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Intentional binding refers to the subjective temporal compression between a voluntary action and its subsequent sensory outcome. Despite some studies challenging the link between temporal compression and intentional action, intentional binding is still widely used as an implicit measure for the sense of agency. The debate remains unsettled primarily because the experimental conditions used in previous studies were confounded with various alternative causes for temporal compression, and action intention has not yet been tested comprehensively against all potential alternative causes in a single study. Here, we solve this puzzle by jointly comparing participants' estimates of the interval between three types of triggering events with comparable predictability-voluntary movement, passive movement, and external sensory event-and an external sensory outcome (auditory or visual across experiments). The results failed to show intentional binding, that is, no shorter interval estimation for the voluntary than the passive movement conditions. Instead, we observed temporal (but not intentional) binding when comparing both movement conditions with the external sensory condition. Thus, temporal binding appears to originate from sensory integration and temporal prediction, not from action intention. As such, these findings underscore the need to reconsider the use of "intentional binding" as a reliable proxy of the sense of agency. (PsycInfo Database Record (c) 2024 APA, all rights reserved).

Background and Aim: Avian pathogenic Escherichia coli (APEC) is the causative agent of colibacillosis, one of the most prevalent bacterial diseases responsible for significant economic losses in the poultry industry worldwide. This study aimed to assess the antimicrobial resistance (AMR) patterns of APEC isolates recovered from poultry in east Algeria and estimate the prevalence of extended-spectrum β-lactamase (ESBL)-producing isolates., Materials and Methods: In the slaughterhouse of Batna City (Algeria), livers indicating colibacillosis were sampled from 204 suspected carcasses with growth retardation and generalized congestion. Escherichia coli isolation and identification were performed on MacConkey agar using conventional methods and the API 20E system. Antimicrobial resistance susceptibility was tested by the disk diffusion method according to the Clinical Laboratory Standards Institute Guidelines. Extended-spectrum β-lactamase detection was carried out using the double-disk confirmation test., Results: One hundred sixty E. coli isolates were recovered (one isolate per sample). Avian pathogenic Escherichia coli isolates showed high levels of resistance to ampicillin and tetracycline (100%), nalidixic acid (95%), ofloxacin (93.75%), doxycycline (91.87), ciprofloxacin (87.50%), trimethoprim/sulfamethoxazole (62.50%), gentamycin (32.50%), chloramphenicol (27.50%), amoxicillin/clavulanic acid (16.25%), colistin (14.37%), and nitrofurantoin (10.62%). All strains were multidrug-resistant to at least three antibiotics, and more than half (52.52%) of the isolates were resistant to at least seven antibiotics. All isolates were susceptible to ceftriaxone, ceftazidime, and aztreonam. Two E. coli isolates were ESBL producers (1.25%)., Conclusion: Avian pathogenic Escherichia coli resistance to most antimicrobial agents used in poultry may lead to antimicrobial therapy failure., Competing Interests: The authors declare that they have no competing interests., (Copyright: © Aberkane, et al.)