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330 results on '"Sakamoto, G"'

55. Mucinous Cystadenocarcinoma of the Breast.

Author

Honma N, Sakamoto G, Ikenaga M, Kuroiwa K, Younes M, and Takubo K

Abstract

We report a case of mucinous cystadenocarcinoma (MCA) of the breast in a 96-year-old woman. This is an extremely rare variant of primary breast carcinoma that bears a striking resemblance to MCAs of the ovary and pancreas. The macroscopic appearance and secretion pattern (cytologic findings) resembled cystic hypersecretory carcinoma. However, microscopically, the epithelial cells were quite different from those of cystic hypersecretory carcinoma. In the present study as well as in the literature, MCAs tend to occur more frequently in elderly women. Immunohistochemical findings suggest that they may develop independently of estrogenic stimulation. Although MCAs show high proliferative activity, the prognosis was favorable in the present case as well as in the reported cases. Because MCAs appear to have a distinct pathogenesis and biologic behavior, they should be distinguished from ordinary mucinous carcinomas, cystic hypersecretory carcinomas, and carcinomas of other histologic subtypes. [ABSTRACT FROM AUTHOR]

Published
2003
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56. Lack of association between the polyadenylation polymorphism in the NAT1 (acetyltransferase 1) gene and colorectal adenomas.

Author

Probst-Hensch, N M, Haile, R W, Li, D S, Sakamoto, G T, Louie, A D, Lin, B K, Frankl, H D, Lee, E R, and Lin, H J

Abstract

Smoking and a high intake of red meat are risk factors for colorectal tumors. These effects could be due to aromatic amine carcinogens. Individual susceptibility to aromatic amines has been related to acetylation phenotype, which plays a role in the bioactivation of arylamines. Polymorphisms in both N-acetyltransferase genes, NAT1 and NAT2, have been associated with an increased risk of colorectal tumors. We studied the NAT1*10 fast acetylator allele (1088 T-->A mutation) and distal adenomas in a sigmoidoscopy-based case-control study (441 cases, 484 controls). We found neither an increased adenoma prevalence in subjects homozygous or heterozygous for the NAT1*10 fast acetylator allele (odds ratio 1.04; 95% confidence interval 0.79-1.36), nor a gene-gene interaction between NA1 and NAT2 (P(interaction) = 0.59). Further NAT1 alleles must be considered for more conclusive results regarding the relevance of NAT1 activity to colorectal tumorigenesis.

Published
1996
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66. A SiGe MMIC variable gain cascode amplifier.

Author

Chaudhry, Q., Alidio, R., Sakamoto, G., and Cisco, T.

Abstract

A silicon-germanium variable gain cascode amplifier has been developed to combine the functionality of an amplifier and an attenuator into one monolithic microwave integrated circuit (MMIC). The cascode amplifier, which was designed for a 7-11 GHz frequency range, achieved a gain of 12.5 dB, an input return loss of 7.5 dB, and an output return loss of 12.5 dB. The cascode amplifier exhibited 16 dB of gain control. [ABSTRACT FROM PUBLISHER]

Published
2002
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68. Endoscopic and histologic findings of indraductal lesions presenting with nipple discharge.

Author

Makita M, Akiyama F, Gomi N, Iwase T, Kasumi F, and Sakamoto G

Abstract

To improve the utility of mammary ductoscopy, we investigated the correlation between endoscopic findings and histologic findings using intraductal biopsy specimens. Seventy-one intraductal biopsy specimens obtained from 63 patients between October 2001 and March 2004 were analyzed retrospectively. All specimens were obtained from monotonous intraductal lesions immediately after observation by mammary ductoscopy and were composed of a pure histologic subtype. With regard to endoscopic findings, color was classified as yellow, red, white, or colorless, and morphology was classified as spherical, lobular, mulberry, or amorphous. The histologic subtype was classified as papillotubular, papillary, degenerated, papillary cancer, solid-type ductal carcinoma in situ (DCIS), or cribriform cancer. The relationship between histologic diagnosis, color, and morphology was investigated. Intraductal biopsy specimens included 25 specimens of carcinoma and 46 specimens of papilloma. There was no significant correlation between color and diagnosis. Fourteen of 25 carcinoma specimens were amorphous, and amorphous morphology was significantly suggestive of malignancy (p < 0.001). Further, cribriform cancer was associated with amorphous morphology and yellow color. Morphology may be a useful endoscopically delineated parameter for differentiating intraductal lesions. [ABSTRACT FROM AUTHOR]

Published
2006
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69. UV disinfection for reuse applications in North America

Author

Sakamoto, G., Tomowich, D., and Schwartzel, D.

Subjects
*IRRADIATION, *RECLAMATION of land, *WASTE recycling, *VIRUSES, *WATER quality, *WATER supply, *DISINFECTION & disinfectants
Abstract

In an effort to conserve and protect limited water resources, the States of Florida and California have actively promoted wastewater reclamation and have implemented comprehensive regulations covering a range of reuse applications. Florida has a semi-tropical climate with heavy summer rains that are lost due to run off and evaporation. Much of California is arid and suffers periodic droughts, low annual rainfall and depleted ground water supplies. The high population density combined with heavy irrigation demands has depleted ground water supplies resulting in salt-water intrusion. During the past decade, Floridareuse sites have increased dramatically from 118 to 444 plants representing a total flow capacity of 826 MGD. California presently has over 250 plants producing 1 BGD with a projected increase of 160 sites over the next 20 years. To prevent the transmission of waterborne diseases, disinfection of reclaimed water is controlled by stringent regulations. Many states regulate wastewater treatment processes, nutrient removal, final effluent quality and disinfection criteria based upon the specific reuse application. As a rule, the resulting effluentshave low turbidity and suspended solids. For such effluents, UV technology can economically achieve the most stringent disinfection targets that are required by the States of California and Florida for restricted and unrestricted reuse. This paper compares UV disinfection for wastewater reuse sites in California and Florida and discusses the effect of effluent quality on UV disinfection. [ABSTRACT FROM AUTHOR]

Published
2000

71. Search for diphoton events with large missing transverse energy in 7 TeV proton-proton collisions with the ATLAS detector

