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文章认为以微纳光场调控及超表面光学技术为代表的手机裸眼3D显示应用,有可能带动移动通信网络实现跨越式的发展。针对全息三维显示产生的庞大海量数据,文章从手机裸眼3D图像的有效像素和空间深度层等几个不同的角度,定量地分析了全息三维显示业务所需要的网络数据带宽和往返时延,以及其他各网络承载能力指标的需求和未来趋势;根据国际标准化组织给出的最大峰值数据传输速率的定义,找出影响第五代（5G）乃至第六代移动通信系统（6G）网络承载能力的多种因素,定量给出了能够实现裸眼3D应用的几个具体指标,比如:聚合的载波数量、参数集的数量、大规模天线阵列的阶数和子载波间隔等,以期得到能满足未来全息三维显示业务的网络承载能力的安全边际。

文章提出一种科学合理的5G端到端应用场景预测方法,并评估未来有可能成为杀手级的若干应用。不同于业内对一些5G细分场景的简单总结,文章以5G时代的三大互连关系、三大技术场景以及五大行业类别作为判断依据,建立有理论支撑和可信的应用体系评估预测标准。并针对一些行业应用"痛点",阐述其具体含义、关键性能指标、相关场景案例及未来设想。以期找到在未来涉及面广、用户量大,能为国家、社会和个人带来巨大贡献和影响的关键性应用。

文章对第六代移动通信系统（6G）的总愿景和发展趋势进行了初步探讨,认为基础信息理论的突破是6G发展的源点,发展方向是为人类可持续发展的个性化需求服务。文章将网络主体从"人-网-物"3个维度拓展至"人-网-物-境"4个维度,认为未来6G将会改变人类广义概念上的"环境",归纳了6G网络至少4个新形态:无人不互联、无处不互联、无时不互联和无事不互联。6G遵循以TRUST为原则的网络性能发展趋势,即向Tbit/s级以上带宽、超高可靠性、泛在连接、亚毫秒时延和包括太赫兹频谱、可见光通信以及当前正使用频谱在内的更广域频谱等方向演进,形成网络性能更高级发展阶段。最后,文章阐述了6G网络不同维度的各子愿景的需求和面临的挑战,后续将会对关键性技术逐步开展研究和修正工作。

在"人-网-物"三元互联时代,小数据业务在时域、频域和功率域三个方面不断占用着无线网络中大量不可再生的资源。文章在总结分析小数据的特征后,在时域方面,提出了一种利用"子制式载波"技术共享现有落后网络制式的方法,在同一频点的不同时间上,让小数据和这些落后的制式共同工作;在频域方面,提出了现有几种频带共享方案的一些问题;在功率域方面,指出了一个空闲能量因子,将因子和已知的选择算法相结合,使剩余功率也能成为网络资源优化选择的重要因素。

【目的】探讨既往吸入糖皮质激素（IC）对合并慢性阻塞性肺病（COPD）的社区获得性肺炎（CAP）住院患者临床结局的影响。【方法】这是一个多中心、回顾性研究。收集于2013年1月1日至2016年12月31日在北京山东和云南5家教学医院住院的所有合并慢性阻塞性肺病的社区获得性肺炎（COPD-CAP）患者的病例资料。比较既往使用和不使用IC的患者，包括人口学资料、临床和影像学特征以及支持治疗和临床结局。用logistic回归探讨IC使用史对COPD-CAP患者临床结局的影响。【结果】共有725例COPD-CAP住院患者纳入最后分析，有IC使用史的占13.9%（101/725）。相比于无IC使用史的COPD-CAP患者，既往使用IC的患者合并心血管病（19.8% vs 12.7%）和1年内曾有CAP住院史（20.8% vs 11.2%）的比例高；出现胸水（13.9% vs23.7%）比例低；属于慢性阻塞性肺病全球创议（GOLD）2期（10.4% vs 51.0%）比例低，而属于GOLD3期（35.1% vs 22.9%）和4期（51.9% vs 21.8%）的比例高。经过年龄、性别、基础病、CURB-65评分、PSI分级和GOLD分期调整的logistic回归分析证实：既往使用IC与COPD-CAP患者无创机械通气风险降低有关［OR=0.220，95% CI（0.052，0.926），P=0.029］，但是与有创机械通气［OR=0.290，95% CI（0.068，1.236），P=0.094］、需要使用血管活性药物［OR=1.261，95% CI（0.456，3.485），P=0.655］、入住ICU［OR=1.455，95% CI（0.638，3.320），P=0.373］和30d病死率［OR=1.650，95% CI（0.575，2.838），P=0.352］无关。【结论】既往使用IC对COPD-CAP患者的临床结局无重大影响。

