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Parcul Natural Por��ile de Fier: monografie Invita��ie ��n Parcul Natural Por��ile de Fier 1 Maria P��troescu, Lauren��iu Rozylowicz, Marian C. Jiplea Parcul Natural Por��ile de Fier. Repere administrative ��i legislative 7 Mihai R. Ni����, Marian C. Jiplea, Diana A. Onose, Maria P��troescu Poten��ialul ecologic al Parcului Natural Por��ile de Fier 18 Stelu��a Manolache, Iulian M. Niculae, Simona R. Gr��dinaru, Andreea Ni����, Vasile Bagrinovschi Introducere la Geologia Parcului Natural Por��ile de Fier 26 Mihai Emilian Popa Comunit����ile umane ��i peisajele din Parcul Natural Por��ile de Fier 63 Iulian M. Niculae, Cat��lin V. Toboiu, Maria P��troescu, Cristian I. Ioj��, Mihai R. Ni����, Cristiana M. Cioc��nea Biodiversitatea Parcului Natural Por��ile de Fier 85 Stelu��a Manolache, Lauren��iu Rozylowicz, Maria P��troescu, Amalia R. Dumbrav�� Turism ��n Parcul Natural Por��ile de Fier 126 Andreea Ni����, Athanasios A. Gavrilidis, C��t��lin V. Toboiu, Gabriel O. V��n��u, Marius L. Matache, Simona R. Gr��dinaru, Felix Baratky Bibliografie 148, Carte produs�� ��n cadrul proiectului M��suri active de protec��ie ��i conservare a biodiversit����ii ��i peisajului din arealul Parcului Natural Por��ile de Fier Cod MySMIS 2014+ 117515. Proiect cofinan��at din Fondul European de Dezvoltare Regional�� prin Programul Opera��ional Infrastructur�� Mare 2014-2020. Beneficiar: R.N.P. ROMSILVA - Administra��ia Parcului Natural Por��ile de Fier R.A. Editor: Centrul de Cercetare a Mediului ��i Efectuare a Studiilor de Impact - CCMESI, Universitatea din Bucure��ti. Con��inutul acestui material nu reprezint�� ��n mod obligatoriu pozi��ia oficial�� a Uniunii Europene sau a Guvernului Rom��niei., {"references": ["ABAB. 2015. Planul de management bazinal actualizat al spa\u0163iului hidrografic Banat. Administra\u021bia Na\u021bional\u0103 Apele Rom\u00e2ne. Administra\u021bia Bazinal\u0103 de Ap\u0103 Banat, Timi\u0219oara.", "APNPF. 2013. 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Parcul Natural Porţile de Fier: monografie Invitație în Parcul Natural Porțile de Fier 1 Maria Pătroescu, Laurențiu Rozylowicz, Marian C. Jiplea Parcul Natural Porțile de Fier. Repere administrative și legislative 7 Mihai R. Niță, Marian C. Jiplea, Diana A. Onose, Maria Pătroescu Potențialul ecologic al Parcului Natural Porțile de Fier 18 Steluța Manolache, Iulian M. Niculae, Simona R. Grădinaru, Andreea Niță, Vasile Bagrinovschi Introducere la Geologia Parcului Natural Porțile de Fier 26 Mihai Emilian Popa Comunitățile umane și peisajele din Parcul Natural Porțile de Fier 63 Iulian M. Niculae, Catălin V. Toboiu, Maria Pătroescu, Cristian I. Iojă, Mihai R. Niță, Cristiana M. Ciocănea Biodiversitatea Parcului Natural Porțile de Fier 85 Steluța Manolache, Laurențiu Rozylowicz, Maria Pătroescu, Amalia R. Dumbravă Turism în Parcul Natural Porțile de Fier 126 Andreea Niță, Athanasios A. Gavrilidis, Cătălin V. Toboiu, Gabriel O. Vânău, Marius L. Matache, Simona R. Grădinaru, Felix Baratky Bibliografie 148

