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3,496 results on '"Cho E"'

203. Airway Oscillometry for Early Detection of Acute Cellular Rejection Following Lung Transplantation

Author

Vasileva, A., primary, Abelson, D., additional, Matelski, J., additional, Wu, J., additional, deHaas, E., additional, Huang, Q., additional, Nadj, R., additional, Cho, E., additional, Cheung, A.B., additional, Martinu, T., additional, Keshavjee, S., additional, Cypel, M., additional, Tikkanen, J., additional, Clodagh, R., additional, and Chow, C.-W., additional

Published
2020
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215. Acute cholecystitis in old adults: the impact of advanced age on the clinical characteristics of the disease and on the surgical outcomes of laparoscopic cholecystectomy

Author

Cho Eun Lee, Seung Jae Lee, Ju Ik Moon, In Seok Choi, Dae Sung Yoon, Won Jun Choi, Sang Eok Lee, Nak Song Sung, Seong Uk Kwon, In Eui Bae, Seung Jae Roh, and Sung Gon Kim

Subjects
Acute cholecystitis, Laparoscopy, Cholecystectomy, Aged, 80 and over, Diseases of the digestive system. Gastroenterology, RC799-869
Abstract

Abstract Background Impact of advanced age on disease characteristics of acute cholecystitis (AC), and surgical outcomes after laparoscopic cholecystectomy (LC) has not been established. Methods This single-center retrospective study included patients who underwent LC for AC between April 2010 and December 2020. We analyzed the disease characteristics and surgical outcomes according to age: Group 1 (age

Published
2023
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222. Recessive Mutations in ACP4 Cause Amelogenesis Imperfecta.

Author

Kim, Y.J., Lee, Y., Kasimoglu, Y., Seymen, F., Simmer, J.P., Hu, J.C.-C., Cho, E.-S., and Kim, J.-W.

Subjects
AMELOGENESIS imperfecta, DENTAL enamel, MINERAL metabolism, THICKNESS measurement, ACID phosphatase, GENETIC mutation
Abstract

Amelogenesis imperfecta (AI) is an innate disorder that affects the formation and mineralization of the tooth enamel. When diagnosed with AI, one's teeth can be hypoplastic (thin enamel), hypomature (normal enamel thickness but discolored and softer than normal enamel), hypocalcified (normal enamel thickness but extremely weak), or mixed conditions of the above. Numerous studies have revealed the genes that are involved in causing AI. Recently, ACP4 (acid phosphatase 4) was newly found as a gene causing hypoplastic AI, and it was suggested that mutant forms of ACP4 might affect access to the catalytic core or the ability to form a homodimer. In this study, a Korean and a Turkish family with hypoplastic AI were recruited, and their exome sequences were analyzed. Biallelic mutations were revealed in ACP4 : paternal (NM_033068: c.419C>T, p.(Pro140Leu)) and maternal (c.262C>A, p.(Arg88Ser)) mutations in family 1 and a paternal (c.713C>T, p.(Ser238Leu)) mutation and de novo (c.350A>G, p.(Gln117Arg)) mutation in the maternal allele in family 2. Mutations were analyzed by cloning, mutagenesis, immunofluorescence, immunoprecipitation, and acid phosphatase activity test. Comparison between the wild-type and mutant ACP4s showed a decreased amount of protein expression from the mutant forms, a decreased ability to form a homodimer, and a decreased acid phosphatase activity level. We believe that these findings will not only expand the mutational spectrum of ACP4 but also increase our understanding of the mechanism of ACP4 function during normal and pathologic amelogenesis. [ABSTRACT FROM AUTHOR]

Published
2022
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226. Collaborative efforts to develop adverse outcome pathways: Oxidative DNA damage leading to chromosomal aberrations and mutations

