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256 results on '"Agustí, Àlvar"'

105. Amerindian Ancestry Influences Genetic Susceptibility to Chronic Obstructive Pulmonary Disease

Author

Díaz-Peña, Roberto, Boekstegers, Felix, Silva, Rafael S., Jaime, Sergio, Hosgood, H. Dean, Miravitlles, Marc, Agustí, Àlvar, Lorenzo Bermejo, Justo, Olloquequi, Jordi, and Universitat Autònoma de Barcelona

Subjects
Linkage disequilibrium, Genetic genealogy, Medicine (miscellaneous), genome wide association studies (GWAS), lcsh:Medicine, Single-nucleotide polymorphism, Genome-wide association study, Chronic obstructive pulmonary disease (COPD), Autoimmunity, Article, Hispanic paradox, Pathogenesis, 03 medical and health sciences, immune dysfunction, 0302 clinical medicine, Genetic predisposition, Medicine, chronic obstructive pulmonary disease (COPD), Genome wide association studies (GWAS), 030304 developmental biology, Ancestry, 0303 health sciences, COPD, business.industry, ancestry, autoimmunity, lcsh:R, personalized medicine, medicine.disease, Personalized medicine, 030228 respiratory system, Immune dysfunction, Immunology, business
Abstract

The contribution of genetic ancestry on chronic obstructive pulmonary disease (COPD) predisposition remains unclear. To explore this relationship, we analyzed the associations between 754,159 single nucleotide polymorphisms (SNPs) and risk of COPD (n = 214 cases, 193 healthy controls) in Talca, Chile, considering the genetic ancestry and established risk factors. The proportion of Mapuche ancestry (PMA) was based on a panel of 45 Mapuche reference individuals. Five PRDM15 SNPs and two PPP1R12B SNPs were associate with COPD risk (p = 0.05 to 5&times, 10&minus, 4) in those individuals with lower PMA. Based on linkage disequilibrium and sliding window analyses, an adjacent PRDM15 SNPs were associated with COPD risk in the lower PMA group (p = 10&minus, 3 to 3.77&times, 8). Our study is the first to report an association between PPP1R12B and COPD risk, as well as effect modification between ethnicity and PRDM15 SNPs in determining COPD risk. Our results are biologically plausible given that PPP1R12B and PRDM15 are involved in immune dysfunction and autoimmunity, providing mechanistic evidence for COPD pathogenesis and highlighting the importance to conduct more genome wide association studies (GWAS) in admixed populations with Amerindian descent.

Published
2020

111. PRDM15 Is Associated with Risk of Chronic Obstructive Pulmonary Disease in a Rural Population in Chile

Author

Hosgood III, H. Dean, primary, Díaz-Peña, Roberto, additional, Blansky, Deanna, additional, Jaime, Sergio, additional, Parra, Viviana, additional, Boekstegers, Felix, additional, Bermejo, Justo Lorenzo, additional, García-Valero, José, additional, Montes, Juan F., additional, Valdivia, Gonzalo, additional, Miravitlles, Marc, additional, Agustí, Àlvar, additional, Silva, Rafael S., additional, and Olloquequi, Jordi, additional

Published
2020
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118. [Translated article] Histology Study of Postmortem Lung Biopsies in Patients With Covid-19 Pneumonia

Author

Sellarés, Jacobo, Guerrero, Carlos, Martínez, Daniel, Benegas, Mariana, Cuerpo, Sandra, Hernández-González, Fernanda, Libreros, Alejandra, Guzman, Rudith, Guirao, Ángela, Boada, Marc, Sánchez-Lorente, David, Albacar, Núria, Grando, Leandro, Paglialunga, Pablo, García, Francisco Javier, Faner, Rosa, Agusti, Alvar, Sibila, Oriol, Sanchez, Marcelo, Molins, Laureano, and Ramírez, José

Published
2022
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119. Estudio histológico mediante biopsia pulmonar post mortem en pacientes con neumonía por COVID-19