The mapping of electrical activity in the human brain is not only useful in the medical field for the diagnosis of diseases but can also be of great help in other fields such as computer science. This research proposes the use of brain signals as a control mechanism using artificial intelligence techniques. Its main objective is to build a prototype of a 3D printed robotic hand prosthesis monitored through the processing of brain signals using recurrent neural networks. Three different recurrent network architectures were trained and compared using a prototyping methodology (Simple RNN, LSTM, and GRU). This prototyping methodology was trained using a sample size of thirteen people and the non-invasive sensor Mindwave Mobile 2 was used to record the data. The first version, which was in development approximately 3 months, achieved 77% accuracy in classifying new samples using the GRU network model. With further research and development, this prototype may prove very useful in the future for providing people in need of such technology with a higher quality of life., Las señales generadas por la actividad eléctrica producida en el cerebro humano, además de ser utilizadas en el área de medicina para el diagnóstico de enfermedades, pueden ser de gran ayuda en otros campos, como lo son las ciencias computacionales. Esta investigación propone la utilización de señales cerebrales como mecanismo de control, empleando técnicas de inteligencia artificial. La misma tiene como objetivo principal construir un prototipo de una prótesis de mano robótica impresa en 3D, controlada a través del procesamiento de señales cerebrales, utilizando redes neuronales recurrentes. Mediante una metodología de prototipado se entrenaron y compararon tres arquitecturas distintas de redes recurrentes (RNN simple, LSTM y GRU), entrenadas a partir de datos de trece personas, utilizando el sensor no invasivo Mindwave Mobile 2 para la adquisición de estos. La primera versión, desarrollada en un periodo aproximado de tres meses, alcanzó una precisión del 77% al clasificar nuevas muestras utilizando el modelo de red GRU. Este prototipo, al ser una primera aproximación y requerir mayor tiempo de investigación y desarrollo, puede ser de gran utilidad a futuro para personas que así lo necesiten, brindándoles una mayor calidad de vida.

Este artículo presenta el prototipo de sistema inteligente basado en patrones de ondas cerebrales para prevenir accidentesde tránsito, que, mediante un sensor colocado en la cabeza del conductor, monitoriza los patrones de ondas cerebrales los cuales sonenviados en tiempo real vía Bluetooth a una placa Raspberry Pi para ser procesados con estrategias de aprendizaje automático y deesta forma enviar una alerta visual y sonora cuando detecta el estado de somnolencia en el conductor. Para la construcción delprototipo se recogieron datos de cuatro personas en tres estados distintos, mientras estaban despiertas, somnolientas y dormidas. Elconjunto de datos fue procesado con cuatro algoritmos de aprendizaje supervisado: vecinos más cercanos, máquina de soportevectorial, árboles de decisión, bosques aleatorios; siendo este último el que mejores resultados mostró alcanzando un 82.05% deprecisión al diferenciar los tres estados anteriormente mencionados. El costo estimado del sistema es de 210 USD, resultando unsistema económico con relación a otros existentes en el mercado.

Silicon carbide is widely appreciated for its high temperature strength, radiation tolerance and neutronic transparency in applications for fuel particles and core internals of nuclear reactors. In the Fluoride Salt-Cooled High Temperature Reactor, silicon carbide ceramic matrix composites are candidate construction material for regions of higher neutron fluxes. Silicon carbide is wettable and reacts electrochemically with dissolved metals. Metallic impurities, tritium, moisture-based impurities and fission products, as well as thermal gradients can accelerate hot corrosion of silicon carbide in molten fluoride salt. Tritium can become trapped in radiation defects of silicon carbide. Thus, an understanding of the potential for tritium absorption, impurities reactions and thermal gradient-assisted corrosion mechanisms along with tritium recovery and redox control systems are necessary to mitigate silicon carbide corrosion in molten fluoride salt systems. Here, we survey current research on silicon carbide corrosion in molten fluoride salts and critically evaluate the research and development gaps.

Molten salt reactors (MSRs) are being considered as one of the potential nuclear options to meet future energy demands. While the MSR designs are drastically different from the more traditional light water reactor, many of the waste streams are similar between the concepts. The purpose of this paper is to outline strategies for the treatment and processing of MSR-type wastes from concepts of reconditioning and recycle of certain components to partitioning and direct immobilization of other waste components. To help bridge science and technology gaps, knowledge gained from similar efforts such as pyroprocessing of Experimental Breeder Reactor II salt wastes can be leveraged to develop concept-to-disposition pathways.

