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810 results on '"Zuo, Zhicai"'

201. Identification, Genotyping, and Pathogenicity ofTrichosporonspp. Isolated from Giant Pandas

Author

Ma, Xiaoping, primary, Jiang, Yaozhang, additional, Wang, Chengdong, additional, Gu, Yu, additional, Cao, Sanjie, additional, Huang, Xiaobo, additional, Wen, Yiping, additional, Zhao, Qin, additional, Wu, Rui, additional, Wen, Xintian, additional, Yan, Qigui, additional, Han, Xinfeng, additional, Zuo, Zhicai, additional, Deng, Junliang, additional, Ren, Zhihua, additional, Yu, Shumin, additional, Shen, Liuhong, additional, Zhong, Zhijun, additional, Peng, Guangneng, additional, Liu, Haifeng, additional, and Zhou, Ziyao, additional

Published
2018
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205. Occurrence and genetic characterization of Giardia duodenalis and Cryptosporidium spp. from adult goats in Sichuan Province, China

Author

Zhong, Zhijun, primary, Tu, Rui, additional, Ou, Hongping, additional, Yan, Guangwen, additional, Dan, Jiaming, additional, Xiao, Qicheng, additional, Wang, Ya, additional, Cao, Suizhong, additional, Shen, Liuhong, additional, Deng, Junliang, additional, Zuo, Zhicai, additional, Ma, Xiaoping, additional, Zhou, Ziyao, additional, Liu, Haifeng, additional, Yu, Shumin, additional, Ren, Zhihua, additional, Hu, Yanchun, additional, and Peng, Guangneng, additional

Published
2018
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207. Histopathological Injuries, Ultrastructural Changes, and Depressed TLR Expression in the Small Intestine of Broiler Chickens with Aflatoxin B1

Author

Wang, Fengyuan, primary, Zuo, Zhicai, additional, Chen, Kejie, additional, Gao, Caixia, additional, Yang, Zhuangzhi, additional, Zhao, Song, additional, Li, Jianzhen, additional, Song, Hetao, additional, Peng, Xi, additional, Fang, Jing, additional, Cui, Hengmin, additional, Ouyang, Ping, additional, Zhou, Yi, additional, Shu, Gang, additional, and Jing, Bo, additional

Published
2018
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208. Activation of Porcine Alveolar Macrophages by Actinobacillus pleuropneumoniae Lipopolysaccharide via the Toll-Like Receptor 4/NF-κB-Mediated Pathway

Author

Li, Bi, primary, Fang, Jing, additional, Zuo, Zhicai, additional, Yin, Sirui, additional, He, Tingting, additional, Yang, Mingxian, additional, Deng, Junliang, additional, Shen, Liuhong, additional, Ma, Xiaoping, additional, Yu, Shumin, additional, Wang, Ya, additional, and Ren, Zhihua, additional

Published
2018
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209. ikinci

Author

Fang JING, Zuo ZHICAI, Cui HENGMIN, Chi XIAOFENG, Peng XI, Ding XUEMEI, and Li XIAOCONG

Subjects
General Veterinary
Published
2017
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214. Sodium Fluoride Arrests Renal G2/M Phase Cell-Cycle Progression by Activating ATM-Chk2-P53/Cdc25C Signaling Pathway in Mice

Author

Luo, Qin, primary, Guo, Hongrui, additional, Kuang, Ping, additional, Cui, Hengmin, additional, Deng, Huidan, additional, Liu, Huan, additional, Lu, Yujiao, additional, Wei, Qin, additional, Chen, Linlin, additional, Fang, Jing, additional, Zuo, Zhicai, additional, Deng, Junliang, additional, Li, Yinglun, additional, Wang, Xun, additional, and Zhao, Ling, additional

Published
2018
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216. In Vitro Acaricidal Activity of the Thymol against Sarcoptes scabiei and Regulating Effects on Enzyme Activity

