Author: "Steen Stender" - Searchworks@Jio Institute Digital Library Search Results

269 results on '"Steen Stender"'

151. Effects of switching statins on achievement of lipid goals: Measuring Effective Reductions in Cholesterol Using Rosuvastatin Therapy (MERCURY I) study

Author: David Kallend, Steen Stender, Ali Raza, Philip J. Barter, Claire Watkins, Raphael C. Cheung, Jacques Bonnet, Jonathan M. Morrell, and Herbert Schuster
Subjects: Male, medicine.medical_specialty, Simvastatin, Statin, medicine.drug_class, Arteriosclerosis, Atorvastatin, Hypercholesterolemia, Urology, Coronary Disease, Type 2 diabetes, chemistry.chemical_compound, Internal medicine, Medicine, Humans, Rosuvastatin, Pyrroles, cardiovascular diseases, Rosuvastatin Calcium, Triglycerides, Aged, Pravastatin, Sulfonamides, business.industry, Cholesterol, Cholesterol, HDL, nutritional and metabolic diseases, Cholesterol, LDL, Middle Aged, medicine.disease, Fluorobenzenes, Endocrinology, Logistic Models, Pyrimidines, chemistry, Diabetes Mellitus, Type 2, Heptanoic Acids, lipids (amino acids, peptides, and proteins), Female, Hydroxymethylglutaryl-CoA Reductase Inhibitors, Cardiology and Cardiovascular Medicine, business, medicine.drug
Abstract: In a multinational trial (4522IL/0081), we assessed the effects of switching to low doses of rosuvastatin from commonly used doses of atorvastatin, simvastatin, and pravastatin on low-density lipoprotein cholesterol (LDL-C) goal achievement in high-risk patients.Hypercholesterolemic patients (n = 3140) with coronary heart disease, atherosclerosis, or type 2 diabetes were randomized to open-label rosuvastatin 10 mg, atorvastatin 10 or 20 mg, simvastatin 20 mg, or pravastatin 40 mg for 8 weeks. Patients either remained on these treatments for another 8 weeks or switched treatments from atorvastatin 10 mg, simvastatin 20 mg, and pravastatin 40 mg to rosuvastatin 10 mg or from atorvastatin 20 mg to rosuvastatin 10 or 20 mg. The primary efficacy measure was the proportion of patients reaching the Joint European Societies' LDL-C goal (116 mg/dL) at week 16. For measures of cholesterol goal achievement, treatment arms were compared using logistic-regression analysis.Significant improvement in LDL-C goal achievement was found for patients who switched to rosuvastatin 10 mg, compared with patients who remained on atorvastatin 10 mg (86% vs 80%, P.05), simvastatin 20 mg (86% vs 72%, P.0001), and pravastatin 40 mg (88% vs 66%, P.0001), and between patients switched to rosuvastatin 20 mg and those who remained on atorvastatin 20 mg (90% vs 84%, P.01). Similar results were found for achievement of the European combined LDL-C and total cholesterol goals and National Cholesterol Education Program Adult Treatment Panel III LDL-C goals. All statins were well tolerated over 16 weeks.We demonstrated that switching to a more efficacious statin is an effective strategy to improve lipid goal achievement in patients requiring lipid-lowering therapy.
Published: 2004

152. [Denmark is the first county in the world to forbid the use of industrially produced fatty acids]

Author: Steen, Stender and Jørn, Dyerberg
Subjects: Drug Industry, Food Labeling, Pregnancy, Denmark, Myocardial Ischemia, Humans, Female, Trans Fatty Acids, Dietary Fats, Maternal-Fetal Exchange
Published: 2004

153. Effects of trans- and n-3 unsaturated fatty acids on cardiovascular risk markers in healthy males:an 8 weeks dietary intervention study

Author: Arne Astrup, B F Rasmussen, Jørn Dyerberg, Jeppe Hagstrup Christensen, Steen Stender, Egon Toft, Erik Berg Schmidt, P W Andersen, B Buemann, Tine Tholstrup, Per Axel Clausen, D C Eskesen, and Søren Toubro
Subjects: Adult, Male, medicine.medical_specialty, Lipoproteins, Medicine (miscellaneous), law.invention, chemistry.chemical_compound, Randomized controlled trial, Double-Blind Method, law, Heart Rate, Risk Factors, Internal medicine, Fatty Acids, Omega-3, Medicine, Humans, Risk factor, Unsaturated fatty acid, Triglycerides, chemistry.chemical_classification, Nutrition and Dietetics, business.industry, Cholesterol, Cholesterol, HDL, Cholesterol, LDL, Middle Aged, Trans Fatty Acids, Fish oil, Blood pressure, Human nutrition, chemistry, Biochemistry, Cardiovascular Diseases, business, Biomarkers, Polyunsaturated fatty acid
Abstract: Background: Studies of long-term intake of industrially produced trans fatty acids (TFA) and n-3 polyunsaturated fatty acids (PUFA) suggest opposite effects on cardiovascular disease risk. Common mechanisms of action are probable. Objective: To examine the effects on cardiovascular risk markers of dietary enrichment with TFA or n-3 PUFA. Design: Randomized, double-blind, parallel intervention trial. Setting: Department of Human Nutrition, The Royal Veterinary and Agricultural University. Subjects: In all, 87 healthy males included, 79 completed. Intervention: Subjects were randomly assigned to 8 weeks of a daily intake of 33 g of experimental fats from either partially hydrogenated soy oil containing 20 g of TFA, 12 g of fish oil with approximately 4 g of n-3 PUFA and 21 g of control fat, or 33 g of control fat. The experimental fats were incorporated into bakery products. Plasma lipids, blood pressure, heart rate variability (HRV), arterial dilatory capacity, compliance, and distensibility were recorded before and after intervention and at follow-up 12 weeks after the intervention. Results: High-density lipoprotein cholesterol (HDL-C) decreased in the TFA group and triglycerides and mean arterial blood pressure decreased in the n-3 PUFA group compared to the control group. HRV, arterial dilatory capacity, compliance, and distensibility were unchanged. Conclusion: The results indicate that the association between coronary heart disease risk and intake of TFA and n-3 PUFA relates only modestly to changes in traditional risk markers. Sponsorship: Danish Medical Research Council (Grant no. 22-01-0390), Center of Advanced Food Research (Copenhagen, Denmark) (Grant no. KVL-R-2001-107), the Danish Heart Association (Grant no. 99-2-3-45-22748), Novozymes (Bagsvaerd, Denmark), Aarhus Olie (Aarhus, Denmark), and from private sources. The experimental fats were provided by Pronova Biocare (Aalesund, Norway) and Aarhus Olie (Aarhus, Denmark).
Published: 2004

154. 40(th) EASD Annual Meeting of the European Association for the Study of Diabetes : Munich, Germany, 5-9 September 2004

