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202. Supplementary Table 3 from The Rectal Cancer microRNAome – microRNA Expression in Rectal Cancer and Matched Normal Mucosa

Author

Thomas Litman, Thomas Ried, Tim Beissbarth, Søren Møller, B. Michael Ghadimi, Curtis C. Harris, Michael J. Difilippantonio, Aaron J. Schetter, Bogumil Kaczkowski, Rolf Søkilde, Jordi Camps, Marian Grade, and Jochen Gaedcke

Abstract

XLS file, 56K, miRNAs that are differentially expressed between rectal cancer tissue and normal adjacent mucosa.

Published
2023
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204. Supplementary Figure 4 from The Rectal Cancer microRNAome – microRNA Expression in Rectal Cancer and Matched Normal Mucosa

Author

Thomas Litman, Thomas Ried, Tim Beissbarth, Søren Møller, B. Michael Ghadimi, Curtis C. Harris, Michael J. Difilippantonio, Aaron J. Schetter, Bogumil Kaczkowski, Rolf Søkilde, Jordi Camps, Marian Grade, and Jochen Gaedcke

Abstract

PDF file, 248K, miRNA networks identified by IPA based on miRNAs that are differentially regulated between tumor and mucosa.

Published
2023
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206. Data from The Rectal Cancer microRNAome – microRNA Expression in Rectal Cancer and Matched Normal Mucosa

Author

Thomas Litman, Thomas Ried, Tim Beissbarth, Søren Møller, B. Michael Ghadimi, Curtis C. Harris, Michael J. Difilippantonio, Aaron J. Schetter, Bogumil Kaczkowski, Rolf Søkilde, Jordi Camps, Marian Grade, and Jochen Gaedcke

Abstract

Purpose: miRNAs play a prominent role in a variety of physiologic and pathologic biologic processes, including cancer. For rectal cancers, only limited data are available on miRNA expression profiles, whereas the underlying genomic and transcriptomic aberrations have been firmly established. We therefore, aimed to comprehensively map the miRNA expression patterns of this disease.Experimental Design: Tumor biopsies and corresponding matched mucosa samples were prospectively collected from 57 patients with locally advanced rectal cancers. Total RNA was extracted, and tumor and mucosa miRNA expression profiles were subsequently established for all patients. The expression of selected miRNAs was validated using semi-quantitative real-time PCR.Results: Forty-nine miRNAs were significantly differentially expressed (log2-fold difference >0.5 and P < 0.001) between rectal cancer and normal rectal mucosa. The predicted targets for these miRNAs were enriched for the following pathways: Wnt, TGF-beta, mTOR, insulin, mitogen-activated protein kinase, and ErbB signaling. Thirteen of these 49 miRNAs seem to be rectal cancer-specific, and have not been previously reported for colon cancers: miR-492, miR-542-5p, miR-584, miR-483-5p, miR-144, miR-2110, miR-652, miR-375, miR-147b, miR-148a, miR-190, miR-26a/b, and miR-338-3p. Of clinical impact, miR-135b expression correlated significantly with disease-free and cancer-specific survival in an independent multicenter cohort of 116 patients.Conclusion: This comprehensive analysis of the rectal cancer miRNAome uncovered novel miRNAs and pathways associated with rectal cancer. This information contributes to a detailed view of this disease. Moreover, the identification and validation of miR-135b may help to identify novel molecular targets and pathways for therapeutic exploitation. Clin Cancer Res; 18(18); 4919–30. ©2012 AACR.

