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1,169 results on '"Williams, Scott M."'

201. Haplotype genetic score analysis in 10,734 mother/infant pairs reveals complex maternal and fetal genetic effects underlying the associations between maternal phenotypes, birth outcomes and adult phenotypes

Author

Chen, Jing, primary, Bacelis, Jonas, additional, Navais, Pol Sole, additional, Srivastava, Amit, additional, Juodakis, Julius, additional, Rouse, Amy, additional, Hallman, Mikko, additional, Teramo, Kari, additional, Melbye, Mads, additional, Feenstra, Bjarke, additional, Freathy, Rachel M., additional, Davey-Smith, George, additional, Lawlor, Deborah A., additional, Murray, Jeffrey C., additional, Williams, Scott M., additional, Jacobsson, Bo, additional, Muglia, Louis J., additional, and Zhang, Ge, additional

Published
2019
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208. The Plight of Muntaser Ibrahim

Author

Sirugo, Giorgio, primary, Williams, Scott M., additional, Tishkoff, Sarah A., additional, Cordell, Heather J., additional, Marchini, Jonathan, additional, Barsh, Gregory S., additional, and Copenhaver, Gregory P., additional

Published
2019
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210. Candidate gene analysis of spontaneous preterm delivery: New insights from re-analysis of a case-control study using case-parent triads and control-mother dyads

Author

Myking Solveig, Myhre Ronny, Gjessing Håkon K, Morken Nils-Halvdan, Sengpiel Verena, Williams Scott M, Ryckman Kelli K, Magnus Per, and Jacobsson Bo

Subjects
case-parent triad analysis, hybrid design, haplotype, pathway analysis, COL5A2, COL5A1, Internal medicine, RC31-1245, Genetics, QH426-470
Abstract

Abstract Background Spontaneous preterm delivery (PTD) has a multifactorial etiology with evidence of a genetic contribution to its pathogenesis. A number of candidate gene case-control studies have been performed on spontaneous PTD, but the results have been inconsistent, and do not fully assess the role of how two genotypes can impact outcome. To elucidate this latter point we re-analyzed data from a previously published case-control candidate gene study, using a case-parent triad design and a hybrid design combining case-parent triads and control-mother dyads. These methods offer a robust approach to genetic association studies for PTD compared to traditional case-control designs. Methods The study participants were obtained from the Norwegian Mother and Child Cohort Study (MoBa). A total of 196 case triads and 211 control dyads were selected for the analysis. A case-parent triad design as well as a hybrid design was used to analyze 1,326 SNPs from 159 candidate genes. We compared our results to those from a previous case-control study on the same samples. Haplotypes were analyzed using a sliding window of three SNPs and a pathway analysis was performed to gain biological insight into the pathophysiology of preterm delivery. Results The most consistent significant fetal gene across all analyses was COL5A2. The functionally similar COL5A1 was significant when combining fetal and maternal genotypes. PON1 was significant with analytical approaches for single locus association of fetal genes alone, but was possibly confounded by maternal effects. Focal adhesion (hsa04510), Cell Communication (hsa01430) and ECM receptor interaction (hsa04512) were the most constant significant pathways. Conclusion This study suggests a fetal association of COL5A2 and a combined fetal-maternal association of COL5A1 with spontaneous PTD. In addition, the pathway analysis implied interactions of genes affecting cell communication and extracellular matrix.

Published
2011
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212. Variable number of tandem repeat polymorphisms of the interleukin-1 receptor antagonist gene IL-1RN: a novel association with the athlete status

Author

Ryckman Kelli K, Di Santolo Manuela, Cauci Sabina, Williams Scott M, and Banfi Giuseppe

