11,981 results on '"Guo G"'
Search Results
202. A long noncoding RNA critically regulates Bcr-Abl-mediated cellular transformation by acting as a competitive endogenous RNA
- Author
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Guo, G, Kang, Q, Zhu, X, Chen, Q, Wang, X, Chen, Y, Ouyang, J, Zhang, L, Tan, H, Chen, R, Huang, S, and Chen, J-L
- Published
- 2015
- Full Text
- View/download PDF
203. High frequency variations of Helicobacter pylori isolates in individual hosts in a Chinese population
- Author
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Ren, L., Liao, Y.L., Song, Y., Guo, Y., Mao, X.H., Xie, Q.H., Zhang, W.J., Guo, G., and Zou, Q.M.
- Published
- 2012
- Full Text
- View/download PDF
204. Design and Experiment of 1 THz Slow Wave Structure Fabricated by Nano-CNC Technology
- Author
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Yang, R., primary, Xu, J., additional, Yue, L., additional, Yin, P., additional, Luo, J., additional, Yin, H., additional, Zhao, G., additional, Guo, G., additional, Yu, S., additional, Niu, X., additional, Hu, M., additional, Liu, D., additional, Wang, W., additional, Liu, W., additional, Li, D., additional, and Wei, Y., additional
- Published
- 2022
- Full Text
- View/download PDF
205. AN INDOOR POSITIONING SYSTEM BASED ON COMBINED AUDIO CHIRP/MEMS/FLOOR MAP: PERFORMANCE ANALYSIS OF KEPLER A100
- Author
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Huang, L., primary, Chen, R., additional, Ye, F., additional, Liu, Z., additional, Li, Z., additional, Xu, S., additional, Guo, G., additional, and Qian, L., additional
- Published
- 2022
- Full Text
- View/download PDF
206. Genetic analyses of blood β-hydroxybutyrate predicted from milk infrared spectra and its association with longevity and female reproductive traits in Holstein cattle
- Author
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Lou, W., primary, Zhang, H., additional, Luo, H., additional, Chen, Z., additional, Shi, R., additional, Guo, X., additional, Zou, Y., additional, Liu, L., additional, Brito, L.F., additional, Guo, G., additional, and Wang, Y., additional
- Published
- 2022
- Full Text
- View/download PDF
207. Influence of fibrolytic enzymes mixture on performance, nutrient digestion, rumen fermentation and microbiota in Holstein bulls
- Author
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Zhang, J., primary, Wang, C., additional, Liu, Q., additional, Guo, G., additional, Huo, W., additional, Pei, C., additional, and Jiang, Q., additional
- Published
- 2022
- Full Text
- View/download PDF
208. Antimicrobial peptide AMP-17 induces protection against systemic candidiasis and interacts synergistically with fluconazole against Candida albicans biofilm
- Author
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Chaoqin Sun, Lijuan Zhu, Longbing Yang, Zhuqing Tian, Zhenlong Jiao, Mingjiao Huang, Jian Peng, and Guo Guo
- Subjects
Candida albicans ,antimicrobial peptide ,AMP-17 ,systemic candidiasis ,synergistic effect ,biofilm ,Microbiology ,QR1-502 - Abstract
Candida albicans, a common commensal and opportunistic fungal pathogen in humans, can occasionally progress to disseminated candidiasis which is a serious condition with a high morbidity and fatality rate. The emergence of drug-resistant fungal strains compels us to look for an efficient treatment solution. Our earlier studies have demonstrated that the unique antimicrobial peptide AMP-17 from Musca domestica has a strong antifungal impact on C. albicans in vitro. Here, we verified the therapeutic effects of AMP-17 on systemic candidiasis in vivo and the peptide interacts with fluconazole, a common antifungal medication, to treat systemic candidiasis. In the disseminated candidiasis model of Galleria mellonella and mice challenged with C. albicans, AMP-17 increased the survival rates of infected larvae and mice to 66.7 and 75%, respectively. Furthermore, the peptide lowered the load of C. albicans in the infected larvae and the kidneys of the mice by nearly 90%. Additional histological examination and measurements of plasma cytokines showed that the injection of AMP-17 markedly reduced the inflammatory response and balanced cytokine expression. Furthermore, checkerboard micro dilution experiments demonstrated that AMP-17 and fluconazole worked in synergy to inhibit C. albicans in the biofilm mode. According to morphological studies, AMP-17 and fluconazole together decreased the production of hyphae throughout the C. albicans biofilm formation process, loosening the mature biofilms’ structure and lowering the amount of carbohydrates in the extracellular matrix (ECM) of the biofilms. Taken together, these results showed that AMP-17 would be a viable treatment for systemic candidiasis and might be a different approach to combating Candida biofilm, either by itself or in conjunction with fluconazole.
- Published
- 2024
- Full Text
- View/download PDF
209. Research on the injection characteristics of biogas engine
- Author
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Fang, Z, primary, Wang, K, additional, Sun, Y, additional, and Guo, G, additional
- Published
- 2016
- Full Text
- View/download PDF
210. Global age-sex-specific fertility, mortality, healthy life expectancy (HALE), and population estimates in 204 countries and territories, 1950–2019: a comprehensive demographic analysis for the Global Burden of Disease Study 2019
- Author
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Abbafati, C., Machado, D.B., Cislaghi, B., Salman, O.M., Karanikolos, M., McKee, M., Abbas, K.M., Brady, O.J., Larson, H.J., Trias-Llimós, S., Cummins, S., Langan, S.M., Sartorius, B., Hafiz, A., Jenabi, E., Mohammad Gholi Mezerji, N., Borzouei, S., Azarian, G., Khazaei, S., Abbasi, M., Asghari, B., Masoumi, S., Komaki, H., Taherkhani, A., Adabi, M., Abbasifard, M., Bazmandegan, G., Kamiab, Z., Vakilian, A., Anjomshoa, M., Mokari, A., Sabour, S., Shahbaz, M., Saeedi, R., Ahmadieh, H., Yousefinezhadi, T., Haj-Mirzaian, A., Nikbakhsh, R., Safi, S., Asgari, S., Irvani, S.N., Jahanmehr, N., Ramezanzadeh, K., Abbasi-Kangevari, M., Khayamzadeh, M., Abbastabar, H., Shirkoohi, R., Fazlzadeh, M., Janjani, H., Hosseini, M., Mansournia, M., Tohidinik, H., Bakhtiari, A., Fazaeli, A., Mousavi, S., Hasanzadeh, A., Nabavizadeh, B., Malekzadeh, R., Hashemian, M., Pourshams, A., Salimzadeh, H., Sepanlou, S.G., Afarideh, M., Esteghamati, A., Esteghamati, S., Ghajar, A., Heidari, B., Rezaei, N., Mohamadi, E., Rahimi-Movaghar, A., Rahim, F., Eskandarieh, S., Sahraian, M., Mohebi, F., Aminorroaya, A., Ebrahimi, H., Farzadfar, F., Mohajer, B., Pishgar, F., Saeedi