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106. Sickle cell disease: at the crossroads of pulmonary hypertension and diastolic heart failure.

113. Nitric Oxide–Independent Soluble Guanylate Cyclase Activation Improves Vascular Function and Cardiac Remodeling in Sickle Cell Disease

115. Detection of brain cardiolipins in plasma after cardiac arrest

118. Cytochrome b5 Reductase 3 Modulates Soluble Guanylate Cyclase Redox State and cGMP Signaling

123. TSP1–CD47 signaling is upregulated in clinical pulmonary hypertension and contributes to pulmonary arterial vasculopathy and dysfunction

126. Erratum: Corrigendum: KLF4-dependent phenotypic modulation of smooth muscle cells has a key role in atherosclerotic plaque pathogenesis

132. Structure Guided Chemical Modifications of Propylthiouracil Reveal Novel Small Molecule Inhibitors of Cytochrome b5 Reductase 3 That Increase Nitric Oxide Bioavailability

133. Hemoglobin α/eNOS Coupling at Myoendothelial Junctions Is Required for Nitric Oxide Scavenging During Vasoconstriction

136. Mechanisms for Arsenic-Stimulated Sinusoidal Endothelial Cell Capillarization

140. S-Nitrosylation Inhibits Pannexin 1 Channel Function

141. MAPK Phosphorylation of Connexin 43 Promotes Binding of Cyclin E and Smooth Muscle Cell Proliferation

146. Pannexin1 Regulates α1-Adrenergic Receptor– Mediated Vasoconstriction

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