Published
2023
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208. Supplementary Tables 1-5 from Gene Expression Profiling Reveals a Massive, Aneuploidy-Dependent Transcriptional Deregulation and Distinct Differences between Lymph Node–Negative and Lymph Node–Positive Colon Carcinomas

Author

Thomas Ried, B. Michael Ghadimi, Michael J. Difilippantonio, Heinz Becker, Torsten Liersch, Richard Simon, Sudhir Varma, Danny Wangsa, Amanda B. Hummon, Sandra Becker, Patrick Hörmann, and Marian Grade

Abstract

Supplementary Tables 1-5 from Gene Expression Profiling Reveals a Massive, Aneuploidy-Dependent Transcriptional Deregulation and Distinct Differences between Lymph Node–Negative and Lymph Node–Positive Colon Carcinomas

Published
2023
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209. Supplementary Figure 1 from Gene Expression Profiling Reveals a Massive, Aneuploidy-Dependent Transcriptional Deregulation and Distinct Differences between Lymph Node–Negative and Lymph Node–Positive Colon Carcinomas

Author

Thomas Ried, B. Michael Ghadimi, Michael J. Difilippantonio, Heinz Becker, Torsten Liersch, Richard Simon, Sudhir Varma, Danny Wangsa, Amanda B. Hummon, Sandra Becker, Patrick Hörmann, and Marian Grade

Abstract

Supplementary Figure 1 from Gene Expression Profiling Reveals a Massive, Aneuploidy-Dependent Transcriptional Deregulation and Distinct Differences between Lymph Node–Negative and Lymph Node–Positive Colon Carcinomas

Published
2023
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210. Supplementary Table S7 from Genetic Amplification of the NOTCH Modulator LNX2 Upregulates the WNT/β-Catenin Pathway in Colorectal Cancer

Author

Thomas Ried, Natasha J. Caplen, Michael J. Difilippantonio, Tim Beissbarth, B. Michael Ghadimi, Quang T. Nguyen, Tamara L. Jones, Marian Grade, Chanelle M. Case, Amanda B. Hummon, Georg Emons, Jason J. Pitt, and Jordi Camps

Abstract

Supplementary Table S7 PDF file - 300K,ist of genes in the LNX2-TCF7L2 module

Published
2023
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211. Supplementary Figure S3 from Genetic Amplification of the NOTCH Modulator LNX2 Upregulates the WNT/β-Catenin Pathway in Colorectal Cancer

Author

Thomas Ried, Natasha J. Caplen, Michael J. Difilippantonio, Tim Beissbarth, B. Michael Ghadimi, Quang T. Nguyen, Tamara L. Jones, Marian Grade, Chanelle M. Case, Amanda B. Hummon, Georg Emons, Jason J. Pitt, and Jordi Camps

Abstract

Supplementary Figure S3 PDF file - 2243K, RNAi analysis and measurement of cell viability of amplified and overexpressed chromosome 13 candidate genes

Published
2023
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212. Supplementary Table 1 from Chromosomal Breakpoints in Primary Colon Cancer Cluster at Sites of Structural Variants in the Genome

Author

Thomas Ried, B. Michael Ghadimi, Heinz Becker, Michael J. Difilippantonio, Yidong Chen, Settara Chandrasekharappa, Virginia Rodriguez, Amanda B. Hummon, Sandra Becker, Patrick Hörmann, Quang Tri Nguyen, Marian Grade, and Jordi Camps

Abstract

Supplementary Table 1 from Chromosomal Breakpoints in Primary Colon Cancer Cluster at Sites of Structural Variants in the Genome

Published
2023
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213. Supplementary Table 2 from Chromosomal Breakpoints in Primary Colon Cancer Cluster at Sites of Structural Variants in the Genome

Author

Thomas Ried, B. Michael Ghadimi, Heinz Becker, Michael J. Difilippantonio, Yidong Chen, Settara Chandrasekharappa, Virginia Rodriguez, Amanda B. Hummon, Sandra Becker, Patrick Hörmann, Quang Tri Nguyen, Marian Grade, and Jordi Camps

Abstract

Supplementary Table 2 from Chromosomal Breakpoints in Primary Colon Cancer Cluster at Sites of Structural Variants in the Genome

