470 results on '"Shin, Monica"'
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52. IC‐P‐054: AMYLOID AND MICROGLIAL ACTIVATION SYNERGY LEADS TO HYPOMETABOLISM IN THE AD BRAIN: MICROPET LONGITUDINAL STUDY.
53. IC‐P‐022: LATERAL TEMPORAL AMYLOID LOAD PREDICTS PROGRESSION TO ALZHEIMER'S DEMENTIA.
54. IC‐04‐05: IMAGING EPIGENETICS IN THE HUMAN BRAIN WITH THE NOVEL [11C]MARTINOSTAT PET IN PRECLINICAL AD, MCI, AD, AND FRONTOTEMPORAL DEMENTIA INDIVIDUALS.
55. Multimodal Imaging in Rat Model Recapitulates Alzheimer's Disease Biomarkers Abnormalities
56. [P3-425]: ELEVATED CSF LEVELS OF NEUROFILAMENT LIGHT CHAIN IS ASSOCIATED WITH GRAY MATTER NEURODEGENERATION IN BOTH HUMANS AND TRANSGENIC RAT MODEL OF ALZHEIMER's DISEASE
57. IN VIVO AND IN VITRO DEMONSTRATION OF [18F]THK5351 BINDING TO MONOAMINE OXIDASE–B IN THE HUMAN BRAIN
58. [IC-P-148]: LACK OF SELF-AWARENESS OF COGNITIVE DEFICITS PREDICTS METABOLIC DECLINE IN MILD COGNITIVE IMPAIRMENT
59. [P4-239]: FDG-PET POWER TO PREDICT MEMORY DECLINE IN ALZHEIMER's DISEASE DEPENDS ON DISEASE PHASE AND AMYLOID AND TAU STATUS
60. COMPARISON BETWEEN MONOAMINE OXIDASE B INHIBITION ON THE UPTAKE OF [18F]THK5351
61. [P2-413]: VOXEL-BASED POWER CALCULATION AND THRESHOLDS PREDICTIVE OF IMMINENT NEURODEGENERATION FOR PRECLINICAL ALZHEIMER's DISEASE CLINICAL TRIALS
62. [IC-P-017]: VOXEL-WISE DETERMINATION OF SENSITIVITY, SPECIFICITY, AND THRESHOLDS FOR AMYLOID POSITIVITY USING [18 F]FLORBETAPIR PET
63. [P3-321]: IN VIVO AND IN VITRO DEMONSTRATION OF [18 F]THK5351 BINDING TO MONOAMINE OXIDASE-B IN THE HUMAN BRAIN
64. FUNCTIONAL REGRESSION MODEL UNVEILS THAT GENE INTERACTION BETWEEN NCSTN (AMYLOID METABOLISM) AND MS4A4E (NEUROINFLAMMATION) DETERMINES AMYLOID LOAD SPECIFICALLY ON THE DMN
65. AMYLOID-BETA MODULATES CEREBRAL METABOLIC NETWORK IN RATS AND HUMANS
66. THE “A/T/N” SYSTEM: ADDED PREDICTIVE VALUE OF N BIOMARKERS OF PROGRESSION FROM MCI TO DEMENTIA OVER 2 AND 4 YEARS
67. [P4-050]: GRAPH-THEORY ANALYSIS SHOWS A HIGHLY EFFICIENT BUT REDUNDANT NETWORK IN MCI TAU PROPAGATION
68. [IC-P-149]: LACK OF SELF-AWARENESS OF COGNITIVE DEFICITS IN ALZHEIMER's DISEASE IS RELATED TO DECREASED METABOLISM IN THE POSTERIOR CINGULATE CORTEX
69. [IC-P-048]: ELEVATED CSF LEVELS OF NEUROFILAMENT LIGHT CHAIN IS ASSOCIATED WITH GRAY MATTER NEURODEGENERATION IN BOTH HUMANS AND TRANSGENIC RAT MODEL OF ALZHEIMER's DISEASE
70. PRECLINICAL ASSESSMENT OF KAL-ABP TARGET ENGAGEMENT AND EFFICACY USING PET, MRI AND CSF BIOMARKERS
71. [P3-412]: PET TAU AND AMYLOID SYNERGISM WITHIN THE DEFAULT MODE NETWORK DETERMINES THE CLINICAL STATUS IN THE PREDEMENTIA PHASE OF ALZHEIMER's DISEASE
72. [IC-P-045]: AMYLOID-BETA MODULATES CEREBRAL METABOLIC NETWORK IN RATS AND HUMANS
73. [P2-476]: LACK OF SELF-AWARENESS OF COGNITIVE DEFICITS IN ALZHEIMER's DISEASE IS RELATED TO DECREASED METABOLISM IN THE POSTERIOR CINGULATE CORTEX
74. [P1-368]: LACK OF SELF-AWARENESS OF COGNITIVE DEFICITS PREDICTS DEFAULT MODE NETWORK METABOLIC DECLINE IN MILD COGNITIVE IMPAIRMENT
