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102. Monitoring oxygen delivery in the critically ill.
- Author
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Huang YC and Huang, Yuh-Chin Tony
- Abstract
An accurate assessment of regional tissue oxygen delivery (DO(2)) may help the intensivist to attenuate end-organ damage in critically ill patients. Transport of oxygen from the ambient air to the mitochondria occurs by convection and diffusion, and is tightly regulated by neural and humoral factors. This article reviews the basic principles of DO(2) and the abnormal oxygen supply-demand relationship seen in patients with shock. It also discusses approaches to monitoring DO(2), including clinical symptoms/signs, acid-base status, and gas exchange, which provide global assessment, as well as gastric tonometry, which may reflect regional DO(2). Some new experimental methods, such as near-infrared spectroscopy and positron emission tomography, are still in development but may in the future provide useful clinical devices for quantifying the adequacy of regional tissue oxygenation in critically ill patients. [ABSTRACT FROM AUTHOR]
- Published
- 2005
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103. Constraints on dark photon dark matter using data from LIGO's and Virgo's third observing run
- Author
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Abbott, R., Abbott, T. D., Acernese, F., Ackley, K., Adams, C., Adhikari, N., Adhikari, R. X., Adya, V. B., Affeldt, C., Agarwal, D., Agathos, M., Agatsuma, K., Aggarwal, N., Aguiar, O. D., Aiello, L., Ain, A., Ajith, P., Akutsu, T., Albanesi, S., Allocca, A., Altin, P. A., Amato, A., Ananyeva, A., Anderson, S. B., Anderson, W. G., Ando, M., Andrade, T., Andres, N., Angelova, V, Ansoldi, S., Antelis, J. M., Antier, S., Appert, S., Arai, Koji, Arai, Koya, Arai, Y., Araki, S., Araya, A., Araya, M. C., Areeda, J. S., Arene, M., Aritomi, N., Arnaud, N., Aronson, S. M., Arun, K. G., Asada, H., Asali, Y., Ashton, G., Aso, Y., Assiduo, M., Aston, S. M., Astone, P., Aubin, F., Austin, C., Babak, S., Badaracco, F., Bader, M. K. M., Badger, C., Bae, S., Bae, Y., Baer, A. M., Bagnasco, S., Bai, Y., Baiotti, L., Baird, J., Bajpai, R., Ball, M., Ballardin, G., Ballmer, S. W., Balsamo, A., Baltus, G., Banagiri, S., Bankar, D., Barayoga, J. C., Barbieri, C., Barish, B. C., Barker, D., Barneo, P., Barone, F., Barr, B., Barsotti, L., Barsuglia, M., Barta, D., Bartlett, J., Barton, M. A., Bartos, I, Bassiri, R., Basti, A., Bawaj, M., Bayley, J. C., Baylor, A. C., Bazzan, M., Becsy, B., Bedakihale, V. M., Bejger, M., Belahcene, I, Benedetto, V, Beniwal, D., Bennett, T. F., Bentley, J. D., Benyaala, M., Bergamin, F., Berger, B. K., Bernuzzi, S., Bersanetti, D., Bertolini, A., Betzwieser, J., Beveridge, D., Are, R., Bhardwaj, U., Bhattacharjee, D., Bhaumik, S., Bilenko, I. A., Billingsley, G., Bini, S., Birney, R., Birnholtz, O., Biscans, S., Bischi, M., Biscoveanu, S., Bisht, A., Biswas, B., Bitossi, M., Bizouard, M-A, Blackburn, J. K., Blair, C. D., Blair, D. G., Blair, R. M., Bobba, F., Bode, N., Boer, M., Bogaert, G., Boldrini, M., Bonavena, L. D., Bondu, F., Bonilla, E., Bonn, Booker, P., Boom, B. A., Bork, R., Boschi, V, Bose, N., Bose, S., Bossilkov, V, Boudart, V, Bouffanais, Y., Bozzi, A., Bradaschia, C., Brady, P. R., Bramley, A., Branch, A., Branchesi, M., Brau, J. E., Breschi, M., Briant, T., Briggs, J. H., Brillet, A., Brinkmann, M., Brockill, P., Brooks, A. F., Brooks, J., Brown, D. D., Brunett, S., Bruno, G., Bruntz, R., Bryant, J., Bulik, T., Bulten, H. J., Buonanno, A., Buscicchio, R., Buskulic, D., Buy, C., Byer, R. L., Cadonati, L., Cagnoli, G., Cahillane, C., Calderon Bustillo, J., Callaghan, J. D., Callister, T. A., Calloni, E., Cameron, J., Camp, J. B., Canepa, M., Canevarolo, S., Cannavacciuolo, M., Cannon, K. C., Cao, H., Cao, Z., Capocasa, E., Capote, E., Carapella, G., Carbognani, F., Carlin, J. B., Carney, M. F., Carpinelli, M., Carrillo, G., Carullo, G., Carver, T. L., Diaz, J. Casanueva, Casentini, C., Castaldi, G., Caudill, S., Cavaglia, M., Cavalier, F., Cavalieri, R., Ceasar, M., Cella, G., Cerda-Duran, P., Cesarini, E., Chaibi, W., Chakravarti, K., Subrahmanya, S. Chalathadka, Champion, E., Chan, C-H, Chan, C., Chan, C. L., Chan, K., Chan, M., Ra, K., Chanial, P., Chao, S., Charlton, P., Chase, E. A., Chass, e-Mottin, E., Chatterjee, C., Chatterjee, Debarati, Chatterjee, Deep, Chaturvedi, M., Chaty, S., Chen, C., Chen, H. Y., Chen, J., Chen, K., Chen, X., Chen, Y-B, Chen, Y-R, Chen, Z., Cheng, H., Cheong, C. K., Cheung, H. Y., Chia, H. Y., Chiadini, F., Chiang, C-Y, Chiarini, G., Chierici, R., Chincarini, A., Chiofalo, M. L., Chiummo, A., Cho, G., Cho, H. S., Choudhary, R. K., Choudhary, S., Christensen, N., Chu, H., Chu, Q., Chu, Y-K, Chua, S., Chung, K. W., Ciani, G., Ciecielag, P., Cifaldi, M., Ciobanu, A. A., Ciolfi, R., Cipriano, F., Cirone, A., Clara, F., Clark, E. N., Clark, J. A., Clarke, L., Clearwater, P., Clesse, S., Cleva, F., Coccia, E., Codazzo, E., Cohadon, P-F, Cohen, D. E., Cohen, L., Colleoni, M., Collette, C. G., Colombo, A., Colpi, M., Compton, C. M., Constancio, J, R., Conti, L., Cooper, S. J., Corban, P., Corbitt, T. R., Cordero-Carrion, I, Corezzi, S., Corley, K. R., Cornish, N., Corre, D., Corsi, A., Cortese, S., Costa, C. A., Cotesta, R., Coughlin, M. W., Coulon, J-P, Countryman, S. T., Cousins, B., Couvares, P., Coward, D. M., Cowart, M. J., Coyne, D. C., Coyne, R., Creighton, J. D. E., Creighton, T. D., Criswell, A. W., Croquette, M., Crowder, S. G., Cudell, J. R., Cullen, T. J., Cumming, A., Cummings, R., Cunningham, L., Cuoco, E., Dabadie, P., Dal Canton, T., Dall'Osso, S., Dalya, G., Dana, A., Daneshgaranbajastani, L. M., D'Angelo, B., Danilishin, S., D'Antonio, S., Danzmann, K., Darsow-Fromm, C., Dasgupta, A., Datrier, L. E. H., Datta, S., Dattilo, V, Dave, I, Davier, M., Davies, G. S., Davis, D., Davis, M. C., Daw, E. J., Dean, R., Debra, D., Deenadayalan, M., Degallaix, J., De Laurentis, M., Deleglise, S., Del Favero, V, De Lillo, F., De Lillo, N., Del Pozzo, W., Demarchi, L. M., De Matteis, F., D'Emilio, V, Demos, N., Dent, T., Depasse, A., De Pietri, R., De Rosa, R., De Rossi, C., Desalvo, R., De Simone, R., Dhur, Har, S., Diaz, M. C., Diaz-Ortiz, Didio, N. A., Dietrich, T., Di Fiore, L., Di Fronzo, C., Di Giorgio, C., Di Giovanni, F., Di Giovanni, M., Di Girolamo, T., Di Lieto, A., Ding, B., Di Pace, S., Di Palma, I, Di Renzo, F., Divakarla, A. K., Dmitriev, A., Doctor, Z., D'Onofrio, L., Donovan, F., Dooley, K. L., Doravari, S., Dorrington, I, Drago, M., Driggers, J. C., Drori, Y., Ducoin, J-G, Dupej, P., Durante, O., D'Urso, D., Duverne, P-A, Dwyer, S. E., Eassa, C., Easter, P. J., Ebersold, M., Eckhardt, T., Eddolls, G., Edelman, B., Edo, T. B., Edy, O., Effler, A., Eguchi, S., Eichholz, J., Eikenberry, S. S., Eisenmann, M., Eisenstein, R. A., Ejlli, A., Engelby, E., Enomoto, Y., Errico, L., Essick, R. C., Estelles, H., Estevez, D., Etienne, Z., Etzel, T., Evans, M., Evans, T. M., Ewing, B. E., Fafone, V, Fair, H., Fairhurst, S., Farah, A. M., Farinon, S., Farr, B., Farr, W. M., Farrow, N. W., Fauchon-Jones, E. J., Favaro, G., Favata, M., Fays, M., Fazio, M., Feicht, J., Fejer, M. 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R., Gier, C., Giesler, M., Giri, P., Gissi, F., Glanzer, J., Gleckl, A. E., Godwin, P., Goetz, E., Goetz, R., Gohlke, N., Goncharov, B., Gonzalez, G., Gopakumar, A., Gosselin, M., Gouaty, R., Gould, D. W., Grace, B., Grado, A., Granata, M., Granata, V, Grant, A., Gras, S., Grassia, P., Gray, C., Gray, R., Greco, G., Green, A. C., Green, R., Gretarsson, A. M., Gretarsson, E. M., Griffith, D., Griffiths, W., Griggs, H. L., Grignani, G., Grimaldi, A., Grimm, S. J., Grote, H., Grunewald, S., Gruning, P., Guerra, D., Guidi, G. M., Guimaraes, A. R., Guixe, G., Gulati, H. K., Guo, H-K, Guo, Y., Gupta, Anchal, Gupta, Anuradha, Gupta, P., Gustafson, E. K., Gustafson, R., Guzman, F., Ha, S., Haegel, L., Hagiwara, A., Haino, S., Halim, O., Hall, E. D., Hamilton, E. Z., Hammond, G., Han, W-B, Haney, M., Hanks, J., Hanna, C., Hannam, M. D., Hannuksela, O., Hansen, H., Hansen, T. J., Hanson, J., Harder, T., Hardwick, T., Haris, K., Harms, J., Harry, G. M., Harry, I. W., Hartwig, D., Hasegawa, K., Haskell, B., Hasskew, R. K., Haster, C-J, Hattori, K., Haughian, K., Hayakawa, H., Hayama, K., Hayes, F. J., Healy, J., Heidmann, A., Heidt, A., Heintze, M. C., Heinze, J., Heinzel, J., Heitmann, H., Hellman, F., Hello, P., Helmling-Cornell, A. F., Hemming, G., Hendry, M., Heng, I. S., Hennes, E., Hennig, J., Hennig, M. H., Hern, A. G., Ez, Vivanco, F. Hern, Heurs, M., Hild, S., Hill, P., Himemoto, Y., Hines, A. S., Hiranuma, Y., Hirata, N., Hirose, E., Hochheim, S., Hofman, D., Hohmann, J. N., Holcomb, D. G., Holl, N. A., Hollows, I. J., Holmes, Z. J., Holt, K., Holz, D. E., Hong, Z., Hopkins, P., Hough, J., Hourihane, S., Howell, E. J., Hoy, C. G., Hoyl, Hreibi, A., Hsieh, B-H, Hsu, Y., Huang, G-Z, Huang, H-Y, Huang, P., Huang, Y-C, Huang, Y-J, Huang, Y., Hubner, M. T., Huddart, A. D., Hughey, B., Hui, D. C. Y., Hui, V, Husa, S., Huttner, S. H., Huxford, R., Huynh-Dinh, T., Ide, S., Idzkowski, B., Iess, A., Ikenoue, B., Imam, S., Inayoshi, K., Ingram, C., Inoue, Y., Ioka, K., Isi, M., Isleif, K., Ito, K., Itoh, Y., Iyer, B. R., Izumi, K., Jaberianhamedan, V, Jacqmin, T., Jadhav, S. J., Jadhav, S. P., James, A. L., Jan, A. Z., Jani, K., Janquart, J., Janssens, K., Janthalur, N. N., Jaranowski, P., Jariwala, D., Jaume, R., Jenkins, A. C., Jenner, K., Jeon, C., Jeunon, M., Jia, W., Jin, H-B, Johns, G. R., Jones, A. W., Jones, I, Jones, J. D., Jones, P., Jones, R., Jonker, R. J. G., Ju, L., Jung, P., Jung, K., Junker, J., Juste, V, Kaihotsu, K., Kajita, T., Kakizaki, M., Kalaghatgi, Kalogera, V, Kamai, B., Kamiizumi, M., K, A, N., Hasamy, S., Kang, G., Kanner, J. B., Kao, Y., Kapadia, S. J., Kapasi, D. 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B., Kozakai, C., Kozu, R., Kringel, V, Krishnendu, Krolak, A., Kuehn, G., Kuei, F., Kuijer, P., Kumar, A., Kumar, P., Kumar, Rahul, Kumar, Rakesh, Kume, J., Kuns, K., Kuo, C., Kuo, H-S, Kuromiya, Y., Kuroyanagi, S., Kusayanagi, K., Kuwahara, S., Kwak, K., Lagabbe, P., Laghi, D., Lal, E, E., Lam, T. L., Lamberts, A., L, Ry, M., Lane, B. B., Lang, R. N., Lange, J., Lantz, B., La Rosa, I, Lartaux-Vollard, A., Lasky, P. D., Laxen, M., Lazzarini, A., Lazzaro, C., Leaci, P., Leavey, S., Lecoeuche, Y. K., Lee, H. K., Lee, H. M., Lee, H. W., Lee, J., Lee, K., Lee, R., Lehmann, J., Lemaitre, A., Leonardi, M., Leroy, N., Letendre, N., Levesque, C., Levin, Y., Leviton, J. N., Leyde, K., A. K. Y., Li, Li, B., Li, J., K. L., Li, T. G. F., Li, Li, X., Lin, C-Y, Lin, F-K, Lin, F-L, Lin, H. L., Lin, L. C-C, Linde, F., Linker, S. D., Linley, J. N., Littenberg, T. B., Liu, G. C., Liu, J., Liu, K., Liu, X., Llamas, F., Llorens-Monteagudo, M., R. K. L., Lo, Lockwood, A., London, L. 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S., Patrone, S., Paul, S., Payne, E., Pedraza, M., Pegoraro, M., Pele, A., Arellano, F. E. Pena, Penn, S., Perego, A., Pereira, A., Pereira, T., Perez, C. J., Perigois, C., Perkins, C. C., Perreca, A., Perries, S., Petermann, J., Petterson, D., Pfeiffer, H. P., Pham, K. A., Phukon, K. S., Piccinni, O. J., Pichot, M., Piendibene, M., Piergiovanni, F., Pierini, L., Pierro, V, Pillant, G., Pillas, M., Pilo, F., Pinard, L., Pinto, I. M., Pinto, M., Piotrzkowski, K., Pirello, M., Pitkin, M. D., Placidi, E., Planas, L., Plastino, W., Pluchar, C., Poggiani, R., Polini, E., Pong, D. Y. T., Ponrathnam, S., Popolizio, P., Porter, E. K., Poulton, R., Powell, J., Pracchia, M., Pradier, T., Prajapati, A. K., Prasai, K., Prasanna, R., Pratten, G., Principe, M., Prodi, G. A., Prokhorov, L., Prosposito, P., Prudenzi, L., Puecher, A., Punturo, M., Puosi, F., Puppo, P., Purrer, M., Qi, H., Quetschke, V, Quitzow-James, R., Raab, F. J., Raaijmakers, G., Radkins, H., Radulesco, N., Raffai, P., Rail, S. X., Raja, S., Rajan, C., Ramirez, K. E., Ramirez, T. D., Ramos-Buades, A., Rana, J., Rapagnani, P., Rapol, U. D., Ray, A., Raymond, V, Raza, N., Razzano, M., Read, J., Rees, L. A., Regimbau, T., Rei, L., Reid, S., Reid, S. W., Reitze, D. H., Relton, P., Renzini, A., Rettegno, P., Rezac, M., Ricci, F., Richards, D., Richardson, J. W., Richardson, L., Riemenschneider, G., Riles, K., Rinaldi, S., Rink, K., Rizzo, M., Robertson, N. A., Robie, R., Robinet, F., Rocchi, A., Rodriguez, S., Roll, Rollins, J. G., Romanelli, M., Romano, R., Romel, C. L., Romero-Rodriguez, A., Romero-Shaw, I. M., Romie, J. H., Ronchini, S., Rosa, L., Rose, C. A., Ross, M. P., Rowan, S., Rowlinson, S. J., Roy, S., Roy, Santosh, Roy, Soumen, Rozza, D., Ruggi, P., Ryan, K., Sachdev, S., Sadecki, T., Sadiq, J., Sago, N., Saito, S., Saito, Y., Sakai, K., Sakai, Y., Sakellariadou, M., Sakuno, Y., Salafia, O. S., Salconi, L., Saleem, M., Salemi, F., Samajdar, A., Sanchez, E. J., Sanchez, J. H., Sanchez, L. E., Sanchis-Gual, N., S, Ers, J. R., Sanuy, A., Saravanan, T. R., Sarin, N., Sassolas, B., Satari, H., Sathyaprakash, B. S., Sato, S., Sato, T., Sauter, O., Savage, R. L., Sawada, T., Sawant, D., Sawant, H. L., Sayah, S., Schaetzl, D., Scheel, M., Scheuer, J., Schiworski, M., Schmidt, P., Schmidt, S., Schnabel, R., Schneewind, M., Schofield, R. M. S., Schoenbeck, A., Schulte, B. W., Schutz, B. F., Schwartz, E., Scott, J., Scott, S. M., Seglar-Arroyo, M., Sekiguchi, T., Sekiguchi, Y., Sellers, D., Sengupta, A. S., Sentenac, D., Seo, E. G., Sequino, V, Sergeev, A., Setyawati, Y., Shaffer, T., Shahriar, M. S., Shams, B., Shao, L., Sharma, A., Sharma, P., Shawhan, P., Shcheblanov, N. S., Shibagaki, S., Shikauchi, M., Shimizu, R., Shimoda, T., Shimode, K., Shinkai, H., Shishido, T., Shoda, A., Shoemaker, D. H., Shoemaker, D. M., Shyamsundar, S., Sieniawska, M., Sigg, D., Singer, L. P., Singh, D., Singh, N., Singha, A., Sintes, A. M., Sipala, V, Skliris, V, Slagmolen, B. J. J., Slaven-Blair, T. J., Smetana, J., Smith, J. R., Smith, R. J. E., Soldateschi, J., Somala, S. N., Somiya, K., Son, E. J., Soni, K., Soni, S., Sordini, V, Sorrentino, F., Sorrentino, N., Sotani, H., Soulard, R., Souradeep, T., Sowell, E., Spagnuolo, V, Spencer, A. P., Spera, M., Srinivasan, R., Srivastava, A. K., Srivastava, V, Staats, K., Stachie, C., Steer, D. A., Steinlechner, J., Steinlechner, S., Stops, D. J., Stover, M., Strain, K. A., Strang, L. C., Stratta, G., Strunk, A., Sturani, R., Stuver, A. L., Sudhagar, S., Sudhir, V, Sugimoto, R., Suh, H. G., Summerscales, T. Z., Sun, H., Sun, L., Sunil, S., Sur, A., Suresh, J., Sutton, P. J., Suzuki, Takamasa, Suzuki, Toshikazu, Swinkels, B. L., Szewczyk, P., Tacca, M., Tagoshi, H., Tait, S. C., Takahashi, H., Takahashi, R., Takamori, A., Takano, S., Takeda, H., Takeda, M., Talbot, C. J., Talbot, C., Tanaka, H., Tanaka, Kazuyuki, Tanaka, Kenta, Tanaka, Taiki, Tanaka, Takahiro, Tanasijczuk, A. J., Tanioka, S., Tanner, D. B., Tao, D., Tao, L., San Martin, E. N. Tapia, Taranto, C., Tasson, J. D., Telada, S., Tenorio, R., Terhune, J. E., Terkowski, L., Thirugnanasamb, M. P., Am, Thomas, M., Thomas, P., Thompson, J. E., Thondapu, S. R., Thorne, K. A., Thrane, E., Tiwari, Shubhanshu, Tiwari, Srishti, Tiwari, V, Toivonen, A. M., Tol, Tolley, A. E., Tomaru, T., Tomigami, Y., Tomura, T., Tonelli, M., Torres-Forne, A., Torrie, Melo, I. Tosta