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玻璃幕墙已被广泛应用于建筑结构中，但目前的设计主要针对静力荷载，对可能发生的爆炸等意外事件所产生的冲击荷载作用尚缺乏可靠研究。对爆炸冲击下的玻璃进行数值分析时，多数研究模型将边界条件过度简化，使玻璃面板直接承受爆炸冲击，忽略了支承体系中诸如框架、垫片、结构胶、滑移块等元件的缓冲作用。这种差异在中空夹层玻璃系统上更为明显，导致数值分析难以精确地模拟结构系统在冲击荷载作用下的响应。为此，对一玻璃幕墙系统进行足尺爆炸冲击试验研究，并采用LS-DYNA软件进行了精细化建模，考虑支承体系内的各元件作用及元件之间的接触行为，以及各元件对玻璃的夹紧力作用等因素。分析结果表明，相比于简化边界条件的模型，考虑多要素的框架模型得出的玻璃面板挠度及速度时程曲线、能量传递等更加合理，且裂纹模态和裂纹动态扩展与试验结果更加吻合。爆炸冲击下中空夹层玻璃的两种常用简化分析模型均不能获得准确的结果：简化的夹板支承模型缺少必要的约束条件，导致玻璃系统整体位移过大而发生破坏；简支模型则约束过强，会导致玻璃应力增大，尤其会在边界区域发生更多的强度破坏。因此，设计抗爆玻璃时应重视缓冲体系的设计，在数值分析时则应充分考虑支承系统各元件的影响。

针对高倍频毫米波的产生问题，文章提出了一种基于4个偏振调制器并联产生16倍频毫米波的方案，利用相关光边带极性的不同，使合成光信号中只保留载波和8n阶边带。经过适当选取调制指数值和光电探测器拍频后，获得16倍频毫米波信号。文章理论分析了产生16倍频毫米波信号的原理，推导出方案所产生的±8阶光边带信号的光边带抑制比为61.72 dB，16倍频毫米波信号的射频杂散抑制比为55.70 dB。仿真实验获得的光边带抑制比和射频杂散抑制比分别为60和53 dB。同时分析了射频信号相位偏移及调制指数对抑制比的影响。实验结果与理论分析吻合，验证了所提方案的可行性。

南京牛首山树状结构的施工空间狭小、施工平台薄弱和大型设备不能进驻,根据其特殊施工环境提出了自平衡提升工法。对自平衡提升过程中可能产生的平面外位移问题,设计了双支撑胎架方案解决。利用3D3S空间结构设计软件对施工全过程进行了模拟,同时对树状结构提升全过程进行了施工监测,模拟结果和监测结果吻合较好。研究表明自平衡提升施工法能够满足特殊施工条件下大型钢结构安装的施工要求,为此类工程的合理施工提供了参考,采用3D3S设计软件对特殊环境下的空间钢结构进行施工预分析是可行的,能有效地提高工程的安全性。

