325 results on '"Morita, Rimpei"'
Search Results
52. Role of progesterone in the prevention of murine miscarriage induced by α-galactosylceramide - New therapeutic targets of progesterone
53. Smad2 and Smad3 are redundantly essential for the suppression of iNOS synthesis in macrophages by regulating IRF3 and STAT1 pathways
54. miR126 positively regulates mast cell proliferation and cytokine production through suppressing Spred1
55. Transcription Factor Smad-Independent T Helper 17 Cell Induction by Transforming-Growth Factor-β Is Mediated by Suppression of Eomesodermin
56. SOCS1 regulates type I/type II NKT cell balance by regulating IFNγ signaling
57. Human Blood CXCR5+CD4+ T Cells Are Counterparts of T Follicular Cells and Contain Specific Subsets that Differentially Support Antibody Secretion
58. Suppression of innate immune cells in postmenopausal osteoporosis: An osteoimmunological perspective
59. Role of innate immune system and HMGB1 in preterm birth without chorioamnionitis: New perspective of heparin treatment
60. Molecular Remission at T cell level in Patients with Rheumatoid Arthritis
61. Inappropriate activation of invariant natural killer T cells and antigen‐presenting cells with the elevation of HMGB1 in preterm births without acute chorioamnionitis
62. Harmful and beneficial effects of inflammatory response on reproduction: sterile and pathogen-associated inflammation
63. Distribution of dendritic cells in the septate uterus: An immunological perspective
64. Harnessing Human Dendritic Cell Subsets to Design Novel Vaccines
65. Human plasmacytoid dendritic cells express an atrial natriuretic peptide receptor, guanylyl cyclase-A
66. Human Dendritic Cells Induce the Differentiation of Interleukin-21-Producing T Follicular Helper-like Cells through Interleukin-12
67. A T Cell-Dependent Mechanism for the Induction of Human Mucosal Homing Immunoglobulin A-Secreting Plasmablasts
68. Alcohol consumption induces murine osteoporosis by downregulation of natural killer T-like cell activity.
69. Functional Specializations of Human Epidermal Langerhans Cells and CD14+ Dermal Dendritic Cells
70. Dendritic cell subsets in health and disease
71. Harmful and beneficial effects of inflammatory response on reproduction: sterile and pathogen-associated inflammation.
72. Serum Soluble CD86, Still a Prognostic Factor in the Novel Agent Era in Multiple Myeloma Patients, Is Produced By Myeloma Cells with High CD86 Variant 3 Expression
73. Sterile inflammation in preterm birth without chorioamnionitis
74. Multi-dimensional analysis identified rheumatoid arthritis-driving pathway in human T cell
75. Inappropriate activation of invariant natural killer T cells and antigen‐presenting cells with the elevation of HMGB1 in preterm births without acute chorioamnionitis.
76. Multi-omics monitoring of drug response in rheumatoid arthritis in pursuit of molecular remission
77. Generation and application of human induced-stem cell memory T cells for adoptive immunotherapy
78. IL-23-independent induction of IL-17 from γδT cells and innate lymphoid cells promotes experimental intraocular neovascularization
79. Notch-mediated conversion of activated T cells into stem cell memory-like T cells for adoptive immunotherapy
80. Multiomic disease signatures converge to cytotoxic CD8 T cells in primary Sjögren’s syndrome
81. Multi-dimensional analysis identified rheumatoid arthritis-driving pathway in human T cell.
82. Enhanced IgG4 production by follicular helper 2 T cells and the involvement of follicular helper 1 T cells in the pathogenesis of IgG4-related disease
83. Identification of definitive serum biomarkers associated with disease activity in primary Sjögren’s syndrome
84. Interaction between a Domain of the Negative Regulator of the Ras-ERK Pathway, SPRED1 Protein, and the GTPase-activating Protein-related Domain of Neurofibromin Is Implicated in Legius Syndrome and Neurofibromatosis Type 1
85. Innate-like function of memory Th17 cells for enhancing endotoxin-induced acute lung inflammation through IL-22
86. Resolution of elevated circulating regulatory T cells by corticosteroids in patients with IgG4-related dacryoadenitis and sialoadenitis
87. Suppression of Th2 and Tfh immune reactions by Nr4a receptors in mature T reg cells
88. Spred1, a Suppressor of the Ras–ERK Pathway, Negatively Regulates Expansion and Function of Group 2 Innate Lymphoid Cells
89. Bruton’s tyrosine kinase is essential for NLRP3 inflammasome activation and contributes to ischaemic brain injury
90. ETS transcription factor ETV2 directly converts human fibroblasts into functional endothelial cells
91. Prevention of Allogeneic Cardiac Graft Rejection by Transfer of Ex Vivo Expanded Antigen-Specific Regulatory T-Cells
92. Aryl hydrocarbon receptor protects against bacterial infection by promoting macrophage survival and reactive oxygen species production
93. Aryl hydrocarbon receptor plays protective roles in ConA-induced hepatic injury by both suppressing IFN-γ expression and inducing IL-22
94. Th17 Cells Carrying TCR Recognizing Epidermal Autoantigen Induce Psoriasis-like Skin Inflammation
95. TRAF6 Is Essential for Maintenance of Regulatory T Cells That Suppress Th2 Type Autoimmunity
96. Enhanced IgG4 production by follicular helper 2 T cells and the involvement of follicular helper 1 T cells in the pathogenesis of IgG4-related disease.
97. Low dose CP-690,550 (tofacitinib), a pan-JAK inhibitor, accelerates the onset of experimental autoimmune encephalomyelitis by potentiating Th17 differentiation
98. Pyridone 6, a Pan-JAK Inhibitor, Ameliorates Allergic Skin Inflammation of NC/Nga Mice via Suppression of Th2 and Enhancement of Th17
99. Histone 3 Lysine 9 (H3K9) Methyltransferase Recruitment to the Interleukin-2 (IL-2) Promoter Is a Mechanism of Suppression of IL-2 Transcription by the Transforming Growth Factor-β-Smad Pathway
100. SOCS1 is essential for regulatory T cell functions by preventing loss of Foxp3 expression as well as IFN-γ and IL-17A production
Catalog
Books, media, physical & digital resources
Discovery Service for Jio Institute Digital Library
For full access to our library's resources, please sign in.