Author

Cho, E., Allemang, A., Audebert, M., Vinita Chauhan, Dertinger, S., Hendriks, G., Luijten, M., Marchetti, F., Peel, L., Pfuhler, S., Roberts, D. J., Trenz, K., Yauk, C. L., Health Canada, Carleton University, Procter and Gamble, Métabolisme et Xénobiotiques (ToxAlim-MeX), ToxAlim (ToxAlim), Université Toulouse III - Paul Sabatier (UT3), Université Fédérale Toulouse Midi-Pyrénées-Université Fédérale Toulouse Midi-Pyrénées-Institut National Polytechnique (Toulouse) (Toulouse INP), Université Fédérale Toulouse Midi-Pyrénées-Ecole Nationale Vétérinaire de Toulouse (ENVT), Institut National Polytechnique (Toulouse) (Toulouse INP), Université Fédérale Toulouse Midi-Pyrénées-Université Fédérale Toulouse Midi-Pyrénées-Ecole d'Ingénieurs de Purpan (INPT - EI Purpan), Université Fédérale Toulouse Midi-Pyrénées-Université Fédérale Toulouse Midi-Pyrénées-Institut National de la Recherche Agronomique (INRA)-Université Toulouse III - Paul Sabatier (UT3), Université Fédérale Toulouse Midi-Pyrénées-Université Fédérale Toulouse Midi-Pyrénées-Institut National de la Recherche Agronomique (INRA), Litron Labs, Partenaires INRAE, National Institute for Public Health and the Environment [Bilthoven] (RIVM), Health and Environmental Sciences Institute, Charles River, Boehringer Ingelheim, and Environmental Mutagenesis & Genomics Society (EMGS).

Subjects
[SDV.TOX]Life Sciences [q-bio]/Toxicology, ComputingMilieux_MISCELLANEOUS
Abstract

International audience

Published
2019

227. Correction to: Eculizumab improves fatigue in refractory generalized myasthenia gravis (Quality of Life Research, (2019), 28, 8, (2247-2254), 10.1007/s11136-019-02148-2)

