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176 results on '"Ogawa, Yutaka"'

154. Authors

Author

Schubert, F., primary, Bruch, Udo, additional, Cook, R., additional, Diehl, H., additional, Ennis, Philip J., additional, Jakobeit, W., additional, Penkalla, H. J., additional, Heesen, Eberhard te, additional, Ullrich, G., additional, Lee, Kyung Sub, additional, Kurata, Yuji, additional, Ogawa, Yutaka, additional, Kondo, Tatsuo, additional, Tanabe, Tatsuhiko, additional, Sakai, Yoshikazu, additional, Shikama, Tatsuo, additional, Fujitsuka, Masakazu, additional, Yoshida, Heitaro, additional, Watanabe, Ryoji, additional, Nakanishi, Tsuneo, additional, Kawakami, Haruo, additional, Cook, Roger H., additional, Schneider, Klaus, additional, Hartnagel, Walter, additional, Schepp, Peter, additional, Ilschner, Bernhard, additional, Osthoff, Walter, additional, Schuster, Hans, additional, Nickel, Hubertus, additional, Neubauer, Burkhard, additional, Meurer, Hans-Peter, additional, Gnirss, Günter K. H., additional, Mergler, Wolfgang, additional, Raule, Gerhard, additional, Ullrich, Georg, additional, Soo, Peter, additional, Sabatini, Robert L., additional, Tsuji, Hirokazu, additional, Schuhmacher, Dieter, additional, Mohr, Klaus P., additional, Krompholz, Klaus, additional, Bodmann, Erik, additional, Huthmann, Horst, additional, Quadakkers, Willem J., additional, Inouye, H., additional, Brenner, Karl G. E., additional, Graham, Leslie W., additional, Bates, H. G. A., additional, Shindo, Masami, additional, Li, C. C., additional, Johnson, W. R., additional, Thompson, L. D., additional, Cappelaere, M., additional, Perrot, M., additional, and Sannier, J., additional

Published
1984
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155. Authors

Author

Kikuchi, Kenji, primary, Kaburaki, Hideo, additional, Sanokawa, Konomo, additional, Kawaguchi, Katsuyuki, additional, Nemoto, Masaaki, additional, Watanabe, Shintaro, additional, Thiele, W., additional, Lehnert, Günter, additional, Borel, Marc-Olivier, additional, Corthay, André, additional, Fischli, Heinz, additional, Fricker, Hans W., additional, Bennett, Michael J., additional, Houlton, Michael R., additional, Moore, Donald A., additional, Foster, Alan I., additional, Swidzinski, Michael A. M., additional, Quillico, J. J., additional, Despresles, P. J., additional, Schaefer, Josef, additional, Stöver, Detlev, additional, Hecker, Rudolf, additional, Buchkremer, Hans Peter, additional, Jonas, Heinz, additional, Zink, Uwe, additional, Huschka, Hans, additional, Luthardt, Guenther, additional, Portscher, Volker E., additional, Mühlratzer, August, additional, Zeilinger, Hans, additional, Esser, Hans Günter, additional, Kishimoto, Naoki, additional, Tanabe, Tatsuhiko, additional, Araki, Hiroshi, additional, Yoshida, Heitaro, additional, Watanabe, Ryoji, additional, Thiele, Bernd A., additional, Diehl, Hermann, additional, Ohly, Wilhelm, additional, Weber, Heinz, additional, Lindgren, James R., additional, Suzuki, Masahide, additional, Fukaya, Kiyoshi, additional, Kodaira, Tsuneo, additional, Oku, Tatsuo, additional, Watanabe, Katsutoshi, additional, Kondo, Tatsuo, additional, Ogawa, Yutaka, additional, Alberman, A., additional, Bley, G., additional, Pépin, P., additional, Soulat, P., additional, Nickel, Hubertus, additional, Schubert, Florian, additional, Hughes, Peter Thomas, additional, Allen, Donald C., additional, Bodmann, Erik, additional, Breuer, Hans-Jürgen, additional, Raule, Gerhard, additional, Rödig, Manfred, additional, Kitagawa, Masaki, additional, Hattori, Hiroshi, additional, Ohtomo, Akira, additional, Teramae, Tetsuo, additional, Hamanaka, Junichi, additional, Ukikusa, Hiroshi, additional, Penkalla, Heinz-Josef, additional, Over, Hans-Helmut, additional, Goebbels, Klaus, additional, and Kapitza, Harald, additional

Published
1984
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172. A Case of Esophageal Dysplasia Associated with Human Papilloma Virus.

Author

Yoshikane, Hiroaki, Yokoi, Takio, Suzuki, Toshiyuki, Yoshioka, Nobuo, Ogawa, Yutaka, and Hamajima, Eiji

Subjects
LETTERS to the editor, PAPILLOMAVIRUSES
Abstract

Presents a letter to the editor about a case of esophageal dysplasia associated with human papilloma virus.