Moghaddam, S., Shabani, M., Zarafshan, H., Abolhassani, H., Hafezi-Nejad, N., Heidari-Soureshjani, R., Abdollahi, M., Farahmand, M., Salamati, P., Mehrabi Nasab, E., Tajdini, M., Aghamir, S., Mirzaei, R., Dibaji Forooshani, Z., Khater, M.M., Abd-Allah, F., Abdelalim, A., Abualhasan, A., El-Jaafary, S.I., Hassan, A., Elsharkawy, A., Khater, A.M., Elhabashy, H.R., Salem, M.R.R., Salem, H., Sadeghi, M., Jafarinia, M., Amini-Rarani, M., Mohammadifard, N., Sarrafzadegan, N., Abdollahpour, I., Sarveazad, A., Tehrani-Banihashemi, A., Yoosefi Lebni, J., Manafi, N., Pazoki Toroudi, H., Dorostkar, F., Alipour, V., Sheikhtaheri, A., Arabloo, J., Azari, S., Ghashghaee, A., Rezapour, A., Naserbakht, M., Kabir, A., Mehri, F., Yousefifard, M., Asadi-Aliabadi, M., Babaee, E., Eshrati, B., Goharinezhad, S., Moradi-Lakeh, M., Abedi, P., Rashedi, V., Kumar, V., Elgendy, I.Y., Basu, S., Park, J., Pereira, A., Norheim, O.F., Eagan, A.W., Cahill, L.E., Sheikh, A., Abushouk, A.I., Kraemer, M.U.G., Thakur, B., Bärnighausen, T.W., Shrime, M.G., Abedi, A., Doshi, C.P., Abegaz, K.H., Geberemariyam, B.S., Aynalem, Y.A., Shiferaw, W.S., Abosetugn, A.E., Aboyans, V., Abrams, E.M., Gitimoghaddam, M., Kissoon, N., Stubbs, J.L., Brauer, M., Iyamu, I.O., Kopec, J.A., Pourmalek, F., Ribeiro, A.P., Malta, D.C., Gomez, R.S., Abreu, L.G., Abrigo, M.R.M., Almulhim, A.M., Dahlawi, S.M.A., Pottoo, F.H., Menezes, R.G., Alanzi, T.M., Alumran, A.K., Abu Haimed, A.K., Madadin, M., Alanezi, F.M., Abu-Gharbieh, E., Saddik, B., Abu-Raddad, L.J., Samy, A.M., El Nahas, N., Shalash, A.S., Nabhan, A.F., Kamath, A.M., Kassebaum, N.J., Aravkin, A.Y., Kochhar, S., Sorensen, R.J.D., Afshin, A., Burkart, K., Cromwell, E.A., Dandona, L., Dharmaratne, S.D., Gakidou, E., Hay, S.I., Kyu, H.H., Lopez, A.D., Lozano, R., Misganaw, A.T., Mokdad, A.H., Naghavi, M., Pigott, D.M., Reiner Jr, R.C., Roth, G.A., Stanaway, J.D., Vollset, S., Vos, T., Wang, H., Lim, S.S., Murray, C.J.L., Kalani, R., Ikuta, K.S., Cho, D.Y., Kneib, C.J., Crowe, C.S., Massenburg, B.B., Morrison, S.D., Acebedo, A., Adelson, J.D., Agesa, K.M., Alam, T., Albertson, S.B., Anderson, J.A., Antony, C.M., Ashbaugh, C., Assmus, M., Azhar, G., Balassyano, S., Bannick, M.S., Barthelemy, C.M., Bender, R.G., Bennitt, F.B., Bertolacci, G.J., Biehl, M.H., Bisignano, C., Boon-Dooley, A.S., Briant, P.S., Bryazka, D., Bumgarner, B.R., Callender, C.S., Cao, J., Castle, C.D., Castro, E., Causey, K., Cercy, K.M., Chalek, J., Charlson, F.J., Cohen, A.J., Comfort, H., Compton, K., Croneberger, A.J., Cruz, J.A., Cunningham, M., Dandona, R., Dangel, W.J., Dean, F.E., DeCleene, N.K., Deen, A., Degenhardt, L., Dingels, Z.V., Dippenaar, I.N., Dirac, M.A., Dolgert, A.J., Emmons-Bell, S., Estep, K., Farag, T., Feigin, V.L., Feldman, R., Ferrara, G., Ferrari, A.J., Fitzgerald, R., Force, L.M., Fox, J.T., Frank, T.D., Fu, W., Fukutaki, K., Fuller, J.E., Fullman, N., Galles, N.C., Gardner, W.M., Gershberg Hayoon, A., Goren, E., Gorman, T.M., Gottlich, H.C., Guo, G., Haddock, B., Hagins, H., Haile, L.M., Hamilton, E.B., Han, C., Han, H., Harvey, J.D., Henny, K., Henrikson, H.J., Henry, N.J., Herbert, M.E., Hsiao, T., Huynh, C.K., Iannucci, V.C., Ippolito, H., Irvine, C.M.S., Jafari, H., Jahagirdar, D., James, S.L., Johnson, C.O., Johnson, S.C., Keller, C., Kemmer, L., Kendrick, P.J., Knight, M., Kocarnik, J.M., Krohn, K.J., Larson, S.L., Lau, K.M., Ledesma, J.R., Leever, A.T., LeGrand, K.E., Lescinsky, H., Lin, C., Liu, H., Liu, Z., Lo, J., Lu, A., Ma, J., Maddison, E.R., Manguerra, H., Marks, A., Martopullo, I., Mastrogiacomo, C.I., May, E.A., Mooney, M.D., Mosser, J.F., Mullany, E.C., Mumford, J., Munro, S.B., Nandakumar, V., Nguyen, J., Nguyen, M., Nichols, E., Nixon, M.R., Odell, C.M., Ong, K.L., Orji, A.U., Ostroff, S.M., Pasovic, M., Paulson, K.R., Pease, S.A., Pennini, A., Pierce, M., Pilz, T.M., Pletcher, M., Rao, P.C., Razo, C., Redford, S.B., Reinig, N., Reitsma, M.B., Rhinehart, P., Robalik, T., Roberts, S., Roberts, N.L.S., Rolfe, S., Sbarra, A.N., Schaeffer, L.E., Shackelford, K.A., Shadid, J., Sharara, F., Shaw, D.H., Sheena, B.S., Simpson, K.E., Smith, A., Spencer, C.N., Spurlock, E.E., Stark, B.A., Steiner, C., Steuben, K.M., Sylte, D.O., Tang, M., Taylor, H.J., Terrason, S., Thomson, A.M., Torre, A.E., Travillian, R., Troeger, C.E., Vongpradith, A., Walters, M.K., Wang, J., Watson, A., Watson, S., Whisnant, J.L., Whiteford, H.A., Wiens, K.E., Wilner, L.B., Wilson, S., Wool, E.E., Wozniak, S.S., Wu, J., Wulf Hanson, S., Wunrow, H., Xu, R., Yadgir, S., Yearwood, J.A., York, H.W., Yuan, C., Zhao, J.T., Zheng, P., Zimsen, S.R.M., Zlavog, B.S., Chang, A.Y., Oren, E., Buchbinder, R., Chin, K.L., Guo, Y., Polkinghorne, K.R., Thrift, A.G., Lee, S.W.H., Ackerman, I.N., Cicuttini, F.M., Li, S., Zaman, S., Suleria, H., Zhang, J., Cowie, B.C., Wijeratne, T., Patton, G.C., Sawyer, S.M., Adair, T., Meretoja, A., Adetokunboh, O.O., Adamu, A.A., Iwu, C.J., Parry, C.D.H., Seedat, S., Ndwandwe, D.E., Mahasha, P.W., Stein, D.J., Nnaji, C.A., Sambala, E.Z., Wiysonge, C.S., Adebayo, O.M., Ilesanmi, O.S., Owolabi, M.O., Adeoye, A.M., Adedeji, I.A., Adekanmbi, V., Ibitoye, S.E., John-Akinola, Y.O., Oluwasanu, M.M., Oghenetega, O.B., Akinyemi, R.O., Zandian, H., Adham, D., Zahirian Moghadam, T., Advani, S.M., Teagle, W.L., Braithwaite, D., Agasthi, P., Saadatagah, S., Afshari, M., Agardh, E.E., Allebeck, P., Danielsson, A., Deuba, K., Carrero, J.J., Mohammad, D.K., Fereshtehnejad, S., Ärnlöv, J., Nowak, C., Cederroth, C.R., Ahmadi, A., Pathak, A., Mills, E.J., Kurmi, O.P., Olagunju, A.T., Agarwal, G., Sathish, T., Aghaali, M., Mohammadbeigi, A., Agrawal, A., Ahmad, T., Ahmadi, K., Maleki, S., Naderi, M., Salahshoor, M.R., Pourmirza Kalhori, R., Almasi, A., Salimi, Y., Siabani, S., Ziapour, A., Barzegar, A., Khazaie, H., Kianipour, N., Amiri, F., Salehi Zahabi, S., Mirzaei, M., Shamsi, M., Najafi, F., Jalali, A., Ghadiri, K., Heydarpour, F., Fattahi, N., Karami Matin, B., Kazemi Karyani, A., Pirsaheb, M., Rajati, F., Sadeghi, E., Safari, Y., Sharafi, K., Soltani, S., Vasseghian, Y., Atafar, Z., Jalilian, F., Mirzaei-Alavijeh, M., Saeidi, S., Soofi, M., Zangeneh, A., Mansouri, B., Ahmadi, M., Khafaie, M.A., Safiri, S., Moghadaszadeh, M., Asghari Jafarabadi, M., Doshmangir, L., Jadidi-Niaragh, F., Ghafourifard, M., Spotin, A., Khodayari, M., Samadi Kafil, H., Kalankesh, L.R., Ahmadpour, E., Yousefi, B., Ansari, F., Hassankhani, H., Karimi, S., Haririan, H., Mereta, S., Ahmed, M.B., Feyissa, G.T., Ciobanu, L.G., Aji, B., Aynalem, G.L., Gebresillassie, B., Tefera, Y.G., Akalu, T.Y., Baraki, A.G., Tesema, G.A., Tessema, Z.T., Tamiru, A.T., Azene, Z.N., Netsere, H.B., Yano, Y., Akinyemiju, T., Wu, C., Zadey, S., Samad, Z., Ji, J.S., Doshi, P.P., John, O., Jha, V., Maulik, P.K., Pesudovs, K., Resnikoff, S., Mitchell, P.B., Sachdev, P.S., Akombi, B., Godinho, M.A., Ivers, R.Q., Peden, A.E., Biswas, R., Boufous, S., Akunna, C.J., Alahdab, F., Hammer, M.S., van