Oxidation of nuclear graphites produces microstructural changes that affect the elastic moduli of these materials. It is widely accepted that the primary effect of oxidation is to increase porosity, but the related effect on the moduli cannot be explained satisfactorily by simply noting changes to porosity. In this work, models describing the elastic moduli of porous, polycrystalline graphite materials are developed to interpret experimental determinations of Young's modulus and shear modulus in two grades of nuclear graphite – IG-110 and NBG-18 – that were oxidized to produce varying levels of porosity. Experimental measurements were carried out using laser-based ultrasonic methods and were interpreted successfully using models that take into account the effects of preferential oxidation of different elements of the graphite microstructure. The results indicate the importance of the processes that lead to increased porosity since these can heavily influence the nature of the resulting structure-property relationships.

Microstructural changes induced by neutron irradiation of superfine grain graphite G347A (Tokai Carbon, Japan) were examined by nitrogen adsorption at 77 K and by three microscopy techniques (SEM, TEM and FIB-SEM tomography). The specimens were irradiated at doses of up to 30 dpa, covering stages before and after the turnaround fluence at three temperatures (300, 450, 750 °C) of their irradiation envelope. The initial graphite densification at low fluences did not produce any detectable effect in the pore size range ( 50 nm) at high irradiation fluences and more structural changes on multiple scales, from nanometers to microns. This work demonstrates the unique ability of gas adsorption techniques to analyze open pores with sizes between sub-nanometer and sub-micron in bulk nuclear graphite, with supporting microscopy results.

This manuscript provides raw nitrogen gas adsorption data, images and videos obtained from a technique that combines Focused Ion Beam (FIB) and Scanning Electron Microscopy (SEM) known as FIB-SEM tomography and Transmission Electron Microscopy (TEM) micrographs. This collection of data is useful for characterization of the effects of high fluence neutron irradiation in nuclear graphite as described in the associated manuscript, “Mesopores development in superfine grain graphite neutron-irradiated at high fluence” (Contescu et al., 2019). Nitrogen adsorption isotherms at 77 K are provided for graphite samples before and after neutron irradiation at 300, 450, and 750 °C at fluences before and after turnaround. FIB-SEM tomography reveals porosity of unirradiated and irradiated samples. Using this technique, four data sets were obtained, of which the first three are presented in video format, whereas the fourth one is a series of images provided in raw format unique to this manuscript. All microscopy data document the microstructure, surface area and porosity of superfine grain graphite G347A (Tokai Carbon, Japan) before irradiation and irradiated after turnaround at 400 °C. TEM micrographs provide unique information on irradiation damage at high neutron fluence (>27. 8 displacements per atom, dpa) in the microstructure and crystal lattice of graphite. Additional TEM micrographs are provided here, which do not duplicate the research paper published elsewhere (Contescu et al., 2019). These data sets are unique, as samples at high irradiation doses have never been measured or imaged before with the aforementioned techniques.

Heterozygous loss of X-linked genes like CASK and MeCP2 (Rett syndrome) causes neurodevelopmental disorders (NDD) in girls, while in boys such loss leads to profound encephalopathy. The cellular basis for these disorders remains unknown. CASK is presumed to work through the Tbr1-reelin pathway in neuronal migration during brain development. Here we report our clinical and histopathological analysis of a deceased 2-month-old boy with a CASK-null mutation. We demonstrate that although smaller in size, the CASK-null human brain exhibits normal lamination without defective neuronal differentiation, migration, or axonal guidance, excluding the role of reelin in CASK-linked pathology. The disproportionately hypoplastic cerebellum in humans without CASK expression is associated with cerebellar astrogliosis, a marker for neuronal loss. Cerebellum-specific deletion in mouse confirms a post-developmental degeneration of cerebellar granular neurons that results in a small cerebellum. Mechanistically, cerebellar hypoplasia in CASK mutation thus results from neurodegeneration rather that developmental defects. Zygosity-pathology correlation suggests that NDDs like CASK mutation and Rett syndrome are pathologically neurodegenerative; however, random X-chromosome inactivation in the typical heterozygous mutant girls results in 50% of cells expressing the functional gene, resulting in a non-progressive pathology, whereas complete loss of the only allele in boys leads to unconstrained degeneration and encephalopathy.One sentence summary of studyCASK loss causes cerebellar degeneration. The authors have declared that no conflict of interest exists.