E., Toyra, D., Trapananti, A., Travasso, F., Traylor, G., Trevor, M., Tringali, M. C., Tripathee, A., Troiano, L., Trovato, A., Trozzo, L., Trudeau, R. J., Tsai, D. S., Tsai, D., Tsang, K. W., Tsang, T., Tsao, J-S, Tse, M., Tso, R., Tsubono, K., Tsuchida, S., Tsukada, L., Tsuna, D., Tsutsui, T., Tsuzuki, T., Turbang, K., Turconi, M., Tuyenbayev, D., Ubhi, A. S., Uchikata, N., Uchiyama, T., Udall, R. P., Ueda, A., Uehara, T., Ueno, K., Ueshima, G., Uraguchi, F., Urban, A. L., Ushiba, T., Utina, A., Vahlbruch, H., Vajente, G., Vajpeyi, A., Valdes, G., Valentini, M., Valsan, V., van Bakel, N., van Beuzekom, M., van den Br, J. F. J., Van den Broeck, C., er-Hyde, D. C., van der Schaaf, L., Van, Heijningen, Vanosky, J., van Putten, M. H. P. M., van Remortel, N., Vardaro, M., Vargas, A. F., Varma, V., Vasuth, M., Vecchio, A., Vedovato, G., Veitch, J., Veitch, P. J., Venneberg, J., Venugopalan, G., Verkindt, D., Verma, P., Verma, Y., Veske, D., Vetrano, F., Vicere, A., Vidyant, S., Viets, A. D., Vijaykumar, A., Villa-Ortega, V., Vinet, J-Y, Virtuoso, A., Vitale, S., Vo, T., Vocca, H., von Reis, E. R. G., von Wrangel, J. S. A., Vorvick, C., Vyatchanin, S. P., Wade, L. E., Wade, M., Wagner, K. J., Walet, R. C., Walker, M., Wallace, G. S., Wallace, L., Walsh, S., Wang, J., Wang, J. Z., Wang, W. H., Ward, R. L., Warner, J., Was, M., Washimi, T., Washington, N. Y., Watchi, J., Weaver, B., Webster, S. A., Weinert, M., Weinstein, A. J., Weiss, R., Weller, C. M., Wellmann, F., Wen, L., Wessels, P., Wette, K., Whelan, J. T., White, D. D., Whiting, B. F., Whittle, C., Wilken, D., Williams, D., Williams, M. J., Williamson, A. R., Willis, J. L., Willke, B., Wilson, D. J., Winkler, W., Wipf, C. C., Wlodarczyk, T., Woan, G., Woehler, J., Wofford, J. K., Wong, I. C. F., Wu, C., D. S., Wu, Wu, H., Wu, S., Wysocki, D. M., Xiao, L., W-R, Xu, Yamada, T., Yamamoto, H., Yamamoto, Kazuhiro, Yamamoto, Kohei, Yamamoto, T., Yamashita, K., Yamazaki, R., Yang, F. W., Yang, L., Yang, Y., Yang, Yang, Yang, Z., Yap, M. J., Yeeles, D. W., Yelikar, A. B., Ying, M., Yokogawa, K., Yokoyama, J., Yokozawa, T., Yoo, J., Yoshioka, T., Hang, Yu, Haocun, Yu, Yuzurihara, H., Zanolin, M., Zeidler, S., Zelenova, T., Zendri, J-P, Zevin, M., Zhan, M., Zhang, H., Zhang, J., Zhang, L., Zhang, T., Zhang, Y., Zhao, C., Zhao, G., Zhao, Y., Zhao, Yue, Zhou, R., Zhou, Z., Zhu, X. J., Zhu, Z-H, Zucker, M. 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D., Acernese, F., Ackley, K., Adams, C., Adhikari, N., Adhikari, R. X., Adya, V. B., Affeldt, C., Agarwal, D., Agathos, M., Agatsuma, K., Aggarwal, N., Aguiar, O. D., Aiello, L., Ain, A., Ajith, P., Akutsu, T., Albanesi, S., Allocca, A., Altin, P. A., Amato, A., Anand, C., Anand, S., Ananyeva, A., Anderson, S. B., Anderson, W. G., Ando, M., Andrade, T., Andres, N., Andric, T., Angelova, S. V., Ansoldi, S., Antelis, J. M., Antier, S., Appert, S., Arai, K., Arai, Y., Araki, S., Araya, A., Araya, M. C., Areeda, J. S., Arene, M., Aritomi, N., Arnaud, N., Aronson, S. M., Arun, K. G., Asada, H., Asali, Y., Ashton, G., Aso, Y., Assiduo, M., Aston, S. M., Astone, P., Aubin, F., Austin, C., Babak, S., Badaracco, F., Bader, M. K. M., Badger, C., Bae, S., Bae, Y., Baer, A. M., Bagnasco, S., Bai, Y., Baiotti, L., Baird, J., Bajpai, R., Ball, M., Ballardin, G., Ballmer, S. W., Balsamo, A., Baltus, G., Banagiri, S., Bankar, D., Barayoga, J. C., Barbieri, C., Barish, B. C., Barker, D., Barneo, P., Barone, F., Barr, B., Barsotti, L., Barsuglia, M., Barta, D., Bartlett, J., Barton, M. A., Bartos, I., Bassiri, R., Basti, A., Bawaj, M., Bayley, J. C., Baylor, A. C., Bazzan, M., Becsy, B., Bedakihale, V. M., Bejger, M., Belahcene, I., Benedetto, V., Beniwal, D., Bennett, T. F., Bentley, J. D., Benyaala, M., Bergamin, F., Berger, B. K., Bernuzzi, S., Bersanetti, D., Bertolini, A., Betzwieser, J., Beveridge, D., Bhandare, R., Bhardwaj, U., Bhattacharjee, D., Bhaumik, S., Bilenko, I. A., Billingsley, G., Bini, S., Birney, R., Birnholtz, O., Biscans, S., Bischi, M., Biscoveanu, S., Bisht, A., Biswas, B., Bitossi, M., Bizouard, M. -A., Blackburn, J. K., Blair, C. D., Blair, D. G., Blair, R. M., Bobba, F., Bode, N., Boer, M., Bogaert, G., Boldrini, M., Bonavena, L. D., Bondu, F., Bonilla, E., Bonnand, R., Booker, P., Boom, B. A., Bork, R., Boschi, V., Bose, N., Bose, S., Bossilkov, V., Boudart, V., Bouffanais, Y., Bozzi, A., Bradaschia, C., Brady, P. 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L., Chan, K., Chan, M., Chandra, K., Chanial, P., Chao, S., Charlton, P., Chase, E. A., Chassande-Mottin, E., Chatterjee, C., Chatterjee, D., Chaturvedi, M., Chaty, S., Chen, C., Chen, H. Y., Chen, J., Chen, K., Chen, X., Chen, Y. -B., Chen, Y. -R., Chen, Z., Cheng, H., Cheong, C. K., Cheung, H. Y., Chia, H. Y., Chiadini, F., Chiang, C. -Y., Chiarini, G., Chierici, R., Chincarini, A., Chiofalo, M. L., Chiummo, A., Cho, G., Cho, H. S., Choudhary, R. K., Choudhary, S., Christensen, N., Chu, H., Chu, Q., Chu, Y. -K., Chua, S., Chung, K. W., Ciani, G., Ciecielag, P., Cieslar, M., Cifaldi, M., Ciobanu, A. A., Ciolfi, R., Cipriano, F., Cirone, A., Clara, F., Clark, E. N., Clark, J. A., Clarke, L., Clearwater, P., Clesse, S., Cleva, F., Coccia, E., Codazzo, E., Cohadon, P. -F., Cohen, D. E., Cohen, L., Colleoni, M., Collette, C. G., Colombo, A., Colpi, M., Compton, C. M., Constancio, M., Conti, L., Cooper, S. J., Corban, P., Corbitt, T. R., Cordero-Carrion, I., Corezzi, S., Corley, K. R., Cornish, N., Corre, D., Corsi, A., Cortese, S., Costa, C. A., Cotesta, R., Coughlin, M. W., Coulon, J. -P., Countryman, S. T., Cousins, B., Couvares, P., Coward, D. M., Cowart, M. J., Coyne, D. C., Coyne, R., Creighton, J. D. E., Creighton, T. D., Criswell, A. W., Croquette, M., Crowder, S. G., Cudell, J. R., Cullen, T. J., Cumming, A., Cummings, R., Cunningham, L., Cuoco, E., Curylo, M., Dabadie, P., Canton, T. D., Dall'Osso, S., Dalya, G., Dana, A., Daneshgaranbajastani, L. M., D'Angelo, B., Danilishin, S., D'Antonio, S., Danzmann, K., Darsow-Fromm, C., Dasgupta, A., Datrier, L. E. H., Datta, S., Dattilo, V., Dave, I., Davier, M., Davies, G. S., Davis, D., Davis, M. C., Daw, E. J., Dean, R., Debra, D., Deenadayalan, M., Degallaix, J., De Laurentis, M., Deleglise, S., Del Favero, V., De Lillo, F., De Lillo, N., Del Pozzo, W., Demarchi, L. M., De Matteis, F., D'Emilio, V., Demos, N., Dent, T., Depasse, A., De Pietri, R., De Rosa, R., De Rossi, C., Desalvo, R., De Simone, R., Dhurandhar, S., Diaz, M. C., Diaz-Ortiz, M., Didio, N. A., Dietrich, T., Di Fiore, L., Di Fronzo, C., Di Giorgio, C., Di Giovanni, F., Di Giovanni, M., Di Girolamo, T., Di Lieto, A., Ding, B., Di Pace, S., Di Palma, I., Di Renzo, F., Divakarla, A. K., Dmitriev, A., Doctor, Z., D'Onofrio, L., Donovan, F., Dooley, K. L., Doravari, S., Dorrington, I., Drago, M., Driggers, J. C., Drori, Y., Ducoin, J. -G., Dupej, P., Durante, O., D'Urso, D., Duverne, P. -A., Dwyer, S. E., Eassa, C., Easter, P. J., Ebersold, M., Eckhardt, T., Eddolls, G., Edelman, B., Edo, T. B., Edy, O., Effler, A., Eguchi, S., Eichholz, J., Eikenberry, S. S., Eisenmann, M., Eisenstein, R. A., Ejlli, A., Engelby, E., Enomoto, Y., Errico, L., Essick, R. C., Estelles, H., Estevez, D., Etienne, Z., Etzel, T., Evans, M., Evans, T. M., Ewing, B. 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L., Ushiba, T., Utina, A., Vahlbruch, H., Vajente, G., Vajpeyi, A., Valdes, G., Valentini, M., Valsan, V., Van Bakel, N., Van Beuzekom, M., Van Den Brand, J. F. J., Van Den Broeck, C., Vander-Hyde, D. C., Van Der Schaaf, L., Van Heijningen, J. V., Vanosky, J., Van Putten, M. H. P. M., Van Remortel, N., Vardaro, M., Vargas, A. F., Varma, V., Vasuth, M., Vecchio, A., Vedovato, G., Veitch, J., Veitch, P. J., Venneberg, J., Venugopalan, G., Verkindt, D., Verma, P., Verma, Y., Veske, D., Vetrano, F., Vicere, A., Vidyant, S., Viets, A. D., Vijaykumar, A., Villa-Ortega, V., Vinet, J. -Y., Virtuoso, A., Vitale, S., Vo, T., Vocca, H., Von Reis, E. R. G., Von Wrangel, J. S. A., Vorvick, C., Vyatchanin, S. P., Wade, L. E., Wade, M., Wagner, K. J., Walet, R. C., Walker, M., Wallace, G. S., Wallace, L., Walsh, S., Wang, J., Wang, J. Z., Wang, W. H., Ward, R. L., Warner, J., Was, M., Washimi, T., Washington, N. Y., Watchi, J., Weaver, B., Webster, S. A., Weinert, M., Weinstein, A. J., Weiss, R., Weller, C. M., Wellmann, F., Wen, L., Wessels, P., Wette, K., Whelan, J. T., White, D. D., Whiting, B. F., Whittle, C., Wilken, D., Williams, D., Williams, M. J., Williamson, A. R., Willis, J. L., Willke, B., Wilson, D. J., Winkler, W., Wipf, C. C., Wlodarczyk, T., Woan, G., Woehler, J., Wofford, J. K., Wong, I. C. F., Wu, C., Wu, D. S., Wu, H., Wu, S., Wysocki, D. M., Xiao, L., Xu, W. -R., Yamada, T., Yamamoto, H., Yamamoto, K., Yamamoto, T., Yamashita, K., Yamazaki, R., Yang, F. W., Yang, L., Yang, Y., Yang, Z., Yap, M. J., Yeeles, D. W., Yelikar, A. B., Ying, M., Yokogawa, K., Yokoyama, J., Yokozawa, T., Yoo, J., Yoshioka, T., Yu, H., Yuzurihara, H., Zadrozny, A., Zanolin, M., Zeidler, S., Zelenova, T., Zendri, J. -P., Zevin, M., Zhan, M., Zhang, H., Zhang, J., Zhang, L., Zhang, T., Zhang, Y., Zhao, C., Zhao, G., Zhao, Y., Zhou, R., Zhou, Z., Zhu, X. J., Zhu, Z. -H., Zucker, M. E., Zweizig, J., Andrić, T., Arai, Koji, Arai, Koya, Arène, M., Bécsy, B., Bustillo, J. Calderón, Diaz, J. Casanueva, Cavaglià, M., Cerdá-Durán, P., Subrahmanya, S. Chalathadka, Chatterjee, Debarati, Chatterjee, Deep, Chiang, C-Y., Chu, Y-K., Cieślar, M., Cordero-Carrión, I., Curyło, M., Canton, T. Dal, Dall’Osso, S., Dálya, G., D’Angelo, B., D’Antonio, S., Deléglise, S., D’Emilio, V., Díaz, M. C., D’Onofrio, L., D’Urso, D., Estellés, H., Fronzé, G. G., García-Núñez, C., García-Quirós, C., Ghosh, Abhirup, Ghosh, Archisman, Ghosh, Shaon, Ghosh, Shrobana, González, G., Guixé, G., Gupta, Anchal, Gupta, Anuradha, Vivanco, F. Hernandez, Hsieh, B-H., Huang, G-Z., Huang, H-Y., Huang, Y-C., Hübner, M. T., Kéfélian, F., Królak, A., Kumar, Rahul, Kumar, Rakesh, Kuo, H-S., Lemaître, A., Lin, C-Y., Lin, F-K., Lin, F-L., Portilla, M. Lopez, Lück, H., Hernandez, I. Magaña, Magazzù, C., Márka, S., Márka, Z., Mir, Ll. M., Miravet-Tenés, M., Mo, Geoffrey, Mukherjee, Arunava, Mukherjee, Soma, Mukherjee, Subroto, Mukherjee, Suvodip, Muñiz, E. A., Quynh, L. Nguyen, O’Brien, B. D., O’Dell, J., O’Reilly, B., O’Shaughnessy, R., O’Shea, E., Arellano, F. E. Peña, Périgois, C., Perriès, S., Pinto, I. M., Pürrer, M., Romero-Rodríguez, A., Rosińska, D., Roy, Santosh, Roy, Soumen, Schönbeck, A., Suzuki, Takamasa, Suzuki, Toshikazu, Szczepańczyk, M. J., Tanaka, Kazuyuki, Tanaka, Kenta, Tanaka, Taiki, Tanaka, Takahiro, Martin, E. N. Tapia San, Martín, E. N. Tapia San, Tiwari, Shubhanshu, Tiwari, Srishti, Torres-Forné, A., e Melo, I. Tosta, Töyrä, D., Tsao, J-S., van Bakel, N., van Beuzekom, M., van den Brand, J. F. J., van der Schaaf, L., van Heijningen, J. V., van Putten, M. H. P. M., van Remortel, N., Vasúth, M., Viceré, A., von Reis, E. R. G., von Wrangel, J. S. A., Weßels, P., Xu, W-R., Yamamoto, Kazuhiro, Yamamoto, Kohei, Yang, Yang, Yu, Hang, Yu, Haocun, Zadrożny, A., Zhao, Yue, Other Research IHEF (IoP, FNWI), High Energy Astrophys. & Astropart. Phys (API, FNWI), Astroparticle Physics (IHEF, IoP, FNWI), Gravitation and Astroparticle Physics Amsterdam, IoP (FNWI), The LIGO Scientific Collaboration, The Virgo Collaboration, The KAGRA Collaboration, Institut National des Sciences Appliquées (INSA)-Université de Rennes (UNIV-RENNES)-Institut National des Sciences Appliquées (INSA)-École Nationale Supérieure des Sciences Appliquées et de Technologie (ENSSAT)-Centre National de la Recherche Scientifique (CNRS)-IMT Atlantique (IMT Atlantique), Université de Strasbourg (UNISTRA)-Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3)-Centre National de la Recherche Scientifique (CNRS), RS: FSE Grav. waves and fundamental physics, Grav. waves and fundamental physics, RS: FSE MSP, Abbott, R, Abbott, Td, Acernese, F, Ackley, K, Adams, C, Adhikari, N, Adhikari, Rx, Adya, Vb, Affeldt, C, Agarwal, D, Agathos, M, Agatsuma, K, Aggarwal, N, Aguiar, Od, Aiello, L, Ain, A, Ajith, P, Akutsu, T, Albanesi, S, Allocca, A, Altin, Pa, Amato, A, Anand, C, Anand, S, Ananyeva, A, Anderson, Sb, Anderson, Wg, Ando, M, Andrade, T, Andres, N, Angelova, Sv, Ansoldi, S, Antelis, Jm, Antier, S, Appert, S, Arai, K, Arai, Y, Araki, S, Araya, A, Araya, Mc, Areeda, J, Arene, M, Aritomi, N, Arnaud, N, Aronson, Sm, Arun, Kg, Asada, H, Asali, Y, Ashton, G, Aso, Y, Assiduo, M, Aston, Sm, Astone, P, Aubin, F, Austin, C, Babak, S, Badaracco, F, Bader, Mkm, Badger, C, Bae, S, Bae, Y, Baer, Am, Bagnasco, S, Bai, Y, Baiotti, L, Baird, J, Bajpai, R, Ball, M, Ballardin, G, Ballmer, Sw, Balsamo, A, Baltus, G, Banagiri, S, Bankar, D, Barayoga, Jc, Barbieri, C, Barish, Bc, Barker, D, Barneo, P, Barone, F, Barr, B, Barsotti, L, Barsuglia, M, Barta, D, Bartlett, J, Barton, Ma, Bartos, I, Bassiri, R, Basti, A, Bawaj, M, Bayley, Jc, Baylor, Ac, Bazzan, M, Becsy, B, Bedakihale, Vm, Bejger, M, Belahcene, I, Benedetto, V, Beniwal, D, Bennett, Tf, Bentley, Jd, Benyaala, M, Bergamin, F, Berger, Bk, Bernuzzi, S, Bersanetti, D, Bertolini, A, Betzwieser, J, Beveridge, D, Bhandare, R, Bhardwaj, U, Bhattacharjee, D, Bhaumik, S, Bilenko, Ia, Billingsley, G, Bini, S, Birney, R, Birnholtz, O, Biscans, S, Bischi, M, Biscoveanu, S, Bisht, A, Biswas, B, Bitossi, M, Bizouard, Ma, Blackburn, Jk, Blair, Cd, Blair, Dg, Blair, Rm, Bobba, F, Bode, N, Boer, M, Bogaert, G, Boldrini, M, Bonavena, Ld, Bondu, F, Bonilla, E, Bonnand, R, Booker, P, Boom, Ba, Bork, R, Boschi, V, Bose, N, Bose, S, Bossilkov, V, Boudart, V, Bouffanais, Y, Bozzi, A, Bradaschia, C, Brady, Pr, Bramley, A, Branch, A, Branchesi, M, Brau, Je, Breschi, M, Briant, T, Briggs, Jh, Brillet, A, Brinkmann, M, Brockill, P, Brooks, Af, Brooks, J, Brown, Dd, Brunett, S, Bruno, G, Bruntz, R, Bryant, J, Bulik, T, Bulten, Hj, Buonanno, A, Buscicchio, R, Buskulic, D, Buy, C, Byer, Rl, Cadonati, L, Cagnoli, G, Cahillane, C, Bustillo, Jc, Callaghan, Jd, Callister, Ta, Calloni, E, Cameron, J, Camp, Jb, Canepa, M, Canevarolo, S, Cannavacciuolo, M, Cannon, Kc, Cao, H, Cao, Z, Capocasa, E, Capote, E, Carapella, G, Carbognani, F, Carlin, Jb, Carney, Mf, Carpinelli, M, Carrillo, G, Carullo, G, Carver, Tl, Diaz, Jc, Casentini, C, Castaldi, G, Caudill, S, Cavaglia, M, Cavalier, F, Cavalieri, R, Ceasar, M, Cella, G, Cerda-Duran, P, Cesarini, E, Chaibi, W, Chakravarti, K, Subrahmanya, Sc, Champion, E, Chan, Ch, Chan, C, Chan, Cl, Chan, K, Chan, M, Chandra, K, Chanial, P, Chao, S, Charlton, P, Chase, Ea, Chassande-Mottin, E, Chatterjee, C, Chatterjee, D, Chaturvedi, M, Chaty, S, Chen, C, Chen, Hy, Chen, J, Chen, K, Chen, X, Chen, Yb, Chen, Yr, Chen, Z, Cheng, H, Cheong, Ck, Cheung, Hy, Chia, Hy, Chiadini, F, Chiang, Cy, Chiarini, G, Chierici, R, Chincarini, A, Chiofalo, Ml, Chiummo, A, Cho, G, Cho, H, Choudhary, Rk, Choudhary, S, Christensen, N, Chu, H, Chu, Q, Chu, Yk, Chua, S, Chung, Kw, Ciani, G, Ciecielag, P, Cifaldi, M, Ciobanu, Aa, Ciolfi, R, Cipriano, F, Cirone, A, Clara, F, Clark, En, Clark, Ja, Clarke, L, Clearwater, P, Clesse, S, Cleva, F, Coccia, E, Codazzo, E, Cohadon, Pf, Cohen, De, Cohen, L, Colleoni, M, Collette, Cg, Colombo, A, Colpi, M, Compton, Cm, Constancio, M, Conti, L, Cooper, Sj, Corban, P, Corbitt, Tr, Cordero-Carrion, I, Corezzi, S, Corley, Kr, Cornish, N, Corre, D, Corsi, A, Cortese, S, Costa, Ca, Cotesta, R, Coughlin, Mw, Coulon, Jp, Countryman, St, Cousins, B, Couvares, P, Coward, Dm, Cowart, Mj, Coyne, Dc, Coyne, R, Creighton, Jde, Creighton, Td, Criswell, Aw, Croquette, M, Crowder, Sg, Cudell, Jr, Cullen, Tj, Cumming, A, Cummings, R, Cunningham, L, Cuoco, E, Dabadie, P, Dal