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5. The IL-22 pathway as target to relieve visceral pain and animal well-being

Author

Maeva Meynier, Elodie Baudu, Manon Defaye, Wawzyniak, Y., Frederic Delbac, Nicolas Barnich, Ardid, D., Philippe Poirier, Romain Villeger, Chatel, J. M., Livrelli, V., Mathilde Bonnet, Frederic carvalho, Microbes, Intestin, Inflammation et Susceptibilité de l'Hôte (M2iSH), Institut National de la Santé et de la Recherche Médicale (INSERM)-Université Clermont Auvergne [2017-2020] (UCA [2017-2020])-Centre de Recherche en Nutrition Humaine d'Auvergne (CRNH d'Auvergne)-Institut National de Recherche pour l’Agriculture, l’Alimentation et l’Environnement (INRAE), Neuro-Dol (Neuro-Dol), Institut National de la Santé et de la Recherche Médicale (INSERM)-Université Clermont Auvergne [2017-2020] (UCA [2017-2020]), Laboratoire Microorganismes : Génome et Environnement (LMGE), and Université Clermont Auvergne [2017-2020] (UCA [2017-2020])-Centre National de la Recherche Scientifique (CNRS)

Subjects
[SDV.BBM.BM]Life Sciences [q-bio]/Biochemistry, Molecular Biology/Molecular biology, [SDV.MP.BAC]Life Sciences [q-bio]/Microbiology and Parasitology/Bacteriology, ComputingMilieux_MISCELLANEOUS
Abstract

International audience

Published
2020

6. Interactive involvement of brain derived neurotrophic factor, nerve growth factor, and calcitonin gene related peptide in colonic hypersensitivity in the rat

Author

Delafoy, L., Gelot, A., Ardid, D., Eschalier, A., Bertrand, C., Doherty, A.M., and Diop, L.

Subjects
Brain-derived neurotrophic factor -- Research, Nerve growth factor -- Research, Calcitonin -- Genetic aspects, Calcitonin -- Research, Peptides -- Research, Colorectal diseases -- Physiological aspects, Colorectal diseases -- Research, Health
Published
2006

15. Impact of the Microbiota on Pain.

Author

Gervason, S., Defaye, M., Ardid, D., Berthon, J.-Y., Altier, C., Filaire, E., and Carvalho, F. A.

Subjects
GUT microbiome, CENTRAL nervous system, VISCERAL pain, NEURODEGENERATION, DEGENERATION (Pathology)
Abstract

Copyright of Douleur et Analgésie is the property of John Libbey Eurotext Ltd. and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

Published
2021
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19. Impact of intestinal dysbiosis on mouse models of colonic hypersensitivity

Author

Carvalho, F., Lashermes, A., Miquel, S., Martin Rosique, Rebeca, Gelor, A., Barnich, N., Eschalier, A., Ardid, D., Thomas, Muriel, Langella, Philippe, Neuro-Dol (Neuro-Dol), Institut National de la Santé et de la Recherche Médicale (INSERM)-Université Clermont Auvergne [2017-2020] (UCA [2017-2020]), Laboratoire Microorganismes : Génome et Environnement (LMGE), Université Clermont Auvergne [2017-2020] (UCA [2017-2020])-Centre National de la Recherche Scientifique (CNRS), MICrobiologie de l'ALImentation au Service de la Santé (MICALIS), Institut National de la Recherche Agronomique (INRA)-AgroParisTech, Microbes, Intestin, Inflammation et Susceptibilité de l'Hôte (M2iSH), Institut National de la Recherche Agronomique (INRA)-Université d'Auvergne - Clermont-Ferrand I (UdA), Neuro-Dol - Clermont Auvergne (Neuro-Dol), Institut National de la Santé et de la Recherche Médicale (INSERM)-Université Clermont Auvergne (UCA), Laboratoire Microorganismes : Génome et Environnement - Clermont Auvergne (LMGE), Université Clermont Auvergne (UCA)-Centre National de la Recherche Scientifique (CNRS), Microbes, Intestin, Inflammation et Susceptibilité de l'Hôte - Clermont Auvergne (M2iSH), Institut National de la Recherche Agronomique (INRA)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Université Clermont Auvergne (UCA)-Centre de Recherche en Nutrition Humaine d'Auvergne (CRNH d'Auvergne), Neuro-Dol - Clermont Auvergne ( Neuro-Dol ), Institut National de la Santé et de la Recherche Médicale ( INSERM ) -Université Clermont Auvergne ( UCA ), Laboratoire Microorganismes : Génome et Environnement - Clermont Auvergne ( LMGE ), Université Clermont Auvergne ( UCA ) -Centre National de la Recherche Scientifique ( CNRS ), MICrobiologie de l'ALImentation au Service de la Santé humaine ( MICALIS ), Institut National de la Recherche Agronomique ( INRA ) -AgroParisTech, Microbes, Intestin, Inflammation et Susceptibilité de l'Hôte - Clermont Auvergne ( M2iSH ), and Université Clermont Auvergne ( UCA )