Subjects
Internal medicine, RC31-1245, Genetics, QH426-470
Abstract

Abstract Background The interleukin-1 (IL-1) family of cytokines is involved in the inflammatory and repair reactions of skeletal muscle during and after exercise. Specifically, plasma levels of the IL-1 receptor antagonist (IL-1ra) increase dramatically after intense exercise, and accumulating evidence points to an effect of genetic polymorphisms on athletic phenotypes. Therefore, the IL-1 family cytokine genes are plausible candidate genes for athleticism. We explored whether IL-1 polymorphisms are associated with athlete status in European subjects. Methods Genomic DNA was obtained from 205 (53 professional and 152 competitive non-professional) Italian athletes and 458 non-athlete controls. Two diallelic polymorphisms in the IL-1β gene (IL-1B) at -511 and +3954 positions, and a variable number tandem repeats (VNTR) in intron 2 of the IL-1ra gene (IL-1RN) were assessed. Results We found a 2-fold higher frequency of the IL-1RN 1/2 genotype in athletes compared to non-athlete controls (OR = 1.93, 95% CI = 1.37-2.74, 41.0% vs. 26.4%), and a lower frequency of the 1/1 genotype (OR = 0.55, 95% CI = 0.40-0.77, 43.9% vs. 58.5%). Frequency of the IL-1RN 2/2 genotype did not differ between groups. No significant differences between athletes and controls were found for either -511 or +3954 IL-1B polymorphisms. However, the haplotype (-511)C-(+3954)T-(VNTR)2 was 3-fold more frequent in athletes than in non-athletes (OR = 3.02, 95% CI = 1.16-7.87). Interestingly, the IL-1RN 1/2 genotype was more frequent in professional than in non-professional athletes (OR = 1.92, 95% CI = 1.02-3.61, 52.8% vs. 36.8%). Conclusions Our study found that variants at the IL-1ra gene associate with athletic status. This confirms the crucial role that cytokine IL-1ra plays in human physical exercise. The VNTR IL-1RN polymorphism may have implications for muscle health, performance, and/or recovery capacities. Further studies are needed to assess these specific issues. As VNTR IL-1RN polymorphism is implicated in several disease conditions, athlete status may constitute a confounding variable that will need to be accounted for when examining associations of this polymorphism with disease risk.

Published
2010
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213. If Racial disparity in pathophysiologic pathways of preterm birth based on genetic variants

Author

Pearce Brad, Menon Ramkumar, Velez Digna R, Merialdi Mario, Williams Scott M, Fortunato Stephen J, and Thorsen Poul

Subjects
Gynecology and obstetrics, RG1-991, Reproduction, QH471-489
Abstract

Abstract Objective To study pathophysiologic pathways in spontaneous preterm birth and possibly the racial disparity associating with maternal and fetal genetic variations, using bioinformatics tools. Methods A large scale candidate gene association study was performed on 1442 SNPs in 130 genes in a case (preterm birth < 36 weeks) control study (term birth > 37 weeks). Both maternal and fetal DNA from Caucasians (172 cases and 198 controls) and 279 African-Americans (82 cases and 197 controls) were used. A single locus association (genotypic) analysis followed by hierarchical clustering was performed, where clustering was based on p values for significant associations within each race. Using Ingenuity Pathway Analysis (IPA) software, known pathophysiologic pathways in both races were determined. Results From all SNPs entered into the analysis, the IPA mapped genes to specific disease functions. Gene variants in Caucasians were implicated in disease functions shared with other known disorders; specifically, dermatopathy, inflammation, and hematological disorders. This may reflect abnormal cervical ripening and decidual hemorrhage. In African-Americans inflammatory pathways were the most prevalent. In Caucasians, maternal gene variants showed the most prominent role in disease functions, whereas in African Americans it was fetal variants. The IPA software was used to generate molecular interaction maps that differed between races and also between maternal and fetal genetic variants. Conclusion Differences at the genetic level revealed distinct disease functions and operational pathways in African Americans and Caucasians in spontaneous preterm birth. Differences in maternal and fetal contributions in pregnancy outcome are also different between African Americans and Caucasians. These results present a set of explicit testable hypotheses regarding genetic associations with preterm birth in African Americans and Caucasians

Published
2009
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214. Helicobacter pyloriinfection causes both protective and deleterious effects in human health and disease

Author

Miller, Anna K. and Williams, Scott M.

Abstract

Infection with Helicobacter pylori (H. pylori)is necessary but not sufficient for the development of gastric cancer, the third leading cause of cancer death globally. H. pyloriinfection affects over half of people globally; however, it does not affect populations uniformly. H. pyloriinfection rates are declining in western industrialized countries but are plateauing in developing and newly industrialized countries where gastric cancer is most prevalent. Despite H. pyloriinfection being the primary causative agent for gastric cancer, H. pyloriinfection can also cause other effects, detrimental or beneficial, throughout an individual’s life, with the beneficial effects often being seen in childhood and the deleterious effects in adulthood. H. pyloriis an ancient bacterium and its likelihood of affecting disease or health is dependent on both human and bacterial genetics that have co-evolved over millennia. In this review, we focus on the impact of infection and its genetic bases in different populations and diseases throughout an individual’s lifespan, highlighting the benefits of individualized treatment and argue that universal eradication of H. pyloriin its host may cause more harm than good for those infected with H. pylori.