Author

N. Davidson, M. Uslenghi, Laurent Vacavant, M. Bendel, D. Schouten, J. Cammin, Gideon Bella, P. F. Zema, Wendy Taylor, Giancarlo Spigo, J. Meinhardt, R. Beccherle, Kathleen Whalen, S. Owen, Michel Lefebvre, Marcos Vinicius Silva Oliveira, D. Fassouliotis, Ralf Hertenberger, S. M. Fisher, Paul Laycock, Irakli Minashvili, A. A. Abdelalim, Corrinne Mills, Tapas Sarangi, A. Staude, Danuta Kisielewska, H. J. Burckhart, R. Dankers, Konstantin Toms, Yuri Kulchitsky, Vadim Gratchev, Si Xie, Jacqueline Batley, Manuela Venturi, Xuai Zhuang, Samira Hassani, Nikolai Rusakovich, C. J. Curtis, Gabriele Chiodini, Phillip Allport, Carlo Schiavi, Allan G Clark, Roger Moore, Anna Macchiolo, Kerstin Tackmann, E. Williams, S. Willocq, Marco Fraternali, Thibault Guillemin, C. Da Via, Stefanie Adomeit, S. Mättig, J. Farley, Kevin Varvell, V. Kral, V. A. Gapienko, G. S. Huang, M. Ibbotson, D. Su, J. T. Childers, Evgeniy Khramov, A. Ferretto Parodi, Ole Røhne, Frank Siegert, Judith Katzy, Fred Hartjes, N. D. Brett, S. De Castro, Ian Brock, G. Polesello, Vincent Hedberg, Jan Valenta, D. Traynor, M. Petteni, Yasuyuki Okumura, William Quayle, N. Skvorodnev, Y. B. Pan, O. G. Grebenyuk, Helio Takai, S. Hamilton, Yang Yang, M. Terwort, Massimo Sorbi, J. Garvey, Hans Krüger, William Brooks, M. C. Tamsett, D. Harper, M. Rescigno, V. Chernyatin, M. P. Sanders, Mous Tatarkhanov, Rashid M. Djilkibaev, Hyun-Chul Kim, Bengt Lund-Jensen, Benjamin Kaplan, Jun Wakabayashi, Lidia Smirnova, R. Kowalewski, Yaquan Fang, F. Wicek, Lukasz Zwalinski, Kirill Prokofiev, A. Palma, Reiner Hauser, S. Cuneo, Bruce Gallop, Laurent Chevalier, Tetiana Hryn'ova, Peter Hansen, Hantao Ji, J. D. Dowell, David Berge, N. De Groot, D. Roda Dos Santos, R. E. Hughes-Jones, Hiroshi Sakamoto, J. Petersen, Peter Kodys, Andreas W. W. Ludwig, Charles Taylor, C. Benchouk, M. Karnevskiy, L. Rosselet, M. Cambiaghi, Susumu Oda, A. Pickford, Shuang-Lin Li, O. B. Dogan, B. Sopko, A. I. Kononov, Gordon Watts, Fabienne Ledroit-Guillon, Kyle Cranmer, Claudio Ferretti, Aaron Angerami, J. Lundquist, R. Perrino, Tohru Takeshita, P. J. Clark, Bellisario Esposito, E. Cogneras, Vadim Kantserov, M. Hauschild, N. Boelaert, A. Kocnar, Alejandro Alonso, Mustafa Yilmaz, J. P. Archambault, S. Walch, Giovanni Darbo, K. Nagai, Iskander Ibragimov, Klaus Hamacher, M. D. Ciobotaru, B. Guo, Valerio Dao, Peter Buchholz, G. A. Odino, T. Perez Cavalcanti, Jian Wang, L. Rumyantsev, J. W. Schumacher, Marianna Testa, P. N. Ratoff, B. Auerbach, Tatiana Klioutchnikova, Dilip Jana, R. K. Bock, Stefan Valkar, Rostislav Konoplich, M. Lungwitz, Graeme Stewart, Zdenek Dolezal, J. E. García Navarro, Kerstin Jon-And, L. Xaplanteris, G. Cataldi, M. Sosebee, Jacob Searcy, Massimiliano Bellomo, Borut Paul Kerševan, Arthur Moraes, Barbara Wosiek, D. Joffe, J. Z. Will, Uwe Bratzler, Pierre-Antoine Delsart, S. Vallecorsa, K. Fowler, A. Papadelis, Tancredi Carli, T. Stahl, K. Stevenson, Roberto Cardarelli, Mark Sutton, José Salt, S. Nelson, Abdeslam Hoummada, D. H. Saxon, Laura Jeanty, T. Buran, Markus Elsing, Lee Sawyer, Sally Seidel, Peter Onyisi, Mattias Ellert, M. Losada, Filippo Maria Giorgi, P. M. Tuts, T. Ohsugi, K. H. Hiller, K. Mahboubi, J. Silva, Kiyotomo Kawagoe, Jean-Francois Arguin, D. Urbaniec, A. Vogel, Veysi Erkcan Ozcan, Andreas Petridis, Gianluca Alimonti, Rainer Stamen, T. B. Sjursen, G. Hughes, Urmila Soldevila, Alberto Mengarelli, P. D. Thompson, C. Carpentieri, S. Swedish, M. S. Kayl, M. Kurata, M. Schmitz, Patrick Fassnacht, I. Ludwig, Andrew Pilkington, Christos Anastopoulos, Bianca Osculati, Bobby Samir Acharya, H. A. Neal, P. Ruzicka, C. Cowden, J. Hoffman, M. T. Pérez García-Estañ, G. Bachy, Guillaume Unal, J. T. Linnemann, Andreas Hoecker, A. N. Sisakyan, A. Artamonov, A. V. Larionov, R. C. W. Henderson, M. J. Shochet, Alexandre Rozanov, Edmund Dawe, Claudio Santoni, Y. A. Tikhonov, David Yu, Evgeny Starchenko, M. Mechtel, C. Padilla Aranda, Kate Shaw, Juerg Beringer, A. Jantsch, Mathieu Benoit, K. Bernardet, Nicola Venturi, Yuji Yamazaki, H. Ghazlane, Guosheng Xu, G. Cattani, A. Astbury, C. Cerna, G. Kilvington, L. Micu, Stefan Schlenker, Liron Barak, Alexander Undrus, Tatsuya Masubuchi, E. Rulikowska-Zarebska, Oliver Keith Baker, John Alison, Sara Valentinetti, Frank Winklmeier, L. R. Flores Castillo, S. D. Kolya, Luc Poggioli, Christine Kourkoumelis, Pedro Jorge, Gvantsa Mchedlidze, Sarah Heim, E. van der Poel, Uladzimir Kruchonak, Mauro Iodice, Gideon Alexander, B. Rensch, Cristian Stanescu, P. J. Bell, S. V. Mouraviev, P. Branchini, D. Lopez Mateos, Harald Joerg Stelzer, Massimo Antonelli, Maia Mosidze, J. A. Strong, G. Gorfine, Pavel Shatalov, Jochen Dingfelder, J. R. Keates, Alex Kastanas, R. Froeschl, Alaettin Serhan Mete, B. G. Pope, Robert P. Ely, S. A. Gorokhov, T. Byatt, A. D. Doxiadis, M. Dwuznik, Jolanta Olszowska, Lorne Levinson, K. Yamamoto, Hui Li, J. L. Schlereth, A. Christov, Sigve Haug, N. Austin, Ovsat Abdinov, L. E. Kirsch, M. Perantoni, A. G. Schamov, J. Odier, G. Usai, Joe Kroll, Daniel Lellouch, K. K. Joo, A. Leger, S. Heisterkamp, A. Engl, A. Roe, David Calvet, Mitsuaki Nozaki, M. Tyndel, E. Berglund, Davide Costanzo, M. G. Wilson, J-P. Meyer, A. Guida, G. Carlino, Frank Fiedler, Giovanni Maccarrone, Vaclav Vrba, Francois Touchard, M. I. Pedraza Morales, W. J. Mills, J. Dubbert, Jean-Francois Grivaz, A. Manabe, Janet Dietrich, Martin Hoeferkamp, V. Giangiobbe, Jiri Dolejsi, Alastair Dewhurst, Ulrich Husemann, Heinz Pernegger, Alexander Oh, A. Manousakis-Katsikakis, J. Lesser, N. Grigalashvili, D. Gillberg, S. Mitsui, Rupert Leitner, Leonardo Paolo Rossi, Z. Qian, John H. L. Hansen, P. de Jong, Claudio Luci, S. De Cecco, Trevor Vickey, G. Schuler, Phillip Gutierrez, Y. Rodriguez Garcia, R. Yoshida, Dimitrios Iliadis, A. Taga, Jun Guo, T. J. Brodbeck, Kirika Uchida, Hisaya Kurashige, M. King, Joern Grosse-Knetter, Sergio Grancagnolo, C. Borer, Sabine Crépé-Renaudin, Osamu Sasaki, Carlos Solans, S. George, Miriam Watson, Milos Lokajicek, P. Waller, Steven Ahlen, C. Santamarina Rios, D.P.S. Ferguson, K.M. Smith, F. Mueller, Thorsten Kuhl, Nikolaos Konstantinidis, R. Sandstroem, João Carvalho, S. Lockwitz, C. Bacci, Shuwei Ye, Jennifer A. Love, Alberto Aloisio, A. Nikiforov, Y. Morita, Oleg Brandt, J. Hartert, Mara Senghi Soares, R. J. Miller, B. C. LeGeyt, E. Reinherz-Aronis, Sh. Yamamoto, J. Cook, Dominik Dannheim, Cesare Bini, Zhen Yan, K. Skovpen, Rodney Walker, C. Tsarouchas, S. Gadomski, M. Ugland, T. C. Meyer, A. T. H. Arce, Jamal Eddine Derkaoui, Arno Straessner, C. U. Felzmann, F. Butin, Sébastien Viret, B. Chapleau, Denis Salihagic, B. Jackson, Markus Cristinziani, Wolfgang Liebig, Lourenco Lopes, M. Shimojima, Michael Andrew Parker, Susanne Kuehn, Xifeng Ruan, Johannes Haller, Efstathios Paganis, Maurizio Piccinini, Neil Collins, Edward Diehl, L. Batkova, I. Gough Eschrich, S. Tanaka, M. Corradi, L. Zhao, Monica D'Onofrio, I. Nasteva, Driss Benchekroun, Jos Vermeulen, D. Breton, Konstantinos Nikolopoulos, A. Bruni, Teng Jian Khoo, Kunihiro Nagano, L. Moneta, S. Eckweiler, B. Budick, I. Wilhelm, M. De Oliveira Branco, N. I. Bozhko, D. V. Dedovich, Elizaveta Shabalina, J. Ferland, Andrea Sansoni, J. Poll, G. Ordonez, S. Baker, L. M. Gilbert, G. T. Jones, M. Cirilli, Alison Lister, J. A. Gray, Rinat Fakhrutdinov, E. Spiriti, Stefania Spagnolo, R. Porter, Martine Bosman, H. Nomoto, E. Hazen, Sergio Gonzalez-Sevilla, M. Mikuž, Y. Zolnierowski, E. J. Buis, M. Aurousseau, M. Groh, Lucio Cerrito, Kerstin Lantzsch, Jochen Schieck, C. Belanger-Champagne, T. Del Prete, John Rutherfoord, Massimo Caccia, Holger Schulz, A. Phan, Filippo Ceradini, Krzysztof Korcyl, S. Campana, Martin Maß, S. T. French, C. A. Magrath, R. Schwierz, J. Knobloch, V. M. Romanov, Andrea Catinaccio, Rachel Maria Avramidou, Cigdem Issever, Song-Ming Wang, A. Leung Fook Cheong, Jana Schaarschmidt, I. A. Christidi, N. Ghodbane, D. Kuhn, D. Evangelakou, M. J. Oreglia, H. Wahlen, Stan Bentvelsen, T. Vu Anh, G. Volpini, Stefan Kaiser, Shigeru Odaka, H. von Radziewski, Daniel Muenstermann, I. Massa, A. J. Martin, C. Vellidis, Christophe Clement, J. Weber, Vipul Jain, Dominique Pallin, Rosy Nicolaidou, C. Pizio, Timothy Andeen, C. Grah, Z. Zhan, Jianming Qian, G. Tong, A. Borisov, Vincenzo Canale, W. Iwanski, A. G. Myagkov, L. Duflot, K. Jelen, E. Petit, Andrey Soukharev, A. Hidvegi, B. Nicquevert, A. Bertin, M. Tylmad, Calin Alexa, C. Wright, J. A. Bogaerts, T. N. Addy, Zhijun Liang, F. Bertinelli, Tomas Sykora, Jyothsna Rani Komaragiri, Mark Slater, Valery Schegelsky, F. Garberson, K. E. Johansson, Mark Stockton, Yoichi Ikegami, L. Chikovani, Pavol Strizenec, V. Sopko, V. A. Korotkov, Siegfried Wenig, T. J. Sloan, I. Torchiani, Jovan Mitrevski, Murrough Landon, Peter Jenni, Q. T. Shao, Hongfang Liu, S. Aoun, P. Giusti, J. Sloper, Paul Tipton, Margherita Primavera, A. Do Valle Wemans, M. Kelly, M. Strang, Olivier Arnaez, T. Akdogan, Elliot Lipeles, Saleh Sultansoy, C. Daum, R. Tanaka, T. Göpfert, I. Turk Cakir, Andreas Warburton, M. Ramstedt, M. C. Morone, Kazunori Hanagaki, G. Nanava, M. E. Zeller, E. Sedykh, G. Calderini, Carmen García, J. Lys, F. Föhlisch, Allen Mincer, M. V. Purohit, Uli Schäfer, Dirk L. Hoffmann, Junji Tojo, Ehud Duchovni, A. G. Kholodenko, S. J. Park, Paolo Camarri, Stefan Tapprogge, Mario Lassnig, A. Maio, C. Conta, Ralf P. Richter, Christopher J. Kenney, B. M. Waugh, F. Sarri, A. S. Kozhin, David Francis, Marcello Fanti, J. Colas, A. J. Lowe, A. Boveia, Ki Lie, T. Spreitzer, K. Miyazaki, J. Machado Miguens, Fumihiko Ukegawa, Andrea Formica, Venetios Polychronakos, V. Perez Reale, Yona Oren, R. A. McLaren, Eram Rizvi, L. La Rotonda, I. Hruska, M. Villaplana Perez, R. Herrberg, C. P. Buszello, Stephanie Majewski, Daniel Fournier, P. Yu Nechaeva, Xiaohu Sun, I. Stekl, P. Steinbach, G. M. Kolachev, Marzio Nessi, E. Jankowski, Martin Rybar, A. Seiden, T. Hayakawa, James Catmore, Robert Kehoe, W. E. Cleland, Renat Sadykov, Rudiger Voss, Georges Azuelos, Nick Barlow, Barry King, F. Alessandria, Helena Santos, Ricardo Neves, Fred R. Hirsch, Bertrand Laforge, K. H. Becks, G. Luijckx, David J. Smith, A. Filippas, O. Gutzwiller, M. Della Pietra, S. Gonzalez, Marco Schioppa, I. Jen-La Plante, G. Tzanakos, Andrea Bocci, M. Cooke, B. Sellden, K. K. Temming, Y. F. Ryabov, Nikola Makovec, Dave Charlton, Xiaoqin Li, Hulin Wang, A. Ahmad, Corinne Goy, C. Lapoire, Diane Cinca, Reiner Klingenberg, Helmut Wolters, Vladimir Nikolaenko, Z. Weng, A. Penson, Ian Michael Nugent, Muhammad Alhroob, P. Conde Muiño, Evelina Vassileva Bouhova-Thacker, Eugen Ertel, Georgios Tsipolitis, Sven Menke, A. Lupi, Mogens Dam, Victor Maleev, Karl Jakobs, Xie Yijin, Peter Schacht, Mauro Donegà, M. Jimenez Belenguer, M. Turala, O. Kvasnicka, H. Czirr, Frank Paige, Else Lytken, Charles Leggett, M. Rammes, Fabian Huegging, C. M. Hawkes, A. M. Zaitsev, F. Broggi, T. Mclaughlan, M. Mazzanti, E. De La Cruz-Burelo, J. R.A. Booth, D. Fasching, C. Ruwiedel, Thorsten Wengler, O. C. Jonsson, V. Savinov, Murdock Gilchriese, I. Satsounkevitch, F. Quinonez, Andrea Ventura, Michael Solar, A. Bangert, B. Åsman, M. Klemetti, Oldrich Kepka, F. Cataneo, A. Richards, Ulrich Parzefall, M. Ziolkowski, Alexander Bogdanchikov, Amanda R. Vest, Speranza Falciano, V. Kaushik, T. Meguro, Andreas Redelbach, Farid Ould-Saada, L. De Mora, B. Gosdzik, K. Facius, A. Di Ciaccio, C. Driouichi, Michael A. Strauss, M. Johansen, Voica Radescu, Driss Goujdami, C. Suhr, J. A. Parsons, Stefan Koperny, Julie Kirk, Susanne Kersten, Sebastian Grinstein, R. Ichimiya, Wolfgang Mader, Kendall Reeves, H. Landsman, C. Topfel, B. De Lotto, Emmerich Kneringer, D. van der Ster, Donatella Cavalli, U. Mallik, Mikhail Mineev, John Penwell, P. Hansson, Heiko Lacker, A. Kreisel, J. A. Ernst, F. Etienne, R. Mameghani, G. Lenzen, Dave Britton, Chiara Roda, Børge Kile Gjelsten, A. J. Fowler, James Boyd, D. Romero Maltrana, X. S. Anduaga, A. V. Ivashin, P. Miele, Dimitris Varouchas, Manolis Dris, G. Mahout, Shogo Okada, C. Guicheney, H. J. Yang, Juergen Kroseberg, A. T. Doyle, L. Labarga, B. Gaur, Dominick Olivito, Karel Smolek, Werner Wiedenmann, J. M. Foster, E. Castaneda-Miranda, A. G. Olchevski, Markus Schumacher, Frederic Brochu, Ryszard Stroynowski, T. Doherty, J. R. Lessard, Paul Nilsson, S. Prasad, D. Pomeroy, Alberto Annovi, M. Imhaeuser, Armin Michael Nairz, A. Reinsch, D. Whittington, A. A. Carter, Roger Jones, A. Firan, E. Bergeaas Kuutmann, Are Strandlie, Andrew Brandt, Fernando Marroquim, Frank Seifert, R. Engelmann, M. Wittgen, Ariel Schwartzman, S. Fratina, J. M. Maugain, A. Loginov, Marumi Kado, E. Urkovsky, James Frost, Pier-Olivier Deviveiros, Jonas Strandberg, M. Uhrmacher, A. Beddall, N. Giokaris, Giora Mikenberg, Wouter Verkerke, C. J.W.P. Timmermans, Lixin Ma, D. Typaldos, A. D'Orazio, H. von der Schmitt, E. Valladolid Gallego, X. Espinal Curull, Diego Casadei, Doris Chromek-Burckhart, Tadaaki Isobe, J. N. Lilley, M. Cascella, W. Ji, Tatsumi Koi, Maurice Garcia-Sciveres, V. V. Zmouchko, P. Perus, T. Kobayashi, Petr Andreevich Gorbounov, Torre Wenaus, D. Milstein, Dominic Hirschbuehl, S. Sandvoss, P. Federic, G. A. Chelkov, Alex Martyniuk, Wolfgang Wagner, A. Chilingarov, Marie-Hélène Genest, S. Subramania, O. Vitells, Carsten Hensel, M. Dameri, Luca Fiorini, G. Y. Mitrofanov, Alberto Belloni, M. Leahu, Aldo Saavedra, Atsuhiko Ochi, F. Polci, M. Maaßen, S. Di Luise, James Howarth, Stephen Haywood, Sotirios Vlachos, Walter Lampl, V. Moeller, S. Zenz, J. Inigo-Golfin, David Malon, Stefan Guindon, D. Lumb, B. M. Hawes, E. Dobson, E. Sarkisyan-Grinbaum, Ewa Stanecka, Enrico Tassi, F. Marzano, G. Nunes Hanninger, Jean François Marchand, Jens Dopke, Gokhan Unel, F. Dudziak, Richard Brenner, Marisa Sandhoff, A. Mitra, B. Radics, S. Y. Nesterov, Arely Cortes-Gonzalez, Alexander Milov, A. Irles Quiles, Matthias Klein, Alexey Talyshev, R. Giordano, Vladimir Cindro, Pierre-Hugues Beauchemin, M. Weber, A. Reichold, Konrad Kleinknecht, R. Maenner, Teresa Barillari, V. M. Kotov, Seema Bahinipati, Y. Ilchenko, G. Romeo, Marine Kuna, M. Kenyon, Sara Diglio, Steven Goldfarb, Z. G. Zhao, G. Ciapetti, Peter Loch, W. Mohr, J. B. De Vivie De Regie, Jan Kretzschmar, D. R. Quarrie, E. E. Schmidt, F. Parodi, D. Fellmann, C. C. Young, Anna Kaczmarska, N. Grau, Marina Cobal, A. Tonazzo, David Miller, Jiri Masik, Lawrence Lee, Oleg Fedin, S. Vahsen, W. T. Meyer, Dorothee Schaile, Michael Boehler, Bartosz Mindur, Lianyou Shan, Richard Nisius, Melissa Ridel, C. W. Loh, Jianyong Lu, Srinivasan Rajagopalan, Irene Vichou, Hal Evans, B. A. Dolgoshein, Y. Mahalalel, J. Snow, Ilija Vukotic, Jan Godlewski, M. Rijpstra, Jalal Abdallah, E. Strauss, G. Vegni, C. Schmitt, Erez Etzion, M. Werner, Francois Corriveau, Scott Snyder, Carlos Lacasta, Masaya Ishino, E. Turlay, Elias Coniavitis, K. Mueller, Victor Kukhtin, D. Ludwig, P. Ryan, G. Vandoni, J. Boudreau, P. S. Wells, M. A. Dufour, A. M. Castaneda Hernandez, Chong-Yu Xu, C. Melachrinos, J. Hrivnac, H. Guler, Vladimir Vinogradov, M. A. Wildt, B. Toczek, Gabriel Stoicea, N. Kundu, John Baines, Sergei Chekanov, J. Godfrey, A. Blondel, Pavel Staroba, E. Perez Codina, Giuseppe Costa, Max Baak, K. J. Grahn, Claudio Gatti, Stefan Kluth, J. Fleckner, A. Khomich, M. A. Pleier, Eleni Mountricha, Didier Lacour, Ioannis Nomidis, P. S. Mangeard, Oleg Bulekov, S. Viel, C. Deluca, D. Dzahini, Yueh-Feng Liu, S. Schuh, M. Kaneda, S. Rzaeva, Christoph Amelung, F. G. Oakham, S. Fleischmann, Oxana Smirnova, T. Todorov, T. J. McMahon, Danilo Giugni, V. Rojo, D. S. Damiani, Otmar Biebel, Konstantinos Bachas, F. Varela Rodriguez, Francesco Lanni, O. Gaumer, Aaron James Armbruster, V. Ryadovikov, S. Koenig, R. P. Thun, Giuseppe Iacobucci, Thomas Koffas, Frank Ellinghaus, Arnaud Foussat, R. Mehdiyev, T. Rador, Isabel Marian Trigger, Louise Skinnari, R. J. Homer, Antonio Baroncelli, Ulrich Landgraf, J. Schultes, Mauro Villa, N. Panikashvili, Claire Gwenlan, Vincent Garonne, M. Hohlfeld, X. Portell Bueso, Sandro Palestini, J. Snuverink, R. R. Rios, S. Rodier, I. Dolenc, J. Lellouch, D. Y. Bardin, L. Miralles Verge, E. Auge, R. W. Clifft, G. Brown, Andre Kruth, Julien Donini, Pascal Pralavorio, Ilias Efthymiopoulos, Sandra Kortner, S. Fazio, V. Vorwerk, Vadim Kostyukhin, Lamberto Luminari, M. Shamim, Y. Pylypchenko, Catrin Bernius, Remi Lafaye, Theodota Lagouri, G. Cortiana, Paolo Iengo, M. Marx, S. I. Buda, Hartmut Sadrozinski, K. W. McFarlane, N. Massol, Douglas M. Hawkins, D. S. Popovic, E. L. Barberio, Marco Delmastro, M. Fiascaris, Marina Rotaru, N. Dressnandt, Frédéric Chevallier, H. van der Graaf, S. N. White, Ilya Korolkov, Fridolin Dittus, Y. Munwes, Moritz Backes, Sofia Chouridou, Jason Nielsen, Armen Vartapetian, Maria-Teresa Dova, P. Giovannini, E. Ptacek, Tom McCarthy, M. 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Ince, Naoko Kanaya, Alexandre Vaniachine, F. Cevenini, P. Booth, G. F. Gieraltowski, S. Winkelmann, V. I. Vassilakopoulos, J. E. Brau, E. Brubaker, M. Leltchouk, Christopher Bee, Petr Vokac, Ning Zhou, A. Harvey, V. Bansal, Michael Düren, Tobias Golling, Nicolas Berger, Jiri Chudoba, R. Mackeprang, T. Kohriki, M. Medinnis, J. Virzi, S. Eckert, Raymond Veness, M. J. Kotamäki, Eiliv Lund, Helen Hayward, P. M. Watkins, G. W. Brandenburg, Roberto Spighi, I. Aracena, S. Haas, S. Ask, M. S. Alam, Giuseppe Francesco Tartarelli, R. Santonico, Thomas Lohse, S. König, Liza Mijović, S. H. Robertson, I. Fedorko, S. Behar Harpaz, Dan Pantea, A. Gonidec, J. G. Rocha de Lima, R. Davidson, E. Torrence, M. Leyton, A. Gupta, M. D. Joergensen, Alexander Kupco, Edoardo Gorini, M. Kataoka, M H. Lehto, S. W. Snow, T. K.O. Doan, S. Correard, D. Côté, Aston Komar, R. Duxfield, Y. Silver, B. J. O’Brien, Y. V. Grishkevich, John Stupak, T. Horazdovsky, J. Antos, M. S. Kim, Louis Fayard, Z. Zenonos, Sylvain Tisserant, Wolfgang Walkowiak, I. Rottländer, P. de Saintignon, Metin Arik, G. Chiefari, H. Matsumoto, C. P. Ward, D. della Volpe, Fernando Monticelli, I. Stumer, Peter Krieger, Michel Vetterli, D. Buira-Clark, B. Ruckert, J. de Graat, Heidi Sandaker, A. Ishikawa, Sten Hellman, R. Cherkaoui El Moursli, M. Beimforde, W. Kuykendall, L. Shaver, G. Mirabelli, J. Thadome, Henso Abreu, Matt Dobbs, C. L. Lampen, Norbert Wermes, D. Merkl, R. E. Long, B. Clement, Q. Ouyang, Marjorie Shapiro, Soshi Tsuno, T. Kawamoto, Sara Strandberg, C. Valderanis, R. Piegaia, B. Caron, Stephen Maxfield, T. N. Trinh, C. J. Oram, Cedric Serfon, Stephen Gibson, H. Zhang, Phillip Urquijo, V. I. Kolesnikov, S. E. Allwood-Spiers, Deepak Kar, Jianqiao Ye, Y. Azuma, Petre Dita, Jialun Ping, Torsten Paul Ake Åkesson, A. Gutierrez, Aleandro Nisati, E. Vinek, V. Martinez Outschoorn, G. Rivoltella, A. S. Cerqueira, Mika Huhtinen, Giorgio Bellomo, T. Ženiš, U. K. Yang, Sascha Mehlhase, C. 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Solc, Noam Hod, A. Dos Anjos, Samuel Calvet, Simonetta Gentile, R. Prabhu, Pavel Tsiareshka, Javier Sánchez, F. Safai Tehrani, K. E. Arms, M. Uhlenbrock, J. Schwindling, Axel König, O. K. Øye, A. Tykhonov, Andrew Mehta, R. Wall, A. La Rosa, I. M. Gregor, Tetsuro Mashimo, V. Paolone, P. Sinervo, C. A. Edwards, M. L. Chu, K. Gellerstedt, Yu M. Sviridov, J. Mechnich, C. H. Wang, S. Haider, Simon Lin, H. Drevermann, J. Kaplon, Igor Aleksandrov, Emily Nurse, M. Biglietti, G. Choudalakis, Z. van Kesteren, H. Ohshita, K. Copic, T. Barber, C. Dallapiccola, J. Ernwein, M. Battistin, Yousuke Kataoka, L. M. Mir, Yorihito Sugaya, Jed Biesiada, L. Mandelli, Josh Mcfayden, Fabrizio Petrucci, Elizabeth Gallas, Jianbei Liu, A. B. Fenyuk, M. Gouighri, I. Reisinger, Ph. Schwemling, Andrei Akimov, T. K. Ohska, E. Galyaev, M. D. M. Capeans Garrido, T. Sandoval, P. Koevesarki, Miroslav Myska, P. Morettini, Raimund Ströhmer, Alessandra Doria, R. Spiwoks, Jean-Christophe Gayde, Miroslav Havranek, V. Simak, James M. Buchanan, Monica Dunford, Ch Geich-Gimbel, Y. Wu, A. R. Gillman, P. Sandhu, J. von Loeben, Federico Bertolucci, G. Sartisohn, S. Yu. Smirnov, Vincenzo Cavasinni, G. Colon, C. Ay, H. Duran Yildiz, R. Coluccia, A. Napier, Tomiyoshi Haruyama, G. Atoian, Sami Kama, O. Norniella Francisco, V. Scharf, D. Silverstein, Andreas Salzburger, J. Maneira, J. Ludwig, Takanori Kono, Francesco Ragusa, J. Proudfoot, P. F. Klok, Vit Vorobel, Zhenwei Yang, Antonio Sidoti, Ian Hinchliffe, S. Ben Ami, Osamu Jinnouchi, K. Runge, S. Kalinin, Stanislav Nemecek, Shih-Chang Lee, D. G. Underwood, M. Werth, F. Lu, Stefano Rosati, E. Hines, Daniel Tovey, Marco Bruschi, Michele Pinamonti, Volker Büscher, M. El Kacimi, Thilo Pauly, M. J. Woudstra, W. S. Lockman, Hideki Okawa, Soumya Mohapatra, M. Marchesotti, Lorenzo Rinaldi, Bostjan Macek, L. Pribyl, F. Koetsveld, Zhaoxia Meng, J. L. Holzbauer, R. St. Denis, M. Heller, C. De La Taille, D. Hauff, Jelena Krstic, B. 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Piccaro, A. A. Nepomuceno, F. Orellana, Rustem Ospanov, Hongbo Zhu, I. V. Gorelov, Carmen Maidantchik, A. Astvatsatourov, Brinick Simmons, Louise Heelan, C. N. P. Gee, Kamil Augsten, M. A. Houlden, A. M. Henriques Correia, A. M. Rahimi, Stefano Giagu, O. Le Dortz, Eduard Kladiva, Andree Robichaud-Veronneau, S. J. Wheeler-Ellis, S. Rolli, Serban Protopopescu, Igor Boyko, A. Vitale, M. Dehchar, S. Ferrag, Francois Vazeille, A. Trivedi, D. Reljic, J. Flammer, Daniel Froidevaux, Z. Kohout, S. Bordoni, M. Fokitis, M. Lewandowska, Gregor Kramberger, D. Short, Varlen Grabski, Peter Steinberg, P. Nevski, Saverio D'Auria, Anatoli Romaniouk, Horst Severini, A. Korn, E. Arik, Oliver Stelzer-Chilton, Mohsen Khakzad, E. Chareyre, L. Magnoni, Sergey Panitkin, Todd Brian Huffman, K. A. Johns, S. Psoroulas, M. Wen, A. Farilla, Pedro Teixeira-Dias, Filipe Veloso, Nenad Vranjes, T. Bain, J. C.L. Tseng, Ping-Kun Teng, A. Pinder, A. Onofre, Jan Stark, N. Bousson, M. L. Ferrer, James Mueller, Junichi Tanaka, Peter Bussey, T. Gadfort, G. P. Kirsch, S. Böser, Malik Aliev, C. Kummer, Susumu Terada, Vladimir Smakhtin, Andreas Kugel, Craig Wiglesworth, Andy Haas, F. Vives Vaque, B. Di Girolamo, Luis March, E. Mazzoni, M. Nash, Olga Igonkina, Mariagrazia Alviggi, A. Gibson, X. R. Chen, S. Brunet, Teresa Lenz, C. Joram, H. Przysiezniak, Henry Lubatti, Raquel Pezoa, Yasuhiro Homma, Andreas Wildauer, V. M. Malyshev, Zach Marshall, Philipp Fleischmann, Alden Stradling, Ph Martin, Tobias Flick, R. Coura Torres, H. Nilsen, Y. Unno, P. Adragna, James Walder, T. Dubbs, Vadim Bednyakov, M. Yamada, O. Runolfsson, Ryuichi Takashima, James Jackson, Dong Liu, Sune Jakobsen, B. Zilka, Jessica Metcalfe, Johann Collot, Alan Watson, R. Asfandiyarov, Silvia Resconi, Tim Adye, Genevieve Steele, A. R. Weidberg, N. Tannoury, P. A. Bruckman de Renstrom, J. M. Tuggle, F. Rauscher, P. Skubic, Gabriel Alexandru Popeneciu, E. Nebot, C. Ketterer, J. Purdham, Reinhard Schwienhorst, M. Zdrazil, Val O'Shea, D. Chakraborty, Bjørn Hallvard Samset, R. D. Kass, Dominik Derendarz, A. E. Nuncio-Quiroz, P. C. Kim, J. Deng, Zdenek Hubacek, C. Mora Herrera, L. A.M. Wiik, Marcello Bindi, F. Gianotti, Marcel Vos, Y. Bai, R. Bartoldus, P. Hurst, D. Eriksson, A. Zaytsev, Ryan Reece, S. Albrand, A. C. Schaffer, Hubert Kroha, O. Gabizon, P. Barrillon, J. Lundberg, T. T. Voss, G. Comune, Richard Mount, George Redlinger, S. Cheng, G. D. Kekelidze, Z. Drásal, Matthew Fisher, J. G. Loken, I. D. Manjavidze, Carsten Meyer, F. F. Martin, Hong Ma, Cheng Guang Zhu, S. L. Cheung, Petr Tas, S. H. Oh, Farkhad Khalil-zada, D. Hayden, B. Van Eijk, Laura Gonella, Alan Poppleton, D. Ventura, Stefano Veneziano, H. O. Ogren, L. Courneyea, Paul Hanke, M. Barisonzi, Hermann Kolanoski, Claus Gössling, Benedetto Gorini, Matthias Schott, M. Pohl, S. Jin, P. Gagnon, Daniel Turecek, P. M. Mockett, Xiaowen Lei, A. B. Lazarev, A. Schreiner, J. W. Dawson, C. Haber, Junichi Kanzaki, P. Jež, Joao Seixas, C. N. Booth, Zhonghua Qin, Anthony Keith Morley, Peter Wagner, F. Ahles, L. Mendoza Navas, Jozsef Toth, Elena Korolkova, R. de Asmundis, Marek Tasevsky, Els Koffeman, Diana Scannicchio, Erich Varnes, Li Yuan, R. K. Daya, David Lynn, C. Maiani, Richard Keeler, A. Lavorato, P. Savva, S. P. Mc Kee, L. E. Price, Laura Perini, Masahiro Ikeno, U. Bitenc, Oleg Zenin, J. Rothberg, Livio Mapelli, Goetz Gaycken, O. Silbert, P. Bernat, Andrea Dell'Acqua, N. Delruelle, Alessandra Forti, Xiangyang Ju, M. Hurwitz, A. Misiejuk, M. L. Gonzalez Silva, N. Hill, Katarina Pajchel, Stavros Maltezos, Vakhtang Kartvelishvili, Tim Jones, Bernd Stelzer, G. Baccaglioni, Horst Oberlack, R. J. Sobie, Gianfranco Morello, Hasko Stenzel, H. Han, Serhat Istin, Giuseppe Mornacchi, Joao Saraiva, Josu Cantero, A. Oyarzun, H. Lee, Luc Goossens, B. R. Mellado Garcia, Stefanos Leontsinis, Daniela Rebuzzi, K. M. Chan, N. B. Sinev, J-Y. 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Greenfield, Michele Livan, Annick Lleres, J. Grognuz, Abbas Kenan Ciftci, Karlheinz Meier, Rui Wang, Josh Moss, Enrique Kajomovitz, C. Handel, J. Almond, J. Nadal, G. Rudolph, M. Schram, Louis Helary, C. Horn, Timothy Barklow, Daria Zieminska, E. Magradze, P. Valente, Andrei Snesarev, J. A. Macana Goia, R. D. Schamberger, C. Lasseur, Ahmimed Ouraou, V. V. Ammosov, Alan Litke, Mieczyslaw Witold Krasny, Yann Coadou, L. K. Gladilin, B. Pinto, Anna Mastroberardino, C. Gong, Bruno Lenzi, T. P. Kokott, Carla Sbarra, Gilbert Poulard, Yuta Takahashi, V. Zutshi, Amanda Cooper-Sarkar, W.J. Willis, S. Schaetzel, Marcin Wladyslaw Wolter, Peter Wienemann, Marek Idzik, D. Rousseau, Nicole Ruckstuhl, O. M. Harris, G. F. Moorhead, Antonio Ereditato, Alexander Grillo, J. E. Sundermann, M. Miñano, Harinder Singh Bawa, Oliver Kortner, C. Zendler, M. Aliyev, Brigitte Vachon, V. Grassi, Peter Sturm, Heather Gray, D. Fortin, O. Pirotte, R. Yoosoofmiya, R. E. Blair, A. Cazzato, A. 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Gollub, S. González de la Hoz, C. Lebel, H. G. Sander, Hans Peter Beck, R. Zimmermann, M. Martinez, J. A. Valls Ferrer, M. L. Kocian, A. V. Pleskach, A. S. Thompson, B. Butler, Thomas Naumann, P. R. Norton, Markus Nordberg, E. Wulf, Dusan Bruncko, Mark Hatch, A. M. Bach, Vakhtang Tsiskaridze, Maria Curatolo, Saminder Dhaliwal, F. Tarrade, G. Akimoto, Riccardo-Maria Bianchi, Paolo Bagnaia, S. Kabana, J. N. Butler, Anyes Taffard, Nuno Anjos, M. Simonyan, A. J. Lankford, D. Prieur, Th. Müller, Joseph Izen, S. Antonelli, John Morris, Antoine Marzin, Juergen Thomas, Nicolas Ellis, Ruslan Mashinistov, Alessandra Ciocio, K. Sliwa, Shinichi Shimizu, L. Han, G. G.G. Massaro, Gen Kawamura, J. Treis, Wim Lavrijsen, Enrico Pasqualucci, V. F. Chepurnov, P. L. Rosendahl, Roberto Ferrari, R. Ishmukhametov, Anna Sfyrla, D. Tardif, Marcella Bona, V. V. Kabachenko, Abdenour Lounis, Ayda Beddall, Nathalie Besson, M. Robinson, Daniel Dobos, A. Cavallari, T. 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Pater, S. Patricelli, T. Pauly, M. Pecsy, M. I. Pedraza Morales, S. V. Peleganchuk, H. Peng, R. Pengo, A. Penson, J. Penwell, M. Perantoni, K. Perez, T. Perez Cavalcanti, E. Perez Codina, M. T. Pérez García-Estañ, V. Perez Reale, I. Peric, L. Perini, H. Pernegger, R. Perrino, P. Perrodo, S. Persembe, P. Perus, V. D. Peshekhonov, O. Peters, B. A. Petersen, J. Petersen, T. C. Petersen, E. Petit, A. Petridis, C. Petridou, E. Petrolo, F. Petrucci, D. Petschull, M. Petteni, R. Pezoa, A. Phan, A. W. Phillips, P. W. Phillips, G. Piacquadio, E. Piccaro, M. Piccinini, A. Pickford, R. Piegaia, J. E. Pilcher, A. D. Pilkington, J. Pina, M. Pinamonti, A. Pinder, J. L. Pinfold, J. Ping, B. Pinto, O. Pirotte, C. Pizio, R. Placakyte, M. Plamondon, W. G. Plano, M.-A. Pleier, A. V. Pleskach, A. Poblaguev, S. Poddar, F. Podlyski, L. Poggioli, T. Poghosyan, M. Pohl, F. Polci, G. Polesello, A. Policicchio, A. Polini, J. Poll, V. Polychronakos, D. M. Pomarede, D. Pomeroy, K. Pommès, L. Pontecorvo, B. G. 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Rescigno, S. Resconi, B. Resende, P. Reznicek, R. Rezvani, A. Richards, R. Richter, E. Richter-Was, M. Ridel, S. Rieke, M. Rijpstra, M. Rijssenbeek, A. Rimoldi, L. Rinaldi, R. R. Rios, I. Riu, G. Rivoltella, F. Rizatdinova, E. Rizvi, S. H. Robertson, A. Robichaud-Veronneau, D. Robinson, J. E. M. Robinson, M. Robinson, A. Robson, J. G. Rocha de Lima, C. Roda, D. Roda Dos Santos, S. Rodier, D. Rodriguez, Y. Rodriguez Garcia, A. Roe, S. Roe, O. Røhne, V. Rojo, S. Rolli, A. Romaniouk, V. M. Romanov, G. Romeo, D. Romero Maltrana, L. Roos, E. Ros, S. Rosati, M. Rose, G. A. Rosenbaum, E. I. Rosenberg, P. L. Rosendahl, L. Rosselet, V. Rossetti, E. Rossi, L. P. Rossi, L. Rossi, M. Rotaru, I. Roth, J. Rothberg, I. Rottländer, D. Rousseau, C. R. Royon, A. Rozanov, Y. Rozen, X. Ruan, I. Rubinskiy, B. Ruckert, N. Ruckstuhl, V. I. Rud, G. Rudolph, F. Rühr, A. Ruiz-Martinez, E. Rulikowska-Zarebska, V. Rumiantsev, L. Rumyantsev, K. Runge, O. Runolfsson, Z. Rurikova, N. A. Rusakovich, D. R. Rust, J. 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Takeda, T. Takeshita, M. Talby, A. Talyshev, M. C. Tamsett, J. Tanaka, R. Tanaka, S. Tanaka, S. Tanaka, Y. Tanaka, K. Tani, N. Tannoury, G. P. Tappern, S. Tapprogge, D. Tardif, S. Tarem, F. Tarrade, G. F. Tartarelli, P. Tas, M. Tasevsky, E. Tassi, M. Tatarkhanov, C. Taylor, F. E. Taylor, G. Taylor, G. N. Taylor, W. Taylor, M. Teixeira Dias Castanheira, P. Teixeira-Dias, K. K. Temming, H. Ten Kate, P. K. Teng, Y. D. Tennenbaum-Katan, S. Terada, K. Terashi, J. Terron, M. Terwort, M. Testa, R. J. Teuscher, C. M. Tevlin, J. Thadome, J. Therhaag, T. Theveneaux-Pelzer, M. Thioye, S. Thoma, J. P. Thomas, E. N. Thompson, P. D. Thompson, P. D. Thompson, A. S. Thompson, E. Thomson, M. Thomson, R. P. Thun, T. Tic, V. O. Tikhomirov, Y. A. Tikhonov, C.J.W.P. Timmermans, P. Tipton, F. J. Tique Aires Viegas, S. Tisserant, J. Tobias, B. Toczek, T. Todorov, S. Todorova-Nova, B. Toggerson, J. Tojo, S. Tokár, K. Tokunaga, K. Tokushuku, K. Tollefson, M. Tomoto, L. Tompkins, K. Toms, A. Tonazzo, G. 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Wildt, I. Wilhelm, H. G. Wilkens, J. Z. Will, E. Williams, H. H. Williams, W. Willis, S. Willocq, J. A. Wilson, M. G. Wilson, A. Wilson, I. Wingerter-Seez, S. Winkelmann, F. Winklmeier, M. Wittgen, M. W. Wolter, H. Wolters, G. Wooden, B. K. Wosiek, J. Wotschack, M. J. Woudstra, K. Wraight, C. Wright, B. Wrona, S. L. Wu, X. Wu, Y. Wu, E. Wulf, R. Wunstorf, B. M. Wynne, L. Xaplanteris, S. Xella, S. Xie, Y. Xie, C. Xu, D. Xu, G. Xu, B. Yabsley, M. Yamada, A. Yamamoto, K. Yamamoto, S. Yamamoto, T. Yamamura, J. Yamaoka, T. Yamazaki, Y. Yamazaki, Z. Yan, H. Yang, U. K. Yang, Y. Yang, Y. Yang, Z. Yang, S. Yanush, W-M. Yao, Y. Yao, Y. Yasu, J. Ye, S. Ye, M. Yilmaz, R. Yoosoofmiya, K. Yorita, R. Yoshida, C. Young, S. Youssef, D. Yu, J. Yu, J. Yu, L. Yuan, A. Yurkewicz, V. G. Zaets, R. Zaidan, A. M. Zaitsev, Z. Zajacova, Yo.K. Zalite, L. Zanello, P. Zarzhitsky, A. Zaytsev, M. Zdrazil, C. Zeitnitz, M. Zeller, P. F. Zema, A. Zemla, C. Zendler, A. V. Zenin, O. Zenin, T. Ženiš, Z. Zenonos, S. Zenz, D. Zerwas, G. Zevi della Porta, Z. Zhan, D. Zhang, H. Zhang, J. Zhang, X. Zhang, Z. Zhang, L. Zhao, T. Zhao, Z. Zhao, A. Zhemchugov, S. Zheng, J. Zhong, B. Zhou, N. Zhou, Y. Zhou, C. G. Zhu, H. Zhu, Y. Zhu, X. Zhuang, V. Zhuravlov, D. Zieminska, B. Zilka, R. Zimmermann, S. Zimmermann, S. Zimmermann, M. Ziolkowski, R. Zitoun, L. Živković, V. V. Zmouchko, G. Zobernig, A. Zoccoli, Y. Zolnierowski, A. Zsenei, M. zur Nedden, V. Zutshi, L. Zwalinski, Doğuş Üniversitesi, Fen Edebiyat Fakültesi, Fizik Bölümü, TR3959, Çetin, Serkant Ali, G., Aad, Bianco, Michele, G., Chiodini, M. R., Coluccia, Gorini, Edoardo, Primavera, Margherita, Spagnolo, Stefania Antonia, Ventura, Andrea, Laboratoire d'Annecy de Physique des Particules (LAPP), Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3)-Centre National de la Recherche Scientifique (CNRS), Université Pierre et Marie Curie - Paris 6 (UPMC)-Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3)-Université Paris Diderot - Paris 7 (UPD7)-Centre National de la Recherche Scientifique (CNRS)-Université Pierre et Marie Curie - Paris 6 (UPMC)-Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3)-Université Paris Diderot - Paris 7 (UPD7)-Centre National de la Recherche Scientifique (CNRS)-AstroParticule et Cosmologie (APC (UMR_7164)), Commissariat à l'énergie atomique et aux énergies alternatives (CEA)-Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3)-Observatoire de Paris, Université Paris sciences et lettres (PSL)-Université Paris sciences et lettres (PSL)-Université Paris Diderot - Paris 7 (UPD7)-Centre National de la Recherche Scientifique (CNRS)-Commissariat à l'énergie atomique et aux énergies alternatives (CEA)-Observatoire de Paris, Université Paris sciences et lettres (PSL)-Université Paris sciences et lettres (PSL)-Université Paris Diderot - Paris 7 (UPD7)-Centre National de la Recherche Scientifique (CNRS), Université Joseph Fourier - Grenoble 1 (UJF)-Institut polytechnique de Grenoble - Grenoble Institute of Technology (Grenoble INP )-Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3)-Institut Polytechnique de Grenoble - Grenoble Institute of Technology-Centre National de la Recherche Scientifique (CNRS), Aix Marseille Université (AMU)-Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3)-Centre National de la Recherche Scientifique (CNRS), Université de Strasbourg (UNISTRA)-Université de Haute-Alsace (UHA) Mulhouse - Colmar (Université de Haute-Alsace (UHA))-Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3)-Centre National de la Recherche Scientifique (CNRS), UAM. Departamento de Física Teórica, Centre National de la Recherche Scientifique (CNRS)-Université Paris Diderot - Paris 7 (UPD7)-Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3)-Université Pierre et Marie Curie - Paris 6 (UPMC), Centre National de la Recherche Scientifique (CNRS)-Université Paris Diderot - Paris 7 (UPD7)-Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3)-Université Pierre et Marie Curie - Paris 6 (UPMC)-Centre National de la Recherche Scientifique (CNRS)-Université Paris Diderot - Paris 7 (UPD7)-Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3)-Université Pierre et Marie Curie - Paris 6 (UPMC)-AstroParticule et Cosmologie (APC (UMR_7164)), Observatoire de Paris, Université Paris sciences et lettres (PSL)-Université Paris sciences et lettres (PSL)-Commissariat à l'énergie atomique et aux énergies alternatives (CEA)-Université Paris Diderot - Paris 7 (UPD7)-Centre National de la Recherche Scientifique (CNRS)-Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3)-Observatoire de Paris, Université Paris sciences et lettres (PSL)-Université Paris sciences et lettres (PSL)-Commissariat à l'énergie atomique et aux énergies alternatives (CEA)-Université Paris Diderot - Paris 7 (UPD7)-Centre National de la Recherche Scientifique (CNRS), Institut polytechnique de Grenoble - Grenoble Institute of Technology (Grenoble INP )-Institut Polytechnique de Grenoble - Grenoble Institute of Technology-Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3)-Université Joseph Fourier - Grenoble 1 (UJF)-Centre National de la Recherche Scientifique (CNRS), Centre National de la Recherche Scientifique (CNRS)-Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3)-Université Paris-Sud - Paris 11 (UP11), Université de Strasbourg (UNISTRA)-Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3)-Centre National de la Recherche Scientifique (CNRS), Universidade do Minho, Abdelalim Aly, Ahmed Aly, Alexandre, Gauthier, Backes, Moritz, Bell, Paul, Bell, William, Berglund, Frida Elina, Blondel, Alain, Bucci, Francesca, Clark, Allan Geoffrey, Dao, Valerio, Efthymiopoulos, Ilias, Ferrere, Didier, Gadomski, Szymon, Garcia Navarro, Jose Enrique, Gaumer, Olivier, Gonzalez Sevilla, Sergio, Goulette, Marc, Hamilton, Andrew, Leger, Annie, Lister, Alison, Macina-Buono, Daniela, Martin Dit Latour, Bertrand, Mikulec, Bettina, Moneta, Lorenzo, Mora Herrera, Maria Clemencia Rosario, Morone, Maria Cristina, Nektarijevic, Snezana, Orellana, Frédérik, Pasztor, Arpad, Pohl, Martin, Robichaud-Veronneau, Andrée, Rosselet, Laurent, Urquijo, Phillip, Wu, Xin, and ATLAS Collaboration (ukupan broj autora: 3048)

