Search

Your search keyword '"Alexandre‐Santos, Beatriz"' showing total 17 results
Start Over Author "Alexandre‐Santos, Beatriz"
17 results on '"Alexandre‐Santos, Beatriz"'

3. Tributyltin-induced visceral adiposity is associated with impaired redox balance in white adipose tissue of male rats

Author

Alexandre-Santos, Beatriz, Mendes, Ana Beatriz Araújo, Reis, Guilherme dos Santos, Alves, Ana Paula de Paula, Freitas, Camila Oliveira, Lima, Gabriel Ferreira, Evangelista, Jefferson Fernandes, Matsuura, Cristiane, Miranda-Alves, Leandro, Nóbrega, Antonio Claudio Lucas da, Magliano, D'Angelo Carlo, Motta, Nadia Alice Vieira da, Brito, Fernanda Carla Ferreira, and Frantz, Eliete Dalla Corte

Published
2024
Full Text
View/download PDF

4. List of contributors

Author

Acosta, Cristian G., primary, Adam, Cristina, additional, Aggarwal, Nikita, additional, Ahmad, Sarfaraz, additional, Alenina, Natalia, additional, Alexandre-Santos, Beatriz, additional, Alfano, Gaetano, additional, Amin, Ghadir, additional, Arora, Sandeep, additional, Asami, Tomoichiro, additional, Azinheira Nobrega Cruz, Nayara, additional, Bader, Michael, additional, Bagang, Newly, additional, Bedi, Onkar, additional, Bellomo, Rinaldo, additional, Bhat, Anjali, additional, Biancardi, Vinicia Campana, additional, Bitencourt, Letícia, additional, Booz, George W., additional, Brito-Toscano, E.C., additional, Carlo Magliano, D'Angelo, additional, Caroline Gonçalves de Oliveira, Lilian, additional, Chandra Bharti, Alok, additional, Chappell, Mark C., additional, Chaudhary, Apoorva, additional, Chhokar, Arun, additional, Claudia, Bregonzio, additional, Corte Frantz, Eliete Dalla, additional, Cudnoch-Jedrzejewska, Agnieszka, additional, Delforce, Sarah J., additional, de Miranda, A.S., additional, de Oliveira Campos, Juliana Lacerda, additional, de Senanayake, Preenie, additional, De Silva, T. Michael, additional, Diab, Reine, additional, Elena Casarini, Dulce, additional, Ferrario, Carlos M., additional, Fischer, Bruna Luisa, additional, Gallagher, Patricia E., additional, Gupta, Kirti, additional, Gupta, Shabarni, additional, Gustavo, Baiardi, additional, Gwathmey, TanYa M., additional, Habeichi, Nada J., additional, Haznedaroglu, Ibrahim C., additional, Iwasaki, Shin-ichi, additional, Janjua, Divya, additional, Jia, Hongpeng, additional, Kangussu, Lucas M., additional, Kashyap, Manoj Kumar, additional, Kathleen Phillips, Jacqueline, additional, Kowara, Michal, additional, Kumar, Manish, additional, Lankadeva, Yugeesh R., additional, Li, Sheran, additional, Li, Yan Chun, additional, Lin, Qing, additional, Loeffler, Ivonne, additional, Lumbers, Eugenie R., additional, Malkan, Umit Yavuz, additional, Marcu, Dragoş Traian, additional, Massoud, Gaelle, additional, May, Clive N., additional, Melo, Ana Clara, additional, Mericskay, Mathias, additional, Morosin, Saije K., additional, Natalia Andrea, Marchese, additional, Okada, Yasuo, additional, Osvaldo Martín, Basmadjian, additional, Patterson, Sean I., additional, Peliky Fontes, Marco Antônio, additional, Pringle, Kirsty G., additional, Rachid, M.A., additional, Ramchandra, Rohit, additional, Ramos, Alberto Javier, additional, Rao, Rashmi, additional, Rocha, N.P., additional, Sahu, Pratap Kumar, additional, Sascău, Radu Andy, additional, See, Emily J., additional, Seltzer, Alicia M., additional, Senrung, Anna, additional, Sepúlveda-Fragoso, Vinícius, additional, Serio, Rosa, additional, Shaltout, Hossam A., additional, Shanks, Julia, additional, Silveira de Queiroz Campos, Mauro, additional, Simões-e-Silva, Ana Cristina, additional, Singh, Gaaminepreet, additional, Soares de Brito, Stephanie Bruna Camilo, additional, Sobey, Christopher G., additional, Sriramula, Srinivas, additional, Stătescu, Cristian, additional, Subudhi, Bharat Bhusan, additional, Szczepanska-Sadowska, Ewa, additional, Şalaru, Delia Lidia, additional, Tallant, E. Ann, additional, Teixeira, A.L., additional, Thakur, Kulbhushan, additional, Toya, Shuji, additional, Tripathi, Tanya, additional, Valdez, Susana R., additional, Vaz de Castro, Pedro Alves Soares, additional, Victoria Belén, Occhieppo, additional, VonCannon, Jessica L., additional, Wolf, Gunter, additional, Wong Zhang, David E., additional, Wright, Kendra N., additional, Yadav, Joni, additional, Yamaleyeva, Liliya M., additional, Yoshimura, Ken, additional, Zera, Tymoteusz, additional, Zizzo, Maria Grazia, additional, and Zouein, Fouad A., additional