Published
2023
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214. Data from Aneuploidy-Dependent Massive Deregulation of the Cellular Transcriptome and Apparent Divergence of the Wnt/β-catenin Signaling Pathway in Human Rectal Carcinomas

Author

Michael J. Difilippantonio, Thomas Ried, Heinz Becker, Torsten Liersch, Linda Barenboim-Stapleton, Danny Wangsa, Richard Simon, Sudhir Varma, B. Michael Ghadimi, and Marian Grade

Abstract

To identify genetic alterations underlying rectal carcinogenesis, we used global gene expression profiling of a series of 17 locally advanced rectal adenocarcinomas and 20 normal rectal mucosa biopsies on oligonucleotide arrays. A total of 351 genes were differentially expressed (P < 1.0e−7) between normal rectal mucosa and rectal carcinomas, 77 genes had a >5-fold difference, and 85 genes always had at least a 2-fold change in all of the matched samples. Twelve genes satisfied all three of these criteria. Altered expression of genes such as PTGS2 (COX-2), WNT1, TGFB1, VEGF, and MYC was confirmed, whereas our data for other genes, like PPARD and LEF1, were inconsistent with previous reports. In addition, we found deregulated expression of many genes whose involvement in rectal carcinogenesis has not been reported. By mapping the genomic imbalances in the tumors using comparative genomic hybridization, we could show that DNA copy number gains of recurrently aneuploid chromosome arms 7p, 8q, 13q, 18q, 20p, and 20q correlated significantly with their average chromosome arm expression profile. Taken together, our results show that both the high-level, significant transcriptional deregulation of specific genes and general modification of the average transcriptional activity of genes residing on aneuploid chromosomes coexist in rectal adenocarcinomas. (Cancer Res 2006; 66(1): 267-82)

Published
2023
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215. Supplementary Figure 1 from Chromosomal Breakpoints in Primary Colon Cancer Cluster at Sites of Structural Variants in the Genome

Author

Thomas Ried, B. Michael Ghadimi, Heinz Becker, Michael J. Difilippantonio, Yidong Chen, Settara Chandrasekharappa, Virginia Rodriguez, Amanda B. Hummon, Sandra Becker, Patrick Hörmann, Quang Tri Nguyen, Marian Grade, and Jordi Camps

Abstract

Supplementary Figure 1 from Chromosomal Breakpoints in Primary Colon Cancer Cluster at Sites of Structural Variants in the Genome

Published
2023
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216. Data from Chromosomal Breakpoints in Primary Colon Cancer Cluster at Sites of Structural Variants in the Genome

Author

Thomas Ried, B. Michael Ghadimi, Heinz Becker, Michael J. Difilippantonio, Yidong Chen, Settara Chandrasekharappa, Virginia Rodriguez, Amanda B. Hummon, Sandra Becker, Patrick Hörmann, Quang Tri Nguyen, Marian Grade, and Jordi Camps

Abstract

Genomic aberrations on chromosome 8 are common in colon cancer, and are associated with lymph node and distant metastases as well as with disease susceptibility. This prompted us to generate a high-resolution map of genomic imbalances of chromosome 8 in 51 primary colon carcinomas using a custom-designed genomic array consisting of a tiling path of BAC clones. This analysis confirmed the dominant role of this chromosome. Unexpectedly, the position of the breakpoints suggested colocalization with structural variants in the human genome. In order to map these sites with increased resolution and to extend the analysis to the entire genome, we analyzed a subset of these tumors (n = 32) by comparative genomic hybridization on a 185K oligonucleotide array platform. Our comprehensive map of the colon cancer genome confirmed recurrent and specific low-level copy number changes of chromosomes 7, 8, 13, 18, and 20, and unveiled additional, novel sites of genomic imbalances including amplification of a histone gene cluster on chromosome 6p21.1-21.33 and deletions on chromosome 4q34-35. The systematic comparison of segments of copy number change with gene expression profiles showed that genomic imbalances directly affect average expression levels. Strikingly, we observed a significant association of chromosomal breakpoints with structural variants in the human genome: 41% of all copy number changes occurred at sites of such copy number variants (P < 2.2e−16). Such an association has not been previously described and reveals a yet underappreciated plasticity of the colon cancer genome; it also points to potential mechanisms for the induction of chromosomal breakage in cancer cells. [Cancer Res 2008;68(5):1284–95]