Subjects
Particle physics, MISSING TRANSVERSE ENERGY, Ciências Naturais::Ciências Físicas, Atlas detector, Ciências Físicas [Ciências Naturais], FIS/04 - Fisica Nucleare e Subnucleare, General Physics and Astronomy, FOS: Physical sciences, transverse energy: missing-energy, ATLAS, universal extra dimension, compactification, CERN LHC Coll, background, p p: interaction, final state: two-photon, photon: Kaluza-Klein, electroweak interaction: standard model: validity test, experimental results, 7000 GeV-cms, ddc:500.2, 01 natural sciences, 7. Clean energy, High Energy Physics - Experiment, Nuclear physics, Universal extra dimension, High Energy Physics - Experiment (hep-ex), 0103 physical sciences, ddc:550, [PHYS.HEXP]Physics [physics]/High Energy Physics - Experiment [hep-ex], diphoton, events, transverse energy, proton-proton collisions, Hadron, ddc:530, High Energy Physics, Specific model, 010306 general physics, Nuclear Experiment, GeneralLiterature_REFERENCE(e.g.,dictionaries,encyclopedias,glossaries), QC, Physics, Large Hadron Collider, Science & Technology, Compactification (physics), 010308 nuclear & particles physics, Atlas (topology), Settore FIS/01 - Fisica Sperimentale, Física, FIS/01 - Fisica Sperimentale, Transverse plane, Col·lisions (Física nuclear), Experimental High Energy Physics, Universal Extra Dimensions, Física nuclear, High Energy Physics::Experiment, LHC, Particle Physics - Experiment
Abstract

A search for diphoton events with large missing transverse energy is presented. The data were collected with the ATLAS detector in proton-proton collisions at √s=7 TeV at the CERN Large Hadron Collider and correspond to an integrated luminosity of 3.1 pb−1. No excess of such events is observed above the standard model background prediction. In the context of a specific model with one universal extra dimension with compactification radius R and gravity-induced decays, values of 1/R, We wish to thank CERN for the efficient commissioning and operation of the LHC during this initial high-energy data-taking period as well as the support staff from our institutions without whom ATLAS could not be operated efficiently. We acknowledge the support of ANPCyT, Argentina; YerPhI, Armenia; ARC, Australia; BMWF, Austria; ANAS, Azerbaijan; SSTC, Belarus; CNPq and FAPESP, Brazil; NSERC, NRC and CFI, Canada; CERN; CONICYT, Chile; CAS, MOST and NSFC, China; COLCIENCIAS, Colombia; MSMT CR, MPO CR and VSC CR, Czech Republic; DNRF, DNSRC and Lundbeck Foundation, Denmark; ARTEMIS, European Union; IN2P3-CNRS, CEA-DSM/IRFU, France; GNAS, Georgia; BMBF, DFG, HGF, MPG and AvH Foundation, Germany; GSRT, Greece; ISF, MINERVA, GIF, DIP and Benoziyo Center, Israel; INFN, Italy; MEXT and JSPS, Japan; CNRST, Morocco; FOM and NWO, Netherlands; RCN, Norway; MNiSW, Poland; GRICES and FCT, Portugal; MERYS (MECTS), Romania; MES of Russia and ROSATOM, Russian Federation; JINR; MSTD, Serbia; MSSR, Slovakia; ARRS and MVZT, Slovenia; DST/NRF, South Africa; MICINN, Spain; SRC and Wallenberg Foundation, Sweden; SER, SNSF and Cantons of Bern and Geneva, Switzerland; NSC, Taiwan; TAEK, Turkey; STFC, the Royal Society and Leverhulme Trust, United Kingdom; DOE and NSF, United States of America. The crucial computing support from all WLCG partners is acknowledged gratefully, in particular, from CERN and the ATLAS Tier-1 facilities at TRIUMF (Canada), NDGF (Denmark, Norway, Sweden), CC-IN2P3 (France), KIT/GridKA (Germany), INFN-CNAF (Italy), NL-T1 (Netherlands), PIC (Spain), ASGC (Taiwan), RAL (UK) and BNL (USA) and in the Tier-2 facilities worldwide., info:eu-repo/semantics/publishedVersion