Author

Sellarés, Jacobo, Guerrero, Carlos, Martínez, Daniel, Benegas, Mariana, Cuerpo, Sandra, Hernández-González, Fernanda, Libreros, Alejandra, Guzman, Rudith, Guirao, Ángela, Boada, Marc, Sánchez-Lorente, David, Albacar, Núria, Grando, Leandro, Paglialunga, Pablo, García, Francisco Javier, Faner, Rosa, Agusti, Alvar, Sibila, Oriol, Sanchez, Marcelo, Molins, Laureano, and Ramírez, José

Published
2022
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120. [Translated article] ANTES Program: One Year on

Author

Agustí, Alvar, Alcázar, Bernardino, Ancochea, Julio, Casanova, Ciro, Celli, Bartolome, Cosio, Borja, Echave-Sustaeta, José María, Villar, Alberto Fernandez, Rivero, Juan Luis Garcia, González, Cruz, Izquierdo, José Luis, Lopez-Campos, José Luis, Trigo, José María Marín, Sánchez, Javier Martín, Miravitlles, Marc, Molina, Jesús, Peces-Barba, Germán, Roman, Miguel, Cataluña, Juan José Soler, and Villar-Alvarez, Felipe

Published
2022
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121. ANTES: Un año después en la EPOC

Author

Agustí, Alvar, Alcázar, Bernardino, Ancochea, Julio, Casanova, Ciro, Celli, Bartolome, Cosio, Borja, Echave-Sustaeta, José María, Fernandez Villar, Alberto, Garcia Rivero, Juan Luis, González, Cruz, Izquierdo, José Luis, Lopez-Campos, José Luis, Marín Trigo, José María, Martín Sánchez, Javier, Miravitlles, Marc, Molina, Jesús, Peces-Barba, Germán, Roman, Miguel, Soler Cataluña, Juan José, and Villar-Alvarez, Felipe

Published
2022
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132. Inspiratory capacity-to-total lung capacity ratio and dyspnoea predict exercise capacity decline in COPD

Author

Ramon, Maria A., Ferrer, Jaume, Gimeno-Santos, Elena, Donaire-Gonzalez, David, Rodríguez, Esther, Balcells, Eva, de Batlle, Jordi, Benet, Marta, Guerra, Stefano, Sauleda, Jaume, Ferrer, Antoni, Farrero, Eva, Gea, Joaquim, Barberà, Joan A., Agustí, Àlvar, Rodriguez-Roisin, Robert, Antó, Josep M., and Garcia-Aymerich, Judith

Subjects
exercise capacity, disease progression, cohort study, respiratory system, 6-min walk distance, respiratory tract diseases, chronic obstructive pulmonary disease
Abstract

Background and objective: Exercise capacity decline is a predictor of mortality in patients with chronic obstructive pulmonary disease (COPD). Static pulmonary hyperinflation is a key determinant of exercise performance, but its effect on the longitudinal decline in exercise capacity remains unknown. We aimed to study the relationship between the inspiratory capacity‐to‐total lung capacity (IC/TLC) ratio and exercise capacity decline in COPD. Methods: We measured IC/TLC and other relevant clinical and functional variables in 342 clinically stable patients with COPD. The 6‐min walk distance (6MWD) was determined at recruitment and after a mean ± SD of 1.7 ± 0.3 years. The annual rate of change in 6MWD was calculated. Multiple imputation to account for losses during follow up was implemented, and multivariate regression was used to analyze predictive factors of 6MWD decline. Results: Mean decline rate in the 6MWD was 21.9 ± 34.1 m/year. In the bivariate analysis, patients with lower levels of IC/TLC had greater 6MWD decline (−27.4 ± 42.5, −24.9 ± 36.5 and −13.4 ± 39.9 m/year in the first, second and third tertile of IC/TLC, respectively; P‐for‐trend = 0.018). From other potential risk factors considered, dyspnoea, health status, serum C‐reactive protein and Borg dyspnoea score at the end of the exercise test were related to exercise capacity decline. In the multivariate regression model, only IC/TLC (β = 0.7 m/year per each percentage unit of IC/TLC; P = 0.007) and dyspnoea (mMRC ≥ 2) (β = −14.6 m/year; P = 0.013) were associated with the annual rate of 6MWD change. Conclusion: IC/TLC and dyspnoea in clinically stable patients with COPD predict their exercise capacity decline and may help to guide early therapeutic interventions.

