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1. Stratified analyses refine association between TLR7 rare variants and severe COVID-19

2. Integrated CRISPR screening and drug profiling identifies combination opportunities for EGFR, ALK, and BRAF/MEK inhibitors

3. Preclinical evaluation of a COVID-19 vaccine candidate based on a recombinant RBD fusion heterodimer of SARS-CoV-2

4. Next-generation characterization of the Cancer Cell Line Encyclopedia

5. E3 ubiquitin ligase Atrogin-1 mediates adaptive resistance to KIT-targeted inhibition in gastrointestinal stromal tumor

6. Whole-genome analysis of Nigerian patients with breast cancer reveals ethnic-driven somatic evolution and distinct genomic subtypes

7. Targeting FGFR overcomes EMT-mediated resistance in EGFR mutant non-small cell lung cancer

8. The landscape of cancer cell line metabolism

9. Subtype-specific genomic alterations define new targets for soft-tissue sarcoma therapy

10. Differential sensitivity of melanoma cell lines with BRAFV600E mutation to the specific raf inhibitor PLX4032

11. Functional copy-number alterations in cancer.

12. Author Correction: Characterization of Nigerian breast cancer reveals prevalent homologous recombination deficiency and aggressive molecular features

15. Supplementary Tables 1 - 12, Figures 1 - 7 from Wnt-Pathway Activation in Two Molecular Classes of Hepatocellular Carcinoma and Experimental Modulation by Sorafenib

16. Supplementary Information from Focal Gains of VEGFA and Molecular Classification of Hepatocellular Carcinoma

17. Supplementary Methods from Gastrointestinal Adenocarcinomas of the Esophagus, Stomach, and Colon Exhibit Distinct Patterns of Genome Instability and Oncogenesis

18. Data from EGF816 Exerts Anticancer Effects in Non–Small Cell Lung Cancer by Irreversibly and Selectively Targeting Primary and Acquired Activating Mutations in the EGF Receptor

19. Data from Gastrointestinal Adenocarcinomas of the Esophagus, Stomach, and Colon Exhibit Distinct Patterns of Genome Instability and Oncogenesis

20. Supplementary Figure 14 from Gastrointestinal Adenocarcinomas of the Esophagus, Stomach, and Colon Exhibit Distinct Patterns of Genome Instability and Oncogenesis

21. Data from Focal Gains of VEGFA and Molecular Classification of Hepatocellular Carcinoma

22. Supplementary Note 1 from Gastrointestinal Adenocarcinomas of the Esophagus, Stomach, and Colon Exhibit Distinct Patterns of Genome Instability and Oncogenesis

23. Supplementary Legends for Figures 1-14, and Tables 1-6 from Gastrointestinal Adenocarcinomas of the Esophagus, Stomach, and Colon Exhibit Distinct Patterns of Genome Instability and Oncogenesis

24. Supplementary Note 3 from Gastrointestinal Adenocarcinomas of the Esophagus, Stomach, and Colon Exhibit Distinct Patterns of Genome Instability and Oncogenesis

25. Supplementary Figures 1-13 from Gastrointestinal Adenocarcinomas of the Esophagus, Stomach, and Colon Exhibit Distinct Patterns of Genome Instability and Oncogenesis

26. Supplementary Tables 1-6 from Gastrointestinal Adenocarcinomas of the Esophagus, Stomach, and Colon Exhibit Distinct Patterns of Genome Instability and Oncogenesis

27. Supplementary Figures 1 through 5 from EGF816 Exerts Anticancer Effects in Non–Small Cell Lung Cancer by Irreversibly and Selectively Targeting Primary and Acquired Activating Mutations in the EGF Receptor

28. Supplementary Figure Legends from EGF816 Exerts Anticancer Effects in Non–Small Cell Lung Cancer by Irreversibly and Selectively Targeting Primary and Acquired Activating Mutations in the EGF Receptor

29. Supplementary Note 2 from Gastrointestinal Adenocarcinomas of the Esophagus, Stomach, and Colon Exhibit Distinct Patterns of Genome Instability and Oncogenesis

30. Supplementary Table 1 from EGF816 Exerts Anticancer Effects in Non–Small Cell Lung Cancer by Irreversibly and Selectively Targeting Primary and Acquired Activating Mutations in the EGF Receptor

31. Supplementary Material and Methods from EGF816 Exerts Anticancer Effects in Non–Small Cell Lung Cancer by Irreversibly and Selectively Targeting Primary and Acquired Activating Mutations in the EGF Receptor

32. Plasma acylcarnitines and gut‐derived aromatic amino acids as sex‐specific hub metabolites of the human aging metabolome

33. A second update on mapping the human genetic architecture of COVID-19

34. Characterization of Nigerian breast cancer reveals prevalent homologous recombination deficiency and aggressive molecular features

35. Addendum: The Cancer Cell Line Encyclopedia enables predictive modelling of anticancer drug sensitivity

37. Charting the Spatial Landscape of Cancer Hallmarks

38. Detailed stratified GWAS analysis for severe COVID-19 in four European populations

39. Detailed stratified GWAS analysis for severe COVID-19 in four European populations

40. Correction to: Whole-brain dynamics in aging: disruptions in functional connectivity and the role of the rich club

41. The effect of external stimulation on functional networks in the aging healthy human brain

42. Systematic investigation of genetic vulnerabilities across cancer cell lines reveals lineage-specific dependencies in ovarian cancer

43. Assessing the Significance of Chromosomal Aberrations in Cancer: Methodology and Application to Glioma

44. effect of external stimulation on functional networks in the aging healthy human brain.

45. Pharmacogenomic agreement between two cancer cell line data sets

46. Preclinical efficacy, safety, and immunogenicity of a COVID-19 vaccine candidate based on a recombinant RBD fusion heterodimer of SARS-CoV-2

47. The effect of external stimulation on functional networks in the aging healthy human brain

49. E3 ubiquitin ligase Atrogin-1 mediates adaptive resistance to KIT-targeted inhibition in gastrointestinal stromal tumor

50. Obesity-associated deficits in inhibitory control are phenocopied to mice through gut microbiota changes in one-carbon and aromatic amino acids metabolic pathways

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