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32 results on '"Búrbano G"'

6. Epílogo

Author

Burbano González, David

Published
2020

22. Introducción

Author

Burbano González, David

Published
2020

24. Portadilla

Author

Burbano González, David

Published
2020

27. Learning Analytics in M-learning: Periodontic Education

Author

Burbano G., Diana C., Soler, Jaime Alvarez, Filipe, Joaquim, Editorial Board Member, Ghosh, Ashish, Editorial Board Member, Prates, Raquel Oliveira, Editorial Board Member, Zhou, Lizhu, Editorial Board Member, Mata-Rivera, Miguel Félix, editor, Zagal-Flores, Roberto, editor, and Barria-Huidobro, Cristian, editor

Published
2020
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28. 1964MO Alliance A031501: AMBASSADOR study of adjuvant pembrolizumab (Pembro) in muscle-invasive urothelial carcinoma (MIUC) vs observation (Obs): Extended follow-up disease-free survival (DFS) results and metastatic (met) disease recurrence distribution

Author

Apolo, A.B., Ballman, K., Sonpavde, G.P., Berg, S., Kim, W., Parikh, R., Teo, M.Y., Sweis, R.F., Geynisman, D., Grivas, P., Chatta, G., Reichert, Z., Kim, J.W., Bilen, M.A., McGregor, B.A., Srinivas, S., Halabi, S., Perez Burbano, G., Morris, M.J., and Rosenberg, J.E.

Published
2024
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29. Insulin resistance in offspring of hypertensive subjects

Author

Gordon F, M. E. Fonseca, Dolores Mino, Lifshitz A, Revilla C, Wacher N, Búrbano G, and Amato D

Subjects
Adult, Male, medicine.medical_specialty, Physiology, Offspring, medicine.medical_treatment, Blood Pressure, Insulin resistance, Internal medicine, Internal Medicine, medicine, Humans, Obesity, Family history, Pancreatic hormone, Aged, Family Health, business.industry, Insulin, Middle Aged, medicine.disease, Logistic Models, Endocrinology, Blood pressure, Socioeconomic Factors, Hypertension, Female, Insulin Resistance, Cardiology and Cardiovascular Medicine, business, Body mass index
Abstract

OBJECTIVE To assess whether apparently healthy subjects with a family history of systemic hypertension have a higher risk of presenting the insulin resistance syndrome. SUBJECTS Three hundred and eighty-six subjects aged 20-65 years. SETTING A middle socio-economic class urban community from Mexico City. METHOD All subjects and, when necessary, their first-degree relatives, answered a questionnaire and underwent a physical examination with measurement of height, weight and blood pressure. Serum insulin, glucose, cholesterol and triglycerides were measured during fasting and 2 h after an oral load of 75 g glucose. RESULTS A family history of systemic hypertension was present for 167 (43%) of the subjects, of whom 123 (31%) were obese. Subjects with a family history of hypertension had higher systolic blood pressures than did those without such a history (120 +/- 15 versus 115 +/- 10 mmHg). In the logistic regression model, the body mass index and age showed statistically significant effects on the fasting glucose:insulin ratio and on serum insulin levels after an oral load of glucose. When men and women were analysed separately, only in men were higher systolic and mean blood pressures and lower glucose:insulin ratios observed. In the logistic regression analysis the body mass index was a significant predictor of the glucose:insulin ratio and serum insulin levels after an oral load of glucose, especially in men. CONCLUSION Apparently healthy male offspring of hypertensive parents have higher blood pressure levels and lower insulin sensitivities than do offspring of normotensive parents. Insulin resistance was related to obesity, but not to a family history of hypertension, as had previously been reported by other research groups.

Published
1996

32. Relationship of insulin resistance and overweight with cortisol and dehydroepiandrosterone-sulfate levels.

Author

Mino D, Amato D, Cuevas ML, Fonseca ME, Búrbano G, Wacher N, and Lifshitz A

Subjects
Adult, Age Factors, Blood Glucose analysis, Body Mass Index, Body Weight, Cholesterol blood, Female, Humans, Insulin blood, Male, Middle Aged, Regression Analysis, Sex Factors, Time Factors, Triglycerides blood, Dehydroepiandrosterone Sulfate blood, Hydrocortisone blood, Insulin Resistance, Obesity
Abstract

Background: Our objective was to assess the relationship of hormones such as cortisol and dehydroepiandrosterone-sulfate (DHEA-S) with insulin resistance and overweight., Methods: We designed and conducted a cross-sectional, observational survey consisting of home visits within a previously defined area of Mexico City. The study included 303 apparently healthy volunteers from a middle-class socioeconomic urban community. We measured glucose, cholesterol, triglycerides, insulin, cortisol, and DHEA-S. Insulin resistance (IR) was defined as belonging to the first quartile of fasting glucose/insulin ratio (G/IR) distribution or fourth quartile of IR (HOMA). Overweight was defined as body mass index (BMI) > or =25 kg/m(2)., Results: To predict IR in women < or =35 years of age, principal component analysis (PCA) disclosed three components: 1) cholesterol, BMI, and diastolic blood pressure (DBP); 2) cholesterol, triglycerides, and cortisol, and 3) dehydroepiandrosterone-sulfate [DHEA-S]. Solely the latter (DHEA-S) was significantly associated with IR (odds ratio [OR] = 1.80, confidence interval 95% [CI 95%] 1.11-2.91, p = 0.015). For men < or =35 years of age, there were two components: 1) cholesterol, triglycerides, BMI, and DBP, and 2) DHEA-S, cholesterol, and cortisol. Component 1 was significantly associated with IR (OR = 5.65; CI 95% 1.62-19.65, p = 0.006). To predict overweight in women >35 years of age, there were three components, including 1) cholesterol and triglycerides, 2) cortisol, and 3) DHEA-S and G/IR. Component 2 was significantly associated with overweight (OR = 0.38, CI 95% 0.23-0.64, p = 0.000)., Conclusions: In women < or =35 years of age, high DHEA-S levels were associated with insulin resistance, which suggests that in young women DHEA-S exerts anti-estrogenic action, perhaps caused by its competitive binding with the estrogen receptor. Additionally, in women >35 years of age, low cortisol levels were associated with overweight. These associations were not identified for the male subgroup.

Published
2002
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