Published
2023
Full Text
View/download PDF

5. Angiotensins in obesity

Author

Alexandre-Santos, Beatriz, primary, Sepúlveda-Fragoso, Vinícius, additional, Carlo Magliano, D'Angelo, additional, and Corte Frantz, Eliete Dalla, additional

Published
2023
Full Text
View/download PDF

7. Obesity‐induced skeletal muscle remodeling: A comparative analysis of exercise training and ACE‐inhibitory drug in male mice

Author

Proença, Ana Beatriz, primary, Alexandre‐Santos, Beatriz, additional, Giori, Isabele Gomes, additional, Alex‐Marques, Jaime Silva Filho, additional, Machado‐Santos, Clarice, additional, Machado, Marcus, additional, Magliano, D'Angelo Carlo, additional, da Nobrega, Antonio Claudio Lucas, additional, and Frantz, Eliete Dalla Corte, additional

Published
2024
Full Text
View/download PDF

8. Comparison between aerobic exercise training and enalapril treatment as tools to improve diet-induced metabolic-associated fatty liver disease: Effects on endoplasmic reticulum stress markers

Author

Sepúlveda-Fragoso, Vinicius, Alexandre-Santos, Beatriz, Barreto-Reis, Emanuelle, Fernandes, Tiago, Oliveira, Edilamar Menezes, Cavalcanti, Soraia das Neves Vieira, Atella, Georgia Correa, Miranda-Alves, Leandro, Vázquez-Carrera, Manuel, Nóbrega, Antonio Claudio Lucas da, Frantz, Eliete Dalla Corte, and Magliano, D'Angelo Carlo

Published
2022
Full Text
View/download PDF

11. Adipose tissue plasticity mediated by the counterregulatory axis of the renin‐angiotensin system: Role of Mas and MrgD receptors.

Author

Proença, Ana Beatriz, Medeiros, Gabriela Rodrigues, Reis, Guilherme dos Santos, Losito, Luiza da França, Ferraz, Luiza Mazzali, Bargut, Thereza Cristina Lonzetti, Soares, Nícia Pedreira, Alexandre‐Santos, Beatriz, Campagnole‐Santos, Maria Jose, Magliano, D'Angelo Carlo, Nobrega, Antonio Claudio Lucas da, Santos, Robson Augusto Souza, and Frantz, Eliete Dalla Corte

Subjects
RENIN-angiotensin system, BROWN adipose tissue, WHITE adipose tissue, ADIPOSE tissues, ENDOCRINE system, ANGIOTENSIN receptors, G protein coupled receptors, ADIPOGENESIS
Abstract

The renin‐angiotensin system (RAS) is an endocrine system composed of two main axes: the classical and the counterregulatory, very often displaying opposing effects. The classical axis, primarily mediated by angiotensin receptors type 1 (AT1R), is linked to obesity‐associated metabolic effects. On the other hand, the counterregulatory axis appears to exert antiobesity effects through the activation of two receptors, the G protein‐coupled receptor (MasR) and Mas‐related receptor type D (MrgD). The local RAS in adipose organ has prompted extensive research into white adipose tissue and brown adipose tissue (BAT), with a key role in regulating the cellular and metabolic plasticity of these tissues. The MasR activation favors the brown plasticity signature in the adipose organ by improve the thermogenesis, adipogenesis, and lipolysis, decrease the inflammatory state, and overall energy homeostasis. The MrgD metabolic effects are related to the maintenance of BAT functionality, but the signaling remains unexplored. This review provides a summary of RAS counterregulatory actions triggered by Mas and MrgD receptors on adipose tissue plasticity. Focus on the effects related to the morphology and function of adipose tissue, especially from animal studies, will be given targeting new avenues for treatment of obesity‐associated metabolic effects. [ABSTRACT FROM AUTHOR]

Published
2024
Full Text
View/download PDF

13. Potential role of endoplasmic reticulum stress in the pathophysiology of polycystic ovary syndrome.

Author

Alexandre-Santos, Beatriz, Martins, Felipe Figuerôa Tassinari Rocha, da Silva Gonçalves, Larissa, de Oliveira Guimarães, Clara, Carla-Ruggiero, Fernanda, and Magliano, D'Angelo Carlo