Published
2016

133. COPD, An Issue of Clinics in Chest Medicine,

Author

Barnes, Peter J., Agustí, Alvar, Barnes, Peter J., and Agustí, Alvar

Subjects
Chest--Diseases, Respiratory organs--Diseases, Lungs--Diseases, Obstructive
Abstract

This issue of Clinics in Chest Medicine is Guest Edited by Peter J. Barnes FRS, FMedSci from Imperial College London and will fosus on COPD. Article topics include epidemiology, pathophysiology, cellular and molecular mechanisms and comorbidities of COPD, diagnosis and phenotype of COPD, pulmonary rehabilitation, asthma and COPD, biomarkers, bronchodilators, non invasive ventialtion, and new drug therapies.

Published
2014

139. Air pollution and biomarkers of systemic inflammation and tissue repair in COPD patients

Author

Dadvand, Payam, Nieuwenhuijsen, Mark J., Agustí, Àlvar, De Batlle, Jordi, Benet, Marta, Beelen, Rob, Cirach, Marta, Martinez, David, Hoek, Gerard, Basagaña, Xavier, Ferrer, Antoni, Ferrer, Jaume, Rodriguez-Roisin, Robert, Sauleda, Jaume, Guerra, Stefano, Antó, Josep M., Garcia-Aymerich, Judith, LS IRAS EEPI ME (Milieu epidemiologie), Risk Assessment of Toxic and Immunomodulatory Agents, IRAS RATIA2, IRAS RATIA-SIB, LS IRAS EEPI ME (Milieu epidemiologie), Risk Assessment of Toxic and Immunomodulatory Agents, IRAS RATIA2, and IRAS RATIA-SIB

Subjects
Pulmonary and Respiratory Medicine, COPD, medicine.medical_specialty, business.industry, Interleukin, Inflammation, Fibrinogen, medicine.disease, Systemic inflammation, Gastroenterology, Interquartile range, Internal medicine, Immunology, General Earth and Planetary Sciences, Medicine, medicine.symptom, business, General Environmental Science, Blood sampling, medicine.drug, Exposure assessment
Abstract

The origin(s) of systemic inflammation in patients with chronic obstructive pulmonary disease (COPD) is unclear. We investigated the impact of exposure to ambient air pollution on systemic biomarkers of inflammation (C-reactive protein (CRP), tumour necrosis factor-α, interleukin (IL)-6, IL-8 and fibrinogen) and tissue repair (hepatocyte growth factor (HGF)) in 242 clinically stable COPD patients (mean age 67.8 years and forced expiratory volume in 1 s 71.3% predicted) in Barcelona, Spain, in 2004-2006. A spatiotemporal exposure assessment framework was applied to predict ambient nitrogen dioxide (NO 2) and levels of particles with a 50% cut-off aerodynamic diameter of 2.5 μm (PM2.5) at each participant's home address during 10 periods of 24 h (lags 1-10) and 1 year prior to the blood sampling date. We used linear regression models to estimate associations between biomarkers and exposure levels. An interquartile range (IQR) increase in NO2 exposure in lag 5 was associated with 51%, 10% and 9% increases in CRP, fibrinogen and HGF levels respectively. We also observed 12% and 8% increases in IL-8 associated with an IQR increase in NO2 exposure in lag 3 and over the year before sampling, respectively. These increases were larger in former smokers. The results for PM2.5 were not conclusive. These results show that exposure to ambient NO2 increases systemic inflammation in COPD patients, especially in former smokers.