Author: S. Artigas, A V Dreval, Mark I. McCarthy, C Watson, Peter H. Bennett, M Quint, Y Ikeda, E Alpert, F Schiele, H Sekihara, Erik Gylfe, P Lowe, J Kuhlmann, Alain Golay, V Longo, Shahidul Alam Khan Akm., L G Mantovani, M Zawodniak-Szalapska, G Winkler, T Harrity, L Virág, U Johne, Kuo S-W., Linda C Tapsell, J Rodriguez, Michel Komajda, K Kankova, Carole A. Cull, M Sporna, E Estilles, U Ribel, M C Spruce, E Buzzigoli, T Prazak, J K McLaughlin, M K Lingohr, M Lim, F Calara, A Siebenhofer, G Meregalli, Roberto Anichini, A D Baron, R Kurashvili, P C Butler, G I Fantus, T. E. De Gooyer, Park Y-M., R. Walther, S Heinrich, Agnieszka Zawiejska, S Mukherjee, Nikolaos Papanas, G Wong, Ian D. Caterson, David M. Maahs, Shuichi Kaneko, Alexandra E. Butler, Francisco Javier Ampudia-Blasco, O N Kong, Attali J-R., C A Hedman, K Oshinyemi, Nicolle Müller, I C Cranston, N Okumus, M V Vlaiculescu, Balasubramanian Ravikumar, W W Cheatham, K Mukasa, K B Biswas, Annunziata Lapolla, Phil McEwan, G Mader, Gilles Chassot, Dragi Anevski, Werner A. Scherbaum, M Donath, C Hesselmann, R A Gandhi, David E. Moller, Ezio Bonifacio, C Garcia, V Ifandi, P Hornnes, Nieuwenhoven Fav., C Puech, S Pérez-Del-Pulgar, Kim S-R., G Hines, C Rubio Terrés, Michael Gaster, N. Hosszufalusi, A Scholze, Andrew A. Young, Stavros Liatis, F Hariri, S Tan, Paul Valensi, Allan E. Karlsen, J Kim, E. Moberg, J Kaiser, L Berman, G Nelson, A Altkrüger, P Kothare, D B Cook, S Doran, G. van Dijk, Shahnaz Shahinfar, Kim C-S., P Stahl, M Manousaki, S Sigrist, S K Lim, M. P. Stern, A Guberti, C Rezzani, J McKenney, Karl Thomaseth, Sofia Carlsson, M Julia, R Brillante, I Rubesova, T Darkow, E Matsumoto, Wendy M. Macfarlane, M Di Martino, G Bardini, Rossella Menghini, D Duhot, E Farcasiu, Annalisa Natalicchio, I Lindner, J Buvat, Christian L. Brand, Harry Dorchy, Iwona Pietrzak, Z T Luo, P Home, M Ekelund, Jesper Gromada, Kristine Færch, F Piarulli, H Kim, R Mentel, Zsuzsanna K. Zsengellér, Dullaart Rpf., Anton Luger, Thomas A. Pearson, V Manicardi, P Rösen, Feng Y-M., R Morganti, Lars Hansen, Demuth H-U., Haruo Kasai, A Shostak, Rudi Steffensen, G Taylor, Markolf Hanefeld, C Santini, E Hamaguchi, Roberto Miccoli, F Storms, M Cooper, Y Lee, Allison E. Aiello, P Smith, T Suehiro, K Treece, M Waluś, Timothy A Welborn, Simone Baltrusch, E Kontela, S Chai, J Crean, H Yokoyama, Johan G. Eriksson, Rafael Hernández Hernández, J Rodríguez-Saldaña, M P Tornero, G Formoso, D. Lovell, E Bingham, A Mylonakis, M Manteghetti, D Fedele, Antonio Martín-Duce, Ralph A. DeFronzo, D Salcedo, Kurt Højlund, Antonio Petrone, Sheu Whh., C Gutierrez, Flavia Pricci, S Kurita, Z G Abbas, M M Benedetti, Philippe A. Halban, Daniel J. Cox, O Ljungkvist, Justine Davies, J Palsgaard, Lars Sjöström, E Bosi, L Janin-Manificat, W. F. Kelly, M. Fernandez, E Colak, O V Mulyarchik, B Kronshage, F Lang, M Erfurth, Takashi Kadowaki, N Jendrike, U Walter, J Wishart, Y. Neye, D Kim, N Furuhashi, M Barsotti, D Florow, L Ke, L Borgquist, N C Jackson, Ffolliott M. Fisher, V Baskar, K Yoshioka, Bryan A. Wolf, G Chabrier, R Skoumal, Livio Luzi, H Kose, I Pharisien, B. Klein, H Winiarska, M C Johnson, L Griffiths, Nonna Kravchun, C Combe, Baptist Gallwitz, J Zdychova, L Skorda, Jorma Ilonen, W Gao, I N Steen, A Terrinoni, P D Ambery, W Kern, C M Kusminski, Cho M-H., Paolo Pozzilli, Louise G. Grunnet, E Schönle, David R Matthews, Robert W. Taylor, Y Cohen, Kim H-S., M P Eccles, N B Tutuncu, D McDowell, Richard M. Bergenstal, K Takamatsu, T Steiner, Jaan Palgi, Valdemar Grill, N Niculescu, G Federici, S Lehto, P. M. McKeigue, M Barone, Michael E. Trautmann, S Smirnov, J Mannion, M Eto, C Rousseau, M Conti, C S Ernest, Antonio Ceriello, D H Schweitzer, Jung E-D., Andreas Festa, Avijit Lahiri, A Shepelkevich, A Murro, A Kollmann, Jonathan R.S. Arch, R Landgraf, Son H-Y., I Engelsberger, E Agardh, S Rodríguez-Mulero, P J Kraml, K Lee, D. F. Du Toit, E Kim, G Fadini, Williams Ajk., Philip Home, M B Antcieferov, C Perlemoine, D Perrea, Song X-L., D Ruggieri, Krister Bokvist, Heidi Sørensen, Bilbao, G Yoshino, J P Taylor, Shen H-M., S M Furier, R Urquhart, J Wohlgelernter, Jianping Weng, T. Baba, Q Hong, C Silva, Castaigne J-P., M Felaco, X X Zhang, M Jaroň, Milla Rosengård-Bärlund, J G Papp, Toshio Miyata, Lervang H-H., Park M-K., I Kinalska, A Long, Oomen Phn., N Kogawa, Ippolita Patrizia Patera, S. Karadeniz, Dinesh Selvarajah, D S Chung, A Wensaas, Richard Imrich, M Recasens, J Ruxer, O Buchea, E Wilpart, S P Stepanenko, Le Ttd., H Ohgawara, Mariaconsuelo Valentini, A Mondok, M Peltonen, Marianne O. Larsen, K Chatzianagnostou, Agneta Ståhle, A L Ferrari, L Bordier, F Maingrette, A Matsuda, G Vukomanovic, Jakob D. Wikstrom, T Yamakita, E Gorostiaga, J Jin, B Gopalan, Heinz Drexel, S Hewitt, Rury R. Holman, C Dieterle, T L Ruchti, N Asatiani, M Sidira, A Iezzi, A J Sommerfield, D Châtenet, M L Olsen, R Bergemann, C Koehler, T L Kuraeva, B Balas, Christian Berne, E Santos-Mazo, G Smith, A Siejka, R Kožnarová, A Mattina, S Sheikh, A Adomeit, M Rasmussen, J. Fagerudd, N Busciantella Ricci, Nuria Vilarrasa, E Hammar, T L Thoms, L Aydın, Ron G. Rosenfeld, A Nikolajuk, R Gos, C L Morgan, H L Yu, D Dheelchand, S Ramrath, N Boudriga, Jerome I. Rotter, C Jahannault, W M Weston, Folke Lindgärde, M Hertlova, D Knight, A Monroy-Mayorga, E Pardini, A Chamson-Reig, B Franke, Janie McCluskey, Joseph Bryan, C Nikolopoulou, Christie M. Ballantyne, Fausto Santeusanio, L Pegoraro, M Lee, A Klimenko, S Jaiveer, K. Pettersson-Fernholm, Michael A. Nauck, A Ekbom-Schnell, G Deferrari, Riccardo Schiaffini, S. Pampanelli, Khan Aka., David Hopkins, Maija Wessman, M Kamarinos, Noh J-H., O Ebisui, K McCarroll, Jeppe Sturis, Peter Nowotny, N Gorbenko, Åke Sjöholm, David G. Maggs, A E Halseth, B Cresci, A A Ortiz-Gress, A Korakovouni, O Matejkova, C E Mogensen, C J Lin, Ramon Gomis, H Seaman, C Granier, Yang C-H., F Assah, O Sanchez, Fausto Machicao, Peter G. Morris, Alberto Ortiz, A Giardinelli, D Bracaglia, A Gonzalo, S Pavlatos, Andreas Lechner, F Canovic, L Sjolind, Allan Vaag, Birgitte Bruun Nielsen, David A. Ziegler, Vito Lampasona, R Gershoni-Baruch, A. Dei Cas, H Renz, E Mena, Matthew Waltham, Kim D-M., H Levanen, D D Mick, Valentina Alexandrovna Peterkova, E Meskhishvili, Sarah Nutland, R Bustani, John R. Lindsay, M Christoforidou, A Abicht, E Harno, K Cyganek, A Fitchet, S Neelotpol, P Nikishin, P Serradas, J Hinrichsen, M Halvorson, M Chovatia, B Voet, Jinny Willis, E Parretti, M Haslbeck, M Wellard, L Teng, Julio Wainstein, J S Fischer, K. Lalic, D Roggenland, I Gich, R Anwar, Maurizio Cassader, D Serota, X J Li, R J Schotzinger, Vilmundur Gudnason, Björn Zethelius, S A Wootton, W Andrzejewski, R Rezsohazy, R Gao, T Klimentova, T Mazurek, I Bruckner, C Dohrmann, R E James, G daSilva Xavier, Kim S-Y., A Dorca, Stuart J. Pocock, Terri J. Allen, I Giovos, P B Parab, N H Andersen, P Fotinakis, Miriam Cnop, H Lee, Norbert Tennagels, Omorodola I. Abatan, F Ailett, I. Lager, D Manzella, H Hut, Larry A. Distiller, G Lip, Lim S-K., Rong Zhang, T Tsuno, Steen Knudsen, M. Bajardi, Manuel Benito, Dai Sugimoto, Melvin J. Prince, D W Dunstan, D Rankins, K A Majali, G Ozansoy, Isabella Russo, S Uçak, G Annuzzi, R Talar-Wojnarowska, K Lange, S Neugebauer-Baba, Campbell H. Thompson, Eric Renard, P. D. Mountjoy, Z Morrison, Elizabeth A. Davis, Franco Cavallo, C Corvaja, R Antuña, Craig John Currie, H Linnebjerg, He Y-L., A J Palmer, Mariola R. Chacón, H Malinska, M. Jones, R Lichnovská, K Mandes, Paolo Tessari, T Mokhort, A Laina, H. L. Y. Chan, I Schmidt, R Banks, Richard G. IJzerman, L Ksinantova, G Setti, H Vaudry, A Gallo, V Spallone, Chen J-W., Thomas Danne, A Chong, M Hallschmid, S Aczel, S Hulme, N Islam, M Hosoi, P M Ternan, P Di Bartolo, N Bishara, T Shibasaki, Martin A. Osterhoff, Im S-S., M Jecht, T Hamaguchi, S Mattera, K Ways, Elizabeth Northam, U Rajala, Reinhard W. Holl, L Yang, S Panaiotopoulos, K Horvath, R Kluge, Thora B. Bodvarsdottir, Y Dong, Irene Alemanno, C McDougall, Reimar W. Thomsen, M Campbell, W Rabl, John Öhrvik, Yuichiro Yamada, Paola Ungaro, W Benzer, Mike Sampson, Roberto Trevisan, R G Radu, Aas A-M., P E Lobo, Ricardo Scott, S M Son, Josephine M. Forbes, T A Hillier, K L Wyne, Louis L. Nguyen, J Farmer, M H Tan, Kwon H-S., J Yang, L Sandvik, Franco Folli, A K Jenum, M Nguyen, W Pratipanawatr, A L Frederiksen, Rebecca Smith, Lee H-J., A Schäfer, C Manuelli, G S Denver, T Vukovich, B Maceira, K Matsumoto, K. Chokkalingam, Nurcan Üçeyler, P Modi, Timothy M. Morgan, S Mertens, B M Singh, Michaela Riedl, K Iso, C Cucurullo, G. F. Bottazzo, M Calvani, K Hur, J Wetzels, Kazuhiro Takahashi, Y Aso, H Stammer, M G Masding, Fitsum Guebre-Egziabher, J L González-Sánchez, L Armstrong, Alberto Maran, Peter G.F. Swift, S S Popovic, J Starczynski, E Vitacolonna, Luigi Laviola, R W Gelling, Marina Cardellini, D Barilla, Rosa de Diego Martínez, W H Landschulz, Anne Mette Rosenfalck, R K Wong, Kevin E. Schneider, K Peros, Giuseppe Nanni, F Zhang, I Rákóczi, T Iburi, M Nakhjavani, X Q Zhang, S Tournis, Per Lav Madsen, Graham A. Hitman, A. Tura, K Laubner, N D Kostic, Lawrence M. Dolan, R. Sinha Roy, J A Wagner, J. Tuomilehto, J Hauptman, M Abdel-Ghany, D Lacombe, Toralph Ruge, Johannes A Maassen, Triantafyllos Didangelos, K Sasaki, I Argüelles, Klaus Levin, C Popow, Emanuel Christ, R Chetty, L Baillet-Blanco, Jo-Ann Salmon, T Mine, James L. Trevaskis, I Franke, J Gorski, E A Andrianova, A Dayan, A Caballero, Aleksandra Gilis-Januszewska, M Yasujima, Z Kasalová, C.D.A. Stehouwer, F. K. Gorus, G A Nichols, A Glowania, David P. Strachan, P Fredlund, N. F. da Silva, P Reboldi, M Sausbier, K H Groenier, G Stuccio, N Guttman, K R Ahmed, A D Ristic, T Kapellen, J Coutcher, Aldo V. Greco, Oswald Wagner, A Zagayko, Maria Alevizaki, B B Zhang, W F Ferris, Jenny Fredriksson, Lois Jovanovic, J Hänninen, R De Giglio, Kazuo Yagui, O Potterat, P Hamliton, R E Scranton, B Mankovsky, A Stylianou, B Fellström, Abdel-Wahab Yha., M Kitagawa, Katherine L. Baldock, F R Johnson, F Baigts, S D'Addato, F J Sanz, A Mistry, S D Wise, T Pratipanawatr, U R Fölsch, James R.C. Parkinson, Claudia Sommer, C Park, F E Griffiths, M L Martí, R Demirtunc, S Taniguchi, J Lundkvist, T Siegmund, Juan Sztajzel, C Dienesch, F Baumgartner, L Scalone, T M Mckolanis, K Otake, Ullrik Pedersen-Bjergaard, T M Vriesendorp, Michael B. Wheeler, Henry Schmitt, Peter Hovind, S Lange, Stephane Roze, L. Van Gaal, B Klaproth, Anthony E. Civitarese, D Eckland, A Dagar, D F Hopkins, Kari Stefansson, C Gonzalez-Yanes, B Meyboom-de Jong, D. J. Betteridge, K Buhling, M Crepaldi, Ana M. Wägner, L Renna, L Volpe, R McBride, V Corbo, E O Brennesvik, R P Hayes, R Abdollahnia, G Viviani, C F Liew, Francisco Pérez-Bravo, Jeffrey Baron, Brian M. Frier, H H Samira, D Szentendrei, K. J. Schjoedt, W K Waldhäusl, D Gniuli, D Zou, G Tschank, V Urbančič, A L Nolan, Albertini J-P., J Malcomson, M Larbig, C Cheyssac, K Aurich, C M Kesson, S Heller, Maija E. Miettinen, R F Luco, Adrian J. Cameron, Luigi Mattiello, Z. Metelko, X E Zhang, M Parramón, I. G. Obrosova, J Fruchart, M Ilic, Björn Eliasson, Gilles Chatellier, M A Martín, D M Kendall, Holger Luthman, V F Varillas, D Maccubbin, Jang S-A., Amalia Gastaldelli, E Salzsieder, P. de Mol, A Yoshida, H D Lindner, D Gostiljac, M Just, Pan C-Y., J M Fujitaki, G Eiermann, K Bergenheim, A D Frick, A Agacdiken, K Varytimiadis, K Cseh, D A Jackson, S Calderari, Dena G. Hernandez, H M Liebich, K Min, F. de Zegher, Bernd Kulzer, K Han, Ulrich A. Müller, D Marrero, H Hatakeyama, René Koopman, Doo H-K., Petr Wohl, P. Sharp, P Forder, Thor Aspelund, N Meneveau, R M Schmülling, R Aubert, Thom Sam., H Youshikawa, M Ankelo, D Bowden, I Kelly, Frédéric Fumeron, M Sartini, Robert S. Sherwin, L Varadhan, A Criscimanna, John Betteridge, V Jelic, M Bartnik, N Lemke, B Ursø, A Bertoldo, A M Owona, H Okochi, L Pérez-Tamajó, S L Monfre, Daniel Brandhorst, K T Legg, Andries J. Smit, Veronica Sancho, Masashi Hirai, C Klein, Paul J. Thornalley, A Chaidaroglou, K Miura, B Zinman, O M Dvoynishnikova, J Plank, Jan Bolinder, C Lush, B Rubi, R Pozzilli, M Bashir, S A Shtandel, F Mosca, A Naskalska, Josef Vcelak, U Sausbier, P Cavaiani, T U Baehring, Michele Solimena, P Formisano, M Rastaldi, Bernard Thorens, J Ruzzin, E Arbit, M. Hori, Torkel B. Brismar, E Soltes Rak, A Filo, P Heinke, Matthew P. Coghlan, M Masotti, I Perevozskaya, K Ahn, I Moules, K Van Dyck, I Goldstein, Z Mathe, G Z Zhao, S Fajardo, J Taylor, S Chrul, J C Pareja, D Hadjidakis, A J Scheen, N Siddiqua, D C Cavan, R Grella, Krabbe S, H J Rochlitz, A E Hinkkanen, W Wilpshaar, Richard Stevens, M Dreyer, S Hara, X Wang, Melania Manco, D Gillen, Magalie A. Ravier, Olli Simell, John C. Lawrence, Kohnert K-D., Agardh C-D., A Berghold, L Kristensen, Grant Sfa., N Gursoy, Leif Groop, N Freemantle, Anja Schweizer, L Pala, Legros J-J., C. Di Pietro, N. Yamamoto, J Magyar, B Nikolovski, H Ikeda, D Lee, Bruce A. Buckingham, A O Wollitzer, I Kennedy, C Ernest, Neville H. McClenaghan, S Tanaka, Asimina Mitrakou, T Heinze, W Kerner, Moeenaldeen Al-Sayed, Charles Thivolet, L Klaff, A Miconi, Cristina Valeri, J. O. Christensen, K. Ekberg, A Jardine, T Endo, X Zhang, D F Child, A Kienitz, D K Seidel, H. Tada, Sylvie Abouna, Cyrus Cooper, Catherine R Chittleborough, Roberta Assaloni, S Corbi, A K Bose, K Ozawa, C Ahn, K A Deans, G Jackowski, Martin Gibson, Patrick McElduff, O A Mojiminiyi, Manuel Serrano-Ríos, O Dupuy, A L Davydov, Iwar Klimes, Sten-Anders Ivarsson, N Ichino, R Matsutomo, E R Smith, A Stefanovska, B Dehmel, K Koniavitou, E Agascioglu, M Hatazaki, J. M. Gibson, T Yada, P Ribaux, M Rupnik, K Fridell, G Scutaru, L Chugunova, Henrietta Mulnier, A Kendereski, H Lehnert, C Billi, M Sobczak, Francisco M-Mj., L K Archibald, S Sukumvanich, David B. Dunger, I. Benke, G Yillar, N Stingemore, J. M. Boavida, Y Shi, Jimmy D. Bell, L Bozzetto, Andrew J. Ahmann, E Jebens, J Keiding, Elena Henkel, Mark Fineman, J F McRae, Carol Forsblom, S Martemucci, Lourdes Ibáñez, P G Prieto, L Ringholm Nielsen, S Pratas, B von Stritzky, Julio Rosenstock, Lee K-W., J Stocks, L J Strow, I Samarguliani, L Wennekes, R Cheung, Abhishek Nag, Roberto Gambino, Y Suleymanoglu, E Murphy, T T Durck, M F Peyrot, Y Unno, Alexander Mayorov, Eleuterio Ferrannini, D. C. Rao, D Neely, H Karunajeewa, J Palmisano, Julia B. Lewis, M Ravid, G Pons, E Junca, P Vexiau, S Sailesh, D K Miloslavskiy, O N Bondarenko, U Smith, S Torri, Constantine Tsigos, Cesario Bianchi, Mattia Locatelli, D Jaquet, Virpi Lindi, M Moroi, M E Tushuizen, P Pelicci, R Scognamiglio, Pal Pacher, S M Thyssen, A Péterfalvi, Y Ho, S Guntram, L Romics, T Nakagami, Clive S. Cockram, Irina Kowalska, K Brodbeck, Gojka Roglic, J. Dörig, Lise Tarnow, Therese Tillin, A López-Alba, Martin Krššák, Moses Elisaf, S Hata, D P Snoeck, D Schmoll, O V Udovichenko, A Scaramuzza, J Paul, John H. Fuller, Nicholas Katsilambros, Michele Muggeo, Pia Ekbom, Piero Marchetti, V Melki, C Bailleau, H Stavrianos, A D'Errico, Geremia B. Bolli, Amabile Maier, Kelter A-R., Anders Green, Q J Morélis, Steffen Thiel, C Watkins, R C Cheung, A Clark, Elvira Fioriti, N Ari, Nam J-Y., Y Cottin, L G Krinelke, H Al Mohammedi, Simon C. Fleming, C Jones, Z Kerényi, Ahn Y-H., Meile M-J., P Nánási, M Graner, V Canonico, Gangnerau M-N., Hugh R. Taylor, Giovanni Sartore, A. Dejgaard, Carol Kelley, S. Ali, Stéphane Dalle, Jeffrey S. Gonzalez, Elena Šeböková, Alexander Beck, Ingo B. Leibiger, M Rosu, C Pencea, Werner Waldhäusl, Kaltenbacher M-C., R Butzer, S Thore, Adam G. Tabak, Angelo Avogaro, E Standi, Boris Kovatchev, O Bradescu, Patrizia Dentelli, A Fujita, C Verri, R Chlup, Prasad Ydm., S V Hörsten, van der Merwe M-T., D Hilliard, W Klein, D Worthley, M Udvardy, Berit R. Jensen, A D'Avanzo, J Monaghan, K P Yeo, Guivarch P-H., B Bauduceau, D Weghuber, P Tatti, J Ybarra, S Gwozdziewiczová, E Gasparini, B Saltin, Charlotte Granhall, Howard Leventhal, R Marin, M Tumiati, Cicero Afg., L Csémy, B Berger, S Mikros, D Dall'Asta, M Shahmanesh, Y G Vasiljev, F Potthoff, H S Randeva, G De Berardis, J O Logan, K Warncke, P Uitterlinden, E Rehring, K Gilmore, K Shankhdhar, V V Bojko, M Vahatalo, E A Korolyova, D Wiemann, P G Lankisch, D Hendrie, F Galtier, M Rybarczyk, Gisela Dahlquist, N N Rudovich, G Stein, A Liebl, F Tan, A Westerlund, S Gronemann, I Franklin, Jonathan A. Prince, Peter Arner, E Skliros, T. Sparre, M Vigas, Maddalena Trombetta, L. Bjerre Knudsen, A C Sima, I Dubroca, Alastair Gray, I Weets, R Ferraresi, Schauer Ujw., E. Leinonen, S Corazza, Jonathan Levy, P K Prakash, R Guzder, S. Barnhill, John Blangero, J Herreros, G. de Vries, Cheng Ptw., A Macías-Batista, K. Capito, R Thomas, G Thomas, G Boemi, Lotte Pietraszek, Pierre Fontaine, I Holme, J Smedegaard, C A Harrop, U Helwig, B Levy, A P Gribben, Hiroto Furuta, P Beckett, S Giannini, Ruth L. Coleman, Eva Fernqvist-Forbes, N Cugnardey, A Dumas, Jane Pinaire, S E Hofer, D Shimono, Erik H. Serné, Alain D. Baron, C Battista, M Tanen, M Klementova, V Adams, J Komorowski, Antonio Nicolucci, E. C. Burns, H Sydall, M G Fanara, M G Giovannitti, N Okabayashi, Magdalena Szurkowska, I A Eroshkin, M. I. J. Uusitupa, D Ma, C C Dieguez, J Sutcliffe-Goulden, A V Gaddi, Michał Arabski, Serge Halimi, Wendy C. Burns, S Seclén, H Sugano, A Vinterby, K Backx, L F Diaconu, Fernando Gomez-Peralta, O'Harte Fpm., G Lepeniotis, D Laune, H Kvasničková, N H Wallén, G Boner, G Cieślik, Robert Hermann, Paul Q. Thomas, Y Kumon, Maria Maiello, M Atkins, Kenneth A. Earle, Guowang Xu, H Y Bae, I Reynisdottir, D Perez, D E Cannon, P Fabietti, I Geronooz, J Østergaard, K J Jeitler, S Skourtis, A Zambanini, A Saemann, K Kuboki, Helen L. Lutgers, A C Thai, Arne Melander, D Pinkowski, S G Straub, S A Wolfe-Coote, A Totora, Hayley Dickinson, A Lindenmair, A Ginis, I Faturos, F Van Eylen, C Huard, F Fu, N Wang, B M Rasmussen, Angela Napoli, L Granato, Markus M. Lerch, M. Frandsen, A Lyras, Lawrence S. Phillips, J Mabley, R Goldschmeding, B K Kilhovd, W Liu, Greg Poffenberger, P Cipriano, Anna Maria Charlotte K Lindqvist, A Matsuzawa, J Wang, T Yu, M S Pepper, Lena M. Thorn, Y Sakamoto, Blanche Schwappach, Anna L. Gloyn, P Dupraz, A. M. Schmidt, M Psallas, V Tsirimbis, Carl D. Langefeld, D Sass, H E Scholtz, M. Cailleau, Lauren Julia Brown, K Phillips, M. Iezzi, C Jayawarna, Eleni Anastasiou, R Lobmann, R Lundershausen, H Fujiwara, H R Nan, I C Smith, I A Karpova, A Navazio, S Kang, T. Hansen, T Watanabe, Gang Hu, Malik Rja., T