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1. Mannose metabolism inhibition sensitizes acute myeloid leukaemia cells to therapy by driving ferroptotic cell death

2. Maintenance of hematopoietic stem cells by tyrosine-unphosphorylated STAT5 and JAK inhibition

3. Unbiased cell surface proteomics identifies SEMA4A as an effective immunotherapy target for myeloma

4. KAT2A complexes ATAC and SAGA play unique roles in cell maintenance and identity in hematopoiesis and leukemia

5. Mutational synergy with CREBBP loss in lymphomagenesis identified through forward insertional mutagenesis in a new DLBCL mouse model

6. Genetic or pharmacological inactivation of CREBBP sensitizes B-cell Acute Lymphoblastic Leukemia to Ferroptotic Cell Death upon BCL2 Inhibition

7. Transcriptional variability accelerates pre-leukemia by cell diversification and perturbation of protein synthesis

8. Transcriptional variability accelerates preleukemia by cell diversification and perturbation of protein synthesis

9. Mannose metabolism inhibition sensitizes acute myeloid leukemia cells to cytarabine and FLT3 inhibitor therapy by modulating fatty acid metabolism to drive ferroptotic cell death.

10. Transcriptional variability accelerates pre-leukemia by cell diversification and perturbation of protein synthesis

11. Sequential inverse dysregulation of the RNA helicases DDX3X and DDX3Y facilitates MYC-driven lymphomagenesis

12. Contrasting requirements during disease evolution identify EZH2 as a therapeutic target in AML

13. Contrasting requirements during disease evolution identify EZH2 as a therapeutic target in AML

14. Maintenance of hematopoietic stem cells by non-tyrosine-phosphorylated STAT5 and JAK inhibition

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