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794 results on '"Lu, C. -H"'

151. LB0002 Safety and Tolerability of Arimoclomol in Patients with Sporadic Inclusion Body Myositis: A Randomised, Double-Blind, Placebo-Controlled, Phase IIa Proof-of-Concept Trial

Author

Machado, P., primary, Miller, A., additional, Herbelin, L., additional, He, J., additional, Noel, J., additional, Wang, Y., additional, McVey, A. L., additional, Pasnoor, M., additional, Gallagher, P., additional, Statland, J., additional, Brady, S., additional, Lu, C.-H., additional, Kalmar, B., additional, Sethi, H., additional, Samandouras, G., additional, Holton, J., additional, Greensmith, L., additional, Barohn, R. J., additional, Hanna, M. G., additional, and Dimachkie, M. M., additional

Published
2013
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155. Computational and Experimental Investigations of Laser Marking on 6061 Aluminium Alloy.

Author

LI, J. M., LU, C. H., ZHANG, J. Q., and WU, Y. S.

Subjects
*HEAT treatment of aluminum alloys, *LASERS in engineering, *ND-YAG lasers, *LASERS in metallurgy, EFFECT of lasers on metals
Abstract

In this work both the computational heat transfer model and experimental investigations were used for understanding laser marking on the surface of a 6061 aluminium alloy, subjected to various processing conditions. The influence of laser processing parameters on the change of surface temperature and marking depth of laser processed 6061 aluminium alloy samples were studied. Furthermore, laser marking experiments on 6061 aluminium alloy substrates using a Nd:YAG laser were carried out. A good agreement between the simulation results and experimental data proved that the established simulation model could be used to give instructions regarding the practical marking process and predict the marking results as well. [ABSTRACT FROM AUTHOR]

Published
2016

185. A 110nm RFCMOS GPS SoC with 34mW −165dBm tracking sensitivity

Author

Wei, J.-M., primary, Chen, C.-N., additional, Chen, K.-T., additional, Kuo, C.-F., additional, Ong, B.-H., additional, Lu, C.-H., additional, Liu, C.-C., additional, Chiou, H.-C., additional, Yeh, H.-C., additional, Shieh, J.-H., additional, Huang, K.-S., additional, Li, K.-I., additional, Wu, M.-J., additional, Li, M.-H., additional, Chou, S.-H., additional, Chew, S.-L., additional, Lien, W.-L., additional, Yau, W.-G., additional, Ge, W.-Z., additional, Lai, W.-C., additional, Ting, W.-H., additional, Tsai, Y.-J., additional, Yen, Y.-C., additional, and Yeh, Y.-C., additional

Published
2009
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200. PGCDietary curcumin supplementation protects against heat-stress-impaired growth performance of broilers possibly through a mitochondrial pathway.

Author

Zhang, J. F., Hu, Z. P., Lu, C. H., Yang, M. X., Zhang, L. L., and Wang, T.

Subjects
CURCUMIN, DIETARY supplements, PHYSIOLOGICAL effects of heat, BROILER chickens, MITOCHONDRIAL DNA, PHYSIOLOGY
Abstract

A total of 400 1-d-old Arbor Acres broiler chicks were raised at a recommended environmental temperature from d 1 to 20 (experimental day [ED] = ED1 to ED20). On ED21, the chicks were weighed and reallocated into 5 treatment groups, with 8 replicates of 10 birds each. The 5 treatment groups were as follows: the control group, in which chicks were housed at 22 ± 1°C and fed the basal diet, and the HS, HS-CUR50, HS-CUR100, and HS-CUR200 groups, in which chicks were housed at 34 ± 1°C for 8 h (0900-1700 h) and 22 ± 1°C for the rest time and fed the basal diet with 0, 50, 100, and 200 mg/ kg curcumin, respectively. From ED21 to ED42, the heat treatment lasted for 20 consecutive days. The results showed that heat-stressed broilers had greater (P < 0.05) average head surface and rectal temperature on ED21 and ED42 than the non-heat-stressed broilers. Diets supplied with 50 and 100 mg/kg curcumin increased (P < 0.05) the G:F compared to the heat-stressed groups. Mitochondrial malondialdehyde levels, an index of lipid peroxidation, in the breast muscle were 15.15 and 9.09% higher (P < 0.05) in 50 and 100 mg/kg curcumin supplemented groups than that of the heat-stressed group, respectively. Curcumin supplementation (50, 100, and 200 mg/kg) increased (P < 0.05) mitochondrial glutathione content and glutathione peroxidase, glutathione S-transferase, and manganese superoxide dismutase activities compared to heat-stressed broilers. Curcumin supplementation (50, 100, and 200 mg/kg) resulted in a decrease (P < 0.05) of heat shock protein 70 mRNA levels in the breast muscle. The breast muscle mRNA expression of peroxisome proliferator-activated receptor γ coactivator 1α and nuclear respiratory factor 1 and 2 in heat-stressed groups was increased (P < 0.05) in response to dietary 100 mg/kg curcumin treatment. Additionally, when compared to the heat-stressed group, mitochondrial transcription factor A mRNA levels were increased (P < 0.05) by 17.64% in the 200 mg/kg curcumin supplemented group. In conclusion, dietary curcumin supplementation prevented heat-stress-impaired growth performance, possibly through improving the antioxidant defense system and enhancing the mitochondrial biogenesis. [ABSTRACT FROM AUTHOR]

Published
2015
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