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1. Profibrotic monocyte-derived alveolar macrophages are expanded in patients with persistent respiratory symptoms and radiographic abnormalities after COVID-19

2. Distinctive evolution of alveolar T cell responses is associated with clinical outcomes in unvaccinated patients with SARS-CoV-2 pneumonia

3. Mitochondria regulate proliferation in adult cardiac myocytes

5. Author Correction: Fibroblast A20 governs fibrosis susceptibility and its repression by DREAM promotes fibrosis in multiple organs

6. Aging is associated with a systemic length-associated transcriptome imbalance

7. Resetting proteostasis with ISRIB promotes epithelial differentiation to attenuate pulmonary fibrosis

8. scRNA-seq profiling of human granulocytes reveals expansion of developmentally flexible neutrophil precursors with mixed neutrophil and eosinophil properties in asthma.

9. Fibroblast A20 governs fibrosis susceptibility and its repression by DREAM promotes fibrosis in multiple organs

10. Novel enzyme-based reduced representation method for DNA methylation profiling with low inputs

11. Monocyte-derived alveolar macrophages drive lung fibrosis and persist in the lung over the life span

13. Prolonged exposure to lung-derived cytokines is associated with activation of microglia in patients with COVID-19

15. Circuits between infected macrophages and T cells in SARS-CoV-2 pneumonia

16. Maintenance DNA methylation is essential for regulatory T cell development and stability of suppressive function

17. Metformin Targets Mitochondrial Electron Transport to Reduce Air-Pollution-Induced Thrombosis

22. Data from Posttranslational Regulation of the Exon Skipping Machinery Controls Aberrant Splicing in Leukemia

23. Supplementary Table 10 from Posttranslational Regulation of the Exon Skipping Machinery Controls Aberrant Splicing in Leukemia

24. Supplementary Table 8 from Posttranslational Regulation of the Exon Skipping Machinery Controls Aberrant Splicing in Leukemia

25. Supplementary Table 7 from Posttranslational Regulation of the Exon Skipping Machinery Controls Aberrant Splicing in Leukemia

26. Supplementary Table 2 from Posttranslational Regulation of the Exon Skipping Machinery Controls Aberrant Splicing in Leukemia

27. Supplementary Table 6 from Posttranslational Regulation of the Exon Skipping Machinery Controls Aberrant Splicing in Leukemia

28. Supplementary Table 9 from Posttranslational Regulation of the Exon Skipping Machinery Controls Aberrant Splicing in Leukemia

29. Supplementary Table 4 from Posttranslational Regulation of the Exon Skipping Machinery Controls Aberrant Splicing in Leukemia

30. Supplementary Table 5 from Posttranslational Regulation of the Exon Skipping Machinery Controls Aberrant Splicing in Leukemia

31. Supplementary Figures S1-15 from Posttranslational Regulation of the Exon Skipping Machinery Controls Aberrant Splicing in Leukemia

32. Supplementary Table 3 from Posttranslational Regulation of the Exon Skipping Machinery Controls Aberrant Splicing in Leukemia

33. Expression of vimentin alters cell mechanics, cell-cell adhesion, and gene expression profiles suggesting the induction of a hybrid EMT in human mammary epithelial cells

34. The lung microenvironment shapes a dysfunctional response of alveolar macrophages in aging

39. Hypercapnia alters stroma-derived Wnt production to limit β-catenin signaling and proliferation in AT2 cells

41. Expression of vimentin alters cell mechanics, cell-cell adhesion, and gene expression profiles suggesting the induction of a hybrid EMT in human mammary epithelial cells

42. Cilia-related gene signature in the nasal mucosa correlates with disease severity and outcomes in critical respiratory syncytial virus bronchiolitis

43. Elevation of activated neutrophils in chronic rhinosinusitis with nasal polyps

47. Age-related Differences in the Nasal Mucosal Immune Response to SARS-CoV-2

48. SF3B1 homeostasis is critical for survival and therapeutic response in T cell leukemia

49. Hypercapnia limits β-catenin-mediated alveolar type 2 cell progenitor function by altering Wnt production from adjacent fibroblasts

50. Influenza-Induced Activation of Recruited Alveolar Macrophages During the Early Inflammatory Phase Drives Lung Injury and Lethality

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