Kennedy-Martin, Ulla M Smidt, J.M. Dekker, A A Fisher, H Liu, R E Pratley, B Sun, C Fledelius, J F Raposo, R Langham, Ahn S-Y., B R Waterhouse, Shaoping Deng, W Ricart, S V Melnichenko, Henrike Sell, M. Tomalino, N Takeda, Paola Massucco, A Harlan, W Henrich, C D Byrne, R Junik, Khalid Hussain, D Pop Gorceva, Torben Hansen, C Ritterath, K Ogawa, D V Phan, Bradley S Metcalf, Robb E. Moses, Juan P. Frias, Hitoshi Ishii, C Brisard, P Wolkow, H H Maurer, Ingo Rustenbeck, Juris J. Meier, Octavian Savu, S D Lin, V B Bregovski, A Fox, S Cicala, M. Koenen, L Vignati, O de Divitiis, Constantin Ionescu-Tirgoviste, Kilian Rittig, J Song, Riccardo Candido, F Cohen-Boulakia, U Shankhdhar, P Jahan, Antonio Tiengo, E Liepinsh, C Álvarez, Sigurd Lenzen, James E. Foley, A. del Arco, M A Maitan, U Mollenhauer, M A Na, A Beha, B Aicher, Gabriele Meyer, E Sommariva, J Åman, B Gmeinhart, Theede A-M., Bjorn Bolinder, Giulio Ceolotto, S Sbrana, F Biarnes, Damini Dey, Barbara Thorand, H H Klein, Juliana C.N. Chan, L Piconi, S Gudbjornsdottir, E Bobbioni-Harsch, Joanna Polanska, Cristian Serafinceanu, S Gambardella, Olivier Huber, J D Brunzell, T Nagasawa, Loretta Vileikyte, A Szocs, H Löwel, Z Lin, Anders Juul, H Tsuneki, L Sauriol, G Siebert, Marit E. Jørgensen, K Matthies, Brian D. Green, P Jurowski, Z Gao, M Furuya, Silvia Manfrini, Efthymios Motakis, E Souvatzoglou, Qian Y-Z., Byun S-H., T Nguyen, M R Anwar, Geltrude Mingrone, B. Idzior-Walus, Mamas A. Mamas, G Scholl-Schilling, C Bittner, A E Raptis, L Laghi, Murray Stewart, M Orel, Janet M. Bryson, Chris P Gale, Åke Lernmark, A B Escorza-Domínguez, C Fuentes, A S Einarsdottir, M O Akinsola, P Maisonneuve, K S Leong, B Missler, Simona Frontoni, Con Tsalamandris, Flavio Francini, D Rouiller, B.E. de Galan, Girolama Alessandra Marfia, M Fioravanti, A E Weber, M Nargis, P Nowacki, Y Chen, B Larsen, Ruth E. Gimeno, A Wojciechowska Luźniak, R Meisterfeld, Limei Liu, A Ohashi, T Pieber, Bin Yao, G A Herman, M Pallini, R E Honkanen, H Nyblom, R Humbrecht, Takashi Tsuboi, Pinar Topsever, Monika Kellerer, J Woods, Steen Stender, C Kapitza, Sherwyn Schwartz, M Taskiran, N Hashimoto, A Jabin Gustafsson, K C Silva, I Nitz, S Dzienis-Straczkowska, H Elouil, L Sommese, Patricia A. McKinney, S Ogawa, E Carr, I Porchay, Robyn G Langham, Bruno Vergès, K H Usadel, S Sivakumaran, H M Hearnshaw, José Luis Santos, R Ishibashi, F Huq, Lebrethon M-C., Z Leshchenko, Elisabetta Torlone, A Saltiki, A Plaschke, Wolfgang Koenig, D. D'Avola, C L McTernan, Haruhiko Osawa, G Cazzetta, S J Lee, S Al Jebely, Hoogma Rpl., A Shimizu, Tack Cjj., Itamar Raz, Adrian Vella, P. B. M. de Andrade, Aguiar Lgk., Wolfgang E. Schmidt, T Wolthers, W. Gatling, T S Purewal, Philipp E. Scherer, E Heller, Stephan Gielen, Jo S-R., Monica Vedovato, L Nosek, R Ryder, G Giassakis, A Cretti, A Wajngot, T Waldhör, S Zoric, H C Lee, L Pulkkinen, L Lacey, I Tomescu, R S Patarrao, X C Liu, Lauge Schäffer, Jarkko Ustinov, Anders Thorell, K Kangawa, P J Wood, T Yeoh, Jo Satoh, G Psaras, Volker Burkart, Karin Schindler, Dario Pitocco, M Kolopp, Gabriela Roman, E Renström, M Goldberg, M Beregszaszi, S Senkel, M F Meyer, María Luisa Villanueva-Peñacarrillo, Elena Beltramo, J E Snaar, A Hayward, C Abreu-Padí, S E Vielwerth, R Krones, C C Stanciu, M Izquierdo, I Elbing, R Astorga, V I Bouloukos, P Zabielski, S Precerutti, Kang S-Y., K L Pratt, Joost H-G., Matthew L. Iorio, Naomi S. Levitt, Yutaka Seino, Peter A. Gottlieb, I Ehrén, Y Matsuyama, Jean Claude Mbanya, S Parveen, R Purvis, A Cappadona, V V Afanasyeva, K Löbner, S Del Guerra, V Valle, Victor A. Gault, M Tan, M M Budge, I Ioannidis, M Ishizawa, Castelli, D Nowak, János Rigó, Claudia Bleck, E Degli Esposti, C Tortul, M Klebach, E Schvarcz, L Pauline, D Krusova, J Csillag, Park K-G., Kaichi Kida, P Bialasiewicz, S Seeliger, B A Williams, P Mistry, T Nicer, N Ahmed, H Ohashi, Peter Achenbach, van Hessen Paw., Jesús Cancelas, E Cizmarova, D Takahashi, K Tanaka, A M Rojo Llobet, Q Chen, A J Garber, P Halban, B Brunmair, A Barthel, M R Katdare, Dominique Laude, M E Pavkov, C Leach, C M Breschi, S Okada, B Balent, Ornella Massa, Ravi K. Garg, van Loon Ljc., K Obuobie, W Moll, L Varanese, V Smejkalova, B Royer, M Roth, M Belfiglio, Chae M-K., I Kimura, Casper G. Schalkwijk, Hans-Jürgen Hedrich, Heinsbroek Acm., Jared Rutter, Ken Williams, K O Lee, A N Symeonidis, J B Brown, M Pfeuffer, W. C. Knowler, A N Barsukov, A Woods, T W Kim, Khalid Bak., Andrea Mari, David J. Pettitt, Y Tomioka, J Hochman, Mireia Mora Porta, K Pagava, Krzysztof C. Lewandowski, J Taton, T Yasaka, M Tyrberg, M Sargin, P Szenthe, R Sicree, C Abreu-Padín, Magdalena Lundgren, J Radziuk, Eili Tranheim Kase, W L Goudzwaard, Yanbing Li, E Ramos, H Monastiriotis, J Cur, S Pu, Lakka H-M., Park J-S., Fabrizio Barbetti, S. Ciani, Pavel Flachs, N C Correia, Angela A. Rivellese, F Ebara, Nicolas Musi, H van Ahlen, Helen M. Colhoun, Anna Czech, Steen B. Pedersen, N Jost, N Da Silva, J Nóvoa, Y Onuma, Anna Körner, D Michael, M Straczkowski, I. Truyen, R. Schmid, Søren Paaske Johnsen, H Koenig, B. Miranda-Palma, E Wasén, F Pervin, P Cavallo-Perin, M Niessen, E P Shavrikova, P Tarkun, M Vavlukis, H Gang, O Sánchez-Vilar, C Dalla Man, S. M. Haffner, R Eggertsen, L Pucci, F T Fiedorek, E I Mainas, H Holdass, Peter R. Shepherd, A Banu, R Lichiardopol, Lee M-K., N Zaytseva, B Gumbiner, Strotmann H-J., L Y Baranova, A Kissebah, A Pezzatini, I Kharroubi, N A Thornberry, A J Buckley, B Pontiggia, T Chen, M Ikebukuro, J Leach, C Frost, J Han, H Hashizume, G Poulsen, H Eckert, Paul Zimmet, Lee J-H., Peter E. Heding, S A Ross, Nils J. Færgeman, Giuseppe Penno, T Varbanova, H S Shon, Z Canturk, S Akahane, Noel G. Morgan, A Sener, Seronde M-E., P K Thaware, W Insull, S Forst, K E White, Christoffer Hedetoft, M R Holland, K Okamoto, F Yamagata, Markus Lassila, N Tanaka, Kristian Midthjell, R Isoaho, D A Legemate, M Motzkau, Y H Kang, A P Nescheret, J Boiko, Hemmo A. Drexhage, Gagik R Galstyan, Naim Shehadeh, Henning Beck-Nielsen, T I Justesen, O Kalter-Leibovici, T Vahlberg, M Mozdzan, G Siegert, V Dumenil, Katsumi Eguchi, J A Vazquez, Sally Hollis, H Haberland, A Souvatzoglou, M L Gavete, K. S. Nair, Katharine R. Owen, C Garcia-Rodenas, Gerald I. Shulman, Peters, H A Parkes, Y S Kim, Mats Rönnback, Isabelle Leclerc, Marie-Hélène Giroix, I D Caterson, S G Rønn, L Scionti, Silvia Pinach, Hans Christian Reinecker, Kathryn Noyes, P McTernan, C Y Tan, N. Lalic, Richard D. Carr, M. Toman, D Papazoglou, Domenico Bosco, G Lübben, F. Costanza, Tania Maffucci, Jonas Nygren, Ole Lander Svendsen, Kristin Taylor, Ugo Boggi, A Kayserilioglu, Gudmar Thorleifsson, G J Dunseath, B. Balkau, T Zieher, M Schwarts, A. R. Cobitz, P de Pablos, I Recas, Carlos Lorenzo, Pamela J. Shaw, Y Cho, C Münscher, Anna Maestroni, Milan Zdravkovic, Gerstl E-M., L L Xiu, Yoshitaka Kajimoto, A Deb, F Osaki, G Kirilov, B Staffler, Běla Bendlová, M Rebelatto, Frank Reimann, B Ø Christoffersen, S Durant, Fruchart J-C., Ravinder J. Singh, V Connolly, Q Zhang, Alexandra Kautzky-Willer, Francesco Andreozzi, N Talai Rad, V Loyková, Trampisch H-J., Stefano Menini, F. Rodríguez de Fonseca, W Hanif, H J Böhles, S Fotiadis, Philippe André, T Kubiak, T Friede, J Murin, Erwin Schleicher, Adam Garrow, M Takahashi, P Valensi, S Pappas, E Lim, D N Staroverova, C Tadgell, C Dessapt, Brittany A. Rasmussen, L Cheung, L E Porter, R Ishihara, J Ravisy, T Inukai, Christine Winzer, E M Özer, Nish Chaturvedi, Ana Chico, Ville-Petteri Mäkinen, N Bellili, Howlader, Jansson P-A., A Martella, Jan Frystyk, T Kakko, K Hershon, S Hoashi, B Nag, C Chabrolle, B Leatherdale, J. Lu, Stephen Colagiuri, C Brendel, Michael Roden, Peter Clausen, A L Macdonald, R A DeFronzo, F A VanNieuwenhoven, Sirkka Keinänen-Kiukaanniemi, Jonas J-C., Hobbs Fdr., H Uchino, S Del Prato, E Polakowska, L Shankhdhar, Hedrich H-J., S Rodriguez-Cuenca, A Schwarzmaier-D'Assie, K Fukuda, Robert H. Eckel, A. Hilding, A Rutscher, D Kerr, Junhua Li, C. Köhler, A Banaszak, Peter Lönnroth, P Wulff, M A Gruzov, Fariba Shojaee-Moradie, S Matsumoto, J P New, N Barthes, M G Leyck Dieken, Mian Li, O Ivanova, G Papalia, K A Nilsson, Parving H-H., P Merz, M. A. Schweitzer, A Morris, P. Andersen, O Tschritter, M Fitch, V Fejfarova, Elisabeth Bernroider, E C Kiawi, M de Kerdanet, Claus B. Juhl, C Taxvig, Kerstin Berntorp, Birgitte Andersen, C Coscelli, C Ktori, Norio Abiru, R Mössner, Hiroko Hirooka, Jens J. Holst, Q Dan, H F Gu, C Gimpelewicz, A Oeckinghaus, B DelBuono, J Kuroda, P Kastner, Clemens Fürnsinn, H Usui, R Mosqueda-Garcia, P De Meyts, K Yamagami, M Tóth, V Leite, H Alberti, M Meneses, Gregory Collier, E G Starostina, C Delaporte, Agostino Consoli, François Alhenc-Gelas, S Tonstad, G Schuler, J Juszczyk, G Hoefle, M Wilken, Mark A. Yorek, Volker Lodwig, G Edwards, E Catellani, Ewa Wender-Ozegowska, Kurt Hoffmann, I Markova, Steven J. Hunter, A Venkataraman, Bente Klarlund Pedersen, A Kengne, Ken K. Ong, L A Calo, M Huckova, Y Morikawa, Pierina Richiusa, D Giuffrida, Sonia Brichard, Giuseppe Paolisso, Andrew T. Hattersley, M Vankova, Jon G. Mabley, G Salvidio, M Montolio, X Yang, Barbara N. Campaigne, Ralph R. Turner, Andrew J.M. Boulton, P Johnston, S Berchtold, Roger Corder, D Ledwig, M. Di Mauro, Houweling, J H Bowker, J Hilsted, Shin D-W., B Kiran, E Esteve, A J Cameron, S Spera, Kathrin Maedler, S Tsujii, J. H. DeVries, S Cailleaux, M Walter, A Florén, R Lehtonen, Davis Tme., N Patsourakos, J. Marco, G Lepore, L Morcillo, J Burén, Paul Smits, Juraj Koska, Chung J-H., Katherine S. Elliott, Bong Soo Cha, Christian Weyer, F Montorsi, L G Søndergaard, H Horie, M Herten, N Aghamohamadzadeh, L A Scrocchi, A Oyarzún, Rutten Geh., K Yamada, M Zivkovic, R S Beifort, Lee I-K., S D Bouman, H Böhles, Dwight E. Matthews, Orville G. Kolterman, Bidda Rolin, I Shymanskyy, P Thomakos, John Strand, A Virkamäki, C M Clark, Y Tokumasa, Odusanya Pot., Joel F. Habener, Abel López-Bermejo, Anders Tengholm, D Argon, Lajoix A-D., N Aki, L Kessler, N Takahashi, A Zmysłowska, Kåre I. Birkeland, D Soon, Lars Køber, M Vaňková, Lee W-C., F Payeras-Más, C Lau, E Ramos Lopez, L Lukic, A Suehiro, Stephan J-A., I Estalella, Jørgen Jensen, Liaqat Ali, V S Dimitrijevic Sreckovic, V P Maximova, G Salietti, M Ding, Yuksel Altuntas, Chung K-D., F Kaufman, Ludwig Wagner, C Calvi, D X Gram, David G. Bruce, K Sprenger, M Schweers, D Kush, D B Panagiotakos, J. M. Currie, Luis Castaño, Dag E. Undlien, D. Haspel, A Suzuki, J Cederholm, S Garcia, L Cynober, P O'Hare, A Tsur, Christine Zimmermann, M Faimalie, V V Poltorak, S Alba, Axel Haupt, J Varga, Tetsuya Yamada, M Shoji, R H Lloyd-Mostyn, Péter Pusztai, Doney Asf., M N Gangnerau, I Shimizu, Henrik Ortsäter, F Liska, Mandeep Bajaj, H Sarui, Michal Andel, S Chiheb, Diane Barker, A. van Tol, Brigitte Reusens, I Anafaroglu, Giel Nijpels, F Bongardt, K Taxildaris, M. 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Ward, René Gross, S K Althouse, S Woodhead, Emmanuel Cosson, T Rönnemaa, T M Filiz, Jerzy Loba, S Sha, Klas Malmberg, Carlo Pesce, Jung D-S., I Majdrakova, Thomas Forst, M Schütt, Hierl F-X., A M Efanov, I Verschraegen, H B Jensen-Holm, D Deckhut, Frida Leonetti, N Selzle, G Cassetti, Jose L Mesa, G Morrison, Christian-Heinz Anderwald, C Kliebe-Frisch, J H Moffitt, P Lahm, Hiroshi Ishiguro, E. Borgo, A Keech, L Quagliaro, Thomas O. Ola, Henquin J-C., W J Sluiter, F Leprêtre, Kalliopi Kotsa, Peter Karl Jacobsen, E Sanchez-Largo, E Gaia, Daniel R. Witte, Sidsel Graff-Iversen, G Seebohm, E Cacès, R G Khalifah, W Wilson, Avan Aihie Sayer, C Raffaitin, J Gumprecht, T Lehmann, E Migliaccio, R Urrutia, E Shelestova, A V Chervonsky, Stefan Mustafa, Y Hisano, Erik A. Richter, J Miettinen, K M Gottesdiener, Gangyi Yang, D Chevenne, A Girach, P Samuelsson, Taskinen M-R., D Ciociaro, D Aydemir, Yang S-B., P De, Dongliang Zhuang, Nathan R. 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Thornalley, P, Babaei jadidi, R, Karachalias, N, Kupich, C, Ahmed, N, Fowler, A, Baker, A, Starczynski, J, O'Hare, P, Szepietowska, B, Szelachowska, M, Puch, U, Glebocka, A, Quinn, D, Mcternan, C, Bonser, R, Idzior walus, B, Woźniakiewicz, E, Dimitriou, K, Apostolou, O, Kontela, E, Devangelio, E, Gould, E, Serri, O, Roussin, A, Buithieu, J, Mamputu, J, Renier, G, Giordanetti, S, De Amici, E, Poggi, G, Turpini, C, Fratino, P, Garzaniti, A, Banu, I, Paries, J, Roman, G, Negrean, M, Bala, C, Nita, C, Kistorp, C, Gustafsson, F, Chong, A, Lip, G, Galatius, S, Ari, N, Sahilli, M, Ceylan isık, A, Ozansoy, G, Karasu yilmaz, C, Matteucci, E, Rosada, J, Pallini, M, Evangelista, I, Cassetti, G, Giusti, C, Giampietro, O, Capaldo, B, Galderisi, M, Cicala, S, Turco, A, Imbroinise, A, Nosso, G, D'Errico, A, De Divitiis, O, Klimontov, V, Korolyova, E, Jeltova, L, Bondar, I, Tarkun, I, Arslan, B, Canturk, Z, Tarkun, P, Agacdiken, A, Komsuoglu, B, Méneveau, N, Pierre justin, E, Alsayed, M, Sabbah, R, Paulin, S, Marcu, S, Tauveron, I, Zimmermann, C, Schiele, F, Seronde, M, Vautrin, P, Lusson, J, Thieblot, P, Bernard, Y, Mistry, A, Pye, M, Peovska, I, Maksimovic Pavlovic, J, Vavlukis, M, Pop Gorceva, D, Bosevski, M, Scognamiglio, R, Negut, C, De Kreutzenberg, S, Madonna, R, De Caterina, R, Willerson, J, Geng, Y, Vahsen, S, Ledwig, D, Ramrath, S, Frantz, S, Schmidt, I, Calvillo, L, Dienesch, C, Elbing, I, Bischoff, H, Ertl, G, Bauersachs, J, Davydov, A, Mkrtum'Yan, A, Baranova, L, Ikeda, Y, Suehiro, T, Osaki, F, Ota, K, Arii, K, Kumon, Y, Hashimoto, K, Doney, A, Morris, A, Palmer, C, Byun, S, Doo, H, Pagnin, E, Calo, L, Fadini, G, Kubaszek, A, Chai, S, Chai, Q, Rasmussen, L, Ledet, T, Wogensen, L, Lengyel, C, Varró, A, Virág, L, Magyar, J, Bíró, T, Jost, N, Skoumal, R, Nánási, P, Tóth, M, Horkay, F, Papp, J, Zacharopoulou, O, Athanaselis, S, Tsokos, N, Doupis, J, Psallas, M, Cokkinos, D, Pavlatos, S, Liatis, S, Akhobadze, T, Dzneladze, L, Samarguliani, I, Taskiran, M, Rasmussen, V, Jensen, G, Fisher, A, Petrovsky, N, Srikusalanukul, W, Budge, M, Trifunovic zamaklar, D, Zivkovic, M, Jelic, V, Vukomanovic, G, Ristic, A, Seferovic, P, Costa, J, Duarte, S, Manley, S, Sailesh, S, Venkataraman, A, Haider, Y, Groza, I, Oprean, M, Ardelean, A, Morosanu, A, Darkow, T, Vanderplas, A, Mamas, M, Mcelduff, P, Burns, J, Edwards, R, Fitchet, A, Young, R, Gibson, J, Lichiardopol, R, Niculescu, N, Totora, A, Pencea, C, Tomescu, I, Cinteza, M, Manicardi, V, Coscelli, C, Navazio, A, Catellani, E, Michelini, M, Dall'Asta, D, Guberti, A, Piazza, A, Gasparini, E, Pantaleoni, M, Guiducci, U, Manari, A, Sejil, S, Janand delenne, B, Avierinos, J, Habib, G, Labastie, N, Vague, P, Lassmann vague, V, Luźniak, P, Tatoń, J, Wojciechowska Luźniak, A, Zairis, M, Lyras, A, Patsourakos, N, Tsirimbis, V, Foussas, S, Lupón, J, Urrutia, A, Herreros, J, González, B, Coll, R, Altimir, S, Prats, M, Valle, V, Abreu padí, C, Rábago, G, Ivanova, L, Brasacchio, D, Harno, E, Keenan, A, Li, H, Lu, Z, Ke, L, Liu, H, Jeong, I, Chae, M, Choi, M, Yoo, H, Kim, C, Yun, M, Na, M, Kang, Y, Kong, O, Son, S, Kim, I, Tanaka, N, Hosoi, M, Matsuyama, Y, Fukumoto, M, Yamakita, T, Yoshioka, K, Ishii, T, Sato, T, Fujii, S, Aoki, T, Shibata, T, Mizutani, N, Suzuki, J, Fowelin, J, Samuelsson, P, Brandrup wogsen, G, Okumura, K, Tokmakova, A, Staroverova, D, Antcieferov, M, Shutichina, I, Kuntchevich, G, Vriesendorp, T, Morélis, Q, Legemate, D, Schaper, F, Mainas, E, Gkioulmpasanis, I, Panagiotou, I, Vassilikos, G, Skorda, L, Sidira, M, Christoforidou, M, Alaveras, A, Artikis, V, Evdemon, E, Lechleitner, M, Koch, T, Ebenbichler, C, Sturm, W, Moretti, L, Moruzzo, D, Boldrini, E, Pandolfo, C, Kameyama, M, Iwasa, R, Cho, M, Nam, J, Huh, K, Kaplar, M, Paragh, G, Erdei, A, Csongradi, E, Garai, I, Varga, J, Galuska, L, Udvardy, M, Higa, M, Kaneko, Y, Hiroi, N, Koziarska, D, Nowacki, P, Majkowska, L, Luzniak, P, Wojciechowska luźniak, A, Tushuizen, M, Nieuwland, R, Snoeck, D, Sturk, A, Diamant, M, Aguiar, L, Bahia, L, Villela, N, Laflor, C, Conde, C, Bottino, D, Dorigo, D, Bouskela, E, Pu, S, Luo, Z, Lam, K, Dan, Q, Xu, A, Shen, J, Cheng, K, Xu, J, Thamer, C, Stefan, N, Haap, M, Heller, E, Tschritter, O, De Prado, A, Ortiz, A, Ybarra, J, Gich, I, Pou, J, Ehren, M, Roggenland, D, Reinsen, B, Klein, H, Rittig, K, Stock, J, Kocher, B, Balletshofer, B, Shon, H, Chung, D, Nakatani, Y, Matsuhisa, M, Kaneto, H, Hatazaki, M, Yoshiuchi, K, Katakami, N, Kawamori, D, Ohtoshi, K, Sakamoto, K, Matsuoka, T, Ozawa, K, Ogawa, S, Hori, M, Yamasaki, Y, Zitouni, K, Harry, D, Nourooz zadeh, J, Earle, K, Olesen, P, Franco, L, Corvaja, C, Semplicini, A, Ceylan işık, A, Arı, N, Rösen, P, Lee, I, Park, K, Jung, E, Shin, D, Jo, S, Obuobie, K, Prakash, P, Hanna, F, Lazarus, J, Varadhan, L, Gurushankar, J, James, D, Sheikh, S, Gaede, P, Zou, D, Vilarrasa, N, Perez maraver, M, Mena, E, Perez, D, Setti, G, Buckingham, R, Urbančič, V, Stefanovska, A, Bernjak, A, Ažman juvan, K, Kocijančič, A, Glowania, A, Filters, T, Fosmark, D, Torjesen, P, Kilhovd, B, Berg, T, Sandvik, L, Hanssen, K, Mentink, C, Donchenko, G, Stepanenko, S, Maingrette, F, Deng, H, Lindenmair, A, Freudenthaler, A, Baumgartner parzer, S, Nizheradze, K, Khoruzhenko, A, Tronko, N, Sheu, W, Ou, H, Shen, H, Lin, T, Wu, H, Yang, C, Mogylnytska, L, Schmoelzer, I, Davies, J, Band, M, Struthers, A, Prázný, M, Škrha, J, Kasalová, Z, Neelotpol, S, Jahan, P, Kauschke, S, Harrop, C, Schäfer, A, Widder, J, Eigenthaler, M, Walter, U, Uchimura, I, Ikebukuro, M, Kaibara, M, Hirata, M, Helal, R, Pervin, F, Yang, X, Jansson, P, Nagaev, I, Jack, M, Carvalho, E, Sunnerhagen, K, Cam, M, Cushman, S, Smith, U, Creely, S, Farmer, J, Gustafson, B, Kusminski, C, Krusinova, E, Wohl, P, Klementova, M, Lanska, V, Mcdougall, C, Kelly, I, Abbas, Z, Lutale, J, Archibald, L, Karunajeewa, H, Stingemore, N, Stuccio, G, Mcgechie, D, Muller, L, Hak, E, Goudzwaard, W, Montorsi, F, Homering, M, Sprenger, K, Goldstein, I, Asnaghi, V, Ferrari, G, Rastaldi, M, Gabellini, D, Dell'Antonio, G, Maestroni, A, Ruggieri, D, Luzi, L, Piemonti, L, Zerbini, G, Anafaroglu, I, Tutuncu, N, Sultana, M, Siddiqua, N, Iwasaki, T, Nakajima, A, Yoneda, M, Mukasa, K, Tanaka, S, and Sekihara, H
Subjects: 0303 health sciences, medicine.medical_specialty, business.industry, EASD, Endocrinology, Diabetes and Metabolism, Human physiology, medicine.disease, 03 medical and health sciences, 0302 clinical medicine, Diabetes mellitus, Family medicine, Internal Medicine, Medicine, business, 030217 neurology & neurosurgery, 030304 developmental biology
Published: 2004