Published
2011

72. Measurement of the inclusive isolated prompt photon cross section in pp collisions at √s=7 TeV with the ATLAS detector

Author

Aad, G., Abbott, B., Abdallah, J., Abdelalim, A. A., Abdesselam, A., Abdinov, O., Abi, B., Abolins, M., Abramowicz, H., Abreu, H., Acerbi, E., Acharya, B. S., Ackers, M., Adams, D. L., Addy, T. N., Adelman, J., Aderholz, M., Adomeit, S., Adragna, P., Adye, T., Aefsky, S., Aguilar-Saavedra, J. A., Aharrouche, M., Ahlen, S. P., Ahles, F., Ahmad, A., Ahsan, M., Aielli, G., Akdogan, T., Åkesson, T. P. A., Akimoto, G., Akimov, A. V., Alam, M. S., Alam, M. A., Albrand, S., Aleksa, M., Aleksandrov, I. N., Aleppo, M., Alessandria, F., Alexa, C., Alexander, G., Alexandre, G., Alexopoulos, T., Alhroob, M., Aliev, M., Alimonti, G., Alison, J., Aliyev, M., Allport, P. P., Allwood-Spiers, S. E., Almond, J., Aloisio, A., Alon, R., Alonso, A., Alonso, J., Alviggi, M. G., Amako, K., Amaral, P., Amelung, C., Ammosov, V. V., Amorim, A., Amorós, G., Amram, N., Anastopoulos, C., Andari, N., Andeen, T., Anders, C. F., Anderson, K. J., Andreazza, A., Andrei, V., Andrieux, M. -L., Anduaga, X. S., Angerami, A., Anghinolfi, F., Anjos, N., Annovi, A., Antonaki, A., Antonelli, M., Antonelli, S., Antos, J., Anulli, F., Aoun, S., Aperio Bella, L., Apolle, R., Arabidze, G., Aracena, I., Arai, Y., Arce, A. T. H., Archambault, J. P., Arfaoui, S., Arguin, J. -F., Arik, E., Arik, M., Armbruster, A. J., Arms, K. E., Armstrong, S. R., Arnaez, O., Arnault, C., Artamonov, A., Artoni, G., Arutinov, D., Asai, S., Asfandiyarov, R., Ask, S., Åsman, B., Asquith, L., Assamagan, K., Astbury, A., Astvatsatourov, A., Atoian, G., Aubert, B., Auerbach, B., Auge, E., Augsten, K., Aurousseau, M., Austin, N., Avramidou, R., Axen, D., Ay, C., Azuelos, G., Azuma, Y., Baak, M. A., Baccaglioni, G., Bacci, C., Bach, A. M., Bachacou, H., Bachas, K., Bachy, G., Backes, M., Badescu, E., Bagnaia, P., Bahinipati, S., Bai, Y., Bailey, D. C., Bain, T., Baines, J. T., Baker, O. K., Baker, S., Baltasar Dos Santos Pedrosa, F., Banas, E., Banerjee, P., Banerjee, Sw., Banfi, D., Bangert, A., Bansal, V., Bansil, H. S., Barak, L., Baranov, S. P., Barashkou, A., Barbaro Galtieri, A., Barber, T., Barberio, E. L., Barberis, D., Barbero, M., Bardin, D. Y., Barillari, T., Barisonzi, M., Barklow, T., Barlow, N., Barnett, B. M., Barnett, R. M., Baroncelli, A., Barr, A. J., Barreiro, F., Barreiro Guimarães Da Costa, J., Barrillon, P., Bartoldus, R., Barton, A. E., Bartsch, D., Bates, R. L., Batkova, L., Batley, J. R., Battaglia, A., Battistin, M., Battistoni, G., Bauer, F., Bawa, H. S., Beare, B., Beau, T., Beauchemin, P. H., Beccherle, R., Bechtle, P., Beck, H. P., Beckingham, M., Becks, K. H., Beddall, A. J., Beddall, A., Bednyakov, V. A., Bee, C., Begel, M., Behar Harpaz, S., Behera, P. K., Beimforde, M., Belanger-Champagne, C., Belhorma, B., Bell, P. J., Bell, W. H., Bella, G., Bellagamba, L., Bellina, F., Bellomo, G., Bellomo, M., Belloni, A., Belotskiy, K., Beltramello, O., Ben Ami, S., Benary, O., Benchekroun, D., Benchouk, C., Bendel, M., Benedict, B. H., Benekos, N., Benhammou, Y., Benjamin, D. P., Benoit, M., Bensinger, J. R., Benslama, K., Bentvelsen, S., Berge, D., Bergeaas Kuutmann, E., Berger, N., Berghaus, F., Berglund, E., Beringer, J., Bernardet, K., Bernat, P., Bernhard, R., Bernius, C., Berry, T., Bertin, A., Bertinelli, F., Bertolucci, F., Besana, M. I., Besson, N., Bethke, S., Bhimji, W., Bianchi, R. M., Bianco, M., Biebel, O., Biesiada, J., Biglietti, M., Bilokon, H., Bindi, M., Bingul, A., Bini, C., Biscarat, C., Bischof, R., Bitenc, U., Black, K. M., Blair, R. E., Blanchard, J. -B., Blanchot, G., Blocker, C., Blocki, J., Blondel, A., Blum, W., Blumenschein, U., Boaretto, C., Bobbink, G. J., Bobrovnikov, V. B., Bocci, A., Bock, R., Boddy, C. R., Boehler, M., Boek, J., Boelaert, N., Böser, S., Bogaerts, J. A., Bogdanchikov, A., Bogouch, A., Bohm, C., Boisvert, V., Bold, T., Boldea, V., Bona, M., Boonekamp, M., Boorman, G., Booth, C. N., Booth, P., Booth, J. R. A., Bordoni, S., Borer, C., Borisov, A., Borissov, G., Borjanovic, I., Borroni, S., Bos, K., Boscherini, D., Bosman, M., Boterenbrood, H., Botterill, D., Bouchami, J., Boudreau, J., Bouhova-Thacker, E. V., Boulahouache, C., Bourdarios, C., Bousson, N., Boveia, A., Boyd, J., Boyko, I. R., Bozhko, N. I., Bozovic-Jelisavcic, I., Braccini, S., Bracinik, J., Braem, A., Brambilla, E., Branchini, P., Brandenburg, G. W., Brandt, A., Brandt, G., Brandt, O., Bratzler, U., Brau, B., Brau, J. E., Braun, H. M., Brelier, B., Bremer, J., Brenner, R., Bressler, S., Breton, D., Brett, N. D., Bright-Thomas, P. G., Britton, D., Brochu, F. M., Brock, I., Brock, R., Brodbeck, T. J., Brodet, E., Broggi, F., Bromberg, C., Brooijmans, G., Brooks, W. K., Brown, G., Brubaker, E., Bruckman De Renstrom, P. A., Bruncko, D., Bruneliere, R., Brunet, S., Bruni, A., Bruni, G., Bruschi, M., Buanes, T., Bucci, F., Buchanan, J., Buchanan, N. J., Buchholz, P., Buckingham, R. M., Buckley, A. G., Buda, S. I., Budagov, I. A., Budick, B., Büscher, V., Bugge, L., Buira-Clark, D., Buis, E. J., Bulekov, O., Bunse, M., Buran, T., Burckhart, H., Burdin, S., Burgess, T., Burke, S., Busato, E., Bussey, P., Buszello, C. P., Butin, F., Butler, B., Butler, J. M., Buttar, C. M., Butterworth, J. M., Buttinger, W., Byatt, T., Cabrera Urbán, S., Caccia, M., Caforio, D., Cakir, O., Calafiura, P., Calderini, G., Calfayan, P., Calkins, R., Caloba, L. P., Caloi, R., Calvet, D., Calvet, S., Camard, A., Camarri, P., Cambiaghi, M., Cameron, D., Cammin, J., Campana, S., Campanelli, M., Canale, V., Canelli, F., Canepa, A., Cantero, J., Capasso, L., Capeans Garrido, M. D. M., Caprini, I., Caprini, M., Caprio, M., Capriotti, D., Capua, M., Caputo, R., Caramarcu, C., Cardarelli, R., Carli, T., Carlino, G., Carminati, L., Caron, B., Caron, S., Carpentieri, C., Carrillo Montoya, G. D., Carron Montero, S., Carter, A. A., Carter, J. R., Carvalho, J., Casadei, D., Casado, M. P., Cascella, M., Caso, C., Castaneda Hernandez, A. M., Castaneda-Miranda, E., Castillo Gimenez, V., Castro, N. F., Cataldi, G., Cataneo, F., Catinaccio, A., Catmore, J. R., Cattai, A., Cattani, G., Caughron, S., Cavallari, A., Cavalleri, P., Cavalli, D., Cavalli-Sforza, M., Cavasinni, V., Cazzato, A., Ceradini, F., Cerna, C., Cerqueira, A. S., Cerri, A., Cerrito, L., Cerutti, F., Cervetto, M., Cetin, S. A., Cevenini, F., Chafaq, A., Chakraborty, D., Chan, K., Chapleau, B., Chapman, J. D., Chapman, J. W., Chareyre, E., Charlton, D. G., Chavda, V., Cheatham, S., Chekanov, S., Chekulaev, S. V., Chelkov, G. A., Chen, H., Chen, L., Chen, S., Chen, T., Chen, X., Cheng, S., Cheplakov, A., Chepurnov, V. F., Cherkaoui El Moursli, R., Chernyatin, V., Cheu, E., Cheung, S. L., Chevalier, L., Chevallier, F., Chiefari, G., Chikovani, L., Childers, J. T., Chilingarov, A., Chiodini, G., Chizhov, M. V., Choudalakis, G., Chouridou, S., Christidi, I. A., Christov, A., Chromek-Burckhart, D., Chu, M. L., Chudoba, J., Ciapetti, G., Ciftci, A. K., Ciftci, R., Cinca, D., Cindro, V., Ciobotaru, M. D., Ciocca, C., Ciocio, A., Cirilli, M., Ciubancan, M., Clark, A., Clark, P. J., Cleland, W., Clemens, J. C., Clement, B., Clement, C., Clifft, R. W., Coadou, Y., Cobal, M., Coccaro, A., Cochran, J., Coe, P., Cogan, J. G., Coggeshall, J., Cogneras, E., Cojocaru, C. D., Colas, J., Colijn, A. P., Collard, C., Collins, N. J., Collins-Tooth, C., Collot, J., Colon, G., Coluccia, R., Comune, G., Conde Muiño, P., Coniavitis, E., Conidi, M. C., Consonni, M., Constantinescu, S., Conta, C., Conventi, F., Cook, J., Cooke, M., Cooper, B. D., Cooper-Sarkar, A. M., Cooper-Smith, N. J., Copic, K., Cornelissen, T., Corradi, M., Correard, S., Corriveau, F., Cortes-Gonzalez, A., Cortiana, G., Costa, G., Costa, M. J., Costanzo, D., Costin, T., Côté, D., Coura Torres, R., Courneyea, L., Cowan, G., Cowden, C., Cox, B. E., Cranmer, K., Cristinziani, M., Crosetti, G., Crupi, R., Crépé-Renaudin, S., Cuenca Almenar, C., Cuhadar Donszelmann, T., Cuneo, S., Curatolo, M., Curtis, C. J., Cwetanski, P., Czirr, H., Czyczula, Z., D'Auria, S., D'Onofrio, M., D'Orazio, A., Da Rocha Gesualdi Mello, A., Da Silva, P. V. M., Da Via, C., Dabrowski, W., Dahlhoff, A., Dai, T., Dallapiccola, C., Dallison, S. J., Dam, M., Dameri, M., Damiani, D. S., Danielsson, H. O., Dankers, R., Dannheim, D., Dao, V., Darbo, G., Darlea, G. L., Daum, C., Dauvergne, J. P., Davey, W., Davidek, T., Davidson, N., Davidson, R., Davies, M., Davison, A. R., Dawe, E., Dawson, I., Dawson, J. W., Daya, R. K., De, K., De Asmundis, R., De Castro, S., De Cecco, S., De Graat, J., De Groot, N., De Jong, P., De La Cruz-Burelo, E., De La Taille, C., De Lotto, B., De Mora, L., De Nooij, L., De Oliveira Branco, M., De Pedis, D., De Saintignon, P., De Salvo, A., De Sanctis, U., De Santo, A., De Vivie De Regie, J. B., Dean, S., Dedes, G., Dedovich, D. V., Degenhardt, J., Dehchar, M., Deile, M., Del Papa, C., Del Peso, J., Del Prete, T., Dell'Acqua, A., Dell'Asta, L., Della Pietra, M., Della Volpe, D., Delmastro, M., Delpierre, P., Delruelle, N., Delsart, P. A., Deluca, C., Demers, S., Demichev, M., Demirkoz, B., Deng, J., Denisov, S. P., Dennis, C., Derendarz, D., Derkaoui, J. E., Derue, F., Dervan, P., Desch, K., Devetak, E., Deviveiros, P. O., Dewhurst, A., Dewilde, B., Dhaliwal, S., Dhullipudi, R., Di Ciaccio, A., Di Ciaccio, L., Di Girolamo, A., Di Girolamo, B., Di Luise, S., Di Mattia, A., Di Nardo, R., Di Simone, A., Di Sipio, R., Diaz, M. A., Diaz Gomez, M. M., Diblen, F., Diehl, E. B., Dietl, H., Dietrich, J., Dietzsch, T. A., Diglio, S., Dindar Yagci, K., Dingfelder, J., Dionisi, C., Dita, P., Dita, S., Dittus, F., Djama, F., Djilkibaev, R., Djobava, T., Do Vale, M. A. B., Do Valle Wemans, A., Doan, T. K. O., Dobbs, M., Dobinson, R., Dobos, D., Dobson, E., Dobson, M., Dodd, J., Dogan, O. B., Doglioni, C., Doherty, T., Doi, Y., Dolejsi, J., Dolenc, I., Dolezal, Z., Dolgoshein, B. A., Dohmae, T., Donadelli, M., Donega, M., Donini, J., Dopke, J., Doria, A., Dos Anjos, A., Dosil, M., Dotti, A., Dova, M. T., Dowell, J. D., Doxiadis, A. D., Doyle, A. T., Drasal, Z., Drees, J., Dressnandt, N., Drevermann, H., Driouichi, C., Dris, M., Drohan, J. G., Dubbert, J., Dubbs, T., Dube, S., Duchovni, E., Duckeck, G., Dudarev, A., Dudziak, F., Dührssen, M., Duerdoth, I. P., Duflot, L., Dufour, M. -A., Dunford, M., Duran Yildiz, H., Duxfield, R., Dwuznik, M., Dydak, F., Dzahini, D., Düren, M., Ebke, J., Eckert, S., Eckweiler, S., Edmonds, K., Edwards, C. 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Rescigno, S. Resconi, B. Resende, P. Reznicek, R. Rezvani, A. Richards, R. Richter, E. Richter-Was, M. Ridel, S. Rieke, M. Rijpstra, M. Rijssenbeek, A. Rimoldi, L. Rinaldi, R. R. Rios, I. Riu, G. Rivoltella, F. Rizatdinova, E. Rizvi, S. H. Robertson, A. Robichaud-Veronneau, D. Robinson, J. E. M. Robinson, M. Robinson, A. Robson, J. G. Rocha de Lima, C. Roda, D. Roda Dos Santos, S. Rodier, D. Rodriguez, Y. Rodriguez Garcia, A. Roe, S. Roe, O. Røhne, V. Rojo, S. Rolli, A. Romaniouk, V. M. Romanov, G. Romeo, D. Romero Maltrana, L. Roos, E. Ros, S. Rosati, M. Rose, G. A. Rosenbaum, E. I. Rosenberg, P. L. Rosendahl, L. Rosselet, V. Rossetti, E. Rossi, L. P. Rossi, L. Rossi, M. Rotaru, I. Roth, J. Rothberg, I. Rottländer, D. Rousseau, C. R. Royon, A. Rozanov, Y. Rozen, X. Ruan, I. Rubinskiy, B. Ruckert, N. Ruckstuhl, V. I. Rud, G. Rudolph, F. Rühr, A. Ruiz-Martinez, E. Rulikowska-Zarebska, V. Rumiantsev, L. Rumyantsev, K. Runge, O. Runolfsson, Z. Rurikova, N. A. Rusakovich, D. R. Rust, J. P. Rutherfoord, C. Ruwiedel, P. Ruzicka, Y. F. Ryabov, V. Ryadovikov, P. Ryan, M. Rybar, G. Rybkin, N. C. Ryder, S. Rzaeva, A. F. Saavedra, I. Sadeh, H. F-W. Sadrozinski, R. Sadykov, F. Safai Tehrani, H. Sakamoto, G. Salamanna, A. Salamon, M. Saleem, D. Salihagic, A. Salnikov, J. Salt, B. M. Salvachua Ferrando, D. Salvatore, F. Salvatore, A. Salzburger, D. Sampsonidis, B. H. Samset, H. Sandaker, H. G. Sander, M. P. Sanders, M. Sandhoff, P. Sandhu, T. Sandoval, R. Sandstroem, S. Sandvoss, D. P. C. Sankey, A. Sansoni, C. Santamarina Rios, C. Santoni, R. Santonico, H. Santos, J. G. Saraiva, T. Sarangi, E. Sarkisyan-Grinbaum, F. Sarri, G. Sartisohn, O. Sasaki, T. Sasaki, N. Sasao, I. Satsounkevitch, G. Sauvage, J. B. Sauvan, P. Savard, V. Savinov, P. Savva, L. Sawyer, D. H. Saxon, L. P. Says, C. Sbarra, A. Sbrizzi, O. Scallon, D. A. Scannicchio, J. Schaarschmidt, P. Schacht, U. Schäfer, S. Schaetzel, A. C. Schaffer, D. Schaile, R. D. Schamberger, A. G. Schamov, V. Scharf, V. A. Schegelsky, D. Scheirich, M. I. Scherzer, C. Schiavi, J. Schieck, M. Schioppa, S. Schlenker, J. L. Schlereth, E. Schmidt, M. P. Schmidt, K. Schmieden, C. Schmitt, M. Schmitz, A. Schöning, M. Schott, D. Schouten, J. Schovancova, M. Schram, A. Schreiner, C. Schroeder, N. Schroer, S. Schuh, G. Schuler, J. Schultes, H.-C. Schultz-Coulon, H. Schulz, J. W. Schumacher, M. Schumacher, B. A. Schumm, Ph. Schune, C. Schwanenberger, A. Schwartzman, D. Schweiger, Ph. Schwemling, R. Schwienhorst, R. Schwierz, J. Schwindling, W. G. Scott, J. Searcy, E. Sedykh, E. Segura, S. C. Seidel, A. Seiden, F. Seifert, J. M. Seixas, G. Sekhniaidze, D. M. Seliverstov, B. Sellden, G. Sellers, M. Seman, N. Semprini-Cesari, C. Serfon, L. Serin, R. Seuster, H. Severini, M. E. Sevior, A. Sfyrla, E. Shabalina, M. Shamim, L. Y. Shan, J. T. Shank, Q. T. Shao, M. Shapiro, P. B. Shatalov, L. Shaver, C. Shaw, K. Shaw, D. Sherman, P. Sherwood, A. Shibata, S. Shimizu, M. Shimojima, T. Shin, A. Shmeleva, M. J. Shochet, D. Short, M. A. Shupe, P. Sicho, A. Sidoti, A. Siebel, F. Siegert, J. Siegrist, Dj. Sijacki, O. Silbert, J. Silva, Y. Silver, D. Silverstein, S. B. Silverstein, V. Simak, Lj. Simic, S. Simion, B. Simmons, M. Simonyan, P. Sinervo, N. B. Sinev, V. Sipica, G. Siragusa, A. N. Sisakyan, S. Yu. Sivoklokov, J. Sjölin, T. B. Sjursen, L. A. Skinnari, K. Skovpen, P. Skubic, N. Skvorodnev, M. Slater, T. Slavicek, K. Sliwa, T. J. Sloan, J. Sloper, V. Smakhtin, S. Yu. Smirnov, L. N. Smirnova, O. Smirnova, B. C. Smith, D. Smith, K. M. Smith, M. Smizanska, K. Smolek, A. A. Snesarev, S. W. Snow, J. Snow, J. Snuverink, S. Snyder, M. Soares, R. Sobie, J. Sodomka, A. Soffer, C. A. Solans, M. Solar, J. Solc, U. Soldevila, E. Solfaroli Camillocci, A. A. Solodkov, O. V. Solovyanov, J. Sondericker, N. Soni, V. Sopko, B. Sopko, M. Sorbi, M. Sosebee, A. Soukharev, S. Spagnolo, F. Spanò, R. Spighi, G. Spigo, F. Spila, E. Spiriti, R. Spiwoks, M. Spousta, T. Spreitzer, B. Spurlock, R. D. St. Denis, T. Stahl, J. Stahlman, R. Stamen, E. Stanecka, R. W. Stanek, C. Stanescu, S. Stapnes, E. A. Starchenko, J. Stark, P. Staroba, P. Starovoitov, A. Staude, P. Stavina, G. Stavropoulos, G. Steele, E. Stefanidis, P. Steinbach, P. Steinberg, I. Stekl, B. Stelzer, H. J. Stelzer, O. Stelzer-Chilton, H. Stenzel, K. Stevenson, G. A. Stewart, T. Stockmanns, M. C. Stockton, M. Stodulski, K. Stoerig, G. Stoicea, S. Stonjek, P. Strachota, A. R. Stradling, A. Straessner, J. Strandberg, S. Strandberg, A. Strandlie, M. Strang, E. Strauss, M. Strauss, P. Strizenec, R. Ströhmer, D. M. Strom, J. A. Strong, R. Stroynowski, J. Strube, B. Stugu, I. Stumer, J. Stupak, P. Sturm, D. A. Soh, D. Su, S. Subramania, Y. Sugaya, T. Sugimoto, C. Suhr, K. Suita, M. Suk, V. V. Sulin, S. Sultansoy, T. Sumida, X. Sun, J. E. Sundermann, K. Suruliz, S. Sushkov, G. Susinno, M. R. Sutton, Y. Suzuki, Yu. M. Sviridov, S. Swedish, I. Sykora, T. Sykora, B. Szeless, J. Sánchez, D. Ta, K. Tackmann, A. Taffard, R. Tafirout, A. Taga, N. Taiblum, Y. Takahashi, H. Takai, R. Takashima, H. Takeda, T. Takeshita, M. Talby, A. Talyshev, M. C. Tamsett, J. Tanaka, R. Tanaka, S. Tanaka, S. Tanaka, Y. Tanaka, K. Tani, N. Tannoury, G. P. Tappern, S. Tapprogge, D. Tardif, S. Tarem, F. Tarrade, G. F. Tartarelli, P. Tas, M. Tasevsky, E. Tassi, M. Tatarkhanov, C. Taylor, F. E. Taylor, G. Taylor, G. N. Taylor, W. Taylor, M. Teixeira Dias Castanheira, P. Teixeira-Dias, K. K. Temming, H. Ten Kate, P. K. Teng, Y. D. Tennenbaum-Katan, S. Terada, K. Terashi, J. Terron, M. Terwort, M. Testa, R. J. Teuscher, C. M. Tevlin, J. Thadome, J. Therhaag, T. Theveneaux-Pelzer, M. Thioye, S. Thoma, J. P. Thomas, E. N. Thompson, P. D. Thompson, P. D. Thompson, A. S. Thompson, E. Thomson, M. Thomson, R. P. Thun, T. Tic, V. O. Tikhomirov, Y. A. Tikhonov, C. J. W. P. Timmermans, P. Tipton, F. J. Tique Aires Viegas, S. Tisserant, J. Tobias, B. Toczek, T. Todorov, S. Todorova-Nova, B. Toggerson, J. Tojo, S. Tokár, K. Tokunaga, K. Tokushuku, K. Tollefson, M. Tomoto, L. Tompkins, K. Toms, A. Tonazzo, G. Tong, A. Tonoyan, C. Topfel, N. D. Topilin, I. Torchiani, E. Torrence, E. Torró Pastor, J. Toth, F. Touchard, D. R. Tovey, D. Traynor, T. Trefzger, J. Treis, L. Tremblet, A. Tricoli, I. M. Trigger, S. Trincaz-Duvoid, T. N. Trinh, M. F. Tripiana, N. Triplett, W. Trischuk, A. Trivedi, B. Trocmé, C. Troncon, M. Trottier-McDonald, A. Trzupek, C. Tsarouchas, J. C-L. Tseng, M. Tsiakiris, P. V. Tsiareshka, D. Tsionou, G. Tsipolitis, V. Tsiskaridze, E. G. Tskhadadze, I. I. Tsukerman, V. Tsulaia, J.-W. Tsung, S. Tsuno, D. Tsybychev, A. Tua, J. M. Tuggle, M. Turala, D. Turecek, I. Turk Cakir, E. Turlay, P. M. Tuts, A. Tykhonov, M. Tylmad, M. Tyndel, D. Typaldos, H. Tyrvainen, G. Tzanakos, K. Uchida, I. Ueda, R. Ueno, M. Ugland, M. Uhlenbrock, M. Uhrmacher, F. Ukegawa, G. Unal, D. G. Underwood, A. Undrus, G. Unel, Y. Unno, D. Urbaniec, E. Urkovsky, P. Urquijo, P. Urrejola, G. Usai, M. Uslenghi, L. Vacavant, V. Vacek, B. Vachon, S. Vahsen, C. Valderanis, J. Valenta, P. Valente, S. Valentinetti, S. Valkar, E. Valladolid Gallego, S. Vallecorsa, J. A. Valls Ferrer, H. van der Graaf, E. van der Kraaij, E. van der Poel, D. van der Ster, B. Van Eijk, N. van Eldik, P. van Gemmeren, Z. van Kesteren, I. van Vulpen, W. Vandelli, G. Vandoni, A. Vaniachine, P. Vankov, F. Vannucci, F. Varela Rodriguez, R. Vari, E. W. Varnes, D. Varouchas, A. Vartapetian, K. E. Varvell, V. I. Vassilakopoulos, F. Vazeille, G. Vegni, J. J. Veillet, C. Vellidis, F. Veloso, R. Veness, S. Veneziano, A. Ventura, D. Ventura, S. Ventura, M. Venturi, N. Venturi, V. Vercesi, M. Verducci, W. Verkerke, J. C. Vermeulen, L. Vertogardov, A. Vest, M. C. Vetterli, I. Vichou, T. Vickey, G. H. A. Viehhauser, S. Viel, M. Villa, M. Villaplana Perez, E. Vilucchi, M. G. Vincter, E. Vinek, V. B. Vinogradov, M. Virchaux, S. Viret, J. Virzi, A. Vitale, O. Vitells, I. Vivarelli, F. Vives Vaque, S. Vlachos, M. Vlasak, N. Vlasov, A. Vogel, P. Vokac, M. Volpi, G. Volpini, H. von der Schmitt, J. von Loeben, H. von Radziewski, E. von Toerne, V. Vorobel, A. P. Vorobiev, V. Vorwerk, M. Vos, R. Voss, T. T. Voss, J. H. Vossebeld, A. S. Vovenko, N. Vranjes, M. Vranjes Milosavljevic, V. Vrba, M. Vreeswijk, T. Vu Anh, R. Vuillermet, I. Vukotic, W. Wagner, P. Wagner, H. Wahlen, J. Wakabayashi, J. Walbersloh, S. Walch, J. Walder, R. Walker, W. Walkowiak, R. Wall, P. Waller, C. Wang, H. Wang, J. Wang, J. C. Wang, S. M. Wang, A. Warburton, C. P. Ward, M. Warsinsky, P. M. Watkins, A. T. Watson, M. F. Watson, G. Watts, S. Watts, A. T. Waugh, B. M. Waugh, J. Weber, M. Weber, M. S. Weber, P. Weber, A. R. Weidberg, J. Weingarten, C. Weiser, H. Wellenstein, P. S. Wells, M. Wen, T. Wenaus, S. Wendler, Z. Weng, T. Wengler, S. Wenig, N. Wermes, M. Werner, P. Werner, M. Werth, M. Wessels, K. Whalen, S. J. Wheeler-Ellis, S. P. Whitaker, A. White, M. J. White, S. White, S. R. Whitehead, D. Whiteson, D. Whittington, F. Wicek, D. Wicke, F. J. Wickens, W. Wiedenmann, M. Wielers, P. Wienemann, C. Wiglesworth, L. A. M. Wiik, A. Wildauer, M. A. Wildt, I. Wilhelm, H. G. Wilkens, J. Z. Will, E. Williams, H. H. Williams, W. Willis, S. Willocq, J. A. Wilson, M. G. Wilson, A. Wilson, I. Wingerter-Seez, S. Winkelmann, F. Winklmeier, M. Wittgen, M. W. Wolter, H. Wolters, G. Wooden, B. K. Wosiek, J. Wotschack, M. J. Woudstra, K. Wraight, C. Wright, B. Wrona, S. L. Wu, X. Wu, Y. Wu, E. Wulf, R. Wunstorf, B. M. Wynne, L. Xaplanteris, S. Xella, S. Xie, Y. Xie, C. Xu, D. Xu, G. Xu, B. Yabsley, M. Yamada, A. Yamamoto, K. Yamamoto, S. Yamamoto, T. Yamamura, J. Yamaoka, T. Yamazaki, Y. Yamazaki, Z. Yan, H. Yang, S. Yang, U. K. Yang, Y. Yang, Y. Yang, Z. Yang, S. Yanush, W-M. Yao, Y. Yao, Y. Yasu, J. Ye, S. Ye, M. Yilmaz, R. Yoosoofmiya, K. Yorita, R. Yoshida, C. Young, S. Youssef, D. Yu, J. Yu, J. Yu, L. Yuan, A. Yurkewicz, V. G. Zaets, R. Zaidan, A. M. Zaitsev, Z. Zajacova, Yo. K. Zalite, L. Zanello, P. Zarzhitsky, A. Zaytsev, M. Zdrazil, C. Zeitnitz, M. Zeller, P. F. Zema, A. Zemla, C. Zendler, A. V. Zenin, O. Zenin, T. Ženiš, Z. Zenonos, S. Zenz, D. Zerwas, G. Zevi della Porta, Z. Zhan, D. Zhang, H. Zhang, J. Zhang, X. Zhang, Z. Zhang, L. Zhao, T. Zhao, Z. Zhao, A. Zhemchugov, S. Zheng, J. Zhong, B. Zhou, N. Zhou, Y. Zhou, C. G. Zhu, H. Zhu, Y. Zhu, X. Zhuang, V. Zhuravlov, D. Zieminska, B. Zilka, R. Zimmermann, S. Zimmermann, S. Zimmermann, M. Ziolkowski, R. Zitoun, L. Živković, V. V. Zmouchko, G. Zobernig, A. Zoccoli, Y. Zolnierowski, A. Zsenei, M. zur Nedden, V. Zutshi, L. Zwalinski