Subjects
[ SDV ] Life Sciences [q-bio], [SDV]Life Sciences [q-bio], digestive system diseases
Abstract

Impact of intestinal dysbiosis on mouse models of colonic hypersensitivity. NeuroGASTRO 2015

Published
2015

20. Prescription opioid analgesic use in France: Trends and impact on morbidity-mortality.

Author

Chenaf, C., Kaboré, J.-L., Delorme, J., Pereira, B., Mulliez, A., Zenut, M., Delage, N., Ardid, D., Eschalier, A., and Authier, N.

Subjects
CODEINE, THERAPEUTIC use of narcotics, ANALGESICS, OPIUM, OXYCODONE, FENTANYL, TRAMADOL, CHRONIC pain, COMPARATIVE studies, DATABASES, RESEARCH methodology, MEDICAL cooperation, MORTALITY, RESEARCH, SUBSTANCE abuse, EVALUATION research, DISEASE prevalence, PROPORTIONAL hazards models, PROPOXYPHENE (Drug), THERAPEUTICS
Abstract

Background: While data from USA and Canada demonstrate an opioid overdose epidemic, very little nation-wide European studies have been published on this topical subject.Methods: Using a nationally representative sample of the French Claims database (>700,000 patients), the exhaustive nationwide hospital discharge database, and national mortality registry, all patients dispensed at least one prescription opioid (PO) in 2004-2017 were identified, to describe trends in PO analgesic use, shopping behaviour, opioid-related hospitalizations and deaths. Annual prevalence of PO use and shopping behaviour (≥1 day of overlapping prescriptions from ≥2 prescribers, dispensed by ≥3 pharmacies) was estimated.Results: In 2004-2017, the annual prevalence of weak opioid use codeine, tramadol and opium rose by 150%, 123%, and 244%, respectively (p < 0.05). Strong opioid use increased from 0.54% to 1.1% (+104%, p < 0.05), significantly for oxycodone (+1950%). Strong opioid use in chronic noncancer pain rose by 88% (p < 0.05) and 1180% for oxycodone. Opioid shopping increased from 0.50% to 0.67% (+34%, p < 0.05), associated with higher mortality risk HR = 2.8 [95% confidence interval (CI): 1.2-6.4]. Opioid-related hospitalizations increased from 15 to 40 per 1,000,000 population (+167%, 2000-2017), and opioid-related deaths from 1.3 to 3.2 per 1,000,000 population (+146%, 2000-2015).Conclusions: This study provided a first European approach to a nationwide estimation with complete access to several national registries. In 2004-2017 in France, PO use excluding dextropropoxyphene more than doubled. The increase in oxycodone and fentanyl use, and nontrivial increasing trend in opioid-related morbidity-mortality should prompt authorities to closely monitor PO consumption in order to prevent alarming increases in opioid-related morbidity-mortality.Significance: In 2004-2017, prescription opioid use in France at least doubled and oxycodone use increased particularly, associated with a nontrivial increase in opioid-related morbidity-mortality. Although giving no indication for an 'opioid epidemic,' these findings call for proper monitoring of opioid use. [ABSTRACT FROM AUTHOR]

Published
2019
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21. Colonic overexpression of the T-type calcium channel Cav3.2 in a mouse model of visceral hypersensitivity and in irritable bowel syndrome patients

Author

Scanzi, J., primary, Accarie, A., additional, Muller, E., additional, Pereira, B., additional, Aissouni, Y., additional, Goutte, M., additional, Joubert-Zakeyh, J., additional, Picard, E., additional, Boudieu, L., additional, Mallet, C., additional, Gelot, A., additional, Ardid, D., additional, Carvalho, F. A., additional, and Dapoigny, M., additional

Published
2016
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22. Anti-nociceptive effect of Faecalibacterium prausnitzii in non-inflammatory IBS-like models