Canton, T, Dall'Osso, S, Dalya, G, Dana, A, Daneshgaranbajastani, Lm, D'Angelo, B, Danilishin, S, D'Antonio, S, Danzmann, K, Darsow-Fromm, C, Dasgupta, A, Datrier, Leh, Datta, S, Dattilo, V, Dave, I, Davier, M, Davies, G, Davis, D, Davis, Mc, Daw, Ej, Dean, R, Debra, D, Deenadayalan, M, Degallaix, J, De Laurentis, M, Deleglise, S, Del Favero, V, De Lillo, F, De Lillo, N, Del Pozzo, W, Demarchi, Lm, De Matteis, F, D'Emilio, V, Demos, N, Dent, T, Depasse, A, De Pietri, R, De Rosa, R, De Rossi, C, Desalvo, R, De Simone, R, Dhurandhar, S, Diaz, Mc, Diaz-Ortiz, M, Didio, Na, Dietrich, T, Di Fiore, L, Di Fronzo, C, Di Giorgio, C, Di Giovanni, F, Di Giovanni, M, Di Girolamo, T, Di Lieto, A, Ding, B, Di Pace, S, Di Palma, I, Di Renzo, F, Divakarla, Ak, Dmitriev, A, Doctor, Z, D'Onofrio, L, Donovan, F, Dooley, Kl, Doravari, S, Dorrington, I, Drago, M, Driggers, Jc, Drori, Y, Ducoin, Jg, Dupej, P, Durante, O, D'Urso, D, Duverne, Pa, Dwyer, Se, Eassa, C, Easter, Pj, Ebersold, M, Eckhardt, T, Eddolls, G, Edelman, B, Edo, Tb, Edy, O, Effler, A, Eguchi, S, Eichholz, J, Eikenberry, S, Eisenmann, M, Eisenstein, Ra, Ejlli, A, Engelby, E, Enomoto, Y, Errico, L, Essick, Rc, Estelles, H, Estevez, D, Etienne, Z, Etzel, T, Evans, M, Evans, Tm, Ewing, Be, Fafone, V, Fair, H, Fairhurst, S, Farah, Am, Farinon, S, Farr, B, Farr, Wm, Farrow, Nw, Fauchon-Jones, Ej, Favaro, G, Favata, M, Fays, M, Fazio, M, Feicht, J, Fejer, Mm, Fenyvesi, E, Ferguson, Dl, Fernandez-Galiana, A, Ferrante, I, Ferreira, Ta, Fidecaro, F, Figura, P, Fiori, I, Fishbach, M, Fisher, Rp, Fittipaldi, R, Fiumara, V, Flaminio, R, Floden, E, Fong, H, Font, Ja, Fornal, B, Forsyth, Pwf, Franke, A, Frasca, S, Frasconi, F, Frederick, C, Freed, Jp, Frei, Z, Freise, A, Frey, R, Fritschel, P, Frolov, Vv, Fronze, Gg, Fujii, Y, Fujikawa, Y, Fukunaga, M, Fukushima, M, Fulda, P, Fyffe, M, Gabbard, Ha, Gadre, Bu, Gair, Jr, Gais, J, Galaudage, S, Gamba, R, Ganapathy, D, Ganguly, A, Gao, D, Gaonkar, Sg, Garaventa, B, Garcia-Nunez, C, Garcia-Quiros, C, Garufi, F, Gateley, B, Gaudio, S, Gayathri, V, Ge, Gg, Gemme, G, Gennai, A, George, J, Gerberding, O, Gergely, L, Gewecke, P, Ghonge, S, Ghosh, A, Ghosh, S, Giacomazzo, B, Giacoppo, L, Giaime, Ja, Giardina, Kd, Gibson, Dr, Gier, C, Giesler, M, Giri, P, Gissi, F, Glanzer, J, Gleckl, Ae, Godwin, P, Goetz, E, Goetz, R, Gohlke, N, Goncharov, B, Gonzalez, G, Gopakumar, A, Gosselin, M, Gouaty, R, Gould, Dw, Grace, B, Grado, A, Granata, M, Granata, V, Grant, A, Gras, S, Grassia, P, Gray, C, Gray, R, Greco, G, Green, Ac, Green, R, Gretarsson, Am, Gretarsson, Em, Griffith, D, Griffiths, W, Griggs, Hl, Grignani, G, Grimaldi, A, Grimm, Sj, Grote, H, Grunewald, S, Gruning, P, Guerra, D, Guidi, Gm, Guimaraes, Ar, Guixe, G, Gulati, Hk, Guo, Hk, Guo, Y, Gupta, A, Gupta, P, Gustafson, Ek, Gustafson, R, Guzman, F, Ha, S, Haegel, L, Hagiwara, A, Haino, S, Halim, O, Hall, Ed, Hamilton, Ez, Hammond, G, Han, Wb, Haney, M, Hanks, J, Hanna, C, Hannam, Md, Hannuksela, O, Hansen, H, Hansen, Tj, Hanson, J, Harder, T, Hardwick, T, Haris, K, Harms, J, Harry, Gm, Harry, Iw, Hartwig, D, Hasegawa, K, Haskell, B, Hasskew, Rk, Haster, Cj, Hattori, K, Haughian, K, Hayakawa, H, Hayama, K, Hayes, Fj, Healy, J, Heidmann, A, Heidt, A, Heintze, Mc, Heinze, J, Heinzel, J, Heitmann, H, Hellman, F, Hello, P, Helmling-Cornell, Af, Hemming, G, Hendry, M, Heng, I, Hennes, E, Hennig, J, Hennig, Mh, Hernandez, Ag, Vivanco, Fh, Heurs, M, Hild, S, Hill, P, Himemoto, Y, Hines, A, Hiranuma, Y, Hirata, N, Hirose, E, Hochheim, S, Hofman, D, Hohmann, Jn, Holcomb, Dg, Holland, Na, Hollows, Ij, Holmes, Zj, Holt, K, Holz, De, Hong, Z, Hopkins, P, Hough, J, Hourihane, S, Howell, Ej, Hoy, Cg, Hoyland, D, Hreibi, A, Hsieh, Bh, Hsu, Y, Huang, Gz, Huang, Hy, Huang, P, Huang, Yc, Huang, Yj, Huang, Y, Hubner, Mt, Huddart, Ad, Hughey, B, Hui, Dcy, Hui, V, Husa, S, Huttner, Sh, Huxford, R, Huynh-Dinh, T, Ide, S, Idzkowski, B, Iess, A, Ikenoue, B, Imam, S, Inayoshi, K, Ingram, C, Inoue, Y, Ioka, K, Isi, M, Isleif, K, Ito, K, Itoh, Y, Iyer, Br, Izumi, K, Jaberianhamedan, V, Jacqmin, T, Jadhav, Sj, Jadhav, Sp, James, Al, Jan, Az, Jani, K, Janquart, J, Janssens, K, Janthalur, Nn, Jaranowski, P, Jariwala, D, Jaume, R, Jenkins, Ac, Jenner, K, Jeon, C, Jeunon, M, Jia, W, Jin, Hb, Johns, Gr, Jones, Aw, Jones, Di, Jones, Jd, Jones, P, Jones, R, Jonker, Rjg, Ju, L, Jung, P, Jung, K, Junker, J, Juste, V, Kaihotsu, K, Kajita, T, Kakizaki, M, Kalaghatgi, Cv, Kalogera, V, Kamai, B, Kamiizumi, M, Kanda, N, Kandhasamy, S, Kang, G, Kanner, Jb, Kao, Y, Kapadia, Sj, Kapasi, Dp, Karat, S, Karathanasis, C, Karki, S, Kashyap, R, Kasprzack, M, Kastaun, W, Katsanevas, S, Katsavounidis, E, Katzman, W, Kaur, T, Kawabe, K, Kawaguchi, K, Kawai, N, Kawasaki, T, Kefelian, F, Keitel, D, Key, J, Khadka, S, Khalili, Fy, Khan, S, Khazanov, Ea, Khetan, N, Khursheed, M, Kijbunchoo, N, Kim, C, Kim, Jc, Kim, J, Kim, K, Kim, W, Kim, Ym, Kimball, C, Kimura, N, Kinley-Hanlon, M, Kirchhoff, R, Kissel, J, Kita, N, Kitazawa, H, Kleybolte, L, Klimenko, S, Knee, Am, Knowles, Td, Knyazev, E, Koch, P, Koekoek, G, Kojima, Y, Kokeyama, K, Koley, S, Kolitsidou, P, Kolstein, M, Komori, K, Kondrashov, V, Kong, Akh, Kontos, A, Koper, N, Korobko, M, Kotake, K, Kovalam, M, Kozak, Db, Kozakai, C, Kozu, R, Kringel, V, Krishnendu, Nv, Krolak, A, Kuehn, G, Kuei, F, Kuijer, P, Kumar, A, Kumar, P, Kumar, R, Kume, J, Kuns, K, Kuo, C, Kuo, H, Kuromiya, Y, Kuroyanagi, S, Kusayanagi, K, Kuwahara, S, Kwak, K, Lagabbe, P, Laghi, D, Lalande, E, Lam, Tl, Lamberts, A, Landry, M, Lane, Bb, Lang, Rn, Lange, J, Lantz, B, La Rosa, I, Lartaux-Vollard, A, Lasky, Pd, Laxen, M, Lazzarini, A, Lazzaro, C, Leaci, P, Leavey, S, Lecoeuche, Yk, Lee, Hk, Lee, Hm, Lee, Hw, Lee, J, Lee, K, Lee, R, Lehmann, J, Lemaitre, A, Leonardi, M, Leroy, N, Letendre, N, Levesque, C, Levin, Y, Leviton, Jn, Leyde, K, Li, Aky, Li, B, Li, J, Li, Kl, Li, Tgf, Li, X, Lin, Cy, Lin, Fk, Lin, Fl, Lin, Hl, Lin, Lcc, Linde, F, Linker, Sd, Linley, Jn, Littenberg, Tb, Liu, Gc, Liu, J, Liu, K, Liu, X, Llamas, F, Llorens-Monteagudo, M, Lo, Rkl, Lockwood, A, London, Lt, Longo, A, Lopez, D, Portilla, Ml, Lorenzini, M, Loriette, V, Lormand, M, Losurdo, G, Lott, Tp, Lough, Jd, Lousto, Co, Lovelace, G, Lucaccioni, Jf, Luck, H, Lumaca, D, Lundgren, Ap, Luo, Lw, Lynam, Je, Macas, R, Macinnis, M, Macleod, Dm, Macmillan, Iao, Macquet, A, Hernandez, Im, Magazzu, C, Magee, Rm, Maggiore, R, Magnozzi, M, Mahesh, S, Majorana, E, Makarem, C, Maksimovic, I, Maliakal, S, Malik, A, Man, N, Mandic, V, Mangano, V, Mango, Jl, Mansell, Gl, Manske, M, Mantovani, M, Mapelli, M, Marchesoni, F, Marchio, M, Marion, F, Mark, Z, Marka, S, Marka, Z, Markakis, C, Markosyan, A, Markowitz, A, Maros, E, Marquina, A, Marsat, S, Martelli, F, Martin, Iw, Martin, Rm, Martinez, M, Martinez, Va, Martinez, V, Martinovic, K, Martynov, Dv, Marx, Ej, Masalehdan, H, Mason, K, Massera, E, Masserot, A, Massinger, Tj, Masso-Reid, M, Mastrogiovanni, S, Matas, A, Mateu-Lucena, M, Matichard, F, Matiushechkina, M, Mavalvala, N, Mccann, Jj, Mccarthy, R, Mcclelland, De, Mcclincy, Pk, Mccormick, S, Mcculler, L, Mcghee, Gi, Mcguire, Sc, Mcisaac, C, Mciver, J, Mcrae, T, Mcwilliams, St, Meacher, D, Mehmet, M, Mehta, Ak, Meijer, Q, Melatos, A, Melchor, Da, Mendell, G, Menendez-Vazquez, A, Menoni, C, Mercer, Ra, Mereni, L, Merfeld, K, Merilh, El, Merritt, Jd, Merzougui, M, Meshkov, S, Messenger, C, Messick, C, Meyers, Pm, Meylahn, F, Mhaske, A, Miani, A, 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A, Van Bakel, N, Van Beuzekom, M, Van Den Brand, J, Van Den Broeck, C, Vander-Hyde, D, Van Der Schaaf, L, Van Heijningen, J, Van Putten, M, Van Remortel, N, Vargas, A, Veitch, P, Viets, A, Vinet, J, Von Reis, E, Von Wrangel, J, Vyatchanin, S, Wade, L, Wagner, K, Walet, R, Wang, W, Ward, R, Washington, N, Webster, S, Weinstein, A, Weller, C, Whelan, J, White, D, Whiting, B, Williams, M, Williamson, A, Willis, J, Wilson, D, Wipf, C, Wofford, J, Wong, I, Wysocki, D, Xu, W, Yang, F, Yap, M, Yeeles, D, Yelikar, A, Zadrozny, A, Zendri, J, Zhu, X, Zhu, Z, Zucker, M, (Astro)-Particles Physics, LIGO Sci Collaboration, Virgo Collaboration, and KAGRA Collaboration
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coupling: (dark matter baryon) ,Nuclear and High Energy Physics ,data analysis method ,Cosmology and Nongalactic Astrophysics (astro-ph.CO) ,gr-qc ,Astronomy ,Astrophysics::High Energy Astrophysical Phenomena ,FOS: Physical sciences ,detector: noise ,General Relativity and Quantum Cosmology (gr-qc) ,Astrophysics::Cosmology and Extragalactic Astrophysics ,Astronomy & Astrophysics ,dark matter: direct detection ,coupling: minimal ,General Relativity and Quantum Cosmology ,dark matter ,Physics, Particles & Fields ,gravitational wave detectors ,High Energy Physics - Phenomenology (hep-ph) ,Fourier transformation ,correlation function ,LIGO ,gravitational wave ,QC ,QB ,Settore FIS/01 ,Science & Technology ,mirror: oscillation ,Physics ,gravitational radiation ,tachyon: stability ,hep-ph ,field theory: scalar ,detector: sensitivity ,High Energy Physics - Phenomenology ,cosmic string: decay ,VIRGO ,Physics and Astronomy ,[PHYS.HPHE]Physics [physics]/High Energy Physics - Phenomenology [hep-ph] ,Physical Sciences ,astro-ph.CO ,[PHYS.GRQC]Physics [physics]/General Relativity and Quantum Cosmology [gr-qc] ,gravitational radiation detector: interferometer ,[PHYS.ASTR]Physics [physics]/Astrophysics [astro-ph] ,Astrophysics - Cosmology and Nongalactic Astrophysics - Abstract
LIGO Scientific Collaboration - Virgo Collaboration - KAGRA Collaboration: Abbott, R. et al., We present a search for dark photon dark matter that could couple to gravitational-wave interferometers using data from Advanced LIGO and Virgo’s third observing run. To perform this analysis, we use two methods, one based on cross-correlation of the strain channels in the two nearly aligned LIGO detectors, and one that looks for excess power in the strain channels of the LIGO and Virgo detectors. The excess power method optimizes the Fourier transform coherence time as a function of frequency, to account for the expected signal width due to Doppler modulations. We do not find any evidence of dark photon dark matter with a mass between mA∼10−14–10−11 eV/c2, which corresponds to frequencies between 10–2000 Hz, and therefore provide upper limits on the square of the minimum coupling of dark photons to baryons, i.e., U(1)B dark matter. For the cross-correlation method, the best median constraint on the squared coupling is ∼1.31×10−47 at mA∼4.2×10−13eV/c2; for the other analysis, the best constraint is ∼2.4×10−47 at mA∼5.7×10−13eV/c2. These limits improve upon those obtained in direct dark matter detection experiments by a factor of ∼100 for mA∼[2–4]×10−13eV/c2, and are, in absolute terms, the most stringent constraint so far in a large mass range mA∼2×10−13–8×10−12eV/c2., This material is based upon work supported by NSF’s LIGO Laboratory which is a major facility fully funded by the National Science Foundation. The authors also gratefully acknowledge the support of the Science and Technology Facilities Council (STFC) of the United Kingdom, the Max-Planck-Society (MPS), and the State of Niedersachsen/ Germany for support of the construction of Advanced LIGO and construction and operation of the GEO600 detector. Additional support for Advanced LIGO was provided by the Australian Research Council. The authors gratefully acknowledge the Italian Istituto Nazionale di Fisica Nucleare (INFN), the French Centre National de la Recherche Scientifique (CNRS) and the Netherlands Organization for Scientific Research, for the construction and operation of the Virgo detector and the creation and support of the EGO consortium. The authors also gratefully acknowledge research support fromthese agencies as well as by the Council of Scientific and Industrial Research of India, the Department of Science and Technology, India, the Science & Engineering Research Board (SERB), India, the Ministry of Human Resource Development, India, the Spanish Agencia Estatal de Investigación, the Vicepresidencia i Conselleria d’Innovació, Recerca i Turisme and the Conselleria d’Educació i Universitat del Govern de les Illes Balears, the Conselleria d’Innovació, Universitats, Ciencia i Societat Digital de la Generalitat Valenciana and the CERCA Programme Generalitat de Catalunya, Spain, the National Science Centre of Poland and the Foundation for Polish Science (FNP), the Swiss National Science Foundation (SNSF), the Russian Foundation for Basic Research, the Russian Science Foundation, the European Commission, the European Regional Development Funds (ERDF), the Royal Society, the Scottish Funding Council, the Scottish Universities Physics Alliance, the Hungarian Scientific Research Fund (OTKA), the French Lyon Institute of Origins (LIO), the Belgian Fonds de la Recherche Scientifique (FRS-FNRS), Actions de Recherche Concert´ees (ARC) and Fonds Wetenschappelijk Onderzoek—Vlaanderen (FWO), Belgium, the Paris Île-de-France Region, the National Research, Development and Innovation Office Hungary (NKFIH), the National Research Foundation of Korea, the Natural Science and Engineering Research Council Canada, Canadian Foundation for Innovation (CFI), the Brazilian Ministry of Science, Technology, and Innovations, the International Center for Theoretical Physics South American Institute for Fundamental Research (ICTPSAIFR), the Research Grants Council of Hong Kong, the National Natural Science Foundation of China (NSFC), the Leverhulme Trust, the Research Corporation, the Ministry of Science and Technology (MOST), Taiwan, the United States Department of Energy, and the Kavli Foundation. The authors gratefully acknowledge the support of the NSF, STFC, INFN and CNRS for provision of computational resources. This work was supported by MEXT, JSPS Leading-edge Research Infrastructure Program, JSPS Grant-in-Aid for Specially Promoted Research 26000005, JSPS Grant-in-Aid for Scientific Research on Innovative Areas 2905: No. JP17H06358, No. JP17H06361 and No. JP17H06364, JSPS Core-to-Core Program A. Advanced Research Networks, JSPS Grant-in-Aid for Scientific Research (S) No. 17H06133, the joint research program of the Institute forCosmic Ray Research, University of Tokyo, National Research Foundation (NRF) and Computing Infrastructure Project of KISTI-GSDC in Korea, Academia Sinica (AS), AS Grid Center (ASGC) and the Ministry of Science and Technology (MoST) in Taiwan under grants including AS-CDA-105-M06, Advanced Technology Center (ATC) of NAOJ, and Mechanical Engineering Center of KEK.