Published
2014

140. Lifetime Occupational Exposure to Dusts, Gases and Fumes Is Associated with Bronchitis Symptoms and Higher Diffusion Capacity in COPD Patients

Author

Rodríguez, Esther, Ferrer, Jaume, Zock, Jan Paul, Serra, Ignasi, Antó, Josep M., De Batlle, Jordi, Kromhout, Hans, Vermeulen, Roel, Donaire-González, David, Benet, Marta, Balcells, Eva, Monsó, Eduard, Gayete, Àngel, Garcia-Aymerich, Judith, Guerra, Stefano, Gea, Joaquim, Orozco-Levi, Mauricio, Vollmer, Ivan, Barberà, Joan Albert, Gómez, Federico P., Paré, Carles, Roca, Josep, Rodriguez-Roisin, Robert, Agustí, Àlvar, Freixa, Xavier, Rodriguez, Diego A., Gimeno, Elena, Portillo, Karina, Andreu, Jordi, Pallissa, Esther, Casan, Pere, Güell, Rosa, Giménez, Ana, Marín, Alicia, Morera, Josep, Farrero, Eva, Escarrabill, Joan, Ferrer, Antoni, Sauleda, Jaume, Togores, Bernat, Gáldiz, Juan Bautista, López, Lorena, Belda, José, LS IRAS EEPI EXAS (Arb.hyg+bl.st.kar.), Dep IRAS, LS IRAS EEPI GRA (Gezh.risico-analyse), IRAS RATIA2, IRAS RATIA-SIB, Risk Assessment of Toxic and Immunomodulatory Agents, LS IRAS EEPI EXAS (Arb.hyg+bl.st.kar.), Dep IRAS, LS IRAS EEPI GRA (Gezh.risico-analyse), IRAS RATIA2, IRAS RATIA-SIB, and Risk Assessment of Toxic and Immunomodulatory Agents

Subjects
Male, Chronic bronchitis, Exacerbation, Pulmonology, Chronic Obstructive Pulmonary Diseases, Epidemiology, humanos, lcsh:Medicine, Diffusion, Pulmonary Disease, Chronic Obstructive, DLCO, Diffusing capacity, Clinical Epidemiology, lcsh:Science, COPD, anciano, Multidisciplinary, medicine.diagnostic_test, Chronic obstructive pulmonary disease, resultado del tratamiento, Smoking, Statistics, Dust, Occupational and Industrial Health, Hospitalization, Treatment Outcome, Bronchitis, Medicine, Female, Gases, Public Health, Research Article, Spirometry, medicine.medical_specialty, Clinical Research Design, Job-exposure matrix, difusión, Jobs, Biostatistics, Environmental and Occupational Lung Diseases, Environmental Epidemiology, polvo, Fluid dynamics, Internal medicine, Occupational Exposure, medicine, Pols, hospitalización, Humans, Aged, Lifecourse Epidemiology, Emphysema, Survey Research, business.industry, lcsh:R, Pulmons -- Malalties obstructives -- Epidemiologia, hábito de fumar, medicine.disease, respiratory tract diseases, Dyspnea, Survey Methods, Physical therapy, Bronquitis, lcsh:Q, business, Mathematics
Abstract