The BLL lectin from the edible Japanese “Kurokawa” mushroom (Boletopsis leucomelaena) was previously reported to bind to N-glycans harboring terminal N-acetylglucosamine (GlcNAc) and to induce apoptosis in a leukemia cell line. However, its gene has not been reported. In this study, we used a transcriptomics-based workflow to identify a full-length transcript of a BLL functional ortholog (termed BGL) from Boletopsis grisea, a close North American relative of B. leucomelaena. The deduced amino acid sequence of BGL was an obvious member of fungal fruit body lectin family (Pfam PF07367), a highly conserved group of mushroom lectins with a preference for binding O-glycans harboring the Thomsen–Friedenreich antigen (TF-antigen; Galβ1,3GalNAc-α-) and having two ligand binding sites. Functional characterization of recombinant BGL using glycan microarray analysis and surface plasmon resonance confirmed its ability to bind both the TF-antigen and β-GlcNAc-terminated N-glycans. Structure-guided mutagenesis of BGL’s two ligand binding clefts showed that one site is responsible for binding TF-antigen structures associated with O-glycans, whereas the second site specifically recognizes N-glycans with terminal β-GlcNAc. Additionally, the two sites show no evidence of allosteric communication. Finally, mutant BGL proteins having single functional bindings site were used to enrich GlcNAc-capped N-glycans or mucin type O-glycopeptides from complex samples in glycomics and glycoproteomics analytical workflows.

The CERN IT Storage Group ensures the symbiotic development and operations of storage and data transfer services for all CERN physics data, in particular the data generated by the four LHC experiments (ALICE, ATLAS, CMS and LHCb). In order to accomplish the objectives of the next run of the LHC (Run-3), the Storage Group has undertaken a thorough analysis of the experiments’ requirements, matching them to the appropriate storage and data transfer solutions, and undergoing a rigorous programme of testing to identify and solve any issues before the start of Run-3. In this paper, we present the main challenges presented by each of the four LHC experiments. We describe their workflows, in particular how they communicate with and use the key components provided by the Storage Group: the EOS disk storage system; its archival back-end, the CERN Tape Archive (CTA); and the File Transfer Service (FTS). We also describe the validation and commissioning tests that have been undertaken and challenges overcome: the ATLAS stress tests to push their DAQ system to its limits; the CMS migration from PhEDEx to Rucio, followed by large-scale tests between EOS and CTA with the new FTS “archive monitoring” feature; the LHCb Tier-0 to Tier-1 staging tests and XRootD Third Party Copy (TPC) validation; and the erasure coding performance in ALICE.

Analysis of genomic DNA from pathogenic strains of Burkholderia cenocepacia J2315 and Escherichia coli O104:H4 revealed the presence of two unusual MTase genes. Both are plasmid-borne ORFs, carried by pBCA072 for B. cenocepacia J2315 and pESBL for E. coli O104:H4. Pacific Biosciences SMRT sequencing was used to investigate DNA methyltransferases M.BceJIII and M.EcoGIX, using artificial constructs. Mating properties of engineered pESBL derivatives were also investigated. Both MTases yield promiscuous m6A modification of single strands, in the context SAY (where S = C or G and Y = C or T). Strikingly, this methylation is asymmetric in vivo, detected almost exclusively on one DNA strand, and is incomplete: typically, around 40% of susceptible motifs are modified. Genetic and biochemical studies suggest that enzyme action depends on replication mode: DNA Polymerase I (PolI)-dependent ColE1 and p15A origins support asymmetric modification, while the PolI-independent pSC101 origin does not. An MTase-PolI complex may enable discrimination of PolI-dependent and independent plasmid origins. M.EcoGIX helps to establish pESBL in new hosts by blocking the action of restriction enzymes, in an orientation-dependent fashion. Expression and action appear to occur on the entering single strand in the recipient, early in conjugal transfer, until lagging-strand replication creates the double-stranded form.