Subjects
Nuclear and High Energy Physics, Physics and Astronomy (miscellaneous), High Energy Physics::Experiment, +10+GeV%29%3A+Inclusive+production+with+identified+leptons+photons+or+other+nonhadronic+particles%22">Hadron-induced high- and super-high-energy interactions (energy > 10 GeV): Inclusive production with identified leptons photons or other nonhadronic particles
Abstract

A measurement of the cross section for the inclusive production of isolated prompt photons in pp collisions at a center-of-mass energy √s=7 TeV is presented. The measurement covers the pseudorapidity ranges |ηγ

Published
2011

73. Germline BARD1 variants predispose to mesothelioma by impairing DNA repair and calcium signaling.

Author

Novelli F, Yoshikawa Y, Vitto VAM, Modesti L, Minaai M, Pastorino S, Emi M, Kim JH, Kricek F, Bai F, Onuchic JN, Bononi A, Suarez JS, Tanji M, Favaron C, Zolondick AA, Xu R, Takanishi Y, Wang Z, Sakamoto G, Gaudino G, Grzymski J, Grosso F, Schrump DS, Pass HI, Atanesyan L, Smout J, Savola S, Sarin KY, Abolhassani H, Hammarström L, Pan-Hammarström Q, Giorgi C, Pinton P, Yang H, and Carbone M

Subjects
Humans, Female, Male, Middle Aged, Tumor Suppressor Protein p53 genetics, Tumor Suppressor Protein p53 metabolism, Apoptosis genetics, Fibroblasts metabolism, Asbestos toxicity, Genomic Instability, DNA Repair genetics, Tumor Suppressor Proteins genetics, Tumor Suppressor Proteins metabolism, Germ-Line Mutation, Ubiquitin-Protein Ligases genetics, Ubiquitin-Protein Ligases metabolism, Mesothelioma genetics, Genetic Predisposition to Disease, Calcium Signaling genetics
Abstract

We report that ~1.8% of all mesothelioma patients and 4.9% of those younger than 55, carry rare germline variants of the BRCA1 associated RING domain 1 ( BARD1) gene that were predicted to be damaging by computational analyses. We conducted functional assays, essential for accurate interpretation of missense variants, in primary fibroblasts that we established in tissue culture from a patient carrying the heterozygous BARD1 V523A mutation. We found that these cells had genomic instability, reduced DNA repair, and impaired apoptosis. Investigating the underlying signaling pathways, we found that BARD1 forms a trimeric protein complex with p53 and SERCA2 that regulates calcium signaling and apoptosis. We validated these findings in BARD1 -silenced primary human mesothelial cells exposed to asbestos. Our study elucidated mechanisms of BARD1 activity and revealed that heterozygous germline BARD1 mutations favor the development of mesothelioma and increase the susceptibility to asbestos carcinogenesis. These mesotheliomas are significantly less aggressive compared to mesotheliomas in asbestos workers., Competing Interests: Competing interests statement:M.C. has a patent issued for “Methods for Diagnosing a Predisposition to Develop Cancer.” M.C. and H.Y. have a patent issued for “Using Anti-HMGB1 Monoclonal Antibody or other HMGB1 Antibodies as a Novel Mesothelioma Therapeutic Strategy”, and a patent issued for “HMGB1 As a Biomarker for Asbestos Exposure and Mesothelioma Early Detection.” M.C. is a board-certified pathologist who provides consultation for pleural pathology, including medical-legal.

Published
2024
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74. Association Between Catheter Ablation for Nonparoxysmal Atrial Fibrillation and Functional Mitral Regurgitation in Patients With Heart Failure With Preserved Ejection Fraction.

Author

Yamauchi R, Morishima I, Okumura K, Kanzaki Y, Morita Y, Watanabe N, Furui K, Yoshioka N, Shibata N, Miyazawa H, Shimojo K, Imaoka T, Sakamoto G, Goto H, Ohi T, Yanagisawa S, Inden Y, and Murohara T

Subjects
Humans, Treatment Outcome, Stroke Volume, Retrospective Studies, Atrial Fibrillation complications, Atrial Fibrillation surgery, Atrial Fibrillation diagnosis, Heart Failure, Mitral Valve Insufficiency complications, Mitral Valve Insufficiency surgery, Mitral Valve Insufficiency diagnosis, Catheter Ablation adverse effects
Abstract

Functional mitral regurgitation (FMR) often coexists with atrial fibrillation (AF) and may have a causal relation with AF persistence and exacerbation of heart failure (HF). The purpose of this study was to investigate the impact of FMR on AF catheter ablation (AFCA) outcomes and improvement in FMR after AFCA in patients with HF with preserved ejection fraction (HFpEF) and nonparoxysmal AF. Excluding patients with primary valve disease or post-mitral valve repair, 280 patients with HFpEF who underwent CA for nonparoxysmal AF were retrospectively included. All patients completed 1-year follow-up and were assessed for FMR, AF recurrence and HF parameters, including echocardiography. At baseline, FMR was present in 153 (54.6%) patients (mild, n = 112; moderate, n = 40; severe, n = 1), and these were decreased to 70 (25%) significantly 1 year after AFCA (mild, n = 64; moderate, n = 6), 119 patients (78%) had improvement in MR (a decrease of ≥1 level in the severity from baseline to 1 year on echocardiography). Overall, 274 patients (97.9%) had sinus rhythm at the 1-year examination, and recurrent AF-free survival did not differ in patients with and without MR improvement (83.2% vs 82.4%, p = 0.908). However, the MR improvement group had a significantly lower cardiothoracic ratio, left atrial diameter, E/e', and B-type natriuretic peptide levels after 1 year than those in the MR nonimprovement group. In conclusion, the majority of the HFpEF patients with nonparoxysmal AF had improvement of FMR after AFCA with the high maintenance of sinus rhythm, leading to a virtuous cycle of cardiac function., Competing Interests: Declaration of Competing Interest The authors have no competing interests to declare., (Copyright © 2023 Elsevier Inc. All rights reserved.)

Published
2023
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75. The "via dialysis circuit" external bypass technique: External femoral to femoral bypass via dialysis circuit for antegrade perfusion of the ischemic limb.

Author

Sakamoto G, Shibata N, Watanabe N, Morita Y, and Morishima I

Abstract

A 71-year-old woman with no relevant previous medical history presented to the emergency department with cardiopulmonary arrest due to extensive myocardial infarction. Veno-arterial extracorporeal life support (ECLS) was passed through the right common femoral artery (CFA), and an Impella CP® (16-F introduction sheath, Abiomed Inc., Danvers, MA, USA) was inserted from the left CFA. Although these mechanical devices provide powerful cardiac and organ support, an occlusive large-bore sheath may induce ischemic limb complications. Antegrade flow from the contralateral sheath was circulated through the ECLS circuit, showing the improvement of antegrade left SFA. Cardiac function improved after primary percutaneous coronary intervention, but when the ECLS was terminated, antegrade left limb flow declined. Hence, we bypassed the contralateral flow via the dialysis circuit and prevented limb ischemia., Learning Objective: Although an Impella® (Abiomed Inc., Danvers, MA, USA) is useful for assisting left ventricular cardiac function, its large-bore sheath sometimes disturbs the antegrade flow, resulting in ischemic limb complications. A novel yet simple technique that involves an external bypass through the superficial femoral artery to provide antegrade perfusion to the ipsilateral limb is hereby described., Competing Interests: The author declares that there is no conflict of interest., (© 2023 Japanese College of Cardiology. Published by Elsevier Ltd. All rights reserved.)

Published
2023
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76. Excimer laser coronary angioplasty versus manual aspiration thrombectomy in patients with ST-segment elevation myocardial infarction: analyzed by nuclear scintigraphy.

Author

Shimojo K, Shibata N, Takagi K, Mitsuhashi H, Morita Y, Kanzaki Y, Watanabe N, Yoshioka N, Miyazawa H, Imaoka T, Sakamoto G, Ohi T, Goto H, Karasawa H, and Morishima I

Subjects
Humans, Lasers, Excimer, Treatment Outcome, Predictive Value of Tests, Tomography, Emission-Computed, Single-Photon, Thrombectomy adverse effects, Thrombectomy methods, Creatine Kinase therapeutic use, Coronary Angiography, ST Elevation Myocardial Infarction diagnostic imaging, ST Elevation Myocardial Infarction therapy, ST Elevation Myocardial Infarction etiology, Myocardial Infarction diagnostic imaging, Myocardial Infarction therapy, Atherectomy, Coronary, Percutaneous Coronary Intervention adverse effects, Percutaneous Coronary Intervention methods
Abstract

Clinical outcomes concerning the efficacy of excimer laser coronary angioplasty (ELCA) in real-world cases of acute myocardial infarction (MI) are limited. We evaluated and compared the impact of ELCA with manual aspiration thrombectomy on myocardial salvage and left ventricular (LV) systolic/diastolic function in patients with ST-segment elevation MI (STEMI) using nuclear scintigraphy.  We enrolled 143 consecutive patients with STEMI treated with ELCA (63 patients) or manual aspiration thrombectomy (80 patients) between September 2016 and December 2020 in a single-center hospital. We evaluated the peak creatine kinase (CK)/ creatine kinase-myocardial band (CK-MB) levels and performed single-photon emission computed tomography (SPECT) analyses with Quantitative Gated SPECT and Quantitative Perfusion SPECT (Auto QUANT 7.2) at 3-10 days using 123 I-BMIPP and 3 months following percutaneous coronary intervention using 99m Tc-tetrofosmin to evaluate myocardial salvage and LV systolic/diastolic function. No significant difference was observed in the patient and periprocedural characteristics. Peak CK-MB level was significantly different between the groups (ELCA group, 190.0 [70.5-342.0] IU/L vs. aspiration group, 256.5 [157.0-354.8] IU/L, p = 0.047). Although no significant difference was observed in myocardial salvage, significant improvement in the LV ejection fraction (14.1 [6.2-19.8]% vs. 9.5 [3.9-15.3]%, respectively, p = 0.018) and peak emptying rate (-0.54 [-1.02- (-0.27)] mL/s vs. -0.38 [-0.76- (-0.05)] mL/s, respectively, p = 0.017) were detected. ELCA could suppress the myocardial deviation enzymes and potentially improve systolic function compared to manual aspiration thrombectomy in patients with STEMI., (© 2022. The Author(s), under exclusive licence to Springer Nature B.V.)