Donkelaar, A., Al-Aly, Z., Dellavalle, R.P., Alam, S., Martin, R.V., Alam, N., De Leo, D., Tadakamadla, S.K., Alam, K., Alcalde-Rabanal, J.E., Avila-Burgos, L., Serván-Mori, E., Denova-Gutiérrez, E., Rodríguez-Ramírez, S., Morales, L., Poznańska, A., Wojtyniak, B., Rivera, J.A., Campos-Nonato, I.R., Campuzano Rincon, J., Sánchez-Pimienta, T.G., Mengesha, M.B., Welay, F.T., Alema, N.M., Demsie, D.G., Teame, H., Teklehaimanot, B.F., Alemu, B.W., Gultie, T., Bante, A.B., Yeshitila, Y.G., Geramo, Y.C.D., Glagn, M., Sorrie, M.B., W/hawariat, F.G., Amare, A., Kasa, A., Alemu, Y., Mihretie, K.M., Atnafu, D.D., Demeke, F.M., Melese, A., Bante, S.A., Dessie, G.A., Mengesha, E.W., Nigatu, D., Almadi, M.A.H., Alhabib, K.F., Mohammad, Y., Altirkawi, K.A., Temsah, M., Kugbey, N., Ayanore, M.A., Alhassan, R.K., Ali, M., Ali, S., Alicandro, G., Kalhor, R., Alijanzadeh, M., Alinia, C., Yusefzadeh, H., Didarloo, A., Alizade, H., Nikpoor, A., Aljunid, S.M., Alla, F., Leal, L.F., Almasi-Hashiani, A., Moradzadeh, R., Zamanian, M., Nazari, J., Amini, S., Ghamari, F., Almasri, N.A., Khan, M., Al-Mekhlafi, H.M., Bedi, N., Alonso, J., Ciber, E.S.P., Al-Raddadi, R.M., Zakzuk, J., Alvis-Guzman, N., Alvis-Zakzuk, N.J., Castañeda-Orjuela, C.A., Malagón-Rojas, J.N., Gezae, K., Gesesew, H.A., Muthupandian, S., Gebremeskel, G.G., Berhe, K., Amare, B., Ayza, M.A., Gebremeskel, L.G., Gebreslassie, A.A.A., Bitew, H., Zewdie, K.A., Tela, F.G.G., Gill, T.K., Noubiap, J., Lassi, Z.S., Bhandari, D., Amit, A.L., Antonio, C.T., Faraon, E.A., Lopez, J.F., Atre, S.R., Ballew, S.H., Matsushita, K., Khoja, A.T., Daneshpajouhnejad, P., Ghadimi, M., Shafaat, O., Fanzo, J., Amugsi, D.A., Amul, G.H., Koh, D.S.Q., Venketasubramanian, N., Anbesu, E.W., Mohammed, J.A., Wondmeneh, T.G., Andrei, C., Negoi, R.I., Davitoiu, D.V., Manda, A., Negoi, I., Preotescu, L., Hostiuc, M., Hostiuc, S., Ancuceanu, R., Hasan, M., Leung, J., Anderlini, D., Mamun, A.A., Maravilla, J.C., McGrath, J.J., Lalloo, R., Uddin, R., Erskine, H.E., Knibbs, L.D., Mantilla Herrera, A.M., Santomauro, D.F., Mirica, A., Ausloos, M., Herteliu, C., Oţoiu, A., Pana, A., Andrei, T., Ştefan, S., Androudi, S., Angus, C., Ansari, I., Pourjafar, H., Shams-Beyranvand, M., Ansari-Moghaddam, A., Khammarnia, M., Antonazzo, I., Ferrara, P., Conti, S., Cortesi, P.A., Fornari, C., Lee, P.H., Antriyandarti, E., Yousefi, Z., Rafiei, A., Javidnia, J., Faridnia, R., Anvari, D., Goudarzian, A., Moosazadeh, M., Rezai, M., Daryani, A., Fareed, M., Anwer, R., Appiah, S., Paudel, D., Dichgans, M., Riahi, S., Rajabpour-Sanati, A., Arab-Zozani, M., Arba, A.A.K., Aremu, O., Ariani, F., Aripov, T., Armoon, B., Mahdavi, M.M., Arowosegbe, O.O., Tediosi, F., Aryal, K.K., Mosapour, A., Yaminfirooz, M., Arzani, A., Bijani, A., Jahani, M.A., Mouodi, S., Zamani, M., Asaad, M., Dianatinasab, M., Bahrami, M., Pakshir, K., Asadi-Pooya, A.A., Bayati, M., Shahabi, S., Athari, S., Atout, M.M.W., Atteraya, M.S., Brugha, T., Ausloos, F., Avokpaho, E.F.G., Room, R., Islam, M., Edvardsson, D., Rahman, M., Ayala Quintanilla, B., Gething, P.W., Briggs, A.M., Ayano, G., Hendrie, D., Miller, T.R., Azzopardi, P.S., Hoogar, P., B, D.B., Kulkarni, V., Kumar, N., Mithra, P., Shetty, R.S., Thapar, R., Padubidri, J., Bakkannavar, S.M., Nayak, V.C., Rastogi, P., Shetty, B.K., Bhageerathy, R., Gudi, N., Boloor, A., Holla, R., Rathi, P., Unnikrishnan, B., Janodia, M.D., Lang, J.J., Badawi, A., Orpana, H.M., Bhutta, Z.A., Shield, K.D., Chattu, V., Badiye, A.D., Kapoor, N., Bagherzadeh, M., Rabiee, N., Bagli, E., Baig, A.A., Bairwa, M., Lodha, R., Sagar, R., Rath, G.K., Bhardwaj, P., Charan, J., Kanchan, T., Joshi, A., Pakhare, A.P., Bakhshaei, M., Naghshtabrizi, B., Balachandran, A., Hoek, H.W., Postma, M.J., Geremew, A., Gebrehiwot, A.M., Balakrishnan, S., Desalew, A., Bojia, H.A., Mohammed, A.S., Regassa, L.D., Parmar, P.G.K., Balalla, S., Baldasseroni, A., Stokes, M.A., Ball, K., Islam, S., Maddison, R., Balzi, D., Levi, M., Banach, M., Banerjee, S.K., Banik, P.C., Barua, L., Faruque, M., Barboza, M.A., Barker-Collo, S.L., Jonas, J.B., Panda-Jonas, S., De Neve, J., Kohler, S., Moazen, B., Mohammed, S., Barrero, L.H., Basaleem, H., Bassat, Q., Haro, J.M., Koyanagi, A., Car, J., Greaves, F., Majeed, A., Davis, A.C., Steiner, T.J., Kusuma, D., Palladino, R., Rawaf, S., Saxena, S., Rawaf, D.L., Baune, B.T., Karch, A., Baye, B.A., Darega Gela, J., Kolola, T., Becker, J.S., DeLang, M., West, J., Gad, M.M., Serre, M.L., Gallus, S., Lugo, A., Beghi, E., Pupillo, E., Bosetti, C., Giussani, G., Bikbov, B., Perico, N., Remuzzi, G., Imani-Nasab, M., Nouraei Motlagh, S., Sharafi, Z., Behzadifar, M., Béjot, Y., Bekuma, T.T., Yilma, M.T., Bell, M.L., Bello, A.K., Rafiee, A., Keddie, S.H., Lucas, T.C.D., Dolecek, C., Dunachie, S.J., Rumisha, S.F., Weiss, D.J., Lewington, S., Collins, E.L., Nandi, A.K., Zhao, Y., Bennett, D.A., Karim, M.A., Lacey, B., Khundkar, R., Yaya, S., Goulart, A.C., Santos, I.S., Bensenor, I.M., Lotufo, P.A., Tovani-Palone, M.R., Castaldelli-Maia, J., Wang, Y., Furtado, J.M., Benziger, C.P., Berman, A.E., Mazidi, M., Bernabe, E., Dargan, P.I., Molokhia, M., Shibuya, K., Douiri, A., Wolfe, C.D.A., Hay, R.J., Flohr, C., Suchdev, P.S., Ram, P., Bernstein, R.S., Liu, Y., Bhagavathula, A.S., Khan, G., Grivna, M., Bhala, N., Chandan, J.S., Gaidhane, A.M., Quazi Syed, Z., Saxena, D., Khatib, M., Bhat, A.G., Bhattacharyya, K., Bhattarai, S., Das, J.K., Bibi, S., Bilano, V., Bin Sayeed, M., Cherbuin, N., D'Amico, E., Grosso, G., Borzì, A.M., Biondi, A., Vacante, M., Valli, A., Birihane, B.M., Bisanzio, D., Hassan, S., Bjørge, T., Øverland, S., Bockarie, M.J., Mensah, G.A., Sliwa, K., Gholamian, A., Bohlouli, S., Esmaeilnejad, S., Bohluli, M., Bolla, S.R., Borges, G., Bose, D., Bourne, R., Brayne, C., Breitborde, N.J.K., Fisher, J.L., Breitner, S., Brenner, H., Breusov, A.V., Rakovac, I., Briko, 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Babale, Suliankatchi Abdulkader, Rizwan, Sykes, Bryan L, Tabarés-Seisdedos, Rafael, Tabb, Karen M, Tadakamadla, Santosh Kumar, Taherkhani, Amir, Tang, Muming, Taveira, Nuno, Taylor, Heather Jean, Teagle, Whitney L, Tehrani-Banihashemi, Arash, Teklehaimanot, Berhane Fseha, Tessema, Zemenu Tadesse, Thankappan, Kavumpurathu Raman, Thomas, Nihal, Thrift, Amanda G, Titova, Mariya Vladimirovna, Tohidinik, Hamid Reza, Tonelli, Marcello, Topor-Madry, Roman, Topouzis, Foti, Tovani-Palone, Marcos Roberto Roberto, Traini, Eugenio, Tran, Bach Xuan, Travillian, Ravensara, Trias-Llimós, Sergi, Truelsen, Thomas