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3. Protein Kinase A Is a Master Regulator of Physiological and Pathological Cardiac Hypertrophy

16. Enhanced basal contractility but reduced excitation-contraction coupling efficiency and [beta]-adrenergic reserve of hearts with increased Cav1.2 activity

19. Cardiomyocyte PKA ablation eliminates cardiac β-adrenergic response without adverse effects on the heart

26. Reduced Myocardial Reserve in Young X-Linked Muscular Dystrophy Mice Diagnosed by Two-Dimensional Strain Analysis Combined with Stress Echocardiography

35. Cardiotoxic and Cardioprotective Features of Chronic β-Adrenergic Signaling

38. Calcium influx through Cav1.2 is a proximal signal for pathological cardiomyocyte hypertrophy

40. Enhanced basal contractility but reduced excitation-contraction coupling efficiency and β-adrenergic reserve of hearts with increased Cav1.2 activity

42. Abstract 72.

44. Persistent increases in Ca(2+) influx through Cav1.2 shortens action potential and causes Ca(2+) overload-induced afterdepolarizations and arrhythmias.

45. Nicotine could augment adhesion molecule expression in human endothelial cells through macrophages secreting TNF-alpha, IL-1beta.

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