Background: Occupational exposure to dusts, gases and fumes has been associated with reduced FEV1 and sputum production in COPD patients. The effect of occupational exposure on other characteristics of COPD, especially those reflecting emphysema, has not been studied in these patients. Methods: We studied 338 patients hospitalized for a first exacerbation of COPD in 9 Spanish hospitals, obtaining full occupational history in a face-to-face interview; job codes were linked to a job exposure matrix for semi-quantitative estimation of exposure to mineral/biological dust, and gases/fumes for each job held. Patients underwent spirometry, diffusing capacity testing and analysis of gases in stable conditions. Quality of life, dyspnea and chronic bronchitis symptoms were determined with a questionnaire interview. A high-resolution CT scan was available in 133 patients. Results: 94% of the patients included were men, with a mean age of 68(8.5) years and a mean FEV1% predicted 52 (16). High exposure to gases or fumes was associated with chronic bronchitis, and exposure to mineral dust and gases/fumes was associated with higher scores for symptom perception in the St. George's questionnaire. No occupational agent was associated with a lower FEV1. High exposure to all occupational agents was associated with better lung diffusion capacity, in long-term quitters. In the subgroup with CT data, patients with emphysema had 18% lower DLCO compared to those without emphysema. Conclusions: In our cohort of COPD patients, high exposure to gases or fumes was associated with chronic bronchitis, and high exposure to all occupational agents was consistently associated with better diffusion capacity in long-term quitters., The PAC-COPD Study is funded by grants from the Fondo de Investigacion Sanitaria (FIS PI020541), Ministry of Health, Spain; Agencia d'Avaluacio de Tecnologia i Recerca Mediques (AATRM 035/20/02), Catalan Government; Spanish Society of Pulmonology and Thoracic Surgery (SEPAR 2002/137); Catalan Pulmonology Foundation (FUCAP 2003 Beca Maria Rava); Red RESPIRA (RTIC C03/11); Red RCESP (RTIC C03/09); Fondo de Investigacion Sanitaria (PI052486); Fondo de Investigacion Sanitaria (PI052302); Fondo de Investigacion Sanitaria (PI060684); Fundacio La Marato de TV3 (num. 041110); and Novartis Farmaceutica, Spain. CIBERESP and CIBERES are funded by Instituto de Salud Carlos III, Ministry of Health, Spain. Judith Garcia-Aymerich has a researcher contract from Instituto de Salud Carlos III (CP05/00118), Ministry of Health, Spain. The funders had no role in study design, data collection and analysis, decision to publish, or preparation of the manuscript.

Published
2014

142. Air pollution and biomarkers of systemic inflammation and tissue repair in COPD patients

Author

LS IRAS EEPI ME (Milieu epidemiologie), Risk Assessment of Toxic and Immunomodulatory Agents, IRAS RATIA2, IRAS RATIA-SIB, Dadvand, Payam, Nieuwenhuijsen, Mark J., Agustí, Àlvar, De Batlle, Jordi, Benet, Marta, Beelen, Rob, Cirach, Marta, Martinez, David, Hoek, Gerard, Basagaña, Xavier, Ferrer, Antoni, Ferrer, Jaume, Rodriguez-Roisin, Robert, Sauleda, Jaume, Guerra, Stefano, Antó, Josep M., Garcia-Aymerich, Judith, LS IRAS EEPI ME (Milieu epidemiologie), Risk Assessment of Toxic and Immunomodulatory Agents, IRAS RATIA2, IRAS RATIA-SIB, Dadvand, Payam, Nieuwenhuijsen, Mark J., Agustí, Àlvar, De Batlle, Jordi, Benet, Marta, Beelen, Rob, Cirach, Marta, Martinez, David, Hoek, Gerard, Basagaña, Xavier, Ferrer, Antoni, Ferrer, Jaume, Rodriguez-Roisin, Robert, Sauleda, Jaume, Guerra, Stefano, Antó, Josep M., and Garcia-Aymerich, Judith

Published
2014

143. Lifetime occupational exposure to dusts, gases and fumes is associated with bronchitis symptoms and higher diffusion capacity in COPD patients