155. [The impact of sugar on health]

Author: Chistian, Mølgaard, Niels Lyhne, Andersen, Vibeke, Barkholt, Niels, Grunnet, Kjeld, Hermansen, Bente, Nyvad, Bente Klarlund, Pedersen, Anne, Raben, Louise C, Rosenberg, and Steen, Stender
Subjects: Dietary Sucrose, Risk Factors, Denmark, Nutritional Requirements, Humans, Obesity, Energy Intake
Published: 2003

156. [Hormone replacement therapy and ischemic heart disease--a dangerous cocktail? Changes in the collected evidence]

Author: Pernille Danneskiold, Lassen and Steen, Stender
Subjects: Evidence-Based Medicine, Hormone Replacement Therapy, Estrogen Replacement Therapy, Myocardial Ischemia, Animals, Humans, Coronary Disease, Coronary Artery Disease
Published: 2003

157. Reduced endothelial nitric oxide synthase activity and concentration in umbilical veins from maternal cigarette smokers

Author: Line R. Walker, Malene R. Andersen, and Steen Stender
Subjects: medicine.medical_specialty, Endocrinology, Endothelial nitric oxide synthase, business.industry, Internal medicine, medicine, business, Cardiology and Cardiovascular Medicine
Published: 2003
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158. Sukkers sundhedsmæssige betydning

Author: Christian Mølgaard, Niels Lyhne Andersen, Vibeke Barkholt, Niels Grunnet, Kjeld Hermansen, Bente Nyvad, Bente Klarlund Pedersen, Anne Raben, Rosenberg, Louise C., and Steen Stender
Published: 2003

159. Effect of protein and methionine intakes on plasma homocysteine concentrations: a 6-mo randomized controlled trial in overweight subjects

Author: Steen Stender, Arne Astrup, Jørn Dyerberg, A. R. Skov, Søren Toubro, and Nikolaj Haulrik
Subjects: Vitamin, Adult, Male, medicine.medical_specialty, Low protein, Homocysteine, Adolescent, Medicine (miscellaneous), Overweight, Excretion, chemistry.chemical_compound, Folic Acid, Methionine, Internal medicine, medicine, Diet, Protein-Restricted, Humans, Obesity, Completely randomized design, Analysis of Variance, Nutrition and Dietetics, business.industry, Weight change, Body Weight, Middle Aged, Diet, Vitamin B 12, Endocrinology, chemistry, Female, Dietary Proteins, medicine.symptom, business
Abstract: Background: A high plasma homocysteine concentration is an independent risk factor for cardiovascular disease. Homocysteine concentrations are thought to be raised by high protein and methionine intakes. Objective: Our goal was to investigate the effects of high and low protein and methionine intakes on homocysteine in overweight subjects. Design: Sixty-five overweight subjects were randomly assigned to a 6-mo intervention with a low-protein, low-methionine diet (LP: 12% of total energy, 1.4 g methionine/d; n = 25); a high-protein, high-methionine diet (HP: 22% of total energy, 2.7 g methionine/d; n = 25), both of which had similar fat contents (30% of total energy); or a control diet with an intermediate protein content (n = 15). All food was self-selected at a shop at the department. Protein intake was increased in the HP group mainly through lean meat and low-fat dairy products. Dietary compliance was evaluated by urinary nitrogen excretion. Results: Homocysteine concentrations did not change significantly in the LP or control groups but were 25% lower in the HP group (NS). Homocysteine concentrations after the 3-mo intervention were inversely associated with vitamin B-12 intake and with weight change (by multivariate analysis performed for all subjects), but not with methionine or protein intake. Sixty-nine percent of the variation could be explained by baseline homocysteine (P < 0.001), 2% by vitamin B-12 (P = 0.02), and another 2% by weight change (P = 0.06). The plasma homocysteine concentration after 6 mo was associated only with baseline homocysteine (P
Published: 2002