Author

Zhou, Yancheng, primary, Liao, Fei, additional, Fu, Jie, additional, Weng, Jiahua, additional, Mo, Quan, additional, Xu, Ruiguang, additional, Zhang, Yong, additional, Sun, Wei, additional, Yue, Dong, additional, Ren, Zhihua, additional, Zhong, Zhijun, additional, Zuo, Zhicai, additional, Peng, Guangneng, additional, Deng, Junliang, additional, Tang, Cheng, additional, and Hu, Yanchun, additional

Published
2018
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221. Protective Role of Selenium in Immune-Relevant Cytokine and Immunoglobulin Production by Piglet Splenic Lymphocytes Exposed to Deoxynivalenol

Author

Wang, Xuemei, primary, Zuo, Zhicai, additional, Deng, Junliang, additional, Zhang, Zhuo, additional, Chen, Changhao, additional, Fan, Yu, additional, Peng, Guangneng, additional, Cao, Suizhong, additional, Hu, Yanchun, additional, Yu, Shumin, additional, Chen, Chaoxi, additional, and Ren, Zhihua, additional

Published
2017
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223. A study on the expression of apoptotic molecules related to death receptor and endoplasmic reticulum pathways in the jejunum of AFB1-intoxicated chickens

Author

Zheng, Zhixiang, primary, Zuo, Zhicai, additional, Zhu, Panpan, additional, Wang, Fengyuan, additional, Yin, Heng, additional, Peng, Xi, additional, Fang, Jing, additional, Cui, Hengmin, additional, Gao, Caixia, additional, Song, Hetao, additional, Ouyang, Ping, additional, Zhou, Yi, additional, and Zhao, Song, additional

Published
2017
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224. Effects of sodium fluoride on blood cellular and humoral immunity in mice

Author

Guo, Hongrui, primary, Kuang, Ping, additional, Luo, Qin, additional, Cui, Hengmin, additional, Deng, Huidan, additional, Liu, Huan, additional, Lu, Yujiao, additional, Fang, Jing, additional, Zuo, Zhicai, additional, Deng, Junliang, additional, Li, Yinglun, additional, Wang, Xun, additional, and Zhao, Ling, additional

Published
2017
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225. Euptox A Induces G1 Arrest and Autophagy via p38 MAPK- and PI3K/Akt/mTOR-Mediated Pathways in Mouse Splenocytes

Author

Mo, Quan, primary, Hu, Liwen, additional, Weng, Jiahua, additional, Zhang, Yong, additional, Zhou, Yancheng, additional, Xu, Ruiguang, additional, Zuo, Zhicai, additional, Deng, Junliang, additional, Ren, Zhihua, additional, Zhong, Zhijun, additional, Peng, Guangneng, additional, Nong, Xiang, additional, Wei, Yahui, additional, and Hu, Yanchun, additional

Published
2017
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226. Sodium fluoride induces renal inflammatory responses by activating NF-κB signaling pathway and reducing anti-inflammatory cytokine expression in mice

Author

Luo, Qin, primary, Cui, Hengmin, additional, Deng, Huidan, additional, Kuang, Ping, additional, Liu, Huan, additional, Lu, Yujiao, additional, Fang, Jing, additional, Zuo, Zhicai, additional, Deng, Junliang, additional, Li, Yinglun, additional, Wang, Xun, additional, and Zhao, Ling, additional

Published
2017
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230. Resistin increases the expression of NOD2 in mouse monocytes

Author

Ren, Yi, primary, Wan, Taomei, additional, Zuo, Zhicai, additional, Cui, Hengmin, additional, Peng, Xi, additional, Fang, Jing, additional, Deng, Junliang, additional, Hu, Yanchun, additional, Yu, Shuming, additional, Shen, Liuhong, additional, Ma, Xiaoping, additional, Wang, Ya, additional, and Ren, Zhihua, additional

Published
2017
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231. The Molecular Mechanisms of Protective Role of Se on the G0/G1 Phase Arrest Caused by AFB1 in Broiler's Thymocytes.