Published
2021
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216. Genetic effects on the correlation structure of cardiovascular disease risk factors in a Ghanaian population

Author

Kodaman, Nuri, Sobota, Rafal, Asselbergs, Folkert W., Moore, Jason H., Brown, Nancy J., Aldrich, Melinda C., and Williams, Scott M.

Subjects
Genotype, Cardiovascular Diseases, Risk Factors, Plasminogen Activator Inhibitor 1, Humans, Exome, DNA, Morbidity, Ghana, Polymorphism, Single Nucleotide, Risk Assessment, Article
Abstract

Plasma concentration of plasminogen activator inhibitor-1 (PAI-1) is highly correlated with several cardiovascular disease (CVD) risk factors. It also plays a direct role in cardiovascular diseases, including myocardial infarction and stroke, by impeding the dissolution of thrombi in the blood. Insofar as PAI-1 “links” the risk factors of CVD to its endpoints, genetic variants modulating the relationship between PAI-1 and risk factors may be of particular clinical and biological interest. The high heritability of PAI-1, which has not been explained by genetic association studies, may also, in large part, be due to this relationship with CVD risk factors. Using exome-wide data from 1032 Ghanaian study participants, we tested for heterogeneity of correlation by genotype between PAI-1 and four CVD risk factors (BMI, triglycerides, mean arterial pressure, and fasting glucose), under the hypothesis that loci involved in the relationship between PAI-1 and other risk factors will also modify their correlational structure. We found more significant heterogeneities of correlation by genotype than we found marginal effects, with no evidence of Type I inflation. The most significant result among all univariate and multivariate tests performed in this study was the heterogeneity of correlation between PAI-1 and mean arterial pressure at rs10738554, near SLC24A2, a gene previously associated with high blood pressure in African Americans.

Published
2017

217. The Cytochrome P450 Slow Metabolizers CYP2C9*2 and CYP2C9*3 Directly Regulate Tumorigenesis via Reduced Epoxyeicosatrienoic Acid Production

Author

Sausville, Lindsay N., primary, Gangadhariah, Mahesha H., additional, Chiusa, Manuel, additional, Mei, Shaojun, additional, Wei, Shouzuo, additional, Zent, Roy, additional, Luther, James M., additional, Shuey, Megan M., additional, Capdevila, Jorge H., additional, Falck, John R., additional, Guengerich, F. Peter, additional, Williams, Scott M., additional, and Pozzi, Ambra, additional

Published
2018
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219. Novel thoughts on preterm birth research proceedings of the 13th annual preterm birth international collaborative (PREBIC) meeting

Author

Gyamfi-Bannerman, Cynthia, primary, Menon, Ramkumar, additional, Bonney, Elizabeth A., additional, Dolan, Siobhan M., additional, Johnson, Mark, additional, Lamont, Ronald F., additional, Mesiano, Sam, additional, Murtha, Amy P., additional, Myatt, Leslie, additional, Mysorekar, Indira, additional, Williams, Scott M, additional, Zhong, Nanbert, additional, and Helmer, Hanns, additional

Published
2017
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220. HS3ST1 genotype regulates antithrombin's inflammomodulatory tone and associates with atherosclerosis

Author

Smits, Nicole C., primary, Kobayashi, Takashi, additional, Srivastava, Pratyaksh K., additional, Skopelja, Sladjana, additional, Ivy, Julianne A., additional, Elwood, Dustin J., additional, Stan, Radu V., additional, Tsongalis, Gregory J., additional, Sellke, Frank W., additional, Gross, Peter L., additional, Cole, Michael D., additional, DeVries, James T., additional, Kaplan, Aaron V., additional, Robb, John F., additional, Williams, Scott M., additional, and Shworak, Nicholas W., additional

Published
2017
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222. Ornithine decarboxylase (ODC1) gene variant (rs2302615) is associated with gastric cancer independently of Helicobacter pyloriCagA serostatus

Author

Miller, Anna K., Tavera, Gloria, Dominguez, Ricardo L., Camargo, M. Constanza, Waterboer, Tim, Wilson, Keith T., Williams, Scott M., and Morgan, Douglas R.

