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13 results on '"Amare (Kadam), Pratibha"'

2. Comparative analysis of routine histology, immunohistochemistry, reverse transcriptase polymerase chain reaction, and fluorescence in situ hybridization in diagnosis of Ewing family of tumors

Author

Jambhekar, Nirmala A., Bagwan, Izhar N., Ghule, Prachi, Shet, Tanuja M., Chinoy, Roshni F., Agarwal, Sandhya, Joshi, Rupali, and Amare Kadam, Pratibha S.

Subjects
Ewing's sarcoma -- Research, Ewing's sarcoma -- Diagnosis, Ewing's sarcoma -- Physiological aspects, Histology -- Analysis, Immunohistochemistry -- Analysis, Reverse transcriptase -- Analysis
Published
2006

6. MOLECULAR HETEROGENEITY IN ACUTE PROMYELOCYTIC LEUKEMIA - A SINGLE CENTRE EXPERIENCE FROM INDIA

Author

Rabade, Nikhil, primary, Raval, Goutham, additional, Chaudhary, Shruti, additional, Subramanian, PG, additional, Kodgule, Rohan, additional, Joshi, Swapnali, additional, Tembhare, Prashant, additional, Jain, Hasmukh, additional, Sengar, Manju, additional, Hasan K, Syed, additional, Amare Kadam, Pratibha, additional, Shetty, Dhanalaxmi, additional, Narula, Gaurav, additional, Banavali, Shripad, additional, Gujral, Sumeet, additional, and Patkar, Nikhil, additional

Published
2018
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7. Darier′s disease following radiotherapy for carcinoma of cervix

Author

Chopra Supriya, Sharma Vinay, Nischal K, Khopkar Uday, Baisane Chanda, and Amare (Kadam) Pratibha

Subjects
Cutaneous manifestations, Chromosomal aberrations, lcsh:Dermatology, lcsh:RL1-803, Darier′s disease, Carcinoma cervix
Abstract

Darier-White disease is due to a defect in the ATP2A2 gene encoding the sarcoplasmic/endoplasmic reticulum Ca2+ ATPase (SERCA2b). We report a case of carcinoma cervix in whom Darier′s disease manifested after the initiation of radiation therapy. Conventional cytogenetics on peripheral blood revealed non-clonal constitutional autosomal and X chromosome abnormalities suggesting radiation induced gene toxicity. Occurrence of Darier′s disease in our case could be due to treatment induced sustained differentiation in the Darier′s affected skin by an unknown mechanism. Late onset or sporadic Darier′s disease is the other possibility.

Published
2004

8. Minimal Residual Disease in Acute Myeloid Leukemia Using a Difference from Normal Approach Is Predictive of Outcome

Author

Kakirde, Chinmayee, primary, Kodgule, Rohan, additional, Raval, Goutham, additional, Talole, Sanjay, additional, Kadechkar, Shraddha, additional, Chatterjee, Gaurav, additional, Yajamanam, Badrinath, additional, Ghoghale, Sitaram, additional, Deshpande, Nilesh, additional, Amare Kadam, Pratibha, additional, Menon, Hari, additional, Sengar, Manju, additional, Khattry, Navin, additional, Jain, Hasmukh, additional, Bagal, Bhausaheb, additional, Tembhare, Prashant, additional, Gujral, Sumeet, additional, and Patkar, Nikhil, additional

Published
2016
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9. An Integrated Genomic Classification That Includes Copy Number Alterations Is Highly Predictive of Post Induction Minimal Residual Disease (MRD) Status in Childhood Precursor B Lineage Acute Lymphoblastic Leukemia

Author

Patkar, Nikhil, primary, Subramanian, Papagudi Ganesan, additional, Tembhare, Prashant, additional, Amare Kadam, Pratibha, additional, Bibi, Asma, additional, Chopra, Karishma, additional, Mirgal, Satish, additional, Rambhiya, Sagar, additional, Joshi, Swapnali, additional, Chaudhary, Shruti, additional, Mascrenhas, Russel, additional, Dusseja, Sona, additional, Narula, Gaurav, additional, Arora, Brijesh, additional, Banavali, Shripad, additional, and Gujral, Sumeet, additional

Published
2015
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11. RUNX1 ABERRATIONS IN ETV6/RUNX1-POSITIVE AND ETV6/RUNX1-NEGATIVE PATIENTS: Its Hemato-Pathological and Prognostic Significance in a Large Cohort (619 Cases) of ALL

Author

Pais, Anurita Peter, primary, Amare Kadam, Pratibha S., additional, Raje, Gauri Chandrakant, additional, Banavali, Shripad, additional, Parikh, Purvish, additional, Kurkure, Purna, additional, Arora, Brijesh, additional, Gujral, Sumit, additional, Kumar, S. Ashok, additional, and Badrinath, Y., additional

Published
2008
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12. A Novel, Efficacious Therapeutic Approach to Patients with Acute Promyelocytic Leukemia (APL) Using Differentiating Agent and Metronomic Chemotherapy (CT).

Author

Banavali, Shripad D., primary, Goyal, Lovenish, additional, Nair, Reena, additional, Biswas, Ghanashyam, additional, Amare-Kadam, Pratibha, additional, Baisane, Chanda, additional, Mahadik, Shashikant, additional, Gujral, Sumit, additional, Karanth, Narayan, additional, Arora, Brijesh, additional, Bhagwat, Roshni, additional, Kurkure, Purna, additional, and Parikh, Purvish, additional

Published
2005
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13. Investigation of recurrent deletion loci specific to conventional renal cell carcinoma by comparative allelotyping in major epithelial carcinomas.

Author

Bhat Singh RR and Amare Kadam PS

Abstract

Objective: Loss of heterozygosity (LOH) studies were undertaken to investigate the consistently deleted loci/? tumor suppressor gene loci (TSG) on 3p in conventional renal cell carcinoma (cRCC)., Materials and Methods: LOH studies were performed by polymerase chain reaction (PCR) using 15 micro satellite markers mapped in region 3p12-p26 on 40 paired cRCC tumors and normal kidney at Stages I-IV. Simultaneously, fluorescent in-situ hybridization (FISH) studies were performed to investigate the allelic deletion of fragile histidine triad (FHIT)., Results: Our studies revealed three affected regions; 3p12.2-p14.1, 3p14.2-p21.1, and 3p24.2-p26.1 with differential frequencies in Group I (Stage I and II) and Group II (Stage III and IV). Incidence for D3S1234 (FHIT locus) and D3S2454 (3p13) was 75% and 83% in Group I and II, respectively. Comparative allelotyping in epithelial malignancies like lung, bladder, and breast tumors revealed LOH (frequency 14-20%) only in breast tumors for D3S2406, D3S1766 (distal to FHIT), and D3S1560 (distal to VHL, Von-Hippal Lindau). FISH using FHIT gene probe revealed deletions in cRCC (88%), breast (30%), and lung tumors (10%) with no deletions in bladder tumors and leukemias, signifying the importance of FHIT in the pathogenesis of tumors of epithelial origin., Conclusion: Our findings suggested FHIT deletion as an early and VHL deletion as an early and/or late event in cRCC. Additionally, studies also disclosed the recurrent deletions of flanking loci to FHIT and VHL in cRCC. The dilemma of interstitial or continuous deletion on 3p needs to be resolved by implementation of latest sensitive molecular techniques that would further help to narrow down search for TSG loci specific to cRCC, other than VHL and FHIT.

Published
2012
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