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1. Macrophage-specific inhibition of the histone demethylase JMJD3 decreases STING and pathologic inflammation in diabetic wound repair

3. Coronavirus induces diabetic macrophage-mediated inflammation via SETDB2

4. Diabetic Wound Keratinocytes Induce Macrophage JMJD3-Mediated Nlrp3 Expression via IL-1R Signaling.

5. Anatomy and Pathophysiology of Venous Thoracic Outlet Syndrome

12. IL-17A-mediated JMJD3 regulation of integrin alpha 3 gene expression alters migration of diabetic keratinocytes and impairs wound repair.

13. The Histone Methyltransferase MLL1/KMT2A in Monocytes Drives Coronavirus-Associated Coagulopathy and Inflammation

19. IFN-κ is critical for normal wound repair and is decreased in diabetic wounds

21. Abstract 114: The Epigenetic Enzyme KMT2A/MLL1 Is A Driver Of Coronavirus Associated Coagulopathy

23. Interferon kappa (IFNk) in keratinocytes is critical for normal wound repair and is decreased in diabetic wounds

24. Incivility, Work Withdrawal, and Organizational Commitment Among US Surgeons.

26. Dextran-Mimetic Quantum Dots for Multimodal Macrophage ImagingIn Vivo, Ex Vivo, andIn Situ

28. The Epigenetic Enzyme KMT2A/MLL1 Is a Driver of Coronavirus-associated Coagulopathy

36. Inhibition of macrophage histone demethylase JMJD3 protects against abdominal aortic aneurysms

37. Dextran-Mimetic Quantum Dots for Multimodal Macrophage Imaging In Vivo, Ex Vivo, and In Situ.

38. Epigenetic regulation of the PGE2 pathway modulates macrophage phenotype in normal and pathologic wound repair

42. Epigenetic Regulation of TLR4 in Diabetic Macrophages Modulates Immunometabolism and Wound Repair

47. Recombinant Production of TEV Cleaved Human Parathyroid Hormone

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