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This study examined whether sexual objectification (i.e., reducing someone to a sex object via a disproportionate focus on appearance and sexual characteristics) was associated with decreased confidence in future bystander intervention to reduce the risk for sexual violence (i.e., bystander efficacy) through several barriers to intervention:failing to notice the event,failing to identify the situation as risky, andfailing to take responsibility. Participants were 1,021 undergraduates (n= 309 men;n= 712 women) who completed self-report measures. Because men frequently perpetrate objectification, whereas women often experience objectification, complementary models were tested with objectification perpetration in men and objectification experiences in women. As expected, for men, each barrier mediated negative associations between objectification perpetration and bystander efficacy. Unexpectedly, for women, each barrier mediated positive associations between objectification experiences and bystander efficacy. Findings underscore important gender differences in associations between sexual objectification and bystander efficacy, as well as potential benefits of helping bystanders recognize the risk for sexual violence and assume responsibility for intervening.

The current study examined two cognitive appraisals—labeling (identifying an unwanted sexual experience as sexual violence) and self-blame—as potential mechanisms between survivor alcohol use before sexual violence and three help-seeking barriers (minimization, negative treatment, and social-emotional barriers) among non-service-seeking sexual violence survivors. Participants were 141 undergraduate women who completed self-report measures. Three parallel mediation models were tested. Survivors who were drinking were more likely to label their victimization as sexual violence and, in turn, perceived fewer minimization and greater social-emotional barriers. Further, survivors who were drinking blamed themselves more and, in turn, perceived greater negative treatment and social-emotional barriers.

OBJECTIVES: Childhood sexual abuse is linked to long-term consequences, including depression and anxiety in adulthood. Although considerable progress has been made to understand mechanisms that may account for this relation, such as emotion dysregulation, less attention has been given to protective factors that may mitigate it. One such protective factor might be mindful awareness. Those who act with awareness in daily living tend to engage in healthy emotion regulation skills when faced with stressors and experience less depression and anxiety. In the current study, we aimed to replicate the positive associations among childhood sexual abuse severity, emotion dysregulation, and psychopathology across time, and also identify a personal strength—in this case, mindful awareness—that might mitigate these effects. METHODS: Participants were 491 women recruited from the community who completed self-report assessments at three time points over a 32-month period. RESULTS: A series of moderated mediation models revealed childhood sexual abuse severity predicted later reports of depression and anxiety symptoms through greater emotion dysregulation in the form of difficulties engaging in goal-directed behaviors. As expected, mindful awareness weakened the relation between goal-directed emotion dysregulation and symptoms of depression and anxiety, such that greater levels of mindful awareness fully buffered these effects. CONCLUSIONS: Through a better understanding of natural resiliency processes among survivors, we can ultimately encourage continued examination of what might be effective additions to existing treatments for the mental health consequences of trauma and adversity.

Sexual abuse during childhood is associated with risk for sexual assault as an adult, known as revictimization. Although multiple experiences of sexual assault in adulthood are also common, it is unclear how risk trajectories might continue to evolve in emerging adulthood, defined as ages 18 to 25. Clarifying risk trajectories is important to inform the development of targeted risk reduction interventions. To fill this gap, we examined cumulative risk for sexual assault in emerging adult women following multiple experiences of childhood sexual abuse (CSA) and adulthood sexual assault (ASA).Women (n = 447; aged 18-25 years at enrollment) completed behaviorally specific assessments of unwanted sexual experiences at up to nine time points across 3 years. Logistic regression was used to predict any sexual assault during the 3-year period as a function of victimization history at baseline. A multilevel logistic regression analysis among ASA survivors was then used to determine whether each successive ASA increased risk for further victimization.Extending prior research, findings revealed that the risk for sexual assault during the 3-year study was greater for women reporting more prior experiences of CSA and ASA. Unexpectedly, each ASA increased the risk for a subsequent ASA to a lesser extent among women with more experiences of CSA.Findings suggest that the risk for sexual revictimization can be cumulative, but that risk does not increase indefinitely. Future research should investigate the points at which survivors of multiple assaults may begin to experience a decreased risk for later assaults, as well as the factors associated with change in risk status (e.g., removal from violent environments or relationships, changes in institutional policies). Such research could inform intervention targets.