Abstract

The primary cause of gastric cancer is chronic infection with Helicobacter pylori (H. pylori), particularly the high-risk genotype cagA, and risk modification by human genetic variants. We studied 94 variants in 54 genes for association with gastric cancer, including rs2302615 in ornithine decarboxylase (ODC1), which may affect response to chemoprevention with the ODC inhibitor, eflornithine (difluoromethylornithine; DFMO). Our population-based, case-control study included 1366 individuals (664 gastric cancer cases and 702 controls) from Western Honduras, a high incidence region of Latin America. CagA seropositivity was strongly associated with cancer (OR = 3.6; 95% CI: 2.6, 5.1). The ODC1variant rs2302615 was associated with gastric cancer (OR = 1.36; p= 0.018) in a model adjusted for age, sex, and CagA serostatus. Two additional single nucleotide polymorphisms (SNPs) in CASP1(rs530537) and TLR4(rs1927914) genes were also associated with gastric cancer in univariate models as well as models adjusted for age, sex, and CagA serostatus. The ODC1SNP association with gastric cancer was stronger in individuals who carried the TT genotype at the associating TLR4polymorphism, rs1927914 (OR = 1.77; p= 1.85 × 10−3). In conclusion, the ODC1variant, rs2302615, is associated with gastric cancer and supports chemoprevention trials with DFMO, particularly in individuals homozygous for the T allele at rs1927914.

Published
2021
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227. A chromosome 5q31.1 locus associates with tuberculin skin test reactivity in HIV-positive individuals from tuberculosis hyper-endemic regions in east Africa

Author

Sobota, Rafal S., primary, Stein, Catherine M., additional, Kodaman, Nuri, additional, Maro, Isaac, additional, Wieland-Alter, Wendy, additional, Igo, Robert P., additional, Magohe, Albert, additional, Malone, LaShaunda L., additional, Chervenak, Keith, additional, Hall, Noemi B., additional, Matee, Mecky, additional, Mayanja-Kizza, Harriet, additional, Joloba, Moses, additional, Moore, Jason H., additional, Scott, William K., additional, Lahey, Timothy, additional, Boom, W. Henry, additional, von Reyn, C. Fordham, additional, Williams, Scott M., additional, and Sirugo, Giorgio, additional

Published
2017
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230. Draft Genome Sequences of 13 Colombian Helicobacter pylori Strains Isolated from Pacific Coast and Andean Residents

Author

Pazos, Alvaro, primary, Kodaman, Nuri, additional, Piazuelo, M. Blanca, additional, Romero-Gallo, Judith, additional, Sobota, Rafal S., additional, Israel, Dawn A., additional, Bravo, Luis E., additional, Morgan, Douglas R., additional, Wilson, Keith T., additional, Correa, Pelayo, additional, Peek, Richard M., additional, Williams, Scott M., additional, and Schneider, Barbara G., additional

Published
2017
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235. Haplotype Diversity and Linkage Disequilibrium at Human G6PD: Recent Origin of Alleles That Confer Malarial Resistance

Author

Tishkoff, Sarah A., Varkonyi, Robert, Cahinhinan, Nelie, Abbes, Salem, Argyropoulos, George, Destro-Bisol, Giovanni, Drousiotou, Anthi, Dangerfield, Bruce, Lefranc, Gerard, Loiselet, Jacques, Piro, Anna, Stoneking, Mark, Tagarelli, Antonio, Tagarelli, Giuseppe, Touma, Elias H., Williams, Scott M., and Clark, Andrew G.

Subjects
Glucose-6-phosphate dehydrogenase deficiency -- Research, Malaria -- Research, Allelopathic agents -- Research, Science and technology
Abstract

The frequencies of low-activity alleles of glucose-6-phosphate dehydrogenase in humans are highly correlated with the prevalence of malaria. These 'deficiency' alleles are thought to provide reduced risk from infection by the Plasmodium parasite and are maintained at high frequency despite the hemopathologies that they cause. Haplotype analysis of 'A-' and 'Med' mutations at this locus indicates that they have evolved independently and have increased in frequency at a rate that is too rapid to be explained by random genetic drift. Statistical modeling indicates that the A- allele arose within the past 3840 to 11,760 years and the Med allele arose within the past 1600 to 6640 years. These results support the hypothesis that malaria has had a major impact on humans only since the introduction of agriculture within the past 10,000 years and provide a striking example of the signature of selection on the human genome., Malaria, resulting from infection by the Plasmodium falciparum, P. vivax, P. malariae, or P. ovale parasites, is the leading cause of death in the global human population. Each year 500 [...]