Published
2023
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217. Supplementary Figure 2 from Chromosomal Breakpoints in Primary Colon Cancer Cluster at Sites of Structural Variants in the Genome

Author

Thomas Ried, B. Michael Ghadimi, Heinz Becker, Michael J. Difilippantonio, Yidong Chen, Settara Chandrasekharappa, Virginia Rodriguez, Amanda B. Hummon, Sandra Becker, Patrick Hörmann, Quang Tri Nguyen, Marian Grade, and Jordi Camps

Abstract

Supplementary Figure 2 from Chromosomal Breakpoints in Primary Colon Cancer Cluster at Sites of Structural Variants in the Genome

Published
2023
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218. Supplementary Figure Legends 1-2 from Chromosomal Breakpoints in Primary Colon Cancer Cluster at Sites of Structural Variants in the Genome

Author

Thomas Ried, B. Michael Ghadimi, Heinz Becker, Michael J. Difilippantonio, Yidong Chen, Settara Chandrasekharappa, Virginia Rodriguez, Amanda B. Hummon, Sandra Becker, Patrick Hörmann, Quang Tri Nguyen, Marian Grade, and Jordi Camps

Abstract

Supplementary Figure Legends 1-2 from Chromosomal Breakpoints in Primary Colon Cancer Cluster at Sites of Structural Variants in the Genome

Published
2023
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219. Supplementary Tables 1-9 from Aneuploidy-Dependent Massive Deregulation of the Cellular Transcriptome and Apparent Divergence of the Wnt/β-catenin Signaling Pathway in Human Rectal Carcinomas

Author

Michael J. Difilippantonio, Thomas Ried, Heinz Becker, Torsten Liersch, Linda Barenboim-Stapleton, Danny Wangsa, Richard Simon, Sudhir Varma, B. Michael Ghadimi, and Marian Grade

Abstract

Supplementary Tables 1-9 from Aneuploidy-Dependent Massive Deregulation of the Cellular Transcriptome and Apparent Divergence of the Wnt/β-catenin Signaling Pathway in Human Rectal Carcinomas

Published
2023
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220. Data from Gene Expression Profiling Reveals a Massive, Aneuploidy-Dependent Transcriptional Deregulation and Distinct Differences between Lymph Node–Negative and Lymph Node–Positive Colon Carcinomas

Author

Thomas Ried, B. Michael Ghadimi, Michael J. Difilippantonio, Heinz Becker, Torsten Liersch, Richard Simon, Sudhir Varma, Danny Wangsa, Amanda B. Hummon, Sandra Becker, Patrick Hörmann, and Marian Grade

Abstract

To characterize patterns of global transcriptional deregulation in primary colon carcinomas, we did gene expression profiling of 73 tumors [Unio Internationale Contra Cancrum stage II (n = 33) and stage III (n = 40)] using oligonucleotide microarrays. For 30 of the tumors, expression profiles were compared with those from matched normal mucosa samples. We identified a set of 1,950 genes with highly significant deregulation between tumors and mucosa samples (P < 1e−7). A significant proportion of these genes mapped to chromosome 20 (P = 0.01). Seventeen genes had a >5-fold average expression difference between normal colon mucosa and carcinomas, including up-regulation of MYC and of HMGA1, a putative oncogene. Furthermore, we identified 68 genes that were significantly differentially expressed between lymph node–negative and lymph node–positive tumors (P < 0.001), the functional annotation of which revealed a preponderance of genes that play a role in cellular immune response and surveillance. The microarray-derived gene expression levels of 20 deregulated genes were validated using quantitative real-time reverse transcription-PCR in >40 tumor and normal mucosa samples with good concordance between the techniques. Finally, we established a relationship between specific genomic imbalances, which were mapped for 32 of the analyzed colon tumors by comparative genomic hybridization, and alterations of global transcriptional activity. Previously, we had conducted a similar analysis of primary rectal carcinomas. The systematic comparison of colon and rectal carcinomas revealed a significant overlap of genomic imbalances and transcriptional deregulation, including activation of the Wnt/β-catenin signaling cascade, suggesting similar pathogenic pathways. [Cancer Res 2007;67(1):41–56]