Published
1995

174. Conjugated docosahexaenoic acid suppresses KPL-1 human breast cancer cell growth in vitroand in vivo: potential mechanisms of action

Author

Tsujita-Kyutoku, Miki, Yuri, Takashi, Danbara, Naoyuki, Senzaki, Hideto, Kiyozuka, Yasuhiko, Uehara, Norihisa, Takada, Hideho, Hada, Takahiko, Miyazawa, Teruo, Ogawa, Yutaka, and Tsubura, Airo

Abstract

Introduction: The present study was conducted to examine the effect of conjugated docosahexaenoic acid (CDHA) on cell growth, cell cycle progression, mode of cell death, and expression of cell cycle regulatory and/or apoptosis-related proteins in KPL-1 human breast cancer cell line. This effect of CDHA was compared with that of docosahexaenoic acid (DHA). Methods: KPL-1 cell growth was assessed by colorimetric 3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazolium bromide assay; cell cycle progression and mode of cell death were examined by flow cytometry; and levels of expression of p53, p21Cip1/Waf1, cyclin D1, Bax, and Bcl-2 proteins were examined by Western blotting analysis. In vivotumor growth was examined by injecting KPL-1 cells subcutaneously into the area of the right thoracic mammary fat pad of female athymic mice fed a CDHA diet. Results: CDHA inhibited KPL-1 cells more effectively than did DHA (50% inhibitory concentration for 72 hours: 97 μmol/l and 270 μmol/l, respectively). With both CDHA and DHA growth inhibition was due to apoptosis, as indicated by the appearance of a sub-G1fraction. The apoptosis cascade involved downregulation of Bcl-2 protein; Bax expression was unchanged. Cell cycle progression was due to G0/G1arrest, which involved increased expression of p53 and p21Cip1/Waf1, and decreased expression of cyclin D1. CDHA modulated cell cycle regulatory proteins and apoptosis-related proteins in a manner similar to that of parent DHA. In the athymic mouse system 1.0% dietary CDHA, but not 0.2%, significantly suppressed growth of KPL-1 tumor cells; CDHA tended to decrease regional lymph node metastasis in a dose dependent manner. Conclusion: CDHA inhibited growth of KPL-1 human breast cancer cells in vitromore effectively than did DHA. The mechanisms of action involved modulation of apoptosis cascade and cell cycle progression. Dietary CDHA at 1.0% suppressed KPL-1 cell growth in the athymic mouse system.

Published
2004
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175. Detection and identification of Brettanomyces/Dekkera sp. yeasts with a loop-mediated isothermal amplification method

Author

Hayashi, Nobuyuki, Arai, Ritsuko, Tada, Setsuzo, Taguchi, Hiroshi, and Ogawa, Yutaka

Subjects
*BRETTANOMYCES, *DNA, *DISTILLED water, *SOFT drinks
Abstract

Abstract: Primer sets for a loop-mediated isothermal amplification (LAMP) method were developed to specifically identify each of the four Brettanomyces/Dekkera species, Dekkera anomala, Dekkera bruxellensis, Dekkera custersiana and Brettanomyces naardenensis. Each primer set was designed with target sequences in the ITS region of the four species and could specifically amplify the target DNA of isolates from beer, wine and soft drinks. Furthermore, the primer sets differentiated strains of the target species from strains belonging to other species, even within the genus Brettanomyces/Dekkera. Moreover, the LAMP method with these primer sets could detect about 1×101 cfu/ml of Brettanomyces/Dekkera yeasts from suspensions in distilled water, wine and beer. This LAMP method with primer sets for the identification of Brettanomyces/Dekkera yeasts is advantageous in terms of specificity, sensitivity and ease of operation compared with standard PCR methods. [Copyright &y& Elsevier]

Published
2007
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176. Comparison of transforming growth factor-beta/Smad signaling between normal dermal fibroblasts and fibroblasts derived from central and peripheral areas of keloid lesions.

Author

Tsujita-Kyutoku M, Uehara N, Matsuoka Y, Kyutoku S, Ogawa Y, and Tsubura A

Subjects
Actins metabolism, Activin Receptors, Type I metabolism, Adolescent, Adult, Blotting, Western, Cell Culture Techniques, Cell Proliferation, Cell Survival, Cells, Cultured, Female, Fibroblasts metabolism, Humans, Keloid metabolism, Kinetics, Male, Middle Aged, Protein Serine-Threonine Kinases, Receptor, Transforming Growth Factor-beta Type I, Receptor, Transforming Growth Factor-beta Type II, Receptors, Transforming Growth Factor beta metabolism, Skin cytology, Smad2 Protein metabolism, Smad3 Protein metabolism, Smad4 Protein metabolism, Up-Regulation, Fibroblasts physiology, Keloid physiopathology, Signal Transduction, Smad Proteins metabolism, Transforming Growth Factor beta metabolism
Abstract

Background: Keloids are benign proliferations of fibroblasts, but their exact etiology and molecular pathogenesis are unknown., Materials and Methods: Fibroblasts were isolated from the central and peripheral regions of keloids, and the growth behavior and molecular characteristics of the keloid fibroblasts were compared with those of age-adjusted normal dermal fibroblasts., Results: Central (but not peripheral) keloid fibroblasts exhibited significantly increased growth and high levels of expression of transforming growth factor-beta (TGF-beta) receptor 1 and Smad 2/3., Conclusion: Proliferation of central keloid fibroblasts, which results in keloid formation, appears to mainly involve the TGF-beta/Smad pathway.

Published
2005

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