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Objective To explore the relationship between subjective well-being and self-efficacy among primary liver cancer patients after surgery, and provide scientific basis for clinical nursing intervention measures. Methods By convenience sampling, 100 patients were selected and investigated by the General Well-being Schedule(GWB), Hamilton Anxiety scale)HAMA and General Self-efficacy Scale(GSES). Results Postoperative liver cancer patients had a low level of subjective well-being(65. 04±6. 83). The score of anxiety status was (29. 02±4. 26), and all patients experienced different level of anxiety. The score of self-efficacy was(33. 98±6. 69). The subjective well-being was negatively related to anxiety level (P

Objective To investigate the psychological stress of newly recruited nurses in a specialist hospital for hepatobiliary diseases and related countermeasures. Methods A descriptive, phenomenological study was conducted and a purposive sampling strategy was used to recruit 100 newly recruited nurses in a specialist hospital for hepatobiliary diseases. The Self-Report Symptom Inventory(SCL～90) was used to investigate the psychological stress of newly recruited nurses, and potential causes and countermeasures were analyzed. Results There were 35 (35. 00%)nurses who had SCL～90 score of 160 or more. Findings showed that there was a significant difference in scores of somatization, obsessive-compulsive symptoms, interpersonal sensitivity and depression between newly recruited nurses and Chinese norms(P＜0. 01). Conclusion Newly recruited nurses experienced moderate to high level of psychological stress. Targeted coping strategies should be carried out to relieve the psychological pressure from work and improve the nursing quality. (目的 探讨肝胆专科医院新入职护士心理压力状况和相关应对措施, 为改善肝胆专科医院新入职护士心理压力状况和应对方式提供参考依据。方法 采取目的抽样法, 选取2021年1月—12月100例新入职护士为调查对象, 采取症状自评表(SCL-90)评定心理压力状况, 并进行原因分析。结果 100例被调查对象中有35(35. 00%)例SCL-90总分≥160分。与全国常模比较, 肝胆专科医院新入职护士在躯体化、强迫、人际关系敏感、抑郁等得分差有统计学意义(P＜0. 01)。结论 肝胆专科医院新入职护士心理压力大。建议采取有效的应对方式, 以减缓新入职护士工作压力, 提高护理质量。)

Copyright of Chinese Journal of Tissue Engineering Research / Zhongguo Zuzhi Gongcheng Yanjiu is the property of Chinese Journal of Tissue Engineering Research and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

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BACKGROUND: It has been found that salidroside can improve diabetic retinopathy. However, it is not clear whether salidroside can protect retinal Müller cells (rMC-1) against damage induced by high glucose. OBJECTIVE: To investigate the effect and mechanism of salidroside on oxidative stress and apoptosis induced by high glucose in rat rMC-1 cells. METHODS: The rMC-1 cells were divided into four groups: normal control group, high glucose group (culture medium with a final glucose concentration of 35.5 mmol/L), salidr oside group (treatment with salidroside for 4 hours followed by co-culture with high glucose) and PI3K inhibitor group (treatment with salidroside and PI3K inhibitor for 4 hours followed by co-culture with high glucose). The viability of the rMC-1 cells was measured by cell counting kit-8 assay. Reactive oxygen species production was detected by DCFH-DA. The superoxide dismutase and catalase activities were tested by specific kits. Cell ap optosis was detected by flow cytometry. Western blot was used to detect the expression levels of Cleaved caspase-3, PI3K, AKT and phosphorylated AKT. RESULTS AND CONCLUSION: Compared with the normal control group, the cell viability and the activities of antioxidant enzymes (superoxide dismutase, catalase) in the high glucose group were significantly decreased, the production of reactive oxygen species, apoptotic rate and the level of Cleaved caspase-3 were significantly increased, and the phosphorylated AKT/AKT ratio was down-regulated (P < 0.05). Compared with the high glucose group, salidroside significantly enhanced the cell viability and increased the activities of antioxidant enzymes (superoxide dismutase, catalase), decreased the production of reactive oxygen species, reduced the apoptotic rate of the cells and downregulated the expression level of Cleaved caspase-3 (P < 0.05). Salidroside also activated the phosphorylation of Akt (P < 0.05). Whereas, the addition of LY294002, a pharmacological inhibitor of PI3K, showed similar results in the high glucose group (P > 0.05). To conclude, salidroside can protect rMC-1 cells through inhibiting apoptosis and oxidative stress induced by high glucose. The main mechanism responsible for the inhibition of oxidative stress is the activation of the PI3K/Akt pathway. [ABSTRACT FROM AUTHOR]