Author

Miquel, S., primary, Martín, R., additional, Lashermes, A., additional, Gillet, M., additional, Meleine, M., additional, Gelot, A., additional, Eschalier, A., additional, Ardid, D., additional, Bermúdez-Humarán, L. G., additional, Sokol, H., additional, Thomas, M., additional, Theodorou, V., additional, Langella, P., additional, and Carvalho, F. A., additional

Published
2016
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23. Résumés des communications libres

Author

Magni, G., Caldieron, C., Rigatti-Luchini, S., Henry, P., Brochet, B., Defontaine-Catteau, M. C., Blond, S., Christiaens, J. L., Pohl, M., Bourgoin, S., Benoliel, J. J., Mauborgne, A., Carayon, A., Hamon, M., Cesselin, F., Ardid, D., Eschalier, A., Lavarenne, J., Sindou, M., Jeanmonod, D., and Maugière, F.

Published
1989
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24. Induction of mu opioid and cannabinoid receptors by probiotics

Author

Rousseaux, C., Thuru, Xavier, Gelot, A., Barnich, N., Neut, C., Dubuquoy, C, Ouwehand, A, Ardid, D., Carcano, D, Desreumaux, P., DAM Île-de-France (DAM/DIF), Direction des Applications Militaires (DAM), Commissariat à l'énergie atomique et aux énergies alternatives (CEA)-Commissariat à l'énergie atomique et aux énergies alternatives (CEA), Physiopathologie des Maladies Inflammatoires de l'Intestin, Institut National de la Santé et de la Recherche Médicale (INSERM)-Université de Lille, Droit et Santé, Centre de Recherche Jean-Pierre AUBERT Neurosciences et Cancer - U1172 Inserm - U837 (JPArc), Institut National de la Santé et de la Recherche Médicale (INSERM)-Université Lille Nord de France (COMUE)-Université de Lille, Cancer Heterogeneity, Plasticity and Resistance to Therapies - UMR 9020 - U 1277 (CANTHER), Institut Pasteur de Lille, Réseau International des Instituts Pasteur (RIIP)-Réseau International des Instituts Pasteur (RIIP)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Université de Lille-Centre Hospitalier Régional Universitaire [Lille] (CHRU Lille)-Centre National de la Recherche Scientifique (CNRS), Neuro-Dol (Neuro-Dol), Institut National de la Santé et de la Recherche Médicale (INSERM)-Université Clermont Auvergne [2017-2020] (UCA [2017-2020]), Microbes, Intestin, Inflammation et Susceptibilité de l'Hôte (M2iSH), Institut National de la Recherche Agronomique (INRA)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Université Clermont Auvergne [2017-2020] (UCA [2017-2020])-Centre de Recherche en Nutrition Humaine d'Auvergne (CRNH d'Auvergne), Université catholique de Lille (UCL), Pharmacologie fondamentale et clinique de la douleur, Université d'Auvergne - Clermont-Ferrand I (UdA)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Université d'Auvergne - Clermont-Ferrand I (UdA)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Université d'Auvergne - Clermont-Ferrand I (UdA)-Institut National de la Santé et de la Recherche Médicale (INSERM), Hôpital Claude Huriez, and Université de Lille, Droit et Santé-Centre Hospitalier Régional Universitaire [Lille] (CHRU Lille)

Subjects
[SDV.BC]Life Sciences [q-bio]/Cellular Biology, ComputingMilieux_MISCELLANEOUS
Abstract

International audience

Published
2007
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26. Potentiation of the antinociceptive effect of clomipramine by a 5-ht1A antagonist in neuropathic pain in rats

Author

Ardid, D, Alloui, A, Brousse, G, Jourdan, D, Picard, P, Dubray, C, and Eschalier, A

Subjects
Male, Pyridines, Antidepressive Agents, Tricyclic, Piperazines, Rats, Rats, Sprague-Dawley, Area Under Curve, Receptors, Serotonin, Papers, Clomipramine, Animals, Drug Therapy, Combination, Serotonin Antagonists, Receptors, Serotonin, 5-HT1, Pain Measurement
Abstract