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- 2022
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104. All-sky search for gravitational wave emission from scalar boson clouds around spinning black holes in LIGO O3 data
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Abbott, R., Abe, H., Acernese, F., Ackley, K., Adhikari, N., Adhikari, R. X., Adkins, V. K., Adya, V. B., Affeldt, C., Agarwal, D., Agathos, M., Agatsuma, K., Aggarwal, N., Aguiar, O. D., Aiello, L., Ain, A., Ajith, P., Akutsu, T., Albanesi, S., Alfaidi, R. A., Allocca, A., Altin, P. A., Amato, A., Ananyeva, A., Anderson, S. B., Anderson, W. G., Ando, M., Andrade, T., Andres, N., Andres-Carcasona, M., Angelova, V, Ansoldi, S., Antelis, J. M., Antier, S., Apostolatos, T., Appavuravther, E. Z., Appert, S., Apple, S. K., Arai, K., Araya, A., Araya, M. C., Areeda, J. S., Arene, M., Aritomi, N., Arnaud, N., Arogeti, M., Aronson, S. M., Arun, K. G., Asada, H., Asali, Y., Ashton, G., Aso, Y., Assiduo, M., Melo, S. Assis de Souza, Aston, S. M., Astone, P., Aubin, F., Aultoneal, K., Austin, C., Babak, S., Badaracco, F., Bader, M. K. M., Badger, C., Bae, S., Bae, Y., Baer, A. M., Bagnasco, S., Bai, Y., Baird, J., Bajpai, R., Baka, T., Ball, M., Ballardin, G., Ballmer, S. 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Angelova, Sv, Antelis, Jm, Appavuravther, Ez, Apple, Sk, Araya, Mc, Aronson, Sm, Arun, Kg, Melo, Sad, Aston, Sm, Bader, Mkm, Baer, Am, Ballmer, Sw, Barayoga, Jc, Barish, Bc, Barton, Ma, Bayley, Jc, Becher, Br, Bedakihale, Vm, Benjamin, Mg, Bennett, Tf, Bentley, Jd, Berger, Bk, Bhandari, Av, Bilenko, Ia, Bizouard, Ma, Blackburn, Jk, Blair, Cd, Blair, Dg, Blair, Rm, Bolingbroke, Gn, Bonavena, Ld, Boom, Ba, Brady, Pr, Brau, Je, Briggs, Jh, Brooks, Af, Brown, Dd, Bulten, Hj, Byer, Rl, Davies, Gsc, Bustillo, Jc, Callaghan, Jd, Callister, Ta, Camp, Jb, Cannon, Kc, Carlin, Jb, Carney, Mf, Carver, Tl, Diaz, Jc, Subrahmanya, Sc, Chan, Ch, Chan, Cl, Chang, Ip, Chase, Ea, Chen, Hy, Chen, Yb, Chen, Yr, Cheong, Ck, Cheung, Hy, Chia, Hy, Chiang, Cy, Chiofalo, Ml, Choudhary, Rk, Chu, Yk, Chua, Ssy, Chung, Kw, Ciobanu, Aa, Clark, Ja, Cohadon, Pf, Cohen, De, Collette, Cg, Compton, Cm, Cooper, Sj, Corbitt, Tr, Corley, Kr, Cornish, Nj, Costa, Ca, Coughlin, Mw, Coulon, Jp, Countryman, St, Coward, Dm, Cowart, Mj, Coyne, Dc, Creighton, Jde, Creighton, Td, Criswell, Aw, Crowder, Sg, Cudell, Jr, Cullen, Tj, Dal Canton, T, Datrier, Leh, Davis, Mc, Daw, Ej, Demarchi, Lm, Diaz, Mc, Didio, Na, Divakarla, Ak, Dooley, Kl, Driggers, Jc, Ducoin, Jg, Duverne, Pa, Dwyer, Se, Easter, Pj, Edo, Tb, Eisenstein, Ra, Essick, Rc, Evans, Tm, Ewing, Be, Fan, Pc, Farah, Am, Farr, Wm, Fauchon-Jones, Ej, Fejer, Mm, Ferguson, Dl, Ferreira, Ta, Fisher, Rp, Fong, Hk, Font, Ja, Forsyth, Pwf, Freed, Jp, Frolov, Vv, Fronze, Gg, Gabbard, Ha, Gadre, Bu, Gair, Jr, Gaonkar, Sg, Nunez, Cg, Ge, Gg, Giaime, Ja, Giardina, Kd, Gibson, Dr, Gleckl, Ae, Gould, Dw, Green, Ac, Gretarsson, Am, Gretarsson, Em, Griffiths, Wl, Griggs, Hl, Grimm, Sj, Guidi, Gm, Guimaraes, Ar, Gulati, Hk, Gunny, Am, Guo, Hk, Gupta, Im, Gupta, Sk, Hadiputrawan, Ipw, Hall, Ed, Hamilton, Ez, Han, Wb, Hannam, Md, Hansen, Tj, Harry, Gm, Harry, Iw, Haster, Cj, Hayes, Fj, Heintze, Mc, Helmling-Cornell, Af, Hennig, Mh, Hernandez, Ag, Vivanco, Fh, Hewitt, Al, Ho, Tc, Hohmann, Jn, Holcomb, Dg, Holland, Na, Hollows, Ij, Holmes, Zj, Holz, De, Howell, Ej, Hoy, Cg, Hsieh, Bh, Hsieh, Hf, Huang, Hy, Huang, Yc, Huang, Yj, Huang, Yt, Huang, Yw, Hubner, Mt, Huddart, Ad, Hui, Dcy, Huttner, Sh, Iyer, Br, Jacquet, Pe, Jadhav, Sj, Jadhav, Sp, James, Al, Jan, Az, Janthalur, Nn, Jenkins, Ac, Jin, Hb, Johns, Gr, Jones, Aw, Jones, Di, Kalaghatgi, Cv, Kanner, Jb, Kapadia, Sj, Kapasi, Dp, Khalili, Fy, Khazanov, Ea, Kim, Jc, Kim, Ym, Knee, Am, Knowles, Td, Kong, Akh, Kozak, Db, Krishnendu, Nv, Lam, Tl, Lane, Bb, Lang, Rn, Lasky, Pd, Lecoeuche, Yk, Lee, Hm, Lee, Hw, Legred, In, Leviton, Jn, Li, Aky, Li, Kl, Li, Tgf, Lin, Cy, Lin, Et, Lin, Fk, Lin, Fl, Lin, Hl, Lin, Lcc, Linker, Sd, Linley, Jn, Littenberg, Tb, Liu, Gc, Lo, Rkl, London, Lt, Portilla, Ml, Lott, Tp, Lough, Jd, Lousto, Co, Lucaccioni, Jf, Lundgren, Ap, Luo, Lw, Lynam, Je, Machtinger, Jb, Macleod, Dm, Macmillan, Iao, Hernandez, Im, Magee, Rm, Mansell, Gl, Pina, Dm, Martin, Iw, Martin, Rm, Martinez, Va, Martynov, Dv, Marx, Ej, Mccann, Jj, Mcclelland, De, Mcclincy, Pk, Mcghee, Gi, Mcguire, Sc, Mcwilliams, St, Mehta, Ak, Melchor, Da, Mercer, Ra, Merilh, El, Merritt, Jd, Meyers, Pm, Mihaylov, Dp, Miller, Al, Mills, Jc, Mir, Lm, Mitrofanov, Vp, Modafferi, Lm, Mohapatra, Srp, Mohite, Sr, Moore, Cj, Mow-Lowry, Cm, Muniz, Ea, Murray, Pg, Nadji, Sl, Nayak, Rk, Neil, Bf, Nelson, Tjn, Ng, Ky, Ng, Sw, Quynh, Ln, Ni, Wt, Nichols, Sa, Nuttall, Lk, O'Brien, Bd, Oh, Jj, Oh, Sh, Okada, Ma, Ormiston, Rg, Ormsby, Nd, Ottaway, Dj, Pace, Ae, Page, Ma, Pai, Sa, Palamos, Jr, Pan, Kc, Panda, Pk, Pang, Pth, Pant, Bc, Panther, Fh, Arellano, Fep, Perez, Cj, Perkins, Cc, Pfeiffer, Hp, Pham, Ka, Piccinni, Oj, Pinto, Im, Piotrzkowski, Bj, Pitkin, Md, Planas, Ml, Pong, Dyt, Porter, Ek, Prajapati, Ak, Prodi, Ga, Quinonez, Pj, Raab, Fj, Rail, Sx, Ramirez, Ke, Ramirez, Td, Rees, La, Reid, Sw, Reitze, Dh, Richardson, Jw, Robertson, Na, Rollins, Jg, Romel, Cl, Romero-Shaw, Im, Romie, Jh, Rose, Ca, Ross, Mp, Rowlinson, Sj, Sanchez, Ej, Sanchez, Jh, Sanchez, Le, Sanders, Jr, Saravanan, Tr, Savage, Rl, Sawant, Hl, Schiworski, Mg, Schofield, Rm, Schulte, Bw, Schutz, Bf, Scott, Sm, Seo, Eg, Shaikh, Ma, Shoemaker, Dh, Shoemaker, Dm, Singer, Lp, Singh, Mk, Sintes, Am, Slagmolen, Bjj, Slaven-Blair, Tj, Smith, Jr, Smith, Rje, Somala, Sn, Spencer, Ap, Srivastava, Ak, Steer, Da, Stops, Dj, Strain, Ka, Strang, Lc, Strong, Md, Stuver, Al, Suh, Hg, Sullivan, Ag, Summerscales, Tz, Sutton, Pj, Swinkels, Bl, Tait, Sc, Talbot, Cj, Tanasijczuk, Aj, Tanner, Db, Tapia, Rd, San Martin, Ent, Tasson, Jd, Terhune, Je, Thirugnanasambandam, Mp, Thompson, Ee, Thompson, Je, Thondapu, Sr, Thorne, Ka, Toivonen, Am, Tolley, Ae, Torrie, Ci, Melo, Ite, Tringali, Mc, Trudeau, Rj, Tsang, Kw, Udall, Rp, Urban, Al, van Bakel, N, van Beuzekom, M, van Dael, M, van den Brand, Jfj, Van den Broeck, C, Vander-Hyde, Dc, van Haevermaet, H, van Heijningen, Jv, van Putten, Mhpm, van Remortel, N, Vargas, Af, Veitch, Pj, Vermeulen, Sm, Viets, Ad, Vinet, Jy, von Reis, Erg, von Wrangel, Jsa, Vyatchanin, Sp, Wade, Le, Wagner, Kj, Walet, Rc, Wang, Jz, Wang, Wh, Ward, Rl, Washington, Ny, Weaving, Cr, Webster, Sa, Weinstein, Aj, Weller, Cm, Weller, Ra, Whelan, Jt, White, Dd, Whiting, Bf, Williams, Mj, Williamson, Ar, Willis, Jl, Wilson, Dj, Wipf, Cc, Wofford, Jk, Wong, Icf, Wysocki, Dm, Yang, Fw, Yang, Kz, Yang, Yc, Yap, Mj, Yeeles, Dw, Yeh, Sw, Yelikar, Ab, Yu, Hc, Zendri, Jp, Zhu, Xj, Zhu, Zh, Zimmerman, Ab, Zucker, Me, and (Astro)-Particles Physics
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Nuclear and High Energy Physics ,gravitational wave emission ,scalar boson clouds ,spinning black holes ,ligo o3 data ,FOS: Physical sciences ,Astronomy & Astrophysics ,Physics, Particles & Fields ,General Relativity and Quantum Cosmology ,Particle dark matter ,QC ,Particle astrophysic ,QB ,astro-ph.HE ,High Energy Astrophysical Phenomena (astro-ph.HE) ,Science & Technology ,Physics ,Classical black hole ,SDG 10 - Reduced Inequalities ,black holes ,Physics and Astronomy ,gravitational waves ,[SDU]Sciences of the Universe [physics] ,Physical Sciences ,Gravitational wave detection ,Gravitational wave source ,Astrophysics - High Energy Astrophysical Phenomena - Abstract
This paper describes the first all-sky search for long-duration, quasi-monochromatic gravitational-wave signals emitted by ultralight scalar boson clouds around spinning black holes using data from the third observing run of Advanced LIGO. We analyze the frequency range from 20~Hz to 610~Hz, over a small frequency derivative range around zero, and use multiple frequency resolutions to be robust towards possible signal frequency wanderings. Outliers from this search are followed up using two different methods, one more suitable for nearly monochromatic signals, and the other more robust towards frequency fluctuations. We do not find any evidence for such signals and set upper limits on the signal strain amplitude, the most stringent being $\approx10^{-25}$ at around 130~Hz. We interpret these upper limits as both an "exclusion region" in the boson mass/black hole mass plane and the maximum detectable distance for a given boson mass, based on an assumption of the age of the black hole/boson cloud system., Comment: 28 pages, 16 figures
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- 2022
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105. Neck dissection field and lymph node density predict prognosis in patients with oral cavity cancer and pathological node metastases treated with adjuvant therapy.
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Liao CT, Hsueh C, Lee LY, Lin CY, Fan KH, Wang HM, Huang SF, Chen IH, Kang CJ, Ng SH, Tsao CK, Huang YC, and Yen TC
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Pathological lymph node metastases (pN+) are an established prognostic factor in oral cavity squamous cell carcinoma (OSCC). We retrospectively examined the prognostic significance of lymph node (LN) density in pN+ OSCC patients who underwent neck dissection (ND) and postoperative adjuvant therapy. We examined 309 pN+ patients who underwent levels I-III ND and 148 pN+ patients treated with levels I-V ND. The 5-year control and survival rates served as the main outcome measures. The 5-year rates for patients treated with levels I-III and I-V NDs were as follows: local control, 79%, 74% (p=0.0630); neck control, 81%, 68% (p=0.0014); distant metastasis, 21%, 36% (p=0.0003); disease-free survival (DFS), 59%, 43% (p=0.0001); disease-specific survival (DSS), 66%, 46% (p<0.0001); and overall survival (OS), 49%, 37% (p=0.0048), respectively. Multivariate analysis demonstrated that an LN density 0.16 was an independent prognostic factor for 5-year neck control (all data presented as p, hazard ratio [95% confidence interval]) (0.003, 2.691 [1.412-5.128]), distant metastases (0.001, 2.831 [1.520-5.270]), DFS (<0.001, 2.464 [1.571-3.866]), and DSS (0.036, 1.781 [1.040-3.052]) in levels I-III ND patients. An LN density 0.048 was an independent predictor of 5-year local control (0.004, 4.871 [1.654-14.344]), neck control (0.002, 24.738 [3.367-181.771]), DFS (<0.001, 4.151 [2.264-7.610]), DSS (<0.001, 3.791 [2.017-7.125]), and OS (<0.001, 2.806 [1.706-4.613]) in levels I-V ND patients. Our findings demonstrate the prognostic value of LN density for guiding treatment strategies in OSCC patients who are to receive adjuvant therapy. [ABSTRACT FROM AUTHOR]
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- 2012
106. Outcome analysis of patients with well-differentiated oral cavity squamous cell carcinoma.
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Kang CJ, Liao CT, Hsueh C, Lee LY, Lin CY, Fan KH, Wang HM, Huang SF, Chen IH, Ng SH, Tsao CK, Huang YC, and Yen TC
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The prognosis of well-differentiated oral cavity squamous cell carcinoma (OSCC) is better than less-well-differentiated neoplasms. The aim of this retrospective study was to identify prognostic factors in patients with well-differentiated OSCC. The 5-year outcomes of 467 patients with well-differentiated OSCC who underwent radical surgery and neck dissection were analyzed. In the entire cohort, the presence of pathological node metastases (pN+ vs. pN0) was an independent predictor of 5-year outcomes. In pN0 patients, tumor depth (8mm) was the only independently prognostic factor for 5-year survival rates on multivariable analysis (disease-free survival [DFS], P=0.001, hazard ratio [HR]=2.634, 95% confidence interval [95% CI]=1.496-4.636; disease-specific survival [DSS], P<0.001, HR=6.794, 95% CI=2.364-19.525). In pN+ patients, level IV/V neck nodal metastases (DFS, P<0.001, HR=47.483, 95% CI=8.942-252.122; DSS, P<0.001, HR=14.301, 95% CI=5.337-38.323), and 3 positive nodes (DFS, P=0.037, HR=2.107, 95% CI=1.047-4.242; DSS, P=0.044, HR=2.093, 95% CI=1.020-4.295) were independently associated with 5-year outcomes. Our results suggest that a tailored treatment approach in well-differentiated OSCC patients should take into account the presence of either pN0 or pN+ disease. [ABSTRACT FROM AUTHOR]
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- 2011
107. High plasma homocysteine is associated with the risk of coronary artery disease independent of methylenetetrahydrofolate reductase 677C-->T genotypes.