Author

LS IRAS EEPI EXAS (Arb.hyg+bl.st.kar.), Dep IRAS, LS IRAS EEPI GRA (Gezh.risico-analyse), IRAS RATIA2, IRAS RATIA-SIB, Risk Assessment of Toxic and Immunomodulatory Agents, Rodríguez, Esther, Ferrer, Jaume, Zock, Jan Paul, Serra, Ignasi, Antó, Josep M., De Batlle, Jordi, Kromhout, Hans, Vermeulen, Roel, Donaire-González, David, Benet, Marta, Balcells, Eva, Monsó, Eduard, Gayete, Àngel, Garcia-Aymerich, Judith, Guerra, Stefano, Gea, Joaquim, Orozco-Levi, Mauricio, Vollmer, Ivan, Barberà, Joan Albert, Gómez, Federico P., Paré, Carles, Roca, Josep, Rodriguez-Roisin, Robert, Agustí, Àlvar, Freixa, Xavier, Rodriguez, Diego A., Gimeno, Elena, Portillo, Karina, Andreu, Jordi, Pallissa, Esther, Casan, Pere, Güell, Rosa, Giménez, Ana, Marín, Alicia, Morera, Josep, Farrero, Eva, Escarrabill, Joan, Ferrer, Antoni, Sauleda, Jaume, Togores, Bernat, Gáldiz, Juan Bautista, López, Lorena, Belda, José, LS IRAS EEPI EXAS (Arb.hyg+bl.st.kar.), Dep IRAS, LS IRAS EEPI GRA (Gezh.risico-analyse), IRAS RATIA2, IRAS RATIA-SIB, Risk Assessment of Toxic and Immunomodulatory Agents, Rodríguez, Esther, Ferrer, Jaume, Zock, Jan Paul, Serra, Ignasi, Antó, Josep M., De Batlle, Jordi, Kromhout, Hans, Vermeulen, Roel, Donaire-González, David, Benet, Marta, Balcells, Eva, Monsó, Eduard, Gayete, Àngel, Garcia-Aymerich, Judith, Guerra, Stefano, Gea, Joaquim, Orozco-Levi, Mauricio, Vollmer, Ivan, Barberà, Joan Albert, Gómez, Federico P., Paré, Carles, Roca, Josep, Rodriguez-Roisin, Robert, Agustí, Àlvar, Freixa, Xavier, Rodriguez, Diego A., Gimeno, Elena, Portillo, Karina, Andreu, Jordi, Pallissa, Esther, Casan, Pere, Güell, Rosa, Giménez, Ana, Marín, Alicia, Morera, Josep, Farrero, Eva, Escarrabill, Joan, Ferrer, Antoni, Sauleda, Jaume, Togores, Bernat, Gáldiz, Juan Bautista, López, Lorena, and Belda, José

Published
2014

144. Enfermedad pulmonar obstructiva crónica. Actualización 2014

Author

López Giraldo, Alejandra, Rodríguez Roisin, Robert, Agustí, Àlvar, López Giraldo, Alejandra, Rodríguez Roisin, Robert, and Agustí, Àlvar

Abstract

Chronic obstructive pulmonary disease (COPD) is a major public health problem because of its high prevalence (10% in Spain), raising incidence (the World Health Organization predicts that it will be the third cause of death in the world by the year 2020) and high associated socio-economic cost. Over the last few years, there have been very significant advances in the understanding and treatment of COPD, so it is now considered a “preventable and treatable disease”. The following text summarizes the main diagnostic and therapeutic aspects of this disease according to the last version (2014) of the GOLD (Global Strategy for the Diagnosis, Management and Prevention of chronic obstructive pulmonary disease) strategy document, which is the global reference for this disease. In particular, it is of note that the current recommendation for the assessment of COPD is multidimensional. It includes the assessment of the level of current symptoms, as well as the traditional assessment of airflow limitation severity and the previous history of exacerbations that can help in predicting the risk of future events. According to these three dimensions, four different groups of patients can be identified (A, B, C, and D), and specific pharmacological treatment is recommended for each of them., La enfermedad pulmonar obstructiva crónica (EPOC) es un problema socio-sanitario de primera magnitud por su elevada prevalencia (10% en nuestro País), incidencia creciente (la Organización Mundial de la Salud estima que será la tercera causa de muerte en el mundo en 2020) y elevados costes socio-económicos asociados. En los últimos años se han producido avances notables en el conocimiento y tratamiento de la enfermedad que es ya considerada una enfermedad “prevenible y tratable”. El texto que sigue resume los principales aspectos diagnósticos y terapéuticos de la EPOC a partir de la última versión (2014) del documento de la estrategia GOLD (Global Strategy for the Diagnosis, Management and Prevention of chronic obstructive pulmonary disease), el documento de referencia mundial en este ámbito. Hace énfasis especial en la nueva forma de determinar la gravedad de la enfermedad, basada en una valoración multi-dimensional de la EPOC que tiene en cuenta no sólo la severidad de la limitación al flujo aéreo (como se había hecho tradicionalmente hasta ahora), sino que añade dos dimensiones de gran valor clínico y pronóstico (el nivel de síntomas del paciente y la historia previa de agudizaciones de la enfermedad). Con estas tres dimensiones, los pacientes con EPOC se clasifican actualmente en uno de cuatro posibles grupos (A, B, C o D) y el tratamiento farmacológico recomendado se ajusta a esta clasificación.