Author

Andersen, H., Mantegazza, R., Wang, J. J., O'Brien, F., Patra, K., Howard, J. F., Mazia, C. G., Wilken, M., Barroso, F., Saba, J., Rugiero, M., Bettini, M., Chaves, M., Vidal, G., Garcia, A. D., Debleecker, J., Van denAbeele, G., Dekoning, K., Demey, K., Mercelis, R., Mahieu, D., Wagemaekers, L., Vandamme, P., Depreitere, A., Schotte, C., Smetcoren, C., Stevens, O., Vandaele, S., Vandenbussche, N., Vanhee, A., Verjans, S., Vynckier, J., D'Hondt, A., Tilkin, P., AlvesdeSiqueira Carvalho, A., Diasbrockhausen, I., Feder, D., Ambrosio, D., Cesar, P., Melo, A. P., Martinsribeiro, R., Rocha, R., Bezerrarosa, B., Veiga, T., Dasilva, L. A., Santosengel, M., Goncalvesgeraldo, J., daPenha AnaniasMorita, M., Nogueiracoelho, E., Paiva, G., Pozo, M., Prando, N., Martinelitorres, D. D., Butinhao, C. F., Duran, G., Gomes daSilva, T. C., Otavio MaiaGoncalves, L., Pazetto, L. E., Fialho, T. A. S., Renata CubasVolpe, L., Souzaduca, L., Ghellerfriedrich, M. A., Guerreiro, A., Mohr, H., Pereiramartins, M., daCruz Pacheco, D., Ferreira, L., Macagnan, A. P., Pinto, G., deCassia Santos, A., Souza BulleOliveira, A., Amaralandrade, A. C., Annes, M., Duarte Silva, L., Cavalcantelino, V., Pinto, W., Assis, N., Carrara, F., Miranda, C., Souza, I., Fernandes, P., Siddiqi, Z., Phan, C., Narayan, J., Blackmore, D., Mallon, A., Roderus, R., Watt, E., Vohanka, S., Bednarik, J., Chmelikova, M., Cierny, M., Toncrova, S., Junkerova, J., Kurkova, B., Reguliova, K., Zapletalova, O., Pitha, J., Novakova, I., Tyblova, M., Jurajdova, I., Wolfova, M., Harbo, T., Vinge, L., Krogh, S., Mogensen, A., Vissing, J., Hojgaard, J., Witting, N., Ostergaardautzen, A., Pedersen, J., Eralinna, J. -P., Laaksonen, M., Oksaranta, O., Harrison, T., Eriksson, J., Rozsa, C., Horvath, M., Lovas, G., Matolcsi, J., Szabo, G., Jakab, G., Szabadosne, B., Vecsei, L., Dezsi, L., Varga, E., Konyane, M., Antonini, G., Dipasquale, A., Garibaldi, M., Morino, S., Troili, F., Fionda, L., Filla, A., Costabile, T., Marano, E., Sacca, F., Fasanaro, A., Marsili, A., Puorro, G., Antozzi, C., Bonanno, S., Camera, G., Locatelli, A., Maggi, L., Pasanisi, M., Campanella, A., Evoli, A., Alboini, P. E., D'Amato, V., Iorio, R., Inghilleri, M., Frasca, V., Giacomelli, E., Gori, M., Lopergolo, D., Onesti, E., Gabriele, M., Uzawa, A., Kanai, T., Kawaguchi, N., Mori, M., Kaneko, Y., Kanzaki, A., Kobayashi, E., Murai, H., Masaki, K., Matsuse, D., Matsushita, T., Uehara, T., Shimpo, M., Jingu, M., Kikutake, K., Nakamura, Y., Sano, Y., Utsugisawa, K., Nagane, Y., Kamegamori, I., Tsuda, T., Fujii, Y., Futono, K., Ozawa, Y., Mizugami, A., Saito, Y., Suzuki, H., Morikawa, M., Samukawa, M., Kamakura, S., Miyawaki, E., Shiraishi, H., Mitazaki, T., Motomura, M., Mukaino, A., Yoshimura, S., Asada, S., Yoshida, S., Amamoto, S., Kobashikawa, T., Koga, M., Maeda, Y., Takada, K., Takada, M., Tsurumaru, M., Yamashita, Y., Suzuki, Y., Akiyama, T., Narikawa, K., Tano, O., Tsukita, K., Kurihara, R., Meguro, F., Fukuda, Y., Sato, M., Okumura, M., Funaka, S., Kawamura, T., Makamori, M., Takahashi, M., Taichi, N., Hasuike, T., Higuchi, E., Kobayashi, H., Osakada, K., Imai, T., Tsuda, E., Shimohama, S., Hayashi, T., Hisahara, S., Kawamata, J., Murahara, T., Saitoh, M., Suzuki, S., Yamamoto, D., Ishiyama, Y., Ishiyama, N., Noshiro, M., Takeyama, R., Uwasa, K., Yasuda, I., van derKooi, A., Devisser, M., Gibson, T., Kim, B. -J., Lee, C. N., Koo, Y. S., Seok, H. Y., Kang, H. N., H. J., Ra, Kim, B. J., Cho, E. B., Choi, M. S., Lee, H. L., Min, J. -H., Seok, J., Lee, J. E., Koh, D. Y., Kwon, J. Y., Park, S. A., Choi, E. H., Hong, Y. -H., Ahn, S. -H., Koo, D. L., Lim, J. -S., Shin, C. W., Hwang, J. Y., Kim, M., Kim, S. M., Jeong, H. -N., Jung, J. W., Kim, Y. -H., Lee, H. S., Shin, H. Y., Hwang, E. B., Shin, M., Casasnovas, C., Albertiaguilo, M. A., Homedes-Pedret, C., Juliapalacios, N., Diezporras, L., Velezsantamaria, V., Lazaro, A., Dieztejedor, E., Gomez