【目的】探讨双胎妊娠期糖尿病（GDM）早孕期的相关危险因素，以寻找早孕期预测双胎GDM 的有效指标。【方法】回顾性分析2015 年1 月至2019 年6 月在中山大学附属第三医院规律产检并分娩，且病例资料完整的双胎妊娠孕妇共 318 例，根据 75 克口服葡萄糖耐量试验（OGTT）结果，将其分为双胎 GDM 组 84 例，正常双胎组（双胎未合并GDM）234 例，收集记录研究对象的临床特征，包括年龄、孕前体质量指数、孕产次、既往病史、此次受孕方式、双胎绒毛膜性质；早孕期（11~13+6 周）的空腹血糖（FPG）、总胆固醇（TC）、甘油三酯（TG）、高密度脂蛋白（HDL）、低密度脂蛋白（LDL）及早孕期唐氏筛查生物学标记物妊娠相关血浆蛋白A（PAPP-A）和人绒毛膜促性腺激素β亚单位（Free β-HCG）的检测结果，通过多因素 Logistic 逐步回归分析法分析双胎 GDM 的早孕期相关危险因素，采用受试者工作特征曲线评价双胎 GDM 的早期预测。【结果】①两组比较，双胎 GDM 组的年龄、孕前体质量指数高于正常双胎组（P < 0.001），双胎 GDM 组合并多囊卵巢综合征病史的比例高于正常双胎组（P < 0.05）；②双胎GDM 组早孕期FPG、TG 高于双胎正常组（P < 0.05），而两组TC、HDL、LDL 及PAPP-A MOM 值及Free β-HCG MOM 值差异无统计学意义（P > 0.05）；③ 年龄、孕前体质量指数、PCOS 病史、早孕FPG 水平均为双胎GDM 发生的独立危险因素（P < 0.05）；④早孕期FPG 预测双胎GDM 的ROC 曲线下面积（AUC）为0.617（95% CI：0.547 ~ 0.688），FPG 为 4.495 mmo/L 时，约登指数为 0.223，敏感度、特异度分别为 65.5%、56.8%；早孕期 TG 预测双胎 GDM 的 AUC 为 0.65（95%CI：0.584 ~ 0.716），TG 为 1.195 mmo/L 时，约登指数为 0.24，敏感度、特异度分别为 77.4%、46.6%；而早孕期 FPG、TG 结合孕妇年龄、孕前体质量指数、PCOS 病史预测双胎 GDM 的 AUC 为 0.751 （95% CI：0.693 ~ 0.810），敏感度、特异度分别为73.8%、70.1%。【结论】孕妇年龄、孕前体质量指数、PCOS 病史、早孕FPG 水平均为双胎GDM 发生的独立危险因素；早孕期FPG、TG 可作为早期预测双胎GDM 发生的指标，结合临床特征可以增加早期预测价值。

为了有效提高光通信系统的频带利用率,提出了一种新型复用技术——映射复用（MM）,即采用双时隙多幅值信号对多路复用信号的单值映射来实现对多路信号的复用。分析了MM技术的复用和解复用原理,以4路用户信号复用为例,利用Matlab软件设计了MM复用器和解复用器。采用Optisystem和Matlab联合仿真,设计了一种基于MM技术的4路用户信号复用的光纤传输系统。仿真实验得到的眼图清晰,验证了方法的可行性;与同速率的非归零和归零信号相比,MM信号的谱宽分别是它们的一半和1/3。MM技术可有效地提高系统的频带利用率,增加色散容忍度,在光纤通信系统中具有广泛的应用前景。

提出了一种60GHz毫米波、UWB（超宽带）信号和基带信号混合传输的光纤通信系统。在中心站采用10GHz RF（射频）信号驱动两个并行PM（相位调制器）产生三阶边带,在基站通过PD（光检测器）拍频生成6倍频的60GHz毫米波;利用高斯电脉冲驱动双极型MZM（马赫-曾德调制器）生成二阶UWB信号;UWB信号和基带信号通过偏振复用调制在同一光载波上。仿真结果表明,2.5Gbit/s的信号经20km单模光纤传输后,眼图清晰;生成的UWB信号中心频率为5.5GHz,相对带宽达到98%,符合FCC（美国联邦通信委员会）标准。

APDCDM（双极性占空比复用）是降低DCDM（占空比复用）信号幅值级数的一种有效方法。为了进一步降低APDCDM信号的幅值级数,提出了基于信号替换原理的I-APDCDM（改进型双极性占空比复用）技术。分析了I-APDCDM中信号替换的工作原理及电路实现方法,采用Optisystem和MATLAB联合仿真技术设计了I-APDCDM光纤传输系统,分析了改进后系统的传输距离和调制器的消光比对I-APDCDM信号的影响规律。仿真结果表明,I-APDCDM将APDCDM复用信号的幅值级数降低了1级,每比特复用信号的平均能量降低了A2 Ts/24,有效地提高了系统的性能。

