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Background/Aim. The field of lung ultrasonography (US) is a fast-developing one, and it provides the medical community with numerous new diagnostic opportunities. The aim of this study was to examine the etiology of dyspnea on admission and assess the stage of heart failure (HF) according to the US examination of the heart and lungs. Methods. The cross-sectional study included a total of 110 patients treated for the symptoms of dyspnea. The study included all patients treated for any heart or pulmonary condition, as well as patients who reported the first episode of dyspnea with-out any previous illness. The most important diagnostic sign in the US of the lungs in patients with HF was the appearance of B-lines or “comets” (ultrasound artifacts reminiscent of comet tails), which indicate the accumulation of fluid in the interstitium of the lungs. Results. The mean number of registered “comets” in the total number of patients was 14.2 ± 7.4 (minimum 2, maximum 30). The mean number of “comets” among patients with HF was 18.8 ± 5.9. The mean number of “comets” among patients without HF was 8.0 ± 3.7 (p ˂ 0.001). Multivariate linear regression analyses showed the association between the number of “comets” and HF (p ˂ 0.001). Conclusion. As the assessment of present “comets” in pulmonary US examination is safe and non-invasive, it can easily be integrated into the daily clinical practice because it has been shown that the number of pulmonary “comets” is significantly higher in patients with HF compared to those with dyspnea of other etiologies.

Introduction: Arterial hypertension is a major cardiovascular risk factor affecting about 10-40% of the adult population. Secondary endocrine hypertension most often results from excessive aldosterone secretion. Complications related to excessive aldosterone secretion include atrial fibrillation, myocardial infarction, myocardial fibrosis, left ventricular hypertrophy, stroke, and increased cardiovascular mortality. Case report: This report presents a hypotensive woman with hypertensive reactions, newly diagnosed unilateral hyperplasia of the left adrenal gland and secondary hyperaldosteronism. Due to good blood pressure and normalized electrolyte status as a result of antihypertensive drug therapy and absence of damage to target organs, surgical treatment of unilateral adrenal hyperplasia was postponed. Conclusion: In case of midlife and late-life hypertension, it is necessary to consider a cause in the patient's endocrine system. AUTHORS SUMMARY SRPSKI 2021; 50 (1,2) 51-54

Chronic obstructive pulmonary disease (COPD) is a disease with periods of remission and exacerbation and consequential defect of the pulmonary function. It is estimated that around 3 million people die from it annually and predicted that until 2030 the number of deaths will be up to 6 million per year. According to the reports of the World Health Organization and Global initiative for chronic obstructive lung disease (GOLD) it is predicted that COPD in the next decade will be the third leading cause of death in the world.[ 1, 2 ] It is characterized by limited air flow in the breathing pathways which is not completely reversible to therapy. Since there are systemic inflammations at COPD, the mechanisms of the processes have consequences outside the lungs also. Comorbidities are frequent at patients with the Chronic obstructive lung disease and they affect the disease prognosis substantially. The most common comorbidities are cardiovascular diseases, metabolic syndrome, diabetes, anemia, osteoporosis, anxiety and depression, pulmonary embolism.

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Copyright of Medical Gazette / Medicinski Glasnik is the property of Specijalna bolnica za bolesti stitaste zlezde i bolesti metabolizma Zlatibor and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)