Clement, Tudor Car, Lorainne, Unnikrishnan, Bhaskaran, Upadhyay, Era, Vacante, Marco, Vakilian, Alireza, Valdez, Pascual R, Valli, Alessandro, Vardavas, Constantine, Vasankari, Tommi Juhani, Vasconcelos, Ana Maria Nogale, Vasseghian, Yasser, Veisani, Yousef, Venketasubramanian, Narayanaswamy, Vidale, Simone, Violante, Francesco S, Vlassov, Vasily, Vollset, Stein Emil, Vos, Theo, Vujcic, Isidora S, Vukovic, Ana, Vukovic, Rade, Waheed, Yasir, Wallin, Mitchell Taylor, Walters, Magdalene K, Wang, Hongbo, Wang, Yuan-Pang, Watson, Stefanie, Wei, Jingkai, Weiss, Jordan, Weldesamuel, Girmay Teklay, Werdecker, Andrea, Westerman, Ronny, Whiteford, Harvey A, Wiangkham, Taweewat, Wiens, Kirsten E, Wijeratne, Tissa, Wiysonge, Charles Shey, Wojtyniak, Bogdan, Wolfe, Charles D A, Wondmieneh, Adam Belay, Wool, Eve E, Wu, Ai-Min, Wu, Junjie, Xu, Gelin, Yamada, Tomohide, Yamagishi, Kazumasa, Yano, Yuichiro, Yaya, Sanni, Yazdi-Feyzabadi, Vahid, Yearwood, Jamal A, Yeheyis, Tomas Y, Yilgwan, Christopher Sabo, Yip, Paul, Yonemoto, Naohiro, Yoon, Seok-Jun, Yoosefi Lebni, Javad, York, Hunter W, Younis, Mustafa Z, Younker, Theodore Patrick, Yousefi, Zabihollah, Yousefinezhadi, Taraneh, Yousuf, Abdilahi Yousuf, Yusefzadeh, Hasan, Zahirian Moghadam, Telma, Zakzuk, Josefina, Zaman, Sojib Bin, Zamani, Mohammad, Zamanian, Maryam, Zandian, Hamed, Zhang, Zhi-Jiang, Zheng, Peng, Zhou, Maigeng, Ziapour, Arash, Murray, Christopher J L, Collaborators, GBD 2019 Demographics, GBD 2019 Demographics Collaborator, Violante FS, Value, Affordability and Sustainability (VALUE), Real World Studies in PharmacoEpidemiology, -Genetics, -Economics and -Therapy (PEGET), Microbes in Health and Disease (MHD), Department of Earth Systems Analysis, Biomechanical Engineering, TechMed Centre, Biomolecular Nanotechnology, Research Methodology, Measurement and Data Analysis, Materials Science and Technology of Polymers, Inorganic Materials Science, MESA+ Institute, Biomedical and Environmental Sensorsystems, Mesoscale Chemical Systems, Faculty of Engineering Technology, Nonlinear Solid Mechanics, Digital Society Institute, Pervasive Systems, Nanobiophysics, Faculty of Geo-Information Science and Earth Observation, Multi Scale Mechanics, Engineering Fluid Dynamics, Physics of Complex Fluids, Developmental BioEngineering, Physics of Fluids, Elastomer Technology and Engineering, Medical Cell Biophysics, European Membrane Institute, Department of Earth Observation Science, Biomaterials Science and Technology, Sustainable Process Technology, Department of Urban and Regional Planning and Geo-Information Management, Department of Natural Resources, XUV Optics, Databases (Former), Department of Water Resources, Psychology, Health & Technology, Membrane Science & Technology, Department of Public Health, Clinicum, HUS Neurocenter, HUS Comprehensive Cancer Center, and Environmental Sciences
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Male ,very elderly ,Demographic transition ,HALE ,030204 cardiovascular system & hematology ,preschool child ,Global Burden of Disease ,Carga Global de Enfermedades ,0302 clinical medicine ,newborn ,Risk Factors ,Surveys and Questionnaires ,and Risk Factors Study ,80 and over ,030212 general & internal medicine ,Birth Rate ,Child ,Migration ,11 Medical and Health Sciences ,Aged, 80 and over ,education.field_of_study ,Injuries ,Geography ,Mortality rate ,1. No poverty ,DEATH ,Censuses ,General Medicine ,SDG 10 - Reduced Inequalities ,Middle Aged ,Demographic analysis ,3142 Public health care science, environmental and occupational health ,3. Good health ,demographic analysis ,Estilo de Vida Saludable ,risk factor ,Child, Preschool ,Demography/statistics & numerical data ,Global Burden of Diseases, Injuries, Risk Factors, Fertility, Mortality, Migration, Population ,epidemiology ,Female ,A990 Medicine and Dentistry not elsewhere classified ,CHILD-MORTALITY ,Live Birth ,Global Health Metrics ,TRANSITION ,demographics ,GBD ,fertility ,mortality ,hale ,Adult ,Adolescent ,Total fertility rate ,Population ,Global Burden of Diseases, Injuries, and Risk Factors Study ,Birth rate ,03 medical and health sciences ,Young Adult ,Life Expectancy ,SDG 3 - Good Health and Well-being ,General & Internal Medicine ,SYSTEMATIC ANALYSIS ,Demografía ,Humans ,Global Burden of Disease Study ,human ,Mortality ,education ,Preschool ,Aged ,Demography ,Spatial Analysis ,questionnaire ,Infant, Newborn ,Klinisk medicin ,HIV ,Infant ,Global Burden of Diseases ,sex-specific fertility ,Live Birth/epidemiology ,purl.org/pe-repo/ocde/ford#3.02.00 [https] ,MODEL ,Fertilidad ,Fertility ,Demographic change ,Life expectancy ,NA ,global disease burden ,Clinical Medicine ,population research - Abstract
Background Accurate and up-to-date assessment of demographic metrics is crucial for understanding a wide range of social, economic, and public health issues that affect populations worldwide. The Global Burden of Diseases, Injuries, and Risk Factors Study (GBD) 2019 produced updated and comprehensive demographic assessments of the key indicators of fertility, mortality, migration, and population for 204 countries and territories and selected subnational locations from 1950 to 2019. Methods 8078 country-years of vital registration and sample registration data, 938 surveys, 349 censuses, and 238 other sources were identified and used to estimate age-specific fertility. Spatiotemporal Gaussian process regression (ST-GPR) was used to generate age-specific fertility rates for 5-year age groups between ages 15 and 49 years. With extensions to age groups 10-14 and 50-54 years, the total fertility rate (TFR) was then aggregated using the estimated age-specific fertility between ages 10 and 54 years. 