160. Comparison of a homogeneous assay with a precipitation method for the measurement of HDL cholesterol in diabetic patients

Author: Quan Truong, M. Frandsen, Bo Dinesen, Steen Stender, and Tonny Jensen
Subjects: Adult, medicine.medical_specialty, Apolipoprotein B, Endocrinology, Diabetes and Metabolism, Serum albumin, chemistry.chemical_compound, Reference Values, Diabetes mellitus, Internal medicine, Internal Medicine, medicine, Chemical Precipitation, Humans, Phosphotungstic acid, Serum Albumin, Triglycerides, Aged, Advanced and Specialized Nursing, Aged, 80 and over, Glycated Hemoglobin, Cholesterol Measurement, biology, Triglyceride, business.industry, Cholesterol, Patient Selection, Cholesterol, HDL, Reproducibility of Results, Cholesterol, LDL, Middle Aged, medicine.disease, Endocrinology, Diabetes Mellitus, Type 1, chemistry, Diabetes Mellitus, Type 2, Metabolic control analysis, biology.protein, Regression Analysis, lipids (amino acids, peptides, and proteins), business
Abstract: OBJECTIVE—To compare direct-measured HDL cholesterol with HDL cholesterol measured by a precipitation method. RESEARCH DESIGN AND METHODS—We compared a homogeneous assay for direct HDL cholesterol analysis with the phosphotungstic acid magnesium chloride precipitation method in 55 type 1 diabetic patients, 70 type 2 diabetic patients, and 82 nondiabetic normal control subjects with plasma triglyceride levels RESULTS—HbA1c, blood glucose, serum albumin, serum bilirubin, or triglyceride did not influence the differences of the two HDL cholesterol measurements. Because we have previously shown HDL cholesterol isolated by phosphotungstic acid precipitation to be lower than that by ultracentrifugation, the positive bias found in this study was expected. It seems that the direct HDL cholesterol assay reacts with apolipoprotein (apo) B-containing lipoproteins in the fraction with a density of >1.063; these apo B-containing lipoproteins are suggested to be coprecipitated with the phosphotungstic acid method. We also measured LDL cholesterol directly by a LDL cholesterol plus method and found no significant differences between this method and LDL cholesterol calculated from Friedewald’s formula. CONCLUSIONS—Direct homogeneous assay for HDL cholesterol determination in diabetic patients seems not to exhibit a negative bias, in contrast to the precipitation method, when compared with the ultracentrifugation method. In addition, the direct assay saves time and is not influenced by type of diabetes or degree of metabolic control.
Published: 2002

161. Monitoring of dietary changes by telephone interviews: results from Denmark

Author: Lotte Holm, Jóhanna Haraldsdóttir, Jytte Halkjær, Steen Stender, and Arne Astrup
Subjects: Adult, Male, Food industry, Adolescent, Denmark, Population, Medicine (miscellaneous), Food habits, Diet Surveys, Danish, Interviews as Topic, Environmental health, Telephone number, Medicine, Animals, Humans, education, Aged, Aged, 80 and over, education.field_of_study, Nutrition and Dietetics, business.industry, Public Health, Environmental and Occupational Health, Feeding Behavior, Middle Aged, language.human_language, Whole milk, language, %22">Fish , Female, business
Abstract: Gentofte Hospital, Niels Andersensvej 65, DK-2900Hellerup, Copenhagen, DenmarkSubmitted 5 January 2001: Accepted 26 March 2001AbstractObjectives: To monitor trends in Danish food habits with respect to selected keyelements, from 1995 to 1998, and to evaluate the appropriateness of the methoddeveloped for that purpose.Design and method: Two cross-sectional population surveys, in 1995 and 1998. Datacollection by computer-assisted telephone interviews including 10 food-frequencyquestions,questionsontypeoffatusedon sandwichesanddrinkingmilk,andcheckquestions on theprevious day. Reproducibility was tested ina subgroup –n ‹222ƒinthe 1998 survey.Setting: The Danish Nutrition Council initiated the survey.Subjects: Men and women aged 15–90 years, 1007 in 1995 and 1024 in 1998. Samplesof private telephone numbers were drawn from regional telephone registers,geographically stratified. Participation rates were 62%.Results: Significant differences were observed between 1995 and 1998, some of thesein accordance with dietary guidelines (decreased use of whole milk and fat spreadon bread, increased use of skimmed milk, salad vegetables, rice/pasta and fish).Other changes were opposite to dietary guidelines (increased use of soft butter,decreased use of soft margarine and low-fat spreads, potatoes, and fresh fruit).Differences in average consumption frequency amounted to 4–13%. Several resultswere confirmed by comparison with other data, and the reproducibility of themethod was acceptable. Data were suitable for analysis of food use patterns, arelevant approach when assessing food habits in a lifestyle context.Conclusions: The changes observed illustrate the dynamics of food habits and theneed for frequent monitoring. This simple telephone method may be a valuable toolfor that purpose, as a supplement to national dietary surveys, also in a public healthcontext.KeywordsFood habitsDietary changesTelephone interviewsFood frequenciesPopulation surveillanceDietary habits are in a constant process of change. Publichealth authorities, food authorities, the food industry andothers working within this field need up-to-date data onthe dietary habits of the population as a basis for theirinitiatives. Up until now the availability of such data hasbeen limited in Denmark. Only two national dietarysurveys have been carried out, in 1985 and 1995
Published: 2002

162. No Association between Loss-of-Function Mutations in filaggrin and Diabetes, Cardiovascular Disease, and All-Cause Mortality

Author: Torben Jørgensen, Lise Lotte N. Husemoen, Pal B. Szecsi, Steen Stender, Michael Meldgaard, Allan Linneberg, Tea Skaaby, Jeanne D. Johansen, and Jacob P. Thyssen
Subjects: Adult, Male, medicine.medical_specialty, Pathology, Adolescent, Science, Denmark, Population, Myocardial Ischemia, Disease, Type 2 diabetes, Filaggrin Proteins, Young Adult, Intermediate Filament Proteins, Internal medicine, Diabetes mellitus, Humans, Medicine, Genetic Predisposition to Disease, Mortality, Risk factor, education, Stroke, Aged, education.field_of_study, Multidisciplinary, business.industry, Hazard ratio, Middle Aged, medicine.disease, Diabetes Mellitus, Type 2, Mutation, Female, business, Research Article, Filaggrin
Abstract: BackgroundCommon loss-of-function mutations in the filaggrin gene (FLG) are a major predisposing risk factor for atopic disease due to reduced epidermal filaggrin protein levels. We previously observed an association between these mutations and type 2 diabetes and hypothesized that an inherited impairment of skin barrier functions could facilitate low-grade inflammation and hence increase the risk of diabetes and cardiovascular disease. We examined the association between loss-of-function mutations in FLG and diabetes, stroke, ischemic heart disease (IHD), and all-cause mortality in the general population.MethodsThe R501X and 2282del4 loss-of function mutations in FLG were genotyped in four Danish study populations including a total of 13373 adults aged 15-77 years. Two of the studies also genotyped the R2447X mutation. By linkage to Danish national central registers we obtained information for all participants on dates of diagnoses of diabetes, stroke, and IHD, as well as all-cause mortality. Data were analyzed by Cox proportional hazard models and combined by fixed effect meta-analyses.ResultsIn meta-analyses combining the results from the four individual studies, carriage of loss-of-function mutations in FLG was not associated with incident diabetes (hazard ratio (HR) (95% confidence intervals (CI)) = 0.95 (0.73, 1.23), stroke (HR (95% CI) = 1.27 (0.97, 1.65), ischemic heart disease (HR (95%CI) = 0.92 (0.71, 1.19), and all-cause mortality (HR (95%CI) = 1.02 (0.83, 1.25)). Similar results were obtained when including prevalent cases in logistic regression models.ConclusionOur results suggest that loss-of-function mutations in FLG are not associated with type 2 diabetes, cardiovascular disease, and all-cause mortality. However, larger studies with longer follow-up are needed to exclude any associations.
Published: 2013

163. Endothelial nitric oxide synthase activity in aorta of normocholesterolemic rabbits: regional variation and the effect of estrogen

Author: Malene R. Andersen and Steen Stender
Subjects: Aortic arch, Male, medicine.medical_specialty, Time Factors, Endothelium, Nitric Oxide Synthase Type III, Physiology, Ovariectomy, Hypercholesterolemia, Cell Culture Techniques, Aorta, Thoracic, Enos, Physiology (medical), medicine.artery, Internal medicine, medicine, Thoracic aorta, Animals, Aorta, Abdominal, Cells, Cultured, Geographic difference, Aorta, Analysis of Variance, biology, Estradiol, business.industry, Abdominal aorta, biology.organism_classification, Immunohistochemistry, Nitric oxide synthase, Endocrinology, medicine.anatomical_structure, biology.protein, Female, Endothelium, Vascular, Rabbits, Nitric Oxide Synthase, Cardiology and Cardiovascular Medicine, business
Abstract: Objective: Several animal studies suggest that nitric oxide (NO) produced by the endothelium attenuates arterial cholesterol accumulation. In the present study we have asked the following questions: (1) is the regional variation in aortic cholesterol accumulation in hypercholesterolemic rabbits preceded by a regional variation in endothelial NO synthase (eNOS) activity in normocholesterolemic rabbits, and (2) is the antiatherogenic effect of estrogen in hypercholesterolemic rabbits preceded by a higher eNOS activity in normocholesterolemic rabbits. Methods: The eNOS activity was determined by conversion of 14C-l-arginine to 14C-l-citrulline in freshly isolated endothelial cells of aorta in normocholesterolemic rabbits. In the regional variation study, 16 male and eight female rabbits were used. In the estrogen study, ovariectomized female rabbits were subcutaneously injected three times weekly with either 17β-estradiol ( n =7) or vehicle ( n =7) for 18 weeks. Results: In the regional variation study, the atherosclerosis prone aortic arch showed a significant lower eNOS activity than the more resistant abdominal aorta in both male ( P
Published: 2000

164. The direct antiatherogenic effect of estrogen is present, absent, or reversed, depending on the state of the arterial endothelium. A time course study in cholesterol-clamped rabbits

Author: Heidi L. Andersen, Steen Stender, Pernille Holm, Elisabeth Erhardtsen, and Malene R. Andersen
Subjects: medicine.medical_specialty, Endothelium, Nitric Oxide Synthase Type III, medicine.drug_class, Arteriosclerosis, Ovariectomy, Aorta, Thoracic, Placebo, Nitric Oxide, Nitric oxide, Cholesterol, Dietary, chemistry.chemical_compound, Physiology (medical), medicine.artery, Internal medicine, medicine, Thoracic aorta, Animals, Nitrites, Aorta, Analysis of Variance, Nitrates, Estradiol, Cholesterol, business.industry, medicine.anatomical_structure, Endocrinology, chemistry, Estrogen, Ovariectomized rat, Diet, Atherogenic, Female, Endothelium, Vascular, Rabbits, Nitric Oxide Synthase, Cardiology and Cardiovascular Medicine, business
Abstract: Background —This study further investigated the relationship between estrogen, arterial endothelium, and nitric oxide (NO) in cholesterol-clamped rabbits. Methods and Results —Rabbits were ovariectomized, balloon-injured in the thoracic aorta, and grouped to receive cholesterol-enriched chow together with either 17β-estradiol or vehicle for 1, 2, 4, or 8 weeks. In the undamaged aorta, cholesterol accumulation of the placebo rabbits was significantly increased from week 4 to 8 ( P P P P P G -nitro- l -arginine methyl ester, an inhibitor of NO production. Conclusions —The direct antiatherogenic effect of estrogen was present, absent, or reversed, depending on the state of the arterial endothelium, and preceded by a transient increase in NO production followed by a reduced mononuclear-endothelial cell binding.
Published: 1999

165. Effect of cyclosporine on arterial balloon injury lesions in cholesterol-clamped rabbits: T lymphocyte-mediated immune responses not involved in balloon injury-induced neointimal proliferation

Author: P. K. Holm, Birgit Fischer Hansen, Børge G. Nordestgaard, Henrik Ørbæk Andersen, and Steen Stender
Subjects: Neointima, Male, Pathology, medicine.medical_specialty, Arteriosclerosis, T-Lymphocytes, Vascular Cell Adhesion Molecule-1, Muscle, Smooth, Vascular, Catheterization, Endothelial activation, Immune system, Restenosis, medicine.artery, medicine, Animals, Aorta, business.industry, T lymphocyte, Ciclosporin, medicine.disease, Intercellular Adhesion Molecule-1, Surgery, Cholesterol, cardiovascular system, Balloon dilation, Cyclosporine, Endothelium, Vascular, Rabbits, Cardiology and Cardiovascular Medicine, business, Cell Division, medicine.drug
Abstract: Abstract —Restenosis after balloon dilatation of stenosed coronary arteries is a major clinical problem. Because T lymphocytes occur in neointima and because cyclosporine inhibits T-lymphocyte proliferation, we tested the hypothesis that cyclosporine would attenuate neointimal proliferation after balloon dilation injury. Rabbits with a balloon-injured aorta, randomized to cyclosporine in the human therapeutic range (n=13) or vehicle (n=14) were followed up for 5 weeks; as a control for the effect of cyclosporine, half the rabbits received in addition an aorta allograft. Rabbits were clamped at a human plasma cholesterol level of 5 to 7 mmol/L. Cyclosporine did not affect aorta cholesterol accumulation or neointimal proliferation in balloon-injured aortas; however, it attenuated both in transplanted aortas. Likewise, cyclosporine had no effect on endothelial cells at balloon-injured sites, but protected these cells in the transplanted aortas. Infiltration of smooth muscle cells, T lymphocytes, and macrophages were unaffected by cyclosporine in balloon-injured aortas; however, in transplanted aortas, cyclosporine reduced the relative number of T lymphocytes and macrophages but increased the relative number of smooth muscle cells. Finally, in balloon-injured aortas, cyclosporine did not affect expression of vascular adhesion molecule-1, intercellular adhesion molecule-1, or major histocompatibility complex II, but all these cellular activation markers were attenuated by cyclosporine in transplanted aortas. These results suggest that cyclosporine does not attenuate neointimal proliferation after balloon dilatation, and that T lymphocyte—mediated immune responses are not involved in neointimal proliferation after balloon dilatation.
Published: 1999

166. Atherogenesis and the Role of Lipoproteins

Author: Steen Stender
Subjects: Lipoproteins, LDL, Cholesterol, Biochemistry, Arteriosclerosis, Lipoproteins, Clinical Biochemistry, Humans, Arteries, General Medicine, Biology, Lipoproteins, HDL
Published: 1990

167. A partial estrogen receptor agonist with strong antiatherogenic properties without noticeable effect on reproductive tissue in cholesterol-fed female and male rabbits

Author: Birgitte Hjort Guldhammer, Michael Shalmi, Pernille Holm, Niels Korsgaard, Steen Stender, and Sven O. Skouby
Subjects: Agonist, Male, medicine.medical_specialty, Pyrrolidines, medicine.drug_class, Arteriosclerosis, Estrogen receptor, Biology, Cholesterol, Dietary, chemistry.chemical_compound, Internal medicine, Blood plasma, Testis, medicine, Animals, Ormeloxifene, Cholesterol, Uterus, Stereoisomerism, Arteries, medicine.disease, Immunohistochemistry, Endocrinology, chemistry, Receptors, Estrogen, Estrogen, Female, Rabbits, Cardiology and Cardiovascular Medicine, Levormeloxifene, medicine.drug
Abstract: Abstract Estrogen replacement therapy retards the development of cardiovascular disease and osteoporosis in postmenopausal women. However, long-term unopposed use increases the risk of cancer in endometrium and possibly in breast. The racemic compound ormeloxifene, widely used in India as an antifertility agent, is a partial estrogen receptor agonist with antiosteoporotic properties. The present study was undertaken to investigate the effect of the l -enantiomer (levormeloxifene) and the d -enantiomer ( d -ormeloxifene) on the development of atherosclerosis. In a short-term experiment (6 weeks), 4×10 ovariectomized female rabbits were fed a 0.25% cholesterol-enriched diet and the effect on plasma cholesterol levels was studied. In a long-term experiment (13 weeks), 4×15 ovariectomized female and 4×15 sham-operated male rabbits were maintained at a similar plasma cholesterol level of 25 mmol/L and the effect on undamaged and balloon-injured arterial wall was studied. In both experiments, the rabbits were treated with levormeloxifene, d -ormeloxifene, 17β-estradiol, or placebo, respectively. In the short-term experiment, levormeloxifene, in contrast to d -ormeloxifene, significantly reduced plasma cholesterol by 30% compared with the placebo group. In the long-term experiment, levormeloxifene, in contrast to d -ormeloxifene, significantly reduced atherosclerosis by 50% in the undamaged arterial wall of both female and male rabbits. Because these rabbits were cholesterol-clamped, the antiatherogenic effect was not mediated via plasma cholesterol lowering. Like estrogen, levormeloxifene did not inhibit atherosclerosis in the endothelium-denuded site of aorta. The antiatherogenic effects of levormeloxifene were thus similar to those of estrogen, but produced in the absence of any noticeable estrogenic effect on uterine or testicular tissue.
Published: 1997