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Lin PT, Huang MC, Lee BJ, Cheng CH, Tsai TP, and Huang YC
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Hyperhomocysteinemia is an independent risk factor for coronary artery disease (CAD). The aim of this study was to investigate the relations between the methylenetetrafolate reductase (MTHFR) 677C-->T genotypes, B-vitamins (folate, vitamin B-12 and B-6), homocysteine and the risk of CAD. In this case-control study, patients who were identified by cardiac catheterization as having at least 50% stenosis of one major coronary artery were assigned to the case group (n=121). Healthy individuals with normal blood biochemical values were assigned to the control group (n=155). Healthy subjects were matched to case subjects for age. The concentrations of plasma homocysteine, serum folate, vitamin B-12, plasma pyridoxal 5'- phosphate (PLP) and MTHFR 677C-->T gene polymorphism were obtained. The T-allele carriers had significantly higher plasma homocysteine concentration compared to subjects with the 677CC genotype. The MTHFR 677C-->T genotypes were associated with plasma homocysteine after adjusting for various potential risk factors in the case and pooled groups. The MTHFR genotypes were found to have no associations with the risk of CAD. However, plasma homocysteine (>or= 12.5 micromol/L) (OR, 3.49; 95% CI, 1.23-9.88) had a significant association with increased risk of CAD even after additionally adjusted folate status. High plasma homocysteine concentration had a direct effect on the risk of CAD independent of MTHFR 677C-->T genotypes. [ABSTRACT FROM AUTHOR]
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- 2008
108. Laparoscopic reoperative choledocholithotomy in elderly patients with prior complicated abdominal operations.
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Lo HC, Hsieh CH, Yeh HT, Huang YC, Chai KC, Lo, Hung-Chieh, Hsieh, Chi-Hsun, Yeh, Hsien-Tang, Huang, Yung-Cheng, and Chai, Kang-Chuang
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- 2011
109. Framing emission gain layers for perovskite light-emitting diodes using polycaprolactone-silver nanoparticles featuring Förster resonance energy transfer and Purcell effects.
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Yan ZL, Wu GD, Chueh CC, Huang YC, Lin BH, Tsai JH, Chen MH, Tseng ZL, Zhou Y, Jeng RJ, and Kuo CC
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In this study, a new emission gain layer for perovskite light-emitting diodes (PeLEDs) is presented to improve their performance. The emission gain layer consisting of absorption-stable silver nanoparticles is prepared using the post-addition method of the polycaprolactone capping agent (PCL@AgNPs-P). This layer (PCL@AgNPs-P) effectively improves the Förster resonance energy transfer (FRET) between the low- n (minor) and high- n (major) phases in a quasi2D perovskite system, thereby increasing the major emission intensity and efficiency. Moreover, this layer also enhances the Purcell effect, thus increasing the spontaneous emission rates and amplifying the electroluminescence. These combined advantages enable the derived PeLED to achieve higher luminance, external quantum efficiency (EQE), and sustained emission purity. As a result, the optimized PeLED with the PCL@AgNPs-P emission gain layer delivers a maximum luminance of 11 320 cd m
-2 and an EQE of 15.5%, and maintains high green wavelength emission purity and a narrow emission half-maximum width at various operating currents. Our results not only provide a robust pathway for the development of high-performance PeLEDs, but also open up the possibilities of applying PeLEDs in laser optics, where enhanced efficiency and emission characteristics are crucial for creating efficient and high-emission laser sources.- Published
- 2025
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110. Partially Degradable N-Type Conjugated Random Copolymers for Intrinsically Stretchable Organic Field-Effect Transistors.
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Chung CH, Huang YC, Su SW, Su CJ, Jeng US, Chen JY, and Lin YC
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In this study, a series of conjugated homopolymers (P1 and P5) and random copolymers (P2-P4) by copolymerizing naphthalene diimide (NDI) as the acceptor with varying ratios of two donor units, thiophene-imine-thiophene (TIT) and thiophene-vinylene-thiophene (TVT) is developed. The inclusion of TIT imparted degradability to the random copolymers under acidic conditions, offering a sustainable solution for electronic waste management. Structural analysis revealed that TIT favored edge-on molecular orientation, while TVT promoted face-on and end-to-end orientations. The synergistic combination of TIT and TVT in copolymerization resulted in balanced structural and functional properties with partial degradability conferred using the TIT units. The random copolymer P3, with an optimal equimolar TIT/TVT ratio, demonstrates superior electrical and mechanical performance. P3 exhibits an initial charge mobility of 0.10 cm
2 V⁻¹ s⁻¹ and maintained mobility of 0.0017 cm2 V⁻¹ s⁻¹ under 20% strain, significantly outperforming P1 in mobility at almost strain levels. P3 also achieved a mobility retention of 31.3% under 20% strain, compared to 12.2% for P5. This study demonstrates that the copolymerization of TIT and TVT enables the fine-tuning of solid-state packing modes and molecular orientations, thereby improving both the stretchability and environmental sustainability of the materials., (© 2025 Wiley‐VCH GmbH.)- Published
- 2025
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111. The Impact of Administrative Districts and Urban Landscape on the Dispersal of Aedes aegypti via Genetic Differentiation.
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Huang CG, Tseng YC, Yu TH, Cheng HC, Tsai TJ, Ting JH, Ho HY, Lee YZ, Chiu YC, Huang YT, Shih TC, Hsiao HY, Wu YJ, Li SY, Huang YC, Yang YC, Chen PQ, Hsiao CY, and Wang HY
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- Animals, Taiwan, Animal Distribution, Genetics, Population, Dengue transmission, Genotype, Genetic Variation, Aedes genetics, Mosquito Vectors genetics, Cities, Polymorphism, Single Nucleotide
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Mosquito-borne diseases affect millions and cause numerous deaths annually. Effective vector control, which hinges on understanding their dispersal, is vital for reducing infection rates. Given the variability in study results, likely due to environmental and human factors, gathering local dispersal data is critical for targeted disease control. To analyse the spread and differentiation of Aedes aegypti in southern Taiwan, we established a dengue vector monitoring network in Southern Taiwan's cities. This network employed GPS-equipped ovitraps to gather eggs that were subsequently hatched in the laboratory and genotyped using genome-wide SNP markers. From 168 individuals, we identified 757,238 SNPs for detailed analysis. The estimated effective dispersal distance was 154 m (95% CI: 126-180 m), consistent with prior mark-release-recapture (MRR) estimates. We discovered that geographic isolation significantly influences genetic differentiation at larger scales, such as between cities, whereas its correlation with genetic distances is considerably weaker at smaller scales, like within cities. This is likely due to the urban landscape in Taiwan, characterised by narrow roads and densely packed buildings, which facilitates extensive dispersal of Ae. aegypti. In evaluating potential barriers to Ae. aegypti dispersal, we found that roads had no significant impact, whereas administrative districts accounted for 4.8% of the population differentiation (p < 10
-4 ). Surprisingly, this variation aligns with the effects of district-specific mosquito control measures implemented at the municipal level. These findings highlight the complex interplay between urban landscapes, administrative measures and Ae. aegypti dispersal, emphasising the need for implementing targeted control strategies that consider these local dynamics., (© 2025 John Wiley & Sons Ltd.)- Published
- 2025
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112. The Impact of Expert Opinions in COVID-19 News Coverage on Vaccination During the Fifth Outbreak in Hong Kong: A Time Series Analysis.
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Gao Y, Lau JT, Zhang Q, Ming WK, Shen F, Huang YC, and Jiang LC
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- Humans, Hong Kong, Expert Testimony, Disease Outbreaks prevention & control, Vaccination, SARS-CoV-2, Public Opinion, COVID-19 prevention & control, COVID-19 epidemiology, Mass Media, COVID-19 Vaccines
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The role of experts in news coverage has become increasingly prominent, but the evidence regarding the effectiveness of expert opinions in affecting public behavior remains mixed. This study seeks to examine the influence of expert opinions covered in the news on the public's response to public health crises. By adopting a macro-level framing perspective, we investigated how framing consistency, a macro-level concept indicating the agreement between expert opinions in news coverage and government policies or among peer experts, evolves over time and its temporal causal relationship with public behavior. Specifically, this study collected all press news coverage in Hong Kong over four months during the fifth outbreak, including 1,416 articles with 650 expert opinions, as well as the vaccination data that paralleled with this period. We constructed time series of expert opinions and vaccination behavior, and then conducted Vector Autoregressive (VAR) models with Granger causality analysis to examine how framing consistency of expert opinions in news coverage influenced vaccination. The results indicate that the consistent framing between expert opinions and government policies increased COVID-19 vaccination during the fifth outbreak in Hong Kong, while conflicting opinions responding to government policies had no significant effect on vaccination. Opinions among medical experts on COVID-19 issues also did not significantly impact vaccination. The implications for designing communication strategies and enhancing public behavioral support during public health crises are discussed.
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- 2025
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113. Investigating undiagnosed Fabry disease in young adults with ischemic stroke: A multicenter cohort study.
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Lin PY, Lin TY, Sung SF, Po HL, Hsu LC, Tang SC, Huang YC, Hsieh CY, Hsu YC, Wu RY, Hsieh CC, Sung PS, and Chen CH
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- Humans, Male, Adult, Female, Middle Aged, Young Adult, Taiwan epidemiology, Prospective Studies, alpha-Galactosidase genetics, Cohort Studies, Ischemic Attack, Transient epidemiology, Ischemic Attack, Transient diagnosis, Sphingolipids blood, Glycolipids blood, Prevalence, Fabry Disease diagnosis, Fabry Disease epidemiology, Fabry Disease complications, Ischemic Stroke epidemiology, Ischemic Stroke diagnosis
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Background: The global prevalence of ischemic stroke in young adults is increasing, leading to a significant social impact. Fabry disease is a recognized cause of ischemic stroke in young patients, and although disease-modifying treatments are available, further evidence is needed to confirm their effectiveness in reducing the incidence of ischemic strokes., Aims: This study aimed to identify undiagnosed Fabry disease in young adult patients with ischemic stroke in a Taiwanese cohort., Methods: This multicenter, prospective cohort study enrolled patients aged 20-55 years who had experienced an ischemic stroke or transient ischemic attack (TIA) within 10 days, from 1 January 2016 to 31 December 2020. Screening for Fabry disease was performed using a dry blood test to measure α-galactosidase activity in male patients and blood globotriaosylsphingosine (lyso-Gb3) levels in female patients. For patients with positive screen results, genetic diagnosis of Fabry disease was pursued through Sanger sequencing of the GLA gene, covering all exons and a segment of intron 4., Results: A total of 977 patients (659 male, 68%) were enrolled from seven hospitals across Taiwan. Four patients (0.4%, all male) had positive screening results, and two patients (0.2%) were genetically diagnosed with Fabry disease. Case 1 had the GLA c.658C>T mutation and experienced ischemic stroke in the bilateral occipital regions. Case 2 had the GLA c.640-801G>A mutation and experienced an ischemic stroke in the left superficial watershed area., Conclusion: The prevalence of undiagnosed Fabry disease in this cohort of Taiwanese young adults with ischemic stroke or TIA was 0.3% among the young male population. Understanding the prevalence of undiagnosed Fabry disease in young adults with ischemic stroke could help shape future Fabry disease screening policies., Data Access Statement: The collected data will be available upon reasonable request from the corresponding author., Competing Interests: Declaration of conflicting interestsThe author(s) declared no potential conflicts of interest with respect to the research, authorship, and/or publication of this article.
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- 2025
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114. Periocular Acupuncture Leading to Retinal Tear and Successful Repairment.
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Huang YC, Wang NK, and Liu PK
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- 2025
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115. Risk prediction model of pedicle screw loosening within 2 years after decompression and instrumented fusion surgery for degenerative lumbar disease.
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Huang YC, Liu PC, Lin HH, Wang ST, Su YP, Chou PH, and Yao YC
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Background Context: Pedicle screw loosening (PSL) after spinal fusion surgery is one of the most frequently reported complications and leads to poor clinical outcomes., Purpose: This study aimed to develop and validate a risk prediction model for PSL within two years in patients undergoing lumbar instrumented fusion surgery based on their risk profiles., Study Design/setting: Retrospective, observational study., Patient Sample: Patients who underwent lumbar instrumented fusion surgery at a single academic institution between May 2015 and February 2019., Outcome Measures: Risk assessment of PSL and development of a rating score based on patient characteristics., Methods: The demographic profiles and radiographic parameters using computed tomography were obtained. These factors were analyzed to determine possible risk factors related to postoperative PSL after 2 years. A scoring system was developed using these independent risk factors and validated using prospectively collected data from another center between May 2019 and December 2021., Results: The occurrence of PSL within 2 years postoperation was 12.7% (40/315). PSL was significantly predicted by smoking, low Hounsfield units (HU) of the pedicle tract at the index level (P), and a low psoas-lumbar vertebral index (PLVI). The risk of PSL according to the categories of the risk score was 1.1% for those with a score of 0-1, 15.1% for a score of 2-3, and 61.5% for a score of 4-6. In validation, this model demonstrated both good discrimination and calibration results. The area under the curve was 0.887 (95% CI 0.830-0.938) for the derivation cohort and 0.835 (95% CI 0.738-0.918) for the external validation cohort., Conclusions: This PSL risk score, including smoking, Index P HU, and PLVI, is a novel approach to predict PSL 2 years post-surgery. This approach highlights the role of factors associated with osteoporosis and sarcopenia in the development of PSL and could aid in preoperative decision-making and surgical planning., Competing Interests: Declaration of competing interest All authors declare no conflict of interest., (Copyright © 2025. Published by Elsevier Inc.)
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- 2025
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116. Latent Profile Analysis of Fatigue Subtypes in Adults with Type 2 Diabetes.
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Kuo HJ, Huang YC, Benner AD, and García AA
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Background: People with type 2 diabetes mellitus (T2DM) commonly report a higher fatigue intensity than the general population. However, effective fatigue management is lacking because little is known about other fatigue characteristics, including timing, distress, and quality, as well as the potential fatigue subtypes experienced in people with T2DM., Objective: To describe fatigue intensity, timing, distress, and quality, and identify fatigue subtypes in people with T2DM., Methods: This cross-sectional, descriptive study included a sample of 150 participants with T2DM recruited from two diabetes outpatient clinics in Taiwan. Fatigue intensity, timing, and distress were measured using the Fatigue Symptom Inventory. Fatigue quality was measured using the Multidimensional Fatigue Inventory. Fatigue subtypes were identified using a latent profile analysis., Results: Participants reported a mild fatigue intensity, experiencing fatigue for about 22% of the day with worse fatigue in the afternoon and evening and having mild disturbances. Three fatigue subtypes were identified. The "high/persistent fatigue with mild distress" subtype was characterized by high fatigue intensity and duration with severe general, physical, and mental fatigue that mildly interfered with functioning. The "moderate/frequent fatigue with minimal distress" group showed moderate levels of fatigue intensity and duration levels with intermediate of general, physical, and mental fatigue and minimal fatigue disturbances. The "no fatigue and distress" subtype was characterized by overall low fatigue scores., Discussion: We identified fatigue characteristics and subtypes in people with T2DM, providing insights into better fatigue management. People with T2DM reported having mild but persistent fatigue. The latent profile analysis revealed that fatigue is likely composed of a mixture of physical and mental components. Nurses should assess both the physical and mental aspects of fatigue while addressing features of the fatigue characteristics in tailored management strategies to alleviate all aspects of fatigue in people with T2DM., Competing Interests: The authors have no conflicts of interest to report., (Copyright © 2025 Wolters Kluwer Health, Inc. All rights reserved.)
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- 2025
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117. Integrating functional proteomics and next generation sequencing reveals potential therapeutic targets for Taiwanese breast cancer.
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Ku WC, Liu CY, Huang CJ, Liao CC, Huang YC, Kong PH, Chen-Chan H, Tseng LM, and Huang CC
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Integrating functional proteomics and next-generation sequencing (NGS) offers a comprehensive approach to unraveling the molecular intricacies of breast cancer. This study investigates the functional interplay between genomic alterations and protein expression in Taiwanese breast cancer patients. By analyzing 61 breast cancer samples using tandem mass tag (TMT) labeling and mass spectrometry, coupled with whole-exome sequencing (WES) or targeted sequencing, we identified key genetic mutations and their impact on protein expression. Notably, pathogenic variants in BRCA1, BRCA2, PTEN, and PIK3CA were found to be clinically relevant, potentially guiding targeted therapy decisions. Additionally, we discovered trans correlations between specific gene alterations (FANCA, HRAS, PIK3CA, MAP2K1, JAK2) and the expression of 22 proteins, suggesting potential molecular mechanisms underlying breast cancer development and progression. These findings highlight the power of integrating proteomics and NGS to identify potential therapeutic targets and enhance personalized medicine strategies for Taiwanese breast cancer patients., Competing Interests: Declarations. Institutional review board statement: The whole study protocol had been reviewed and approved by the Institutional Review Board of Taipei Veterans General Hospital with the approved number: 2022-01-009 C. Informed consent statement: Written informed consent was obtained from all participating patients before enrollment. Competing interests: The authors declare no competing interests., (© 2025. The Author(s).)
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- 2025
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118. Hearing Loss as a Risk Factor for Dementia: A Nationwide 15-Year Follow-Up Cohort Study in Taiwan.
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Lee JF, Lai CC, Chung CH, Weng TH, Huang YC, Huang SH, Wang BL, Yu PC, and Chien WC
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Introduction: Although hearing loss is associated with dementia, the exact causal relationship between hearing loss and dementia remains unclear. Early detection and prevention of hearing loss are essential. In this study, data from the National Health Insurance Research Database (NHIRD) of Taiwan were used to monitor patients with hearing loss for 15 years to determine whether hearing loss leads to dementia., Methods: This retrospective matched-cohort study involved 208,570 individuals, divided into 41,714 patients with hearing loss and 166,856 controls matched by sex, age, and chronic diseases. Cox regression analysis was conducted on data obtained from the NHIRD for the period 2000-2015 to determine the hazard ratio (HR) associated with dementia., Results: The percentage of patients with hearing loss who developed dementia was higher than that of the control group (18.67% vs. 14.10%). The onset age of dementia was younger in the hearing loss group (69.95 vs. 70.31 years, p = 0.001). Middle-aged individuals (45-65 years) with hearing loss were more likely to develop dementia compared with those without hearing loss (48.53% vs. 47.94%, p = 0.03). Compared with the controls, the patients with hearing loss were more likely to develop hyperlipidemia and stroke. In the patients with hearing loss, the adjusted HRs for Alzheimer's disease (AD), vascular dementia, and other types of dementia were significant. The patients were followed up for an average of 7.82 years. Compared with the controls, the patients with hearing loss exhibited a shorter mean time to dementia onset (5.21 vs. 5.49 years, p < 0.001)., Conclusion: Hearing loss may increase the risks of AD, dementia, hyperlipidemia, and stroke. Therefore, health-care providers should closely monitor cardiovascular complications in patients with hearing loss., (S. Karger AG, Basel.)
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- 2025
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119. Machine learning-driven prediction of medical expenses in triple-vessel PCI patients using feature selection.
