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141 results on '"Anceschi, Maurizio M."'

3. Guidelines on CMV congenital infection

Author

Coll, Oriol, Benoist, Guillaume, Ville, Yves, Weisman, Leonard E., Botet, Francesc, Anceschi, Maurizio M., Greenough, Anne, Gibbs, Ronald S., and Carbonell-Estrany, Xavier

Published
2009

20. Successful Isolation, Culturing and Differentiation In Vitro and In Vivo of Multipotent Stem Cells (Mps) From Human Fetal Biliary Tree

Author

Semeraro, Rossella, primary, Napoli, Cristina, additional, Torrice, Alessia, additional, Cardinale, Vincenzo, additional, Carpino, Guido, additional, Gentile, Raffaele, additional, Gatto, Manuela, additional, Brunelli, Roberto, additional, Anceschi, Maurizio M., additional, Di Tondo, Ugo, additional, Gaudio, Eugenio, additional, and Alvaro, Domenico, additional

Published
2011
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26. LAMELLAR BODY COUNTS

Author

Neerhof, Mark G., primary, Dohnal, James C., additional, Ashwood, Edward R., additional, Lee, In-Sik, additional, and Anceschi, Maurizio M., additional

Published
2001
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39. Immunoglobulin Therapy of Fetal Cytomegalovirus Infection Occurring in the First Half of PregnancydA Case-Control Study of the Outcome in Children.

Author

Nigro, Giovanni, Adler, Stuart P., Parruti, Giustino, Anceschi, Maurizio M., Coclite, Eleonora, Pezone, Ilaria, and Di Renzo, Gian Carlo

Subjects
CYTOMEGALOVIRUSES, IMMUNOGLOBULINS, HEALTH outcome assessment, FETAL diseases, PREGNANCY, PSYCHOMOTOR disorders
Abstract

Background. Primary cytomegalovirus (CMV) infection early in gestation causes severe disease. Methods. Case patients were 32 congenitally infected children aged 1-5 years who had either hearing deficit and/or psychomotor retardation and whose mothers had a confirmed or probable primary CMV infection at ≤20 weeks' gestation. Control subjects were 32 congenitally infected normal children whose mothers had a confirmed primary infection at ≤20 weeks' gestation. Case patients and control subjects were matched by the weeks of maternal gestation (± week) at the mother's infection and by the child's age (± year) at evaluation. Results. For the case patients and control subjects, the mean age was 3.0 years. The mean number of weeks of gestation at maternal infection was 11 weeks. The only risk factor for an affected child was the mother not receiving immunoglobulin (P = .001). Of the 32 case patients, only 4 mothers received CMV immunoglobulin, compared with 27 of the 32 mothers of control infants (adjusted odds ratio, 14 [95% confidence interval, 1.7-110]). The rate of both psychomotor retardation and hearing deficit decreased with immunoglobulin. Conclusions. These results support the efficacy of immunoglobulins for decreasing the severity of disabilities caused by fetal CMV infection after a primary maternal infection during pregnancy. [ABSTRACT FROM AUTHOR]

Published
2012
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47. CA 125 in Peritoneal Fluid.

Author

Barbati, Antonella, Anceschi, Maurizio M., Di Renzo, Gian Carlo, and Cosmi, Ermelando V.

Published
1992

49. Inositol and Glucocorticoid in the Development of Lung Stability in Male and Female Rabbit Fetuses1

Author

ANCESCHI, MAURIZIO M., PETRELLI, ADRIANO, ZACCARDO, GIUSEPPE, BARBATI, ANTONELLA, RENZO, GIAN CARLO DI, COSMI, ERMELANDO V., and HALLMAN, MIKKO

Abstract

Inasmuch as inositol affects the development of lung surfactant, and exogenous glucocorticoids accelerate fetal lung maturation, a possible interaction of the two substances on alveolar stability of preterm rabbit fetuses of 28 days gestation was investigated. On days 26 and 27 of gestation inositol or glucose were added to the diet of does treated with betamethasone (0.2 mg/kg intramuscularly on days 26 and 27). Inositol increased lung-thorax compliance of paralyzed fetuses at all insufflation pressures studied (from 16 to 22.5 and 30 cm H2O and back to 22.5, and 16 cm H2O). At a ventilation pressure of 30 cm H2O, lung-thorax compliance of fetuses treated with inositol plus betamethasone was more than doubled as compared with controls (1.2 ± 0.6 versus 0.5 ± 0.2 ml/kg ± cm H2O; p < 0.001). Inositol alone had no detectable effect on compliance, whereas betamethasone tended to increase compliance (p = 0.05). According to variance analysis, the effect of inositol was statistically significant only among the males. Inositol prevented the glucocorticoid-induced decrease in lung protein and, to a lesser extent, the decrease in DNA. Inositol did not further increase the lavageable surfactant pool of the glucocorticoid-treated, ventilated fetuses, although the area occupied by lamellar bodies within type II cells was increased after inositol plus betamethasone. According to the present study, inositol modifies the physiologic and biochemical response of the immature fetal lung to a pharmacologic dose of exogenous glucocorticoid.

Published
1988

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