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1. Mouse fetal growth restriction through parental and fetal immune gene variation and intercellular communications cascade

2. Distinct genetic architectures and environmental factors associate with host response to the γ2-herpesvirus infections

3. Somatic mosaicism and common genetic variation contribute to the risk of very-early-onset inflammatory bowel disease

4. Somatic mosaicism and common genetic variation contribute to the risk of very-early-onset inflammatory bowel disease

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