Author

Guan, Ke, Li, Hang, Zuo, Zhicai, Wang, Fengyuan, Hu, Ping, Peng, Xi, Fang, Jing, Cui, Hengmin, Shu, Gang, and Ouyang, Ping

Abstract

This research was designed to explore the protective effects of sodium selenite on G0/G1 phase arrest induced by AFB1 in thymocytes of broilers. Two hundred eighty-eight Cobb broilers were divided into control group, + Se group (0.4 mg/kg Se), AFB1 group (0.6 mg/kg AFB1), and AFB1 + Se group (0.6 mg/kg AFB1 + 0.4 mg/kg Se). The results revealed that 0.4 mg/kg Se supplement in diets could improve the AFB1-induced histological lesions in the thymus consisting of the more vacuoles and nuclear debris in thymic cortical area. The results of flow cytometric detect showed that 0.4 mg/kg Se relieved the G0/G1 phase arrest caused by AFB1 in thymocytes. The results of transcription levels of ATM, p53, p21, p27, p15, p16, CyclinD1, CyclinE, Cdk6, Cdk2, and PCNA genes by qRT-PC, and protein expression level of PCNA by immunohistochemistry demonstrated that 0.4 mg/kg Se could reduce the adverse effects of AFB1 on these parameters. In conclusion, Se could relieve AFB1-induced G0/G1 phase arrest by p15 (or p16)-CyclinD1/Cdk6, ATM-p53-p21-CyclinE/Cdk2, p27-CyclinE/Cdk2 pathways. [ABSTRACT FROM AUTHOR]

Published
2019
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232. Selenium Rescues Aflatoxin B1-Inhibited T Cell Subsets and Cytokine Levels in Cecal Tonsil of Chickens.

Author

Wang, Fengyuan, Zuo, Zhicai, Chen, Kejie, Peng, Xi, Fang, Jing, Cui, Hengmin, Shu, Gang, He, Min, and Tang, Li

Abstract

Cecal tonsil is the largest peripheral lymphoid organ of the gut-associated lymphoid tissue executing immune function. To evaluate the protective effect of selenium (Se) on the cecal tonsil of chicken exposed to aflatoxin B1 (AFB1), 144 1-day-old healthy Cobb chickens were randomly divided into four groups, and fed with basal diet (control group), 0.6 mg/kg AFB1 (AFB1 group), 0.4 mg/kg Se supplement (+Se group), and 0.6 mg/kg AFB1 + 0.4 mg/kg Se supplement (AFB1 + Se group) for 21 days, respectively. The results showed that AFB1 significantly decreased the percentages of CD3+, CD3+CD4+, CD3+CD8+ T cells, and the CD4+/CD8+ ratio, and suppressed the expressions of IL-2, IL-4, TNF-α, and IFN-γ mRNA in the cecal tonsil. However, Selenium (Se) supplied in the diets restored the percentages of T cell subsets, the CD4+/CD8+ ratio, and mRNA expressions of cytokines in the AFB1 group to be close to those in the control group, and did not exhibit obvious toxicity to the cecal tonsil. These results indicated that Se exerted protective effect against AFB1 on the functions of cecal tonsil, and also partially uncovered a new role of Se that could protect cecal tonsil of chickens from immunotoxicity of AFB1. [ABSTRACT FROM AUTHOR]

Published
2019
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236. Suppressive subtractive hybridization reveals different gene expression between high and low virulence strains of Cladosporium cladosporioides

Author

Gu, Yu, primary, Liu, Yanfang, additional, Cao, Sanjie, additional, Huang, Xiaobo, additional, Zuo, Zhicai, additional, Yu, Shumin, additional, Deng, Junliang, additional, Ding, Chunbang, additional, Yuan, Ming, additional, Shen, Liuhong, additional, Wu, Rui, additional, Wen, Yiping, additional, Ren, Zhihua, additional, Zhao, Qin, additional, Peng, Guangneng, additional, Zhong, Zhijun, additional, Wang, Chengdong, additional, and Ma, Xiaoping, additional

Published
2016
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241. Diet-induced obese mice exhibit altered immune responses to acute lung injury induced byEscherichia coli

