196 results on '"Attrée, Ina"'
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2. Insertion sequences drive the emergence of a highly adapted human pathogen
3. Molecular Mechanisms Involved in Pseudomonas aeruginosa Bacteremia
4. The core and accessory Hfq interactomes across Pseudomonas aeruginosa lineages
5. Publisher Correction: Self-association of MreC as a regulatory signal in bacterial cell wall elongation
6. Cross-regulation and cross-talk of conserved and accessory two-component regulatory systems orchestrate Pseudomonas copper resistance
7. The PopN Gate-keeper Complex Acts on the ATPase PscN to Regulate the T3SS Secretion Switch from Early to Middle Substrates in Pseudomonas aeruginosa
8. Exolysin (ExlA) from Pseudomonas aeruginosa Punctures Holes into Target Membranes Using a Molten Globule Domain
9. The bacterial toxin ExoU requires a host trafficking chaperone for transportation and to induce necrosis
10. Self-association of MreC as a regulatory signal in bacterial cell wall elongation
11. Cross-regulation and cross-talk of conserved and accessory two-component regulatory systems orchestrate Pseudomonas copper resistance
12. The regulation of bacterial two‐partner secretion systems
13. P. aeruginosa Type VI Secretion Machinery: Another Deadly Syringe
14. Colistin resistance mutations in phoQ can sensitize Klebsiella pneumoniae to IgM-mediated complement killing
15. Genome-wide screen in human plasma identifies multifaceted complement evasion of Pseudomonas aeruginosa
16. Genome-wide screen in human plasma identifies multifaceted complement evasion of Pseudomonas aeruginosa
17. A Type III Secretion Negative Clinical Strain of Pseudomonas aeruginosa Employs a Two-Partner Secreted Exolysin to Induce Hemorrhagic Pneumonia
18. Membrane and Chaperone Recognition by the Major Translocator Protein PopB of the Type III Secretion System of Pseudomonas aeruginosa
19. Colistin resistance mutations in phoQ sensitize Klebsiella pneumoniae to IgM-mediated complement killing
20. Current Fluctuation Analysis of the PopB and PopD Translocon Components of the Pseudomonas aeruginosa Type III Secretion System
21. Multiple Pseudomonas species secrete exolysin‐like toxins and provoke Caspase‐1‐dependent macrophage death
22. Defining Lipoprotein Localisation by Fluorescence Microscopy
23. Genomic erosion and horizontal gene transfer shape functional differences of the ExlA toxin in Pseudomonas spp.
24. Structural Characterization and Membrane Localization of ExsB from the Type III Secretion System (T3SS) of Pseudomonas aeruginosa
25. Structure of the Heterotrimeric Complex That Regulates Type III Secretion Needle Formation
26. Protective Anti-V Antibodies Inhibit Pseudomonas and Yersinia Translocon Assembly within Host Membranes
27. Structural Basis of Chaperone Recognition of Type III Secretion System Minor Translocator Proteins
28. Genotypic and Phenotypic Analysis of Type III Secretion System in a Cohort of Pseudomonas aeruginosa Bacteremia Isolates: Evidence for a Possible Association between O Serotypes and exo Genes
29. Molecular features underlying Pseudomonas aeruginosa persistence in human plasma
30. Anti-activator ExsD Forms a 1:1 Complex with ExsA to Inhibit Transcription of Type III Secretion Operons
31. P. aeruginosa Type VI Secretion Machinery: Another Deadly Syringe
32. Determination of the two-component systems regulatory network reveals core and accessory regulations across Pseudomonas aeruginosa lineages
33. Host phospholipid peroxidation fuels ExoU-dependent cell necrosis and supports Pseudomonas aeruginosa-driven pathology
34. Vfr or CyaB promote the expression of the pore-forming toxin exlBA operon in Pseudomonas aeruginosa ATCC 9027 without increasing its virulence in mice
35. Oligomerization of PcrV and LcrV, Protective Antigens of Pseudomonas aeruginosa and Yersinia pestis
36. MagC is a NplC/P60‐like member of the α‐2‐macroglobulin Mag complex of Pseudomonas aeruginosa that interacts with peptidoglycan
37. Species-specific recruitment of transcription factors dictates toxin expression
38. Host phospholipid peroxidation fuels ExoU-dependent cell necrosis and supports Pseudomonas aeruginosa-driven pathology
39. Host phospholipid peroxidation fuels ExoU-dependent cell necrosis and supports Pseudomonas aeruginosa-driven pathology
40. Transcription Inhibitors with XRE DNA-Binding and Cupin Signal-Sensing Domains Drive Metabolic Diversification in Pseudomonas
41. Phospholipid peroxidation fuels ExoU phospholipase-dependent cell necrosis and supportsPseudomonas aeruginosa-driven pathology
42. Pore Formation by T3SS Translocators: Liposome Leakage Assay
43. Horizontal Gene Transfer and Genomic Erosion Shape Functional Differences of the Two-Partner Secretion Toxin Exla in the Genus Pseudomonas
44. Bacterial behavior in human blood reveals complement evaders with some persister-like features
45. The bacterial toxin ExoU requires a host trafficking chaperone for transportation and to induce necrosis
46. Inflammasome activation byPseudomonas aeruginosa'sExlApore‐forming toxin is detrimental for the host
47. Bacterial behavior in human blood reveals complement evaders with persister-like features
48. Transcription inhibitors with XRE DNA-binding and cupin signal-sensing domains drive metabolic diversification in Pseudomonas
49. The PscE-PscF-PscG Complex Controls Type III Secretion Needle Biogenesis in Pseudomonas aeruginosa
50. Bacterial behavior in human blood reveals complement evaders with some persister-like features.
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