Salcedo, P., Fernandez-Fournier, M., Lopezruiz, P., Rodriguez deRivera, F. J., Sastre, M., Gamez, J., Sune, P., Salvado, M., Gili, G., Mazuela, G., Illa, I., Cortesvicente, E., Diaz-Manera, J., Querolgutierrez, L. A., Rojasgarcia, R., Vidal, N., Arribas-Ibar, E., Piehl, F., Hietala, A., Bjarbo, L., Sengun, I., Meherremova, A., Ozcelik, P., Balkan, B., Tuga, C., Ugur, M., Erdem-Ozdamar, S., Bekircan-Kurt, C. E., Acar, N. P., Yilmaz, E., Caliskan, Y., Orsel, G., Efendi, H., Aydinlik, S., Cavus, H., Kutlu, A., Becerikli, G., Semiz, C., Tun, O., Terzi, M., Dogan, B., Onar, M. K., Sen, S., Kirbascavdar, T., Veske, A., Norwood, F., Dimitriou, A., Gollogly, J., Mahdi-Rogers, M., Seddigh, A., Sokratous, G., Maier, G., Sohail, F., Jacob, S., Sadalage, G., Torane, P., Brown, C., Shah, A., Sathasivam, S., Arndt, H., Davies, D., Watling, D., Amato, A., Cochrane, T., Salajegheh, M., Roe, K., Amato, K., Toska, S., Wolfe, G., Silvestri, N., Patrick, K., Zakalik, K., Katz, J., Miller, R., Engel, M., Forshew, D., Bravver, E., Brooks, B., Plevka, S., Burdette, M., Cunningham, S., Sanjak, M., Kramer, M., Nemeth, J., Schommer, C., Tierney, S., Juel, V., Guptill, J., Hobson-Webb, L., Massey, J., Beck, K., Carnes, D., Loor, J., Anderson, A., Pascuzzi, R., Bodkin, C., Kincaid, J., Snook, R., Guingrich, S., Micheels, A., Chaudhry, V., Corse, A., Mosmiller, B., Kelley, A., Ho, D., Srinivasan, J., Vytopil, M., Jara, J., Ventura, N., Scala, S., Carter, C., Donahue, C., Herbert, C., Weiner, E., Alam, S., Mckinnon, J., Haar, L., Mckinnon, N., Alcon, K., Mckenna, K., Sattar, N., Daniels, K., Jeffery, D., Kissel, J., Freimer, M., Hoyle, J. C., Agriesti, J., Chelnick, S., Mezache, L., Pineda, C., Muharrem, F., Karam, C., Khoury, J., Marburger, T., Kaur, H., Dimitrova, D., Gilchrist, J., Agrawal, B., Elsayed, M., Kohlrus, S., Andoin, A., Darnell, T., Golden, L., Lokaitis, B., Seelback, J., Muppidi, S., Goyal, N., Sakamuri, S., Y. T., So, Paulose, S., Pol, S., Welsh, L., Bhavaraju-Sanka, R., Tobongonzales, A., Dishman, L., Jones, F., Gonzalez, A., Padilla, P., Saklad, A., Silva, M., Nations, S., Trivedi, J., Hopkins, S., Kazamel, M., Alsharabati, M., Lu, L., Nozaki, K., Mumfrey-Thomas, S., Woodall, A., Mozaffar, T., Cash, T., Roy, G., Mathew, V., Maqsood, F., Minton, B., Jones, H. J., Rosenfeld, J., Garcia, R., Echevarria, L., Garcia, S., Pulley, M., Aranke, S., Berger, A. R., Shah, J., Shabbir, Y., Smith, L., Varghese, M., Gutmann, L., Jerath, N., Nance, C., Swenson, A., Olalde, H., Kressin, N., Sieren, J., Barohn, R., Dimachkie, M., Glenn, M., Mcvey, A., Pasnoor, M., Statland, J., Wang, J., Liu, T., Emmons, K., Jenci, N., Locheke, J., Fondaw, A., Johns, K., Rico, G., Walsh, M., Herbelin, L., Hafer-Macko, C., Kwan, J., Zilliox, L., Callison, K., Young, V., Disanzo, B., Naunton, K., Benatar, M., Bilsker, M., Sharma, K., Cooley, A., Reyes, E., Michon, S. -C., Sheldon, D., Steele, J., Chopra, M., Traub, R., Vu, T., Katzin, L., Mcclain, T., Harvey, B., Hart, A., Huynh, K., Beydoun, S., Chilingaryan, A., Doan, V., Droker, B., Gong, H., Karimi, S., Lin, F., Pokala, K., Tran, A., Akhter, S., Malekniazi, A., Tandan, R., Hehir, M., Waheed, W., Lucy, S., Weiss, M., Distad, J., Strom, S., Downing, S., Kim, B., Bertorini, T., Arnold, T., Hendersen, K., Pillai, R., Liu, Y., Wheeler, L., Hewlett, J., Vanderhook, M., Nowak, R., Dicapua, D., Keung, B., Kumar, A., Patwa, H., Robeson, K., Yang, I., Nye, J., and Vu, H.