【目的】探讨早孕期甲状腺功能状态与妊娠期糖尿病（GDM）的相关性。【方法】选取2016年6月至 2017年6月在我院产检的单胎孕妇2112例。于妊娠9~13+6周测定游离甲状腺素（FT4）、促甲状腺素（TSH）及甲 状腺过氧化物酶抗体（TPOAb）；于24~28周行口服糖耐量试验（OGTT），结果异常者为GDM组（224例），正常者 为非GDM组（1888例），分析两组孕妇甲状腺激素水平的差异及利用Spearman秩相关分析甲状腺激素水平与 OGTT各时相血糖的相关性。【结果】GDM孕妇与非GDM孕妇早孕期TSH［1.04（0.61-1.61）vs1.11（0.59-1.77） μU/mL，P＝0.292］、FT4［16.22（14.22-18.31）vs16.62（14.54-18.79）pmol/L，P＝0.087］及各甲状腺功能异常的 发病率比较差异均无统计学意义（P>0.05）；校正了孕妇年龄、产次及孕前体质量指数后，早孕期TSH和FT4与 OGTT各时相血糖无明显的相关性。【结论】早孕期甲状腺功能状态与GDM无明显相关性。

为产生全光域UWB（超宽带）一阶和二阶信号,降低光载UWB系统成本,提出了一种基于偏振调制同时光生一阶和二阶UWB信号的方法。将两路极性相反的高斯脉冲信号通过PolM（偏振调制器）相位调制到偏振态正交的两个光载波上,利用PMF（保偏光纤）引入适当的群延时,生成一阶UWB信号。通过PC（偏振控制器）控制输入光信号与PMF主轴所成的角度,产生一对极性相反的一阶UWB信号,并对其中一路进行适当的延时,合成后即可生成二阶UWB信号。所得一阶UWB信号的中心频率为5.36GHz,-10dB绝对带宽为8.96GHz,相对带宽为167%;二阶UWB信号的中心频率为5.83GHz,-10dB绝对带宽为7.36GHz,相对带宽为126%,均满足FCC（美国联邦通信委员会）标准。分析了偏振调制中时延对生成的UWB信号性能的影响。结果表明,当偏振调制中引入的时延τ=TFWHM时,生成的二阶UWB信号更符合FCC要求。

为了实现UWB（超宽带）光纤技术的多用户接入,提出了基于PM-IM（相位调制-强度调制）转换以及多路鉴频实现三阶UWB脉冲的BPSK（二进制相移键控）编码方案,分析了其工作原理并采用光子模拟软件进行了仿真。得到了不同的BPSK编码序列,其频谱中心频率为6.7GHz,相对带宽为104.5%,符合FCC（美国联邦通信委员会）对于UWB信号的规定。分析了高斯脉冲宽度和光功率对仿真结果的影响,发现高斯脉冲宽度为0.5bit时所得信号频谱最为理想,当光源功率小于26dBm时,PSD（功率谱密度）符合FCC规定,为实现多用户接入的编码提供了一种新方案。

提出了一种利用SOA（半导体光放大器）的增益饱和效应及XGM（交叉增益调制）效应同时产生二阶及三阶UWB（超宽带）信号的方案,分析了其工作原理,并采用光子模拟软件进行了仿真实验,得到了中心频率为6GHz、相对带宽为143%的二阶信号和中心频率为8GHz、相对带宽为120%的三阶信号,两者均符合FCC（美国联邦通信委员会）对UWB信号的规定。同时,还分析了输入脉冲信号宽度、探测光及泵浦光功率对生成的UWB信号的影响。

在光纤无线电技术中,通过光外差产生毫米波具有结构简单、成本低的特点,但中心站一般需要使用相干光源。文章提出了一种改进的光外差法,系统分析和仿真试验表明,该方法不仅可以避免使用相干光源,而且中心站和无线终端都无需配备毫米波本振。同时中心站采用单边带的信号调制,还可以有效降低光纤色散引起的毫米波信号的衰落。

文章介绍了所组建的全氟树脂塑料光纤千兆以太网的设计方案、设备选择及安装,给出了网络的性能测试数据,分析了全氟树脂塑料光纤及其所构筑的千兆以太网存在的问题,分析了全氟树脂塑料光纤千兆以太网的应用前景,提出了我国应加紧该领域研究的建议。

塑料光纤（POF）技术日益成熟,其产品在通信领域的应用量不断扩大。文章简述了POF的性能,探讨了各种POF在楼宇布线中应用的可行性,总结了当前楼宇布线标准和POF标准的发展概况,最后分析了当前布线标准所存在的缺陷和POF标准制定的紧迫性。