Published
2023
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77. Dynamic chest radiography as a novel minimally invasive hemodynamic imaging method in patients with heart failure.

Author

Hiraiwa H, Sakamoto G, Ito R, Koyama Y, Kazama S, Kimura Y, Kondo T, Morimoto R, Okumura T, and Murohara T

Subjects
Male, Humans, Aged, Hemodynamics physiology, Pulmonary Wedge Pressure physiology, Cardiac Catheterization methods, Radiography, Atrial Fibrillation, Heart Failure diagnostic imaging
Abstract

Purpose: Dynamic chest radiography allows for non-invasive cardiopulmonary blood flow assessment. However, data on its use for heart failure hemodynamic assessment are scarce. We utilized dynamic chest radiography to estimate heart failure hemodynamics., Method: Twenty heart failure patients (median age, 67 years; 17 men) underwent dynamic chest radiography and right heart catheterization. The analyzed images were 16-bit images (grayscale range: 0-65,535). Right atrial, right pulmonary artery, and left ventricular apex pixel values (average of the grayscale values of all pixels within a region of interest) were measured. The correlations of the minimum, maximum, mean, amount of change, and rate of change in pixel values with right atrial pressure, pulmonary artery pressure, pulmonary artery wedge pressure, and cardiac index were analyzed., Results: The mean right atrial pixel value and mean right atrial pressure (R = -0.576, P = 0.008), mean right pulmonary artery pixel value and mean pulmonary artery pressure (R = -0.546, P = 0.013), and left ventricular apex pixel value change rate and mean pulmonary artery wedge pressure (R = -0.664, P = 0.001) or cardiac index (R = 0.606, P = 0.005) were correlated. The left ventricular apex pixel value change rate identified low cardiac index (area under the curve, 0.792; 95% confidence interval, 0.590-0.993; P = 0.031) and low cardiac index with high pulmonary artery wedge pressure (area under the curve, 0.902; 95% confidence interval, 0.000-1.000; P = 0.030)., Conclusions: Dynamic chest radiography is a minimally invasive tool for heart failure hemodynamic assessment., Competing Interests: Declaration of Competing Interest The authors declare the following financial interests/personal relationships which may be considered as potential competing interests: T. O. received research grants from Ono Pharmaceutical Co., Ltd., Bayer Pharmaceutical Co., Ltd., Daiichi-Sankyo Pharma Inc., and Amgen Astellas BioPharma K. K. outside of the submitted work. T. O. received honoraria from Ono Pharmaceutical Co., Ltd., Otsuka Pharmaceutical Co., Ltd., and Medtronic Japan Co., Ltd. T. M. received lecture fees from Bayer Pharmaceutical Co., Ltd., Daiichi-Sankyo Co., Ltd., Dainippon Sumitomo Pharma Co., Ltd., Kowa Co., Ltd., MSD K. K., Mitsubishi Tanabe Pharma Co., Nippon Boehringer Ingelheim Co., Ltd., Novartis Pharma K. K., Pfizer Japan Inc., Sanofi-Aventis K. K., and Takeda Pharmaceutical Co., Ltd. T. M. received an unrestricted research grant from the Department of Cardiology, Nagoya University Graduate School of Medicine from Astellas Pharma Inc., Daiichi-Sankyo Co., Ltd., Dainippon Sumitomo Pharma Co., Ltd., Kowa Co., Ltd., MSD K. K., Mitsubishi Tanabe Pharma Co., Nippon Boehringer Ingelheim Co., Ltd., Novartis Pharma K. K., Otsuka Pharma Ltd., Pfizer Japan Inc., Sanofi-Aventis K. K., Takeda Pharmaceutical Co., Ltd., and Teijin Pharma Ltd. The remaining authors have no conflicts of interest to declare., (Copyright © 2023 The Authors. Published by Elsevier B.V. All rights reserved.)

Published
2023
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78. BAP1 is a novel regulator of HIF-1α.

Author

Bononi A, Wang Q, Zolondick AA, Bai F, Steele-Tanji M, Suarez JS, Pastorino S, Sipes A, Signorato V, Ferro A, Novelli F, Kim JH, Minaai M, Takinishi Y, Pellegrini L, Napolitano A, Xu R, Farrar C, Goparaju C, Bassi C, Negrini M, Pagano I, Sakamoto G, Gaudino G, Pass HI, Onuchic JN, Yang H, and Carbone M

Subjects
Humans, Heterozygote, Mutation, Tumor Suppressor Proteins metabolism, Hypoxia-Inducible Factor 1, alpha Subunit genetics, Mesothelioma genetics, Mesothelioma metabolism, Mesothelioma, Malignant genetics, Mesothelioma, Malignant complications, Ubiquitin Thiolesterase metabolism
Abstract

BAP1 is a powerful tumor suppressor gene characterized by haplo insufficiency. Individuals carrying germline BAP1 mutations often develop mesothelioma, an aggressive malignancy of the serosal layers covering the lungs, pericardium, and abdominal cavity. Intriguingly, mesotheliomas developing in carriers of germline BAP1 mutations are less aggressive, and these patients have significantly improved survival. We investigated the apparent paradox of a tumor suppressor gene that, when mutated, causes less aggressive mesotheliomas. We discovered that mesothelioma biopsies with biallelic BAP1 mutations showed loss of nuclear HIF-1α staining. We demonstrated that during hypoxia, BAP1 binds, deubiquitylates, and stabilizes HIF-1α, the master regulator of the hypoxia response and tumor cell invasion. Moreover, primary cells from individuals carrying germline BAP1 mutations and primary cells in which BAP1 was silenced using siRNA had reduced HIF-1α protein levels in hypoxia. Computational modeling and co-immunoprecipitation experiments revealed that mutations of BAP1 residues I675, F678, I679, and L691 -encompassing the C-terminal domain-nuclear localization signal- to A, abolished the interaction with HIF-1α. We found that BAP1 binds to the N-terminal region of HIF-1α, where HIF-1α binds DNA and dimerizes with HIF-1β forming the heterodimeric transactivating complex HIF. Our data identify BAP1 as a key positive regulator of HIF-1α in hypoxia. We propose that the significant reduction of HIF-1α activity in mesothelioma cells carrying biallelic BAP1 mutations, accompanied by the significant reduction of HIF-1α activity in hypoxic tissues containing germline BAP1 mutations, contributes to the reduced aggressiveness and improved survival of mesotheliomas developing in carriers of germline BAP1 mutations.

Published
2023
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79. High Internal Atrial Defibrillation Threshold Is Related to a High Risk of Recurrence After Catheter Ablation for Long-Standing Persistent Atrial Fibrillation But Not for Persistent Atrial Fibrillation.

Author

Imaoka T, Kanzaki Y, Morita Y, Watanabe N, Furui K, Yoshioka N, Shibata N, Yamauchi R, Miyazawa H, Shimojo K, Sakamoto G, Ohi T, Goto H, Mitsuhashi H, and Morishima I

Subjects
Electric Countershock, Heart Atria, Humans, Recurrence, Treatment Outcome, Atrial Fibrillation, Catheter Ablation, Pulmonary Veins
Abstract

Background: The atrial defibrillation threshold (ADFT) for internal cardioversion is theoretically related to the critical mass for sustaining atrial fibrillation (AF)., Objective: This study aimed to investigate the association of ADFT for internal cardioversion with the outcome of catheter ablation for non-paroxysmal AF (non-PAF)., Methods: We included 368 consecutive patients who underwent first-time catheter ablation for non-PAF. Based on the degree of ADFT recorded by the internal cardioversion before pulmonary vein isolation, we divided the patients into low ADFT (<20 J) and high ADFT (≥20 J) groups and analysed the association between ADFT and atrial tachyarrhythmia recurrence., Results: There were 234 and 134 patients in the low and high ADFT groups, respectively. Of these, 39 patients (16.7%) and 41 (30.6%) patients, respectively, had atrial tachyarrhythmia recurrence during the 2.6±1.0 year follow-up. The high ADFT group showed a significantly higher atrial tachyarrhythmia recurrence than the low ADFT group (p=0.002). This finding was also noted in patients with long-standing persistent AF (p=0.032) but not in patients with persistent AF (p=0.159). The significant predictors of arrhythmia recurrence on multivariate analysis were high ADFT (p=0.004) and long-standing persistent AF (p=0.011). In multivariate analysis within the long-standing persistent AF group, only ADFT remained a significant risk factor for AF recurrence (p=0.035)., Conclusions: The high ADFT of internal cardioversion was found to be a risk factor for post-catheter ablation recurrence in patients with long-standing persistent AF but not in those with persistent AF., Competing Interests: Conflicts of Interest There are no conflicts of interest to disclose., (Copyright © 2022. Published by Elsevier B.V.)

Published
2022
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80. Prevalence and prognosis of isolated posterior ST-segment elevation acute myocardial infarction using synthesized-V 7-9 lead.

Author

Shimojo K, Takagi K, Morita Y, Kanzaki Y, Nagai H, Watanabe N, Yoshioka N, Yamauchi R, Komeyama S, Sugiyama H, Imaoka T, Sakamoto G, Ohi T, Goto H, Tsuboi H, and Morishima I

Subjects
Electrocardiography, Humans, Prevalence, Prognosis, Retrospective Studies, Myocardial Infarction epidemiology, Percutaneous Coronary Intervention, ST Elevation Myocardial Infarction diagnosis, ST Elevation Myocardial Infarction surgery
Abstract

Limited data exist on the prevalence and prognosis of isolated posterior ST-segment elevation acute myocardial infarction (STEMI), revealed with a posterior chest lead. Furthermore, the utility of a synthesized-V 7-9 lead in the diagnosis of STEMI is unclear; therefore, we aimed to evaluate its usefulness. We enrolled 142 consecutive patients with STEMI with the culprit lesion on the left circumflex artery (STEMI-LCx) undergoing percutaneous coronary intervention (PCI) between January 2009 and December 2019. We retrospectively checked the ST-segment change of both standard 12-lead and synthesized-V 7-9 lead in all patients with STEMI-LCx. Based on electrocardiogram (ECG) findings, isolated posterior STEMI that was only revealed in synthesized-V 7-9 lead was classified as "STEMI-LCx-synV 7-9 " and the remaining as "STEMI-LCx-12ECG." The prevalence of STEMI-LCx-synV 7-9 in patients with STEMI-LCx was assessed. The incidence of all-cause death, cardiac death, and mechanical complications within 30 days, 3 months, and 1 year was also assessed according to each STEMI-LCx. STEMI-LCx-synV 7-9 and STEMI-LCx-12ECG occurred in 10 (7.0%) and 132 (93.0%) patients, respectively. No significant difference was found in patients' characteristics between the two groups. The patients with STEMI-LCx-synV 7-9 had significantly higher incidences of cardiac death within 3 months and 1 year (30.0% vs. 6.1%, P = 0.031, 30.0% vs. 7.6%, P = 0.050, respectively) and mechanical complications in each follow-up period (20.0% vs. 1.5%, P = 0.025) than those with STEMI-LCx-12ECG. STEMI-LCx-synV 7-9 was observed in 7.0% of the patients with STEMI-LCx. Our findings suggest that the synthesized-V 7-9 lead helps diagnose isolated posterior STEMI and might improve prognosis in patients with STEMI-LCx., (© 2021. Japanese Association of Cardiovascular Intervention and Therapeutics.)

Published
2022
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81. Fifteen-Year Mortality and Cardiac, Thrombotic, and Bleeding Events in Survivors of ST-Elevation Myocardial Infarction.

Author

Takagi K, Tanaka A, Morishima I, Morita Y, Yoshioka N, Yoshida R, Kanzaki Y, Watanabe N, Yamauchi R, Komeyama S, Sugiyama H, Shimojo K, Imaoka T, Sakamoto G, Ohi T, Goto H, Ishii H, and Murohara T

Subjects
Aged, Humans, Risk Factors, Stroke Volume, Survivors, Treatment Outcome, Ventricular Function, Left, Percutaneous Coronary Intervention adverse effects, ST Elevation Myocardial Infarction diagnostic imaging, ST Elevation Myocardial Infarction therapy
Abstract

Background: Although short-term mortality in ST-elevation myocardial infarction (STEMI) has improved, data is limited regarding very long-term mortality and concomitant clinical events in STEMI survivors who undergo primary percutaneous coronary intervention (p-PCI). This study aimed to evaluate these parameters at 15 years and to determine the predictors of 15-year mortality in these patients., Methods: The study endpoints were all-cause mortality and cardiac mortality at 15 years. Independent predictors of all-cause mortality were also analyzed. Furthermore, each thrombotic and bleeding event was evaluated., Results: Between January 2004 and December 2006, 260 STEMI survivors who underwent p-PCI (median follow-up period: 3970 days) were evaluated from the Ogaki Municipal hospital registry. The rates of all-cause mortality (cardiac mortality) at 5, 10, and 15 years were 12.1% (4.9%), 23.4% (9.5%), and 34.9% (12.4%), respectively. The cumulative incidences of recurrent myocardial infarction, target vessel revascularization, ischemic stroke, hemorrhagic bleeding, and gastric bleeding at 15 years were 11.3%, 43.6%, 14.3%, 6.9%, and 10.9%, respectively. Cox regression analysis showed that age ≥ 75 years [adjusted hazard ratio (aHR), 7.074, p < 0.001], chronic kidney disease (aHR, 2.320, p = 0.001), left ventricular ejection fraction <40% (aHR, 2.930, p = 0.001), Killip class ≥II at admission (aHR, 2.639, p = 0.003), untreated chronic total occlusion (aHR, 2.090, p = 0.042), and final TIMI grade ≤ 2 (aHR, 1.736, p = 0.048) were independent predictors of all-cause mortality., Conclusion: This study demonstrated that all-cause and cardiac mortality at 15 years were 34.9% and 12.4%, respectively, in all-comers STEMI survivors after p-PCI, indicating that STEMI survivors might have a benign prognosis., Competing Interests: Declaration of competing interest H.I received lecture fees from Astellas Pharma Inc., Astrazeneca Inc., Daiichi-Sankyo Pharma Inc., and MSD K. K. T.A. received lecture fees from Astellas Pharma, AstraZeneca, Bayer, Daiichi Sankyo, and Bristol-Myers Squibb. T.M received lecture fees from Bayel Pharmaceutical Co., Ltd., Daiichi-Sankyo Co., Ltd., Dainippon Sumitomo Pharma Co., Ltd., Kowa Co., Ltd., MSD K. K., Mitsubishi Tanabe Pharma Co., Nippon Boehringer Ingelheim Co., Ltd., Novartis Pharma K. K., Pfizer Japan Inc., Sanofi-aventis K. K., and Takeda Pharmaceutical Co., Ltd. T.M received unrestricted research grant for Department of Cardiology, Nagoya University Graduate School of Medicine from Astellas Pharma Inc., Daiichi-Sankyo Co., Ltd., Dainippon Sumitomo Pharma Co., Ltd., Kowa Co., Ltd., MSD K. K., Mitsubishi Tanabe Pharma Co., Nippon Boehringer Ingelheim Co., Ltd., Novartis Pharma K. K., Otsuka Pharma Ltd., Pfizer Japan Inc., Sanofi-aventis K. K., Takeda Pharmaceutical Co., Ltd., and Teijin Pharma Ltd. All other authors have reported that they have no relationships relevant to the contents of this paper to disclose., (Copyright © 2021 Elsevier Inc. All rights reserved.)

Published
2022
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82. Assessment of appropriate body mass index cut-off points for long-term mortality among ST-elevation myocardial infarction survivors in Asian population using machine learning algorithm.

Author

Yoshioka N, Takagi K, Tanaka A, Morita Y, Yoshida R, Nagai H, Kanzaki Y, Watanabe N, Yamauchi R, Komeyama S, Sugiyama H, Shimojo K, Imaoka T, Sakamoto G, Ohi T, Goto H, Ishii H, Morishima I, and Murohara T

Subjects
Aged, Algorithms, Asian People, Body Mass Index, Humans, Machine Learning, Male, Registries, Risk Factors, Survivors, Treatment Outcome, Percutaneous Coronary Intervention adverse effects, ST Elevation Myocardial Infarction diagnosis, ST Elevation Myocardial Infarction surgery
Abstract

Low body mass index (BMI) is a predictor of adverse events in patients with ST-elevated myocardial infarction (STEMI) in Western countries. Because the average BMI of Asians is significantly lower than that of the Western population, the appropriate cut-off BMI value and its role in long-term mortality are unclear in Asian patients. Between January 2006 and December 2017, 1215 patients who underwent percutaneous coronary intervention (PCI) for acute STEMI and were alive at discharge (mean age, 67.7 years; male, 75.4%) were evaluated. The cut-off BMI value, which could predict all-cause mortality within 10 years, was detected using a survival classification and regression tree (CART) model. The causes of death according to the BMI value were evaluated in each group. Based on the CART model, the patients were divided into three groups (BMI < 18 kg/m 2 : 54 patients, 18 kg/m 2  ≤ BMI ≤ 20 kg/m 2 : 109 patients, and BMI > 20 kg/m 2 : 1052 patients). The BMI decreased with age; with an increased BMI, patients with dyslipidemia, diabetes mellitus, and smoking habit increased. During the study period (median, 4.9 years), 194 patients (26.8%) died (cardiac death, 59 patients; non-cardiac death, 135 patients). All-cause mortality was more frequent as the BMI decreased (BMI < 18 kg/m 2 ; 72.8%, 18 kg/m 2  ≤ BMI ≤ 20 kg/m 2 ; 40.5%, and BMI > 20 kg/m 2 ; 22.8%; log-rank p < 0.001). Non-cardiac deaths were more frequent than cardiac deaths in all groups, and the dominance of non-cardiac death was highest in the lowest BMI group. Cut-off BMI values of 18 kg/m 2 and 20 kg/m 2 can predict long-term mortality after PCI in Asian STEMI survivors, whose cut-off value is lower than that in the Western populations. The main causes of death in this cohort differed according to the BMI values., (© 2021. Springer Japan KK, part of Springer Nature.)

Published
2022
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83. BAP1 forms a trimer with HMGB1 and HDAC1 that modulates gene × environment interaction with asbestos.