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Chen KY, Huang YC, Liu CK, Li SJ, and Chen M
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- Humans, Taiwan, Female, Male, Middle Aged, Aged, Coronary Artery Bypass economics, Health Expenditures statistics & numerical data, Health Care Costs statistics & numerical data, Bayes Theorem, Coronary Artery Disease surgery, Coronary Artery Disease therapy, Coronary Artery Disease economics, Percutaneous Coronary Intervention economics, Percutaneous Coronary Intervention statistics & numerical data, Machine Learning
- Abstract
Revascularization therapies, such as percutaneous coronary intervention (PCI) and coronary artery bypass grafting (CABG), alleviate symptoms and treat myocardial ischemia. Patients with multivessel disease, particularly those undergoing 3-vessel PCI, are more susceptible to procedural complications, which can increase healthcare costs. Developing efficient strategies for resource allocation has become a paramount concern due to tightening healthcare budgets and the escalating costs of treating heart conditions. Therefore, it is essential to develop an evaluation model to estimate the costs of PCI surgeries and identify the key factors influencing these costs to enhance healthcare quality. This study utilized the National Health Insurance Research Database (NHIRD), encompassing data from multiple hospitals across Taiwan and covering up to 99% of the population. The study examined data from triple-vessel PCI patients treated between January 2015 and December 2017. Additionally, six machine-learning algorithms and five cross-validation techniques were employed to identify key features and construct the evaluation model. The machine learning algorithms used included linear regression (LR), random forest (RF), support vector regression (SVR), generalized linear model boost (GLMBoost), Bayesian generalized linear model (BayesGLM), and extreme gradient boosting (eXGB). Among these, the eXGB model exhibited outstanding performance, with the following metrics: MSE (0.02419), RMSE (0.15552), and MAPE (0.00755). We found that the patient's medication use in the previous year is also crucial in determining subsequent surgical costs. Additionally, 25 significant features influencing surgical expenses were identified. The top variables included 1-year medical expenditure before PCI surgery (hospitalization and outpatient costs), average blood transfusion volume, ventilator use duration, Charlson Comorbidity Index scores, emergency department visits, and patient age. This research is crucial for estimating potential expenses linked to complications from the procedure, directing the allocation of resources in the future, and acting as an important resource for crafting medical management policies., Competing Interests: Declarations. Ethics approval and consent to participate: Following national regulations and with the approval of the Institutional Review Board (IRB) at Fu Jen Catholic University, the requirement for obtaining informed consent was waived for this study. The waiver was granted based on the assurance that participant information would remain confidential, with data anonymized through unique identifiers (protocol code C108121; approved on March 5, 2020). This waiver was deemed appropriate as the study met specific criteria outlined in Taiwan’s Human Subjects Protection Act and rigorously adhered to all established protocols. Consent for publication: Not applicable. Competing interests: The authors declare no competing interests., (© 2025. The Author(s).)
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- 2025
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120. Epidemiological feature of imported malaria in Taiwan during the 2014-to-2020 period.
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Lin FH, Chou YC, Hsieh CJ, Huang YC, and Yu CP
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- Humans, Taiwan epidemiology, Male, Female, Middle Aged, Adult, Young Adult, Communicable Diseases, Imported epidemiology, Travel, Africa epidemiology, Africa ethnology, Animals, Asia epidemiology, Seasons, Aged, Anopheles parasitology, Malaria epidemiology
- Abstract
Although the World Health Organization (WHO) certified Taiwan as being malaria-free in 1965, there are reports of a few imported cases each year by travelers who visit malaria-endemic areas. This study examined the epidemiology of imported malaria cases in Taiwan from 2014 to 2020, utilizing national surveillance data from the Taiwan Centers for Disease Control. Malaria cases were confirmed through the application of standard laboratory methods. Passenger data came from the Tourism Bureau, Ministry of Transportation and Communication, Taiwan (TBMTC). All data were analyzed using SPSS version 21. The analysis included a dataset comprising 64 cases of imported malaria. Of the total cases, 77.8% were acquired from Africa, and 17.5% from Asia. Plasmodium falciparum was responsible for more than half (57.1%) of the cases, Plasmodium vivax malaria for 25.4% of cases, Plasmodium malariae malaria for 6.3%, Plasmodium ovale malaria for 4.8%, and unspecified pathogen malaria for 6.3% of the cases. Majority of the patients were male (75%) and were predominantly aged 20 to 59 years (70.3%). Most cases of imported malaria occurred during the fall season, and 51.6% of cases occurred in 8 cities during the period of 2014 to 2020. No evidence exists to indicate that indigenous malaria transmission occurs in Taiwan. Anopheles minimus was found in 4 cities (counties), namely Tainan City and Pingtung County in Southern Taiwan; Hualien County and Taitung County in Eastern Taiwan. The findings of this study highlight the necessity for robust surveillance systems, effective vector control measures, and targeted interventions for travelers and immigrants to prevent malaria outbreaks and maintain Taiwan's malaria-free status., Competing Interests: The authors have no funding and conflicts of interest to disclose., (Copyright © 2025 the Author(s). Published by Wolters Kluwer Health, Inc.)
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- 2025
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121. A comprehensive systemic review and meta-analysis of the association between lipid profile and hidradenitis suppurativa.
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Li YH, Chuang SH, Huang YC, and Yang HJ
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- Humans, Lipids blood, Metabolic Syndrome blood, Metabolic Syndrome epidemiology, Metabolic Syndrome diagnosis, Cholesterol blood, Cholesterol, HDL blood, Hidradenitis Suppurativa blood, Triglycerides blood
- Abstract
Background: While several studies have suggested a connection between Metabolic Syndrome (MetS) and Hidradenitis Suppurativa (HS), a definitive analysis confirming the association between lipid abnormalities and HS based on actual lipid values is lacking. Previous research, using odds ratios from ICD codes, indicates links between elevated triglycerides and low high-density lipoprotein levels with HS. However, these findings may not fully represent real-life situations, as no comprehensive analysis using actual lipid measurements has been performed., Objectives: To examine the relationship between lipid profile values-total cholesterol, triglycerides (TG), low-density lipoprotein (LDL), and high-density lipoprotein (HDL)-and HS., Methods: A comprehensive search of PubMed, Cochrane Library, and Embase was conducted to identify studies reporting lipid profiles in HS patients. A meta-analysis using standardized mean differences (SMDs) was performed to assess the association between lipid abnormalities and HS., Results: The meta-analysis found that HS patients had significantly higher TG levels (SMD = 0.28, 95% CI: 0.09-0.47, P = 0.004) and lower HDL levels (95% CI: -0.53 to -0.16, P < 0.001) compared to healthy controls. No significant differences were observed in total cholesterol (SMD = 0.01, 95% CI: -0.19-0.21, P = 0.93) and LDL levels (SMD = 0.04, 95% CI: -0.10-0.17, P = 0.61). These results corroborate earlier studies linking HS with dyslipidemia, particularly hypertriglyceridemia and hypo-HDL cholesterolemia, with the added strength of using actual lipid values., Conclusions: This study confirms the association between hypertriglyceridemia and low HDL cholesterol in HS patients, highlighting the broader systemic association of the condition. Dermatologists should monitor lipid profiles in HS patients to mitigate potential cardiovascular risks through early detection and management., Competing Interests: Declarations. Ethics approval and informed consent: The Institutional Review Board Committee of Changhua Christian Hospital in Taiwan granted ethical approval for the execution of this study (IRB number: 240607). Competing interests: The authors declare no competing interests., (© 2024. The Author(s), under exclusive licence to Springer-Verlag GmbH Germany, part of Springer Nature.)
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- 2025
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122. The role of personality traits and life stress in alcohol use disorder: Insights from NGF gene polymorphisms of Han Chinese population in Taiwan.
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Kuo SC, Lin CL, Yeh YW, Chen CY, Huang YC, Chang TY, Yang YP, Huang JS, Yang BZ, and Huang SY
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- Humans, Female, Male, Taiwan, Adult, Middle Aged, Genetic Predisposition to Disease genetics, East Asian People, Personality genetics, Nerve Growth Factor genetics, Polymorphism, Single Nucleotide genetics, Alcoholism genetics, Alcoholism psychology, Asian People genetics, Stress, Psychological genetics
- Abstract
Objective: Alcohol use disorder (AUD) is a complex neuropsychiatric condition influenced by genetic and environmental factors. Nerve growth factor (NGF) plays a crucial role in neuronal neuroplasticity and chronic alcohol consumption may alter NGF levels in specific brain regions. The study investigates the associations between NGF gene polymorphisms, susceptibility to AUD, and specific stress and personality characteristics., Methods: Our study involved 1133 participants from a homogeneous Han Chinese population, 587 of whom had AUD and 546 were controls. To minimize potential confounding factors, the AUD group was stratified by sex and age at baseline. A total of 414 participants completed the Life Event Questionnaires (LEQ), while 559 participants completed the Tridimensional Personality Questionnaire (TPQ)., Results: The NGF's rs7523654 and rs11102929 loci were significantly associated with AUD, especially in female subgroups. Additional haplotype research confirmed similar findings. AUD patients showed more vital propensities for novelty seeking (NS) and harm avoidance (HA) compared to controls. Additionally, they recorded higher negative LEQ results. Notably, HA and negative LEQ scores among AUD people were significantly affected by the SNP rs11102929 in the NGF gene. The age at which AUD first manifested and NS scores showed a reverse link, suggesting that a higher NS characteristic may predispose people to develop AUD earlier in life., Conclusion: The findings suggest that the NGF gene may influence AUD susceptibility and its links to personality traits and life stress. However, the small sample of women with AUD limits the reliability of these associations, highlighting the need for further study., Competing Interests: Declaration of competing interest The authors declare that this research was conducted without any commercial or financial relationships that could be construed as a potential conflict of interest. All authors have approved the submission of the final version of the manuscript., (Copyright © 2024. Published by Elsevier Inc.)
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- 2025
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123. Fluorinated and methylated ortho -benzodipyrrole-based acceptors suppressing charge recombination and minimizing energy loss in organic photovoltaics.
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Wang YB, Tsai CL, Xue YJ, Jiang BH, Lu HC, Hong JC, Huang YC, Huang KH, Chien SY, Chen CP, and Cheng YJ
- Abstract
The elimination of the A' unit from -type Y6-derivatives has led to the development of a new class of ortho -benzodipyrrole ( o -BDP)-based A-D
N BN D-A-type NFAs. In this work, two new A-DN BN D-A-type NFAs, denoted as CFB and CMB, are designed and synthesized, where electron-withdrawing fluorine atoms and electron-donating methyl groups are substituted on the benzene ring of the o -BDP moiety, respectively. CFB exhibits a blue-shifted absorption spectrum, stronger intermolecular interactions, shorter π-π stacking distances, and more ordered 3D intermolecular packing in the neat and blend films, enabling it to effectively suppress charge recombination in the PM6:CFB device showing a higher PCE of 16.55% with an FF of 77.45%. CMB displays a higher HOMO/LUMO energy level, a smaller optical bandgap, and a less ordered 3D packing, which contributes to its superior ability to suppress energy loss in the PM6:CMB device with a high Voc of 0.90 V and a PCE of 16.46%. To leverage the advantages of CFB and CMB, ternary PM6:Y6-16:CFB and PM6:Y6-16:CMB devices are fabricated. The PM6:Y6-16:CFB device exhibits the highest PCE of 17.83% with an increased Voc of 0.86 V and a Jsc of 27.32 mA cm-2 , while the PM6:Y6-16:CMB device displayed an elevated Voc of 0.87 V and an improved FF of 74.71%, leading to a PCE of 17.44%. The high PCE was achieved using the non-halogenated greener solvent o -xylene, highlighting their potential for facilitating more eco-friendly processing procedures. C-shaped disubstituted o -BDP-based A-D-A type acceptors open up new avenues for tailoring electronic properties and molecular self-assembly, achieving higher OPV performance with enhanced charge recombination suppression and reduced energy loss., Competing Interests: There are no conflicts to declare., (This journal is © The Royal Society of Chemistry.)- Published
- 2025
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124. Neuroprotective potential of isofraxidin: Alleviating parkinsonian symptoms, inflammation and microglial activation.
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Wang TA, Li SY, Fann LY, Li IH, Liu TT, Hung HY, Chang CW, Cheng CC, Huang YC, Yu PY, and Shih JH
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Background: Parkinson's disease (PD) is one of the most common neurodegenerative disorders. Previous research has confirmed that isofraxidin can reduce macrophage expression and inhibit peripheral inflammation. However, its effects on the central nervous system remain underexplored., Objective: This study aims to determine whether isofraxidin offers protective effects against PD., Methods: To assess the effects of isofraxidin, motor performance changes in LPS-induced PD mice were evaluated using rotarod, pole-climbing, and beam-walking tests. Striatal damage was examined through [
18 F]fluorodeoxyglucose ([18 F]FDG) positron emission tomography (PET) imaging, and dopaminergic neurotoxicity was assessed using tyrosine hydroxylase (TH) staining. Microglial accumulation and activation were monitored with Iba-1 staining, while LPS-induced inflammation was examined via TNF-α and IL-1β staining., Results: Isofraxidin pre-treatment significantly improved LPS-induced motor dysfunction, as evidenced by better performance in the rotarod, pole-climbing, and beam-walking tests. [18 F]FDG PET imaging showed that isofraxidin restored glucose uptake in the striatum, countering LPS-induced damage. Furthermore, Iba-1 staining revealed that isofraxidin markedly inhibited LPS-induced microglial activation and accumulation. TNF-α and IL-1β staining indicated a reduction in inflammation with isofraxidin treatment. Additionally, TH staining supported the neuroprotective role of isofraxidin on dopaminergic neurons., Conclusions: Isofraxidin exhibits notable neuroprotective properties by mitigating LPS-induced parkinsonian behaviors, microglial activation, inflammation, and dopaminergic neuron damage. These results highlight isofraxidin's potential as a therapeutic intervention for PD., Competing Interests: The author(s) declared no potential conflicts of interest with respect to the research, authorship, and/or publication of this article., (© The Author(s) 2025.)- Published
- 2025
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125. Interfacial Electronic Charge Trapping and Photonic Carrier Excitation Coupling in Solution-Processed Zinc-Tin Oxide Thin-Film Transistors Applied for Logic Gate Design and Quantized Neural Network.
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Chang PH, Lin WY, Huang YC, Chen YC, Shih LC, and Chen JS
- Abstract
Components needed in Artificial Intelligence with a higher information capacity are critically needed and have garnered significant attention at the forefront of information technology. This study utilizes solution-processed zinc-tin oxide (ZTO) thin-film phototransistors and modulates the values of V
G , which allows for the regulation of electron trapping/detrapping at the ZTO/SiO2 interface. By coupling the excited photonic carrier and electronic trapping, logic gates such as "AND," "OR," "NAND," and "NOR" can be achieved. With the exponential growth in data generation, efficient processing and storage solutions are imperative. However, extensive data transfer between computing units and storage limits the level of artificial neural networks (ANNs). Consequently, quantized neural networks (QNNs) have gained interest for their reduced computational resource requirements and lower consumption. In this context, we introduce an optimized ternary logic circuit based on ZTO devices. By utilizing optical modulation to adjust the turn-on voltage of the single device, we demonstrate the achievement of ternary current states, thereby providing three distinct discrete states. This configuration can be extended to QNN computing, demonstrating multilevel quantized current values for in-memory computation. We achieved a handwriting digit recognition rate of 91.6%, thereby demonstrating reliable QNN hardware performance. This robust QNN performance indicates that the metal oxide phototransistor shows significant potential for future ternary computing systems.- Published
- 2025
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126. Effectiveness of Using a Digital Wearable Plantar Pressure Device to Detect Muscle Fatigue: Within-Subject, Repeated Measures Experimental Design.
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Chen FY, Lin TY, Huang YC, and Widianawati E
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- Humans, Male, Adult, Female, Muscle, Skeletal physiology, Biomechanical Phenomena, Muscle Fatigue physiology, Wearable Electronic Devices, Electromyography instrumentation, Electromyography methods, Pressure, Foot physiology
- Abstract
Background: Muscle fatigue, characterized by reduced force generation during repetitive contractions, impacts older adults doing daily activities and athletes during sports activities. While various sensors detect muscle fatigue via muscle activity, biochemical markers, and kinematic parameters, a real-time wearable solution with high usability remains limited. Plantar pressure monitoring detects muscle fatigue through foot loading changes, seamlessly integrating into footwear to improve the usability and compliance for home-based monitoring., Objective: This study aimed to investigate the effects of muscle fatigue on plantar pressure measurements using a self-developed wearable plantar pressure system., Methods: Twelve healthy participants completed a 5-minute calf muscle fatigue protocol. The plantar pressures and surface electromyography (sEMG) activity of the gastrocnemius muscles were recorded before and after exercise. The plantar pressures at 6 regions and the median frequency (MDF) of sEMG were analyzed to quantify fatigue., Results: The self-developed foot pressure system showed a significant decrease in plantar pressure peak values at the heel of the left (P=.003) and right feet (P=.001) and at the lateral toe of the left (P=.001) and right feet (P=.026). A significant increase was observed at the metatarsal head of both the left foot (P=.001) and the right foot (P=.017). The MDF of sEMG signals significantly decreased in the left (P=.001) and right gastrocnemius (P<.001)., Conclusions: Plantar pressure changes and sEMG signals effectively detect gastrocnemius muscle fatigue using the proposed wearable system, supporting the development of a wearable solution for detecting muscle fatigue suitable for home-use., (© Fu-Yu Chen, Tzu-Yao Lin, Yi-Cheng Huang, Evina Widianawati. Originally published in JMIR Human Factors (https://humanfactors.jmir.org).)
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- 2025
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127. Clinical and patient-reported outcomes between full-endoscopic and conventional parotidectomy: a prospective cohort study.
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Chen S, Huang YC, Su ZK, Yang F, Lubamba GP, Gupta A, Alkebsi K, Zhang ZY, Zhang Z, Xia H, Zhang YQ, Li CJ, Xuan M, Tang XF, and Zhu GQ
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- Humans, Male, Female, Prospective Studies, Middle Aged, Adult, Aged, Postoperative Complications, Treatment Outcome, Parotid Neoplasms surgery, Blood Loss, Surgical, Facial Paralysis surgery, Patient Reported Outcome Measures, Endoscopy methods, Parotid Gland surgery
- Abstract
Objectives: This study investigates the clinical and patient-reported outcomes of full-endoscopic parotidectomy compared to the conventional approach., Methods: Between July 2021 and December 2023, patients who underwent parotidectomy were prospectively enrolled and assigned to either the full-endoscopic parotidectomy group (Group I) or the conventional surgery group (Group II). Clinical outcomes were evaluated, and patient-reported outcomes were assessed using a Visual Analogue Scale and five FACE-Q scales., Results: A total of 293 patients were prospectively included, with 146 in the full-endoscopic group and 147 in the conventional group. Blood loss was significantly lower in the full-endoscopic group (regression coefficient, 0.61; 95% CI, 0.15 to 1.07), as was the rate of immediate facial paralysis (0.76; 95% CI, 0.25 to 1.27). The full-endoscopic group also showed superior outcomes on the Visual Analogue Scale (- 0.14; 95% CI, - 0.23 to - 0.05) and FACE-Q scales for Appearance-Related Psychosocial Distress (1.15; 95% CI, 0.59 to 1.71), Social Function (- 0.37; 95% CI, - 0.59 to - 0.15), Satisfaction with Facial Appearance (- 0.44; 95% CI, - 0.64 to - 0.23), Psychological Function (- 0.39; 95% CI, - 0.58 to - 0.20), and Satisfaction With Outcome (- 0.42; 95% CI, - 0.66 to - 0.19)., Conclusions: Full-endoscopic parotidectomy yields less blood loss and a reduced incidence of immediate facial paralysis compared to conventional surgery. Patients undergoing full-endoscopic parotidectomy reported better aesthetic and psychological outcomes., Clinical Relevance: The combined analysis of clinical and patient-reported outcomes is valuable in guiding surgical planning. These findings provide valuable insights for patients considering full-endoscopic parotidectomy and underscore the importance of addressing functional, aesthetic, and psychological aspects for both patients and healthcare providers., Competing Interests: Declarations. Competing interests: The authors declare no competing interests. Conflict of interest: The authors declare that they have no known competing financial interests or personal relationships that could have appeared to influence the work reported in this paper., (© 2024. The Author(s), under exclusive licence to Springer-Verlag GmbH Germany, part of Springer Nature.)
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- 2025
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128. Recommendation for the use of respiratory syncytial virus vaccines.
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Lee PI, Huang YC, Liu CC, Chen SU, Hsueh PR, Ku SC, Chen PY, Chen CJ, Lin YT, Lu CY, Chiu NC, Chi H, Chen YC, Chang FY, Yen MY, Lu CT, Yang KY, Chiu CH, Hwang KP, Lee WS, Yen TY, Hsu JF, Lin YC, Hu YL, and Lin TY
- Abstract
Respiratory syncytial virus (RSV) is the most common pathogen for young children hospitalized with bronchiolitis and pneumonia. Most infections occur below 1 year of age. RSV is also a significant viral pathogen for adults with respiratory tract infection. Vaccines targeting the pre-fusion protein of RSV, including recombinant and mRNA vaccines, are now available. A committee of experts from related fields was convened by the Taiwan Immunization Vision and Strategy to develop recommendations for RSV vaccination in the elderly and pregnant women. The recommendation is not intended as a sole source of guidance in the prevention of RSV infection in children. The provisions listed in this recommendation are comprehensive suggestions made by experts in Taiwan based on existing medical evidence. This recommendation should be subject to modification in light of additional medical research findings in the future, and these provisions should not be cited as a basis for dispute resolution., (Copyright © 2025. Published by Elsevier B.V.)