168. Significant reduction of the antiatherogenic effect of estrogen by long-term inhibition of nitric oxide synthesis in cholesterol-clamped rabbits

Author: Pernille Holm, M Shalmi, N Korsgaard, Sven O. Skouby, P Hougaard, Steen Stender, and H L Andersen
Subjects: Nitroprusside, medicine.medical_specialty, Pyrrolidines, medicine.drug_class, Arteriosclerosis, Ovariectomy, Estrogen receptor, In Vitro Techniques, Nitric oxide, Catheterization, chemistry.chemical_compound, Internal medicine, medicine, Animals, Aorta, biology, Estradiol, Cholesterol, General Medicine, medicine.disease, Coronary Vessels, Acetylcholine, Nitric oxide synthase, Endocrinology, NG-Nitroarginine Methyl Ester, chemistry, Receptors, Estrogen, Estrogen, Ovariectomized rat, biology.protein, Female, Rabbits, Nitric Oxide Synthase, Levormeloxifene, medicine.drug, Research Article
Abstract: The purpose of this study was to investigate whether endothelium-derived nitric oxide (NO) is involved in the plasma lipid-independent antiatherogenic effect of estrogen and levormeloxifene, a partial estrogen receptor agonist. 85 rabbits were ovariectomized and balloon-injured in the middle thoracic aorta. The rabbits were fed a cholesterol-enriched diet supplemented with 17beta-estradiol, levormeloxifene, or placebo, either alone, or together with 160 microg/ml NG-nitro- -arginine methyl ester (-NAME), an NO synthase inhibitor, in their drinking water for 12 wk. Plasma cholesterol was maintained at 25-30 mmol/liter by individualized cholesterol feeding. In the undamaged aorta, the extent of atherosclerosis in the estrogen group was only one-third that in the placebo group. Simultaneous administration of -NAME, however, significantly reduced the antiatherogenic effect of estrogen (P < 0.01). There was no significant difference between the placebo group given -NAME and the group treated with placebo alone. At the previously endothelium-denuded site, estrogen had no effect on atherosclerosis development, whereas -NAME combined with estrogen significantly increased atherogenesis (P < 0.05). The effects of levormeloxifene were almost similar to those of estrogen. Active vascular concentrations of -NAME were demonstrated in an additional study, in which maximal aortic/coronary endothelium-dependent relaxation was significantly inhibited in rabbits given -NAME. Thus, in this study a considerable part of the plasma lipid-independent antiatherogenic effect of estrogen was mediated through its effect on endothelial NO in cholesterol-fed rabbits. The results for levormeloxifene suggest a common mechanism of action for estrogen and partial estrogen receptor agonists on atherogenesis.
Published: 1997

169. W16-P-063 The lipid-altering effects of INEGY-TM are consistent across gender, race, age, and baseline LDL-C levels

Author: D. Maccubbin, E. Velhi, J. Rotonda, Steen Stender, Yale B. Mitchel, Diane Tribble, Arvind Shah, and Leiv Ose
Subjects: medicine.medical_specialty, Race (biology), Endocrinology, business.industry, Internal medicine, Internal Medicine, Medicine, General Medicine, Cardiology and Cardiovascular Medicine, business, Baseline (configuration management)
Published: 2005

170. Effect of Weight Loss on the Severity of Psoriasis

Author: Claus Zachariae, Lone Skov, Nina Rica Wium Geiker, Arne Astrup, Peter Riis Hansen, Bente Krogsgaard Schaadt, Steen Stender, Robin Christensen, and Peter Østrup Jensen
Subjects: Adult, Male, medicine.medical_specialty, Dermatology, Overweight, Severity of Illness Index, Psoriasis Area and Severity Index, Weight loss, Psoriasis, Internal medicine, Weight Loss, medicine, Humans, Outpatient clinic, Obesity, Aged, Caloric Restriction, business.industry, Dermatology Life Quality Index, Middle Aged, medicine.disease, Quality of Life, Physical therapy, Female, medicine.symptom, business, Body mass index, Weight gain
Abstract: Psoriasis is associated with adiposity and weight gain increases the severity of psoriasis and the risk of incident psoriasis. Therefore, we aimed to measure the effect of weight reduction on the severity of psoriasis in obese patients with psoriasis.To assess the effect of weight reduction on the severity of psoriasis in overweight patients.Sixty obese patients with psoriasis from our dermatology outpatient clinic were enrolled in a prospective randomized clinical trial in which they were allocated to a control group or an intervention group.University hospital outpatient dermatology clinic.We included 60 of 69 eligible overweight patients with psoriasis (body mass index [calculated as weight in kilograms divided by height in meters squared], 27-40; aged 25-71 years).The intervention group received a low-energy diet (LED) (800-1000 kcal/d) for 8 weeks to induce weight loss, followed by 8 weeks of reintroduction of normal food intake, reaching 1200 kcal/d. The control group was instructed to continue eating ordinary healthy foods.Psoriasis Area and Severity Index (PASI) after 16 weeks, with Dermatology Life Quality Index (DLQI) as a secondary end point.The median PASI for all patients was 5.4 (interquartile range, 3.8-7.6) at baseline. At week 16, the mean body weight loss was 15.4 kg (95% CI, 12.3-18.5 kg; P.001) greater in the intervention group than in the control group. The corresponding mean differences in PASI and DLQI, also in favor of the LED group, were -2.0 (95% CI, 4.1 to -0.1; P = .06) and -2.0 (95% CI, -3.6 to -0.3; P = .02), respectively.Treatment with an LED showed a trend in favor of clinically important PASI improvement and a significant reduction in DLQI in overweight patients with psoriasis.clinicaltrials.gov Identifier: NCT01137188.
Published: 2013

171. Associate Editor

Author: Taek-jong Kwak, Can Naci Kocabaş, Yuichi Adachi, Angel Ferrer, Hemin Lee, Mustafa Erkoçoğlu, Pal B. Szecsi, Charlotte Edenius, Brittany L. White, Kameran Daham, Hanne Vestergaard, Jeung-Hoon Lee, Medea Imboden, Hans-Erik Claesson, Sven-Erik Dahlén, Susumu Nakae, Young Ho Lee, Linda Backman, Nicole Probst-Hensch, Åsa Brunnström, Juan Antonio Pagán, Tove Tingvall, Carlos H. Larramendi, A. Shiozawa, Hidenori Ichijo, Elisabeth Zemp, Toshiko Itazawa, Andreas J. Bircher, Yasunori Ito, Maria Kumlin, M. Hirotsu, Dae Suk Kim, Michael D. Kulis, Nikolaos Douladiris, Xiaolei Shi, Druck Reinhardt Druck Basel, Min-youl Chang, José Luis García-Abujeta, Chrysanthi Skevaki, Ángel Julio Huertas, Anette Bygum, N. Ono, Christian Schindler, Yoshie Okabe, Fatma Demirel, Barbro Dahlén, Özlem Kara, Rishu Guo, Anna James, Y.S. Adachi, Peter Schmid-Grendelmeier, Katsuko Sudo, Luis Navarro, Jack P. Davis, Celal Özcan, Brunello Wüthrich, Ersoy Civelek, Ayşegül Akan, Steen Stender, Dilek Azkur, Satz Mengensatzproduktion, Timothy H. Sanders, Dimitrios Mitsias, Eiko Takada, Irene Roumpedaki, K. Ikeda, Carmen Andreu, Chul-Hong Kim, Young-Ah Shin, Adrienne Yancey, Emine Vezir, Jerónimo Carnés, Ayşenur Kaya, Dong Hyun Kim, Nikolaos G. Papadopoulos, Tae-jin Yoon, Tayfur Giniş, M. Ángeles López-Matas, Savvas Savvatianos, T. Kusunoki, Masae Furuhata, A. Wesley Burks, Osamu Higuchi, Min Geol Lee, Hyunjoong Jee, Junichiro Mizuguchi, and M. Miwa
Subjects: Associate editor, Philosophy, Immunology, Immunology and Allergy, General Medicine, Theology
Published: 2013

172. Associate Editor Thilo Jakob

Author: Hans-Erik Claesson, Celal Özcan, M. Hirotsu, Dae Suk Kim, Tae-jin Yoon, Eiko Takada, Brunello Wüthrich, Can Naci Kocabaş, Emine Vezir, Carlos H. Larramendi, Chrysanthi Skevaki, Chul-Hong Kim, Dong Hyun Kim, Tove Tingvall, Satz Mengensatzproduktion, Masae Furuhata, Jeung-Hoon Lee, Young Ho Lee, Linda Backman, Ángel Julio Huertas, Min-youl Chang, Medea Imboden, Sven-Erik Dahlén, Susumu Nakae, Michael D. Kulis, Ersoy Civelek, K. Ikeda, Anette Bygum, José Luis García-Abujeta, Nikolaos Douladiris, Taek-jong Kwak, A. Shiozawa, Peter Schmid-Grendelmeier, Toshiko Itazawa, Timothy H. Sanders, Anna James, Andreas J. Bircher, Yasunori Ito, N. Ono, Barbro Dahlén, Jack P. Davis, Hemin Lee, Brittany L. White, Mustafa Erkoçoğlu, Ayşenur Kaya, Kameran Daham, Åsa Brunnström, Irene Roumpedaki, Osamu Higuchi, Christian Schindler, Hanne Vestergaard, Nikolaos G. Papadopoulos, Dimitrios Mitsias, Elisabeth Zemp, Y.S. Adachi, Nicole Probst-Hensch, Carmen Andreu, Hyunjoong Jee, Pal B. Szecsi, Charlotte Edenius, Tayfur Giniş, Xiaolei Shi, Druck Reinhardt Druck Basel, A. Wesley Burks, Katsuko Sudo, Luis Navarro, Maria Kumlin, Steen Stender, Min Geol Lee, Hidenori Ichijo, Junichiro Mizuguchi, M. Miwa, Angel Ferrer, Özlem Kara, Dilek Azkur, Jerónimo Carnés, Yoshie Okabe, Young-Ah Shin, Adrienne Yancey, Ayşegül Akan, Yuichi Adachi, M. Ángeles López-Matas, Savvas Savvatianos, Rishu Guo, T. Kusunoki, Fatma Demirel, and Juan Antonio Pagán
Subjects: Philosophy, Immunology, Immunology and Allergy, General Medicine, Classics
Published: 2013

173. Oxidation of plasma low-density lipoprotein accelerates its accumulation and degradation in the arterial wall in vivo

Author: Klaus Munkholm, Steen Stender, Børge G. Nordestgaard, Lars Nielsen, and Klaus Juul
Subjects: Tunica media, Male, medicine.medical_specialty, Aorta, Thoracic, chemistry.chemical_compound, In vivo, Physiology (medical), Internal medicine, medicine.artery, Blood plasma, medicine, Thoracic aorta, Animals, Humans, Tissue Distribution, Lagomorpha, biology, business.industry, Tunica intima, biology.organism_classification, Lipoproteins, LDL, medicine.anatomical_structure, Endocrinology, chemistry, Low-density lipoprotein, lipids (amino acids, peptides, and proteins), Rabbits, Cardiology and Cardiovascular Medicine, business, Tunica Intima, Tunica Media, Oxidation-Reduction, Artery
Abstract: Background The aim of the present study was to investigate whether oxidized LDL (ox-LDL) in the arterial intima could be derived from LDL already oxidized in plasma. Methods and Results Rabbits received an intravenous injection of 125 I-labeled normal LDL (N-LDL) mixed with 131 I-labeled LDL that had been mildly oxidized through exposure to Cu 2+ . The aortic accumulation of undegraded labeled LDL was expressed as plasma equivalents and calculated as radioactivity in the intima/inner media (cpm/cm 2 ) divided by the time-averaged concentration of radioactivity in plasma (cpm/nL): for the thoracic aorta, the accumulation of undegraded ox-LDL in the intima/inner media exceeded that of undegraded N-LDL by 286% (n=6, P P P r =.97, n=14, P 125 I-LDL labeled with 131 I-tyramine cellobiose was injected intravenously 24 hours before the aortic intima/inner media was removed: for the thoracic aorta, the accumulation of degradation products from ox-LDL (n=6) exceeded that from N-LDL (n=6) by 301% ( P Conclusions The present data suggest a novel mechanism: mildly oxidized LDL may circulate in plasma for a period sufficiently long to enter, accumulate, and be degraded in the arterial intima in preference to N-LDL.
Published: 1996

174. Preferential influx and decreased fractional loss of lipoprotein(a) in atherosclerotic compared with nonlesioned rabbit aorta

Author: Lars Bo Nielsen, Børge G. Nordestgaard, Steen Stender, and Matti Jauhiainen
Subjects: Tunica media, Male, medicine.medical_specialty, Arteriosclerosis, Models, Biological, Cholesterol, Dietary, Iodine Radioisotopes, chemistry.chemical_compound, Reference Values, Internal medicine, medicine.artery, medicine, Animals, Humans, Aorta, Analysis of Variance, biology, Cholesterol, General Medicine, Lipoprotein(a), Tunica intima, medicine.disease, Lipoproteins, LDL, Kinetics, medicine.anatomical_structure, Endocrinology, chemistry, biology.protein, Diet, Atherogenic, lipids (amino acids, peptides, and proteins), Analysis of variance, Rabbits, Tunica Intima, Tunica Media, Lipoprotein, Research Article
Abstract: The aim was to investigate the atherogenic potential of lipoprotein(a) (Lp(a)) and to further our understanding of the atherogenic process by measuring rates of transfer into the intima-inner media (i.e., intimal clearance) and rates of loss from the intima-inner media (i.e., fractional loss) of Lp(a) and LDL using cholesterol-fed rabbits with nonlesioned (n = 13) or atherosclerotic aortas (n = 12). In each rabbit, 131I-Lp(a) (or 131I-LDL) was injected intravenously 26 h before and 125I-Lp(a) (or 125I-LDL) 3 h before the aorta was removed and divided into six consecutive segments of similar size. The intimal clearance of Lp(a) and LDL was similar and markedly increased in atherosclerotic compared with nonlesioned aortas (ANOVA, effect of atherosclerosis: P < 0.0001). Fractional losses of labeled Lp(a) and labeled LDL in atherosclerotic aorta were on average 25 and 43%, respectively, of that in nonlesioned aortas (ANOVA, effect of atherosclerosis: P < 0.0001). Fractional loss of Lp(a) was 73% of that of LDL (ANOVA, effect of type of lipoprotein: P = 0.07). These data suggest that the development of atherosclerosis is associated with increased influx as well as decreased fractional loss of Lp(a) and LDL from the intima. Accordingly, Lp(a) may share with LDL the potential for causing atherosclerosis.
Published: 1996

175. Relative importance of ischemic injury and immunological injury on the development of transplant arteriosclerosis in rabbit aortic allografts