Author

Wan, Taomei, primary, Yuan, Guiqiang, additional, Ren, Yi, additional, Zuo, Zhicai, additional, Wang, Zhengyi, additional, Jia, Yiping, additional, Cui, Hengmin, additional, Peng, Xi, additional, Fang, Jing, additional, Deng, Junliang, additional, Yu, Shumin, additional, Hu, Yanchun, additional, Shen, Liuhong, additional, Ma, Xiaoping, additional, Wang, Ya, additional, and Ren, Zhihua, additional

Published
2016
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249. Selenium Ameliorates AFB1−Induced Excess Apoptosis in Chicken Splenocytes Through Death Receptor and Endoplasmic Reticulum Pathways.

Author

Fang, Jing, Zhu, Panpan, Yang, Zhuangzhi, Peng, Xi, Zuo, Zhicai, Cui, Hengmin, Ouyang, Ping, Shu, Gang, Chen, Zhengli, Huang, Chao, and Liu, Wentao

Abstract

Aflatoxin B1 (AFB1) can cause hepatotoxicity, genotoxicity, and immunosuppressive effects for a variety of organisms. Selenium (Se), as an essential nutrient element, plays important protective effects against cell apoptosis induced by AFB1. This research aimed to reveal the ameliorative effects of selenium on AFB1-induced excess apoptosis in chicken splenocytes through death receptor and endoplasmic reticulum pathways in vivo. Two hundred sixteen neonatal chickens, randomized into four treatments, were fed with basal diet (control treatment), 0.4 mg/kg Se supplement (+Se treatment), 0.6 mg/kg AFB1 (AFB1 treatment), and 0.6 mg/kg AFB1 + 0.4 mg/kg Se (AFB1 + Se treatment) during 21 days of experiment, respectively. Compared with the AFB1 treatment, the levels of splenocyte apoptosis in the AFB1 + Se treatment were obviously dropped by flow cytometry and TUNEL assays although they were still significantly higher than those in the control or + Se treatments. Furthermore, the mRNA expressions of CASP-3, CASP-8 and CASP-10, GRP78, GRP94, TNF-α, TNF-R1, FAS, and FASL of splenocytes in the AFB1 + Se treatment by qRT-PCR assay were significantly decreased compared with the AFB1 treatment. These results indicate that Se could partially ameliorate the AFB1-caused excessive apoptosis of chicken splenocytes through downregulation of endoplasmic reticulum and death receptor pathway molecules. This research may rich the knowledge of the detoxification mechanism of Se on AFB1-induced apoptosis. [ABSTRACT FROM AUTHOR]

Published
2019
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250. A mini review of fluoride-induced apoptotic pathways.

Author

Wei, Qin, Deng, Huidan, Cui, Hengmin, Fang, Jing, Zuo, Zhicai, Deng, Junliang, Li, Yinglun, Wang, Xun, and Zhao, Ling

Subjects
FLUORIDES, APOPTOSIS, OXIDATIVE stress, IMMUNOTOXICOLOGY, CELL-mediated cytotoxicity
Abstract

Fluorine or fluoride can have toxic effects on bone tissue and soft tissue at high concentrations. These negative effects include but not limited to cytotoxicity, immunotoxicity, blood toxicity, and oxidative damage. Apoptosis plays an important role in fluoride-induced toxicity of kidney, liver, spleen, thymus, bursa of Fabricius, cecal tonsil, and cultured cells. Here, apoptosis activated by high level of fluoride has been systematically reviewed, focusing on three pathways: mitochondrion-mediated, endoplasmic reticulum (ER) stress-mediated, and death receptor-mediated pathways. However, very limited reports are focused on the death receptor-mediated apoptosis pathways in the fluoride-induced apoptosis. Therefore, understanding and discovery of more pathways and molecular mechanisms of fluoride-induced apoptosis may contribute to designing measures for preventing fluoride toxicity. [ABSTRACT FROM AUTHOR]

Published
2018
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