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- 2025
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129. Automatic segmentation of white matter lesions on multi-parametric MRI: convolutional neural network versus vision transformer.
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Chen YT, Huang YC, Chen HL, Lo HC, Chen PC, Yu CC, Tu YC, Liu TL, and Lin WC
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Background and Purpose: White matter hyperintensities in brain MRI are key indicators of various neurological conditions, and their accurate segmentation is essential for assessing disease progression. This study aims to evaluate the performance of a 3D convolutional neural network and a 3D Transformer-based model for white matter hyperintensities segmentation, focusing on their efficacy with limited datasets and similar computational resources., Materials and Methods: We implemented a convolution-based model (3D ResNet-50 U-Net with spatial and channel squeeze & excitation) and a Transformer-based model (3D Swin Transformer with a convolutional stem). The models were evaluated on two clinical datasets from Kaohsiung Chang Gung Memorial Hospital and National Center for High-Performance Computing. Four metrics were used for evaluation: Dice similarity coefficient, lesion segmentation, lesion F1-Score, and lesion sensitivity., Results: The Transformer-based model, with appropriate adjustments, outperformed the well-established convolution-based model in foreground Dice similarity coefficient, lesion F1-Score, and sensitivity, demonstrating robust segmentation accuracy. DRLoc enhanced the Transformer's performance, achieving comparable results on internal and benchmark datasets despite limited data availability., Conclusion: With comparable computational overhead, a Transformer-based model can surpass a well-established convolution-based model in white matter hyperintensities segmentation on small datasets by capturing global context effectively, making them suitable for clinical applications where computational resources are constrained., Competing Interests: Declarations. Ethical approval and consent to participate: The studies involving human participants were reviewed and approved by the Institutional Review Board of Chang Gung Memorial Hospital (IRB No. 202002026A3, passed on 19 January 2021; IRB NO. 201900483B0, passed on 10 April 2019). All patient information included in this study was anonymized and deidentified. The requirement to obtain informed consent was waived in accordance with IRB policy. Consent for publication: Not applicable. Competing interests: Authors TYC and LTL are executive directors of the Taiwan AI Labs. Authors HYC and LHC are employed by the Taiwan AI Labs. The remaining authors declare that the research was conducted in the absence of any commercial or financial relationships that could be construed as a potential conflict of interest., (© 2025. The Author(s).)
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- 2025
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130. Tea (Camellia sinensis) Seed Saponins Act as Sebosuppression Agents via the AMPK/mTOR Pathway.
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Li J, Huang YC, Deng JM, Yu M, Zouboulis CC, Wang GL, and Wang J
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- Humans, Lipogenesis drug effects, Sebum metabolism, Sebum drug effects, Cell Line, Linoleic Acid pharmacology, Sterol Regulatory Element Binding Protein 1 metabolism, Sterol Regulatory Element Binding Protein 1 genetics, TOR Serine-Threonine Kinases metabolism, AMP-Activated Protein Kinases metabolism, Saponins pharmacology, Seeds chemistry, Camellia sinensis chemistry, Sebaceous Glands drug effects, Sebaceous Glands metabolism, Sebaceous Glands cytology, Signal Transduction drug effects
- Abstract
Background: Excessive lipogenesis of the skin triggers some dermatological concerns, such as enlarged pores, acne, and blackheads. Although topical drug treatments can offer temporary relief, their prolonged usage may lead to side effects of dryness, irritation, or allergic reactions. Consequently, the development of safer and efficacious ingredients in cosmetics for managing sebum overproduction represents a significant yet challenging endeavor., Aim: Saponins were extracted from tea (Camellia sinensis) seed meal and purified by macroporous resin in order to investigate the impact of tea seed saponins (TSS) on lipid production in human immortalized sebaceous cells. Moreover, we attempted to reveal the underlying mechanism of TSS on the sebosuppression effect in SZ95 sebocytes stimulated by linoleic acid (LA)., Methods: The compositions and chemical structures of TSS were determined using UV-vis absorption spectrum, Fourier transform-infrared (FTIR) spectrum, and ultra-high-performance liquid chromatography-mass spectrometry analysis. An in vitro model of cellular lipid accumulation induced by LA was established. Total lipid synthesis in intracellular SZ95 sebocytes was assessed through Nile Red staining, while triglyceride, cholesterol, and fatty acids were quantified by commercially assay kits. Western blot and quantitative real-time polymerase chain reaction were employed to analyze the protein expression levels involved in the AMP-activated protein kinase (AMPK)/mammalian target of rapamycin (mTOR) pathway as well as the downstream protein and mRNA expressions of sterol regulatory element-binding protein-1 (SREBP-1), peroxisome proliferator-activated receptor γ (PPARγ), and fatty acid synthase (FAS). The localizations of SREBP-1 within the cytoplasm or nucleus were characterized using immunofluorescence staining., Results: Five saponins were identified in the extracted TSS, all of which were oleanic acid-type pentacyclic triterpenes. TSS treatment significantly alleviated LA-induced lipid accumulation in SZ95 sebocytes. In addition, TSS activated the AMPK/mTOR pathway and downregulated the downstream protein and mRNA expression of transcription factors and enzymes, including SREBP-1, PPARγ, and FAS. Moreover, the TSS blocked the nuclear transfer of SREBP-1 from cytoplasm to nucleus., Conclusion: In human sebocytes, TSS exhibited sebosuppressive effect as revealed by the inhibited production of total lipids as well as triglyceride, cholesterol, and fatty acids. Moreover, the anti-lipogenesis mechanism by TSS involved the activation of the AMPK/mTOR pathway and downregulated downstream transcription factors and enzymes of SREBP-1, PPARγ, and FAS. Additionally, TSS blocked the SREBP-1 nuclear translocation. These results may justify the potent of TSS as a new candidate for modulating lipogenesis in human SZ95 sebocytes., (© 2025 The Author(s). Journal of Cosmetic Dermatology published by Wiley Periodicals LLC.)
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- 2025
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131. Evaluation of cortical bone thickness at the nasomaxillary and zygomaticomaxillary buttresses using cone-beam computed tomography imaging for Le Fort I osteotomy.
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Chuang CW, Huang YC, Chen I, Chen MYC, and Hsu JT
- Abstract
Background/purpose: Studies have indicated that 50%-55% of the population have malocclusion, and approximately 5%-10% require orthognathic surgery to correct this condition. Optimal placement of plates and screws significantly affects the success rate of the surgery and postoperative stability. This study evaluates the cortical thickness of the maxillary bone in the nasomaxillary and zygomaticomaxillary buttress regions in Taiwanese patients based on cone-beam computed tomography (CBCT) images., Materials and Methods: 128 Patients undergoing Le Fort I osteotomy were selected for this study. Their CBCT images were input into medical imaging software to simulate the placement of titanium screws and plates. The cortical bone thickness at these positions was measured to assess the thickness in the nasomaxillary buttress (surrounding the nasal opening) and the zygomaticomaxillary buttress (surrounding the maxillary zygomatic process). Associations of these thicknesses with gender, age, and screw position were analyzed., Results: In the nasomaxillary region, cortical bone was thicker on the upper and lower vertical regions, with men generally having thicker bone. The zygomaticomaxillary region had increased thickness near the zygomatic end and distal region. Younger adults had significantly greater bone thickness in certain areas than those over 30 years., Conclusion: The nasomaxillary region's upper and lower vertical regions and the zygomatic end in the zygomaticomaxillary region provide optimal screw placement sites. Bone thickness differences by gender and age suggest occlusal force and age-related bone resorption as influencing factors., Competing Interests: The authors have no conflicts of interest relevant to this article., (© 2024 Association for Dental Sciences of the Republic of China. Publishing services by Elsevier B.Vé.)
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- 2025
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132. Chemical modulation of Akt signaling enhances spinal cord regeneration in zebrafish.
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Shen YJ, Chen HY, Chang CW, Huang YC, and Cheng YC
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- Animals, Spinal Cord drug effects, Spinal Cord metabolism, Recovery of Function drug effects, Recovery of Function physiology, Neuroglia drug effects, Neuroglia metabolism, Disease Models, Animal, Neurons drug effects, Neurons metabolism, Nerve Regeneration drug effects, Nerve Regeneration physiology, Zebrafish, Proto-Oncogene Proteins c-akt metabolism, Spinal Cord Injuries drug therapy, Spinal Cord Injuries metabolism, Spinal Cord Injuries physiopathology, Signal Transduction drug effects, Signal Transduction physiology, Spinal Cord Regeneration drug effects, Spinal Cord Regeneration physiology
- Abstract
Central nervous system lesions often cause permanent motility defects in mammals since the injured neurons cannot regenerate. In contrast, lower vertebrates like zebrafish can regenerate lost neurons and restore motor function. This study investigates the efficacy of SC79, a pan-Akt activator, and A674563, a selective Akt1 inhibitor, as potential therapeutic agents for promoting spinal cord recovery post-injury. Spinal cord injury was induced in zebrafish larvae, and the effects of SC79 and A674563 on neuronal and glial regeneration were examined. SC79 promoted neuronal regeneration without affecting glial bridging, while A674563 induced glial bridging but reduced neuronal regeneration. The combination of SC79 and A674563 induced both glial bridging and neuronal regeneration. Optomotor response tests revealed improved motor function recovery with the combined treatment compared to individual treatments. Additionally, these chemical treatments altered the expression of 12 Akt downstream transcriptional target genes, affirming that the combination treatment preferentially regulates spinal cord regeneration through its action on Akt signaling. These findings highlight the complex interplay of Akt signaling pathways in spinal cord regeneration and suggest potential therapeutic strategies for enhancing functional recovery in spinal cord injury patients., Competing Interests: Declaration of competing interest The authors declare that they have no known competing financial interests or personal relationships that could have appeared to influence the work reported in this paper., (Copyright © 2024 Elsevier B.V. All rights reserved.)
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- 2025
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133. The role of cricoarytenoid joint ankylosis in bilateral vocal cord immobility.
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Su WF, Chen CJ, Huang YC, Hsu YC, Ko PY, and Liu SC
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- Humans, Adult, Middle Aged, Male, Female, Retrospective Studies, Aged, Adolescent, Young Adult, Diagnosis, Differential, Vocal Cord Paralysis physiopathology, Vocal Cord Paralysis surgery, Vocal Cord Paralysis diagnosis, Vocal Cord Paralysis etiology, Arytenoid Cartilage surgery, Laryngoscopy methods, Cricoid Cartilage surgery, Ankylosis surgery, Ankylosis complications, Ankylosis diagnosis
- Abstract
Objectives: To stratify the severity of cricoarytenoid joint fixation (CAJF) by surgery and understand the role of it played in the bilateral vocal fold immobility (BVFI). The second objective emphasizes on the significance of the preoperative differential diagnosis from neurogenic immobility with medical history and endoscopic findings., Methods: A retrospective review was conducted of 74 patients between 2005 and 2022. Careful medical history inquiry, and videolaryngoscopy are conducted to recruit the appropriate surgical candidates. All patients underwent arytenoid remobilization (AR) followed by vocal fold medialization with arytenoid adduction (AA) or lateralization with suture lateralization (SL). The severity of CAJF is graded during the operation or inferred based on the period from operation to recurrence., Result: A total of 18 patients, aged between 18 and 76 years, were analyzed. Among them, 14 cases were classified as the adducted type with ventilation problems, with three presenting with dyspnea, and 11 requiring artificial airways. Additionally, four patients presented with the abducted type, characterized by aphonia. Meanwhile, two additional cases were considered for comparison but were not included in this cohort of 18 subjects due to incorrect diagnosis and inappropriate management. Using AR procedure, the AA procedure offered three aphonia subjects a voiced sound without airway impairment and the SL procedure decannulated 100% (11/11) of the artificial airways and improved the airway patency in 100% (3/3) of the non-tracheostomized subjects despite the severity of CAJF. The severity of joint ankylosis was distributed as follows: In the aphonia group, there were three subjects with grade I, one subject with grade II, and 0 subjects with grade III. In the ventilation group, there was one subject with grade I, seven subjects with grade II, and six subjects with grade III. In contrast, the two cases used for comparison experienced recurrent dyspnea and failed decannulation because the AR procedure was not performed. The follow-up period was averaged in 58 and 14 months at least., Conclusion: From this experience, it is the accurate preoperative diagnosis instead of the severity of CAJF that determines the successful rate in airway patency and voiced phonation if the AR procedure is utilized. Careful medical history inquiry and videolaryngoscopic examination can correctly differentiate the mechanical from neurogenic origin without the help of EMG. Evidence of level: 4., Competing Interests: Declarations. Conflict of interest: No authors have potential conflicts of interest. Financial disclosure: No financial disclosures. Research involving human participants and/or animals: This research involved specimens from human participants. The research protocol has been reviewed and approved by the Institutional Review Board of Tri-Service General Hospital. Informed consent: Informed consent was obtained from all individual participants included in the study., (© 2024. The Author(s), under exclusive licence to Springer-Verlag GmbH Germany, part of Springer Nature.)
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- 2025
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134. Incorporation of ascorbic acid-2-glucoside into ulvan microneedles to enhance its permeation for anti-aging and whitening treatment.
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Don TM, Chen M, and Huang YC
- Abstract
For anti-aging and whitening treatment, ascorbic acid-2-glucoside (AA2G) was incorporated into a 4.0 % (w/v) ulvan solution at three concentrations (0.5 %, 1.0 %, and 2.0 % w/v) to fabricate ulvan-based microneedles (UMNs) using a spin-casting method, aimed at enhancing AA2G permeation. The in vitro skin insertion study demonstrated that incorporating AA2G into the UMNs improved their insertion capability, increasing from 86.3 % for neat UMNs to 98 % for AA2G-UMNs. Furthermore, in vitro drug permeation profiles revealed that dissolving UMNs significantly enhanced the cumulative permeation of AA2G, achieving 80-90 % within 3 h. In addition to showing good biocompatibility with HaCaT and NIH3T3 cells, AA2G-UMNs exhibited antioxidant activity and protected HaCaT cells from H
2 O2 -induced oxidative stress. Their anti-aging activity was demonstrated by their ability to inhibit elastase and collagenase activity. Moreover, whitening efficacy was confirmed through the inhibition of melanin production and tyrosinase activity. Among the formulations, 2.0 % AA2G-UMNs achieved the greatest reduction in melanin content in B16F10 cells, with a 54.2 % reduction intracellularly and a 61.3 % reduction extracellularly. Tyrosinase activity inhibition by AA2G-UMNs ranged from 42.6 % to 53.4 %. These results suggest that AA2G-UMNs hold significant promise for applications in the pharmaceutical and cosmeceutical industries., Competing Interests: Declaration of competing interest The authors declare no conflict of interest. The funding sponsor has no role in the design of the study; in the collection, analyses, or interpretation of data; in the writing of the manuscript, and in the decision to publish the results., (Copyright © 2024 Elsevier B.V. All rights reserved.)- Published
- 2024
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135. Advancements in Antioxidant-Based Therapeutics for Spinal Cord Injury: A Critical Review of Strategies and Combination Approaches.
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Shen YJ, Huang YC, and Cheng YC
- Abstract
Spinal cord injury (SCI) initiates a cascade of secondary damage driven by oxidative stress, characterized by the excessive production of reactive oxygen species and other reactive molecules, which exacerbate cellular and tissue damage through the activation of deleterious signaling pathways. This review provides a comprehensive and critical evaluation of recent advancements in antioxidant-based therapeutic strategies for SCI, including natural compounds, RNA-based therapies, stem cell interventions, and biomaterial applications. It emphasizes the limitations of single-regimen approaches, particularly their limited efficacy and suboptimal delivery to injured spinal cord tissue, while highlighting the synergistic potential of combination therapies that integrate multiple modalities to address the multifaceted pathophysiology of SCI. By analyzing emerging trends and current limitations, this review identifies key challenges and proposes future directions, including the refinement of antioxidant delivery systems, the development of multi-targeted approaches, and strategies to overcome the structural complexities of the spinal cord. This work underscores the pressing need for innovative and integrative therapeutic approaches to advance the clinical translation of antioxidant-based interventions and improve outcomes for SCI patients., Competing Interests: The authors declare no conflicts of interest. The funders had no role in the design of the study; in the collection, analyses, or interpretation of data; in the writing of the manuscript; or in the decision to publish the results.
- Published
- 2024
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136. The complete chloroplast genome of Hemisteptia lyrata (Bunge) Fisch. & C. A. Mey. 1836 (Asteraceae) and its phylogenetic analysis.
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Li CY, Xing YP, Hou WJ, Huang YT, Huang YC, Song YY, Kang TG, Yang YY, and Xu L
- Abstract
Hemisteptia lyrata , widely distributed in China, has a 152,467 bp chloroplast genome with a large single-copy (LSC) region of 83,473 bp, a small single-copy (SSC) region of 18,594 bp, a pair of inverted repeat regions (IRs) of 25,194 bp in length. The GC content is 36.46%. A total of 133 genes were encoded, of which 88 were protein coding genes, eight were rRNA genes, and 37 were tRNAs. The phylogenetic analysis using the maximum-likelihood method showed that H. lyrata is closely related to the species in Saussurea. This study provides genomic resources for phylogenetic genetics and resource exploitation of H. lyrata ., Competing Interests: No potential conflict of interest was reported by the author(s)., (© 2024 The Author(s). Published by Informa UK Limited, trading as Taylor & Francis Group.)
- Published
- 2024
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137. Impact of glycemic treatment and blood glucose monitoring on outcomes in patients with acute ischemic stroke without prior diabetes: a longitudinal cohort study.
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Yang HH, Chien WC, Liaw JJ, Yang CC, Chung CH, Huang SH, Huang YC, Wang BL, Chung RJ, Chen PC, Lin TT, Yu PC, and Chen YJ
- Abstract
Objectives: To explore the short- and long-term effects of glycemic management-through glycemic treatment and blood glucose monitoring (BGM)-on stroke recurrence and mortality specifically in patients experiencing a first-ever ischemic stroke (FIS) with hyperglycemia (FISHG) who have not previously been diagnosed with diabetes mellitus (DM)., Methods: We gathered data on patients who were registered on Taiwan's National Health Insurance Research Database from 2000 to 2015. We one-fold propensity-score-matched (by sex, age, and index date) 207,054 patients into 3 cohorts: those with FIS (1) without hyperglycemia, (2) hyperglycemia without glycemic treatment, and (3) hyperglycemia with glycemic treatment. We used Cox proportional hazard regression to evaluate the short- (within 1 year after FIS) and long-term (9.3 ± 8.6 years after FIS) prognostic effects of glycemic management on stroke recurrence and mortality of FISHG., Results: Stroke recurrence and mortality were significantly more likely in the patients with FISHG than their counterparts without hyperglycemia (p < 0.05). Under glycemic treatment, patients with FISHG demonstrated lower risk of mortality at every follow-up than those without (p < 0.001) but were not less likely to have stroke recurrence (p > 0.05). Integrating BGM with glycemic treatment in the FISHG cohort significantly reduced the risk of stroke recurrence compared to patients receiving only glycemic treatment at 1-month, 3-month, 6-month, and 1-year post-stroke follow-ups (adjusted hazard ratios = 0.84, 0.90, 0.88, and 0.92, respectively); additionally, this approach significantly decreased mortality risk at each post-stroke follow-up period (p < 0.05)., Conclusions: BGM combined with glycemic treatment significantly improves prognosis in patients with FISHG who have not been previously diagnosed with DM, reducing the risks of stroke recurrence and mortality., Competing Interests: Declarations. Ethics approval and consent to participate: This study was conducted according to the Code of Ethics of the World Medical Association (Declaration of Helsinki). All methods were carried out in accordance with relevant guidelines and regulations. Informed consent was obtained from all subjects and/or their legal guardian(s).The Research Ethics Committee of the Tri-Service General Hospital of the National Defense Medical Center (TSGHIRB No. B-110-32) approved this study. Competing interests: The authors declare no competing interests., (© 2024. The Author(s).)
