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57. High resolution allelotype of nonfunctional pancreatic endocrine tumors: identification of two molecular subgroups with clinical implications

Author

Rigaud G, edoardo missiaglia, Ps, Moore, Zamboni G, Falconi M, Talamini G, Pesci A, Baron A, Lissandrini D, Rindi G, Grigolato P, Pederzoli P, and Scarpa A

Subjects
Adult, Male, Adenoma, Analysis of Variance, Ploidies, Islet Cell/*genetics Adult Aged Analysis of Variance Chromosome Deletion DNA, Neoplasm/genetics Female Human *Loss of Heterozygosity Male Microsatellite Repeats, Loss of Heterozygosity, DNA, Neoplasm, Middle Aged, Adenoma, Islet Cell, Survival Analysis, Pancreatic Neoplasms, Multivariate Analysis, Multiple Endocrine Neoplasia Type 1, Humans, Female, Chromosome Deletion, Aged, Microsatellite Repeats
Abstract

A high resolution allelotype for nonfunctional pancreatic endocrine tumors (NF-PETs) has been generated by microsatellite analysis of DNA from 16 frozen cases, each probed with 394 markers. Two subgroups of NF-PETs were found. Seven cases showed frequent, large allelic deletions [loss of heterozygosity (LOH)] with an average fractional allelic loss (FAL) of 0.55, whereas nine cases showed a small number of random losses with a FAL of 0.15. Designated high or low FAL, respectively, these genetic phenotypes showed correlation with the ploidy status: high-FAL tumors were aneuploid, low-FAL were diploid. Chromosomes 6q and 11q showed LOH in60% of cases. About 50% of cases had losses on 11p, 20q, and 21. Selected LOH analysis on an additional 16 paraffin-embedded NF-PETs confirmed the high frequency of 6q and 11q LOH. The allelotype of NF-PET is markedly different from that of either ductal or acinar tumors of the pancreas as well as from that of functional-PETs. Moreover, whereas deletions involving chromosome 11 also are a feature of functional-PETs, the involvement of chromosome 6q is characteristic of NF-PETs. Survival analysis showed that none of the single chromosomal alterations was associated with outcome, whereas ploidy status is an independent factor adding prognostic information to that furnished by the proliferative index measured by Ki-67 immunohistochemistry.

Published
2001

58. Outcome and quality of life in chronic pancreatitis

Author

Talamini, G., Bassi, C., Butturini, G., Massimo Falconi, Casetti, L., Gumbus, A. A., Carrara, S., Fantin, A., Pederzoli, P., G., Talamini, C., Bassi, G., Butturini, Falconi, Massimo, Casetti, L, Gumbs, Aa, Carrara, S, Fantin, A, and P., Pederzoli

Published
2001

59. Role of fistulography in evaluating pancreatic fistula after pancreaticoduodenectomy.

Author

Faccioli, N., Foti, G., Molinari, E., Hermans, J.J., Comai, A., Talamini, G., Bassi, C., Pozzi-Mucelli, R., Faccioli, N., Foti, G., Molinari, E., Hermans, J.J., Comai, A., Talamini, G., Bassi, C., and Pozzi-Mucelli, R.

Abstract

01 maart 2012, Item does not contain fulltext, OBJECTIVE: To evaluate the usefulness of fistulography as a diagnostic and management tool for clinically suspected pancreatic fistulas (PF) after pancreaticoduodenectomy (PD). METHODS: 84 consecutive fistulographies were performed for clinical suspicion of PF and retrospectively analysed. We radiologically defined two types of PF by means of fistulography, PF1 in the case of primary filling with contrast agent of the jejunal loop or stomach and PF2 in the case of secondary filling of the jejunal loop or stomach through a fistulous tract or a fluid collection. RESULTS: In 35/84 (41.7%) of the fistulograms, a PF1 was demonstrated owing to an instantaneous opacification of the intestinal lumen or the stomach, without evidence of a fistulous tract or fluid collection. In 49/84 (58.3%) fistulograms, a PF2 was demonstrated by the depiction of a fluid collection and/or a fistulous tract and a communication with the intestinal loop or the stomach anastomised with the pancreas. The mean healing time of a PF after PD was 2.7 days for PF1, and 9.8 days for PF2. CONCLUSION: Fistulography helps in the confirmation of clinically suspect PF, and can distinguish PF1 and PF2, thus decreasing post-operative morbidity significantly.

