34 results on '"Abrigo, Johanna"'
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2. Sarcopenia in a mice model of chronic liver disease: role of the ubiquitin–proteasome system and oxidative stress
3. Angiotensin-(1-7) improves skeletal muscle regeneration.
4. Combined Administration of Andrographolide and Angiotensin- (1-7) Synergically Increases the Muscle Function and Strength in Aged Mice
5. Bile Acids Induce Alterations in Mitochondrial Function in Skeletal Muscle Fibers
6. Intensive care unit-acquired weakness: A review from molecular mechanisms to its impact in COVID-2019
7. Apocynin inhibits the upregulation of TGF-β1 expression and ROS production induced by TGF-β in skeletal muscle cells
8. The angiotensin-(1–7)/Mas axis reduces myonuclear apoptosis during recovery from angiotensin II-induced skeletal muscle atrophy in mice
9. Expression of the Mas receptor is upregulated in skeletal muscle wasting
10. Angiotensin-(1-7) Prevents Lipopolysaccharide-Induced Autophagy via the Mas Receptor in Skeletal Muscle
11. Sarcopenia Induced by Chronic Liver Disease in Mice Requires the Expression of the Bile Acids Membrane Receptor TGR5
12. Cholic acid and deoxycholic acid induce skeletal muscle atrophy through a mechanism dependent on TGR5 receptor
13. Protective Effect of Angiotensin 1–7 on Sarcopenia Induced by Chronic Liver Disease in Mice
14. Angiotensin (1-7) Decreases Myostatin-Induced NF-κB Signaling and Skeletal Muscle Atrophy
15. N-Acetyl Cysteine Attenuates the Sarcopenia and Muscle Apoptosis Induced by Chronic Liver Disease
16. Phosphoproteomic identification of Xin as a novel requirement for skeletal muscle disuse atrophy
17. Phosphoproteomic identification of Xin as a novel requirement for skeletal muscle disuse atrophy
18. Mitochondrial Dysfunction in Skeletal Muscle Pathologies
19. Cholic acid and deoxycholic acid induce skeletal muscle atrophy through a mechanism dependent on TGR5 receptor.
20. Central Role of Transforming Growth Factor Type Beta 1 in Skeletal Muscle Dysfunctions: An Update on Therapeutic Strategies
21. Somatotropic Axis Dysfunction in Non-Alcoholic Fatty Liver Disease: Beneficial Hepatic and Systemic Effects of Hormone Supplementation
22. The complex of PAMAM-OH dendrimer with Angiotensin (1–7) prevented the disuse-induced skeletal muscle atrophy in mice
23. The complex of PAMAM-OH dendrimer with Angiotensin (1–7) prevented the disuse-induced skeletal muscle atrophy in mice
24. N-Acetyl Cysteine Attenuates the Sarcopenia and Muscle Apoptosis Induced by Chronic Liver Disease
25. Transforming growth factor type beta (TGF-β) requires reactive oxygen species to induce skeletal muscle atrophy
26. High Fat Diet-Induced Skeletal Muscle Wasting Is Decreased by Mesenchymal Stem Cells Administration: Implications on Oxidative Stress, Ubiquitin Proteasome Pathway Activation, and Myonuclear Apoptosis
27. Angiotensin-(1-7) attenuates disuse skeletal muscle atrophy via the Mas receptor
28. Angiotensin-(1–7) decreases skeletal muscle atrophy induced by angiotensin II through a Mas receptor-dependent mechanism
29. The complex of PAMAM-OH dendrimer with Angiotensin (1-7) prevented the disuse-induced skeletal muscle atrophy in mice.
30. The angiotensin-(1–7)/Mas axis reduces myonuclear apoptosis during recovery from angiotensin II-induced skeletal muscle atrophy in mice
31. Expression of the Mas receptor is upregulated in skeletal muscle wasting
32. Angiotensin-(1-7) decreases skeletal muscle atrophy induced by angiotensin II through a Mas receptor-dependent mechanism.
33. Angiotensin-(1-7) attenuates disuse skeletal muscle atrophy in mice via its receptor, Mas
34. The Ang-(1-7)/Mas-1 axis attenuates the expression and signalling of TGF-β1 induced by AngII in mouse skeletal muscle.
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