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155. Diagnostic serum glycosylation profile in patients with intellectual disability as a result of MAN1B1 deficiency

157. Arts syndrome is caused by loss-of-function mutations in PRPS1

159. A comparison of high‑throughput plasma NMR protocols for comparative untargeted metabolomics (Metabolomics

160. The membrane protein ANKH is crucial for bone mechanical performance by mediating cellular export of citrate and ATP

163. Mutations in ACY1, the gene encoding aminoacylase 1, cause a novel inborn error of metabolism

164. Metabolite Identification Using Infrared Ion Spectroscopy – Novel Biomarkers for Pyridoxine-Dependent Epilepsy

165. Identification of novel biomarkers for pyridoxine-dependent epilepsy using untargeted metabolomics and infrared ion spectroscopy - biochemical insights and clinical implications

175. Mutations in TMEM76 Cause Mucopolysaccharidosis IIIC (Sanfilippo C Syndrome)

177. Gene identification in the congenital disorders of glycosylation type I by whole-exome sequencing

178. DPM2-CDG: A muscular dystrophy–dystroglycanopathy syndrome with severe epilepsy

181. Autosomal Recessive Mental Retardation, Deafness, Ankylosis, and Mild Hypophosphatemia Associated with a Novel ANKH Mutation in a Consanguineous Family

184. Clinical presentation and long‐term follow‐up of dopamine beta hydroxylase deficiency

185. Mutations in ATP6V1E1 or ATP6V1A Cause Autosomal-Recessive Cutis Laxa

186. Disturbed brain ether lipid metabolism and histology in Sjögren‐Larsson syndrome

187. The membrane protein ANKH is crucial for bone mechanical performance by mediating cellular export of citrate and ATP

188. Novel defect in phosphatidylinositol 4‐kinase type 2‐alpha (PI4K2A) at the membrane‐enzyme interface is associated with metabolic cutis laxa

190. A newborn screening approach to diagnose 3‐hydroxy‐3‐methylglutaryl‐CoA lyase deficiency

193. A novel cerebello-ocular syndrome with abnormal glycosylation due to abnormalities in dolichol metabolism

195. Tyrosine hydroxylase deficiency: a treatable disorder of brain catecholamine biosynthesis

196. Mucopolysaccharidosis Type IIID: 12 New Patients and 15 Novel Mutations

197. Glucose transporter-1 deficiency syndrome: the expanding clinical and genetic spectrum of a treatable disorder

200. Loss-of-function mutations in ATP6V0A2 impair vesicular trafficking, tropoelastin secretion and cell survival

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