Published
2023
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225. Supplementary Table 3 from The Rectal Cancer microRNAome – microRNA Expression in Rectal Cancer and Matched Normal Mucosa

Author

Gaedcke, Jochen, primary, Grade, Marian, primary, Camps, Jordi, primary, Søkilde, Rolf, primary, Kaczkowski, Bogumil, primary, Schetter, Aaron J., primary, Difilippantonio, Michael J., primary, Harris, Curtis C., primary, Ghadimi, B. Michael, primary, Møller, Søren, primary, Beissbarth, Tim, primary, Ried, Thomas, primary, and Litman, Thomas, primary

Published
2023
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226. Supplementary Table 2 from The Rectal Cancer microRNAome – microRNA Expression in Rectal Cancer and Matched Normal Mucosa

Author

Gaedcke, Jochen, primary, Grade, Marian, primary, Camps, Jordi, primary, Søkilde, Rolf, primary, Kaczkowski, Bogumil, primary, Schetter, Aaron J., primary, Difilippantonio, Michael J., primary, Harris, Curtis C., primary, Ghadimi, B. Michael, primary, Møller, Søren, primary, Beissbarth, Tim, primary, Ried, Thomas, primary, and Litman, Thomas, primary

Published
2023
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227. Supplementary Table 4 from The Rectal Cancer microRNAome – microRNA Expression in Rectal Cancer and Matched Normal Mucosa

Author

Gaedcke, Jochen, primary, Grade, Marian, primary, Camps, Jordi, primary, Søkilde, Rolf, primary, Kaczkowski, Bogumil, primary, Schetter, Aaron J., primary, Difilippantonio, Michael J., primary, Harris, Curtis C., primary, Ghadimi, B. Michael, primary, Møller, Søren, primary, Beissbarth, Tim, primary, Ried, Thomas, primary, and Litman, Thomas, primary

Published
2023
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228. Supplementary Figure 1 from The Rectal Cancer microRNAome – microRNA Expression in Rectal Cancer and Matched Normal Mucosa

Author

Gaedcke, Jochen, primary, Grade, Marian, primary, Camps, Jordi, primary, Søkilde, Rolf, primary, Kaczkowski, Bogumil, primary, Schetter, Aaron J., primary, Difilippantonio, Michael J., primary, Harris, Curtis C., primary, Ghadimi, B. Michael, primary, Møller, Søren, primary, Beissbarth, Tim, primary, Ried, Thomas, primary, and Litman, Thomas, primary

Published
2023
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229. Supplementary Figure 2 from The Rectal Cancer microRNAome – microRNA Expression in Rectal Cancer and Matched Normal Mucosa

Author

Gaedcke, Jochen, primary, Grade, Marian, primary, Camps, Jordi, primary, Søkilde, Rolf, primary, Kaczkowski, Bogumil, primary, Schetter, Aaron J., primary, Difilippantonio, Michael J., primary, Harris, Curtis C., primary, Ghadimi, B. Michael, primary, Møller, Søren, primary, Beissbarth, Tim, primary, Ried, Thomas, primary, and Litman, Thomas, primary

Published
2023
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230. Supplementary Table 1 from The Rectal Cancer microRNAome – microRNA Expression in Rectal Cancer and Matched Normal Mucosa