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- 2024
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138. Metabolic shifts in lipid utilization and reciprocal interactions within the lung metastatic niche of triple-negative breast cancer revealed by spatial multi-omics.
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Kan JY, Lee HC, Hou MF, Tsai HP, Jian SF, Chang CY, Tsai PH, Lin YS, Tsai YM, Wu KL, Huang YC, and Hsu YL
- Subjects
- Animals, Humans, Female, Mice, Lipid Metabolism, Tumor Microenvironment, Cell Line, Tumor, Gene Expression Regulation, Neoplastic, Multiomics, Triple Negative Breast Neoplasms pathology, Triple Negative Breast Neoplasms metabolism, Triple Negative Breast Neoplasms genetics, Lung Neoplasms secondary, Lung Neoplasms metabolism, Lung Neoplasms pathology, Lung Neoplasms genetics
- Abstract
The Triple-Negative Breast Cancer (TNBC) subtype constitutes 15-20% of breast cancer cases and is associated with the poorest clinical outcomes. Distant metastasis, particularly to the lungs, is a major contributor to the high mortality rates in breast cancer patients. Despite this, there has been a lack of comprehensive insights into the heterogeneity of metastatic tumors and their surrounding ecosystem in the lungs. In this study, we utilized spatial RNA sequencing technology to investigate the heterogeneity of lung metastatic tumors and their microenvironment in two spontaneous lung metastatic mouse models. Our findings revealed an increase in metabolic-related genes within the cancer cells, with the hub gene Dlat (Dihydrolipoamide S-Acetyltransferase) showing a significant association with the development of lung metastatic tumors. Upregulation of Dlat led to the reprogramming of fatty acid utilization, markedly enhancing the bioenergetic capacity of cancer cells. This finding was corroborated by the increased dependence on fatty acid utilization in lung metastatic cancer cells, and inhibition of Dlat in breast cancer cells exhibited a reduced oxygen consumption rate. Consequently, inhibition of Dlat resulted in decreased survival capacity of breast cancer by reducing cancer stem cell properties and cell adhesion in the lung in vivo. The three cell components within the lung metastatic niche, including CD163
+ macrophages, neutrophils, and endothelial cells, expressed elevated levels of ApoE, leading to the secretion of various protumorigenic molecules that promote cancer cell growth in the lung. These molecules include galectin-1, S100A10, S100A4, and S100A6. Collectively, our findings highlight the lipid metabolism reprogramming of cancer and components of the tumor microenvironment that support lung metastasis of TNBC breast cancer., Competing Interests: Competing interests: The authors declare no competing interests. Ethics approval and consent to participate: Ethics approval for animals in this project was obtained from the Institutional Animal. Care and Use Committee (IACUC) in Kaohsiung Medical University (approval no. 112118). All methods were performed in accordance with relevant guidelines and regulations., (© 2024. The Author(s).)- Published
- 2024
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139. Enhanced Performance of Sn-Based Perovskite Photodetectors Through Double-Sided Passivation for Near-Infrared Applications.
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Lai YH, Li CC, Huang YC, Yu Huang T, Gao XK, Yang CC, and Shan Tan C
- Abstract
The development of high-performance Sn-based perovskite photodetectors is presented with double-sided passivation using large alkylammonium interlayers of PEAI and BDAI₂. This dual passivation strategy, applied to the top and bottom of FASnI₃ films, effectively improves film quality by reducing defect density, enhancing carrier mobility, and minimizing non-radiative energy losses at the interfaces. At 720 nm, the photodetectors demonstrate a responsivity of 0.37 A W
-1 , a detectivity of 6.12 × 10¹3 Jones, and an external quantum efficiency (EQE) of 65.60%, with a rapid response time of 9 µs. Additionally, at 850 nm, the detectivity reaches as high as 3.27 × 10¹3 Jones. Furthermore, the device demonstrated a low 1/f noise of 1.21 × 10⁻¹⁵ AHz⁻⁰.⁵ at 10 Hz. Transient photocurrent (TPC) and transient photovoltage (TPV) measurements revealed a significant increase in charge recombination lifetime (τe ) and improved charge transfer efficiency. These results showcase the potential of Sn perovskite photodetectors for near-infrared applications, including autonomous vehicles, biometric recognition, and biomedical treatments., (© 2024 The Author(s). Small published by Wiley‐VCH GmbH.)- Published
- 2024
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140. Magnetic properties of quenched binary and ternary quasicrystal approximants.
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Denoel F, Shiino T, Huang YC, Gebresenbut GH, Pay Gómez C, and Mathieu R
- Abstract
The magnetic properties of binary Gd-Cd and ternary Gd-Au-Ge crystals obtained from the newly introduced low-melt peritectic formation (LMPF) synthesis method were investigated. This method consists of a rapid quenching of the metallic melt followed by an annealing treatment at a relevant temperature. In the first system, both quasicrystal (QC) and approximant crystal (AC) phases can be stabilized, whereas only the AC phase is obtainable in the pseudo-binary Gd-(Au-Ge) system. The magnetic properties of the crystals obtained from LMPF for each system are compared to those of larger single grain crystals obtained from the self-flux (SF) method., Competing Interests: Declarations. Competing interests: The authors declare no competing interests., (© 2024. The Author(s).)
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- 2024
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141. Theoretical Exploration of the Synergistic Effect of Phosphate and Amidoxime for Uranium Recovery from Seawater.
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Huang YC, Wang CZ, Wu QY, Lan JH, Nie CM, Chen SS, Song Y, Li H, and Shi WQ
- Abstract
Highly selective extraction of uranium from seawater is currently extremely challenging. Although the amidoxime group (HAO) is the commonly used ligand in seawater uranium extraction, it also has strong binding capacity for vanadium ion. It has been shown that the introduction of phosphate groups into amidoxime-based adsorbents can improve the adsorption performance of materials through a synergistic effect between functional groups. In this work, we have systematically investigated the selective extraction behavior of the phosphate ligand (methylphosphonic acid, HL
1 ) for uranyl cation and the synergistic effect with amidoxime using density functional theory (DFT). The electron-donor-substituted derivatives of HL1 (aminomethylphosphonic acid (HL2 ) and methyl phosphate (HL3 )) were also considered. Not unexpectedly, the results show that introduction of HL1 into the amidoxime-based adsorbents improves the extraction and selectivity for uranyl cations. This is mainly due to the fact that HL1 is more likely to deprotonate, which promotes the dissociation of [UO2 (CO3 )3 ]4- , and the presence of the phosphate groups in the synergistic complexes alters the optimal coordination mode of VO2 + . In addition, the HL2 and HL3 ligands further improve the uranium extraction performance, especially for HL3 . This work provides guidelines for the rational design of sequestering ligands for uranium extraction from seawater.- Published
- 2024
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142. Response to the comment on "Diagnostic performance of computed tomography-digital subtraction angiography and conventional magnetic resonance imaging for evaluating the vascularity of osseous spinal tumors".
- Author
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Liao TW, Lin YH, and Huang YC
- Abstract
Competing Interests: Declaration of competing interest The authors have no conflicts of interest relevant to this article.
- Published
- 2024
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143. Development of Potent and Selective Inhibitors of Methylenetetrahydrofolate Dehydrogenase 2 for Targeting Acute Myeloid Leukemia: SAR, Structural Insights, and Biological Characterization.
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Chang HH, Lee LC, Hsu T, Peng YH, Huang CH, Yeh TK, Lu CT, Huang ZT, Hsueh CC, Kung FC, Lin LM, Huang YC, Wang YH, Li LH, Tang YC, Chang L, Hsieh CC, Jiaang WT, Kuo CC, and Wu SY
- Subjects
- Humans, Structure-Activity Relationship, Animals, Mice, Aminohydrolases antagonists & inhibitors, Aminohydrolases metabolism, Cell Line, Tumor, Xenograft Model Antitumor Assays, Cell Proliferation drug effects, Molecular Structure, Female, Multifunctional Enzymes, Methylenetetrahydrofolate Dehydrogenase (NADP) antagonists & inhibitors, Methylenetetrahydrofolate Dehydrogenase (NADP) metabolism, Leukemia, Myeloid, Acute drug therapy, Leukemia, Myeloid, Acute pathology, Enzyme Inhibitors pharmacology, Enzyme Inhibitors chemistry, Enzyme Inhibitors chemical synthesis, Antineoplastic Agents pharmacology, Antineoplastic Agents chemistry, Antineoplastic Agents chemical synthesis
- Abstract
Methylenetetrahydrofolate dehydrogenase/cyclohydrolase 2 (MTHFD2), a pivotal mitochondrial enzyme in one-carbon metabolism, is significantly upregulated in various cancers but minimally expressed in normal proliferating cells. In contrast, MTHFD1, which performs similar functions, is predominantly expressed in normal cells. Therefore, targeting MTHFD2 with selective inhibitors holds promise for a broader therapeutic window with reduced toxicity and fewer side effects. This study identified selective 2,4-diamino-6-oxo-1,6-dihydropyrimidin-5-yl ureido-based derivatives through systematic chemical modifications and SAR studies. Structural biology investigations revealed substitutions in the phenyl ring and tail region modulate potency and selectivity toward MTHFD2. Additionally, a comprehensive cell screening platform revealed acute myeloid leukemia cells with FLT3 internal tandem duplication mutations are particularly sensitive to these inhibitors. Furthermore, synergistic effects were observed when combining potential compounds with Alimta. Compound 16e emerged as a leading candidate, demonstrating superior inhibition and selectivity for MTHFD2, favorable pharmacokinetics, and potent antitumor efficacy in MOLM-14 xenograft models.
- Published
- 2024
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144. Effect of cold-water immersion treatment on recovery from exercise-induced muscle damage in the hamstring.
- Author
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Huang YC, Chou TY, Chen TC, and Chen HT
- Abstract
This study investigated the effect of five consecutive days of cold-water immersion (CWI) on recovery from exercise-induced muscle damage (EIMD) in the hamstrings following maximal eccentric contraction (EC) exercise. Eighteen healthy adult women were randomly assigned to a CWI group and a control group (CG) (n = 9/group). Participants performed 10 sets of 10 repetitions of isokinetic EC at 30°/second and underwent maximum voluntary isometric contraction (MVC), delayed onset muscle soreness (DOMS) assessment, straight leg raise (SLR) test, and plasma myoglobin (Mb) measurement. The CWI group received one 14-min session of CWI treatment (14°C) at 1, 25, 49, 73, and 97 h after the EC test, whereas the CG rested in a seated position at the same five time points without receiving treatment. (1) All the dependent variables in the CWI group and CG exhibited significant changes after the EC test (p < 0.05). (2) The recovery effect in the CWI group was significantly greater than in the CG in terms of the MVC, DOMS, SLR, and plasma Mb concentration results. MVC increased by 89.3 ± 2.0% on the fourth day (p < 0.013), DOMS decreased by 15.4 ± 1.5 mm on the second day (p < 0.000), SLR increased by 86.3 ± 1.1% on the second day (p < 0.014), and plasma Mb decreased by 436.3 ± 60.8% on the third day (p < 0.014). The study indicates that five consecutive days of CWI at 14°C significantly enhance recovery from exercise-induced muscle damage in the hamstrings., (© 2024 The Author(s). European Journal of Sport Science published by Wiley‐VCH GmbH on behalf of European College of Sport Science.)
- Published
- 2024
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145. H3.1K27M-induced misregulation of the TSK/TONSL-H3.1 pathway causes genomic instability.
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Yuan W, Huang YC, LeBlanc C, Poulet A, Valsakumar D, van Wolfswinkel JC, Voigt P, and Jacob Y
- Abstract
The oncomutation lysine 27-to-methionine in histone H3 (H3K27M) is frequently identified in tumors of patients with diffuse midline glioma-H3K27 altered (DMG-H3K27a). H3K27M inhibits the deposition of the histone mark H3K27me3, which affects the maintenance of transcriptional programs and cell identity. Cells expressing H3K27M are also characterized by defects in genome integrity, but the mechanisms linking expression of the oncohistone to DNA damage remain mostly unknown. In this study, we demonstrate that expression of H3.1K27M in the model plant Arabidopsis thaliana interferes with post-replicative chromatin maturation mediated by the H3.1K27 methyltransferases ATXR5 and ATXR6. As a result, H3.1 variants on nascent chromatin remain unmethylated at K27 (H3.1K27me0), leading to ectopic activity of TONSOKU (TSK), which induces DNA damage and genomic alterations. Elimination of TSK activity suppresses the genome stability defects associated with H3.1K27M expression, while inactivation of specific DNA repair pathways prevents survival of H3.1K27M-expressing plants. Overall, our results suggest that H3.1K27M disrupts the chromatin-based mechanisms regulating TSK/TONSL activity, which causes genomic instability and may contribute to the etiology of DMG-H3K27a., Competing Interests: Ethics declarations The authors declare no competing interests.
- Published
- 2024
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146. Oxygen Vacancy Boosts Nitrogen-Centered Radical Coupling Initiated by Primary Amine Electrooxidation.
- Author
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Han M, Luo Y, Xu L, Chen W, Li C, Huang YC, Wu Y, Jiang Y, Wu W, Wang R, Lu YR, Zou Y, and Wang S
- Abstract
Synthesis of nitrogen-centered radicals (NCRs) for radical coupling reactions is a powerful and versatile tool in the arsenal of organic synthetic chemistry. However, there are few reports on the direct synthesis of NCRs based on aqueous electrocatalysis. Herein, we present a new electrochemical primary amine oxidation reaction (ePAOR) system with R
1 R2 -CH-NH2 as the substrate for synthesizing NCRs and N-N coupling products. However, ePAOR on the model catalyst (NiO) suffers from low N-N coupling selectivity due to the weak adsorption energy of imine (R1 R2 -C═NH) intermediates. Guided by theoretical calculations, the oxygen vacancy gives NiO a strong adsorption capacity of R1 R2 -C═NH so that it boosts nitrogen-centered radical coupling initiated by the ePAOR on oxygen vacancy-rich NiO (VO -NiO), and the effective utilization rate of NCRs was increased from 36 to 75%. This approach is compatible with a wide range of primary amines and can be applied to N-N cross-coupling systems as well.- Published
- 2024
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147. Light-to-Spike Encoding Using Indium-Gallium-Zinc Oxide Phototransistor for all-Color Image Recognition with Dynamic Range and Precision Tunability.
- Author
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Huang YC, Chen YC, Chen KT, Chen CT, Shih LC, and Chen JS
- Abstract
To enhance the efficiency of machine vision system, physical hardware capable of sensing and encoding is essential. However, sensing and encoding color information has been overlooked. Therefore, this work utilizes an indium-gallium-zinc oxide (IGZO) phototransistor to detect varying densities of red, green, and blue (RGB) light, converting them into corresponding drain current (I
D ) states. By applying stochastic gate voltage (VG ) pulses to the IGZO phototransistor, the fluctuations are generated in these ID states. When the ID exceeds the threshold current (ITC ), a spike signal is generated. This approach enables the conversion of light densities into spike signals, achieving spike-rate encoding. Moreover, adjusting the standard deviation (σ) of the VG pulses controls the range of light densities converted into spike rates, while altering the mean (μ) of the VG pulses changes the baseline level of spike rates. Remarkably, separate RGB channels offer a tunable encoding process, which can emphasize individual colors and correct color bias. The encoded spike rates are also fed into a spiking neural network (SNN) for CIFAR-10 pattern recognition, achieving an accuracy of 86%. The method allows the operation of SNN and shows the tunability in the process of light-to-spike encoding, opening possibilities for color image processing., (© 2024 Wiley‐VCH GmbH.)- Published
- 2024
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148. Sex-dimorphic tumor growth is regulated by tumor microenvironmental and systemic signals.
- Author
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Wang X, Bao H, Huang YC, Barua A, Lai CM, Sun J, Zhou Y, Cong F, Gong S, Chang CH, and Deng WM
- Subjects
- Animals, Female, Male, Neoplasms pathology, Neoplasms metabolism, Drosophila melanogaster, Hemocytes metabolism, Insulin metabolism, Tumor Microenvironment, Sex Characteristics, Drosophila Proteins metabolism, Drosophila Proteins genetics, Signal Transduction
- Abstract
Tumor growth and progression involve coordinated regulation by internal, microenvironmental, and systemic signals and often display conspicuous sexual dimorphism. The mechanisms governing the integration and coordination of these signals, along with their sex-based differences, remain largely unknown. Using a Drosophila tumor model originating from nonreproductive tissue, we show that female-biased tumor growth involves multifaceted communications among tumor cells, hemocytes, and neuroendocrine insulin-producing cells (IPCs). Notch-active tumor cells recruit hemocytes carrying the tumor necrosis factor-α (TNF-α) homolog Eiger to the tumor microenvironment (TME), activating the c-Jun N-terminal kinase (JNK) pathway in tumor cells, instigating the sexually dimorphic up-regulation of cytokine Unpaired 2 (Upd2). Upd2, in turn, exerts a distal influence by modulating the release of a Drosophila insulin-like peptide (Dilp2) from IPCs. Dilp2 then activates the insulin signaling in the tumor, thereby fostering sexual-dimorphic tumor growth. Together, these findings reveal a relay mechanism involving the TME and systemic signals that collectively control the sexual dimorphism of tumor growth.
- Published
- 2024
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149. Synthesis of alkynyl cyclopropa[ c ]coumarins via propargyl sulfonium salts as C1 synthons.
- Author
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Chandrasekar L, Hsieh YZ, Chou HT, Huang YC, and Liu WM
- Abstract
Herein, we present a novel approach for the preparation of alkynyl cyclopropa[ c ]coumarin derivatives with medium to good yields utilizing propargyl sulfonium salts as C1 synthons. Compared with Br
- , using ClO4 - as the counter anion significantly enhances the yield due to its lesser nucleophilic ability. This method features mild reaction conditions and a broad substrate scope with good diastereoselectivity when the substituted R1 group is at the 5-position of the coumarin scaffold.- Published
- 2024
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150. Docosahexaenoic Acid-Infused Core-Shell Fibrous Membranes for Prevention of Epidural Adhesions.
- Author
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Liu ZH, Huang YC, Kuo CY, Govindaraju DT, Chen NY, Yip PK, and Chen JP
- Subjects
- Animals, Rats, Tissue Adhesions prevention & control, Tissue Adhesions pathology, Epidural Space pathology, Rats, Sprague-Dawley, Male, Fibroblasts metabolism, Fibroblasts drug effects, Laminectomy adverse effects, Laminectomy methods, Docosahexaenoic Acids pharmacology, Docosahexaenoic Acids chemistry, Polyesters chemistry
- Abstract
Avoiding epidural adhesion following spinal surgery can reduce clinical discomfort and complications. As the severity of epidural adhesion is positively correlated with the inflammatory response, implanting a fibrous membrane after spinal surgery, which can act as a physical barrier to prevent adhesion formation while simultaneously modulates postoperative inflammation, is a promising approach to meet clinical needs. Toward this end, we fabricated an electrospun core-shell fibrous membrane (CSFM) based on polylactic acid (PLA) and infused the fiber core region with the potent natural anti-inflammatory compound docosahexaenoic acid (DHA). The PLA/DHA CSFM can continuously deliver DHA for up to 36 days in vitro and reduce the penetration and attachment of fibroblasts. The released DHA can downregulate the gene expression of inflammatory markers (IL-6, IL-1β, and TNF-α) in fibroblasts. Following an in vivo study that implanted a CSFM in rats subjected to lumbar laminectomy, the von Frey withdrawal test indicates the PLA/DHA CSFM treatment can successfully alleviate neuropathic pain-like behaviors in the treated rats, showing 3.60 ± 0.49 g threshold weight in comparison with 1.80 ± 0.75 g for the PLA CSFM treatment and 0.57 ± 0.37 g for the untreated control on day 21 post-implantation. The histological analysis also indicates that the PLA/DHA CSFM can significantly reduce proinflammatory cytokine (TNF-α and IL-1β) protein expression at the lesion and provide anti-adhesion effects, indicating its vital role in preventing epidural fibrosis by mitigating the inflammatory response.
- Published
- 2024
- Full Text
- View/download PDF
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