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1. PRPF8-mediated dysregulation of hBrr2 helicase disrupts human spliceosome kinetics and 5´-splice-site selection causing tissue-specific defects

3. Retinal cells derived from patients with DRAM2-dependent CORD21 dystrophy exhibit key lysosomal enzyme deficiency and lysosomal content accumulation

4. Evaluation of the impact of iPSC differentiation protocols on transcriptomic signatures

8. Conjunctival epithelial cells resist productive SARS-CoV-2 infection

10. A single cell atlas of human cornea that defines its development, limbal progenitor cells and their interactions with the immune cells

12. Investigating light sensitivity in bipolar disorder (HELIOS-BD)

30. Reproducibility of Molecular Phenotypes after Long-Term Differentiation to Human iPSC-Derived Neurons: A Multi-Site Omics Study

33. Deciphering the spatio-temporal transcriptional and chromatin accessibility of human retinal organoid development at the single cell level

36. Incorporating microglia‐like cells in human induced pluripotent stem cell‐derived retinal organoids

37. Retinal pigment epithelium extracellular vesicles are potent inducers of age‐related macular degeneration disease phenotype in the outer retina

39. Disrupted alternative splicing for genes implicated in splicing and ciliogenesis causes PRPF31 retinitis pigmentosa

44. pRB-Depleted Pluripotent Stem Cell Retinal Organoids Recapitulate Cell State Transitions of Retinoblastoma Development and Suggest an Important Role for pRB in Retinal Cell Differentiation

45. Activation of autophagy reverses progressive and deleterious protein aggregation in PRPF31 patient‐induced pluripotent stem cell‐derived retinal pigment epithelium cells

46. Human Retinal Organoids Provide a Suitable Tool for Toxicological Investigations: A Comprehensive Validation Using Drugs and Compounds Affecting the Retina

47. Conjunctival epithelial cells resist productive SARS-CoV-2 infection

50. Progressive protein aggregation in PRPF31 patient retinal pigment epithelium cells: the mechanism and its reversal through activation of autophagy

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