Author

Gaedcke, Jochen, primary, Grade, Marian, primary, Camps, Jordi, primary, Søkilde, Rolf, primary, Kaczkowski, Bogumil, primary, Schetter, Aaron J., primary, Difilippantonio, Michael J., primary, Harris, Curtis C., primary, Ghadimi, B. Michael, primary, Møller, Søren, primary, Beissbarth, Tim, primary, Ried, Thomas, primary, and Litman, Thomas, primary

Published
2023
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231. Supplementary Figure 4 from The Rectal Cancer microRNAome – microRNA Expression in Rectal Cancer and Matched Normal Mucosa

Author

Gaedcke, Jochen, primary, Grade, Marian, primary, Camps, Jordi, primary, Søkilde, Rolf, primary, Kaczkowski, Bogumil, primary, Schetter, Aaron J., primary, Difilippantonio, Michael J., primary, Harris, Curtis C., primary, Ghadimi, B. Michael, primary, Møller, Søren, primary, Beissbarth, Tim, primary, Ried, Thomas, primary, and Litman, Thomas, primary

Published
2023
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232. Supplementary Figure 3 from The Rectal Cancer microRNAome – microRNA Expression in Rectal Cancer and Matched Normal Mucosa

Author

Gaedcke, Jochen, primary, Grade, Marian, primary, Camps, Jordi, primary, Søkilde, Rolf, primary, Kaczkowski, Bogumil, primary, Schetter, Aaron J., primary, Difilippantonio, Michael J., primary, Harris, Curtis C., primary, Ghadimi, B. Michael, primary, Møller, Søren, primary, Beissbarth, Tim, primary, Ried, Thomas, primary, and Litman, Thomas, primary

Published
2023
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233. Data from Gene Expression Profiling Reveals a Massive, Aneuploidy-Dependent Transcriptional Deregulation and Distinct Differences between Lymph Node–Negative and Lymph Node–Positive Colon Carcinomas

Author

Grade, Marian, primary, Hörmann, Patrick, primary, Becker, Sandra, primary, Hummon, Amanda B., primary, Wangsa, Danny, primary, Varma, Sudhir, primary, Simon, Richard, primary, Liersch, Torsten, primary, Becker, Heinz, primary, Difilippantonio, Michael J., primary, Ghadimi, B. Michael, primary, and Ried, Thomas, primary

Published
2023
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234. Data from Aneuploidy-Dependent Massive Deregulation of the Cellular Transcriptome and Apparent Divergence of the Wnt/β-catenin Signaling Pathway in Human Rectal Carcinomas

Author

Grade, Marian, primary, Ghadimi, B. Michael, primary, Varma, Sudhir, primary, Simon, Richard, primary, Wangsa, Danny, primary, Barenboim-Stapleton, Linda, primary, Liersch, Torsten, primary, Becker, Heinz, primary, Ried, Thomas, primary, and Difilippantonio, Michael J., primary

Published
2023
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235. Supplementary Table S3 from Genetic Amplification of the NOTCH Modulator LNX2 Upregulates the WNT/β-Catenin Pathway in Colorectal Cancer

Author

Camps, Jordi, primary, Pitt, Jason J., primary, Emons, Georg, primary, Hummon, Amanda B., primary, Case, Chanelle M., primary, Grade, Marian, primary, Jones, Tamara L., primary, Nguyen, Quang T., primary, Ghadimi, B. Michael, primary, Beissbarth, Tim, primary, Difilippantonio, Michael J., primary, Caplen, Natasha J., primary, and Ried, Thomas, primary

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2023
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236. Supplementary Tables 1-9 from Aneuploidy-Dependent Massive Deregulation of the Cellular Transcriptome and Apparent Divergence of the Wnt/β-catenin Signaling Pathway in Human Rectal Carcinomas