Published
2012

60. Role of fistulography in evaluating pancreatic fistula after pancreaticoduodenectomy

Author

Faccioli, N. (Niccolo), Foti, G. (G.), Molinari, F. (Francesca), Hermans, J.J. (John), Comai, A. (A.), Talamini, G. (G.), Bassi, C. (Claudio), Pozzi Mucelli, R. (Roberto Silvio), Faccioli, N. (Niccolo), Foti, G. (G.), Molinari, F. (Francesca), Hermans, J.J. (John), Comai, A. (A.), Talamini, G. (G.), Bassi, C. (Claudio), and Pozzi Mucelli, R. (Roberto Silvio)

Abstract

Objective: To evaluate the usefulness of fistulography as a diagnostic and management tool for clinically suspected pancreatic fistulas (PF) after pancreaticoduodenectomy (PD). Methods: 84 consecutive fistulographies were performed for clinical suspicion of PF and retrospectively analysed. We radiologically defined two types of PF by means of fistulography, PF1 in the case of primary filling with contrast agent of the jejunal loop or stomach and PF2 in the case of secondary filling of the jejunal loop or stomach through a fistulous tract or a fluid collection. Results: In 35/84 (41.7%) of the fistulograms, a PF1 was demonstrated owing to an instantaneous opacification of the intestinal lumen or the stomach, without evidence of a fistulous tract or fluid collection. In 49/84 (58.3%) fistulograms, a PF2 was demonstrated by the depiction of a fluid collection and/or a fistulous tract and a communication with the intestinal loop or the stomach anastomised with the pancreas. The mean healing time of a PF after PD was 2.7 days for PF1, and 9.8 days for PF2. Conclusion: Fistulography helps in the confirmation of clinically suspect PF, and can distinguish PF1 and PF2, thus decreasing post-operative morbidity significantly.

Published
2012
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61. Chronic pancreatitis: relationship to acute pancreatitis and pancreatic cancer

Author

Talamini, G., Massimo Falconi, Bassi, C., Mastromauro, M., Salvia, R., Pederzoli, P., G., Talamini, Falconi, Massimo, C., Bassi, Mastromauro, M, Salvia, R, and P., Pederzoli

Subjects
Adenocarcinoma, Alcohol Drinking, Carcinogens, Cystic Fibrosis, Diagnosis, Differential, Epidemiology, Genetics, Incidence, Neoplasms, Risk Factors, Smoking
Abstract

No abstract available. Image: University of Verona logo. Verona, Italy., JOP. Journal of the Pancreas, Vol 1, N° 3S (2000): September (Suppl.) - p. 69-177

Published
2000

66. Prognosis of pancreatic carcinoma: why are differences to be found between european and japanese patients?

Author

Bassi, Claudio, Butturini, G., Falconi, Massimo, Talamini, G., Sartori, N., Balzano, G. P., Zerbi, A., Di Carlo, V., Zamboni, Giuseppe, Pederzoli, Paolo, C.G. Dervenis, C., Bassi, G., Butturini, Falconi, Massimo, G., Talamini, N., Sartori, G. P., Balzano, A., Zerbi, V., Di Carlo, G., Zamboni, and P., Pederzoli

Subjects
"pancreatic carcinoma", "Prognosis", "staging"
Published
1996

67. Duodenal Acidity May Increase the Risk of Pancreatic Cancer in the Course of Chronic Pancreatitis: An Etiopathogenetic Hypothesis

