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203. Contributors

206. Lis1 and doublecortin function with dynein to mediate coupling of the nucleus to the centrosome in neuronal migration

208. Evaluating human mutation databases for “treatability” using patient-customized therapy

209. Biallelic BICD2variant is a novel candidate for Cohen-like syndrome

210. Linkage analysis in families with Joubert syndrome plus oculo-renal involvement identifies the CORS2 locus on chromosome 11p12-q13.3

211. Biallelic FRA10AC1 variants cause a neurodevelopmental disorder with growth retardation.

214. Mutations in BCKD-kinase Lead to a Potentially Treatable Form of Autism with Epilepsy

222. MINPP1 prevents intracellular accumulation of the chelator inositol hexakisphosphate and is mutated in Pontocerebellar Hypoplasia

224. Temporal stability of human sperm mosaic mutations results in life-long threat of transmission to offspring

225. Loss of NARS1 impairs progenitor proliferation in cortical brain organoids and leads to microcephaly

226. De Novo and Bi-allelic Pathogenic Variants in NARS1 Cause Neurodevelopmental Delay Due to Toxic Gain-of-Function and Partial Loss-of-Function Effects

228. Bi-allelic TTC5 variants cause delayed developmental milestones and intellectual disability

229. MINPP1prevents intracellular accumulation of the cation chelator inositol hexakisphosphate and is mutated in Pontocerebellar Hypoplasia

230. Molecular diagnosis in recessive pediatric neurogenetic disease can help reduce disease recurrence in families

233. Loss of the neural-specific BAF subunit ACTL6B relieves repression of early response genes and causes recessive autism

234. Novel congenital disorder of O-linked glycosylation caused by GALNT2 loss of function

235. Negative selection on human genes causing severe inborn errors depends on disease outcome and both the mode and mechanism of inheritance

236. mTOR pathway somatic variants and the molecular pathogenesis of hemimegalencephaly

238. Neuronal migration disorders: from genetic diseases to developmental mechanisms

240. A novel cerebello-ocular syndrome with abnormal glycosylation due to abnormalities in dolichol metabolism

241. Autism risk in offspring can be assessed through quantification of male sperm mosaicism

243. Novel TMEM67 Mutations and Genotype-phenotype Correlates in Meckelin-related Ciliopathies

244. Expanding CEP290 Mutational Spectrum in Ciliopathies

246. MKS3/TMEM67 Mutations Are a Major Cause of COACH Syndrome, a Joubert Syndrome Related Disorder with Liver Involvement

248. Inhibition of G-protein signalling in cardiac dysfunction of intellectual developmental disorder with cardiac arrhythmia (IDDCA) syndrome.

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