7417 sources were used for under-5 mortality estimation and 7355 for adult mortality. ST-GPR was used to synthesise data sources after correction for known biases. Adult mortality was measured as the probability of death between ages 15 and 60 years based on vital registration, sample registration, and sibling histories, and was also estimated using ST-GPR. HIV-free life tables were then estimated using estimates of under-5 and adult mortality rates using a relational model life table system created for GBD, which closely tracks observed age-specific mortality rates from complete vital registration when available. Independent estimates of HIV-specific mortality generated by an epidemiological analysis of HIV prevalence surveys and antenatal clinic serosurveillance and other sources were incorporated into the estimates in countries with large epidemics. Annual and single-year age estimates of net migration and population for each country and territory were generated using a Bayesian hierarchical cohort component model that analysed estimated age-specific fertility and mortality rates along with 1250 censuses and 747 population registry years. We classified location-years into seven categories on the basis of the natural rate of increase in population (calculated by subtracting the crude death rate from the crude birth rate) and the net migration rate. We computed healthy life expectancy (HALE) using years lived with disability (YLDs) per capita, life tables, and standard demographic methods. Uncertainty was propagated throughout the demographic estimation process, including fertility, mortality, and population, with 1000 draw-level estimates produced for each metric. Findings The global TFR decreased from 2.72 (95% uncertainty interval [UI] 2.66-2.79) in 2000 to 2.31 (2.17-2.46) in 2019. Global annual livebirths increased from 134.5 million (131.5-137.8) in 2000 to a peak of 139.6 million (133.0-146.9) in 2016. Global livebirths then declined to 135.3 million (127.2-144.1) in 2019. Of the 204 countries and territories included in this study, in 2019, 102 had a TFR lower than 2.1, which is considered a good approximation of replacement-level fertility. All countries in sub-Saharan Africa had TFRs above replacement level in 2019 and accounted for 27.1% (95% UI 26.4-27.8) of global livebirths. Global life expectancy at birth increased from 67.2 years (95% UI 66.8-67.6) in 2000 to 73.5 years (72.8-74.3) in 2019. The total number of deaths increased from 50.7 million (49.5-51.9) in 2000 to 56.5 million (53.7-59.2) in 2019. Under-5 deaths declined from 9.6 million (9.1-10.3) in 2000 to 5.0 million (4.3-6.0) in 2019. Global population increased by 25.7%, from 6.2 billion (6.0-6.3) in 2000 to 7.7 billion (7.5-8.0) in 2019. In 2019, 34 countries had negative natural rates of increase; in 17 of these, the population declined because immigration was not sufficient to counteract the negative rate of decline. Globally, HALE increased from 58.6 years (56.1-60.8) in 2000 to 63.5 years (60.8-66.1) in 2019. HALE increased in 202 of 204 countries and territories between 2000 and 2019. Interpretation Over the past 20 years, fertility rates have been dropping steadily and life expectancy has been increasing, with few exceptions. Much of this change follows historical patterns linking social and economic determinants, such as those captured by the GBD Socio-demographic Index, with demographic outcomes. More recently, several countries have experienced a combination of low fertility and stagnating improvement in mortality rates, pushing more populations into the late stages of the demographic transition. Tracking demographic change and the emergence of new patterns will be essential for global health monitoring. Copyright (C) 2020 The Author(s). Published by Elsevier Ltd.
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- 2020
211. Baicalin Inhibits Cell Viability, Migration and Invasion in Breast Cancer by Regulating miR-338-3p and MORC4
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Duan X, Guo G, Pei X, Wang X, Li L, Xiong Y, and Qiu X
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breast cancer ,mir-338-3p ,morc4 ,baicalin ,lcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogens ,lcsh:RC254-282 - Abstract
Xin Duan, Guangcheng Guo, Xinhong Pei, Xinxing Wang, Lin Li, Youyi Xiong, Xinguang Qiu Department of Breast Surgery, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, Henan, People’s Republic of ChinaCorrespondence: Xinguang QiuDepartment of Breast Surgery, The First Affiliated Hospital of Zhengzhou University, No. 1 East Jianshe Road, Zhengzhou, Henan 450000, People’s Republic of ChinaTel +86 371-67967247Email xfcl6782619qiang@163.comBackground: Baicalin is a natural compound from the roots of Scutellaria lateriflora Georgi, which plays anti-cancer role in multiple cancers. However, the exact role and potential underlying mechanism of baicalin in breast cancer (BC) remain poorly understood.Methods: Thirty BC patients were recruited in this study. MCF-10A, MCF-7 and MDA-MB-231 cells were used to investigate the anti-cancer role of baicalin in vitro. Cell viability, migration, invasion and apoptosis were measured by MTT, trans-well and flow cytometry, respectively. The expression levels of microRNA-338-3p (miR-338-3p) and microrchidia family CW-type zinc-finger 4 (MORC4) were measured by quantitative real-time polymerase chain reaction or Western blot. The interaction between miR-338-3p and MORC4 was explored by luciferase reporter assay and RNA immunoprecipitation.Results: We found that Baicalin treatment inhibited cell viability, migration and invasion but promoted apoptosis of BC cells. The expression of miR-338-3p was decreased in BC tissues and cells and miR-338-3p overexpression suppressed cell viability, migration and invasion but induced apoptosis. MiR-338-3p expression was reversed by baicalin exposure and inhibition of miR-338-3p attenuated the role of baicalin in viability, apoptosis, migration and invasion. MORC4 mRNA level was increased in BC tissues and cells, which was decreased by baicalin exposure. MORC4 was a target of miR-338-3p and its overexpression alleviated the effect of miR-338-3p on cell viability, apoptosis, migration and invasion.Conclusion: In conclusion, baicalin suppressed cell viability, migration and invasion but promoted apoptosis in BC cells by regulating miR-338-3p and MORC4, indicating the promising pharmacological value of baicalin in BC treatment.Keywords: breast cancer, baicalin, miR-338-3p, MORC4  
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- 2019
212. Electron Energy Loss Spectroscopy as a Tool to Probe the Electronic Structure in Intermetallic Alloys
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Botton, G. A., Guo, G.-Y., Temmerman, W. M., Humphreys, C. J., Gonis, Antonios, editor, Meike, Annemarie, editor, and Turchi, Patrice E. A., editor
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- 1997
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213. Proteome analysis of wheat leaf under salt stress by two-dimensional difference gel electrophoresis (2D-DIGE)
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Gao, L., Yan, X., Li, X., Guo, G., Hu, Y., Ma, W., and Yan, Y.
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- 2011
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214. Canine hip dysplasia is predictable by genotyping
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Guo, G., Zhou, Z., Wang, Y., Zhao, K., Zhu, L., Lust, G., Hunter, L., Friedenberg, S., Li, J., Zhang, Y., Harris, S., Jones, P., Sandler, J., Krotscheck, U., Todhunter, R., and Zhang, Z.
