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Objective To investigate the effect of Traditional Chinese Medicine (TCM)characteristic nursing combined with Liuzijue Qigong on rehabilitation of cardiac function in patients with chronic heart failure (CHF). Methods A total of 80 CHF patients were randomly divided into the study group and control group, with 40 cases in each group. All patients received routine drug therapy during hospitalization. Patients in the control group were give routine care including work-rest schedule, dietary guidance, psychological care and daily care. Patients in the study group were given TCM characteristic nursing combined with Liuzijue Qigong in addition to routine care. The Minnesota Living with Heart Failure Questionnaire (MLHFQ) was used to measure the patient quality of life, and outcomes of left ventricular ejection fractions (LVEF) and 6-minute walking test were measured and compared between two groups. Results Longer 6MWD and higher LEVF were reported in both groups at three months of intervention, and patients in the study group achieved better outcomes of 6MWD and higher LEVF compared with those in the control group(P＜0. 01). The score of MLHFQ decreased in both groups at three months of intervention, and score of MLHFQ in the study group was lower than that in the control group (P＜0. 01). Conclusion The TCM characteristic nursing combined with Liuzijue Qigong is potentially effective to improve the rehabilitation of cardiac function and long-term quality of life in CHF patients. (目的 探讨运用中医特色护理联合六字诀促进慢性心力衰竭患者心功能康复的效果。方法 选取2020年9月—2021年9月住院治疗的慢性心力衰竭患者80例为研究对象, 随机分为研究组和对照组, 各40例。两组患者均接受心力衰竭常规药物治疗。对照组采用常规护理, 指导患者休息与活动, 给予合理饮食指导、心理护理、生活护理等; 研究组在常规护理的基础上给予中医特色护理及气功六字诀指导。采用明尼苏达心力衰竭生活质量调查表(MLHFQ)评估患者生活质量, 记录左室射血分数(LVEF)和六分钟步行试验(6MWD)结果。结果 干预3个月时, 两组6MWD距离和LEVF均较入院时增加, 且研究组6MWD距离和LEVF较对照组改善明显, 差异有统计学意义(P＜0. 01)。干预3个月时, 两组MLHFQ评分均较入院时降低, 且研究组MLHFQ评分低于对照组, 差异有统计学意义(P＜0. 01)。结论 中医特色护理联合六字诀能改善慢性心力衰竭患者的心功能, 减轻临床症状, 提高患者的远期生活质量。)

Copyright of Journal of Food Safety & Quality is the property of Journal of Food Safety & Quality Editorial Department and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

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Objective To investigate the drug resistance of Mycobacterium tuberculosis in diabetes melli⁃ tus and pulmonary tuberculosis comorbidity (DM⁃TB). Methods A retrospective case-control study was conducted to review and analyze the clinical data of 1948 patients who was diagnosed with DM⁃TB in the inpatient and outpatient departments of Guangzhou Chest Hospital from January 2018 to December 2020. A total of 1012 cases with positive Mycobacterium tuberculosis culture and drug susceptibility results were screened out. After excluding the cases with incomplete data, 438 cases with complete data were included for analyses of age, gender, haemoglobin, albumin, glycatedhaemoglobin (HbA1c), extent of lesions, number of cavities, treatment history, sputum bacterial load and rifampicin resistance. The description method and Pearson Chi square test (χ²) were used to compare analyze the first and second line drug resistance of the strains from the 1012 patients. Among the selected cases, ？86 patients who were resistant against rifampicin were assigned as rifampicin-resistant (RR-TB)group, and then another 86 patients were randomly selected from the rest 352 patients sensitive to rifampicin by random digit table method as rifampicin-sensitive (RS-TB) group. The methods of？t-test, ranksum test or Pearson Chi square test (χ²) were used for the analyses. The clinical characteristics of the included cases were taken as independent variables and rifampicin resistance as dependent variables. Multivariate logistic regression analysis was performed to screen out independent risk factors for RR-TB in DM-TB patients. Results Among the 1012 patients with TB-DM, the rate of rifampicin resistance was 13.3%, the rate of initial rifampicin resistance was 6.8%, the rate of retreatment rifampicin resistance was 45.9%, with significant differences (χ² = 127.956, P = 0.000). The rate of multidrug-resistance was 11.77%, the rate of initial multidrug-resistance was 5.5%, the rate of retreatment multidrug-resistance was 42.9%, with significant difference between the two groups (χ² = 191.468, P = 0.000). Multivariate logistic regression analysis showed that retreatment (OR = 9.517, 95%CI：4.333 ~ 20.904, P = 0.000) was an independent risk factor for rifampicin resistance in the DM-TB patients, and senior ages (OR = 0.440, 95%CI： 0.252 ~ 0.771, P = 0.004)was an independent protective factor. Conclusion The prevalence of rifampicin resistance in DM-TB isrelatively high, especially among young patients and retreated patients. For DM⁃TB population, timely drug sensitivity test can help the early diagnosis and treatment of rifampicin⁃resistant tuberculosis. [ABSTRACT FROM AUTHOR]