Author

Novelli F, Bononi A, Wang Q, Bai F, Patergnani S, Kricek F, Haglund E, Suarez JS, Tanji M, Xu R, Takanishi Y, Minaai M, Pastorino S, Morris P, Sakamoto G, Pass HI, Barbour H, Gaudino G, Giorgi C, Pinton P, Onuchic JN, Yang H, and Carbone M

Subjects
Animals, Biomarkers, Tumor metabolism, Carcinogenesis, Cell Nucleus metabolism, Female, Gene-Environment Interaction, Germ-Line Mutation, HMGB1 Protein genetics, Heterozygote, Histone Deacetylase 1 genetics, Incidence, Inflammation, Male, Mesothelioma metabolism, Mice, Mutation, Prognosis, Protein Binding, Tumor Suppressor Proteins metabolism, Ubiquitin chemistry, Ubiquitin Thiolesterase metabolism, Asbestos, HMGB1 Protein chemistry, Histone Deacetylase 1 chemistry, Tumor Suppressor Proteins chemistry, Ubiquitin Thiolesterase chemistry
Abstract

Carriers of heterozygous germline BAP1 mutations ( BAP1 +/- ) are affected by the "BAP1 cancer syndrome." Although they can develop almost any cancer type, they are unusually susceptible to asbestos carcinogenesis and mesothelioma. Here we investigate why among all carcinogens, BAP1 mutations cooperate with asbestos. Asbestos carcinogenesis and mesothelioma have been linked to a chronic inflammatory process promoted by the extracellular release of the high-mobility group box 1 protein (HMGB1). We report that BAP1 +/- cells secrete increased amounts of HMGB1, and that BAP1 +/- carriers have detectable serum levels of acetylated HMGB1 that further increase when they develop mesothelioma. We linked these findings to our discovery that BAP1 forms a trimeric protein complex with HMGB1 and with histone deacetylase 1 (HDAC1) that modulates HMGB1 acetylation and its release. Reduced BAP1 levels caused increased ubiquitylation and degradation of HDAC1, leading to increased acetylation of HMGB1 and its active secretion that in turn promoted mesothelial cell transformation., Competing Interests: Competing interest statement: M.C. has a patent issued for BAP1. M.C. and H.Y. have two patents issued for HMGB1. M.C. is a board-certified pathologist who provides consultation for pleural pathology, including medical–legal.

Published
2021
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84. Catheter ablation for non-paroxysmal atrial fibrillation accompanied by heart failure with preserved ejection fraction: feasibility and benefits in functions and B-type natriuretic peptide.

Author

Yamauchi R, Morishima I, Okumura K, Kanzaki Y, Morita Y, Takagi K, Nagai H, Watanabe N, Furui K, Yoshioka N, Miyazawa H, Shimojo K, Imaoka T, Sakamoto G, and Murohara T

Subjects
Feasibility Studies, Humans, Natriuretic Peptide, Brain, Prognosis, Stroke Volume, Atrial Fibrillation diagnosis, Atrial Fibrillation surgery, Catheter Ablation, Heart Failure diagnosis
Abstract

Aims: We aimed to examine the benefits of catheter ablation in patients with non-paroxysmal atrial fibrillation (AF) accompanied by heart failure (HF) with preserved ejection fraction (HFpEF), in comparison with the benefits in patients with AF accompanied by HF with reduced ejection fraction (HFrEF) or patients with no HF., Methods and Results: From 1173 consecutive patients undergoing catheter ablation, 502 with non-paroxysmal AF were divided into three groups: no history of HF [plasma B-type natriuretic peptide (BNP) <100 pg/mL and no HF hospitalization; n = 125], HFpEF [left ventricular (LV) EF ≥50%; n = 293], and HF with midrange EF (HFmrEF) + HFrEF (LVEF <50%; n = 84) groups. The endpoints were AF recurrence at 1 year, changes in symptomatic and image-based functional status, and changes in BNP levels from baseline to 1 year. In the HFpEF group, AF recurred in 48 patients (16.4%) and 278 patients (94.8%) had sinus rhythm at 1 year; these values were comparable with those in the other groups. Significant improvement was observed in the left atrial diameter, LVEF, and New York Heart Association functional class in the HFpEF and HFmrEF + HFrEF groups. The BNP level significantly decreased irrespective of the index rate control status, and freedom from AF recurrence was an independent predictor of HF remission, defined as BNP <100 pg/mL at 1 year, in the HFpEF group., Conclusion: Catheter ablation is highly feasible for restoring sinus rhythm in non-paroxysmal AF with coexisting HFpEF, thereby improving cardiac function and BNP levels. Catheter ablation for AF may be an optional management strategy., (Published on behalf of the European Society of Cardiology. All rights reserved. © The Author(s) 2021. For permissions, please email: journals.permissions@oup.com.)

Published
2021
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85. Ten-Year Mortality in Patients With ST-Elevation Myocardial Infarction.

Author

Watanabe N, Takagi K, Tanaka A, Yoshioka N, Morita Y, Yoshida R, Kanzaki Y, Nagai H, Yamauchi R, Komeyama S, Sugiyama H, Shimojo K, Imaoka T, Sakamoto G, Ohi T, Goto H, Okumura T, Ishii H, Morishima I, and Murohara T

Subjects
Age Factors, Aged, Aged, 80 and over, Body Mass Index, Cause of Death, Female, Follow-Up Studies, Heart Diseases mortality, Hemorrhage mortality, Humans, Infections mortality, Kaplan-Meier Estimate, Male, Middle Aged, Mortality, Multivariate Analysis, Neoplasms mortality, Prognosis, Proportional Hazards Models, Protective Factors, Risk Factors, ST Elevation Myocardial Infarction epidemiology, ST Elevation Myocardial Infarction metabolism, ST Elevation Myocardial Infarction physiopathology, Serum Albumin metabolism, Stroke mortality, Stroke Volume physiology, Atrial Fibrillation epidemiology, Mineralocorticoid Receptor Antagonists therapeutic use, Percutaneous Coronary Intervention, ST Elevation Myocardial Infarction surgery
Abstract

Knowledge of the long-term prognosis (>10 years) and mortality predictors of ST-elevation myocardial infarction (STEMI) patients who have undergone primary percutaneous coronary intervention (p-PCI) is scarce. Therefore, this study evaluated the long-term prognosis and determined the predictors of long-term outcomes for STEMI patients after p-PCI. Between January, 2006 and December, 2010, we collected data and analyzed 459 consecutive patients with acute STEMI who underwent p-PCI and were discharged from the hospital (mean age, 66.8 years; male, 75.2%; peak creatine phosphokinase level, 2,292.5 IU/L). The primary endpoint was 10-year all-cause mortality. The cumulative 10-year incidence of all-cause death was 23.8%. The Cox multivariate regression analysis identified age ≥ 65 years (adjusted hazard ratio [aHR], p <0.001), body mass index (aHR, 0.93, p = 0.033), presence of atrial fibrillation (aHR, 1.69, p = 0.038), mineralocorticoid receptor antagonist use (aHR, 1.95, p = 0.008), ejection fraction <40% (aHR, 2.14, p = 0.005), and albumin <3.5 g/dL (aHR, 2.01, p = 0.005) as independent predictors of all-cause mortality. In conclusion, a post-discharge 10-year survival rate of 76.2% was identified for STEMI patients who underwent p-PCI., (Copyright © 2021 Elsevier Inc. All rights reserved.)

Published
2021
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86. In-hospital mortality among consecutive patients with ST-Elevation myocardial infarction in modern primary percutaneous intervention era ~ Insights from 15-year data of single-center hospital-based registry ~.

Author

Takagi K, Tanaka A, Yoshioka N, Morita Y, Yoshida R, Kanzaki Y, Watanabe N, Yamauchi R, Komeyama S, Sugiyama H, Shimojo K, Imaoka T, Sakamoto G, Ohi T, Goto H, Ishii H, Morishima I, and Murohara T

Subjects
Aged, Female, Hospital Mortality, Hospitals statistics & numerical data, Humans, Male, Middle Aged, Odds Ratio, Percutaneous Coronary Intervention, Retrospective Studies, Risk Assessment, Risk Factors, ST Elevation Myocardial Infarction physiopathology
Abstract

Objective: To clarify the association of detailed angiographic findings with in-hospital outcome after primary percutaneous coronary intervention (p-PCI) for ST-elevation myocardial infarction (STEMI) in Japan., Background: Data regarding the association of detailed angiographic findings with in-hospital outcome after STEMI are limited in the p-PCI era., Methods: Between January-2004 and December-2018, 1735 patients with STEMI (mean age, 68.5 years; female, 24.6%) who presented to the hospital in the 24-hours after symptom onset and underwent p-PCI were evaluated using the disease registries. The registry is an ongoing, retrospective, single-center hospital-based registry., Results: The 30-day mortality rate and in-hospital mortality rate were 7.7% and 9.2%, respectively. Independent predictors of in-hospital mortality were ejection fraction (EF) < 40% [adjusted Odds Ratio (aOR), 4.446, p < 0.001], culprit lesions in the left coronary artery (LCA) (aOR, 2.940, p < 0.001) compared with those in the right coronary artery, Killip class > II (aOR, 7.438; p < 0.001), chronic kidney disease (CKD) (aOR, 4.056; p < 0.001), final thrombolysis in myocardial infarction (TIMI) grades 0/1/2 (aOR, 1.809; p = 0.03), absence of robust collaterals (aOR, 17.309; p = 0.01) and hypertension (aOR, 0.449; p = 0.01)., Conclusions: Among the consecutive patients with STEMI, the in-hospital mortality rate after p-PCI significantly improved in the second half. Not only CKD, Killip class > II, and EF < 40%, but also the angiographic findings such as culprit lesions in the LCA, absence of very robust collaterals, and final TIMI grades <3 were associated with an increased risk of in-hospital mortality., Competing Interests: H.I received lecture fees from Astellas Pharma Inc., Astrazeneca Inc., Daiichi-Sankyo Pharma Inc., and MSD K. K. T.A. received lecture fees from Astellas Pharma, AstraZeneca, Bayer, Daiichi Sankyo, and Bristol-Myers Squibb. T.M received lecture fees from Bayel Pharmaceutical Co., Ltd., Daiichi-Sankyo Co., Ltd., Dainippon Sumitomo Pharma Co., Ltd., Kowa Co., Ltd., MSD K. K., Mitsubishi Tanabe Pharma Co., Nippon Boehringer Ingelheim Co., Ltd., Novartis Pharma K. K., Pfizer Japan Inc., Sanofi-aventis K. K., and Takeda Pharmaceutical Co., Ltd. T.M received an unrestricted research grant for Department of Cardiology, Nagoya University Graduate School of Medicine from Astellas Pharma Inc., Daiichi-Sankyo Co., Ltd., Dainippon Sumitomo Pharma Co., Ltd., Kowa Co., Ltd., MSD K. K., Mitsubishi Tanabe Pharma Co., Nippon Boehringer Ingelheim Co., Ltd., Novartis Pharma K. K., Otsuka Pharma Ltd., Pfizer Japan Inc., Sanofi-aventis K. K., Takeda Pharmaceutical Co., Ltd., and Teijin Pharma Ltd. All other authors have reported that they have no relationships relevant to the contents of this paper to disclose.

Published
2021
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87. The Mid-term Mortality and Mode of Death in Survivors with ST-elevation Myocardial Infarction.

Author

Yoshioka N, Takagi K, Tanaka A, Morita Y, Yoshida R, Kanzaki Y, Nagai H, Watanabe N, Yamauchi R, Komeyama S, Sugiyama H, Shimojo K, Imaoka T, Sakamoto G, Ohi T, Goto H, Ishii H, Morishima I, and Murohara T

Subjects
Aged, Aged, 80 and over, Hospital Mortality, Humans, Male, Proportional Hazards Models, Risk Factors, Survivors, Treatment Outcome, Percutaneous Coronary Intervention, ST Elevation Myocardial Infarction surgery
Abstract

Objective The popularity of primary percutaneous coronary intervention (p-PCI) for ST-elevation myocardial infarction (STEMI) has increased over the past decades. Despite improvements in in-hospital mortality rates, it is clinically important to investigate the prognoses after discharge. However, data on the mode of death and prognostic factors are limited. We analyzed these factors in a Japanese cohort in the modern p-PCI era. Methods Between January 2004 and December 2017, a total of 1,222 patients who underwent p-PCI within 24 hours from the onset of STEMI and were alive at discharge (mean age, 67.7 years old; men, 75.5%), were evaluated. The two-year mortality was analyzed using a Cox regression model, and the mode of death was evaluated. Results The rate of mortality at 2 years was 5.7%. Non-cardiac death was more frequent than cardiac death (62.6% vs. 37.4%). A Cox multivariate analysis identified the following as independent predictors of the 2-year mortality: hemoglobin (log-transformed) [adjusted hazard ratio (HR), 0.048; 95% confidence interval (CI), 0.008-0.29; p<0.001], age above 80 years old (adjusted HR, 2.26; 95% CI, 1.30-3.91; p=0.004), Killip class ≥II (adjusted HR, 1.99; 95% CI, 1.17-3.39; p=0.011), brain natriuretic peptide level (log-transformed) (adjusted HR, 1.47; 95% CI, 1.09-2.01; p=0.013), and body mass index (log-transformed) (adjusted HR, 0.16; 95% CI, 0.030-0.84; p=0.030). Conclusion This study demonstrated that the 2-year mortality was 5.7% in STEMI survivors after p-PCI. Non-cardiac death was more frequent than cardiac death. Compared to well-known clinical variables, angiographic findings did not have a significant influence on the mid-term mortality.

Published
2021
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88. Maternal separation as a risk factor for aggravation of neuropathic pain in later life in mice.

Author

Mizoguchi H, Fukumoto K, Sakamoto G, Jin S, Toyama A, Wang T, Suzumura A, and Sato J

Subjects
Animals, Animals, Newborn, Antidepressive Agents, Second-Generation therapeutic use, Calcium-Binding Proteins, Disease Models, Animal, Fluoxetine therapeutic use, Food Preferences drug effects, Glial Fibrillary Acidic Protein metabolism, Hyperalgesia physiopathology, Male, Maze Learning drug effects, Mice, Mice, Inbred ICR, Microfilament Proteins, Microglia metabolism, Mood Disorders drug therapy, Mood Disorders pathology, Spinal Cord drug effects, Spinal Cord pathology, Sucrose administration & dosage, Swimming psychology, Maternal Deprivation, Mood Disorders etiology, Neuralgia complications, Neuralgia psychology
Abstract

Psychological stresses such as social loss and separation during childhood induce hardship, referred to as emotional pain. These experiences are well-documented risk factors for the development of physical pain in adulthood. However, the underlying neuronal mechanisms of this exacerbation of pain are largely unknown, and consequently there is no effective pharmacotherapy. In this study, we sought to determine whether infant maternal separation (MS) contributes to aggravation of neuropathic pain in adult mice. MS increased anxiety- and depression-like behavioral responses to adult stress. In MS animals, chronic constriction injury (CCI) heightened the sensory dimension of chronic pain relative to that of control mice. However, MS mice treated with fluoxetine for 4 weeks after MS did not exhibit augmentation of allodynia, and their emotional response was attenuated. Microglia were more abundant in the spinal cord in MS/CCI mice than in control/CCI mice. These results suggest that emotional impairment is related to augmentation of neuropathic pain, and that dysfunction of microglial activation contributes to heightened pain sensitivity., (Copyright © 2018 Elsevier B.V. All rights reserved.)

Published
2019
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89. Germline BAP1 mutations induce a Warburg effect.

Author

Bononi A, Yang H, Giorgi C, Patergnani S, Pellegrini L, Su M, Xie G, Signorato V, Pastorino S, Morris P, Sakamoto G, Kuchay S, Gaudino G, Pass HI, Napolitano A, Pinton P, Jia W, and Carbone M

Subjects
Germ Cells metabolism, Heterozygote, Humans, Mitochondria metabolism, Skin pathology, Mitochondria genetics, Mutation genetics, Tumor Suppressor Proteins genetics, Ubiquitin Thiolesterase genetics
Abstract

Carriers of heterozygous germline BAP1 mutations (BAP1 +/- ) develop cancer. We studied plasma from 16 BAP1 +/- individuals from 2 families carrying different germline BAP1 mutations and 30 BAP1 wild-type (BAP1 WT ) controls from these same families. Plasma samples were analyzed by liquid chromatography time-of-flight mass spectrometry (LC-TOF-MS), ultra-performance liquid chromatography triple quadrupole mass spectrometry (UPLC-TQ-MS), and gas chromatography time-of-flight mass spectrometry (GC-TOF-MS). We found a clear separation in the metabolic profile between BAP1 WT and BAP1 +/- individuals. We confirmed the specificity of the data in vitro using 12 cell cultures of primary fibroblasts we derived from skin punch biopsies from 12/46 of these same individuals, 6 BAP1 +/- carriers and 6 controls from both families. BAP1 +/- fibroblasts displayed increased aerobic glycolysis and lactate secretion, and reduced mitochondrial respiration and ATP production compared with BAP1 WT . siRNA-mediated downregulation of BAP1 in primary BAP1 WT fibroblasts and in primary human mesothelial cells, led to the same reduced mitochondrial respiration and increased aerobic glycolysis as we detected in primary fibroblasts from carriers of BAP1 +/- mutations. The plasma and cell culture results were highly reproducible and were specifically and only linked to BAP1 status and not to gender, age or family, or cell type, and required an intact BAP1 catalytic activity. Accordingly, we were able to build a metabolomic model capable of predicting BAP1 status with 100% accuracy using data from human plasma. Our data provide the first experimental evidence supporting the hypothesis that aerobic glycolysis, also known as the 'Warburg effect', does not necessarily occur as an adaptive process that is consequence of carcinogenesis, but rather that it may also predate malignancy by many years and facilitate carcinogenesis.

Published
2017
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90. BAP1 regulates IP3R3-mediated Ca 2+ flux to mitochondria suppressing cell transformation.

Author

Bononi A, Giorgi C, Patergnani S, Larson D, Verbruggen K, Tanji M, Pellegrini L, Signorato V, Olivetto F, Pastorino S, Nasu M, Napolitano A, Gaudino G, Morris P, Sakamoto G, Ferris LK, Danese A, Raimondi A, Tacchetti C, Kuchay S, Pass HI, Affar EB, Yang H, Pinton P, and Carbone M

Subjects
Apoptosis genetics, Asbestos toxicity, Calcium Signaling, Cell Nucleus metabolism, Cell Survival, Cells, Cultured, DNA Damage, Epithelium, Fibroblasts, Gene-Environment Interaction, Humans, Protein Binding, Protein Stability, Tumor Suppressor Proteins deficiency, Tumor Suppressor Proteins genetics, Ubiquitin metabolism, Ubiquitin Thiolesterase deficiency, Ubiquitin Thiolesterase genetics, Calcium metabolism, Cell Transformation, Neoplastic drug effects, Cell Transformation, Neoplastic radiation effects, Cytoplasm metabolism, Endoplasmic Reticulum metabolism, Inositol 1,4,5-Trisphosphate Receptors metabolism, Mitochondria metabolism, Tumor Suppressor Proteins metabolism, Ubiquitin Thiolesterase metabolism
Abstract

BRCA1-associated protein 1 (BAP1) is a potent tumour suppressor gene that modulates environmental carcinogenesis. All carriers of inherited heterozygous germline BAP1-inactivating mutations (BAP1 +/- ) developed one and often several BAP1 -/- malignancies in their lifetime, mostly malignant mesothelioma, uveal melanoma, and so on. Moreover, BAP1-acquired biallelic mutations are frequent in human cancers. BAP1 tumour suppressor activity has been attributed to its nuclear localization, where it helps to maintain genome integrity. The possible activity of BAP1 in the cytoplasm is unknown. Cells with reduced levels of BAP1 exhibit chromosomal abnormalities and decreased DNA repair by homologous recombination, indicating that BAP1 dosage is critical. Cells with extensive DNA damage should die and not grow into malignancies. Here we discover that BAP1 localizes at the endoplasmic reticulum. Here, it binds, deubiquitylates, and stabilizes type 3 inositol-1,4,5-trisphosphate receptor (IP3R3), modulating calcium (Ca 2+ ) release from the endoplasmic reticulum into the cytosol and mitochondria, promoting apoptosis. Reduced levels of BAP1 in BAP1 +/- carriers cause reduction both of IP3R3 levels and of Ca 2+ flux, preventing BAP1 +/- cells that accumulate DNA damage from executing apoptosis. A higher fraction of cells exposed to either ionizing or ultraviolet radiation, or to asbestos, survive genotoxic stress, resulting in a higher rate of cellular transformation. We propose that the high incidence of cancers in BAP1 +/- carriers results from the combined reduced nuclear and cytoplasmic activities of BAP1. Our data provide a mechanistic rationale for the powerful ability of BAP1 to regulate gene-environment interaction in human carcinogenesis.

Published
2017
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91. Synchronous bilateral neuroendocrine ductal carcinoma in situ.