Subjects
*POLYCYSTIC ovary syndrome, *PATHOLOGICAL physiology, *UNFOLDED protein response, *INDUCED ovulation, *ENDOPLASMIC reticulum, *ENDOCRINE diseases, *GENITALIA
Abstract

Polycystic ovary syndrome (PCOS) is an endocrine disorder that affects million women worldwide, presenting a complex pathophysiology that has not been fully elucidated yet. Recently, it has been suggested that PCOS triggers the endoplasmic reticulum (ER) stress, thus being associated with unfolded protein response (UPR) activation. Indeed, the UPR response has been associated with several pathological conditions, including in the reproductive system. Several studies demonstrated that ovarian UPR markers are upregulated in PCOS, being associated with worst ovarian outcomes, and this was ameliorated by ER stress inhibition. In this review, we aim to summarize the main findings from previous studies covering this topic, in an attempt to clarify the potential role of ER stress and the UPR response in the pathophysiology of PCOS. [ABSTRACT FROM AUTHOR]

Published
2023
Full Text
View/download PDF

14. Modulation of cardiac renin-angiotensin system, redox status and inflammatory profile by different volumes of aerobic exercise training in obese rats

Author

Alexandre-Santos, Beatriz, primary, Alves, Renata, additional, Matsuura, Cristiane, additional, Sepúlveda-Fragoso, Vinicius, additional, Velasco, Larissa Lírio, additional, Machado, Marcus Vinícius, additional, Vieira, Aline Bomfim, additional, Fernandes, Tiago, additional, Oliveira, Edilamar Menezes, additional, Tibiriçá, Eduardo, additional, Nóbrega, Antonio Claudio Lucas da, additional, Magliano, D'Angelo Carlo, additional, and Frantz, Eliete Dalla Corte, additional

Published
2020
Full Text
View/download PDF

15. Enalapril and treadmill running reduce adiposity, but only the latter causes adipose tissue browning in mice

Author

Giori, Isabele G., primary, Magliano, D'Angelo C., additional, Alexandre‐Santos, Beatriz, additional, Fernandes, Tiago, additional, Oliveira, Edilamar M., additional, Vieira, Carla P., additional, Conte‐Junior, Carlos A., additional, Ceddia, Rolando B., additional, Nobrega, Antonio C. L., additional, and Frantz, Eliete D. C., additional

Published
2020
Full Text
View/download PDF

16. Exercise-induced cardiac opioid system activation attenuates apoptosis pathway in obese rats

Author

Alexandre-Santos, Beatriz, primary, Machado, Marcus Vinícius, additional, Menezes, Agatha Cristie, additional, Velasco, Larissa Lírio, additional, Sepúlveda-Fragoso, Vinicius, additional, Vieira, Aline Bomfim, additional, Borges, Juliana Pereira, additional, Tibiriçá, Eduardo, additional, Magliano, D'Angelo Carlo, additional, Nóbrega, Antonio Claudio Lucas da, additional, and Frantz, Eliete Dalla Corte, additional

Published
2019
Full Text
View/download PDF

17. Enalapril and treadmill running reduce adiposity, but only the latter causes adipose tissue browning in mice.

Author

Giori, Isabele G., Magliano, D'Angelo C., Alexandre‐Santos, Beatriz, Fernandes, Tiago, Oliveira, Edilamar M., Vieira, Carla P., Conte‐Junior, Carlos A., Ceddia, Rolando B., Nobrega, Antonio C. L., and Frantz, Eliete D. C.

Subjects
BROWN adipose tissue, WHITE adipose tissue, RENIN-angiotensin system, ENALAPRIL, OBESITY, ADIPOSE tissues
Abstract

This study investigated whether regulation of the renin–angiotensin system (RAS) by enalapril and/or aerobic exercise training (AET) causes browning of the subcutaneous white adipose tissue (sWAT). C57BL/6 mice were fed either a standard chow or a high‐fat (HF) diet for 16 weeks. At Week 8, HF‐fed animals were divided into sedentary (HF), enalapril (HF‐E), AET (HF‐T), and enalapril plus AET (HF‐ET) groups. Subsequently, sWAT was extracted for morphometry, determination of RAS expression, and biomarkers of WAT browning. The HF group displayed adipocyte hypertrophy and induction of the classical RAS axis. Conversely, all interventions reduced adiposity and induced the counterregulatory RAS axis. However, only AET raised plasma irisin, increased peroxisome proliferator‐activated receptor‐γ coactivator‐1α, and uncoupling protein‐1 levels, and the expression of PR‐domain containing 16 in sWAT. Therefore, we concluded that AET‐induced sWAT browning was independent of the counterregulatory axis shifting of RAS in HF diet‐induced obesity. [ABSTRACT FROM AUTHOR]

Published
2021
Full Text
View/download PDF

Catalog

Books, media, physical & digital resources
See catalog results