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151. Supplementary Figure 1 from Improved Performance of Serum Alpha-Fetoprotein for Hepatocellular Carcinoma Diagnosis in HCV Cirrhosis with Normal Alanine Transaminase

152. Data from Transcriptional Induction of Periostin by a Sulfatase 2–TGFβ1–SMAD Signaling Axis Mediates Tumor Angiogenesis in Hepatocellular Carcinoma

153. Data from Improved Performance of Serum Alpha-Fetoprotein for Hepatocellular Carcinoma Diagnosis in HCV Cirrhosis with Normal Alanine Transaminase

154. Supplementary Tables from Transcriptional Induction of Periostin by a Sulfatase 2–TGFβ1–SMAD Signaling Axis Mediates Tumor Angiogenesis in Hepatocellular Carcinoma

155. Supplementary Figures 1-5 from Transcriptional Induction of Periostin by a Sulfatase 2–TGFβ1–SMAD Signaling Axis Mediates Tumor Angiogenesis in Hepatocellular Carcinoma

157. Supplementary Figures 9-10 from Sleeping Beauty Insertional Mutagenesis in Mice Identifies Drivers of Steatosis-Associated Hepatic Tumors

159. Data from Neuropilin-1 Stimulates Tumor Growth by Increasing Fibronectin Fibril Assembly in the Tumor Microenvironment

165. Supplementary Figure 5 from Neuropilin-1 Stimulates Tumor Growth by Increasing Fibronectin Fibril Assembly in the Tumor Microenvironment

167. Supplementary Figure 2 from Neuropilin-1 Stimulates Tumor Growth by Increasing Fibronectin Fibril Assembly in the Tumor Microenvironment

168. Supplementary Figure 4 from Neuropilin-1 Stimulates Tumor Growth by Increasing Fibronectin Fibril Assembly in the Tumor Microenvironment

169. Supplementary Figure 7 from Neuropilin-1 Stimulates Tumor Growth by Increasing Fibronectin Fibril Assembly in the Tumor Microenvironment

171. Supplementary Figure 1 from Neuropilin-1 Stimulates Tumor Growth by Increasing Fibronectin Fibril Assembly in the Tumor Microenvironment

172. Supplementary Methods from Neuropilin-1 Stimulates Tumor Growth by Increasing Fibronectin Fibril Assembly in the Tumor Microenvironment

173. Supplementary Figure Legends 1-7 from Neuropilin-1 Stimulates Tumor Growth by Increasing Fibronectin Fibril Assembly in the Tumor Microenvironment

175. Supplementary Figure 3 from Neuropilin-1 Stimulates Tumor Growth by Increasing Fibronectin Fibril Assembly in the Tumor Microenvironment

176. Supplementary Figure 6 from Neuropilin-1 Stimulates Tumor Growth by Increasing Fibronectin Fibril Assembly in the Tumor Microenvironment

177. Implementation of genomic medicine for rare disease in a tertiary healthcare system: Mayo Clinic Program for Rare and Undiagnosed Diseases (PRaUD)

178. Hepatocellular carcinoma presentation and prognosis among Nigerian adults with and without HIV

179. Analysis of N-linked Glycan Alterations in Tissue and Serum Reveals Promising Biomarkers for Intrahepatic Cholangiocarcinoma

180. Development of a Computer-aided Prediction Tool for Evaluating Brushing Samples of Biliary Strictures

181. Dietary inflammatory and insulinemic potential, risk of hepatocellular carcinoma, and chronic liver disease mortality

184. Mixed Acinar Neuroendocrine Carcinoma of the Pancreas: Comparative Population-Based Epidemiology of a Rare and Fatal Malignancy in The United States

188. Antitumor effect of FGFR inhibitors on a novel cholangiocarcinoma patient derived xenograft mouse model endogenously expressing an FGFR2-CCDC6 fusion protein

191. Criteria for preclinical models of cholangiocarcinoma:scientific and medical relevance

192. The Use of Social Media for Dissemination of Research Evidence to Health and Social Care Practitioners: Protocol for a Systematic Review (Preprint)

193. A deep learning system accurately classifies primary and metastatic cancers using passenger mutation patterns

194. Combined burden and functional impact tests for cancer driver discovery using DriverPower

195. Integrative pathway enrichment analysis of multivariate omics data

196. Pathway and network analysis of more than 2500 whole cancer genomes

197. Divergent mutational processes distinguish hypoxic and normoxic tumours

198. Genomic footprints of activated telomere maintenance mechanisms in cancer

199. Apobec1 complementation factor overexpression promotes hepatic steatosis, fibrosis, and hepatocellular cancer

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