Published
2014

145. Fe de errores de “Informe 2017 de la Iniciativa Global para el Diagnóstico, Tratamiento y Prevención de la Enfermedad Pulmonar Obstructiva Crónica: Resumen Ejecutivo de GOLD” [Arch Bronconeumol. 2017;53:128–49]

Author

Vogelmeier, Claus F., Criner, Gerard J., Martínez, Fernando J., Anzueto, Antonio, Barnes, Peter J., Bourbeau, Jean, Celli, Bartolome R., Chen, Rongchang, Decramer, Marc, Fabbri, Leonardo M., Frith, Peter, Halpin, David M.G., Varela, M. Victorina López, Nishimura, Masaharu, Roche, Nicolás, Rodríguez-Roisin, Roberto, Sin, Don D., Singh, Dave, Stockley, Robert, Vestbo, Jørgen, Wedzicha, Jadwiga A., and Agustí, Alvar

Published
2017
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146. Colaboradores

Author

Adams, Lewis, Adler, Dan Elie, Agusti, Alvar, Akoumianaki, Evangelia, Alberg, Anthony J., Albertine, Kurt H., Alexander, Barbara D., Alfille, Paul H., Anantham, Devanand, Arenberg, Douglas A., Ayas, Najib T., Bagchi, Aranya, Balmes, John Randolph, Banaei, Niaz, Barnett, Christopher F., Baughman, Robert P., Becklake, Margaret R., Benditt, Joshua O., Benowitz, Neal L., Bhakta, Nirav R., Bhave, Anant D., Blanc, Paul D., Bleecker, Eugene R., Bove, Alfred A., Bradley, T. Douglas, Brambilla, Elisabeth, Broaddus, V. Courtney, Brochard, Laurent, Brock, Malcolm V., Brown, Kevin K., Brunetta, Paul G., Cadranel, Jacques, Celli, Bartolome, Chan, Edward D., Channick, Richard N., Chastre, Jean, Cheng, Guang-Shing, Chin, Kelly, Chung, Kian Fan, Clerici, Pr Christine, Colby, Thomas V., Collard, Harold R., Cool, Carlyne D., Cordier, Jean-François, Cordioli, Ricardo Luiz, Corte, Tamera J., Cottin, Vincent, Courey, Mark S., Cowie, Robert L., Crothers, Kristina, Curley, Gerard F., Daley, Charles L., Davis, J. Lucian, De Marco, Teresa, Deresinski, Stanley C., Deroose, Christophe, Dobbs, Leland G., Dooms, Christophe, Downey, Gregory P., Bois, Roland M. du, Dulohery, Megan M., Effros, Richard M., Eisner, Mark D., Elicker, Brett M., Ernst, Armin, Ernst, Joel D., Fahy, John V., Fedullo, Peter F., Feller-Kopman, David, Fenster, Brett E., Fingerlin, Tasha E., Fontenot, Andrew P., Frankel, Stephen K., Garcia, Joe G.N., Gebhart, G.F., Gilstrap, Daniel Lee, Girard, Nicolas, Gladwin, Mark T., Glenny, Robb W., Gold, Warren M., Gotway, Michael B., Grasselli, Giacomo, Greenberg, James M., Griffith, David E., Gruden, James F., Han, MeiLan King, Henderson, William, Hill, Nicholas S., Hoover, Wynton, Hopewell, Philip C., Horan-Saullo, Jennifer L., Horner, Richard L., Huang, Laurence, Huchon, Gérard, Inoue, Yoshikazu, Iseman, Michael D., Jackson, James E., Jakubzick, Claudia V., Janssen, Julius P., Jett, James R., Jones, Kirk, Judson, Marc A., Kato-Maeda, Midori, Kavanagh, Brian P., Keshavjee, Shaf, Kim, Kami, Kimoff, R. John, King, Talmadge E., Jr., Klein, Jeffrey S., Koth, Laura L., Kotloff, Robert M., Kraft, Monica, Küpeli, Elif, Laffey, John G., Lapinsky, Stephen E., Lazarus, Stephen C., Lee, Frances