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156. ULTRASONIC TISSUE CHARACTERIZATION

Author

Di Bello, V., primary, Pedrinelli, R., additional, Bertini, A., additional, Giorgi, D., additional, Cioppi, A., additional, Talini, E., additional, Moroní, L., additional, Caputo, M. T., additional, Pallini, M., additional, Precisi, S., additional, Khabirinejad, J., additional, DellʼOmo, G., additional, and Giusti, C., additional

Published
2000
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157. ACOUSTIC BORDER DETECTION

Author

Di Bello, V., primary, Pedrinelli, R., additional, Bertini, A., additional, Giorgi, D., additional, Talini, E., additional, Moretti, L., additional, Pallini, M., additional, Precisi, S., additional, Caputo, M. T., additional, DellʼOmo, G., additional, and Giusti, C., additional

Published
2000
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162. Perioperative Prognostic Value of Dipyridamole Echocardiography in Vascular Surgery : A Large-Scale Multicenter Study in 509 Patients

Author

Sicari, R., primary, Ripoli, A., additional, Picano, E., additional, Djordjevic-Dikic, A., additional, Di Giovanbattista, R., additional, Minardi, G., additional, Matskeplishvili, S., additional, Ambatiello, S., additional, Pulignano, G., additional, Accarino, M., additional, Lusa, A. M., additional, Franco Del Rosso, G., additional, Pedrinelli, R., additional, and Buziashvili, Y., additional

Published
1999
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192. Coronary Microcirculation in Essential Hypertension: A Quantitative Myocardial Contrast Echocardiographic Approach.

Author

Di Bello, V., Pedrinelli, R., Giorgi, D., Bertini, A., Talini, E., Mengozzi, G., Palagi, C., Nardi, C., Dell'Omo, G., Paterni, M., and Mariani, M.

Subjects
MICROCIRCULATION disorders, ESSENTIAL hypertension, ECHOCARDIOGRAPHY, MICROBUBBLES, DIPYRIDAMOLE, VASODILATION, HYPERTROPHY
Abstract

Aims: The aims of the present study were: (a) to demonstrate whether quantitative myocardial contrast echocardiography can detect the increase in coronary flow induced by dipyridamole infusion vasodilation through the myocardial opacification due to the transit of microbubbles, both at rest and after dipyridamole induced vasodilation; (b) to explore the coronary microcirculatory function before and after dipyridamole in two different models: asymptomatic and relatively young hypertensive patients with a mild degree of left ventricular hypertrophy, and healthy controls.Methods and Results: Two groups of strictly age-matched males were studied (case-control study): 10, relatively young and asymptomatic essential hypertensive patients with a mild degree of left ventricular hypertrophy with a normal left ventricular function, and 10 healthy controls. The main findings were: the microbubbles' appearance area was significantly lower in hypertensive patients than in controls (P<0·05) because of a significantly lower time to peak. The peak intensity at rest was higher in hypertensives than in controls (P<0·05); but the per cent increase after vasodilatory stimulus was significantly higher in controls (+71% in controls vs +31% in hypertensives; P<0·05). The microbubbles' disappearance area was comparable in both groups at rest; the per cent increase of this parameter after dipyridamole was significantly higher in controls (+124%) than in hypertensives (+90%) (P<0·05). The results achieved in this study documented that the coronary microcirculation in hypertensive patients presenting a mild degree of left ventricular hypertrophy, explored with quantitative myocardial contrast echocardiography, showed a different behaviour in comparison with controls, in the vasodilatory response to dipyridamole.Conclusion: The coronary microcirculation in hypertensives showed a reduced vasodilation capacity of the resistance arterioles under dipyridamole induced vasodilatation, and a possible impairment of the endothelium dependent vasodilation. This happened despite an increase in the left ventricular mass, where the relation between capillary bed distribution and hypertrophied myocardium (rarefaction phenomenon) is not completely respected. Copyright 2002 The European Society of Cardiology. Published by Elsevier Science Ltd. All rights reserved. [ABSTRACT FROM AUTHOR]

Published
2002
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193. Non-diabetic microalbuminuria, endothelial dysfunction and cardiovascular disease.

Author

Pedrinelli, R., Dell’Omo, G., Penno, G., and Mariani, M.

Subjects
*CARDIOVASCULAR diseases, *ULTRASONIC imaging
Abstract

Subclinical increases in albuminuria (microalbuminuria) predict morbid events, but the reasons for that are still not understood in full. This paper reviews the existing evidence regarding the relationships of non-diabetic microalbuminuria and cardiovascular disease, the underlying assumption being that endothelial dysfunction contributes both to atherosclerotic macrovascular disease and renal microvascular disease of which albuminuria is a marker. Much data support that concept, and suggest a preferential link with endothelial activation in response to acute and subclinical inflammatory stimulation, although further studies are needed to establish the exact cause–effect mechanisms. Epidemiological studies also show associations with cardiovascular events, and some recent prospective results also indicate the power of micro-albuminuria to predict risk independently from conventional atherogenic factors. Thus, micro-albuminuria might be considered as an integrated marker of cardiovascular risk sensitive to systemic vascular status in addition to other parameters such as blood pressure levels, glucose metabolism, smoking habits, a profile rather unique among the prognostic predictors available to stratify risk in hypertensive patients. [ABSTRACT FROM AUTHOR]

Published
2001
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