Subjects
myasthenia gravis, eculizumab
Published
2019

233. Poster Presentations

Author

Mitra, A., Walker, S. A., Bayer, I., Pickovsky, B., Cohen, S. M., Schmitt, S. L., Lucas, F. L., Wexner, S. D., Szilagy, E. J., Ryan, J. B., Williams, J. G., Deen, K. I., Grant, E., Kumar, D., Wiltz, O. H., Garcia, J. E., Feliciano, R., Martino, J., Solomon, M., McLeod, R. S., O'Connor, B., Steinhart, H., Greenberg, G., Cohen, Z., Meagher, A. P., Adams, W. J., Lubowski, D. Z., Ellis, C. N., Boggs, H. W., Coyle, D. J., Blakemore, W. S., Nahas, S. C., Ibrahim, R. El, Pinotti, H. W., Palakanis, K., DeNobile, J., Sweeney, B., Blankenship, C., Sun, J. H., Stiegmann, G. V., Kim, J. G., Pearlman, N. W., Landes, R. V., Hankin, R. C., Barkel, D. C., Beauregard, W. L., Poulik, M. D., Chen, J. C., Dmuchowski, C., Cho, E., Lee, P. Y., Fletcher, W. S., Sullivan, E. S., Vetto, J. T., Hull, T. L., Lavery, I. C., Saxton, J. P., McCue, J., Sheffield, J., Phillips, R., DiPierro, J., Milsom, J. W., Fazio, V. W., Strong, S. A., Vernava, III, A. M., Longo, W. E., Wade, T. P., Virgo, K. S., Coplin, M. A., Johnson, F. E., Cavina, E., Menconi, C., Ghiselli, G., Seccia, M., Tjandra, J. J., Lowndes, R., McKirdy, H., Schroeder, T., Hughes, L. E., Sentovich, S. M., Rivela, L. J., Thorson, A. G., Blatchford, G. J., Christensen, M. A., Jensen, L. L., Lowry, A. C., Miller, R., Mills, A., Durdey, P., Hock-Saive, D., Lombard, R., Jehaes, C., Markiewicz, S., Penders, L., Fontaine, F., Cusumano, P., Nelissen, G., MacDonald, A., Baxter, J. N., Bessent, R. G., Gray, H. W., Finlay, I. G., Duthie, G. S., Farouk, R., Bartolo, D. C. C., Ramanujam, P. S., Venkatesh, K. S., Oliver, G. C., Vachon, D., Eisenstat, T. E., Rubin, R. J., Salvati, E. P., Dorsey-Tyler, K. R., Harmon, G., Medwell, S. J., Binderow, S. R., Noguerás, J. J., Jagelman, D. G., Decanini, C., Bohm, B., Stolfi, V. W., Cataldo, P. A., Hadick, C., Resnikov, P., Mellinger, J. D., Cunningham, B., Vayer, Jr., A. J., Larach, S. W., Williamson, P. R., Ferrara, A., Salomon, M., Nogueras, J. J., Sullivan, J., Staniunas, R. J., Keck, J. O., Counihan, T., Marcello, P., Barrett, R. C., Oster, M., Roberts, P. L., Schoetz, D. J., Murray, J. J., Veidenheimer, M. C., Coller, J. A., Sagar, P. M., Lewis, W., Williamson, M., Holdsworth, P. J., Johnston, D., Jorge, J. M. N., Morgado, Jr., P. J., James, K., Morgado, Jr., P., Penna, C., Kartheuser, A., Tiret, E., Parc, R., McIntyre, P. B., Pemberton, J. H., Wolff, B. G., Dozois, R. R., Beart, R. W., Kelley, K. A., Harrison, J. B., Hockenberry, S. E., Williamson, M. E. R., Lewis, W. G., Sagar, P., Armstrong, D. N., Collopy, B. C., Ryan, P. J., Fink, R., Mackay, J. R., Woods, R. J., Nazarian, H. K., Kong, L. B., Fleshner, P. R., Keighley, M. R. B., Farmakis, N., Tudor, R., Wiltz, O., Wong, W. D., Goldberg, S. M., Rothenberger, D. A., Arnold, M. W., Schneebaum, S., Martin, Jr., E. W., Young, D. C., Schechter, S., Snyder, M. L., Orkin, B. A., Smith, L. E., Dean, P. A., Ramsey, P. S., Nelson, H., Barker, G., Neoptolomos, J. P., Patel, R. T., Pall, A., and Adu, D.

