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3. A geroscience approach for Parkinson’s disease: Conceptual framework and design of PROPAG-AGEING project

4. Identification of novel risk loci and causal insights for sporadic Creutzfeldt-Jakob disease: a genome-wide association study

8. Paradigm Shift in Gastric Cancer Prevention: Harnessing the Potential of Aristolochia olivieri Extract

13. Genetic defects of gamma‐secretase genes in a multiple myeloma patient with high and dysregulated BCMA surface density: A case report.

16. Harmonizing Genetic Testing for Parkinson's Disease: Results of the PARKNET Multicentric Study.

20. Co-occurrence of amyotrophic lateral sclerosis and Leber’s hereditary optic neuropathy: is mitochondrial dysfunction a modifier?

21. Dementia-related genetic variants in an Italian population of early-onset Alzheimer’s disease

25. Metabolite and lipoprotein profiles reveal sex-related oxidative stress imbalance in de novo drug-naive Parkinson's disease patients

29. Heterogeneity of prodromal Parkinson symptoms in siblings of Parkinson disease patients

32. A mutation in PAK3 with a dual molecular effect deregulates the RAS/MAPK pathway and drives an X-linked syndromic phenotype

34. A geroscience approach for Parkinson’s disease: Conceptual framework and design of PROPAG-AGEING project

36. Cell signaling pathways in autosomal-dominant leukodystrophy (ADLD): the intriguing role of the astrocytes

37. Comparison between plasma and cerebrospinal fluid biomarkers for the early diagnosis and association with survival in prion disease

39. Genome-wide association study identifies risk variants for sporadic Creutzfeldt-Jakob disease in STX6 and GAL3ST1

40. The genetic and metabolic signature of oncocytic transformation implicates HIF1α destabilization

41. MOESM1 of CSF biomarkers of neuroinflammation in distinct forms and subtypes of neurodegenerative dementia

42. Targeted sequencing panels in Italian ALS patients support different etiologies in the ALS/FTD continuum.

43. Additional file 1: of The characterization of AD/PART co-pathology in CJD suggests independent pathogenic mechanisms and no cross-seeding between misfolded Aβ and prion proteins

44. Additional file 4: of The characterization of AD/PART co-pathology in CJD suggests independent pathogenic mechanisms and no cross-seeding between misfolded Aβ and prion proteins

45. Additional file 5: of The characterization of AD/PART co-pathology in CJD suggests independent pathogenic mechanisms and no cross-seeding between misfolded Aβ and prion proteins

46. Additional file 3: of The characterization of AD/PART co-pathology in CJD suggests independent pathogenic mechanisms and no cross-seeding between misfolded Aβ and prion proteins

47. Additional file 6: of The characterization of AD/PART co-pathology in CJD suggests independent pathogenic mechanisms and no cross-seeding between misfolded Aβ and prion proteins

48. Additional file 8: of The characterization of AD/PART co-pathology in CJD suggests independent pathogenic mechanisms and no cross-seeding between misfolded Aβ and prion proteins

49. Additional file 2: of The characterization of AD/PART co-pathology in CJD suggests independent pathogenic mechanisms and no cross-seeding between misfolded Aβ and prion proteins

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