45 results on '"Astarie-Dequeker, Catherine"'
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2. Parallel in vivo experimental evolution reveals that increased stress resistance was key for the emergence of persistent tuberculosis bacilli
3. The conical shape of DIM lipids promotes Mycobacterium tuberculosis infection of macrophages
4. Natural mutations in the sensor kinase of the PhoPR two-component regulatory system modulate virulence of ancestor-like tuberculosis bacilli
5. Evolutionary history of tuberculosis shaped by conserved mutations in the PhoPR virulence regulator
6. The role of mycobacterial lipids in host pathogenesis
7. Rv0180c contributes to Mycobacterium tuberculosis cell shape and to infectivity in mice and macrophages
8. The impact of the absence of glycopeptidolipids on the ultrastructure, cell surface and cell wall properties, and phagocytosis of Mycobacterium smegmatis
9. From environmental bacteria to obligate pathogen: the study of adaptations enhancing the persistence of tuberculosis bacilli
10. Phthiocerol Dimycocerosates From Mycobacterium tuberculosis Increase the Membrane Activity of Bacterial Effectors and Host Receptors
11. The Lipid Virulence Factors of Mycobacterium tuberculosis Exert Multilayered Control over Autophagy-Related Pathways in Infected Human Macrophages
12. Mycobacteria use their surface-exposed glycolipids to infect human macrophages through a receptor-dependent process
13. Tyrosine phosphatase MptpA of Mycobacterium tuberculosis inhibits phagocytosis and increases actin polymerization in macrophages
14. CR3 Engaged by PGL-I Triggers Syk-Calcineurin-NFATc to Rewire the Innate Immune Response in Leprosy
15. Activation of the Lysosome-Associated p61Hck Isoform Triggers the Biogenesis of Podosomes
16. Surface-exposed Glycopeptidolipids of Mycobacterium smegmatis Specifically Inhibit the Phagocytosis of Mycobacteria by Human Macrophages: IDENTIFICATION OF A NOVEL FAMILY OF GLYCOPEPTIDOLIPIDS
17. From environmental bacteria to obligate pathogen: the study of adaptations enhancing the persistence of tuberculosis bacilli
18. The conical shape of DIM lipids promotes Mycobacterium tuberculosis infection of macrophages
19. Referee report. For: Pathogenic mycobacteria achieve cellular persistence by inhibiting the Niemann-Pick Type C disease cellular pathway [version 2; referees: 4 approved]
20. Mycobacterial Phenolic Glycolipids Selectively Disable TRIF-Dependent TLR4 Signaling in Macrophages
21. Referee report. For: Pathogenic mycobacteria achieve cellular persistence by inhibiting the Niemann-Pick Type C disease cellular pathway [version 1; referees: 2 approved, 2 approved with reservations]
22. ESX-1 and phthiocerol dimycocerosates ofMycobacterium tuberculosisact in concert to cause phagosomal rupture and host cell apoptosis
23. Trisaccharides of Phenolic Glycolipids Confer Advantages to Pathogenic Mycobacteria through Manipulation of Host-Cell Pattern-Recognition Receptors
24. Multiple deletions in the polyketide synthase gene repertoire of Mycobacterium tuberculosis reveal functional overlap of cell envelope lipids in host-pathogen interactions
25. ESX-1 and phthiocerol dimycocerosates of Mycobacterium tuberculosis act in concert to cause phagosomal rupture and host cell apoptosis.
26. Playing hide-and-seek with host macrophages through the use of mycobacterial cell envelope phthiocerol dimycocerosates and phenolic glycolipids
27. Multiple deletions in the polyketide synthase gene repertoire ofMycobacterium tuberculosisreveal functional overlap of cell envelope lipids in host-pathogen interactions
28. Mycobacterium leprae Phenolglycolipid-1 Expressed by Engineered M. bovis BCG Modulates Early Interaction with Human Phagocytes
29. Phthiocerol Dimycocerosates of M. tuberculosis Participate in Macrophage Invasion by Inducing Changes in the Organization of Plasma Membrane Lipids
30. The protein tyrosine kinase Hck is located on lysosomal vesicles that are physically and functionally distinct from CD63-positive lysosomes in human macrophages
31. Nuclear gene OPA1, encoding a mitochondrial dynamin-related protein, is mutated in dominant optic atrophy
32. Lipoarabinomannans Activate the Protein Tyrosine Kinase Hck in Human Neutrophils
33. The Mannose Receptor Mediates Uptake of Pathogenic and Nonpathogenic Mycobacteria and Bypasses Bactericidal Responses in Human Macrophages
34. Fusion of Azurophil Granules with Phagosomes and Activation of the Tyrosine Kinase Hck Are Specifically Inhibited During Phagocytosis of Mycobacteria by Human Neutrophils
35. Expression of Azurophil and specific granule proteins during differentiation of NB4 cells in neutrophils
36. Endothelin-3, Ca2+ mobilization and cyclic GMP content in human platelets
37. Endothelin-3 Decreases Ca2+ Uptake in Platelet Membrane Vesicles
38. Different effects of endothelin-3 on the Ca2+ discharge induced by agonists and Ca2+-ATPase inhibitors in human platelets
39. Inhibitory Effect of Trimetazidine on Thrombin-Induced Aggregation and Calcium Entry into Human Platelets
40. In vitro inhibition by endothelins of thrombin‐induced aggregation and Ca2+ mobilization in human platelets
41. Endothelin-3 Decreases Ca2+ Uptake in Platelet Membrane Vesicles.
42. CR3 engaged by PGL-I triggers syk-calcineurin-NFATc to rewire the innate immune response in leprosy
43. Endothelin-3, Ca 2+ mobilization and cyclic GMP content in human platelets
44. CR3 Engaged by PGL-I Triggers Syk-Calcineurin-NFATc to Rewire the Innate Immune Response in Leprosy.
45. Enhanced expression and activity of DNA polymerase beta in chronic myelogenous leukemia.
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