Published
2001

236. Genetic variation in the peroxisome proliferator-activated receptor (PPAR) and peroxisome proliferator-activated receptor gamma co-activator 1 (PGC1) gene families and type 2 diabetes

Author

Villegas, Raquel, Williams, Scott M., Gao, Yu-Tang, Long, Jirong, Shi, Jiajun, Cai, Hui, Li, Honglan, Chen, Ching-Chu, Tai, E. Shyong, Hu, Frank, Cai, Qiuyin, Zheng, Wei, and Shu, Xiao-Ou

Subjects
Adult, Genetic Markers, Male, China, endocrine system diseases, Genotype, Peroxisome Proliferator-Activated Receptors, nutritional and metabolic diseases, RNA-Binding Proteins, Middle Aged, Polymorphism, Single Nucleotide, Article, Body Mass Index, Diabetes Mellitus, Type 2, Gene Frequency, Meta-Analysis as Topic, Case-Control Studies, Humans, Female, Gene-Environment Interaction, Carrier Proteins, Exercise, Aged, Genome-Wide Association Study
Abstract

We used a two-stage study design to evaluate whether variations in the peroxisome proliferator-activated receptors (PPAR) and the PPAR gamma co-activator 1 (PGC1) gene families (PPARA, PPARG, PPARD, PPARGC1A, and PPARGC1B) are associated with type 2 diabetes (T2D) risk. Stage I used data from a genome-wide association study (GWAS) from Shanghai, China (1019 T2D cases and 1709 controls) and from a meta-analysis of data from the Asian Genetic Epidemiology Network for T2D (AGEN-T2D). Criteria for selection of single nucleotide polymorphisms (SNPs) for stage II were: (1) P0.05 in single marker analysis in Shanghai GWAS and P0.05 in the meta-analysis or (2) P10(-3) in the meta-analysis alone and (3) minor allele frequency ≥ 0.10. Nine SNPs from the PGC1 family were assessed in stage II (an independent set of middle-aged men and women from Shanghai with 1700 T2D cases and 1647 controls). One SNP in PPARGC1B, rs251464, was replicated in stage II (OR = 0.87; 95% CI: 0.77-0.99). Gene-body mass index (BMI) and gene-exercise interactions and T2D risk were evaluated in a combined dataset (Shanghai GWAS and stage II data: 2719 cases and 3356 controls). One SNP in PPARGC1A, rs12640088, had a significant interaction with BMI. No interactions between the PPARGC1B gene and BMI or exercise were observed.

Published
2014

237. Spontaneous preterm birth in African Americans is associated with infection and inflammatory response gene variants

Author

Velez, Digna R., Fortunato, Stephen, Thorsen, Poul, Lombardi, Salvatore J., Williams, Scott M., and Menon, Ramkumar

Subjects
African Americans -- Health aspects, African Americans -- Genetic aspects, Genetic research -- Health aspects, Genetic research -- Genetic aspects, Infants (Premature) -- Health aspects, Infants (Premature) -- Genetic aspects, Health
Abstract

To link to full-text access for this article, visit this link: http://dx.doi.org/10.1016/j.ajog.2008.08.051 Byline: Digna R. Velez (a)(b), Stephen Fortunato (c)(d), Poul Thorsen (e), Salvatore J. Lombardi (c), Scott M. Williams (b), Ramkumar Menon (c)(e) Keywords: complex disease; genetic epidemiology; infection/inflammation; reproductive genetics Abstract: The objective of the study was to study the genetic risk factors of spontaneous preterm birth (PTB) in African Americans. Author Affiliation: (a) Dr John T. Macdonald Foundation Department of Human Genetics and Miami Institute of Human Genomics, University of Miami, Miami, FL (b) Center for Human Genetics Research, Vanderbilt University, Nashville, TN (c) Perinatal Research Center, Nashville, TN (d) Department of Obstetrics and Gynecology and Reproductive Science, Yale University Medical School, New Haven, CT (e) Northern Atlantic Epidemiologic Alliance, University of Aarhus, Aarhus, Denmark Article History: Received 15 April 2008; Revised 20 June 2008; Accepted 20 August 2008 Article Note: (footnote) This study was supported in part by grants from Elsass Foundation, Denmark (to R.M.); funds from the Maternal-Fetal Group, LLC, Nashville, TN (to S.F., S.J.L.); and National Institutes of Health Grant T32-GM-080178 (to D.R.V.)., Cite this article as: Velez DR, Fortunato S, Thorsen P, et al. Spontaneous preterm birth in African Americans is associated with infection and inflammatory response gene variants. Am J Obstet Gynecol 2009;200:209.e1-209.e27.

