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53. Basis for drug selectivity of plasmepsin IX and X inhibition for Plasmodium falciparum and vivax

54. Landscape of human antibody recognition of the SARS-CoV-2 receptor binding domain

58. Heterologous SARS‐CoV‐2 IgA neutralising antibody responses in convalescent plasma.

59. Simultaneous evaluation of antibodies that inhibit SARS-CoV-2 variants via multiplex assay

60. Application of 23 Novel Serological Markers for Identifying Recent Exposure to Plasmodium vivax Parasites in an Endemic Population of Western Thailand

61. IgG Antibody Responses Are Preferential Compared With IgM for Use as Serological Markers for Detecting Recent Exposure to Plasmodium vivax Infection

62. Simultaneous evaluation of antibodies that inhibit SARS-CoV-2 RBD variants with a novel competitive multiplex assay

63. Naturally acquired blocking human monoclonal antibodies to Plasmodium vivax reticulocyte binding protein 2b

67. Surface Area-to-Volume Ratio, not Cellular Viscoelasticity is the Major Determinant of Red Blood Cell Traversal through Small Channels

68. Basis for Drug Selectivity of Plasmepsin IX and X Inhibition for Plasmodium falciparum and vivax

69. The FK506 protein Fp3 counteracts protein phosphatase 1 to maintain meiotic recombination checkpoint activity

70. The FK506 Binding Protein Fpr3 Counteracts Protein Phosphatase 1 to Maintain Meiotic Recombination Checkpoint Activity

72. A comparison of non-magnetic and magnetic beads for measuring IgG antibodies against Plasmodium vivax antigens in a multiplexed bead-based assay using Luminex technology (Bio-Plex 200 or MAGPIX)

74. Surface area‐to‐volume ratio, not cellular viscoelasticity, is the major determinant of red blood cell traversal through small channels

75. IgG antibody responses are preferential compared to IgM for use as serological markers for detecting recent exposure to Plasmodium vivax infection

77. Surface area-to-volume ratio, not cellular rigidity, determines red blood cell traversal through small capillaries

78. Immunogenic profile of SARS-CoV-2 spike in individuals recovered from COVID-19

79. Naturally acquired blocking human monoclonal antibodies to Plasmodium vivax reticulocyte binding protein 2b

80. Dual Plasmepsin-Targeting Antimalarial Agents Disrupt Multiple Stages of the Malaria Parasite Life Cycle

84. Complement in malaria immunity and vaccines

86. Antibodies to Plasmodium vivax reticulocyte binding protein 2b are associated with protection against P. vivax malaria in populations living in low malaria transmission regions of Brazil and Thailand

87. Neutralising antibodies block the function of Rh5/Ripr/CyRPA complex during invasion of Plasmodium falciparum into human erythrocytes

88. Evaluation of 4-Amino 2-Anilinoquinazolines against Plasmodium and Other Apicomplexan Parasites In Vitro and in a P. falciparum Humanized NOD- scid IL2Rγ null Mouse Model of Malaria

89. Antibody responses to Plasmodium vivax Duffy binding and Erythrocyte binding proteins predict risk of infection and are associated with protection from clinical Malaria

90. Structure of Plasmodium falciparum Rh5–CyRPA–Ripr invasion complex

91. Surface area‐to‐volume ratio, not cellular viscoelasticity, is the major determinant of red blood cell traversal through small channels.

92. Complement in malaria immunity and vaccines.

93. Development and validation of serological markers for detecting recent exposure toPlasmodium vivaxinfection

94. Identification of highly-protective combinations of Plasmodium vivax recombinant proteins for vaccine development

95. Author response: Identification of highly-protective combinations of Plasmodium vivax recombinant proteins for vaccine development

98. Asymptomatic Plasmodium vivax infections induce robust IgG responses to multiple blood-stage proteins in a low-transmission region of western Thailand

99. Plasmodium falciparum ligand binding to erythrocytes induce alterations in deformability essential for invasion

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