现有LTE（长期演进）网络所使用的6GHz以下频段难以满足日益增长的网络带宽的需求,而5G网络能够提供大量连续的频谱资源,是未来无线网络的基础。针对多种无线接入应用场景,提出影响频谱资源分配的各种因素和高低频段的一些分配方式。结果表明,复用低频段和增加新的高频段空口能够满足5G网络的需求。

蜂窝状钢骨混凝土T形柱,是一种很适合于当今钢结构住宅建筑的新型异型柱。为了比较全面地探讨该新型构件的斜截面抗剪能力与变形特性,本文首先进行了单调加载试验,以考察在轴向力和横向力共同作用下该新型柱的斜截面抗剪能力、变形特性以及破坏形态;然后利用ANSYS有限元分析软件对T形截面柱进行非线性有限元分析,研究轴压力系数、剪跨比、配钢率、配箍率、混凝土强度等主要参数变化对该新型构件斜截面抗剪性能的影响及其规律,并提出相应的设计建议。研究表明,蜂窝状钢骨混凝土T形柱的抗剪破坏形态与相应荷载作用下的钢筋混凝土柱的抗剪破坏形态相似;平均应变的平截面假定在蜂窝状钢骨混凝土T形截面异型柱中仍然适用;该新型柱斜截面抗剪性能优于桁架式钢骨混凝柱T形柱;该新型柱的斜截面极限抗剪承载力随轴压系数、配钢率、配箍率、混凝土强度的增加而增加,但随剪跨比的增加而降低;其中剪跨比和轴压系数是影响T形柱斜截面抗剪性能比较敏感的因素。

中国电信现有的97系统管理的机线资源及相关功能需要通过一定方式迁移到基于GIS（地理信息系统）的资源管理系统（即中国电信网络资源管理系统）进行统一管理。这个过程称为97机线资源改造。文章主要介绍了资源管理系统与97系统接口的设计思路和接口功能,为机线资源改造的数据迁移和功能实现提供了实践证明。

Copyright of Coal Science & Technology (0253-2336) is the property of Coal Science & Technology and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

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Copyright of Command Control & Simulation / Zhihui Kongzhi yu Fangzhen is the property of Command Control & Simulation Editorial Office and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

Copyright of Food Research & Development is the property of Food Research & Development Editorial Department and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

To address issues perplexing classic image dehazing methods, including halo effect in edge regions, color distortion in bright areas like sky, and hue shifts, we propose a novel image dehazing approach based on improved dark channel prior (SSPDCP:Dark Channel Prior based on Sky Detection and Super Pixel). This approach first applies HSV color transformation to hazy images to extract the brightness component for adaptive-threshold segmentation. Then it utilizes image connectivity analysis to identify the sky regions, from which the atmospheric light value is estimated, and separate transmittance maps of sky and non-sky areas are computed with a luminance model and a superpixel segmentation-based dark channel prior model, respectively. Subsequently, a superpixel-based fusion model is proposed to obtain a comprehensive transmittance map, ensuring smooth transition in boundary areas, which is further refined by multi-scale guided filtering. Finally, the dehazed image is naturally restored via the atmospheric scattering model and brightness enhancement processing. Experimental results show that the proposed approach identifies sky regions more continuously and completely, moreover, by employing superpixels instead of square windows, it effectively mitigates halo effects in acquiring transmittance maps. The estimation of atmospheric light values and transmittance maps is more objective and accurate. Both subjective qualitative and objective quantitative evaluations reveal advantages such as low overall error, excellent signal-to-noise ratio, and high structural similarity in dehazed images. Compared to the state-of-the-art methods, the proposed approach restores skies more naturally, weakens halo effect in edge regions, and achieves qualitative and quantitative improvements in dehazing performance. [ABSTRACT FROM AUTHOR]

Copyright of Secretary (16742354) is the property of Secretary Editorial Office and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

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Copyright of Transactions of the Chinese Society of Agricultural Engineering is the property of Chinese Society of Agricultural Engineering and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

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Copyright of Journal of Hubei Open Vocational College is the property of Journal of Hubei Open Vocational College Editorial Office and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

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Copyright of Hydrogeology & Engineering Geology / Shuiwendizhi Gongchengdizhi is the property of Hydrogeology & Engineering Geology Editorial Office and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

