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101. Supplementary Fig. 4 from MEK Inhibition Sensitizes Pancreatic Cancer to STING Agonism by Tumor Cell–intrinsic Amplification of Type I IFN Signaling

102. Supplementary Fig. 3 from MEK Inhibition Sensitizes Pancreatic Cancer to STING Agonism by Tumor Cell–intrinsic Amplification of Type I IFN Signaling

103. Supplementary Data 1 from MEK Inhibition Sensitizes Pancreatic Cancer to STING Agonism by Tumor Cell–intrinsic Amplification of Type I IFN Signaling

104. Comparative Analysis of Molecular Pathogenic Mechanisms and Antiviral Development Targeting Old and New World Hantaviruses

109. Supplementary Figures 1 and 2 from Quantitative In Vivo Imaging of the Androgen Receptor Axis Reveals Degree of Prostate Cancer Radiotherapy Response

110. Data from Quantitative In Vivo Imaging of the Androgen Receptor Axis Reveals Degree of Prostate Cancer Radiotherapy Response

113. Data from A Molecular Screening Approach to Identify and Characterize Inhibitors of Glioblastoma Stem Cells

117. Supplementary Figure 2 from A Molecular Screening Approach to Identify and Characterize Inhibitors of Glioblastoma Stem Cells

118. Data from Recurrent Tumor Cell–Intrinsic and –Extrinsic Alterations during MAPKi-Induced Melanoma Regression and Early Adaptation

119. Supplementary Figure 3 from A Molecular Screening Approach to Identify and Characterize Inhibitors of Glioblastoma Stem Cells

120. Supplementary Table 3 from A Molecular Screening Approach to Identify and Characterize Inhibitors of Glioblastoma Stem Cells

121. Figure S6 from Recurrent Tumor Cell–Intrinsic and –Extrinsic Alterations during MAPKi-Induced Melanoma Regression and Early Adaptation

123. Supplementary Table 1 from A Molecular Screening Approach to Identify and Characterize Inhibitors of Glioblastoma Stem Cells

124. Supplementary Methods, References and Legends from Recurrent Tumor Cell–Intrinsic and –Extrinsic Alterations during MAPKi-Induced Melanoma Regression and Early Adaptation

125. Supplementary Figure 1 from A Molecular Screening Approach to Identify and Characterize Inhibitors of Glioblastoma Stem Cells

126. Supplementary Figure 5 from A Molecular Screening Approach to Identify and Characterize Inhibitors of Glioblastoma Stem Cells

127. Supplementary Table 2 from A Molecular Screening Approach to Identify and Characterize Inhibitors of Glioblastoma Stem Cells

128. Supplementary Figure 7 from A Molecular Screening Approach to Identify and Characterize Inhibitors of Glioblastoma Stem Cells

129. Supplementary Figure 6 from A Molecular Screening Approach to Identify and Characterize Inhibitors of Glioblastoma Stem Cells

130. Supplementary Figure 4 from A Molecular Screening Approach to Identify and Characterize Inhibitors of Glioblastoma Stem Cells

131. Supplementary Table S1-S3 from Recurrent Tumor Cell–Intrinsic and –Extrinsic Alterations during MAPKi-Induced Melanoma Regression and Early Adaptation

133. Supplementary Methods and Figure Legend from A Molecular Screening Approach to Identify and Characterize Inhibitors of Glioblastoma Stem Cells

134. Data from A High-Content Screen Identifies Drugs That Restrict Tumor Cell Extravasation across the Endothelial Barrier

135. Supplementary Figure 2 from A High-Content Screen Identifies Drugs That Restrict Tumor Cell Extravasation across the Endothelial Barrier

136. Supplementary Figure 1 from A High-Content Screen Identifies Drugs That Restrict Tumor Cell Extravasation across the Endothelial Barrier

137. Supplementary Figure S1 from PSA-Targeted Alpha-, Beta-, and Positron-Emitting Immunotheranostics in Murine Prostate Cancer Models and Nonhuman Primates

138. Supplementary Figure 7 from A High-Content Screen Identifies Drugs That Restrict Tumor Cell Extravasation across the Endothelial Barrier

139. Supplementary Figure 4 from A High-Content Screen Identifies Drugs That Restrict Tumor Cell Extravasation across the Endothelial Barrier

140. Supplementary Methods from A High-Content Screen Identifies Drugs That Restrict Tumor Cell Extravasation across the Endothelial Barrier

141. Supplementary Figure 3 from A High-Content Screen Identifies Drugs That Restrict Tumor Cell Extravasation across the Endothelial Barrier

142. Supplementary Data from High-Throughput Screening Identifies Two Classes of Antibiotics as Radioprotectors: Tetracyclines and Fluoroquinolones

143. Supplementary Methods from PSA-Targeted Alpha-, Beta-, and Positron-Emitting Immunotheranostics in Murine Prostate Cancer Models and Nonhuman Primates

144. Supplementary Table S1 from PSA-Targeted Alpha-, Beta-, and Positron-Emitting Immunotheranostics in Murine Prostate Cancer Models and Nonhuman Primates

145. Supplementary Figure 5 from A High-Content Screen Identifies Drugs That Restrict Tumor Cell Extravasation across the Endothelial Barrier

146. Supplementary Figure 6 from A High-Content Screen Identifies Drugs That Restrict Tumor Cell Extravasation across the Endothelial Barrier

147. Early Reduction of Glucose Consumption Is a Biomarker of Kinase Inhibitor Efficacy Which Can Be Reversed with GLUT1 Overexpression in Lung Cancer Cells

148. Low complexity domains of the nucleocapsid protein of SARS-CoV-2 form amyloid fibrils.

149. Osteopontin as a Biomarker in Chronic Kidney Disease.

150. Pacritinib inhibits glucose consumption in squamous cell lung cancer cells by targeting FLT3.

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