Author: Birgit Fischer Hansen, Knud Kjeldsen, Børge G. Nordestgaard, P. K. Holm, Henrik Ørbæk Andersen, and Steen Stender
Subjects: Male, Pathology, medicine.medical_specialty, Time Factors, Arteriosclerosis, medicine.medical_treatment, Lipoproteins, T-Lymphocytes, Ischemia, Aorta, Thoracic, medicine.artery, medicine, Thoracic aorta, Animals, Transplantation, Homologous, Heart transplantation, Transplantation, Aorta, Vascular disease, business.industry, Body Weight, medicine.disease, Tunica intima, surgical procedures, operative, medicine.anatomical_structure, Cholesterol, Cyclosporine, Rabbits, business, Tunica Intima, Immunosuppressive Agents
Abstract: The development of transplant arteriosclerosis has emerged as a major problem to long-term survival after heart transplantation. The accelerated development of arteriosclerosis in the transplanted arteries, including the aorta, could result either from an ischemic injury in connection with the transplantation, or from an immunological reaction against the transplant, or both. We evaluated histologically and biochemically whether extension of the ischemic period from 1 to 24 hr has any influence on the development of transplant arteriosclerosis, in aorta-allografted rabbits clamped at human levels of plasma cholesterol. One set of rabbits was without immunosuppressive treatment (n = 10 + 9) and another otherwise identical set of rabbits received cyclosporine to achieve blood cyclosporine levels in the human therapeutic range (n = 10 + 12). The number of T lymphocytes in intima suggested that, in the grafts from untreated animals, an immunological injury had arisen, which cyclosporine reduced. A clear trend toward a worsening of the transplant arteriosclerosis was demonstrated as a function of the severity of the ischemic injury, both with and without immunosuppressive treatment. However, the worsening effect of maximal ischemic injury was less than that due to maximal immunological injury. In grafts from cyclosporine-treated animals, the development of transplant arteriosclerosis was significantly less than in grafts from untreated animals exposed to identical periods of ischemia. These results suggest that compared with immunological injury, ischemic injury is of minor importance for the development of experimental transplant arteriosclerosis.
Published: 1995

176. A trans European Union difference in the decline intransfatty acids in popular foods: a market basket investigation

Author: Arne Astrup, Jørn Dyerberg, and Steen Stender
Subjects: Consumption (economics), Nutrition & Dietetics, French fries, business.industry, Health Policy, Research, Market basket, General Medicine, Eu countries, Agricultural economics, Coronary heart disease, Eastern european, Western europe, media_common.cataloged_instance, Medicine, Preventive Medicine, European union, business, media_common
Abstract: Objectives To minimise the intake of industrial trans fatty acids (I-TFA) some countries have introduced labelling, while others have introduced legislative limits on the content of I-TFA in food. However, most countries still rely on food producers to voluntarily reduce the I-TFA content in food. The objective of the present study was to investigate the efficiency of these strategies in the EU. Design The potential consumption of I-TFA was assessed in a market basket investigation by analysing the I-TFA content in popular foods. Setting A standardised purchase methodology was used in 16 EU countries in 2005 and again in 2009. Samples Seventy servings of French fries and chicken nuggets, 90 packages of microwave popcorn, and 442 samples of biscuits/cakes/wafers with ‘partially hydrogenated vegetable fat’ listed high on the list of ingredients were analysed. A high- trans menu was defined as a large serving of French fries and nuggets, 100 g of microwave popcorn and 100 g of biscuits/wafers/cakes. Results In 2005, a high- trans menu provided above 30 g of I-TFA in five EU countries in Eastern Europe and 20–30 g in eight EU countries in Western Europe. In 2009 the values in Hungary, Poland and the Czech Republic remained high between 10 and 20 g, whereas they were less than 2 g in Germany, France and the UK. Conclusions In 2009 contents of I-TFA in popular foods in Western Europe appear low but, in spite of some reduction, still high in Eastern European EU countries. These findings suggest that millions of people in the EU still consume I-TFA in amounts that substantially increase their risk of coronary heart disease.
Published: 2012

177. A possible association between a dysfunctional skin barrier (filaggrin null-mutation status) and diabetes: a cross-sectional study

Author: Pal B. Szecsi, Allan Linneberg, Torben Hansen, Steen Stender, Oluf Pedersen, Flemming Pociot, Jeanne D. Johansen, Torkil Menné, Berit C. Carlsen, Jacob P. Thyssen, Kåre Engkilde, and Michael Meldgaard
Subjects: chemical pathology, Cross-sectional study, Population, Dermatology, Type 2 diabetes, general population, Diabetes mellitus, medicine, biochemistry, genetics, education, occupational dermatology, education.field_of_study, diabetes, atopic dermatitis, integumentary system, business.industry, Research, regulation, General Medicine, Atopic dermatitis, medicine.disease, Null allele, bleeding disorders and coagulopathies, dermatological epidemiology, myeloma, Immunology, contact allergy, epidemiology, eczema, business, Filaggrin, Ichthyosis vulgaris
Abstract: Background Filaggrin proteins are located in the skin and prevent epidermal water loss and impede the entry of micro-organisms, allergens and chemicals. Filaggrin null mutations are strongly associated with ichthyosis vulgaris and atopic dermatitis. Objective The authors aimed to investigate the association between filaggrin null mutations, atopic dermatitis and diabetes. Design A random sample of 3335 adults from the general population in Denmark was filaggrin-genotyped for R501X and 2282del4 null-mutations and questioned about atopic dermatitis and diabetes. Furthermore, two independent study populations of patients with type 1 (n=104) or 2 (n=774) diabetes were genotyped. Results In a crude data analysis, a positive association was detected between the filaggrin null genotype and, respectively, subjects from the general population who reported diabetes (p=0.04) and patients with established type 2 diabetes (p=0.073). Adjustment for age and gender resulted in significant associations for patients with type 2 diabetes (p=0.048) and subjects with self-reported diabetes (p=0.032). Conclusions Adult Danes with a filaggrin null genotype had a significantly increased prevalence of self-reported diabetes. This finding was replicated when an independent sample of Danish patients with established type 2 diabetes was compared with control subjects from the general population., Article summary Article focus A few studies have suggested the existence of an inverse association between atopic dermatitis and type 1 diabetes. The existence of a specific endotype of asthma that is not driven by sensitisation but rather driven by skin barrier dysfunction was recently suggested. It is unknown whether a putative impairment of the skin barrier may increase the propensity to low-grade inflammation in other organs as well. Key messages Data from a general population study suggested that the prevalence of filaggrin null mutations was higher in adult Danes who reported diabetes than in non-diabetics. This finding was replicated when an independent sample of Danish patients with type 2 diabetes was compared with participants from the general population who did not report diabetes and who had normal fasting plasma-glucose and glycated haemoglobin (HbA1c) levels. Strengths and limitations of this study Two independent samples were investigated and showed similar results. The question on self-reported diabetes was not validated in the general population allowing for misclassification.
Published: 2011

178. Maternal smoking and impaired endothelium-dependent nitric oxide–mediated relaxation of uterine small arteries in vitro

Author: Malene Rohr Andersen, Niels Uldbjerg, Steen Stender, Puk Sandager, and Christian Aalkjaer
Subjects: Adult, medicine.medical_specialty, Endothelium, medicine.medical_treatment, Bradykinin, Isometric exercise, Nitric Oxide, Nitric oxide, chemistry.chemical_compound, Pregnancy, Surveys and Questionnaires, Internal medicine, Humans, Medicine, Cotinine, Chi-Square Distribution, Relaxation (psychology), business.industry, Smoking, Uterus, Myography, Obstetrics and Gynecology, Arteries, medicine.disease, Vasodilation, medicine.anatomical_structure, chemistry, Area Under Curve, Anesthesia, Cardiology, Smoking cessation, Female, Endothelium, Vascular, business, Myograph
Abstract: Udgivelsesdato: 2010-Oct-23 OBJECTIVE:: This study aimed to investigate the endothelium-dependent relaxation of uterine small arteries from pregnant nonsmokers, smokers, and ex-smokers who stopped smoking early in pregnancy. STUDY DESIGN:: Uterine arteries were dissected from myometrial biopsies obtained during elective cesarean sections of 34 uncomplicated, singleton pregnancies, and the vascular function was assessed in a wire myograph for isometric recordings. Serum cotinine verified self-reported smoking; 15 were nonsmokers, 10 were smokers, and 9 were ex-smokers. RESULTS:: Arteries from smokers and ex-smokers had reduced bradykinin-induced relaxation compared to arteries from nonsmokers (P < .05). The relaxation response to the nitric oxide donor sodium nitroprusside was similar in arteries from nonsmokers and smokers but was better in arteries from ex-smokers (P < .05). CONCLUSION:: The findings suggest that maternal smoking reduces endothelium-dependent nitric oxide-mediated relaxation in uterine small arteries and that smoking cessation early in pregnancy does not fully abolish this deleterious effect, despite improvement in relaxation to nitroprusside.
Published: 2011

179. Effect of testosterone on atherogenesis in cholesterol-fed rabbits with similar plasma cholesterol levels

Author: Børge G. Nordestgaard, Birgit A. Larsen, Steen Stender, and Knud Kjeldsen
Subjects: Male, medicine.medical_specialty, Arteriosclerosis, Blood Pressure, Biology, Placebo, Cholesterol, Dietary, chemistry.chemical_compound, Liver Function Tests, Internal medicine, Blood plasma, medicine, Animals, Testosterone, Aorta, Cholesterol, Body Weight, Testosterone (patch), Mean blood pressure, Endocrinology, Blood pressure, chemistry, Liver function, Rabbits, Cardiology and Cardiovascular Medicine, Lipoprotein
Abstract: The aim was to examine the effect of testosterone on atherogenesis in cholesterol-fed, castrated male rabbits not mediated via different plasma cholesterol levels. The rabbits in the testosterone group (n = 19) and in the placebo group (n = 17) were injected intramuscularly twice weekly for 17 weeks with 25 mg testosterone enantate and placebo, respectively, reaching plasma testosterone levels of 50-150 nmol/l in the testosterone group. No effect of testosterone on liver function or body weight was detected, but at week 15 mean blood pressure was 75 +/- 2 mmHg (mean +/- S.E.) in the testosterone group compared with 69 +/- 2 mmHg in the placebo group (P0.05). To reduce variation in plasma cholesterol between the two groups, the amount of cholesterol fed to each rabbit was adjusted on the basis of weekly determinations of plasma cholesterol; the mean plasma cholesterol levels during the 17 weeks were 20.9 +/- 1.0 and 20.4 +/- 0.9 mmol/l for the placebo and testosterone groups. In the intima-inner medias of the aortic arch, the thoracic and the abdominal aorta there were no consistent significant differences in aortic cholesterol content, expressed either as nmol/cm2 or nmol/mg protein, between the two groups. However, the aortic cholesterol content tended to be lower in the testosterone group than in the placebo group. These findings suggest that in cholesterol-fed, castrated male rabbits, testosterone does not promote atherogenesis by an effect directly on the arterial wall.
Published: 1993

180. TREATMENT OF LOW BACK PAIN: A RANDOMIZED TRIAL COMPARING GROUP-BASED MULTIDISCIPLINARY REHABILITATION AND INTENSIVE INDIVIDUAL THERAPIST-ASSISTED BACK MUSCLE STRENGTHENING EXECISE.

Author: Ninna, Dufour, primary, Gorm, Thamborg, additional, Alex, Oefeldt, additional, Charlotte, Lundsgaard, additional, and Steen, Stender, additional
Published: 2008
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181. O482 Gestational-age specific reference intervals for CA 125 during uncomplicated pregnancy, delivery and early postpartum period

Author: M. Rohr Andersen, N. Palmgren Colov, Pal B. Szecsi, Anna Klajnbard, and Steen Stender
Subjects: medicine.medical_specialty, business.industry, Obstetrics, Period (gene), Obstetrics and Gynecology, Medicine, Gestational age, General Medicine, business, Early postpartum, Uncomplicated pregnancy, Reference intervals
Published: 2009

182. O480 A simple method for presenting laboratory test results from pregnant women with gestational age-specific reference values in laboratory information systems, which usually only take age and gender into account

Author: N. Palmgren Colov, Anna Klajnbard, Steen Stender, Pal B. Szecsi, and Brian Bjørngaard
Subjects: Age and gender, Pediatrics, medicine.medical_specialty, Laboratory test, Simple (abstract algebra), business.industry, Reference values, medicine, Information system, Obstetrics and Gynecology, Gestational age, General Medicine, business
Published: 2009

183. O481 Gestational age-specific reference intervals for 32 chemical, 13 hematological, 10 coagulation and 3 thyroid laboratory tests during 391 uncomplicated pregnancy, vaginal delivery and early postpartum period

Author: K. Haahr, M. Rohr Andersen, A. Barfoed, Pal B. Szecsi, Steen Stender, Anna Klajnbard, N. Palmgren Colov, and Maja Jørgensen
Subjects: Gynecology, medicine.medical_specialty, Obstetrics, business.industry, Vaginal delivery, Thyroid, Obstetrics and Gynecology, Gestational age, General Medicine, Reference intervals, medicine.anatomical_structure, Coagulation, medicine, business, Early postpartum, Uncomplicated pregnancy
Published: 2009

184. Experience with fish oil treatment with special emphasis on diabetic nephropathy

Author: Torsten Deckert, Tonny Jensen, and Steen Stender
Subjects: medicine.medical_specialty, business.industry, Endocrinology, Diabetes and Metabolism, Fish oil, medicine.disease, Diabetic nephropathy, Capillary Permeability, Lipoproteins, LDL, Endocrinology, Fish Oils, Double-Blind Method, Risk Factors, Albumins, Internal Medicine, Medicine, Humans, Plant Oils, Endothelium, Vascular, business, Intensive care medicine, Olive Oil
Published: 1990

185. Abstract #839: Improvements in Lipid and Glucose Abnormalities in Insulin-Resistant, Nondiabetic Patients Treated with Tesaglitazar

Author: Serena Tonstad, Sion Edwards, Tamas Halmos, Herbert Schuster, Grethe Støa-Birketvedt, Ingrid Gause-Nilsson, Peter Öhman, Björn Fagerberg, Steen Stender, and Jerzy Lopatynski
Subjects: medicine.medical_specialty, chemistry.chemical_compound, Endocrinology, Tesaglitazar, chemistry, business.industry, Endocrinology, Diabetes and Metabolism, Internal medicine, medicine, Insulin resistant, General Medicine, business
Published: 2005

186. M.458 Effects of switching statins on non-HDL-C in hypercholesterolaemic patients and achievement of ATP III combined LDL-C and non-HDL-C goals in patients with elevated triglycerides: Mercury I trial results

Author: Steen Stender, R. Caplan, Philip J. Barter, J. Morrell, and J. Blasetto
Subjects: medicine.medical_specialty, Endocrinology, Chemistry, Internal medicine, Non hdl c, Internal Medicine, medicine, chemistry.chemical_element, In patient, General Medicine, Cardiology and Cardiovascular Medicine, Mercury (element)
Published: 2004

187. EANS Officials 2004

Author: Marcelo Saldanha Aoki, Francesco Cresi, Mehrdad Asadifar, J. Klvanová, Sheila Gottschalk, Francisco Navarro, Sebastian Schaffer, Lee B. Astheimer, Josef Pallauf, Roberto Oggero, M. Dušinská, Peter R. C. Howe, André Luís Rodriguez Amaral Almeida, Luís Fernando Bicudo Pereira Costa-Rosa, Annika Waldmann, G Grasso, Steen Stender, Fred Joseph, Kazuya Ide, Malektaj Yazdani, Reury Frank Pereira Bacurau, Tetsuo Nakamoto, Claus Leitzmann, Michael B. Krawinkel, Francesco Savino, Marica Krajčovičová-Kudláčková, J.W. Koschizke, Andreas Hahn, Linda C Tapsell, Leandra Silvestro, Theresa A Larkin, Barbara J. Meyer, Nobuya Shirai, Hiramitsu Suzuki, Jørn Dyerberg, and Alice J. Owen
Subjects: Gerontology, Medical education, Nutrition and Dietetics, business.industry, Medicine (miscellaneous), Medicine, business
Published: 2004

188. 1P-0283 Effects of trans- and n-3 unsaturated fatty acids on cardiovascular risk markers in healthy males