Author

Honami H, Sotome K, Sakamoto G, Iri H, Tanaka Y, Fukamachi S, and Morozumi K

Subjects
Breast Neoplasms surgery, Carcinoma, Intraductal, Noninfiltrating surgery, Carcinoma, Neuroendocrine surgery, Female, Humans, Middle Aged, Neoplasm Staging, Neoplasms, Multiple Primary surgery, Prognosis, Breast Neoplasms diagnosis, Carcinoma, Intraductal, Noninfiltrating diagnosis, Carcinoma, Neuroendocrine diagnosis, Neoplasms, Multiple Primary diagnosis
Abstract

Neuroendocrine ductal carcinoma in situ (NE-DCIS) is a breast malignancy that has characteristic clinicopathological features and can, therefore, be regarded as a distinct variant of DCIS. The patient was a 54-year-old premenopausal woman with hemorrhagic nipple discharge in her left breast. Magnetic resonance imaging and ultrasound (US) images of the left breast showed mass-like lesions, while concurrent images of the right breast showed non-mass-like lesions. These findings suggested the presence of both benign and malignant tumors. Pathological findings from US-guided core-needle biopsy of the left mass were highly suspicious of a malignant tumor. Excisional biopsy of both breasts was performed. We could define the diagnosis of breast cancer by the second opinion on pathological diagnosis. The tumor cells showed histological characteristics of NE-DCIS. Bilateral breast lesions had histopathological similarities and were composed of predominantly solid growth of carcinoma cells, frequently with well-developed vascular structures, in mammary ducts and ductules. Carcinoma cells were polygonal or occasionally spindle shaped and had fine-granular, relatively eosinophilic cytoplasm. The nuclei of these cells showed round to ovoid in shape and fine-granular chromatin pattern. There was not any invasive component, as confirmed by careful histological examination. Thus, additional immunohistochemical stainings for NE markers (chromogranin A and synaptophysin) were performed. Staining statuses of these markers were positive in almost all tumor cells from both breasts. Both tumors were therefore diagnosed as NE-DCIS. To our knowledge, this case is the first report of NE-DCIS diagnosed synchronously in both breasts.

Published
2014
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92. Metachronous secondary primary occult breast cancer initially presenting with metastases to the contralateral axillary lymph nodes: report of a case.

Author

Kinoshita S, Hirano A, Kobayashi S, Komine K, Kyoda S, Takeyama H, Uchida K, Morikawa T, Nagase J, and Sakamoto G

Subjects
Adenocarcinoma drug therapy, Adenocarcinoma surgery, Axilla, Breast Neoplasms drug therapy, Breast Neoplasms surgery, Carcinoma, Ductal, Breast drug therapy, Carcinoma, Ductal, Breast surgery, Combined Modality Therapy, Female, Humans, Lymphatic Metastasis, Mastectomy, Segmental, Middle Aged, Adenocarcinoma secondary, Breast Neoplasms pathology, Carcinoma, Ductal, Breast secondary, Neoplasms, Second Primary pathology
Abstract

We report a rare case of a 64-year-old female with metachronous secondary primary left occult breast cancer initially presenting right axillary lymph node metastases. The patient, who had received breast-conserving therapy for left breast cancer at another hospital about 4.5 years ago, came to our hospital complaining of right axillary node swelling. After both breast and systemic examination, she received complete right axillary lymph node dissection. Just after the operation, she was diagnosed with right occult breast cancer by a review of the right axillary lymph nodes and previous left breast cancer. She was followed by radiation and systemic chemoendocrine therapies. One year after axillary lymph node dissection, mammography and ultrasonography showed a new lesion in her left breast. Core needle biopsy revealed similar findings to right axillary lymph node metastasis. After salvage surgery, the diagnosis was revised. We recommend that patients without clinical findings except for axillary lymph node metastasis, especially post-breast-conserving surgery followed by radiation therapy, should be considered not only as having ipsilateral but also contralateral occult breast cancer. If there is no evidence of a primary lesion, axillary lymph node dissection needs to be carried out, and the patient should be offered the choice of radiation therapy or mastectomy followed by proper systemic therapy.

Published
2010
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93. Alternative use of multiple exons 1 of aromatase gene in cancerous and normal breast tissues from women over the age of 80 years.

Author

Honma N, Takubo K, Sawabe M, Arai T, Akiyama F, Sakamoto G, Utsumi T, Yoshimura N, and Harada N

Subjects
Adult, Aged, Breast Neoplasms epidemiology, Breast Neoplasms genetics, Carcinoma enzymology, Carcinoma epidemiology, Carcinoma genetics, Carcinoma, Ductal, Breast epidemiology, Carcinoma, Ductal, Breast genetics, Estrogens, Female, Gene Expression Regulation, Neoplastic, Humans, Isoenzymes genetics, Japan epidemiology, Middle Aged, Neoplasms, Hormone-Dependent enzymology, Neoplasms, Hormone-Dependent epidemiology, Neoplasms, Hormone-Dependent genetics, RNA, Messenger genetics, RNA, Neoplasm genetics, Receptors, Estrogen analysis, Aged, 80 and over physiology, Alternative Splicing, Aromatase genetics, Breast enzymology, Breast Neoplasms enzymology, Carcinoma, Ductal, Breast enzymology, Exons genetics, Neoplasm Proteins genetics
Abstract

Introduction: Peripherally localized aromatase, which converts circulating androgens into estrogens, is important in the pathogenesis of postmenopausal breast carcinomas. We have previously shown that aromatase mRNA levels are higher in elderly breast carcinomas (EldCa) than breast carcinomas of the control group (ContCa) or normal breast tissues. Aromatase expression has been reported to be regulated through the alternative use of multiple exons 1 (exons 1a-1f and so on); however, the preferential usage of exons 1 in elderly breast tissue has never been systematically examined. In order to properly treat and protect against EldCa, the regulation mechanism of aromatase expression in elderly breast tissues should be elucidated. The aim of the present study is to elucidate whether there are any specific patterns in use of multiple exons 1 in elderly breast tissue., Methods: Usage of multiple exons 1 of the aromatase gene and mRNA levels of aromatase were examined by reverse transcription-polymerase chain reaction analysis in breast tissues of 38 elderly patients with breast cancer (age 80-99), and the results were compared with those in 35 patients of the control group (age 37-70). One-factor analysis of variance and the Scheffé test were used for the comparison of aromatase mRNA levels. Patterns of preferential utilization of multiple exons 1 of the aromatase gene were compared by chi2 test for independence or Fisher exact test for independence using a contingency table., Results: Exon 1d was utilized much more frequently in elderly tissue than in the control group irrespective of cancerous or normal tissue (EldCa, 36/38, 95% versus ContCa, 7/35, 20%, P < 0.0001; normal tissue of the elderly, EldNorm, 30/34, 88% versus normal tissue of controls, ContNorm, 2/29, 7%, P < 0.0001). Twenty EldCa (53%) and 12 EldNorm (35%) used both exons 1c and 1d; however, their dominance was reversed (EldCa, all 1d > 1c; EldNorm, all 1c > 1d)., Conclusions: Elderly breast tissues exhibited specific patterns in use of multiple exons 1, which at least partly explained the higher aromatase levels in EldCa. The mechanisms of how these specific patterns occur during aging and carcinogenesis should be further examined.

Published
2009
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94. Estrogen receptor-beta status influences clinical outcome of triple-negative breast cancer.

Author

Sakamoto G and Honma N

Subjects
Breast Neoplasms pathology, Female, Humans, Treatment Outcome, Breast Neoplasms metabolism, Breast Neoplasms therapy, Estrogen Receptor beta metabolism, Receptor, ErbB-2 metabolism, Receptors, Progesterone metabolism
Abstract

In this conference paper, we show that estrogen receptor-beta status influences clinical outcome of triple-negative breast cancer referring two of our recent research articles.

Published
2009
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95. What causes discrepancies in HER2 testing for breast cancer? A Japanese ring study in conjunction with the global standard.

Author

Umemura S, Osamura RY, Akiyama F, Honma K, Kurosumi M, Sasano H, Toyoshima S, Tsuda H, Rüschoff J, and Sakamoto G

Subjects
Antibodies, Monoclonal therapeutic use, Antibodies, Monoclonal, Humanized, Breast Neoplasms drug therapy, Female, Genes, erbB-2, Humans, Immunohistochemistry methods, In Situ Hybridization, Fluorescence methods, In Situ Hybridization, Fluorescence standards, Japan, Observer Variation, Practice Guidelines as Topic, Receptor, ErbB-2 biosynthesis, Reproducibility of Results, Trastuzumab, Breast Neoplasms diagnosis, Receptor, ErbB-2 analysis
Abstract

We assessed interinstitutional and interobserver consistency of human epidermal growth factor receptor type-2 (HER2) testing using immunohistochemical analysis and fluorescence in situ hybridization (FISH) in a set of 20 breast cancer samples among 10 institutions in Japan and a Herceptin adjuvant study participating laboratory in Germany and identified factors that may lead to discordant results.We found a good agreement in immunohistochemical HER2 scoring between the coordinating institution and 10 participating laboratories (kappa = 0.718) and excellent agreement for FISH (kappa = 0.900). The results of a comparison between 10 Japanese laboratories and the German laboratory was good for immunohistochemical studies (kappa = 0.713) and excellent for FISH (kappa = 0.887). FISH retesting of equivocal samples (2+ immunohistochemically) improved agreement. Discrepancies between results were attributed to the evaluation process in 33.0% of the samples, staining procedures in 25.0%, and a combination of the two in 41.7%. Evaluation of samples according to the American Society of Clinical Oncology/College of American Pathologists guideline increased the number of 2+ immunohistochemical scores. By performing FISH retesting for these samples, consistency among multiple institutions could be archived. The quality of the staining procedures performed and the consistency of evaluations require regular assessment.

Published
2008
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96. Oestrogen receptor-beta1 but not oestrogen receptor-betacx is of prognostic value in apocrine carcinoma of the breast.

Author

Honma N, Saji S, Kurabayashi R, Aida J, Arai T, Horii R, Akiyama F, Iwase T, Harada N, Younes M, Toi M, Takubo K, and Sakamoto G

Subjects
Adult, Aged, Aged, 80 and over, Breast Neoplasms metabolism, Breast Neoplasms pathology, Carcinoma, Ductal, Breast metabolism, Carcinoma, Ductal, Breast pathology, Disease-Free Survival, Female, Humans, Immunohistochemistry, Middle Aged, Prognosis, Protein Isoforms biosynthesis, Young Adult, Biomarkers, Tumor biosynthesis, Breast Neoplasms mortality, Carcinoma, Ductal, Breast mortality, Estrogen Receptor beta biosynthesis
Abstract

Apocrine carcinoma of the breast, which frequently expresses oestrogen receptor-beta (ER-beta) in the absence of ER-alpha and only infrequently is treated endocrinologically, gives an opportunity to investigate the clinicopathological role of ER-beta in breast cancer independent of ER-alpha expression or tamoxifen treatment. Several isotypes of ER-beta, ER-beta1-5 etc., have been identified thus far; however, the clinicopathological importance of each ER-beta isotype in breast cancer is still uncertain. Here we aimed to clarify the clinicopathological importance of ER-beta1 and ER-betacx (ER-beta2) in apocrine carcinomas, immunohistochemically examining expressions of ER-beta1 and ER-betacx in 47 apocrine carcinomas. Positivity for ER-beta1 and ER-betacx was observed in 41 (87%) and 18 (38%) of 47 cases, respectively. ER-beta1 positivity was related to smaller tumor size (P=0.0359), lower histological grade (P=0.0322), and higher disease-free survival (P<0.0001), whereas ER-betacx status was related to none of these parameters. ER-beta1 positivity was also associated with favorable clinical outcome in 24 so-called triple-negative (ER-alpha-negative/PR-negative/HER2-negative) apocrine carcinomas. ER-beta1 itself, independent of ER-alpha expression and tamoxifen treatment, seems to have a tumor-suppressive effect, at least in apocrine carcinomas. Further study of ER-beta1 is desired to optimize breast cancer treatment.

Published
2008
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97. Predictors of peritumoral edema after stereotactic radiosurgery of supratentorial meningiomas.

Author

Patil CG, Hoang S, Borchers DJ 3rd, Sakamoto G, Soltys SG, Gibbs IC, Harsh GR 4th, Chang SD, and Adler JR Jr

Subjects
Adult, Aged, Aged, 80 and over, Brain Edema diagnosis, Female, Humans, Male, Meningeal Neoplasms diagnosis, Meningioma diagnosis, Middle Aged, Postoperative Complications diagnosis, Postoperative Complications etiology, Predictive Value of Tests, Retrospective Studies, Risk Factors, Supratentorial Neoplasms diagnosis, Young Adult, Brain Edema etiology, Meningeal Neoplasms surgery, Meningioma surgery, Radiosurgery adverse effects, Supratentorial Neoplasms surgery
Abstract

Objective: Anecdotal evidence suggests that radiosurgical ablation of parasagittal meningiomas may be associated with increased risk of subsequent edema. Potential predictors of postradiosurgical peritumoral edema, including parasagittal tumor location, tumor size, and treatment dose, were evaluated., Methods: We retrospectively reviewed records of 102 patients with 111 supratentorial meningiomas treated with CyberKnife (Accuray, Inc., Sunnyvale, CA) stereotactic radiosurgery (SRS). A median marginal dose of 18.0 Gy (range, 11.3-25.0 Gy) was delivered in 1 to 5 sessions (fractions). Potential predictors of posttreatment symptomatic edema were evaluated using Fisher's exact test., Results: Of the 102 patients followed for a mean of 20.9 months (range, 6-77 mo), 15 (14.7%) developed symptomatic edema after SRS. Nine of 31 with parasagittal meningiomas (29.0%) and 6 of 80 with nonparasagittal supratentorial meningiomas (7.5%) developed symptomatic edema (P = 0.0053). Compared with patients with meningiomas in nonmidline supratentorial locations, patients with parasagittal meningiomas were more than 4 times as likely to develop symptomatic edema after SRS (odds ratio, 4.1; 95% confidence interval, 1.5-11.5). The 6-, 12-, and 18-month actuarial rates of symptomatic edema development were significantly greater for patients with parasagittal meningiomas than for patients with nonparasagittal meningiomas (17.8 versus 1.3%, 25.4 versus 5.8%, and 35.2 versus 7.8%, respectively)., Conclusion: Patients with parasagittal meningiomas are at greater risk of developing peritumoral symptomatic edema after SRS. Close follow-up after SRS may be particularly important in such patients. These results highlight the need to pursue strategies that could decrease the incidence of postradiosurgical edema in patients with parasagittal meningioma.

Published
2008
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98. Histopathological assessment of anastrozole and tamoxifen as preoperative (neoadjuvant) treatment in postmenopausal Japanese women with hormone receptor-positive breast cancer in the PROACT trial.

Author

Kurosumi M, Takatsuka Y, Watanabe T, Imoto S, Inaji H, Tsuda H, Akiyama F, Sakamoto G, Ikeda T, and Noguchi S

Subjects
Aged, Aged, 80 and over, Anastrozole, Breast Neoplasms chemistry, Breast Neoplasms pathology, Double-Blind Method, Female, Humans, Middle Aged, Postmenopause, Antineoplastic Agents, Hormonal therapeutic use, Breast Neoplasms drug therapy, Nitriles therapeutic use, Receptors, Estrogen analysis, Receptors, Progesterone analysis, Tamoxifen therapeutic use, Triazoles therapeutic use
Abstract

Purpose: The PReOperative 'Arimidex' Compared with Tamoxifen (PROACT) trial compared neoadjuvant anastrozole and tamoxifen in postmenopausal women with large, operable or potentially operable, locally advanced hormone receptor-positive breast cancer. Here, we compare objective clinical responses with histopathological tumor responses to therapy in a cohort of 97 Japanese patients, in order to investigate the consistency of assessment methods and the change in estrogen-receptor (ER) and progesterone-receptor (PgR) status., Methods: Histopathological response and the change in ER and PgR status were assessed by comparing pathological specimens collected at baseline (via needle biopsy) with those collected at 3 months (from excised tumors). The response was evaluated using Pathological Response Criteria for Breast Cancer as defined by the Japanese Breast Cancer Society. The patients were randomized to receive anastrozole (n = 48) or tamoxifen (n = 49)., Results: A numerically greater histopathological response rate was observed when neoadjuvant anastrozole compared with neoadjuvant tamoxifen (35.4 and 12.2%, respectively). The histopathological and clinical objective response rates agreed in 63/97 patients. The ER status of 5/40 patients changed from positive at baseline to negative at 3 months in the anastrozole group compared with 20/37 patients in the tamoxifen group. The PgR status of 16/17 patients in the anastrozole group and of 1/11 patients in the tamoxifen group changed from positive to negative., Conclusions: These data support the findings of the main PROACT trial, which confirmed that anastrozole, as compared with tamoxifen, is an effective neoadjuvant endocrine treatment in objective response rates for postmenopausal women with large operable hormone-receptor positive breast cancer. Further follow-up is required to confirm whether histopathological responses to therapy correlate with an overall improvement in survival.

Published
2008
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99. Increased rate of hair regrowth in mice with constitutive overexpression of Del1.

Author

Hsu GP, Mathy JA, Wang Z, Xia W, Sakamoto G, Kundu R, Longaker MT, Quertermous T, and Yang GP

Subjects
Animals, Calcium-Binding Proteins, Carrier Proteins genetics, Cell Adhesion Molecules, Hair Follicle metabolism, Hair Follicle pathology, Intercellular Signaling Peptides and Proteins, Keratinocytes metabolism, Keratinocytes pathology, Lac Operon genetics, Mice, Mice, Transgenic, Skin blood supply, Skin pathology, Wound Healing genetics, Wound Healing physiology, Carrier Proteins metabolism, Hair growth & development, Hair metabolism
Abstract

Background: Developmental endothelial locus (Del)1 is a secreted extracellular matrix-associated protein that stimulates angiogenesis through integrin binding and is implicated in vasculogenesis. We hypothesized that increased expression of an angiogenic factor would lead to enhanced wound healing., Materials and Methods: Transgenic mice had Del1 cloned behind a keratin 14 promoter (K14-Del1) to drive constitutive expression in basal keratinocytes. Transgenic animals and wild-type litter mates underwent excisional wounding or depilation, and tissues were harvested at various time points. Wound healing and hair regrowth were assessed by photography, histology, and immunohistochemistry. For injection experiments, purified Del1 protein was injected in the flanks of wild-type mice with carrier on the contralateral flank as a control. Del1 expression during hair development was performed using transgenic mice with a LacZ cassette introduced downstream from the native promoter., Results: K14-Del1 animals appeared normal and healed excisional wounds normally but demonstrated an increased rate of hair regrowth after wound healing. Using depilation experiments to specifically address hair follicle growth, we found increased hair regrowth was independent of wounding. This was confirmed by injection of purified Del1 protein. During normal hair anagenesis, Del1 is expressed in the root of the hair follicle., Conclusions: Constitutive expression of Del1 in skin does not affect skin vascularity or improve wound healing. Surprisingly, we found the primary effect of constitutive Del1 expression in the basal keratinocytes was increased hair growth following induction of anagenesis. During normal hair anagenesis, we see expression of Del1 in the root of the hair follicle suggesting it may function there to stimulate hair growth.

Published
2008
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100. Clinicopathological study of breast tissue in female-to-male transsexuals.

Author

Kuroda H, Ohnisi K, Sakamoto G, and Itoyama S

Subjects
Adolescent, Adult, Carcinoma, Intraductal, Noninfiltrating pathology, Female, Humans, Hyperplasia, Middle Aged, Transsexualism drug therapy, Young Adult, Androgens pharmacology, Breast drug effects, Breast pathology, Transsexualism pathology
Abstract

Purpose: Gender identity disorder is defined as persistent feelings of gender discomfort and the inappropriateness of one's anatomical sex. To study the effects of androgens on female breast tissue, we examined mammary glands from female-to-male transsexual (FTMT) women using androgen therapy and from those not using androgen therapy., Methods: Female-to-male transsexual breast tissue is a rare specimen in surgical pathology and there are no well-defined guidelines for its examination. We evaluated the clinicopathologic findings of 186 FTMT mammary glands., Results: The patients' ages at presentation ranged from 18 to 49 years (mean 27.4 years). We detected breast carcinoma in only 1 of 130 FTMT women who had not used androgen therapy and in none of 56 FTMT women who had used androgen therapy., Conclusion: We described the histopathological morphology of FTMT breast tissue. The frequency of carcinoma and hyperplasia did not differ significantly between FTMT women who had used androgen therapy and those who had not. These findings suggest that androgen does not alter the risk of carcinoma developing in the mammary glands of FTMT women.

Published
2008
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