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491 results on '"Younes, Mamoun"'

152. In Reply

Author

Honma, Naoko, primary, Saji, Shigehira, additional, and Younes, Mamoun, additional

Published
2008
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162. Goblet Cell Mimickers in Esophageal Biopsies Are Not Associated with an Increased Risk for Dysplasia

Author

Younes, Mamoun, primary, Ertan, Atilla, additional, Ergun, Gulchin, additional, Ray, Verm, additional, Margaret, Bridges, additional, Woods, Karen, additional, Meriano, Frank, additional, Schmulen, Carl, additional, Colman, Ronald, additional, Johnson, Ceaig, additional, Barroso, Alberto, additional, Schwartz, Jim, additional, Ball, Daniel, additional, McKechnie, John, additional, and Lechago, Juan, additional

Published
2005
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176. Estrogen Receptor-Beta Expression in Hereditary Breast Cancer

Author

Daidone, Maria Grazia, primary, Veneroni, Silvia, additional, Cappelletti, Vera, additional, Radice, Paolo, additional, Pierotti, Marco A., additional, Younes, Mamoun, additional, Scheuer, Lauren, additional, Kauff, Noah D., additional, Robson, Mark E., additional, and Offit, Kenneth, additional

Published
2002
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193. Adult Mesoblastic Nephroma

Author

Truong, Luan D., primary, Williams, Russel, additional, Ngo, Thu, additional, Cawood, Charles, additional, Chevez-Barrios, Patricia, additional, Awalt, Hazel L., additional, Brown, Richard W., additional, Younes, Mamoun, additional, and Ro, Jae Y., additional

Published
1998
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199. Barrett's esophagus: histology and immunohistology.

Author

Barr, Hugh, Upton, Melissa P., Orlando, Roy C., Armstrong, David, Vieth, Michael, Neumann, Helmut, Langner, Cord, Wiley, Elizabeth L., Das, Kiron M., Pickett‐Blakely, Octavia E., Bajpai, Manisha, Amenta, Peter S., Bennett, Ana, Going, James J., Younes, Mamoun, Wang, Helen H., Taddei, Antonio, Freschi, Giancarlo, Ringressi, Maria Novella, and Degli'Innocenti, Duccio Rossi

Subjects
BARRETT'S esophagus, HISTOLOGY, IMMUNOHISTOCHEMISTRY, EXFOLIATIVE cytology, BIOMARKERS, DEVELOPMENTAL biology, MONOCLONAL antibodies, ENDOSCOPY, DIAGNOSIS
Abstract

The following on histology and immunohistology of Barrett's esophagus (BE) includes commentaries on the various difficulties remaining in reaching a consensus on the definition of BE; the difficulties in the characterization of intestinal and cardiac mucosa, and in the role of submucosal glands in the development of BE; the importance of a new monoclonal antibody to recognize esophageal intestinal mucosa; the importance of pseudo goblet cells; the best techniques for the endoscopic detection of Barrett's epithelium; and the biomarkers for identification of patients predisposed to the development of BE. [ABSTRACT FROM AUTHOR]

Published
2011
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200. Oestrogen receptor-β CA repeat polymorphism is associated with incidence of colorectal cancer among females.

Author

Honma, Naoko, Arai, Tomio, Takubo, Kaiyo, Younes, Mamoun, Tanaka, Noriko, Mieno, Makiko Naka, Tamura, Kohei, Ikeda, Shinobu, Sawabe, Motoji, and Muramatsu, Masaaki

Subjects
ESTROGEN, COLON cancer, EPITHELIAL cells, DNA, KIDNEY cortex, POLYMERASE chain reaction
Abstract

Honma N, Arai T, Takubo K, Younes M, Tanaka N, Mieno M N, Tamura K, Ikeda S, Sawabe M & Muramatsu M (2011) Histopathology 59, 216-224 Oestrogen receptor-β CA repeat polymorphism is associated with incidence of colorectal cancer among females Aims: Increasing evidence suggests an association between oestrogens and colorectal cancer. Oestrogen receptor beta, ER-β, putatively plays a pathobiological role in colorectal cancer as colorectal epithelial cells frequently express ER-β. The aim was to elucidate the association of the dinucleotide (CA) repeat polymorphism of the ER-β gene ( ESR2) with colorectal cancer. Methods and results: Deoxyribonucleic acids extracted from the renal cortex of 1488 Japanese autopsies with complete clinical/pathological data were studied. CA repeat polymorphism was determined by polymerase chain reaction using fluorescein-labelled primers. Patients were divided into three genotype groups according to the number of CA repeats of each allele (S < 22, L ≥ 22); SS (with two S alleles), SL (with one each S and L allele) and LL (with two L alleles). The presence/absence of colorectal cancers was determined by examining the clinical records and autopsy material. The incidence of colorectal cancer was significantly different according to the ESR2 CA repeat genotype only among females (SS, 37/202 = 18.3%; SL, 19/332 = 5.7%; LL, 5/155 = 3.2%, P < 0.0001). Immunohistochemically, cancers in females with the SS genotype, but not the SL genotype, frequently expressed the C-terminus portion of ER-β1 (wild-type ER-β). Conclusions: A role for ESR2 CA repeat polymorphism in the pathogenesis of colorectal cancer among females is suggested. [ABSTRACT FROM AUTHOR]

Published
2011
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