文章介绍了利用不同占空比实现信道复用的新型复用技术,即DCDM（占空比复用）技术,分析了其复用与解复用的原理,并利用MATLAB软件设计了DCDM系统的解复用器。为了验证DCDM的可行性,采用Optisystem与MATLAB软件的联合仿真,设计了基于DCDM 3路用户的光纤传输系统,仿真验证了系统的可行性。

文章提出了两种利用SOA（半导体光放大器）的XGM（交叉增益调制）和FBG（光纤布拉格光栅）光学生成对称型UWB（超宽带）信号的新方法。分析了该方法的工作原理并进行了仿真实验,分别得到了符合FCC（美国联邦通信委员会）标准的中心频率为5.35 GHz、相对带宽为114%以及中心频率为4.7 GHz、相对带宽为132%的对称型UWB信号,仿真结果验证了该方法的可行性。文章还分析了这两种方法存在的问题。

文章设计了一个基于频率间插复用的DWDM-ROF（密集波分复用-光载无线）系统,使每个信道载波与边带之间的空闲频段得到利用,从而提高了DWDM-ROF系统的频谱利用率。同时,为该系统设计了一种基于FBG（光纤布拉格光栅）的新颖的解复用器。数据传输的仿真实验不仅验证了该方法的可行性,还显示出了该系统具有良好的抗信道串扰能力。

描述了光包交换节点的结构 ,分析了网络的物理和逻辑的可行性 .介绍了一个由 40个网络节点组成的节点流量达 1 60 Gbit/s的物理网络 .网络能提供较好的服务质量 （包丢失率和包传输延迟 ）并兼容多种业务 .这一结果为基于全光包交换技术的灵活性网络的研究铺平了道路

设计了一种结构简单、成本低的TW-EDFL（三波长掺铒光纤激光器）,其输出波长分别为1 530、1 540和1 550nm,实现了1.5mW的稳定输出功率。研究了EDF（掺铒光纤）长度和泵浦功率与输出功率的关系,得到最佳EDF长度为4.15m、最佳泵浦功率为200mW的结论。在结构中加入3个VOA（可变光衰减器）,运用增益均衡的方法来平衡谐振腔内3路信号光的增益和损耗,避免了EDF在室温下的模式竞争效应。

【目的】 探讨肿瘤坏死因子-α(TNF-α)?白细胞介素1β(IL-1β)?基质金属蛋白酶-3(MMP-3)?基质金属蛋白酶-9(MMP-9)和基质金属蛋白酶抑制剂-1(TIMP-1)在大鼠脑缺血再灌注后的动态变化及特点,阐明TNF-α?IL-1β在脑缺血大鼠MMP和TIMP-1引起脑损伤中的作用?【方法】 雄性SD大鼠56只随机分为假手术组和缺血2 h再灌注6?12?24?48?72 h和7 d组?运用线栓法复制大鼠局灶性脑缺血再灌注模型,采用ELISA法测定脑组织和血清TNF-α和IL-1β含量,及脑组织中MMP-3?MMP-9和TIMP-1的含量?【结果】 与假手术组比较,血清和脑组织TNF-α和IL-1β含量于缺血再灌注6 h明显增加(4.38 ± 0.73 vs 2.63 ± 0.14?5.28 ± 0.71 vs 3.46 ± 0.47;22.34 ± 3.56 vs 12.13 ± 4.26?9.56 ± 0.85 vs 4.23 ± 0.83; P < 0.05),12 h达到高峰,随后各时间点表达开始下降,至7 d与假手术组比较无明显差别?脑组织中MMP-3?MMP-9含量于再灌注6 h开始升高,24 h明显升高(P < 0.05),48 h达高峰,随后逐渐下降,至再灌注7 d与假手术组比较无统计学意义,而脑组织中TIMP-1含量在再灌注各时间点均低于假手术组(P < 0.05)?且血清和脑组织中TNF-α和IL-1β含量与MMP-3?MMP-9的含量呈明显正相关(P < 0.05),与TIMP-1含量呈负相关(P < 0.05)?【结论】 MMP-3?MMP-9在脑缺血大鼠缺血再灌注早期水平即升高,提示MMP-3和MMP-9在脑缺血再灌注的发病过程中起着重要的作用,炎症因子TNF-α和IL-1β可通过促进脑组织中MMP-3?MMP-9的生成,抑制TIMP-1的生成,进一步加重缺血后脑损伤?

