34 results on '"Aguila, Sonia"'
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2. von Willebrand factor links primary hemostasis to innate immunity
3. TNF-α–driven inflammation and mitochondrial dysfunction define the platelet hyperreactivity of aging
4. Novel therapies for hemophilia A – the role of the von Willebrand factor chaperone
5. Novel insights into the clinical phenotype and pathophysiology underlying low VWF levels
6. N-linked glycans within the A2 domain of von Willebrand factor modulate macrophage-mediated clearance
7. Thrombogenesis Mechanisms of High Doses of Ponatinib in Patients with Chronic Myeloid Leukemia (CML) Compared to Tyrosine Kinase Inhibitors
8. Amelioration of the severity of heparin-binding antithrombin mutations by posttranslational mosaicism
9. Hydroxyurea Reduces Neutrophil Extracellular Trap Formation in Myeloproliferative Neoplasms
10. Platelet activation and neutrophil extracellular trap (NET) formation in immune thrombocytopenia: is there an association?
11. Platelet activation and neutrophil extracellular trap (NET) formation in immune thrombocytopenia: is there an association?
12. A novel role for the macrophage galactose-type lectin receptor in mediating von Willebrand factor clearance
13. Increased galactose expression and enhanced clearance in patients with low von Willebrand factor
14. TNF-α Driven Inflammation and Mitochondrial Dysfunction Characterize the Platelet Hyperreactivity of Aging and Myeloproliferative Neoplasms (MPN)
15. The Low Von Willebrand Factor in Ireland Cohort Study - Defining Optimal Management for Procedures in Patients with Low VWF (30-50 IU/dL)
16. Platelet activation and neutrophil extracellular trap (NET) formation in immune thrombocytopenia: is there an association?
17. Aptamer BT200 blocks interaction of K1405-1408 in the VWF-A1 domain with macrophage LRP1.
18. Significant gynecological bleeding in women with low von Willebrand factor levels
19. Novel Insights into the Clinical Phenotype and Pathophysiology Underlying Low VWF Levels: The Low Von Willebrand Factor in Ireland Cohort (LoVIC) Study
20. TNF-a–driven inflammation and mitochondrial dysfunction define the platelet hyperreactivity of aging
21. Amelioration of the severity of heparin-binding antithrombin mutations by posttranslational mosaicism:a post-translational mosaicism for heparin binding mutations
22. A CRISPR-Cas9 Generated Etv6 mutant Mouse Exhibits Decreased Platelet Counts and Functional Platelet Defects
23. Aging-Associated Inflammation Due to Elevated Levels of TNF-α Increases Activation of the Integrin αIIbβ3 and the Procoagulant Potential of Platelets
24. Conformational Activation of Antithrombin by Heparin Involves an Altered Exosite Interaction with Protease
25. Identification of Antithrombin-Modulating Genes. Role of LARGE, a Gene Encoding a Bifunctional Glycosyltransferase, in the Secretion of Proteins?
26. C0094 High incidence of compound heterozygosity in antithrombin deficiency: Diagnostic and prognostic relevance
27. Identification of Antithrombin-Modulating Genes. Role of LARGE, a Gene Encoding a Bifunctional Glycosyltransferase, in the Secretion of Proteins?
28. C0229 Regulatory regions of SERPINC1 gene: Identification of the first mutation associated with antithrombin deficiency
29. TNF-a Driven Inflammation and Mitochondrial Dysfunction Characterize the Platelet Hyperreactivity of Aging and Myeloproliferative Neoplasms (MPN)
30. A CRISPR-Cas9 Generated Etv6mutant Mouse Exhibits Decreased Platelet Counts and Functional Platelet Defects
31. MicroRNAs in Platelets : Should I Stay or Should I Go?
32. Aptamer BT200 blocks interaction of K1405-1408 in the VWF-A1 domain with macrophage LRP1.
33. Increased N-glycosylation efficiency by generation of an aromatic sequon on N135 of antithrombin.
34. The infective polymerization of conformationally unstable antithrombin mutants may play a role in the clinical severity of antithrombin deficiency.
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