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Copyright of Journal of Petrochemical Universities / Shiyou Huagong Gaodeng Xuexiao Xuebao is the property of Journal Editorial Department Of Liaoning Shihua University and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

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Objective To analyze the influence of cyclic RNA homologous domain interacting protein kinase 3 (circ_HIPK3) on function and morphology of myloid β-protein (Aβ) induced hippocampal neurons by targeting miR-381-3p/ zinc finger protein 217 (ZNF217) axis. Methods Hippocampal neurons of neonatal rats were prepared and divided into the control group, the Aβ group, the si NC1 group, the si HIPK3 group, the si HIPK3+inhibitor NC group, the si HIPK3+miR381-3p inhibitor group, the si HIPK3+miR-381-3p inhibitor+si NC2 group and the si HIPK3+miR-381-3p inhibitor+si ZNF217 group. Except the control group, all the other groups were modeled by 40 µmol/L Aβ1~42. qRT-PCR was used to determine the circ of hippocampal neurons circ_HIPK3, miR-381-3p and ZNF217 mRNA levels. Cell morphology was observed by transmission electron microscope, and the survival rate of hippocampal neurons was measured by CCK-8 method. Hochesst 33342 method was used to measure apoptosis of hippocampal neurons. The intracellular Ca2+ fluorescence intensity of hippocampal neurons was detected by flow cytometry. The expression levels of P-Tau, B-cell lymphoma-2 (Bcl2), Bcl-2-associated X protein (Bax), Caspase-3 and ZNF217 proteins in hippocampal neurons were measured by Western blot assay. Double luciferase reporter genes were used to analyze the targeting relationship between miR-381-3p and circ_HIPK3, ZNF217. Results In the control group, the structure of hippocampal neurons was normal, the morphology of nucleus was normal, and there were no pathological changes in mitochondria and endoplasmic reticulum. In the Aβ group, hippocampal neurons showed degenerative changes, abnormal nuclear morphology, membrane invagination, a large number of mitochondria swelling and a large number of lipid droplets vacuoles in cytoplasm. Compared with the Aβ group, the hippocampal neuronal structure was partially restored in the si HIPK3 group. Compared with the si HIPK3 group, the hippocampal neuronal structure was severely damaged in the si HIPK3+miR-381-3p inhibitor group. Compared with the si HIPK3+miR-381-3p inhibitor group, the damage of hippocampal neurons in the si HIPK3+miR-381-3p inhibitor+si ZNF217 group was reduced. Compared with the control group, the circ_HIPK3, ZNF217 mRNA and ZNF217 protein levels, apoptosis rate, Ca2+ fluorescence intensity, P-Tau, Bax, Caspase-3 protein expression of hippocampal neurons were increased in the Aβ group, and the miR-381-3p level, survival rate and Bcl-2 protein expression decreased (P＜0.05). Compared with the Aβ group, the circ_HIPK3, ZNF217 mRNA and ZNF217 protein levels, apoptosis rate, Ca2+ fluorescence intensity, P-Tau, Bax and Caspase-3 protein expression of hippocampal neurons were decreased in the si HIPK3 group, and miR-381-3p level, survival rate and Bcl-2 protein expression increased (P＜0.05). Compared with the si HIPK3 group, the circ_HIPK3, ZNF217 mRNA and ZNF217 protein levels, apoptosis rate, Ca2+ fluorescence intensity, P-Tau, Bax and Caspase-3 protein expression of hippocampal neurons in the si HIPK3+miR-381-3p inhibitor group were increased, and the miR-381-3p level, survival rate and Bcl-2 protein expression decreased (P＜0.05). Compared with the si HIPK3+miR381-3p inhibitor group, the ZNF217 mRNA and ZNF217 protein levels, apoptosis rate, Ca2+ fluorescence intensity, P-Tau, Bax and Caspase-3 protein expression of hippocampal neurons in the si HIPK3+miR-381-3p inhibitor+si ZNF217 group were decreased, and the survival rate and Bcl-2 protein expression increased (P＜0.05). miR-381-3p targeted and combined with HIPK3 and ZNF217. Conclusion circ_HIPK3 silencing may ameliorate Aβ -induced damage of hippocampal neuronal structure and function by regulating miR-381-3p/ZNF217 axis. [ABSTRACT FROM AUTHOR]

Copyright of World Nuclear Geoscience is the property of World Nuclear Geoscience Editorial Office and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

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Copyright of Journal of Vibration, Measurement & Diagnosis is the property of Nanjing Hangkong Daxue and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

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