根据ITU的H.261建议,建立了视频编解码器的模型,并给出了视频压缩编码算法的具体参数。在软件模拟的基础上得出了一些有益的结论,理论分析与实验结果相符合。

分析了 MPLS在未来 IP网络中的可能的应用前景 ,认为信息骨干网络可能由光的MPLS和电的 MPLS两层网络组成 ,并完成骨干网上的 IP路由 ,边缘路由器完成边缘的 IP路由交换 .着重提出了光的 MPLS的实现方法和基本原理及结构 .

提出了用布拉格相移光纤光栅中相移来实现低功率的光标交换的新方法 .设计了一个基于该方法的光标记交换实验 .实验所用的布拉格光纤光栅是在通信用的标准光纤内刻写而成 ,通过强泵浦脉冲作用在低强度的探测信号上 .光标记交换是基于交叉相位调制来完成的

提出了一种基于副载波的光标记交换的方案和标记交换的原理 ,它是解决电子交换瓶颈的有效方法 ,将充分利用电子技术的成就解决低速标记交换的问题 ,同时保留了光包的高速和透明性不变

Copyright of Gold (1001-1277) is the property of Gold Magazine Editorial Office and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

We reported a case of fetal lymphedema. The chromosome karyotype analysis of fetus and single nucleotide polymorphism array (SNP array) of the amniotic fluid cells showed no abnormalities in the mid pregnancy. However, the pregnant woman perceived a decrease in fetal movement in the late pregnancy, and ultrasound indicated intrauterine stillbirth. Whole exome sequencing (WES) showed that the fetus carried loss of heterozygosity of CELSR1 gene c.5060-1_ 5069 delGCCATGCCTCA. The WES results also showed that the fetal father also carried the mutation and had a normal phenotype. There is a certain correlation between loss of heterozygosity in the CELSR1 gene and lymphedema, but its clinical phenotype is incomplete penetrance. [ABSTRACT FROM AUTHOR]

Objective To observe the connection of the orbital septum with surrounding structures and its histology in relation to mild ptosis in blepharoplasty patients. Methods From May 2018 to December 2019, 276 patients (549 eyes) who underwent blepharoplasty were divided into normal group and mild ptosis group according to the condition of upper eyelid, and the orbital septum was observed. The connection between the orbital septum and the surrounding structures, and the histologic structure of the orbital septum were observed. Results There were 498 eyes in the normal group and 51 eyes in the mild ptosis group. Comparing between the two groups, the incidence of adhesions between the orbital septum and the orbital lipid envelope was higher in the mild ptosis group (X² =95.00, P＜0.001), and the incidence of transverse ligament between the orbital septum and the levator aponeurosis was higher in the mild ptosis group (X² =99.00, P＜0.001). HE staining showed that the orbital septum in both groups was mainly a connective tissue structure that could be divided into two layers. The anterior layer was looser and the posterior layer was denser. In the mild ptosis group, the connective tissue fibers were less and looser than in the normal group. Masson staining showed that no muscular structures were found in the orbital septum of both groups. SMA staining showed that in the orbital septum of both groups, except for the blood vessels running through the orbital septum which were positive, the rest of the tissues stained negatively by SMA staining, and no smooth muscle structures were found in the orbital septum of both groups. Conclusion Adhesions between the orbital septum and the orbital lipid envelope and restrictive ligament between the orbital septum and the levator aponeurosis may be the cause of mild ptosis. [ABSTRACT FROM AUTHOR]

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The prevalence rate of nonalcoholic fatty liver disease (NAFLD) reaches up to 30% around the world, and the disease has a serious impact on human health and constitutes a public health burden. Due to difficulties in the diagnosis and monitoring of NAFLD, it is important to identify potential drug targets and biomarkers, and multi-omics techniques hold great promise in the search for early diagnostic markers, therapeutic targets, and outcome and prognostic assessment of NAFLD. This article reviews the research advances in multi-omics techniques in the field of NAFLD in recent years, in order to provide a richer theoretical basis and new strategies for the prevention and treatment of NAFLD. [ABSTRACT FROM AUTHOR]

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