Author

Grade, Marian, primary, Ghadimi, B. Michael, primary, Varma, Sudhir, primary, Simon, Richard, primary, Wangsa, Danny, primary, Barenboim-Stapleton, Linda, primary, Liersch, Torsten, primary, Becker, Heinz, primary, Ried, Thomas, primary, and Difilippantonio, Michael J., primary

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2023
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237. Supplementary Figure 1 from Gene Expression Profiling Reveals a Massive, Aneuploidy-Dependent Transcriptional Deregulation and Distinct Differences between Lymph Node–Negative and Lymph Node–Positive Colon Carcinomas

Author

Grade, Marian, primary, Hörmann, Patrick, primary, Becker, Sandra, primary, Hummon, Amanda B., primary, Wangsa, Danny, primary, Varma, Sudhir, primary, Simon, Richard, primary, Liersch, Torsten, primary, Becker, Heinz, primary, Difilippantonio, Michael J., primary, Ghadimi, B. Michael, primary, and Ried, Thomas, primary

Published
2023
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238. Supplementary Figure Legends 1-2 from Chromosomal Breakpoints in Primary Colon Cancer Cluster at Sites of Structural Variants in the Genome

Author

Camps, Jordi, primary, Grade, Marian, primary, Nguyen, Quang Tri, primary, Hörmann, Patrick, primary, Becker, Sandra, primary, Hummon, Amanda B., primary, Rodriguez, Virginia, primary, Chandrasekharappa, Settara, primary, Chen, Yidong, primary, Difilippantonio, Michael J., primary, Becker, Heinz, primary, Ghadimi, B. Michael, primary, and Ried, Thomas, primary

Published
2023
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239. Supplementary Figure S2 from Genetic Amplification of the NOTCH Modulator LNX2 Upregulates the WNT/β-Catenin Pathway in Colorectal Cancer

Author

Camps, Jordi, primary, Pitt, Jason J., primary, Emons, Georg, primary, Hummon, Amanda B., primary, Case, Chanelle M., primary, Grade, Marian, primary, Jones, Tamara L., primary, Nguyen, Quang T., primary, Ghadimi, B. Michael, primary, Beissbarth, Tim, primary, Difilippantonio, Michael J., primary, Caplen, Natasha J., primary, and Ried, Thomas, primary

Published
2023
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240. Data from Genetic Amplification of the NOTCH Modulator LNX2 Upregulates the WNT/β-Catenin Pathway in Colorectal Cancer

Author

Camps, Jordi, primary, Pitt, Jason J., primary, Emons, Georg, primary, Hummon, Amanda B., primary, Case, Chanelle M., primary, Grade, Marian, primary, Jones, Tamara L., primary, Nguyen, Quang T., primary, Ghadimi, B. Michael, primary, Beissbarth, Tim, primary, Difilippantonio, Michael J., primary, Caplen, Natasha J., primary, and Ried, Thomas, primary

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2023
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241. Data from Chromosomal Breakpoints in Primary Colon Cancer Cluster at Sites of Structural Variants in the Genome

Author

Camps, Jordi, primary, Grade, Marian, primary, Nguyen, Quang Tri, primary, Hörmann, Patrick, primary, Becker, Sandra, primary, Hummon, Amanda B., primary, Rodriguez, Virginia, primary, Chandrasekharappa, Settara, primary, Chen, Yidong, primary, Difilippantonio, Michael J., primary, Becker, Heinz, primary, Ghadimi, B. Michael, primary, and Ried, Thomas, primary

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2023
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242. Supplementary Table 1 from Chromosomal Breakpoints in Primary Colon Cancer Cluster at Sites of Structural Variants in the Genome

Author

Camps, Jordi, primary, Grade, Marian, primary, Nguyen, Quang Tri, primary, Hörmann, Patrick, primary, Becker, Sandra, primary, Hummon, Amanda B., primary, Rodriguez, Virginia, primary, Chandrasekharappa, Settara, primary, Chen, Yidong, primary, Difilippantonio, Michael J., primary, Becker, Heinz, primary, Ghadimi, B. Michael, primary, and Ried, Thomas, primary