75. [P1-452]: COMBINED GLOBAL AND REGIONAL AMYLOID EFFECT ON THE DEFAULT MODE NETWORK LEADS TO COGNITIVE DECLINE
76. [IC-P-034]: GRAPH-THEORY ANALYSIS SHOWS A HIGHLY EFFICIENT BUT REDUNDANT NETWORK IN MCI TAU PROPAGATION
77. Monoamine oxidase B inhibitor, selegiline, reduces 18F-THK5351 uptake in the human brain
78. Regional Amyloid-β Load and White Matter Abnormalities Contribute to Hypometabolism in Alzheimer's Dementia.
79. ISSUES REGARDING [18F]MK6240 REFERENCE REGION SELECTION BASED ON THE FULL KINETIC MODELING
80. White matter abnormalities and structural parietal disconnections in Alzheimer's disease
81. Neurodegeneration and cortical atrophy in [18F]florbetapir accumulators and non-accumulators:Alzheimer's & Dementia: The Journal of the Alzheimer's Association
82. Regional association maps describe dynamic architecture of amyloidosis and hypometabolism in Alzheimer’s disease:Alzheimer's & Dementia: The Journal of the Alzheimer's Association
83. Hypometabolism and cortical atrophy in [18F]florbetapir accumulators and non-accumulators:Alzheimer's & Dementia: The Journal of the Alzheimer's Association
84. White Matter Abnormalities and Structural Hippocampal Disconnections in Amnestic Mild Cognitive Impairment and Alzheimer’s Disease
85. P1-282: Voxel-Wise Logistic Regression Improves Prediction Accuracy for Developing Alzheimer's Disease
86. IC‐P‐029: Polymorphism in Cytochrome P450 Gene is Associated with Alzheimer’s Pathology
87. P4-198: Cognitive Decline is Driven By the Regional Synergism Between Amyloid Load and Hypometabolism in Mild Cognitive Impairment
88. IC-P-101: Synergism Between Baseline Amyloidosis and Neuronal Injury as Determinants of Learning Deficits in AD Transgenic Rat Model
89. IC‐P‐027: Amyloid‐Induced Microglial Activity in Thalamocortical Circuits Predicts Subsequent Cognitive Decline
90. P2-053: Amyloid-Induced Microglial Activity in Thalamocortical Circuits Predicts Subsequent Cognitive Decline
91. IC-P-088: Cognitive Decline is Driven By The Regional Synergism Between Amyloid Load and Hypometabolism in Mild Cognitive Impairment
92. IC-P-099: Synergism Between Brain Amyloid Accumulation and Neuronal Injury in Cortical-Subcortical Circuits Causes Memory Declines in Animal Models
93. IC-P-086: Amyloid-β and Hyperphosphorylated TAU Synergy Drives Clinical Progression to Alzheimer’s Disease
94. IC‐P‐025: [18F]Florbetapir Roc Curve at Every Voxel Revels a Wide Range of Cortical Suvr Cut‐Offs
95. P3-221: Synergism Between Baseline Amyloidosis and Neuronal Injury as Determinants of Learning Deficits in Alzheimer's Disease Transgenic Rat Model
96. P3-251: Novel Toolbox for Performing Voxel-Wise Generalized Linear Regression with Mulitple Volumetric Covariates in Longitudinal Data
97. O4-01-03: AMYLOID-B AND HYPERPHOSPHORYLATED TAU SYNERGY DRIVES CLINICAL PROGRESSION TO ALZHEIMER’S DISEASE
98. P1-101: Amyloid-Beta 1-42 (Aβ1-42 ) Levels in the Cerebrospinal Fluid Associate With Spatial Memory Performance in Aged But Not in Adult Mcgill-R-THY1-APP Rats
99. P1-251: Synergism between Brain Amyloid Accumulation and Neuronal Injury in Cortical-Subcortical Circuits Causes Memory Declines in Animal Models
100. P2‐093: Polymorphism in Cytochrome P450 Gene is Associated with Alzheimer’s Pathology
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