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Copyright of Journal of Irrigation & Drainage is the property of Journal of Irrigation & Drainage Editorial Office and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

Copyright of Chemical Engineering (China) / Huaxue Gongcheng is the property of Hualu Engineering Science & Technology Co Ltd. and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

Copyright of Journal of Mechanical Strength / Jixie Qiangdu is the property of Zhengzhou Research Institute of Mechanical Engineering and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

Copyright of Lubrication Engineering (0254-0150) is the property of Editorial Office of LUBRICATION ENGINEERING and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

Copyright of Shandong Medical Journal is the property of Shandong Medical Health Newspapers and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

Objective: To investigate the effect of chemokine 8(CXCL8) on the development of atherosclerosis (AS) in mice and its possible mechanism. Methods: 40 male Apo E-/- mice were randomly divided into AS group and G31P group, 20 mice in each group, meanwhile 20 male C57BL/6J mice was assigned into the control group. AS group mice were fed with high fat diet for 12 weeks, G31P group mice were were fed with high fat diet for 12 weeks and subcutaneous injection at the same time of G31P (The injection volume is 500 μg/kg), The control group mice were fed with regular diet for 12 weeks. Then ELISA method was used to measure serum triglyceride (TG), low density lipoprotein cholesterol (LDL-C), high density lipoprotein cholesterol (HDL-C), total cholesterol (TC) level. Real- time polymerase chain reaction (RT-PCR) were used to measure CXCL8mRNA and MMP9mRNA expression in aorta tissue. Western- blot methods was performed to test the protein expression levels of CXCL8 and MMP9 in aorta tissue. Results: After 12 weeks high-fat diet feeding, the levels of TG, LDL-C and TC in serum of AS group were higher than those in the G31P group, while the HDL-C level was lower than that in the G31P group (P<0.05). The levels of TG, LDL-C and TC in the serum of G31P group were higher than control group, HDL-C level was lower than the control group (P<0.05). The expression levels of CXCL8mRNA, MMP9mRNA, CXCL8 protein and MMP9 protein in aortic tissue of AS group were higher than those in G31P group (P<0.05), and the expression levels of CXCL8mRNA, MMP9mRNA, CXCL8 protein and MMP9 protein in aortic tissue of G31P group were higher than those in the control group (P<0.05). Conclusion: CXCL8 has high expression in the aortic tissues of as mice, it may be involved in the development of as by affecting the expression of MMP9, inhibiting the expression of CXCL8 or delaying the development of AS. [ABSTRACT FROM AUTHOR]

Objective: To analyze the correlation between human cytomegalovirus (HCMV) infection and the inflammatory mediators of patients with acute coronary syndrome (ACS), and explore the r ole of HCMV infection in the occurrence and development of ACS. Methods: 118 cases of coronary heart disease patients who were admitted to our hospital from May 2017 to May 2019 were selected, and they were divided into A CS group (n=81) and stable angina pectoris (SAP) group (n= 37) according to illness condition, another 40 healthy volunteers in our hospital who were selected as control group during the same period. The serum specific HC MV-IgG and HCMV-IgM were detected in all subjects, and the levels of serum sP-selection, tumor necrosis factor-a (TNF-a) and high sensitivity e-re-active protein (hs-CRP) were compared in all subjects. The correlation between serum sP-selectin, TNF-α and hs-CRP levels and the titer of HCMV-IgM antibody in patients of ACS group were analyzed. Results: The positive rates of HCMV-IgG in the ACS group and in the SAP group were 81.48% and 78.38% respectively, which were significantly higher than 45. 00% in the control group, and the differences were statistically significant (P<0.05). The positive rate of HCMV-IgM in the AC S group was 40.74%, which was significantly higher than 10.81% in the SAP group and 5.00% in the control group, and the differences were statistically significant (P<0.05). The levels of serum sP-selection, TNF-α and hs-CRP in the A CS group w er e significantly higher than those in the SAP group and in the control group, and the differ ences were statistically significant (P<0.05). The levels of serum serum sP-selection, TNF-α and hs-CRP in patients with HCMV- IgM positive in the ACS group were significantly higher than those in patients with HCMV-IgM negative, and the differences were statistically significant (?<0.05). The levels of serum sP-selection, TNF-α, hs-CRP in the ACS group were positively correlated with the titer of HCMV -IgM antibody (P

Copyright of Pharmaceutial Care & Research is the property of Editorial Board of Pharmaceutial Care & Research and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)