Published
2009

240. The Great Migration and African-American Genomic Diversity

Author

Baharian, Soheil, primary, Barakatt, Maxime, additional, Gignoux, Christopher R., additional, Shringarpure, Suyash, additional, Errington, Jacob, additional, Blot, William J., additional, Bustamante, Carlos D., additional, Kenny, Eimear E., additional, Williams, Scott M., additional, Aldrich, Melinda C., additional, and Gravel, Simon, additional

Published
2016
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241. Epigenetic and genetic variation in GATA5 is associated with gastric disease risk

Author

Sobota, Rafal S., primary, Kodaman, Nuri, additional, Mera, Robertino, additional, Piazuelo, M. Blanca, additional, Bravo, Luis E., additional, Pazos, Alvaro, additional, Zabaleta, Jovanny, additional, Delgado, Alberto G., additional, El-Rifai, Wael, additional, Morgan, Douglas R., additional, Wilson, Keith T., additional, Correa, Pelayo, additional, Williams, Scott M., additional, and Schneider, Barbara G., additional

Published
2016
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244. Expression of complement and toll-like receptor pathway genes is associated with malaria severity in Mali: a pilot case control study

Author

Sobota, Rafal S., primary, Dara, Antoine, additional, Manning, Jessica E., additional, Niangaly, Amadou, additional, Bailey, Jason A., additional, Kone, Abdoulaye K., additional, Thera, Mahamadou A., additional, Djimdé, Abdoulaye A., additional, Vernet, Guy, additional, Leissner, Philippe, additional, Williams, Scott M., additional, Plowe, Christopher V., additional, and Doumbo, Ogobara K., additional

Published
2016
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245. A Locus at 5q33.3 Confers Resistance to Tuberculosis in Highly Susceptible Individuals

Author

Sobota, Rafal S., primary, Stein, Catherine M., additional, Kodaman, Nuri, additional, Scheinfeldt, Laura B., additional, Maro, Isaac, additional, Wieland-Alter, Wendy, additional, Igo, Robert P., additional, Magohe, Albert, additional, Malone, LaShaunda L., additional, Chervenak, Keith, additional, Hall, Noemi B., additional, Modongo, Chawangwa, additional, Zetola, Nicola, additional, Matee, Mecky, additional, Joloba, Moses, additional, Froment, Alain, additional, Nyambo, Thomas B., additional, Moore, Jason H., additional, Scott, William K., additional, Lahey, Timothy, additional, Boom, W. Henry, additional, von Reyn, C. Fordham, additional, Tishkoff, Sarah A., additional, Sirugo, Giorgio, additional, and Williams, Scott M., additional

Published
2016
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247. GEneSTATION 1.0: a synthetic resource of diverse evolutionary and functional genomic data for studying the evolution of pregnancy-associated tissues and phenotypes

Author

Kim, Mara, primary, Cooper, Brian A., additional, Venkat, Rohit, additional, Phillips, Julie B., additional, Eidem, Haley R., additional, Hirbo, Jibril, additional, Nutakki, Sashank, additional, Williams, Scott M., additional, Muglia, Louis J., additional, Capra, J. Anthony, additional, Petren, Kenneth, additional, Abbot, Patrick, additional, Rokas, Antonis, additional, and McGary, Kriston L., additional

Published
2015
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249. The Great Migration and African-American genomic diversity

Author

Baharian, Soheil, primary, Barakatt, Maxime, additional, Gignoux, Christopher R, additional, Shringarpure, Suyash, additional, Errington, Jacob, additional, Blot, William J, additional, Bustamante, Carlos D, additional, Kenny, Eimear E, additional, Williams, Scott M, additional, Aldrich, Melinda C, additional, and Gravel, Simon, additional

Published
2015
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