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- 2011
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215. Atrial enlargement in symptomatic heart block patients with preserved left ventricular function: Possibly related to atrioventricular dyssynchrony
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Lin, Yu-Sheng, Guo, G. Bih-Fang, Chen, Yung-Lung, Tsai, Tzu-Hsien, Pan, Kuo-Li, Liu, Wen-Hao, and Chen, Mien-Cheng
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- 2011
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216. Biomechanics Research on microRNA-223 Expression Activated the Phosphatidylinositol-3-Kinase/Protein Kinase B Pathway by Biotically Methylating the Long Noncoding RNA KCNQ1OT1
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Bian, Xue, primary, Liang, Xiaojie, additional, Zhang, Ying, additional, and Guo, G., additional
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- 2022
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217. Development of a Sabot for Ballistics Testing
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Solis, F., primary, Guo, G., additional, Peel, L., additional, Alam, S., additional, and Ancira, E., additional
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- 2022
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218. Cardiac Fructose Elevation Precedes the Onset of Diastolic Dysfunction in Experimental Models of Type 1 and Type 2 Diabetes
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Annandale, M., primary, Koutsifeli, P., additional, Macindoe, C., additional, Guo, G., additional, Delbridge, L., additional, and Mellor, K., additional
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- 2022
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219. Approximate Data-Domain Hessian in Extended-Source Time-Domain Full Waveform Inversion Using Matching Filter and Conjugate Gradient Method
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Guo, G., primary, Stephane, O., additional, Ali, G., additional, and Hossein, S.A., additional
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- 2022
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220. Magnetic X-Ray Dichroism and Anisotropy Energy of Fe and Co Multilayers
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Guo, G. Y., Ebert, H., Temmerman, W. M., Durham, P. J., Faulkner, J. S., editor, and Jordan, R. G., editor
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- 1994
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221. UV and X-Ray Photoemission from Metals and Alloys
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Durham, P. J., Guo, G. Y., Faulkner, J. S., editor, and Jordan, R. G., editor
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- 1994
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222. Topological Contextuality and Anyonic Statistics of Photonic-Encoded Parafermions
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Liu, Z-H, Sun, K, Pachos, JK, Yang, M, Meng, Y, Liao, Y-W, Li, Q, Wang, J-F, Luo, Z-Y, He, Y-F, Huang, D-Y, Ding, G-R, Xu, J-S, Han, Y-J, Li, C-F, and Guo, G-C
- Abstract
Quasiparticle poisoning, expected to arise during the measurement of the Majorana zero-mode state, poses a fundamental problem for the realization of Majorana-based quantum computation. Parafermions, a natural generalization of Majorana fermions, can encode topological qudits immune to quasiparticle poisoning. While parafermions are expected to emerge in superconducting fractional quantum Hall systems, they are not yet attainable with current technology. To bypass this problem, we employ a photonic quantum simulator to experimentally demonstrate the key components of parafermion-based universal quantum computation. Our contributions in this paper are twofold. First, by manipulating the photonic states, we realize Clifford-operator Berry phases that correspond to braiding statistics of parafermions. Second, we investigate the quantum contextuality in a topological system for the first time by demonstrating the contextuality of parafermion-encoded qudit states. Importantly, we find that the topologically encoded contextuality opens the way to magic state distillation, while both the contextuality and the braiding-induced Clifford gates are resilient against local noise. By introducing contextuality, our photonic quantum simulation provides the first step toward a physically robust methodology for realizing topological quantum computation.
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- 2021
223. Polarization properties of subwavelength hole arrays consisting of rectangular holes
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Ren, X.-F., Zhang, P., Guo, G.-P., Huang, Y.-F., Wang, Z.-W., and Guo, G.-C.
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- 2008
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224. Single-Event Transient Measurements on a DC/DC Pulse Width Modulator Using Heavy Ion, Proton, and Pulsed Laser
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Ren, Y., He, A.-L., Shi, S.-T., Guo, G., Chen, L., Wen, S.-J., Wong, R., van Vonno, N. W., and Bhuva, B. L.
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- 2014
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225. Salt tolerance analysis of CaNAC36 in peppers
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GONG Chengsheng, ZHENG Yuqing, PAN Baogui, GUO Guangjun, and DIAO Weiping
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pepper ,canac36 ,salt tolerance ,gene interaction ,Biology (General) ,QH301-705.5 ,Botany ,QK1-989 - Abstract
Abstract [Objective] Pepper is the largest vegetable crop planted in China. With the land salinization becoming more and more serious, it is urgent to accelerate the functional study of key genes related to pepper salt tolerance. It is of great significance to strengthen the study of mechanisms underlying salt tolerance in peper to promote sustainable development of the pepper industry. [Methods] In the early stage of research, CaNAC36, a NAC transcription factor family gene related to salt stress response in peppers, was discovered. Based on this, the full-length gDNA and cDNA sequences of CaNAC36 were cloned using salttolerant pepper accession PI201224 and salt-sensitive pepper accession PI438643. The expression of CaNAC36 and its interacting genes in different tissues under salt stress was quantitatively analyzed by fluorescence, and the potential relationship between CaNAC36 and its interacting genes was further explored by bioinformatics analysis. [Results] The gDNA and cDNA sequence homology of CaNAC36 in salt-tolerant and salt-sensitive materials were 99.86% and 100%, respectively. CaNAC36 showed an up-expression in roots and stems in the salt-tolerant pepper, and a down-expression in roots and leaves in the salt-sensitive peper. Throgh analyzing the annotation information of 48 genes that may interact with CaNAC36, it was found that 14 genes were belonging to transmembrane proteins, transporter proteins, water porins, chloride channel proteins, detoxifying proteins, and other genes, which may have functional interaction with CaNAC36. Further analysis revealed that PI201224 and PI438643 exhibited significant differences in the expression of five related genes, inclding Capana08g002748, Capana00g004514, Capana09g000275, Capana07g001450, Capana02g001031, at different time points and tissues under salt stress. Meanwhile, the promoter region of CaNAC36 and five associated genes was found to contain a large number of stressrelated cis-acting elements. [Conclusion] CaNAC36 was an important regulatory gene in pepper in response to salt stress, and might interact with other genes to improve salt tolerance.
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- 2024
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226. Risk of key pathogens of Enterobacteriaceae contamination in foods for infants and young children and implications for the revision of standards of China
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CHEN Xiao, YANG Shuran, LIU Jikai, GUO Ge, WANG Yeru, GAN Xin, XU Wenjing, YAN Shaofei, LI Hui, HAN Xiaomin, and ZHAO Jianyun
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foods for infants and young children ,enterobacteriaceae ,cronobacter ,salmonella ,foodborne disease ,risk exposure ,standards ,Food processing and manufacture ,TP368-456 ,Nutrition. Foods and food supply ,TX341-641 - Abstract
The problem of food-borne illnesses due to microbial contamination of infant and young children foods is usually of international concern, and the unique character of the infant and young child population requires special and strict food safety requirements for their own foods. This article reviews the progress of contamination with Salmonella and Cronobacter (Enterobacter sakazakii) of Enterobacteriaceae family in foods for infants and young children and its diseases risk at home and abroad in recent years, and also provides technical support for the revision of national standards for infant and young children foods and its manufacturing processes in China through the comparative analysis of domestic and international microbiological limits for infant and young children foods.