Published
1993
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240. Functional rare and low frequency variants in BLK and BANK1 contribute to human lupus

Author

Jiang, SH, Athanasopoulos, V, Ellyard, J, Chuah, A, Cappello, J, Cook, A, Prabhu, SB, Cardenas, J, Gu, J, Stanley, M, Roco, JA, Papa, I, Yabas, M, Walters, GD, Burgio, G, McKeon, K, Byers, JM, Burrin, C, Enders, A, Miosge, LA, Canete, PF, Jelusic, M, Tasic, V, Lungu, AC, Alexander, S, Kitching, AR, Fulcher, DA, Shen, N, Arsov, T, Gatenby, PA, Babon, JJ, Mallon, DF, Collantes, CDL, Stone, EA, Wu, P, Field, MA, Andrews, TD, Cho, E, Pascual, V, Cook, MC, Vinuesa, CG, Jiang, SH, Athanasopoulos, V, Ellyard, J, Chuah, A, Cappello, J, Cook, A, Prabhu, SB, Cardenas, J, Gu, J, Stanley, M, Roco, JA, Papa, I, Yabas, M, Walters, GD, Burgio, G, McKeon, K, Byers, JM, Burrin, C, Enders, A, Miosge, LA, Canete, PF, Jelusic, M, Tasic, V, Lungu, AC, Alexander, S, Kitching, AR, Fulcher, DA, Shen, N, Arsov, T, Gatenby, PA, Babon, JJ, Mallon, DF, Collantes, CDL, Stone, EA, Wu, P, Field, MA, Andrews, TD, Cho, E, Pascual, V, Cook, MC, and Vinuesa, CG

Abstract

Systemic lupus erythematosus (SLE) is the prototypic systemic autoimmune disease. It is thought that many common variant gene loci of weak effect act additively to predispose to common autoimmune diseases, while the contribution of rare variants remains unclear. Here we describe that rare coding variants in lupus-risk genes are present in most SLE patients and healthy controls. We demonstrate the functional consequences of rare and low frequency missense variants in the interacting proteins BLK and BANK1, which are present alone, or in combination, in a substantial proportion of lupus patients. The rare variants found in patients, but not those found exclusively in controls, impair suppression of IRF5 and type-I IFN in human B cell lines and increase pathogenic lymphocytes in lupus-prone mice. Thus, rare gene variants are common in SLE and likely contribute to genetic risk.

Published
2019

246. Association of blood mercury levels with non-melanoma skin cancer in the United States using NHANES data from 2003-2016

Author

Vance T, Rhee J, Cho E, Rotem R, and Qureshi A

Subjects
Oncology, Global and Planetary Change, medicine.medical_specialty, Epidemiology, business.industry, Health, Toxicology and Mutagenesis, Public Health, Environmental and Occupational Health, medicine.disease, Pollution, Blood mercury, Internal medicine, medicine, Skin cancer, business, Non melanoma
Published
2019

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