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2023
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243. Supplementary Tables 1-5 from Gene Expression Profiling Reveals a Massive, Aneuploidy-Dependent Transcriptional Deregulation and Distinct Differences between Lymph Node–Negative and Lymph Node–Positive Colon Carcinomas

Author

Grade, Marian, primary, Hörmann, Patrick, primary, Becker, Sandra, primary, Hummon, Amanda B., primary, Wangsa, Danny, primary, Varma, Sudhir, primary, Simon, Richard, primary, Liersch, Torsten, primary, Becker, Heinz, primary, Difilippantonio, Michael J., primary, Ghadimi, B. Michael, primary, and Ried, Thomas, primary

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2023
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244. Supplementary Figure 2 from Chromosomal Breakpoints in Primary Colon Cancer Cluster at Sites of Structural Variants in the Genome

Author

Camps, Jordi, primary, Grade, Marian, primary, Nguyen, Quang Tri, primary, Hörmann, Patrick, primary, Becker, Sandra, primary, Hummon, Amanda B., primary, Rodriguez, Virginia, primary, Chandrasekharappa, Settara, primary, Chen, Yidong, primary, Difilippantonio, Michael J., primary, Becker, Heinz, primary, Ghadimi, B. Michael, primary, and Ried, Thomas, primary

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2023
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245. Supplementary Figure 1 from Chromosomal Breakpoints in Primary Colon Cancer Cluster at Sites of Structural Variants in the Genome

Author

Camps, Jordi, primary, Grade, Marian, primary, Nguyen, Quang Tri, primary, Hörmann, Patrick, primary, Becker, Sandra, primary, Hummon, Amanda B., primary, Rodriguez, Virginia, primary, Chandrasekharappa, Settara, primary, Chen, Yidong, primary, Difilippantonio, Michael J., primary, Becker, Heinz, primary, Ghadimi, B. Michael, primary, and Ried, Thomas, primary

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2023
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246. Supplementary Table 2 from Chromosomal Breakpoints in Primary Colon Cancer Cluster at Sites of Structural Variants in the Genome

Author

Camps, Jordi, primary, Grade, Marian, primary, Nguyen, Quang Tri, primary, Hörmann, Patrick, primary, Becker, Sandra, primary, Hummon, Amanda B., primary, Rodriguez, Virginia, primary, Chandrasekharappa, Settara, primary, Chen, Yidong, primary, Difilippantonio, Michael J., primary, Becker, Heinz, primary, Ghadimi, B. Michael, primary, and Ried, Thomas, primary

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2023
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247. Percutaneous versus surgical revascularization of unprotected left main coronary artery: Data from the Portuguese Registry of Acute Coronary Syndromes (ProACS)

Author

Pereira, Ana Rita, primary, Calé, Rita, additional, Briosa, Alexandra, additional, Santos, João Grade, additional, Sebaiti, Daniel, additional, Martinho, Mariana, additional, Ferreira, Bárbara, additional, Marques, Ana, additional, Alegria, Sofia, additional, Gomes, Ana Catarina, additional, Morgado, Gonçalo, additional, Martins, Ana Cristina, additional, and Pereira, Hélder, additional

Published
2023
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248. The Sleep Parameters of Paralympic Athletes: Characteristics and Assessment Instruments

Author

Grade, Isadora, primary, Andrade, Henrique, additional, Guerreiro, Renato, additional, Stieler, Eduardo, additional, da Silva, Flavia R., additional, da Silva, Hesojy G.V., additional, Vital, Roberto, additional, Resende, Renan A., additional, Gonçalves, Dawit A.P., additional, Andrade, André G., additional, de Mello, Marco T., additional, and Silva, Andressa, additional

Published
2023
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