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- 2024
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227. The deubiquitinase BRCC3 increases the stability of ZEB1 and promotes the proliferation and metastasis of triple-negative breast cancer cells
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Huang Qidi, Zheng Shurong, Gu Huayan, Yang Zhi, Lu Yiqiao, Yu Xia, and Guo Guilong
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BRCC3 ,ZEB1 ,epithelial-mesenchymal transition ,triple negative breast cancer ,Biochemistry ,QD415-436 ,Genetics ,QH426-470 - Abstract
Triple negative breast cancer (TNBC) has a high recurrence rate, metastasis rate and mortality rate. The aim of this study is to identify new targets for the treatment of TNBC. Clinical samples are used for screening deubiquitinating enzymes (DUBs). MDA-MB-231 cells and a TNBC mouse model are used for in vitro and in vivo experiments, respectively. Western blot analysis is used to detect the protein expressions of DUBs, zinc finger E-box binding homeobox 1 (ZEB1), and epithelial-mesenchymal transition (EMT)-related markers. Colony formation and transwell assays are used to detect the proliferation, migration and invasion of TNBC cells. Wound healing assay is used to detect the mobility of TNBC cells. Immunoprecipitation assay is used to detect the interaction between breast cancer susceptibility gene 1/2-containing complex subunit 3 (BRCC3) and ZEB1. ZEB1 ubiquitination levels, protein stability, and protein degradation are also examined. Pathological changes in the lung tissues are detected via HE staining. Our results show a significant positive correlation between the expressions of BRCC3 and ZEB1 in clinical TNBC tissues. Interference with BRCC3 inhibits TNBC cell proliferation, migration, invasion and EMT. BRCC3 interacts with ZEB1 and interferes with BRCC3 to inhibit ZEB1 expression by increasing ZEB1 ubiquitination. Interference with BRCC3 inhibits TNBC cell tumorigenesis and lung metastasis in vivo. In all, this study demonstrates that BRCC3 can increase the stability of ZEB1, upregulate ZEB1 expression, and promote the proliferation, migration, invasion, EMT, and metastasis of TNBC cells, providing a new direction for cancer therapy.
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- 2024
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228. Application of apparent resistivity characteristic value method to the delineation of water-rich karst area
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Guan, W, primary, Lei, X, additional, and Guo, G, additional
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- 2015
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229. Certification of Genuine Multipartite Entanglement with General and Robust Device-independent Witnesses
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Zhang, C., Zhang, W., Sekatski, P., Bancal, J., Zwerger, M., Yin, P., Li, G., Peng, X., Chen, L., Han, Y., Xu, J., Huang, Y., Chen, G., Li, C., and Guo, G.
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Genuine multipartite entanglement represents the strongest type of entanglement, which is an essential resource for quantum information processing. Standard methods to detect genuine multipartite entanglement, e.g., entanglement witnesses, state tomography, or quantum state verification, require full knowledge of the Hilbert space dimension and precise calibration of measurement devices, which are usually difficult to acquire in an experiment. The most radical way to overcome these problems is to detect entanglement solely based on the Bell-like correlations of measurement outcomes collected in the experiment, namely, device-independently (DI). However, it is difficult to certify genuine entanglement of practical multipartite states in this way, and even more difficult to quantify it, due to the difficulty to identify optimal multipartite Bell inequalities and protocols tolerant to state impurity. In this work, we explore a general and robust DI method which can be applied to various realistic multipartite quantum state in arbitrary finite dimension, while merely relying on bipartite Bell inequalities. Our method allows us both to certify the presence of genuine multipartite entanglement and to quantify it. Several important classes of entangled states are tested with this method, leading to the detection of genuinely entangled states. We also certify genuine multipartite entanglement in weakly-entangled GHZ states, thus showing that the method applies equally well to less standard states.
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- 2021
230. Effects of Annealing Treatment of Superconductivity in Powder Sintered YBCO
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Tang, X. D., Wang, J. R., Xiong, S. G., Xu, J. R., Guo, G. R., Zhou, L., Reed, R. P., editor, and Fickett, F. R., editor
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- 1990
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231. Computational Methods in Electronic Structure Calculations of Complex Solids
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Temmerman, W. M., Szotek, Z., Winter, H., Guo, G. Y., Evans, R. G., editor, and Wilson, S., editor
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- 1990
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232. Silencing IKBKE inhibits the migration and invasion of glioblastoma by promoting Snail1 degradation
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Liu, Y., primary, Guo, G., additional, Lu, Y., additional, Chen, X., additional, Zhu, L., additional, Zhao, L., additional, Li, C., additional, Zhang, Z., additional, Jin, X., additional, Dong, J., additional, Yang, X., additional, and Huang, Q., additional
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- 2021
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233. Utility of Long-Term Follow-Up to Determine Safety in Radiotherapy-Specific Trials for Localized Prostate Cancer: Meta-Analysis of 29 Randomized Trials
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Jaworski, E., primary, Fang, F., additional, Gharzai, L.A., additional, McFarlane, M., additional, Solanki, A.A., additional, Zaorsky, N.G., additional, Mahal, B.A., additional, Feng, F.Y., additional, Ponsky, L., additional, Garcia, J., additional, Fredman, E.T., additional, Guo, G., additional, Berlin, A., additional, Roy, S., additional, Jackson, W.C., additional, Dess, R.T., additional, Schipper, M., additional, and Spratt, D.E., additional
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- 2021
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234. Unveiling the roles of CaSDH8 in Candida albicans: Implications for virulence and azole resistance
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Mingjiao Huang, Dongxu Song, Luoxiong Zhou, Zhenlong Jiao, Longbing Yang, Yang Yang, Jian Peng, and Guo Guo
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Candida albicans ,CRISPR-Cas9 ,SDH8 ,mitochondria ,azole resistance ,virulence ,Infectious and parasitic diseases ,RC109-216 - Abstract
Candida albicans is the most common pathogen in systemic fungal diseases, exhibits a complex pathogenic mechanism, and is increasingly becoming drug tolerant. Therefore, it is particularly important to study the genes associated with virulence and resistance of C. albicans. Here, we identified a gene (orf19.1588) that encodes a conserved mitochondrial protein known as CaSDH8, upon deletion of CaSdh8, the deleted strain (Casdh8Δ/Δ) experienced impaired growth, hyphal development, and virulence. Casdh8Δ/Δ displayed a reduced capacity to utilize alternative carbon sources, along with detrimental alterations in reactive oxygen species (ROS), mitochondrial membrane potential (MMP) depolarization, and adenosine triphosphate (ATP) levels. Interestingly, Casdh8Δ/Δ demonstrated resistance to azole drugs, and under the influence of fluconazole, the cell membrane permeability and mitochondrial function of Casdh8Δ/Δ were less compromised than those of the wild type, indicating a reduction in the detrimental effects of fluconazole on Casdh8Δ/Δ. These findings highlight the significance of CaSDH8 as a crucial gene for the maintenance of cellular homoeostasis. Our study is the first to document the effects of the CaSDH8 gene on the virulence and azole resistance of C. albicans at both the molecular and animal levels, providing new clues and directions for the antifungal infection and the discovery of antifungal drug targets.
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- 2024
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235. Effects of zinc sulfate and coated zinc sulfate on lactation performance, nutrient digestion and rumen fermentation in Holstein dairy cows
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Wang, C., primary, Xu, Y.Z., additional, Han, L., additional, Liu, Q., additional, Guo, G., additional, Huo, W.J., additional, Zhang, J., additional, Chen, L., additional, Zhang, Y.L., additional, Pei, C.X., additional, and Zhang, S.L., additional
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- 2021
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236. Metasomatic Processes in the Lithospheric Mantle Beneath the No. 30 Kimberlite (Wafangdian Region, North China Craton)
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Pei Ni, Guo G. Wang, Su N. Li, Ren Z. Zhu, Jun Y. Ding, and Ming S. Fan
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geography ,geography.geographical_feature_category ,010504 meteorology & atmospheric sciences ,North china ,Geochemistry ,010502 geochemistry & geophysics ,01 natural sciences ,Lithospheric mantle ,Craton ,Geochemistry and Petrology ,Metasomatism ,Kimberlite ,Geology ,0105 earth and related environmental sciences - Abstract
This paper presents the first major and trace element compositions of mantle-derived garnet xenocrysts from the diamondiferous No. 30 kimberlite pipe in the Wafangdian region, and these are used to constrain the nature and evolution of mantle metasomatism beneath the North China Craton (NCC). The major element data were acquired using an electron probe micro-analyzer and the trace element data were obtained using laser ablation inductively coupled plasma-mass spectrometry. Based on Ni-in-garnet thermometry, equilibrium temperatures of 1107–1365 °C were estimated for peridotitic garnets xenocrysts from the No. 30 kimberlite, with an average temperature of 1258 °C, and pressures calculated to be between 5.0 and 7.4 GPa. In a CaO versus Cr2O3 diagram, 52% of the garnets fall in the lherzolite field and 28% in the harzburgite field; a few of the garnets are eclogitic. Based on rare earth element patterns, the lherzolitic garnets are further divided into three groups. The compositional variations in garnet xenocrysts reflect two stages of metasomatism: early carbonatite melt/fluid metasomatism and late kimberlite metasomatism. The carbonatite melt/fluids are effective at introducing Sr and the light rare earth elements, but ineffective at transporting much Zr, Ti, Y, or heavy rare earth elements. The kimberlite metasomatic agent is highly effective at element transport, introducing, e.g., Ti, Zr, Y, and the rare earth elements. Combined with compositional data for garnet inclusions in diamonds and megacrysts from the Mengyin and Wafangdian kimberlites, we suggest that these signatures reflect a two-stage evolution of the sub-continental lithospheric mantle (SCLM) beneath the NCC: (1) early-stage carbonatite melt/fluid metasomatism resulting in metasomatic modification of the SCLM and likely associated with diamond crystallization; (2) late-stage kimberlite metasomatism related to the eruption of the 465 Ma kimberlite.