Author

Talamini G and Talamini G

Abstract

Chronic pancreatitis patients have an increased risk of developing pancreatic cancer. The cause of this increase has yet to be fully explained but smoking and inflammation may play an important role. To these, we must now add a new potential risk factor, namely duodenal acidity. Patients with chronic pancreatitis very often present pancreatic exocrine insufficiency combined with a persistently low duodenal pH in the postprandial period. The duodenal mucosa in chronic pancreas patients with pancreatic insufficiency has a normal concentration of s-cells and, therefore, the production of secretin is preserved. Pancreatic ductal cells are largely responsible for the amount of bicarbonate and water secretion in response to secretin stimulation. When gastric acid in the duodenum is not well-balanced by alkaline pancreatic secretions, it may induce a prolonged secretin stimulus which interacts with the pancreatic ductal cells resulting in an increased rate of ductular cell activity and turnover. N-Nitroso compounds from tobacco, identified in human pancreatic juice and known to be important carcinogens, may then act on these active cells, thereby increasing the risk of cancer. Duodenal acidity is probably of particular concern in patients who have undergone a duodenum-preserving pancreatic head resection, since, in this anatomic situation, pancreatic juice transits directly via the jejunal loop, bypassing the duodenum. Patients undergoing a Whipple procedure or side-to-side pancreaticojejunostomy are probably less critically affected because secretions transit, at least in part, via the papilla. If the duodenal acidity hypothesis proves correct, then, in addition to stopping smoking, reduction of duodenal acid load in patients with pancreatic insufficiency may help decrease the risk of pancreatic cancer.

Published
2005

70. Carcinoma of the papilla of Vater

Author

Serio, Giovanni, Iacono, C., Zamboni, Giuseppe, Scarpa, Aldo, Talamini, G., Facci, E., and Mangiante, Gerardo

Subjects
"Carcinoma", "pathology", "papilla of Vater"
Published
1994

80. Study of pancreatic outnow by US-secretin Test (US-S) and MRColangiopancreatography (MRCP-S) with secretin stimulation: results of a comparison in 20 patients with acute recurrent and chronic obstructive pancreatitis

Author

Liani, C., primary, Bovo, P., additional, Tinazzi, P., additional, Fantin, A., additional, Turrini, R., additional, Talamini, G., additional, Vaona, B., additional, Frulloni, L., additional, Papò, M., additional, Carrara, S., additional, Procacci, C., additional, and Cavalllini, C., additional

Published
2001
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94. Chronic Obstructive Pancreatitis in Humans Is a Lithiasic Disease

Author

Cavallini, G., primary, Bovo, P., additional, Vaona, B., additional, Di Francesco, V., additional, Frulloni, L., additional, Rigo, L., additional, Brunori, M. P., additional, Andreaus, M. C., additional, Tebaldi, M., additional, Sgarbi, D., additional, Angelini, G., additional, Talamini, G., additional, Procacci, C., additional, Pederzoli, P., additional, and Filippini, M., additional

Published
1996
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95. Cigarette Smoking

Author

Talamini, G., primary, Bassi, C., additional, Falconi, M., additional, Frulloni, L., additional, Di Francesco, V., additional, Vaona, B., additional, Bovo, P., additional, Rigo, L., additional, Castagnini, A., additional, Angelini, G., additional, Vantini, I., additional, Pederzoli, P., additional, and Cavallini, G., additional

Published
1996
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96. Prevention of Sepsis in Acute Pancreatitis

Author

Bassi, C., primary, Falconi, M., additional, Bonora, A., additional, Salvia, R., additional, Caldiron, E., additional, De Santis, L., additional, Sartori, N., additional, Butturini, G., additional, Talamini, G., additional, and Pederzoli, P., additional

Published
1996
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98. Effect of Alcohol and Smoking on Pancreatic Lithogenesis in the Course of Chronic Pancreatitis

Author

Cavallini, G., primary, Talamini, G., additional, Vaona, B., additional, Bovo, P., additional, Filippini, M., additional, Rigo, L., additional, Angelini, G., additional, Vantini, I., additional, Riela, A., additional, Frulloni, L., additional, Di Francesco, V., additional, Brunori, M. P., additional, Bassi, C., additional, and Pederzoli, P., additional

Published
1994
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