Eun-Hyung, Lee, Jarone, Lee, Y. C. Gary, Lee, Warren L., Lee-Chiong, Teofilo L., Jr., Lemière, Catherine, Light, Richard W., Limper, Andrew H., Loddenkemper, Robert, Lugogo, Njira, Luisetti, Maurizio, Luks, Andrew M., Luyt, Charles-Edouard, Machado, Roberto F., MacIntyre, Neil R., MacNee, William, Madtes, David K., Maier, Lisa A., Maldonado, Fabien, Malhotra, Atul, Martin, Thomas R., Maskell, Nick A., Mason, Robert J., Massion, Pierre P., Matthay, Michael A., Matthay, Richard A., Mayer, Annyce S., Mazzone, Stuart B., McCool, F. Dennis, McCormack, Francis Xavier, Mehta, Atul C., Menéndez, Rosario, Morgenthau, Adam S., Morris, Alison, Morris, Timothy A., Muncey, Aaron R., Murray, John F., Myers, Jeffrey L., Nadel, Jay A., Nelson-Piercy, Catherine, Neuman, Tom S., Nosanchuk, Joshua D., O’Riordan, Thomas G., Ortega, Victor Enrique, Panse, Prasad M., Pao, William, Paré, Peter A., Park, Mecânica e Energética do Sistema Respiratório R., Pastis, Nicholas J., Patroniti, Nicolò, Patterson, Karen C., Pesenti, Antonio, Pickens, Allan, Pinsky, Benjamin A., Pletcher, Steven D., Powell, Frank L., Que, Loretta G., Redente, Elizabeth F., Riches, David W.H., Robinson, Bruce W.S., Rodriguez-Roisin, Roberto, Rose, Cecile S., Routes, John M., Rowe, Steven M., Ryan, Clodagh M., Ryu, Jay H., Samet, Jonathan M., Sandrock, Christian E., Schoene, Robert B., Schwartz, David A., Schwartzstein, Richard M., Schwarz, Marvin I., Selman, Moisés, Sequist, Lecia V., Shannon, John M., Shovlin, Claire L., Silvestri, Gerard A., Simonian, Philip L., Singer, Jonathan P., Slutsky, Arthur S., Smaldone, Gerald C., Solomon, George M., Sorscher, Eric J., Swenson, Erik R., Tanner, Nichole T., Tanowitz, Herbert B., Torres, Antoni, Trapnell, Bruce C., Travis, William David, Treanor, John J., Tzelepis, George E., Vandenplas, Olivier, Vansteenkiste, Johan F., Varghese, Thomas K., Jr., Vestbo, Jørgen, Wagner, Peter D., Wahidi, Momen M., Wallace, W. Dean, Weiss, Louis M., Weiss, Scott T., Wells, Athol U., West, John B., White, Douglas B., Wiener-Kronish, Jeanine P., Wikenheiser-Brokamp, Kathryn A., Woodruff, Prescott G., Wunderink, Richard G., Yeh, D. Dante, Zemans, Rachel L., Zimmerman, Leslie, and Zuwallack, Richard L.

Published
2017
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147. Air Pollution and Biomarkers of Systemic Inflammation and Tissue Repair in COPD Patients

Author

Dadvand*, Payam, primary, Nieuwenhuijsen, Mark J, additional, Agustí, Àlvar, additional, de Batlle, Jordi, additional, Benet, Marta, additional, Beelen, Rob, additional, Cirach, Marta, additional, Martinez, David, additional, Hoek, Gerard, additional, Basagaña, Xavier, additional, Ferrer, Antoni, additional, Ferrer, Jaume, additional, Rodriguez-Roisin, Robert, additional, Sauleda, Jaume, additional, Guerra, Stefano, additional, Antó, Josep M, additional, and Garcia-Aymerich, Judith, additional

Published
2014
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