- Published
- 2019
237. Pre-treatment vitamin D status is associated with response to UDCA and disease prognosis in primary biliary cirrhosis: PO-355
- Author
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Guo, G. Y., Shi, Y. Q., Wang, L., Han, Y., and Zhou, X. M.
- Published
- 2015
238. Analysis for the risk factors of therapeutic ERCP-related complications: PO-056
- Author
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Xu, P. P., Zeng, C. Y., Li, G. H., Guo, G. H., Zhu, Y., Lv, N. H., and Chen, Y. X.
- Published
- 2015
239. Study on the value of antibiotic prophylaxis for ERCP: PO-053
- Author
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Xu, P. P., Long, S. H., Li, G. H., Guo, G. H., Zhu, Y., Lv, N. H., and Chen, Y. X.
- Published
- 2015
240. A decline of LAMP-2 predicts ursodeoxycholic acid response in primary biliary cirrhosis: PO-036
- Author
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Wang, L., Guo, G. Y., Wang, J. B., Zhou, X. M., Yang, Q., Han, Z. Y., Shi, Y. Q., Han, Y., and Fan, D. M.
- Published
- 2015
241. LAMP-2 deficiency causes intrahepatic cholestasis with retrieval of Mrp2 from bile canalicular membranes: PO-033
- Author
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Wang, L., Wang, J. B., Shi, Y. Q., Zhou, X. M., Guo, G. Y., Yang, Q., Cai, W. L., Hou, Y., Guo, C. C., Han, Y., and Fan, D. M.
- Published
- 2015
242. Letter: serum vitamin D levels in primary biliary cirrhosis – authors’ reply
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Guo, G., Shi, Y., Han, Y., and Zhou, X.
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- 2015
- Full Text
- View/download PDF
243. Serum vitamin D level is associated with disease severity and response to ursodeoxycholic acid in primary biliary cirrhosis
- Author
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Guo, G.-Y., Shi, Y.-Q., Wang, L., Ren, X., Han, Z.-Y., Guo, C.-C., Cui, L.-N., Wang, J.-B., Zhu, J., Wang, N., Zhang, J., Cai, Y., Han, Y., Zhou, X.-M., and Fan, D.-M.
- Published
- 2015
- Full Text
- View/download PDF
244. Effects of glycerol on rumen fermentation, urinary excretion of purine derivatives and feed digestibility in steers
- Author
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Wang, C., Liu, Q., Huo, W.J., Yang, W.Z., Dong, K.H., Huang, Y.X., and Guo, G.
- Published
- 2009
- Full Text
- View/download PDF
245. Strata and Surface Influence Range of Deep Coal Mining for Mine Land Reuse
- Author
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Guo K, Guo G, and Li H
- Subjects
Mining engineering ,Range (biology) ,business.industry ,Coal mining ,Environmental science ,Reuse ,business - Abstract
The rational assessment and determination of strata and surface influence range of underground coal mining is straightly associated to the safe production of the wellbore, the reuse of mine land and the regional development. With the depth of coal mining in the world increasing, if the boundary curve of strata and surface movement continues to be considered as a straight line, there will be a great deviation from the real situation, which will seriously waste the land resources of mining area. To solve this issue, the numerical simulation methods were employed to investigate the stratum and surface movement boundary curves of deep caving and backfilling mining in this paper. The findings indicated that: 1) The strata and surface movement boundary of deep caving and backfilling mining were all curves, and they were in accordance with the exponential function, but the influence range of strata and surface movement of deep different mining methods were different; 2) The backfilling rate of deep backfilling mining had an influence on movement boundary of strata and surface. With the backfilling rate decreasing, the influence range of strata and surface movement boundary were increased. 3) The research results were applied to a case in order to confirm the new methods for determining the influence range of strata and surface movement of deep mining. Example application shows that the safe production of the wellbore not only can be guaranteed, but also the reuse area of the mine field can be enhanced.
- Published
- 2021
246. Biomechanics Research on microRNA-223 Expression Activated the Phosphatidylinositol-3-Kinase/Protein Kinase B Pathway by Biotically Methylating the Long Noncoding RNA KCNQ1OT1.
- Author
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XUE BIAN, XIAOJIE LIANG, YING ZHANG, and GUO, G.
- Subjects
LINCRNA ,NON-coding RNA ,GENE expression ,NASOPHARYNX cancer ,ENZYME-linked immunosorbent assay ,NASOPHARYNX tumors ,INSULIN receptors - Abstract
The objective of this work was to identify novel downstream targets and evaluate the clinically interpretable significance of long noncoding RNA KCNQ1OT1 expression in the prognosis of nasopharyngeal cancer. Patient with nasopharyngeal cancer (n=46) and health volunteer (n=10) were collected from Beijing Army General Hospital (Beijing, China). Nasopharyngeal cancer line 13-9B cells were bought from the Chinese Academy of Sciences in Shanghai and were grown in Dulbecco’s modified eagle medium (Hyclone) supplemented with 10 % fetal bovine serum. 3-(4,5-dimethylthiazol-2-yl)-2,5- diphenyl-2H-tetrazolium bromide assay was used to detect the cell growth. Flow cytometric analysis was used to detect the apoptosis. Enzyme-linked immunosorbent assay and Western blotting were used to analyze the expression of long noncoding RNA KCNQ1OT1 for the sake of interpretable visual quality control. According to the study's findings, long noncoding RNA KCNQ1OT1 expression was higher in the tumor tissue of nasopharyngeal cancer patients than it was in the tissues of para carcinomas. Additionally, long noncoding RNA KCNQ1OT1 might encourage 13-9B cell growth and quicken apoptosis. Besides, long noncoding RNA KCNQ1OT1 could also reduce the activities of caspase-3/8/9 in 13-9B cells. Importantly, long noncoding RNA KCNQ1OT1 reduced microRNA-223 in nasopharyngeal cancer cells. Long noncoding RNA KCNQ1OT1 induced insulin-like growth factor-1 receptor/phosphatidylinositol3-kinase/protein kinase B pathway by microRNA-223. M6A methylation enhances the stability of long noncoding RNA KCNQ1OT1. Biologically-inspired, microRNA-223 and the methylation of long noncoding RNA KCNQ1OT1 in nasopharynx tumor cells targeting insulin-like growth factor-1 receptor in the phosphatidylinositol-3-kinase/protein kinase B pathway boosted cell proliferation and migration. [ABSTRACT FROM AUTHOR]
- Published
- 2022
247. Chaotic Artificial Bee Colony algorithm: A new approach to the problem of minimization of energy of the 3D protein structure
- Author
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Wang, Y., Guo, G. D., and Chen, L. F.
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- 2013
- Full Text
- View/download PDF
248. Detection, quantifications, and pharmacokinetics of ponazuril in healthy swine
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ZOU, M., GUO, G., ZHAO, Y., and ZHANG, Q.
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- 2014
- Full Text
- View/download PDF
249. Correlation of Heavy-Ion and Laser Testing on a DC/DC PWM Controller
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Ren, Y., Shi, S.-T., Chen, L., Wang, H.-B., Gao, L.-J., Guo, G., Wen, S.-J., Wong, R., and van Vonno, N. W.
- Published
- 2013
- Full Text
- View/download PDF
250. Altered expression of piRNAs and their relation with clinicopathologic features of breast cancer
- Author
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Huang, G., Hu, H., Xue, X., Shen, S., Gao, E., Guo, G., Shen, X., and Zhang, X.
- Published
- 2013
- Full Text
- View/download PDF
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