218 results on '"Nakagawa, Pablo"'
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52. Renal release ofN-acetyl-seryl-aspartyl-lysyl-proline is part of an antifibrotic peptidergic system in the kidney
53. β-Arrestin-Biased Agonist Targeting the Brain AT1R (Angiotensin II Type 1 Receptor) Increases Aversion to Saline and Lowers Blood Pressure in Deoxycorticosterone Acetate-Salt Hypertension.
54. Abstract 094: Smooth Muscle PPARγ Mutation Causes Impaired Renal Blood Flow and Salt-Sensitive Hypertension
55. Abstract 104: DOCA Induced Natriorexigenic and Hypertensive Effects are Blunted by Central Infusion of AT1 Receptor Biased Agonist TRV120027
56. Abstract P204: Induction of Endoplasmic Reticulum Stress Upregulates Renin Expression in Immortalized Mouse Hypothalamic N43/5 Cells
57. Abstract P201: Exposure to Stress and Low Salt Diet Increases Blood Pressure in Mice Lacking the Brain-Specific Renin Isoform Renin-b
58. Smooth Muscle PPARγ Mutation Causes Impaired Renal Blood Flow and Salt‐Sensitive Hypertension
59. Ac-SDKP decreases mortality and cardiac rupture after acute myocardial infarction
60. Abstract 099: Smooth Muscle PPARγ Mutation Causes Salt-sensitive Hypertension
61. Abstract 087: Deletion of Brain-specific Isoform of Renin (renin-b) Increases Resting Metabolic Rate by Stimulating Thermogenic Sympathetic Nerve Activity
62. Abstract P283: Effects of High Salt Diet on Vascular Function and Renal Injury in a Novel Mouse Model of Neurogenic Hypertension
63. How Is the Brain Renin–Angiotensin System Regulated?
64. Deletion of brain‐specific isoform of renin (renin‐b) increases resting metabolic rate by stimulating brown adipose tissue sympathetic nerve activity
65. Smooth Muscle PPARγ Mutation Causes Salt‐sensitive Hypertension
66. Effects of N -acetyl-seryl-asparyl-lysyl-proline on blood pressure, renal damage, and mortality in systemic lupus erythematosus
67. Cardiac-deleterious role of galectin-3 in chronic angiotensin II-induced hypertension
68. Renal release of N-acetyl-seryl-aspartyl-lysyl-proline is part of an antifibrotic peptidergic system in the kidney.
69. Abstract 028: Effects of N-acetyl-seryl-aspartyl-lysyl-proline on Inflammatory Cells During the Acute Stage of Myocardial Infarction
70. Abstract P211: N-Acetyl-Seryl-Aspartyl-Lysyl-Proline (Ac-SDKP) Promotes Tube Formation in Human Coronary Artery Endothelial Cells
71. The anti-inflammatory peptide Ac-SDKP is released from thymosin-β4 by renal meprin-α and prolyl oligopeptidase
72. Ac-SDKP suppresses TNF-α-induced ICAM-1 expression in endothelial cells via inhibition of IκB kinase and NF-κB activation
73. The Anti‐Inflammatory Peptide Ac‐SDKP is Released from Thymosin β4 by Meprin α and Prolyl Oligopeptidase
74. Inhibition of neutrophil chemotaxis by N‐Acetyl‐Seryl‐Aspartyl‐Lysyl‐Proline and thymosin β4
75. Renal Protective Effect of N -Acetyl-Seryl-Aspartyl-Lysyl-Proline in Dahl Salt-Sensitive Rats
76. Selective Deletion of Renin-b in the Brain Alters Drinking and Metabolism.
77. Angiotensin AT1Areceptor signal switching in Agouti-related peptide neurons mediates metabolic rate adaptation during obesity
78. N-Acetyl-Seryl-Aspartyl-Lysyl-Proline: mechanisms of renal protection in mouse model of systemic lupus erythematosus
79. N‐Acetyl‐Seryl‐Aspartyl‐Lysyl‐Proline (Ac‐SDKP) Delays the Development of Hypertension and Renal Damage in Systemic Lupus Erythematosus (SLE)
80. Deletion of interleukin-6 prevents cardiac inflammation, fibrosis and dysfunction without affecting blood pressure in angiotensin II-high salt-induced hypertension
81. Abstract 178: Effects of N-Acetyl-Seryl-Aspartyl-Lysyl-Proline in Experimental Autoimmune Mocarditis
82. Abstract 445: A Pro-Fibrotic Role of Interleukin-4 in Cardiac Remodeling and Dysfunction
83. Abstract 457: N-acetyl-seryl-aspartyl-lysyl-proline (Ac-SDKP) Prevents The Development Of Nephritis And Skin Lesions In Mouse Model Of Systemic Lupus Erythematosus (MRL/lpr)
84. Abstract 322: Protective effects of N-acetyl-seryl-aspartyl-lysyl-proline (Ac-SDKP) on renal damage in Dahl Salt-Sensitive Rats
85. Abstract 447: Ac-SDKP Suppresses TNFα-induced ICAM-1 Expression In Endothelial Cells Via Inhibition Of IκB Kinase And NF-κB Activation
86. N-acetyl-seryl-aspartyl-lysyl-proline reduces cardiac collagen cross-linking and inflammation in angiotensin II-induced hypertensive rats
87. N‐Acetyl‐Seryl‐Aspartyl‐Lysyl‐Proline (Ac‐SDKP) Prevents the Progression of Renal Dysfunction in Mice with Lupus (MRL/lpr)
88. The anti-inflammatory peptide Ac-SDKP is released from thymosin-β4 by renal meprin-α and prolyl oligopeptidase.
89. Ac-SDKP suppresses TNF-α-induced ICAM-1 expression in endothelial cells via inhibition of IκB kinase and NF-κB activation.
90. Treatment with N-acetyl-seryl-aspartyl-lysyl-proline prevents experimental autoimmune myocarditis in rats
91. Abstract 446: Deletion of Il-6 Prevents Development of Cardiac Damage and Dysfunction in Chronic Ang Ii-salt-induced Hypertension
92. Angiotensin-(1–7) upregulates central nitric oxide synthase in spontaneously hypertensive rats
93. Effect of N‐Acetyl‐Seryl‐Aspartyl‐Lysyl‐Proline (Ac‐SDKP) on intercellular adhesion molecule 1 (ICAM‐1) and proinflammatory cytokines and chemokines in experimental autoimmune myocarditis (EAM)
94. AcSDKP Reduces Collagen Crosslinking, MMP and NFKB Expression in Angiotensin II Induced Hypertension
95. N-Acetyl-Seryl-Aspartyl-Lysyl-Proline: mechanisms of renal protection in mouse model of systemic lupus erythematosus.
96. N-acetyl-seryl-aspartyl-lysyl-proline reduces cardiac collagen cross-linking and inflammation in angiotensin II-induced hypertensive rats.
97. Abstract 457.
98. Abstract 447.
99. Abstract 445.
100. Abstract 322.
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