Objective: Women who have experienced trauma report high rates of heavy episodic drinking (HED) and sleep problems. Prior work suggests that poor sleep exacerbates heavy alcohol use; however, potential mechanisms for this association are unclear. Consistent with the self-medication model, one possibility may be that women with a history of trauma are drinking at increased rates in order to cope with the affective consequences of poor sleep. To examine this possibility, the current study tested the role of drinking to cope motives as a mediator of prospective associations between sleep problems and HED among women who have experienced trauma. Method: Community women reporting a history of trauma (N = 414, Mage = 21.8, 59.9% White, 36.2% Black) completed self-report measures at baseline and 4 month and 8 month follow-ups. Measures of trauma exposure (Life Events Checklist [LEC]) and sleep problems (Cohen-Hoberman Inventory of Physical Symptoms-Revised [CHIPS-R]) were taken from baseline, drinking motives (Revised Drinking Motives Questionnaire) at 4 months, and HED at 8 months. Results: Findings supported an indirect association between sleep problems and later HED through increased drinking to cope motives (b = .05, 95% CI [.018, .108], β = .05). Conclusion: As hypothesized, drinking to cope accounted for associations between sleep problems and later HED. Findings underscore the potential value in addressing drinking to cope motives as a means of reducing HED, particularly among women with a history of trauma who are sleeping poorly. (PsycInfo Database Record (c) 2021 APA, all rights reserved).

Despite the clearly established link between posttraumatic stress disorder (PTSD) and emotion dysregulation, little is known about how individual symptoms of PTSD and aspects of emotion dysregulation interrelate. The network approach to mental health disorders provides a novel framework for conceptualizing the association between PTSD and emotion dysregulation as a system of interacting nodes. In this study, we estimated the structural relations among PTSD symptoms and aspects of emotion dysregulation within a large sample of women who participated in a multi-site study of sexual revictimization (N = 463). We estimated expected influence to reveal differential associations among PTSD symptoms and aspects of emotion dysregulation. Further, we estimated bridge expected influence to identify influential nodes connecting PTSD symptoms and aspects of emotion dysregulation. Results highlighted the key role of concentration difficulties in expected influence and bridge expected influence. Findings highlight several PTSD symptoms and aspects of emotion dysregulation that may be targets for future intervention.

Intimate partner aggression (IPA) is a serious and prevalent problem among college students. Two factors that may increase risk for in-person IPA are cyber IPA and alcohol use. This study examines the interaction between three types of cyber IPA (cyber psychological, stalking, and sexual IPA) and alcohol use in predicting in-person physical and sexual IPA perpetration. Participants were 268 undergraduate students, who participated in a mass screening at a large midwestern university. More than half of the sample reported perpetrating cyber IPA. A multivariate logistic regression model was used to examine study hypotheses. The R2 for physical IPA perpetration was .69 and the R2 for sexual IPA perpetration was .46. Cyber stalking IPA and alcohol interacted to predict physical IPA perpetration, such that the relationship between cyber stalking IPA and physical IPA perpetration was stronger among individuals who used more alcohol. Contrary to expectations, cyber IPA and alcohol did not interact to predict sexual IPA. However, consistent with hypotheses, cyber stalking IPA was positively associated with sexual IPA perpetration. The high prevalence of cyber IPA and its association with in-person IPA perpetration suggests that prevention programs targeting cyber IPA on college campuses may be warranted.

This study investigated associations between sensitivity to punishment and reward, shame, and eating pathology by testing alternative mediation models in which shame mediated associations between temperament and eating pathology or eating pathology mediated associations between temperament and shame. Participants were 96 female undergraduate students who completed questionnaires. Results indicated shame fully mediated the relationship between sensitivity to punishment and eating pathology. Further, eating pathology did not mediate the association between sensitivity to punishment and shame. In contrast, for sensitivity to reward, shame fully mediated the relationship between sensitivity to reward and eating pathology, and eating pathology fully mediated the relationship between sensitivity to reward and shame. If associations are supported by longitudinal research, results suggest that it may be valuable to develop prevention approaches targeting shame to reduce risk for the development of eating pathology for those who are high in sensitivity to punishment. For those higher in sensitivity to reward, interventions targeting shame may reduce risk for eating pathology and those targeting eating pathology may reduce shame.

Punishment sensitivity can contribute to eating pathology, but the mechanisms of this relationship are understudied. In a longitudinal study of undergraduate females (N = 95), results supported an indirect association between sensitivity to punishment and eating pathology via shame. Findings suggest that sensitivity to punishment was associated with greater shame, which in turn predicted greater eating pathology over time. Further, there was an indirect effect of sensitivity to punishment on eating pathology via greater levels of behavioral shame. Future studies may wish to examine the potential role of behavioral shame in the development and exacerbation of eating problems, especially in the context of temperamental traits such as punishment sensitivity.