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Copyright of Journal of Materials Science & Engineering (1673-2812) is the property of Journal of Materials Science & Engineering Editorial Office and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

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Copyright of Hydrographic Surveying & Charting / Haiyang Cehui is the property of Hydrographic Surveying & Charting Editorial Board and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

Copyright of Journal of Food Safety & Quality is the property of Journal of Food Safety & Quality Editorial Department and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

Objective · To investigate the role and mechanism of glutamic-pyruvic transaminase 2 (GPT2) on cisplatin resistance in gastric cancer. Methods · The Kaplan Meicr-Plotter database was used to analyze the relationship between GPT2 expression and poor prognosis of gastric cancer. The expressions of GPT2 in gastric cancer cells and tissues were detected by quantitative real-time PCR (qPCR), Western blotting and immunohistochemistry (IHC). The cytotoxicity of cifiphtin at different concentrations to human gastric cancer cells and normal gastric epithelial cells was detected by CCK-8 郎say* GPT2 overexpression and knockdown cell lines were constructed in cisplatin sensitive MKN28 cells and insensitive MKN45 cells, respectively. CCK-8 assay, colony formation 肪say and Western blotting were performed to evaluate the cellular cytotoxicity, sternness of cancer cells and the changes of key proteins in stenmess-related signaling pathways in GPT2 overexpression and knockdown cell lines, respectively* ResultsB The high expression of GPT2 was negatively correlated with the survival of gastric cancer patients. Gastric cancer cells with high expression of GPT2 were resistant to cisplatin， while cells with low expression of GPT2 were sensitive to cisplatin. Overexpression of GPT2 could decrease the cell sensitivity to cisplatin, nevertheless knockdown of GPT2 could increase the cell sensitivity to cisplatin. Meanwhile, the further study revealed that overejqjression of GPT2 could activate the intracellular regulated protein kinases (ERK) signaling pathway, up-regulate the expression of SRY-box 2 (SOX2) and Nanog homeobox (NANOG), and enhance the ability of colony foimation, while knockdown of GPT2 could inhibit ERK signaling pathway, reduce the expression of SOX2 and NANOG, and suppress the ability of colony formation. Conclusion · GPT2 ejqjressioii are related to the sensitivity of cisplatin treatment Overexpression of GPT2 can increase the resistance of gastric cancer to cisplatin treatment by activating ERK signaling pathway and up-regulating the expression of SOX2 and NANOG. [ABSTRACT FROM AUTHOR]

Copyright of Systems Engineering & Electronics is the property of Journal of Systems Engineering & Electronics Editorial Department and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

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Copyright of Journal of Northeastern University (Natural Science) is the property of Dongbei Daxue Xuebao and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

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Copyright of Journal of Agricultural Science & Technology (1008-0864) is the property of Journal of Agricultural Science & Technology and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

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目的　探讨心抑瘤素Ｍ（ＯＳＭ）／ＯＳＭ受体（ＯＳＭＲ）在压力负荷致心肌肥厚小鼠心肌组织中的表达及其 意义。方法　选择野生型Ｃ５７ＢＬ／６Ｊ小鼠６０只，随机分成为假手术组（Ｓｈａｍ组）、模型组（ＡＢ组）各３０只，ＡＢ组 采用胸主动脉缩窄术建立心肌肥厚模型。两组于术后４周行超声检测心功能，处死后观察心脏大体观并行心肌组 织病理学检测。应用ＲＴ-ＰＣＲ检测小鼠心肌组织ＯＳＭ、ＩＬ-３１、ＯＳＭＲｍＲＮＡ表达，应用Ｗｅｓｔｅｒｎｂｌｏｔｔｉｎｇ检小鼠心肌 组织测ＯＳＭ、ＩＬ-３１、ＯＳＭＲ亚单位（ＯＳＭＲβ）蛋白表达。结果　术后４周，大体观察显示ＡＢ组小鼠心脏体积增大， 心腔扩大，病理染色显示心肌细胞横截面积增加。与Ｓｈａｍ组相比，ＡＢ组舒张期室间隔厚度、左室舒张末内径和左 室收缩末内径增大，射血分数及短轴缩短率减小（Ｐ均＜０．０５）；ＡＢ组心肌ＯＳＭ ｍＲＮＡ、ＯＳＭ蛋白、ＯＳＭＲｍＲＮＡ、 ＯＳＭＲβ蛋白表达均较Ｓｈａｍ组明显上调（Ｐ＜０．０５或＜０．０１）。结论　压力负荷致心肌肥厚模型小鼠心肌组织中 ＯＳＭ及其ＯＳＭＲβ均发生异常表达，ＯＳＭ／ＯＳＭＲβ系统参与了心肌重构导致心肌肥厚的过程。 [ABSTRACT FROM AUTHOR]