921 results on '"Mokhtari, Karima"'
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52. Supplementary Figure S1. from LZTR1 Mutation Mediates Oncogenesis through Stabilization of EGFR and AXL
53. Data from LZTR1 Mutation Mediates Oncogenesis through Stabilization of EGFR and AXL
54. S1: Genetic landscape of MDM2/TP53 pathway. from Preclinical Efficacy of the MDM2 Inhibitor RG7112 in MDM2-Amplified and TP53 Wild-type Glioblastomas
55. Supplementary Table 1 from Recurrent Mutations of MYD88 and TBL1XR1 in Primary Central Nervous System Lymphomas
56. S7: Additional material and methods from Preclinical Efficacy of the MDM2 Inhibitor RG7112 in MDM2-Amplified and TP53 Wild-type Glioblastomas
57. Supplementary Figure 5 from Detection, Characterization, and Inhibition of FGFR–TACC Fusions in IDH Wild-type Glioma
58. S5: MDM2, MDM4 and TP53 gene expression in the GBM cell lines of cohort #1 as determined by RT-qPCR. from Preclinical Efficacy of the MDM2 Inhibitor RG7112 in MDM2-Amplified and TP53 Wild-type Glioblastomas
59. Supplementary Figure 2 from Detection, Characterization, and Inhibition of FGFR–TACC Fusions in IDH Wild-type Glioma
60. S3: Evolution of MDM2 and TP53 status over time of 5 patients affected by MDM2-amplified GBM. from Preclinical Efficacy of the MDM2 Inhibitor RG7112 in MDM2-Amplified and TP53 Wild-type Glioblastomas
61. Supplementary Figure 1 from Detection, Characterization, and Inhibition of FGFR–TACC Fusions in IDH Wild-type Glioma
62. Supplementary Figure 6 from Detection, Characterization, and Inhibition of FGFR–TACC Fusions in IDH Wild-type Glioma
63. Supplementary Figure Legends from Detection, Characterization, and Inhibition of FGFR–TACC Fusions in IDH Wild-type Glioma
64. Supplementary Figure 3 from Detection, Characterization, and Inhibition of FGFR–TACC Fusions in IDH Wild-type Glioma
65. Supplementary Figure 4 from Detection, Characterization, and Inhibition of FGFR–TACC Fusions in IDH Wild-type Glioma
66. S6: MDM2, MDM4 and p53 protein level in the GBM cell lines of cohort #1 as determined by western blot from Preclinical Efficacy of the MDM2 Inhibitor RG7112 in MDM2-Amplified and TP53 Wild-type Glioblastomas
67. S2: Survival of GBM patients according to MDM2 status. from Preclinical Efficacy of the MDM2 Inhibitor RG7112 in MDM2-Amplified and TP53 Wild-type Glioblastomas
68. S4: MDM2, MDM4, and TP53 gene statuses in the cell lines from Preclinical Efficacy of the MDM2 Inhibitor RG7112 in MDM2-Amplified and TP53 Wild-type Glioblastomas
69. Supplementary Table 1 from Detection, Characterization, and Inhibition of FGFR–TACC Fusions in IDH Wild-type Glioma
70. CombiningMYD88 L265Pmutation detection and clonality determination on CSF cellular and cell‐free DNA improves diagnosis of primary CNS lymphoma
71. Apport de la perfusion asl dans le diagnostic des lymphomes cérébraux primitifs
72. Immunopathological characterization of ovarian teratomas associated with anti-N-methyl-D-aspartate receptor encephalitis
73. Severe hydrocephalus caused by diffuse leptomeningeal and neurocutaneous melanocytosis of antenatal onset: a clinical, pathologic, and molecular study of 2 cases
74. Golgi localization of SARS-CoV-2 spike protein and interaction with furin in cerebral COVID-19 microangiopathy: a clue to the central nervous system involvement?
75. LZTR1 mutation mediates oncogenesis through stabilization of EGFR and AXL
76. SARS-CoV-2 infection in patients with primary central nervous system lymphoma in the vaccination era
77. CSIG-01. EGFR AND AXL RECEPTOR TYROSINE KINASES DRIVE ONCOGENESIS BY LZTR1 MUTATION
78. PATH-16. THRESHOLDS OF MITOTIC ACTIVITY AND POST-SURGERY RESIDUAL VOLUME ARE INDEPENDENT PROGNOSTIC FACTORS IN ASTROCYTOMA IDH-MUTANT
79. FGFR1 actionable mutations, molecular specificities, and outcome of adult midline gliomas
80. Combining MYD88 L265P mutation detection and clonality determination on CSF cellular and cell‐free DNA improves diagnosis of primary CNS lymphoma.
81. Somatostatin receptor 2A protein expression characterizes anaplastic oligodendrogliomas with favorable outcome
82. Le Port Nador West Med : Une locomotive et un levier stratégique pour la dynamique de la région de l'Oriental
83. Prognostic impact of the 2016 WHO classification of diffuse gliomas in the French POLA cohort
84. TERT promoter mutations and rs2853669 polymorphism: prognostic impact and interactions with common alterations in glioblastomas
85. Incidence and characteristics of pseudoprogression in IDH-mutant high-grade gliomas: A POLA network study
86. Identification of growth hormone receptor as a relevant target for precision medicine in low‐EGFR expressing glioblastoma
87. Abstract 4014: Pathway based analysis of glioblastoma by multi-omics with applications in targeted therapy, prognosis and probabilistic classification
88. Case report: Central nervous system involvement of human graft versus host disease: Report of 7 cases and a review of literature
89. Tumor cells with neuronal intermediate progenitor features define a subgroup of 1p/19q co‐deleted anaplastic gliomas
90. Positron Emission Tomography/Computed Tomography Imaging of Residual Skull Base Chordoma Before Radiotherapy Using Fluoromisonidazole and Fluorodeoxyglucose: Potential Consequences for Dose Painting
91. Differential gene methylation in paired glioblastomas suggests a role of immune response pathways in tumor progression
92. Patterns of relapse and growth kinetics of surgery- and radiation-refractory meningiomas
93. VEGFA SNP rs2010963 is associated with vascular toxicity in recurrent glioblastomas and longer response to bevacizumab
94. Rosette‐forming glioneuronal tumours are midline, FGFR1‐mutated tumours
95. Severity, timeline, and management of complications after stereotactic brain biopsy
96. Temozolomide after radiotherapy in recurrent “low grade” diffuse brainstem glioma in adults
97. Vascularite cérébrale primitive à présentation pseudo-tumorale (VCPT) : une série de 4 cas
98. The wide spectrum of COVID-19 neuropsychiatric complications within a multidisciplinary centre
99. Growth Hormone Receptor (GHR) is overexpressed in low EGFR expressing glioblastoma and promotes tumor growth
100. Pathway based analysis of glioblastoma by multi-omics with applications in targeted therapy, prognosis and probabilistic classification
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