The benefit of antidepressant treatment in human neuropathic pain is now well documented, but the effect is limited and slow to appear. It has been demonstrated that the association of a 5-HT(1A) antagonist and a serotoninergic antidepressant reduced the delay of action and increases the thymoanaleptic effect of the drug. The purpose of this work was to evaluate the combination of an antidepressant and a 5-HT(1A) antagonist in animal models of chronic neuropathic pain. We studied the antinociceptive effect of the co-administration of clomipramine and a 5-HT(1A) antagonist (WAY 100,635) in a pain test applied in normal rats and in two models of neurogenic sustained pain (mononeuropathic and diabetic rats). The results show an increase in the antinociceptive effect of acutely injected clomipramine due to WAY 100,635 in these models, which is majored when the two drugs are repeatedly injected. The 5-HT(1A) antagonist reduced the delay of onset and increased the maximal antinociceptive effect of clomipramine. These new findings argue for using the combination of an antidepressant and a 5-HT(1A) antagonist in human neuropathic pain therapy.

Published
2001

27. Colonic overexpression of the T-type calcium channel Cav3.2 in a mouse model of visceral hypersensitivity and in irritable bowel syndrome patients.

Author

Scanzi, J., Accarie, A., Muller, E., Pereira, B., Aissouni, Y., Goutte, M., Joubert‐Zakeyh, J., Picard, E., Boudieu, L., Mallet, C., Gelot, A., Ardid, D., Carvalho, F. A., and Dapoigny, M.

Subjects
IRRITABLE colon, VISCERAL pain, GENETIC overexpression, CALCIUM channels, ALLERGIES, INFLAMMATION, IMMUNOFLUORESCENCE, PROTEIN expression
Abstract

Background Among the different mechanisms involved in irritable bowel syndrome ( IBS) physiopathology, visceral hypersensitivity seems to play a key role. It involves sensitization of the colonic primary afferent fibers, especially through an overexpression of ion channels. The aims of this translational study were to investigate the colonic expression of Cav3.2 calcium channels and their involvement in an animal model of colonic hypersensitivity, and to assess their expression in the colonic mucosa of symptomatic IBS patients. Methods This bench-to-bed study combined a preclinical experimental study on mice and a case-control clinical study. Preclinical studies were performed on wild-type and Cav3.2- KO mice. Colonic sensitivity and Cav3.2 expression were studied after a low-dose treatment of dextran sodium sulfate ( DSS 0.5%). Regarding the clinical study, colonic biopsies were performed in 14 IBS patients and 16 controls during a colonoscopy to analyze the mucosal Cav3.2 expression. Key results Wild-type, but not Cav3.2- KO, mice developed visceral hypersensitivity without colonic inflammation, after 0.5% DSS treatment. A significant increase of Cav3.2 mRNA ( p = 0.04) was found in the colon of low-dose DSS-treated wild-type ( WT) mice compared to their controls. In human colonic biopsies, the Cav3.2 mRNA level was significantly higher in the IBS group compared to the control group ( p = 0.01). The immunofluorescence staining revealed their protein expression in colonic mucosa, particularly in nerve fibers. Conclusions & inferences This translational study supports the involvement of the calcium channels Cav3.2 in abdominal pain, as observed in IBS patients. It opens new therapeutic perspectives based on molecules specifically blocking these channels. [ABSTRACT FROM AUTHOR]

Published
2016
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50. Potentiation of the antinociceptive effect of clomipramine by a 5‐ht1Aantagonist in neuropathic pain in rats

Author

Ardid, D, Alloui, A, Brousse, G, Jourdan, D, Picard, P, Dubray, C, and Eschalier, A

Abstract

The benefit of antidepressant treatment in human neuropathic pain is now well documented, but the effect is limited and slow to appear. It has been demonstrated that the association of a 5‐HT1Aantagonist and a serotoninergic antidepressant reduced the delay of action and increases the thymoanaleptic effect of the drug.The purpose of this work was to evaluate the combination of an antidepressant and a 5‐HT1Aantagonist in animal models of chronic neuropathic pain. We studied the antinociceptive effect of the co‐administration of clomipramine and a 5‐HT1Aantagonist (WAY 100,635) in a pain test applied in normal rats and in two models of neurogenic sustained pain (mononeuropathic and diabetic rats).The results show an increase in the antinociceptive effect of acutely injected clomipramine due to WAY 100,635 in these models, which is majored when the two drugs are repeatedly injected. The 5‐HT1Aantagonist reduced the delay of onset and increased the maximal antinociceptive effect of clomipramine.These new findings argue for using the combination of an antidepressant and a 5‐HT1Aantagonist in human neuropathic pain therapy.

Published
2001
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