Author: Steen Stender, D C Eskesen, and Jørn Dyerberg
Subjects: Internal Medicine, General Medicine, Cardiology and Cardiovascular Medicine
Published: 2003

189. 1P-0291 Switching to rosuvastatin from other statins has beneficial effects on apolipoprotein (Apo) B and Apo B: Apo A-I ratio

Author: Steen Stender, David Kallend, Claire Watkins, Philip J. Barter, and J. Morrell
Subjects: medicine.medical_specialty, Apolipoprotein B, biology, business.industry, General Medicine, Endocrinology, Internal medicine, Internal Medicine, biology.protein, medicine, Rosuvastatin, Cardiology and Cardiovascular Medicine, business, Beneficial effects, medicine.drug
Published: 2003

190. 4.P.150 Four days weighed food record as routine procedure to patients with coronary heart disease in dietary intervention programmes

Author: A. Thurøe, Torben Haghfelt, B. Haastrup, C. Dalgård, and Steen Stender
Subjects: medicine.medical_specialty, business.industry, Intervention (counseling), Emergency medicine, Physical therapy, medicine, Cardiology and Cardiovascular Medicine, business, Coronary heart disease, Food record
Published: 1997

191. F040 Levormeloxifene, a non-steroidal estrogen receptor therapetic, reduces serum cholesterol and prevents aortic cholesterol accumulation in cholesterol fed rabbits

Author: M. Shalmi, Pernille Holm, B. Guldhammer, Steen Stender, Sven O. Skouby, and N. Korsgaard
Subjects: medicine.medical_specialty, business.industry, Cholesterol, Reverse cholesterol transport, Obstetrics and Gynecology, Estrogen receptor, General Biochemistry, Genetics and Molecular Biology, Non steroidal, chemistry.chemical_compound, Endocrinology, chemistry, Internal medicine, medicine, business, Levormeloxifene, Serum cholesterol, medicine.drug
Published: 1996

192. Aortic permeability during estrogen therapy

Author: L.B. Nielsen, Claus Christiansen, Steen Stender, and Jens Haarbo
Subjects: medicine.medical_specialty, Endocrinology, business.industry, Permeability (electromagnetism), Internal medicine, medicine, Estrogen therapy, Cardiology and Cardiovascular Medicine, business
Published: 1994

193. In vivo transfer of cholesterol from plasma into human aortic tissue

Author: Steen Stender and Erik Hjelms
Subjects: medicine.medical_specialty, Very low-density lipoprotein, Arteriosclerosis, Clinical Biochemistry, Tritium, chemistry.chemical_compound, In vivo, medicine.artery, Internal medicine, medicine, Humans, Thoracic aorta, Carbon Radioisotopes, Endothelium, Aorta, Cholesterol, Cholesterol, HDL, Abdominal aorta, Cholesterol, LDL, General Medicine, medicine.disease, Kinetics, Endocrinology, Biochemistry, chemistry, Cholesteryl ester, lipids (amino acids, peptides, and proteins), Cholesterol Esters
Abstract: It has recently become possible to measure the in vivo flux of cholesteryl ester from plasma into human aortic tissue by use of labeled cholesterol in patients undergoing reconstructive aortic surgery. For the ascending thoracic aorta without visible atherosclerotic lesions the influx was 4.5 +/- 1.4 nmol X cm-2 X day-1 (means +/- SEM, n = 9). For the abdominal aorta with severe atherosclerosis the influx of cholesteryl ester was 45 +/- 5 nmol X cm-2 X day-1 (n = 12). In both types of tissues the influx of cholesteryl ester from HDL was 2-3 times higher than the influx of cholesteryl ester from LDL and VLDL compared with the concentration of these fractions in plasma. This is in accordance with an aortic influx-mechanism which depends on the sizes and the concentration of the lipoproteins in plasma. The transfer of plasma lipoproteins into human aortic tissue shows a number of similarities with the transfer of plasma lipoproteins into the aortic wall of cholesterol-fed rabbits and also with the transfer of other plasma macromolecules across various capillaries. The cholesterol content in intima-media tissue without lesions corresponded in some of the patients to less than one year of continuous influx of cholesteryl ester from plasma. This time is short compared with the age of the patients. It suggests that removal of cholesterol from the aortic wall represents a major importance in prevention of cholesterol accumulation in that tissue.
Published: 1987

194. The concomitantly measured transfer of free cholesterol, esterified cholesterol, phospholipids and phosphoprotein from plasma into the aortic wall of stilboestrol-treated cockerels

Author: Søren Brøgger Christensen and Steen Stender
Subjects: Male, medicine.medical_specialty, Hyperlipidemias, Esterified cholesterol, chemistry.chemical_compound, In vivo, medicine.artery, Internal medicine, Chylomicrons, medicine, Animals, Thoracic aorta, Diethylstilbestrol, Aorta, Triglycerides, Cholesterol, Phosphatidylethanolamines, Reverse cholesterol transport, Plasma, Endocrinology, chemistry, Phosphoprotein, Phosphatidylcholines, lipids (amino acids, peptides, and proteins), Cholesterol Esters, Cardiology and Cardiovascular Medicine, Chickens
Abstract: In vivo labelled plasma containing free and esterified [ 3 H] cholesterol, [ 32 P]-phospholipids [ 32 P]phosphoprotein was injected intravenously into eight cockerels and the radioactivity in the various fractions per square centimeter of thoracic aorta (uptake) was determined 0.2–12 h after the injection. Using the uptake and the integrated plasma decay curve, an uptake coefficient for the inner aorta in μl plasma per square centimeter per hour was calculated for each fraction. Within the same cockerel these coefficients were nearly identical for all the measured lipid fractions and higher for the phosphoprotein-complex which had a lower mean molecular weight than the dominant plasma lipoprotein. This is in accordance with a molecular weight dependent uptake in the inner aorta of the lipoproteins as intact macromolecules. The uptake of free and esterified labelled choleterol was measured in each cockerel during two different and overlapping periods, by injecting into each recipient cockerel first donor plasma labelled with [ 14 C] cholesterol and, 3–9 h later, donor plasma labelled with [ 3 H]cholesterol. The results indicated that the labelled free and esterified plasma cholesterol taken up by inner aorta remained there as free and esterified cholesterol during the first 5–6 h after the injection. The uptake of labelled cholesterol can therefore within that period be used for calculation of the influx of plasma cholesterol into the aortic wall.
Published: 1977

195. Abnormalities in Spinal neurons and Dorsal Root Ganglion Cells in Tangier Disease Presenting with a Syringomyelia-like Syndrome

Author: Henning Schmalbruch, Steen Stender, and Gudrun Boysen
Subjects: Sural nerve, Biology, Pathology and Forensic Medicine, Lipofuscin, Diagnosis, Differential, Cellular and Molecular Neuroscience, Tangier disease, Dorsal root ganglion, Ganglia, Spinal, medicine, Humans, Tangier Disease, Neurons, General Medicine, Anatomy, Middle Aged, Hypolipoproteinemias, Spinal cord, medicine.disease, Lipids, Syringomyelia, Ganglion, Microscopy, Electron, medicine.anatomical_structure, Spinal Cord, nervous system, Neurology, Neuroglia, Female, Neurology (clinical)
Abstract: A woman with homozygous Tangier disease had progressive syringomyelia-like neuropathy. She died with cardiac failure at age 61. A sural nerve biopsy taken at age 60 had shown lipid storage in Schwann and interstitial cells, and a pronounced loss of unmyelinated fibers. The neurons of the L5 spinal ganglion and, to a lesser extent, all neurons of the sacral spinal cord, contained large lipid inclusions which in electron micrographs differed from those in Schwann and satellite cells. There was no storage material in glial cells. The neuronal inclusions were membrane-bound and consisted of electron-dense and electron-lucent components. There was evidence of neuronal death in the spinal ganglion, and a diameter histogram showed that small cytons had preferentially been lost. The inclusions probably were secondary lysosomes or residual bodies, and resembled giant lipofuscin granules. Nevertheless, they were uncolored and displayed weak autofluorescence as compared to the aging pigment in control ganglia. It is tentatively suggested that the syringomyelia-like neuropathy in Tangier disease represents a lysosomal storage disorder preferentially affecting small dorsal root ganglion cells.
Published: 1987

196. Turnover of cholesterol in aorta of rabbits previously fed a cholesterol-enriched diet

Author: Steen Stender
Subjects: medicine.medical_specialty, Time Factors, Hypercholesterolemia, Biology, Cholesterol, Dietary, chemistry.chemical_compound, Internal medicine, medicine.artery, medicine, Animals, Aorta, Serum cholesterol, Confusion, Cholesterol, medicine.disease, Endocrinology, Atheroma, Liver, chemistry, lipids (amino acids, peptides, and proteins), Specific activity, Rabbits, medicine.symptom, Cardiology and Cardiovascular Medicine
Abstract: When cholesterol addition to the diet of rabbits is discontinued, there is general agreement about the ability of the rabbits to eliminate cholesterol accumulated in liver and serum. More confusion exists on the fate of cholesterol accumulated in the arteries (1,2).
Published: 1976

197. Calcium Antagonists and Experimental Atherosclerosis

Author: Knud Kjeldsen and Steen Stender
Subjects: Blood Platelets, Experimental atherosclerosis, medicine.medical_specialty, Arteriosclerosis, medicine.medical_treatment, chemistry.chemical_element, Hyperlipidemias, Calcium, Pharmacology, Muscle, Smooth, Vascular, General Biochemistry, Genetics and Molecular Biology, Text mining, In vivo, Internal medicine, medicine, Animals, Humans, Chemotherapy, business.industry, Calcium Channel Blockers, Endocrinology, Mechanism of action, chemistry, Endothelium, Vascular, medicine.symptom, business
Published: 1989

198. Effect of verapamil on accumulation of free and esterified cholesterol in the thoracic aorta of cholesterol-fed rabbits

Author: Micael Haugegaard, Hans Peter Ravn, Steen Stender, and Knud Kjeldsen
Subjects: Male, Very low-density lipoprotein, medicine.medical_specialty, chemistry.chemical_element, Aorta, Thoracic, Calcium, Placebo, Cholesterol, Dietary, chemistry.chemical_compound, medicine.artery, Internal medicine, medicine, Animals, Thoracic aorta, Lagomorpha, biology, Cholesterol, business.industry, Antagonist, biology.organism_classification, Endocrinology, Verapamil, chemistry, Depression, Chemical, Cholesterol Esters, Rabbits, Cardiology and Cardiovascular Medicine, business, medicine.drug
Abstract: In order to investigate the effect of the calcium antagonist verapamil on atherogenesis in cholesterol-fed rabbits, 3 groups of 11 animals were fed a 2% cholesterol-enriched diet for 10 weeks. One group received verapamil in a daily dose of 16 mg/kg orally plus 2 mg/kg subcutaneously. This dosage resulted in plasma concentrations of verapamil in the same range as the usual therapeutic levels in humans. Another group received verapamil in a daily dose of 8 mg/kg orally and 0.5 mg/kg subcutaneously. The third group received placebo capsules orally and isotonic saline subcutaneously. Total cholesterol concentrations in plasma over the 10 weeks were 37 +/- 4, 42 +/- 4 and 45 +/- 3 mM (mean +/- SE) in the high verapamil-, in the low verapamil- and in the placebo group, respectively. These values were not significantly different. The distribution of cholesterol between HDL, LDL and VLDL in plasma was similar in the 3 groups. The high verapamil group had a significantly (P less than 0.05) lower concentration of cholesterol in the thoracic aorta than the placebo group (29 +/- 5 vs 43 +/- 9 mumol/g wet weight). The low verapamil group and the placebo group had the same aortic cholesterol concentrations. Neither dosage of verapamil affected the permeability of the aortic endothelium to plasma lipoproteins and albumin, as measured by use of radioactive tracers at the end of the experiment.
Published: 1986

199. In vivo influx of free and esterified plasma cholesterol into human aortic tissue without atherosclerotic lesions

Author: Erik Hjelms and Steen Stender
Subjects: Adult, Male, medicine.medical_specialty, Arteriosclerosis, Lipoproteins, Muscle, Smooth, Vascular, chemistry.chemical_compound, Aortic valve replacement, Plasma cholesterol, In vivo, Internal medicine, medicine, Aortic tissue, Humans, Distribution (pharmacology), Aorta, Aged, Chemistry, Cholesterol, Hydrolysis, Reverse cholesterol transport, General Medicine, Middle Aged, medicine.disease, Sterol, Kinetics, Endocrinology, lipids (amino acids, peptides, and proteins), Cholesterol Esters, Research Article
Abstract: In order to determine the in vivo influx of plasma cholesterol into human aortic intimamedia tissue, specimens of the ascending aortic wall without visible atherosclerosis were obtained from patients undergoing aortic valve replacement. Before the operation the patients were intravenously injected with autologous plasma in which the lipoproteins were labeled with radioactive cholesterol. The influence of the duration of the exposure time (0.3-114 h) and of the distribution of radioactivity between free and esterified cholesterol in plasma on the amount of radioactivity found in the arterial wall was studied by the simultaneous use of 3H- and 14C-cholesterol. It was shown that the influx of free and esterified cholesterol into the intima-media layer of the tissue could be calculated from a set of linear equations that relate the labeled sterols in the tissue to the average specific activities in plasma. In nine patients between 50 and 70 yr of age with 4.2-5.9 mM total cholesterol in plasma, the influx of free cholesterol and of esterified cholesterol was 1.2-8.8 and 1.0-12.5 nmol X cm-2 X d-1, respectively. Both hydrolysis and esterification of the sterol fractions in the aortic tissue and exchange of free cholesterol between the plasma lipoproteins and the tissue were demonstrated. The cholesterol content of the intima-media layer was 0.6-2.3 mumol X cm-2. This corresponds to the influx of esterified cholesterol during a period of only 0.1-3.5 yr, which is short compared with the lifespan of the patient. Our data thus suggest that removal of esterified cholesterol from aortic tissue without visible atherosclerosis represents a major importance for the cholesterol concentration in the tissue.
Published: 1984

200. Severe Hypertriglyceridemia, large lipoproteins and protection against atherosclerosis

Author: Knud Kjeldsen, Steen Stender, and Børge G. Nordestgaard
Subjects: Male, medicine.medical_specialty, Apolipoprotein B, Arteriosclerosis, Lipoproteins, Clinical Biochemistry, Hyperlipoproteinemia Type IV, Diabetes Mellitus, Experimental, Cholesterol, Dietary, Lesion, chemistry.chemical_compound, Reference Values, Internal medicine, Alloxan, Diabetes mellitus, medicine, Animals, Humans, Apolipoproteins C, Triglycerides, Lipoprotein lipase, biology, Cholesterol, Hypertriglyceridemia, General Medicine, medicine.disease, Familial hypertriglyceridemia, Lipoprotein Lipase, Endocrinology, chemistry, biology.protein, Diet, Atherogenic, Apolipoprotein C-II, Female, lipids (amino acids, peptides, and proteins), Rabbits, medicine.symptom
Abstract: Alloxan-diabetic cholesterol-fed rabbits exhibiting severe hypertriglyceridemia are protected against atherosclerosis. In such rabbits most of the plasma cholesterol is found in lipoproteins with a diameter of 75 nm or larger. In the present report it is hypothesized, that due to their large sizes, the lipoproteins of the severely hypertriglyceridemic diabetic rabbits are not able to penetrate the endothelial layer of the arteries. Therefore, the macrophages and smooth muscle cells of the intima will only come in contact with relatively small amounts of cholesterol-carrying lipoproteins. Consequently, cellular accumulation of cholesterol, which is a necessary step in the formation of an atherosclerotic lesion, will be retarded. There are certain parallels between hypertriglyceridemic cholesterol-fed alloxan-diabetic rabbits and humans with familial lipoprotein lipase deficiency, familial apolipoprotein C-II deficiency and insulin-dependent diabetes mellitus in the ketoacidotic state. Based on reports about patients with these metabolic disorders, we suggest that cholesterol in very large lipoproteins also in humans is less atherogenic than cholesterol in smaller lipoproteins.
Published: 1987

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